Filtros : "Pharmacological Research" Limpar

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  • Source: Pharmacological Research. Unidade: FMRP

    Subjects: DOR, COMPLICAÇÕES PÓS-OPERATÓRIAS, QUIMIOCINAS, NEUTRÓFILOS, QUIMIOTAXIA

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    • ABNT

      LOPES, Alexandre H. et al. DF2755A, a novel non-competitive allosteric inhibitor of CXCR1/2, reduces inflammatory and post-operative pain. Pharmacological Research, v. 103, p. 69-79, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2015.11.005. Acesso em: 19 abr. 2024.
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      Lopes, A. H., Brandolini, L., Aramini, A., Bianchini, G., Silva, R. L., Zaperlon, A. C., et al. (2016). DF2755A, a novel non-competitive allosteric inhibitor of CXCR1/2, reduces inflammatory and post-operative pain. Pharmacological Research, 103, 69-79. doi:10.1016/j.phrs.2015.11.005
    • NLM

      Lopes AH, Brandolini L, Aramini A, Bianchini G, Silva RL, Zaperlon AC, Verri Júnior WA, Alves Filho JCF, Cunha F de Q, Teixeira MM, Allegretti M, Cunha TM. DF2755A, a novel non-competitive allosteric inhibitor of CXCR1/2, reduces inflammatory and post-operative pain [Internet]. Pharmacological Research. 2016 ; 103 69-79.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2015.11.005
    • Vancouver

      Lopes AH, Brandolini L, Aramini A, Bianchini G, Silva RL, Zaperlon AC, Verri Júnior WA, Alves Filho JCF, Cunha F de Q, Teixeira MM, Allegretti M, Cunha TM. DF2755A, a novel non-competitive allosteric inhibitor of CXCR1/2, reduces inflammatory and post-operative pain [Internet]. Pharmacological Research. 2016 ; 103 69-79.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2015.11.005
  • Source: Pharmacological Research. Unidade: FMRP

    Subjects: ANTI-INFLAMATÓRIOS, LEUCÓCITOS, INFLAMAÇÃO, QUIMIOCINAS, TRANSDUÇÃO DE SINAL CELULAR

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      PERES, Raphael S. et al. Pharmacological opportunities to control inflammatory diseases through inhibition of the leukocyte recruitment. Pharmacological Research, v. 112, p. 37-48, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2016.01.015. Acesso em: 19 abr. 2024.
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      Peres, R. S., Menezes, G. B., Teixeira, M. M., & Cunha, F. de Q. (2016). Pharmacological opportunities to control inflammatory diseases through inhibition of the leukocyte recruitment. Pharmacological Research, 112, 37-48. doi:10.1016/j.phrs.2016.01.015
    • NLM

      Peres RS, Menezes GB, Teixeira MM, Cunha F de Q. Pharmacological opportunities to control inflammatory diseases through inhibition of the leukocyte recruitment [Internet]. Pharmacological Research. 2016 ; 112 37-48.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2016.01.015
    • Vancouver

      Peres RS, Menezes GB, Teixeira MM, Cunha F de Q. Pharmacological opportunities to control inflammatory diseases through inhibition of the leukocyte recruitment [Internet]. Pharmacological Research. 2016 ; 112 37-48.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2016.01.015
  • Source: Pharmacological Research. Unidade: FMRP

    Subjects: DOENÇAS CEREBRAIS, TRANSTORNOS MENTAIS, FARMACOTERAPIA, CANABINOIDES

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      CAMPOS, Alline Cristina de et al. Cannabidiol, neuroprotection and neuropsychiatric disorders. Pharmacological Research, v. 112, p. 119-127, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2016.01.033. Acesso em: 19 abr. 2024.
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      Campos, A. C. de, Fogaça, M. V., Sonego, A. B., & Guimarães, F. S. (2016). Cannabidiol, neuroprotection and neuropsychiatric disorders. Pharmacological Research, 112, 119-127. doi:10.1016/j.phrs.2016.01.033
    • NLM

      Campos AC de, Fogaça MV, Sonego AB, Guimarães FS. Cannabidiol, neuroprotection and neuropsychiatric disorders [Internet]. Pharmacological Research. 2016 ; 112 119-127.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2016.01.033
    • Vancouver

