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Artigo de periodico
Temozolomide resistance mechanisms: unveiling the role of translesion DNA polymerase kappa in glioblastoma spheroids in vitro (2024)
Ribeiro, Luis Diego ; Latância, Marcela Teatin ; Souza, Izadora de ; Ariwoola, Abu-Bakr Adetayo ; Mendes, Davi ; Rocha, Clarissa Ribeiro Reily ; Lengert, André van Helvoort ; Menck, Carlos Frederico Martins
Source: Bioscience Reports: molecular and cellular biology of the cell surface. Unidades: ICB, BIOTECNOLOGIA
Subjects: MICROBIOLOGIA, ANTINEOPLÁSICOS, NEOPLASIAS CEREBRAIS, GLIOMA, DNA POLIMERASES, APOPTOSE, DANO AO DNA, PROLIFERAÇÃO CELULAR
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ABNT
RIBEIRO, Luis Diego et al. Temozolomide resistance mechanisms: unveiling the role of translesion DNA polymerase kappa in glioblastoma spheroids in vitro. Bioscience Reports: molecular and cellular biology of the cell surface, v. 44, n. 5, p. 18 , 2024Tradução . . Disponível em: https://doi.org/10.1042/BSR20230667. Acesso em: 10 jul. 2024.
APA
Ribeiro, L. D., Latância, M. T., Souza, I. de, Ariwoola, A. -B. A., Mendes, D., Rocha, C. R. R., et al. (2024). Temozolomide resistance mechanisms: unveiling the role of translesion DNA polymerase kappa in glioblastoma spheroids in vitro. Bioscience Reports: molecular and cellular biology of the cell surface, 44( 5), 18 . doi:10.1042/BSR20230667
NLM
Ribeiro LD, Latância MT, Souza I de, Ariwoola A-BA, Mendes D, Rocha CRR, Lengert A van H, Menck CFM. Temozolomide resistance mechanisms: unveiling the role of translesion DNA polymerase kappa in glioblastoma spheroids in vitro [Internet]. Bioscience Reports: molecular and cellular biology of the cell surface. 2024 ; 44( 5): 18 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.1042/BSR20230667
Vancouver
Ribeiro LD, Latância MT, Souza I de, Ariwoola A-BA, Mendes D, Rocha CRR, Lengert A van H, Menck CFM. Temozolomide resistance mechanisms: unveiling the role of translesion DNA polymerase kappa in glioblastoma spheroids in vitro [Internet]. Bioscience Reports: molecular and cellular biology of the cell surface. 2024 ; 44( 5): 18 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.1042/BSR20230667
Artigo de periodico
Trypanosoma cruzi infection changes the chromatin proteome profile of infected human cells (2023)
Florentino, Pilar Tavares Veras ; Vitorino, Francisca Nathália de Luna ; Mendes, D.; Cunha, Julia Pinheiro Chagas da ; Menck, Carlos Frederico Martins
Source: Journal of Proteomics. Unidade: ICB
Subjects: MICROBIOLOGIA, CROMATINA, DANO AO DNA, TRYPANOSOMA CRUZI, ANIMAIS PARASITOS, DOENÇA DE CHAGAS, APOPTOSE, PROTEÔMICA, DNA
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FLORENTINO, Pilar Tavares Veras et al. Trypanosoma cruzi infection changes the chromatin proteome profile of infected human cells. Journal of Proteomics, v. 272, p. 1-11, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.jprot.2022.104773. Acesso em: 10 jul. 2024.
APA
Florentino, P. T. V., Vitorino, F. N. de L., Mendes, D., Cunha, J. P. C. da, & Menck, C. F. M. (2023). Trypanosoma cruzi infection changes the chromatin proteome profile of infected human cells. Journal of Proteomics, 272, 1-11. doi:10.1016/j.jprot.2022.104773
NLM
Florentino PTV, Vitorino FN de L, Mendes D, Cunha JPC da, Menck CFM. Trypanosoma cruzi infection changes the chromatin proteome profile of infected human cells [Internet]. Journal of Proteomics. 2023 ; 272 1-11.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.jprot.2022.104773
Vancouver
Florentino PTV, Vitorino FN de L, Mendes D, Cunha JPC da, Menck CFM. Trypanosoma cruzi infection changes the chromatin proteome profile of infected human cells [Internet]. Journal of Proteomics. 2023 ; 272 1-11.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.jprot.2022.104773
Artigo de periodico
Cooperation and interplay between base and nucleotide excision repair pathways: from DNA lesions to proteins (2020)
Kumar, Namrata ; Moreno, Natália Cestari ; Feltes, Bruno C. ; Menck, Carlos Frederico Martins ; Houten, Bennett Van
Source: Genetics and Molecular Biology. Unidades: ICB, IQ
Subjects: MICROBIOLOGIA, DANO AO DNA, MUTAGÊNESE, MUTAÇÃO GENÉTICA, NUCLEOTÍDEOS, CARCINOGÊNESE, ESTRESSE OXIDATIVO, APOPTOSE, GENOMAS
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KUMAR, Namrata et al. Cooperation and interplay between base and nucleotide excision repair pathways: from DNA lesions to proteins. Genetics and Molecular Biology, v. 43, n. 1, p. 14 , 2020Tradução . . Disponível em: https://doi.org/10.1590/1678-4685-gmb-2019-0104. Acesso em: 10 jul. 2024.
