Filtros : "Journal of Clinical Endocrinology and Metabolism" Limpar

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  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Subjects: HIPOTIREOIDISMO (CONGÊNITO;GENÉTICA), SEQUENCIAMENTO GENÉTICO, POLIMORFISMO, MUTAÇÃO GENÉTICA, BRASIL

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    • ABNT

      GUTNISKY, Viviana J. et al. Two distinct compound heterozygous constellation (R277X / IVS34-1G>C and R277X / R1511X) in the thyroglobulin gene in affected individuals of a Brazilian kindred with congenital goiter and defective thyroglobulin synthesis. Journal of Clinical Endocrinology and Metabolism, v. 89, n. 2, p. 646-657, 2004Tradução . . Acesso em: 28 mar. 2024.
    • APA

      Gutnisky, V. J., Moya, C. M., Rivolta, C. M., Domené, S., Varela, V., Toniolo, J. V., et al. (2004). Two distinct compound heterozygous constellation (R277X / IVS34-1G>C and R277X / R1511X) in the thyroglobulin gene in affected individuals of a Brazilian kindred with congenital goiter and defective thyroglobulin synthesis. Journal of Clinical Endocrinology and Metabolism, 89( 2), 646-657.
    • NLM

      Gutnisky VJ, Moya CM, Rivolta CM, Domené S, Varela V, Toniolo JV, Medeiros-Neto G, Targovnik HM. Two distinct compound heterozygous constellation (R277X / IVS34-1G>C and R277X / R1511X) in the thyroglobulin gene in affected individuals of a Brazilian kindred with congenital goiter and defective thyroglobulin synthesis. Journal of Clinical Endocrinology and Metabolism. 2004 ; 89( 2): 646-657.[citado 2024 mar. 28 ]
    • Vancouver

      Gutnisky VJ, Moya CM, Rivolta CM, Domené S, Varela V, Toniolo JV, Medeiros-Neto G, Targovnik HM. Two distinct compound heterozygous constellation (R277X / IVS34-1G>C and R277X / R1511X) in the thyroglobulin gene in affected individuals of a Brazilian kindred with congenital goiter and defective thyroglobulin synthesis. Journal of Clinical Endocrinology and Metabolism. 2004 ; 89( 2): 646-657.[citado 2024 mar. 28 ]
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FMRP

    Subjects: ACROMEGALIA, ENDOCRINOLOGIA

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    • ABNT

      COSTA, Augusto C. F. et al. Assessment of disease activity in treated acromegalic patients using a sensitive GH assay: should we achieve strict normal GH levels for a biochemical cure?. Journal of Clinical Endocrinology and Metabolism, v. 87, n. 7, p. 3142-3147, 2002Tradução . . Acesso em: 28 mar. 2024.
    • APA

      Costa, A. C. F., Rossi, A., Martinelli, C. E., Machado, H. R., & Moreira, A. C. (2002). Assessment of disease activity in treated acromegalic patients using a sensitive GH assay: should we achieve strict normal GH levels for a biochemical cure? Journal of Clinical Endocrinology and Metabolism, 87( 7), 3142-3147.
    • NLM

      Costa ACF, Rossi A, Martinelli CE, Machado HR, Moreira AC. Assessment of disease activity in treated acromegalic patients using a sensitive GH assay: should we achieve strict normal GH levels for a biochemical cure? Journal of Clinical Endocrinology and Metabolism. 2002 ; 87( 7): 3142-3147.[citado 2024 mar. 28 ]
    • Vancouver

      Costa ACF, Rossi A, Martinelli CE, Machado HR, Moreira AC. Assessment of disease activity in treated acromegalic patients using a sensitive GH assay: should we achieve strict normal GH levels for a biochemical cure? Journal of Clinical Endocrinology and Metabolism. 2002 ; 87( 7): 3142-3147.[citado 2024 mar. 28 ]
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FMRP

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      CHANSON, Philipe et al. Normal pituitary hypertrophy as a frequent cause of pituitary incidentaloma: a follow-up study. Journal of Clinical Endocrinology and Metabolism, v. 86, n. 7, p. 3009-3015, 2001Tradução . . Acesso em: 28 mar. 2024.
    • APA