      Campos AC de, Fogaça MV, Sonego AB, Guimarães FS. Cannabidiol, neuroprotection and neuropsychiatric disorders [Internet]. Pharmacological Research. 2016 ; 112 119-127.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2016.01.033
  • Source: Pharmacological Research. Unidade: FMRP

    Subjects: ANALGÉSICOS, INFLAMAÇÃO, TRANSDUÇÃO DE SINAL CELULAR, DOR CRÔNICA

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      QUADROS, Andreza U. e CUNHA, Thiago Mattar. C5a and pain development: an old molecule, a new target. Pharmacological Research, v. 112, p. 58-67, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2016.02.004. Acesso em: 19 abr. 2024.
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      Quadros, A. U., & Cunha, T. M. (2016). C5a and pain development: an old molecule, a new target. Pharmacological Research, 112, 58-67. doi:10.1016/j.phrs.2016.02.004
    • NLM

      Quadros AU, Cunha TM. C5a and pain development: an old molecule, a new target [Internet]. Pharmacological Research. 2016 ; 112 58-67.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2016.02.004
    • Vancouver

      Quadros AU, Cunha TM. C5a and pain development: an old molecule, a new target [Internet]. Pharmacological Research. 2016 ; 112 58-67.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2016.02.004
  • Source: Pharmacological Research. Unidade: FCF

    Subjects: METALOPROTEINASES, MELANOMA

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      SANDRI, Silvana et al. Vemurafenib resistance increases melanoma invasiveness and modulates the tumor microenvironment by MMP-2 upregulation. Pharmacological Research, v. 111, p. 523-533, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2016.07.017. Acesso em: 19 abr. 2024.
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      Sandri, S., Flores, F. F., Tiago, M., Pennacchi, P. C., Massaro, R. R., Fernandes, D. K. A., et al. (2016). Vemurafenib resistance increases melanoma invasiveness and modulates the tumor microenvironment by MMP-2 upregulation. Pharmacological Research, 111, 523-533. doi:10.1016/j.phrs.2016.07.017
    • NLM

      Sandri S, Flores FF, Tiago M, Pennacchi PC, Massaro RR, Fernandes DKA, Berardinelli GN, Evangelista AF, Vazquez V de L, Reis RM, Maria-Engler SS. Vemurafenib resistance increases melanoma invasiveness and modulates the tumor microenvironment by MMP-2 upregulation [Internet]. Pharmacological Research. 2016 ; 111 523-533.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2016.07.017
    • Vancouver

      Sandri S, Flores FF, Tiago M, Pennacchi PC, Massaro RR, Fernandes DKA, Berardinelli GN, Evangelista AF, Vazquez V de L, Reis RM, Maria-Engler SS. Vemurafenib resistance increases melanoma invasiveness and modulates the tumor microenvironment by MMP-2 upregulation [Internet]. Pharmacological Research. 2016 ; 111 523-533.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2016.07.017
  • Source: Pharmacological Research. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      GOMES, Luciana R. e VESSONI, Alexandre T. e MENCK, Carlos Frederico Martins. Microenvironment and autophagy cross-talk: implications in cancer therapy. Pharmacological Research, v. 107, p. 300-307, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2016.03.031. Acesso em: 19 abr. 2024.
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      Gomes, L. R., Vessoni, A. T., & Menck, C. F. M. (2016). Microenvironment and autophagy cross-talk: implications in cancer therapy. Pharmacological Research, 107, 300-307. doi:10.1016/j.phrs.2016.03.031
    • NLM

      Gomes LR, Vessoni AT, Menck CFM. Microenvironment and autophagy cross-talk: implications in cancer therapy [Internet]. Pharmacological Research. 2016 ; 107 300-307.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2016.03.031
    • Vancouver

      Gomes LR, Vessoni AT, Menck CFM. Microenvironment and autophagy cross-talk: implications in cancer therapy [Internet]. Pharmacological Research. 2016 ; 107 300-307.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2016.03.031
  • Source: Pharmacological Research. Unidade: ICB