APA
Kumar, N., Moreno, N. C., Feltes, B. C., Menck, C. F. M., & Houten, B. V. (2020). Cooperation and interplay between base and nucleotide excision repair pathways: from DNA lesions to proteins. Genetics and Molecular Biology, 43( 1), 14 . doi:10.1590/1678-4685-gmb-2019-0104
NLM
Kumar N, Moreno NC, Feltes BC, Menck CFM, Houten BV. Cooperation and interplay between base and nucleotide excision repair pathways: from DNA lesions to proteins [Internet]. Genetics and Molecular Biology. 2020 ; 43( 1): 14 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.1590/1678-4685-gmb-2019-0104
Vancouver
Kumar N, Moreno NC, Feltes BC, Menck CFM, Houten BV. Cooperation and interplay between base and nucleotide excision repair pathways: from DNA lesions to proteins [Internet]. Genetics and Molecular Biology. 2020 ; 43( 1): 14 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.1590/1678-4685-gmb-2019-0104
Artigo de periodico
Melanopsin mediates UVA-dependent modulation of proliferation, pigmentation, apoptosis, and molecular clock in normal and malignant melanocytes (2020)
Assis, Leonardo Vinícius Monteiro de ; Mendes, Davi ; Silva, Matheus Molina ; Kinker, Gabriela Sarti ; Lima, Isabella Pereira ; Borges, Maria Nathália de Carvalho Magalhães Moraes Figueira ; Menck, Carlos Frederico Martins ; Castrucci, Ana Maria de Lauro
Source: Biochimica et Biophysica Acta. Molecular Cell Research. Unidades: ICB, IB
Subjects: MICROBIOLOGIA, APOPTOSE, RITMOS BIOLÓGICOS, PIGMENTOS, MELANINAS, MELANOMA, CICLO CELULAR, RADIAÇÃO ULTRAVIOLETA, PELE, CÉLULAS CULTIVADAS DE TUMOR, NEOPLASIAS CUTÂNEAS
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ASSIS, Leonardo Vinícius Monteiro de et al. Melanopsin mediates UVA-dependent modulation of proliferation, pigmentation, apoptosis, and molecular clock in normal and malignant melanocytes. Biochimica et Biophysica Acta. Molecular Cell Research, v. 1867, n. 10, p. 10 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.bbamcr.2020.118789. Acesso em: 10 jul. 2024.
APA
Assis, L. V. M. de, Mendes, D., Silva, M. M., Kinker, G. S., Lima, I. P., Borges, M. N. de C. M. M. F., et al. (2020). Melanopsin mediates UVA-dependent modulation of proliferation, pigmentation, apoptosis, and molecular clock in normal and malignant melanocytes. Biochimica et Biophysica Acta. Molecular Cell Research, 1867( 10), 10 . doi:10.1016/j.bbamcr.2020.118789
NLM
Assis LVM de, Mendes D, Silva MM, Kinker GS, Lima IP, Borges MN de CMMF, Menck CFM, Castrucci AM de L. Melanopsin mediates UVA-dependent modulation of proliferation, pigmentation, apoptosis, and molecular clock in normal and malignant melanocytes [Internet]. Biochimica et Biophysica Acta. Molecular Cell Research. 2020 ; 1867( 10): 10 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.bbamcr.2020.118789
Vancouver
Assis LVM de, Mendes D, Silva MM, Kinker GS, Lima IP, Borges MN de CMMF, Menck CFM, Castrucci AM de L. Melanopsin mediates UVA-dependent modulation of proliferation, pigmentation, apoptosis, and molecular clock in normal and malignant melanocytes [Internet]. Biochimica et Biophysica Acta. Molecular Cell Research. 2020 ; 1867( 10): 10 .[citado 2024 jul. 10 ] Available from: https://doi.org/10.1016/j.bbamcr.2020.118789
Artigo de periodico
ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways (2015)
Andrade-Lima, Leonardo Carmo de; Andrade, Luciana N.; Menck, Carlos Frederico Martins
Source: Journal of Cell Science. Unidade: ICB
Subjects: MICROBIOLOGIA, APOPTOSE
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ANDRADE-LIMA, Leonardo Carmo de e ANDRADE, Luciana N. e MENCK, Carlos Frederico Martins. ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways. Journal of Cell Science, v. 28, n. 1, p. 150-159, 2015Tradução . . Disponível em: https://doi.org/10.1242/jcs.161596. Acesso em: 10 jul. 2024.