      Chanson, P., Daujat, F., Young, J., Bellucci, Â. D., Kujas, M., & Schaison, G. (2001). Normal pituitary hypertrophy as a frequent cause of pituitary incidentaloma: a follow-up study. Journal of Clinical Endocrinology and Metabolism, 86( 7), 3009-3015.
    • NLM

      Chanson P, Daujat F, Young J, Bellucci ÂD, Kujas M, Schaison G. Normal pituitary hypertrophy as a frequent cause of pituitary incidentaloma: a follow-up study. Journal of Clinical Endocrinology and Metabolism. 2001 ; 86( 7): 3009-3015.[citado 2024 mar. 28 ]
    • Vancouver

      Chanson P, Daujat F, Young J, Bellucci ÂD, Kujas M, Schaison G. Normal pituitary hypertrophy as a frequent cause of pituitary incidentaloma: a follow-up study. Journal of Clinical Endocrinology and Metabolism. 2001 ; 86( 7): 3009-3015.[citado 2024 mar. 28 ]
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      SALVATORI, Roberto et al. Familial dwarfism due to a novel mutation of the growth hormone-releasing hormone receptor gene. Journal of Clinical Endocrinology and Metabolism, v. 84, n. 3, p. 917-923, 1999Tradução . . Acesso em: 28 mar. 2024.
    • APA

      Salvatori, R., Hayashida, C. Y., Aguiar-Oliveira, M. H., Phillips III, J. A., Souza, A. H. O., Gondo, R. G., et al. (1999). Familial dwarfism due to a novel mutation of the growth hormone-releasing hormone receptor gene. Journal of Clinical Endocrinology and Metabolism, 84( 3), 917-923.
    • NLM

      Salvatori R, Hayashida CY, Aguiar-Oliveira MH, Phillips III JA, Souza AHO, Gondo RG, Toledo SPA, Conceição MM, Prince M, Maheshwari HG, Baumann G, Levine M. Familial dwarfism due to a novel mutation of the growth hormone-releasing hormone receptor gene. Journal of Clinical Endocrinology and Metabolism. 1999 ; 84( 3): 917-923.[citado 2024 mar. 28 ]
    • Vancouver

      Salvatori R, Hayashida CY, Aguiar-Oliveira MH, Phillips III JA, Souza AHO, Gondo RG, Toledo SPA, Conceição MM, Prince M, Maheshwari HG, Baumann G, Levine M. Familial dwarfism due to a novel mutation of the growth hormone-releasing hormone receptor gene. Journal of Clinical Endocrinology and Metabolism. 1999 ; 84( 3): 917-923.[citado 2024 mar. 28 ]
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      SALVATORI, Roberto et al. Familial dwarfism due to a novel mutation of the growth hormone-releasing hormone receptor gene. Journal of Clinical Endocrinology and Metabolism, v. 84, n. 3, p. 917-923, 1998Tradução . . Acesso em: 28 mar. 2024.
    • APA

      Salvatori, R., Hayashida, C. Y., Aguiar-Oliveira, M. H., Phillips III, J. A., Souza, A. H. O., Gondo, R. G., et al. (1998). Familial dwarfism due to a novel mutation of the growth hormone-releasing hormone receptor gene. Journal of Clinical Endocrinology and Metabolism, 84( 3), 917-923.
    • NLM

      Salvatori R, Hayashida CY, Aguiar-Oliveira MH, Phillips III JA, Souza AHO, Gondo RG, Toledo SPA, Conceição MM, Prince M, Maheshwari HG, Baumann G, Levine M. Familial dwarfism due to a novel mutation of the growth hormone-releasing hormone receptor gene. Journal of Clinical Endocrinology and Metabolism. 1998 ; 84( 3): 917-923.[citado 2024 mar. 28 ]
    • Vancouver