    Assunto: FARMACOLOGIA

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      COAVOY-SANCHEZ, S. A. et al. Hydrogen sulfide donors alleviate itch secondary to the activation of type-2 protease activated receptors (PAR-2) in mice. Pharmacological Research, v. 113, p. 686-694, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2016.09.030. Acesso em: 19 abr. 2024.
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      Coavoy-Sanchez, S. A., Rodrigues, L., Teixeira, S. A., Soares, A. G., Torregrossa, R., Wood, M. E., et al. (2016). Hydrogen sulfide donors alleviate itch secondary to the activation of type-2 protease activated receptors (PAR-2) in mice. Pharmacological Research, 113, 686-694. doi:10.1016/j.phrs.2016.09.030
    • NLM

      Coavoy-Sanchez SA, Rodrigues L, Teixeira SA, Soares AG, Torregrossa R, Wood ME, Whiteman M, Costa SKP, Muscará MN. Hydrogen sulfide donors alleviate itch secondary to the activation of type-2 protease activated receptors (PAR-2) in mice [Internet]. Pharmacological Research. 2016 ; 113 686-694.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2016.09.030
    • Vancouver

      Coavoy-Sanchez SA, Rodrigues L, Teixeira SA, Soares AG, Torregrossa R, Wood ME, Whiteman M, Costa SKP, Muscará MN. Hydrogen sulfide donors alleviate itch secondary to the activation of type-2 protease activated receptors (PAR-2) in mice [Internet]. Pharmacological Research. 2016 ; 113 686-694.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2016.09.030
  • Source: Pharmacological Research. Unidade: FMRP

    Subjects: AORTA, ANGIOTENSINA II, HIPERTENSÃO, ÓXIDO NÍTRICO

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      WYNNE, Brandi M. et al. Aorta from angiotensin II hypertensive mice exhibit preserved nitroxyl anion mediated relaxation responses. Pharmacological Research, v. 65, n. 1, p. 41-47, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2011.07.002. Acesso em: 19 abr. 2024.
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      Wynne, B. M., Labazi, H., Tostes, R. de C. A., & Webb, R. C. (2012). Aorta from angiotensin II hypertensive mice exhibit preserved nitroxyl anion mediated relaxation responses. Pharmacological Research, 65( 1), 41-47. doi:10.1016/j.phrs.2011.07.002
    • NLM

      Wynne BM, Labazi H, Tostes R de CA, Webb RC. Aorta from angiotensin II hypertensive mice exhibit preserved nitroxyl anion mediated relaxation responses [Internet]. Pharmacological Research. 2012 ; 65( 1): 41-47.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2011.07.002
    • Vancouver

      Wynne BM, Labazi H, Tostes R de CA, Webb RC. Aorta from angiotensin II hypertensive mice exhibit preserved nitroxyl anion mediated relaxation responses [Internet]. Pharmacological Research. 2012 ; 65( 1): 41-47.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2011.07.002
  • Source: Pharmacological Research. Unidade: FMRP

    Subjects: HIPERTENSÃO, NUCLEOTÍDEOS, FÁRMACOS (SISTEMA CARDIOVASCULAR)

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      MATSUMOTO, Takayuki e TOSTES, Rita de Cassia Aleixo e WEBB, R. Clinton. Alterations in vaso constrictor responses to the endothelium-derived contracting factor uridine adenosine tetraphosphate are region specific in DOCA-salt hypertensive rats. Pharmacological Research, v. 65, n. 1, p. 81-90, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2011.09.005. Acesso em: 19 abr. 2024.
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      Matsumoto, T., Tostes, R. de C. A., & Webb, R. C. (2012). Alterations in vaso constrictor responses to the endothelium-derived contracting factor uridine adenosine tetraphosphate are region specific in DOCA-salt hypertensive rats. Pharmacological Research, 65( 1), 81-90. doi:10.1016/j.phrs.2011.09.005
    • NLM

      Matsumoto T, Tostes R de CA, Webb RC. Alterations in vaso constrictor responses to the endothelium-derived contracting factor uridine adenosine tetraphosphate are region specific in DOCA-salt hypertensive rats [Internet]. Pharmacological Research. 2012 ; 65( 1): 81-90.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2011.09.005
    • Vancouver