APA
Andrade-Lima, L. C. de, Andrade, L. N., & Menck, C. F. M. (2015). ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways. Journal of Cell Science, 28( 1), 150-159. doi:10.1242/jcs.161596
NLM
Andrade-Lima LC de, Andrade LN, Menck CFM. ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways [Internet]. Journal of Cell Science. 2015 ; 28( 1): 150-159.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1242/jcs.161596
Vancouver
Andrade-Lima LC de, Andrade LN, Menck CFM. ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways [Internet]. Journal of Cell Science. 2015 ; 28( 1): 150-159.[citado 2024 jul. 10 ] Available from: https://doi.org/10.1242/jcs.161596
Trabalho de evento-resumo periodico
The effect of hyperglycemia on apoptosis in peripheral blood monuclear cells of newborns: an in vitro study (2013)
Moreli, J. B.; Correa-Silva, S.; Rocha, C. R.; Fagundes, D. L. G.; Damasceno, D. C.; Rudge, M. V. C.; Calderon, I. M. P.; Menck, Carlos Frederico Martins ; Bevilacqua, Estela Maris Andrade Forell
Conference titles: Latin American Symposium mon Maternal Fetal Interaction and Placenta (SLIMP). Unidade: ICB
Subjects: HIPERGLICEMIA, APOPTOSE
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ABNT
MORELI, J. B. et al. The effect of hyperglycemia on apoptosis in peripheral blood monuclear cells of newborns: an in vitro study. . Foz do Iguaçu: LASTRI. . Acesso em: 10 jul. 2024. , 2013
APA
Moreli, J. B., Correa-Silva, S., Rocha, C. R., Fagundes, D. L. G., Damasceno, D. C., Rudge, M. V. C., et al. (2013). The effect of hyperglycemia on apoptosis in peripheral blood monuclear cells of newborns: an in vitro study. Foz do Iguaçu: LASTRI.
NLM
Moreli JB, Correa-Silva S, Rocha CR, Fagundes DLG, Damasceno DC, Rudge MVC, Calderon IMP, Menck CFM, Bevilacqua EMAF. The effect of hyperglycemia on apoptosis in peripheral blood monuclear cells of newborns: an in vitro study. 2013 ; 34[citado 2024 jul. 10 ]
Vancouver
Moreli JB, Correa-Silva S, Rocha CR, Fagundes DLG, Damasceno DC, Rudge MVC, Calderon IMP, Menck CFM, Bevilacqua EMAF. The effect of hyperglycemia on apoptosis in peripheral blood monuclear cells of newborns: an in vitro study. 2013 ; 34[citado 2024 jul. 10 ]
Trabalho de evento-resumo
RhoA GTPase-deficient human tumor cells have increased apoptosis and an inefficient repair of lesions induced by ultraviolet radiation (2012)
Espinha, Gisele T. da S; Rocha, C. R. R; Menck, Carlos Frederico Martins ; Forti, Fabio Luis
Source: Programa. Conference titles: Congresso Brasileiro de Genética. Unidades: ICB, IQ
Subjects: APOPTOSE, RADIAÇÃO ULTRAVIOLETA
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ESPINHA, Gisele T. da S et al. RhoA GTPase-deficient human tumor cells have increased apoptosis and an inefficient repair of lesions induced by ultraviolet radiation. 2012, Anais.. Ribeirão Preto: Sociedade Brasileira de Genética, 2012. . Acesso em: 10 jul. 2024.
APA
Espinha, G. T. da S., Rocha, C. R. R., Menck, C. F. M., & Forti, F. L. (2012). RhoA GTPase-deficient human tumor cells have increased apoptosis and an inefficient repair of lesions induced by ultraviolet radiation. In Programa. Ribeirão Preto: Sociedade Brasileira de Genética.
NLM
Espinha GT da S, Rocha CRR, Menck CFM, Forti FL. RhoA GTPase-deficient human tumor cells have increased apoptosis and an inefficient repair of lesions induced by ultraviolet radiation. Programa. 2012 ;[citado 2024 jul. 10 ]
Vancouver
Espinha GT da S, Rocha CRR, Menck CFM, Forti FL. RhoA GTPase-deficient human tumor cells have increased apoptosis and an inefficient repair of lesions induced by ultraviolet radiation. Programa. 2012 ;[citado 2024 jul. 10 ]

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