      Salvatori R, Hayashida CY, Aguiar-Oliveira MH, Phillips III JA, Souza AHO, Gondo RG, Toledo SPA, Conceição MM, Prince M, Maheshwari HG, Baumann G, Levine M. Familial dwarfism due to a novel mutation of the growth hormone-releasing hormone receptor gene. Journal of Clinical Endocrinology and Metabolism. 1998 ; 84( 3): 917-923.[citado 2024 mar. 28 ]
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: OBSTETRÍCIA

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    • ABNT

      MEDEIROS NETO, Geraldo Antônio et al. Prenatal diagnosis and treatment of dyshormonogenetic fetal goiter due to defective thyroglobulin synthesis. Journal of Clinical Endocrinology and Metabolism, v. 82, n. 12, p. 4239-4242, 1997Tradução . . Acesso em: 28 mar. 2024.
    • APA

      Medeiros Neto, G. A., Bunduki, V., Tomimori, E., Gomes, S., Knobel, M., Martin, R. T., & Zugaib, M. (1997). Prenatal diagnosis and treatment of dyshormonogenetic fetal goiter due to defective thyroglobulin synthesis. Journal of Clinical Endocrinology and Metabolism, 82( 12), 4239-4242.
    • NLM

      Medeiros Neto GA, Bunduki V, Tomimori E, Gomes S, Knobel M, Martin RT, Zugaib M. Prenatal diagnosis and treatment of dyshormonogenetic fetal goiter due to defective thyroglobulin synthesis. Journal of Clinical Endocrinology and Metabolism. 1997 ; 82( 12): 4239-4242.[citado 2024 mar. 28 ]
    • Vancouver

      Medeiros Neto GA, Bunduki V, Tomimori E, Gomes S, Knobel M, Martin RT, Zugaib M. Prenatal diagnosis and treatment of dyshormonogenetic fetal goiter due to defective thyroglobulin synthesis. Journal of Clinical Endocrinology and Metabolism. 1997 ; 82( 12): 4239-4242.[citado 2024 mar. 28 ]
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FMRP

    Subjects: GLÂNDULAS ENDÓCRINAS, DIABETES MELLITUS

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    • ABNT

      ELIAS, L. L. K. et al. Effect of plasma osmolality on pituitary-adrenal responses to corticotropin releasing hormone and atrial natriuretic peptide changes in central diabetes insipidus. Journal of Clinical Endocrinology and Metabolism, v. 82, n. 4 , p. 1243-1247, 1997Tradução . . Acesso em: 28 mar. 2024.
    • APA

      Elias, L. L. K., Antunes-Rodrigues, J., Elias, P. C. L., & Moreira, A. C. (1997). Effect of plasma osmolality on pituitary-adrenal responses to corticotropin releasing hormone and atrial natriuretic peptide changes in central diabetes insipidus. Journal of Clinical Endocrinology and Metabolism, 82( 4 ), 1243-1247.
    • NLM

      Elias LLK, Antunes-Rodrigues J, Elias PCL, Moreira AC. Effect of plasma osmolality on pituitary-adrenal responses to corticotropin releasing hormone and atrial natriuretic peptide changes in central diabetes insipidus. Journal of Clinical Endocrinology and Metabolism. 1997 ; 82( 4 ): 1243-1247.[citado 2024 mar. 28 ]
    • Vancouver

      Elias LLK, Antunes-Rodrigues J, Elias PCL, Moreira AC. Effect of plasma osmolality on pituitary-adrenal responses to corticotropin releasing hormone and atrial natriuretic peptide changes in central diabetes insipidus. Journal of Clinical Endocrinology and Metabolism. 1997 ; 82( 4 ): 1243-1247.[citado 2024 mar. 28 ]
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FMRP

    Assunto: GENÉTICA

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    • ABNT

      GREBE, Stefan K G et al. Frequent loss of heterozygosity on chromosomes 3p and 17p without VHL or p53 mutations suggests involvement of unidentified tumor suppressor genes in follicular thyroid carcinoma. Journal of Clinical Endocrinology and Metabolism, v. 82, n. 11, p. 3684-3691, 1997Tradução . . Acesso em: 28 mar. 2024.
    • APA