      Matsumoto T, Tostes R de CA, Webb RC. Alterations in vaso constrictor responses to the endothelium-derived contracting factor uridine adenosine tetraphosphate are region specific in DOCA-salt hypertensive rats [Internet]. Pharmacological Research. 2012 ; 65( 1): 81-90.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2011.09.005
  • Source: Pharmacological Research. Unidade: FMRP

    Subjects: VASODILATAÇÃO, ADRENORECEPTORES, ARTÉRIAS, HIPERTENSÃO

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      MATSUMOTO, Takayuki et al. Impaired β-adrenoceptor-induced relaxation in small mesenteric arteries from DOCA-salt hypertensive rats is due to reduced Kca channel activity. Pharmacological Research, v. 65, n. 5, p. 537-545, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2012.02.004. Acesso em: 19 abr. 2024.
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      Matsumoto, T., Szasz, T., Tostes, R. de C. A., & Webb, R. C. (2012). Impaired β-adrenoceptor-induced relaxation in small mesenteric arteries from DOCA-salt hypertensive rats is due to reduced Kca channel activity. Pharmacological Research, 65( 5), 537-545. doi:10.1016/j.phrs.2012.02.004
    • NLM

      Matsumoto T, Szasz T, Tostes R de CA, Webb RC. Impaired β-adrenoceptor-induced relaxation in small mesenteric arteries from DOCA-salt hypertensive rats is due to reduced Kca channel activity [Internet]. Pharmacological Research. 2012 ; 65( 5): 537-545.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2012.02.004
    • Vancouver

      Matsumoto T, Szasz T, Tostes R de CA, Webb RC. Impaired β-adrenoceptor-induced relaxation in small mesenteric arteries from DOCA-salt hypertensive rats is due to reduced Kca channel activity [Internet]. Pharmacological Research. 2012 ; 65( 5): 537-545.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2012.02.004
  • Source: Pharmacological Research. Unidades: FMRP, FORP

    Subjects: HIPERTENSÃO, METALOPROTEINASES

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      CASTRO, Michele M. e TANUS-SANTOS, José Eduardo e GERLACH, Raquel Fernanda. Matrix metalloproteinases: targets for doxycycline to prevent the vascular alterations of hypertension. Pharmacological Research, v. 64, n. 6, p. 567-572, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2011.04.002. Acesso em: 19 abr. 2024.
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      Castro, M. M., Tanus-Santos, J. E., & Gerlach, R. F. (2011). Matrix metalloproteinases: targets for doxycycline to prevent the vascular alterations of hypertension. Pharmacological Research, 64( 6), 567-572. doi:10.1016/j.phrs.2011.04.002
    • NLM

      Castro MM, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinases: targets for doxycycline to prevent the vascular alterations of hypertension [Internet]. Pharmacological Research. 2011 ; 64( 6): 567-572.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2011.04.002
    • Vancouver

      Castro MM, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinases: targets for doxycycline to prevent the vascular alterations of hypertension [Internet]. Pharmacological Research. 2011 ; 64( 6): 567-572.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2011.04.002
  • Source: Pharmacological Research. Unidades: ICB, FCFRP

    Subjects: PLANTAS MEDICINAIS (ESTUDO;PROPRIEDADES), ALERGIA E IMUNOLOGIA, LEUCÓCITOS

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      ROGERIO, Alexandre de Paula e NUNES, Anderson de Sá e FACCIOLI, Lucia Helena. The activity of medicinal plants and secondary metabolites on eosinophilic. Pharmacological Research, v. 62, n. 4, p. 298-307, 2010Tradução . . Acesso em: 19 abr. 2024.
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      Rogerio, A. de P., Nunes, A. de S., & Faccioli, L. H. (2010). The activity of medicinal plants and secondary metabolites on eosinophilic. Pharmacological Research, 62( 4), 298-307.
    • NLM

      Rogerio A de P, Nunes A de S, Faccioli LH. The activity of medicinal plants and secondary metabolites on eosinophilic. Pharmacological Research. 2010 ; 62( 4): 298-307.[citado 2024 abr. 19 ]
    • Vancouver

