Filtros : "Journal of Hypertension" "Indexado na Base de Dados Scopus" Removido: "BELLOTTI, GIOVANNI MAURO VITTORIO" Limpar

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  • Source: Journal of Hypertension. Unidade: ICB

    Subjects: PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA, RATOS WISTAR, NEURÔNIOS, GÂNGLIOS, TRONCO CEREBRAL, SISTEMA NERVOSO CENTRAL

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      MOREIRA, Thiago dos Santos et al. Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat. Journal of Hypertension, v. 36, n. 12, p. 2444-2452, 2018Tradução . . Disponível em: https://doi.org/10.1097/HJH.0000000000001871. Acesso em: 04 out. 2024.
    • APA

      Moreira, T. dos S., Antunes, V. R., Falquetto, B., & Gonzalez, N. M. (2018). Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat. Journal of Hypertension, 36( 12), 2444-2452. doi:10.1097/HJH.0000000000001871
    • NLM

      Moreira T dos S, Antunes VR, Falquetto B, Gonzalez NM. Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat [Internet]. Journal of Hypertension. 2018 ; 36( 12): 2444-2452.[citado 2024 out. 04 ] Available from: https://doi.org/10.1097/HJH.0000000000001871
    • Vancouver

      Moreira T dos S, Antunes VR, Falquetto B, Gonzalez NM. Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat [Internet]. Journal of Hypertension. 2018 ; 36( 12): 2444-2452.[citado 2024 out. 04 ] Available from: https://doi.org/10.1097/HJH.0000000000001871
  • Source: Journal of Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA (HUMANO)

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      KOBAYASI, Renata et al. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice. Journal of Hypertension, v. 28, n. 10, p. 2111-2119, 2010Tradução . . Disponível em: https://doi.org/10.1097/HJH.0b013e32833ca68c. Acesso em: 04 out. 2024.
    • APA

      Kobayasi, R., Akamine, E. H., Davel, A. P. C., Rodrigues, M. A. M., Carvalho, C. R. de O., & Rossoni, L. V. (2010). Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice. Journal of Hypertension, 28( 10), 2111-2119. doi:10.1097/HJH.0b013e32833ca68c
    • NLM

      Kobayasi R, Akamine EH, Davel APC, Rodrigues MAM, Carvalho CR de O, Rossoni LV. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice [Internet]. Journal of Hypertension. 2010 ; 28( 10): 2111-2119.[citado 2024 out. 04 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c
    • Vancouver

      Kobayasi R, Akamine EH, Davel APC, Rodrigues MAM, Carvalho CR de O, Rossoni LV. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice [Internet]. Journal of Hypertension. 2010 ; 28( 10): 2111-2119.[citado 2024 out. 04 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c

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