      Grebe, S. K. G., McIver, B., Hay, I. D., Wu, P. S. C., Maciel, L. M. Z., Drabkin, H. A., et al. (1997). Frequent loss of heterozygosity on chromosomes 3p and 17p without VHL or p53 mutations suggests involvement of unidentified tumor suppressor genes in follicular thyroid carcinoma. Journal of Clinical Endocrinology and Metabolism, 82( 11), 3684-3691.
    • NLM

      Grebe SKG, McIver B, Hay ID, Wu PSC, Maciel LMZ, Drabkin HA, Goellner JR, Grant CS, Jenkins RB, Eberhardt NL. Frequent loss of heterozygosity on chromosomes 3p and 17p without VHL or p53 mutations suggests involvement of unidentified tumor suppressor genes in follicular thyroid carcinoma. Journal of Clinical Endocrinology and Metabolism. 1997 ; 82( 11): 3684-3691.[citado 2024 mar. 28 ]
    • Vancouver

      Grebe SKG, McIver B, Hay ID, Wu PSC, Maciel LMZ, Drabkin HA, Goellner JR, Grant CS, Jenkins RB, Eberhardt NL. Frequent loss of heterozygosity on chromosomes 3p and 17p without VHL or p53 mutations suggests involvement of unidentified tumor suppressor genes in follicular thyroid carcinoma. Journal of Clinical Endocrinology and Metabolism. 1997 ; 82( 11): 3684-3691.[citado 2024 mar. 28 ]
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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      TOLEDO, S. P. A. et al. Inactivating mutation of the luteinizing hormone receptor causes amenorrhea in a 46, xx female. Journal of Clinical Endocrinology and Metabolism, v. no 1996, n. 11, p. 3850-4, 1996Tradução . . Acesso em: 28 mar. 2024.
    • APA

      Toledo, S. P. A., Bunner, H. G., Kraaij, R., Post, M., Dahia, P. L. M., Hayashida, C. Y., et al. (1996). Inactivating mutation of the luteinizing hormone receptor causes amenorrhea in a 46, xx female. Journal of Clinical Endocrinology and Metabolism, no 1996( 11), 3850-4.
    • NLM

      Toledo SPA, Bunner HG, Kraaij R, Post M, Dahia PLM, Hayashida CY, Kremer H, Themmen APN. Inactivating mutation of the luteinizing hormone receptor causes amenorrhea in a 46, xx female. Journal of Clinical Endocrinology and Metabolism. 1996 ; no 1996( 11): 3850-4.[citado 2024 mar. 28 ]
    • Vancouver

      Toledo SPA, Bunner HG, Kraaij R, Post M, Dahia PLM, Hayashida CY, Kremer H, Themmen APN. Inactivating mutation of the luteinizing hormone receptor causes amenorrhea in a 46, xx female. Journal of Clinical Endocrinology and Metabolism. 1996 ; no 1996( 11): 3850-4.[citado 2024 mar. 28 ]
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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      BOYAGES, S C e MEDEIROS NETO, Geraldo Antônio. Pathogenesis of myxedematous endemic cretinism. Journal of Clinical Endocrinology and Metabolism, v. 81, n. 4 , p. 1671-2, 1996Tradução . . Disponível em: https://doi.org/10.1210/jcem.81.4.8636390. Acesso em: 28 mar. 2024.
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      Boyages, S. C., & Medeiros Neto, G. A. (1996). Pathogenesis of myxedematous endemic cretinism. Journal of Clinical Endocrinology and Metabolism, 81( 4 ), 1671-2. doi:10.1210/jcem.81.4.8636390
    • NLM

      Boyages SC, Medeiros Neto GA. Pathogenesis of myxedematous endemic cretinism [Internet]. Journal of Clinical Endocrinology and Metabolism. 1996 ;81( 4 ): 1671-2.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.81.4.8636390
    • Vancouver

      Boyages SC, Medeiros Neto GA. Pathogenesis of myxedematous endemic cretinism [Internet]. Journal of Clinical Endocrinology and Metabolism. 1996 ;81( 4 ): 1671-2.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.81.4.8636390
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: ICB