      Rogerio A de P, Nunes A de S, Faccioli LH. The activity of medicinal plants and secondary metabolites on eosinophilic. Pharmacological Research. 2010 ; 62( 4): 298-307.[citado 2024 abr. 19 ]
  • Source: Pharmacological Research. Unidade: FMRP

    Subjects: CANNABIS, NEUROTRANSMISSORES, SEROTONINA

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      ALVES, Fernando H. F. et al. Cannabidiol injected into the bed nucleus of the stria terminalis modulates baroreflex activity through 5-HT1A receptors. Pharmacological Research, v. 62, n. 3, p. 228-236, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2010.05.003. Acesso em: 19 abr. 2024.
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      Alves, F. H. F., Crestani, C. C., Gomes, F. V., Guimarães, F. S., Corrêa, F. M. de A., & Resstel, L. B. M. (2010). Cannabidiol injected into the bed nucleus of the stria terminalis modulates baroreflex activity through 5-HT1A receptors. Pharmacological Research, 62( 3), 228-236. doi:10.1016/j.phrs.2010.05.003
    • NLM

      Alves FHF, Crestani CC, Gomes FV, Guimarães FS, Corrêa FM de A, Resstel LBM. Cannabidiol injected into the bed nucleus of the stria terminalis modulates baroreflex activity through 5-HT1A receptors [Internet]. Pharmacological Research. 2010 ; 62( 3): 228-236.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2010.05.003
    • Vancouver

      Alves FHF, Crestani CC, Gomes FV, Guimarães FS, Corrêa FM de A, Resstel LBM. Cannabidiol injected into the bed nucleus of the stria terminalis modulates baroreflex activity through 5-HT1A receptors [Internet]. Pharmacological Research. 2010 ; 62( 3): 228-236.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2010.05.003
  • Source: Pharmacological Research. Unidades: FMRP, EERP, FCFRP

    Subjects: ANTIOXIDANTES, EXTRATOS (FORMAS FARMACÊUTICAS), PLANTAS

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      ANDRADE, Claudia Roberta de et al. Total stenosis triggers compensatory responsiveness of carotid and basilar arteries to endothelin-1 and phenylephrine. Pharmacological Research, v. 57, n. 1, p. 32-42, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2007.10.009. Acesso em: 19 abr. 2024.
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      Andrade, C. R. de, Tirapelli, C. R., Corrêa, F. M. de A., Ramalho, L. N. Z., & Oliveira, A. M. de. (2008). Total stenosis triggers compensatory responsiveness of carotid and basilar arteries to endothelin-1 and phenylephrine. Pharmacological Research, 57( 1), 32-42. doi:10.1016/j.phrs.2007.10.009
    • NLM

      Andrade CR de, Tirapelli CR, Corrêa FM de A, Ramalho LNZ, Oliveira AM de. Total stenosis triggers compensatory responsiveness of carotid and basilar arteries to endothelin-1 and phenylephrine [Internet]. Pharmacological Research. 2008 ; 57( 1): 32-42.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2007.10.009
    • Vancouver

      Andrade CR de, Tirapelli CR, Corrêa FM de A, Ramalho LNZ, Oliveira AM de. Total stenosis triggers compensatory responsiveness of carotid and basilar arteries to endothelin-1 and phenylephrine [Internet]. Pharmacological Research. 2008 ; 57( 1): 32-42.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2007.10.009
  • Source: Pharmacological Research. Unidade: FCFRP

    Subjects: EXTRATOS (FORMAS FARMACÊUTICAS), PLANTAS, ANTIOXIDANTES, FERRO

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      PARDO-ANDREU, Gilberto L. et al. Interaction of Vimang (Mangifera indica L. extract) with Fe(III) improves its antioxidant and cytoprotecting activity. Pharmacological Research, n. 5, p. 389-395, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2006.08.001. Acesso em: 19 abr. 2024.
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      Pardo-Andreu, G. L., Sánchez-Baldoquín, C., Ávila González, R., Yamamoto, E. T. S., Revilla, A., Uyemura, S. A., et al. (2006). Interaction of Vimang (Mangifera indica L. extract) with Fe(III) improves its antioxidant and cytoprotecting activity. Pharmacological Research, ( 5), 389-395. doi:10.1016/j.phrs.2006.08.001
    • NLM