    Assunto: FISIOLOGIA

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      RIBELA, Maria Teresa de Carvalho Pinto e BIANCO, Antônio Carlos e BARTOLINI, P. Use of recombinant human thyrotropin produced by chinese hamster ovary cells for the preparation of immunoassay reagents. Journal of Clinical Endocrinology and Metabolism, v. 81, p. 249-56, 1996Tradução . . Disponível em: https://doi.org/10.1210/jcem.81.1.8550760. Acesso em: 28 mar. 2024.
    • APA

      Ribela, M. T. de C. P., Bianco, A. C., & Bartolini, P. (1996). Use of recombinant human thyrotropin produced by chinese hamster ovary cells for the preparation of immunoassay reagents. Journal of Clinical Endocrinology and Metabolism, 81, 249-56. doi:10.1210/jcem.81.1.8550760
    • NLM

      Ribela MT de CP, Bianco AC, Bartolini P. Use of recombinant human thyrotropin produced by chinese hamster ovary cells for the preparation of immunoassay reagents [Internet]. Journal of Clinical Endocrinology and Metabolism. 1996 ;81 249-56.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.81.1.8550760
    • Vancouver

      Ribela MT de CP, Bianco AC, Bartolini P. Use of recombinant human thyrotropin produced by chinese hamster ovary cells for the preparation of immunoassay reagents [Internet]. Journal of Clinical Endocrinology and Metabolism. 1996 ;81 249-56.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.81.1.8550760
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      SPAMBALG, D et al. Structural studies of the rhyrotropin receptor and gs alfa in human thyroid cancer: low prevalence of mutations predicts infrequent involvement in malignant transformation. Journal of Clinical Endocrinology and Metabolism, v. no 1996, n. 11, p. 3898-901, 1996Tradução . . Disponível em: https://doi.org/10.1210/jcem.81.11.8923835. Acesso em: 28 mar. 2024.
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      Spambalg, D., Sharifi, N., Elisei, R., Gross, J. L., Medeiros Neto, G. A., & Fagin, J. A. (1996). Structural studies of the rhyrotropin receptor and gs alfa in human thyroid cancer: low prevalence of mutations predicts infrequent involvement in malignant transformation. Journal of Clinical Endocrinology and Metabolism, no 1996( 11), 3898-901. doi:10.1210/jcem.81.11.8923835
    • NLM

      Spambalg D, Sharifi N, Elisei R, Gross JL, Medeiros Neto GA, Fagin JA. Structural studies of the rhyrotropin receptor and gs alfa in human thyroid cancer: low prevalence of mutations predicts infrequent involvement in malignant transformation [Internet]. Journal of Clinical Endocrinology and Metabolism. 1996 ; no 1996( 11): 3898-901.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.81.11.8923835
    • Vancouver

      Spambalg D, Sharifi N, Elisei R, Gross JL, Medeiros Neto GA, Fagin JA. Structural studies of the rhyrotropin receptor and gs alfa in human thyroid cancer: low prevalence of mutations predicts infrequent involvement in malignant transformation [Internet]. Journal of Clinical Endocrinology and Metabolism. 1996 ; no 1996( 11): 3898-901.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.81.11.8923835
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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      LATRONICO, A C et al. Novel mutation of the luteinizing hormone receptor gene causing male gonadotropin-independent precocious puberty. Journal of Clinical Endocrinology and Metabolism, v. 80, n. 8 , p. 2490-4, 1995Tradução . . Disponível em: https://doi.org/10.1210/jcem.80.8.7629248. Acesso em: 28 mar. 2024.
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      Latronico, A. C., Anasti, J., Arnhold, I. J. P., Mendonça, B. B., Domenice, S., Albano, M. C., et al. (1995). Novel mutation of the luteinizing hormone receptor gene causing male gonadotropin-independent precocious puberty. Journal of Clinical Endocrinology and Metabolism, 80( 8 ), 2490-4. doi:10.1210/jcem.80.8.7629248
    • NLM

      Latronico AC, Anasti J, Arnhold IJP, Mendonça BB, Domenice S, Albano MC, Zachman K, Wajchenberg BL, Tsigos C. Novel mutation of the luteinizing hormone receptor gene causing male gonadotropin-independent precocious puberty [Internet]. Journal of Clinical Endocrinology and Metabolism. 1995 ;80( 8 ): 2490-4.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.80.8.7629248
    • Vancouver

      Latronico AC, Anasti J, Arnhold IJP, Mendonça BB, Domenice S, Albano MC, Zachman K, Wajchenberg BL, Tsigos C. Novel mutation of the luteinizing hormone receptor gene causing male gonadotropin-independent precocious puberty [Internet]. Journal of Clinical Endocrinology and Metabolism. 1995 ;80( 8 ): 2490-4.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.80.8.7629248
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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      TARGOVNIK, H M et al. 138- nucleotid deletion in the thyroglobulin ribunocleic acid messenger in a congenital goiter with defective thyroglobulin synthesis. Journal of Clinical Endocrinology and Metabolism, v. no 1995, n. 11, p. 3356-60, 1995Tradução . . Disponível em: https://doi.org/10.1210/jcem.80.11.7593451. Acesso em: 28 mar. 2024.
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      Targovnik, H. M., Vono, J., Billerbeck, A. E. C., Cerrone, G. E., Varela, V., Mendive, F., et al. (1995). 138- nucleotid deletion in the thyroglobulin ribunocleic acid messenger in a congenital goiter with defective thyroglobulin synthesis. Journal of Clinical Endocrinology and Metabolism, no 1995( 11), 3356-60. doi:10.1210/jcem.80.11.7593451
    • NLM

      Targovnik HM, Vono J, Billerbeck AEC, Cerrone GE, Varela V, Mendive F, Wajchenberg BL, Medeiros Neto GA. 138- nucleotid deletion in the thyroglobulin ribunocleic acid messenger in a congenital goiter with defective thyroglobulin synthesis [Internet]. Journal of Clinical Endocrinology and Metabolism. 1995 ; no 1995( 11): 3356-60.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.80.11.7593451
    • Vancouver

      Targovnik HM, Vono J, Billerbeck AEC, Cerrone GE, Varela V, Mendive F, Wajchenberg BL, Medeiros Neto GA. 138- nucleotid deletion in the thyroglobulin ribunocleic acid messenger in a congenital goiter with defective thyroglobulin synthesis [Internet]. Journal of Clinical Endocrinology and Metabolism. 1995 ; no 1995( 11): 3356-60.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.80.11.7593451
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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      WAJCHENBERG, Bernardo Leo et al. Estimation of body fat and lean tissue distribution by dual energy x-ray absorptiometry and abdominal body fat evaluation by computed tomography in cushing's disease. Journal of Clinical Endocrinology and Metabolism, v. 80, n. 9, p. Se 1995, 1995Tradução . . Disponível em: https://doi.org/10.1210/jcem.80.9.7673425. Acesso em: 28 mar. 2024.
    • APA

      Wajchenberg, B. L., Bosco, A., Marone, M. M., Levin, S., Rocha, M. de S., Lerario, A. C., et al. (1995). Estimation of body fat and lean tissue distribution by dual energy x-ray absorptiometry and abdominal body fat evaluation by computed tomography in cushing's disease. Journal of Clinical Endocrinology and Metabolism, 80( 9), Se 1995. doi:10.1210/jcem.80.9.7673425
    • NLM

      Wajchenberg BL, Bosco A, Marone MM, Levin S, Rocha M de S, Lerario AC, Nery M, Goldman J, Liberman B. Estimation of body fat and lean tissue distribution by dual energy x-ray absorptiometry and abdominal body fat evaluation by computed tomography in cushing's disease [Internet]. Journal of Clinical Endocrinology and Metabolism. 1995 ; 80( 9): Se 1995.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.80.9.7673425
    • Vancouver

      Wajchenberg BL, Bosco A, Marone MM, Levin S, Rocha M de S, Lerario AC, Nery M, Goldman J, Liberman B. Estimation of body fat and lean tissue distribution by dual energy x-ray absorptiometry and abdominal body fat evaluation by computed tomography in cushing's disease [Internet]. Journal of Clinical Endocrinology and Metabolism. 1995 ; 80( 9): Se 1995.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.80.9.7673425
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ONCOLOGIA