      Pardo-Andreu GL, Sánchez-Baldoquín C, Ávila González R, Yamamoto ETS, Revilla A, Uyemura SA, Naal Z, Delgado R, Curti C. Interaction of Vimang (Mangifera indica L. extract) with Fe(III) improves its antioxidant and cytoprotecting activity [Internet]. Pharmacological Research. 2006 ;( 5): 389-395.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2006.08.001
    • Vancouver

      Pardo-Andreu GL, Sánchez-Baldoquín C, Ávila González R, Yamamoto ETS, Revilla A, Uyemura SA, Naal Z, Delgado R, Curti C. Interaction of Vimang (Mangifera indica L. extract) with Fe(III) improves its antioxidant and cytoprotecting activity [Internet]. Pharmacological Research. 2006 ;( 5): 389-395.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2006.08.001
  • Source: Pharmacological Research. Unidade: FCFRP

    Subjects: METABOLISMO, BIOENERGÉTICA

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      ANDREU, Gilberto Pardo et al. Mangifera indica L. extract (Vimang) inhibits 'Fe POT 2+'-citrate-induced lipoperoxidation in isolated rat liver mitochondria. Pharmacological Research, v. 51, p. 427-435, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2004.11.002. Acesso em: 19 abr. 2024.
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      Andreu, G. P., Delgado, R., Velho, J., Inada, N. M., Curti, C., & Vercesi, A. E. (2005). Mangifera indica L. extract (Vimang) inhibits 'Fe POT 2+'-citrate-induced lipoperoxidation in isolated rat liver mitochondria. Pharmacological Research, 51, 427-435. doi:10.1016/j.phrs.2004.11.002
    • NLM

      Andreu GP, Delgado R, Velho J, Inada NM, Curti C, Vercesi AE. Mangifera indica L. extract (Vimang) inhibits 'Fe POT 2+'-citrate-induced lipoperoxidation in isolated rat liver mitochondria [Internet]. Pharmacological Research. 2005 ;51 427-435.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2004.11.002
    • Vancouver

      Andreu GP, Delgado R, Velho J, Inada NM, Curti C, Vercesi AE. Mangifera indica L. extract (Vimang) inhibits 'Fe POT 2+'-citrate-induced lipoperoxidation in isolated rat liver mitochondria [Internet]. Pharmacological Research. 2005 ;51 427-435.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2004.11.002
  • Source: Pharmacological Research. Unidades: FCFRP, FMRP

    Subjects: TRANSTORNO DO PÂNICO, FARMACOLOGIA

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      JORGE, Salim Demétrio et al. Effects of sibutramine on anxiety-related behaviours in rats. Pharmacological Research, v. 50, p. 517-522, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2004.05.003. Acesso em: 19 abr. 2024.
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      Jorge, S. D., Pobbe, R. L. H., Soares, V. de P., Oliveira, A. M. de, & Zangrossi Júnior, H. (2004). Effects of sibutramine on anxiety-related behaviours in rats. Pharmacological Research, 50, 517-522. doi:10.1016/j.phrs.2004.05.003
    • NLM

      Jorge SD, Pobbe RLH, Soares V de P, Oliveira AM de, Zangrossi Júnior H. Effects of sibutramine on anxiety-related behaviours in rats [Internet]. Pharmacological Research. 2004 ; 50 517-522.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2004.05.003
    • Vancouver

      Jorge SD, Pobbe RLH, Soares V de P, Oliveira AM de, Zangrossi Júnior H. Effects of sibutramine on anxiety-related behaviours in rats [Internet]. Pharmacological Research. 2004 ; 50 517-522.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/j.phrs.2004.05.003
  • Source: Pharmacological Research. Unidade: FFCLRP