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      LATRONICO, A C et al. No evicence for oncogenic mutations in the adrenocorticotripin receptor gene in human adrenocortical neoplasms. Journal of Clinical Endocrinology and Metabolism, v. 80, n. 3 , p. 875-7, 1995Tradução . . Disponível em: https://doi.org/10.1210/jcem.80.3.7883845. Acesso em: 28 mar. 2024.
    • APA

      Latronico, A. C., Reincke, M., Mendonça, B. B., Arai, K., Mora, P., Allolio, B., et al. (1995). No evicence for oncogenic mutations in the adrenocorticotripin receptor gene in human adrenocortical neoplasms. Journal of Clinical Endocrinology and Metabolism, 80( 3 ), 875-7. doi:10.1210/jcem.80.3.7883845
    • NLM

      Latronico AC, Reincke M, Mendonça BB, Arai K, Mora P, Allolio B, Wajchenberg BL, Chrousos GP, Tsigos C. No evicence for oncogenic mutations in the adrenocorticotripin receptor gene in human adrenocortical neoplasms [Internet]. Journal of Clinical Endocrinology and Metabolism. 1995 ;80( 3 ): 875-7.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.80.3.7883845
    • Vancouver

      Latronico AC, Reincke M, Mendonça BB, Arai K, Mora P, Allolio B, Wajchenberg BL, Chrousos GP, Tsigos C. No evicence for oncogenic mutations in the adrenocorticotripin receptor gene in human adrenocortical neoplasms [Internet]. Journal of Clinical Endocrinology and Metabolism. 1995 ;80( 3 ): 875-7.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.80.3.7883845
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      SEMER, M et al. Growth hormone pulsatility in active and cured acromegalic subjects. Journal of Clinical Endocrinology and Metabolism, v. 80, n. 12, p. 3767-70, 1995Tradução . . Disponível em: https://doi.org/10.1210/jcem.80.12.8530632. Acesso em: 28 mar. 2024.
    • APA

      Semer, M., Faria, A. C. S., Nery, M., Salgado, L. R., Knoepfelmacher, M., Wajchenberg, B. L., & Liberman, B. (1995). Growth hormone pulsatility in active and cured acromegalic subjects. Journal of Clinical Endocrinology and Metabolism, 80( 12), 3767-70. doi:10.1210/jcem.80.12.8530632
    • NLM

      Semer M, Faria ACS, Nery M, Salgado LR, Knoepfelmacher M, Wajchenberg BL, Liberman B. Growth hormone pulsatility in active and cured acromegalic subjects [Internet]. Journal of Clinical Endocrinology and Metabolism. 1995 ;80( 12): 3767-70.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.80.12.8530632
    • Vancouver

      Semer M, Faria ACS, Nery M, Salgado LR, Knoepfelmacher M, Wajchenberg BL, Liberman B. Growth hormone pulsatility in active and cured acromegalic subjects [Internet]. Journal of Clinical Endocrinology and Metabolism. 1995 ;80( 12): 3767-70.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.80.12.8530632
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidades: FM, ICB

    Subjects: BIOFÍSICA, FISIOLOGIA

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    • ABNT

      LATRONICO, A C et al. Calcium-dependent protein kinase-c activity in human adrenocortical neoplasms , hyperplastic adrenals , and normal adrenocortical tissue. Journal of Clinical Endocrinology and Metabolism, v. 79, n. 3 , p. 736-9, 1994Tradução . . Disponível em: https://doi.org/10.1210/jcem.79.3.7521351. Acesso em: 28 mar. 2024.
    • APA

      Latronico, A. C., Mendonça, B. B., Bianco, A. C., Villares, S. M., Lucon, M. A., Nicolau, W., & Wajchenberg, B. L. (1994). Calcium-dependent protein kinase-c activity in human adrenocortical neoplasms , hyperplastic adrenals , and normal adrenocortical tissue. Journal of Clinical Endocrinology and Metabolism, 79( 3 ), 736-9. doi:10.1210/jcem.79.3.7521351
    • NLM