    Assunto: FARMACOLOGIA

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      ROSSA, Marcelo M. et al. Comparison of the cytotoxicity of two nitroheterociclic drugs (NHCD) towards transformed and non-transformed cells. Tradução . Pharmacological Research, London, 2003. , v. 48, p. 369-375. Acesso em: 19 abr. 2024.
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      Rossa, M. M., Rocha-e-Sillva, T. A. A., Terruggi, C. H. B., Tedesco, A. C., Selistre-de- Araújo, H. S., Borissevich, I. E., & Degterev, I. A. (2003). Comparison of the cytotoxicity of two nitroheterociclic drugs (NHCD) towards transformed and non-transformed cells. Pharmacological Research, p. 369-375. London: Faculdade de Filosofia, Ciências e Letras de Ribeirão Preto, Universidade de São Paulo.
    • NLM

      Rossa MM, Rocha-e-Sillva TAA, Terruggi CHB, Tedesco AC, Selistre-de- Araújo HS, Borissevich IE, Degterev IA. Comparison of the cytotoxicity of two nitroheterociclic drugs (NHCD) towards transformed and non-transformed cells. Pharmacological Research. 2003 ; 48 369-375.[citado 2024 abr. 19 ]
    • Vancouver

      Rossa MM, Rocha-e-Sillva TAA, Terruggi CHB, Tedesco AC, Selistre-de- Araújo HS, Borissevich IE, Degterev IA. Comparison of the cytotoxicity of two nitroheterociclic drugs (NHCD) towards transformed and non-transformed cells. Pharmacological Research. 2003 ; 48 369-375.[citado 2024 abr. 19 ]
  • Source: Pharmacological Research. Unidade: FCFRP

    Assunto: MICROBIOLOGIA

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      MORA, Luciana de Oliveira et al. The effects of oral glutamine on cisplatin-induced nephrotoxicity in rats. Pharmacological Research, v. 47, p. 517-522, 2003Tradução . . Disponível em: https://doi.org/10.1016/s1043-6618(03)00040-9. Acesso em: 19 abr. 2024.
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      Mora, L. de O., Antunes, L. M. G., Francescato, H. D. C., & Bianchi, M. de L. P. (2003). The effects of oral glutamine on cisplatin-induced nephrotoxicity in rats. Pharmacological Research, 47, 517-522. doi:10.1016/s1043-6618(03)00040-9
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      Mora L de O, Antunes LMG, Francescato HDC, Bianchi M de LP. The effects of oral glutamine on cisplatin-induced nephrotoxicity in rats [Internet]. Pharmacological Research. 2003 ; 47 517-522.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/s1043-6618(03)00040-9
    • Vancouver

      Mora L de O, Antunes LMG, Francescato HDC, Bianchi M de LP. The effects of oral glutamine on cisplatin-induced nephrotoxicity in rats [Internet]. Pharmacological Research. 2003 ; 47 517-522.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/s1043-6618(03)00040-9
  • Source: Pharmacological Research. Unidades: FM, FCFRP

    Assunto: QUÍMICA ANALÍTICA

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      ZAINAGHI, Isis Amaral e LANCHOTE, Vera Lúcia e QUEIROZ, Regina Helena Costa. Determination of paroxetine in geriatric depression by high-performance liquid chromatography. Pharmacological Research, v. 48, p. 217-221, 2003Tradução . . Disponível em: https://doi.org/10.1016/s1043-6618(03)00098-7. Acesso em: 19 abr. 2024.
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      Zainaghi, I. A., Lanchote, V. L., & Queiroz, R. H. C. (2003). Determination of paroxetine in geriatric depression by high-performance liquid chromatography. Pharmacological Research, 48, 217-221. doi:10.1016/s1043-6618(03)00098-7
    • NLM

      Zainaghi IA, Lanchote VL, Queiroz RHC. Determination of paroxetine in geriatric depression by high-performance liquid chromatography [Internet]. Pharmacological Research. 2003 ; 48 217-221.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/s1043-6618(03)00098-7
    • Vancouver

      Zainaghi IA, Lanchote VL, Queiroz RHC. Determination of paroxetine in geriatric depression by high-performance liquid chromatography [Internet]. Pharmacological Research. 2003 ; 48 217-221.[citado 2024 abr. 19 ] Available from: https://doi.org/10.1016/s1043-6618(03)00098-7

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