      Latronico AC, Mendonça BB, Bianco AC, Villares SM, Lucon MA, Nicolau W, Wajchenberg BL. Calcium-dependent protein kinase-c activity in human adrenocortical neoplasms , hyperplastic adrenals , and normal adrenocortical tissue [Internet]. Journal of Clinical Endocrinology and Metabolism. 1994 ;79( 3 ): 736-9.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.79.3.7521351
    • Vancouver

      Latronico AC, Mendonça BB, Bianco AC, Villares SM, Lucon MA, Nicolau W, Wajchenberg BL. Calcium-dependent protein kinase-c activity in human adrenocortical neoplasms , hyperplastic adrenals , and normal adrenocortical tissue [Internet]. Journal of Clinical Endocrinology and Metabolism. 1994 ;79( 3 ): 736-9.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.79.3.7521351
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      TARGOVNIK, H M et al. Nonsense mutation causes human hereditary congenital goiter with preferential production of a 171 nucleotide-deleted thyroglobulin ribunocleic acid messenger. Journal of Clinical Endocrinology and Metabolism, v. 77, n. 1 , p. 210-5, 1993Tradução . . Disponível em: https://doi.org/10.1210/jcem.77.1.8325944. Acesso em: 28 mar. 2024.
    • APA

      Targovnik, H. M., Medeiros Neto, G. A., Varela, V., Cochaux, P., Wajchenberg, B. L., & Vassart, G. (1993). Nonsense mutation causes human hereditary congenital goiter with preferential production of a 171 nucleotide-deleted thyroglobulin ribunocleic acid messenger. Journal of Clinical Endocrinology and Metabolism, 77( 1 ), 210-5. doi:10.1210/jcem.77.1.8325944
    • NLM

      Targovnik HM, Medeiros Neto GA, Varela V, Cochaux P, Wajchenberg BL, Vassart G. Nonsense mutation causes human hereditary congenital goiter with preferential production of a 171 nucleotide-deleted thyroglobulin ribunocleic acid messenger [Internet]. Journal of Clinical Endocrinology and Metabolism. 1993 ;77( 1 ): 210-5.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.77.1.8325944
    • Vancouver

      Targovnik HM, Medeiros Neto GA, Varela V, Cochaux P, Wajchenberg BL, Vassart G. Nonsense mutation causes human hereditary congenital goiter with preferential production of a 171 nucleotide-deleted thyroglobulin ribunocleic acid messenger [Internet]. Journal of Clinical Endocrinology and Metabolism. 1993 ;77( 1 ): 210-5.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.77.1.8325944
  • Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      GROLLMAN, E F et al. Hyposialylated thyroglobulin in a patient with congenital goiter and hypothyroidism. Journal of Clinical Endocrinology and Metabolism, v. 74, n. ja 1992, p. 43-8, 1992Tradução . . Disponível em: https://doi.org/10.1210/jcem.74.1.1727828. Acesso em: 28 mar. 2024.
    • APA

      Grollman, E. F., Doi, S. Q., Weiss, P., Ashwell, G., Wajchenberg, B. L., & Medeiros Neto, G. A. (1992). Hyposialylated thyroglobulin in a patient with congenital goiter and hypothyroidism. Journal of Clinical Endocrinology and Metabolism, 74( ja 1992), 43-8. doi:10.1210/jcem.74.1.1727828
    • NLM

      Grollman EF, Doi SQ, Weiss P, Ashwell G, Wajchenberg BL, Medeiros Neto GA. Hyposialylated thyroglobulin in a patient with congenital goiter and hypothyroidism [Internet]. Journal of Clinical Endocrinology and Metabolism. 1992 ;74( ja 1992): 43-8.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.74.1.1727828
    • Vancouver

      Grollman EF, Doi SQ, Weiss P, Ashwell G, Wajchenberg BL, Medeiros Neto GA. Hyposialylated thyroglobulin in a patient with congenital goiter and hypothyroidism [Internet]. Journal of Clinical Endocrinology and Metabolism. 1992 ;74( ja 1992): 43-8.[citado 2024 mar. 28 ] Available from: https://doi.org/10.1210/jcem.74.1.1727828

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