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  • Source: Clinical Endocrinology. Unidade: FM

    Subjects: BÓCIO MODULAR (TERAPIA), TIROTROFINA (APLICAÇÕES TERAPÊUTICAS), RADIOISÓTOPOS (APLICAÇÕES TERAPÊUTICAS)

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    • ABNT

      SILVA, Marcia N. C. et al. Administration of a single dose of recombinant human thyrotrophin enhances the efficacy of radioiodine treatment of large compressive multinodular goitres. Clinical Endocrinology, v. 60, p. 300-308, 2004Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2003.01918.x. Acesso em: 29 mar. 2024.
    • APA

      Silva, M. N. C., Rubió, I. G. S., Romão, R., Gebrin, E. M. M. S., Buchpiguel, C. A., Tomimori, E., et al. (2004). Administration of a single dose of recombinant human thyrotrophin enhances the efficacy of radioiodine treatment of large compressive multinodular goitres. Clinical Endocrinology, 60, 300-308. doi:10.1046/j.1365-2265.2003.01918.x
    • NLM

      Silva MNC, Rubió IGS, Romão R, Gebrin EMMS, Buchpiguel CA, Tomimori E, Camargo R, Cardia MS, Medeiros-Neto G. Administration of a single dose of recombinant human thyrotrophin enhances the efficacy of radioiodine treatment of large compressive multinodular goitres [Internet]. Clinical Endocrinology. 2004 ; 60 300-308.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01918.x
    • Vancouver

      Silva MNC, Rubió IGS, Romão R, Gebrin EMMS, Buchpiguel CA, Tomimori E, Camargo R, Cardia MS, Medeiros-Neto G. Administration of a single dose of recombinant human thyrotrophin enhances the efficacy of radioiodine treatment of large compressive multinodular goitres [Internet]. Clinical Endocrinology. 2004 ; 60 300-308.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01918.x
  • Source: Clinical Endocrinology. Unidades: FCF, FM

    Subjects: DIABETES MELLITUS, LIPOPROTEÍNAS

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      WAJCHENBERG, Bernardo Leo et al. Metabolism of chylomicrons in patients with congenital lipoatrophic diabetes: a study with emulsion models of chylomicrons. Clinical Endocrinology, v. 61, n. 3, p. 347-352, 2004Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2004.02103.x. Acesso em: 29 mar. 2024.
    • APA

      Wajchenberg, B. L., Amâncio, R. F., Santomauro, A. T., & Maranhão, R. C. (2004). Metabolism of chylomicrons in patients with congenital lipoatrophic diabetes: a study with emulsion models of chylomicrons. Clinical Endocrinology, 61( 3), 347-352. doi:10.1111/j.1365-2265.2004.02103.x
    • NLM

      Wajchenberg BL, Amâncio RF, Santomauro AT, Maranhão RC. Metabolism of chylomicrons in patients with congenital lipoatrophic diabetes: a study with emulsion models of chylomicrons [Internet]. Clinical Endocrinology. 2004 ; 61( 3): 347-352.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1111/j.1365-2265.2004.02103.x
    • Vancouver

      Wajchenberg BL, Amâncio RF, Santomauro AT, Maranhão RC. Metabolism of chylomicrons in patients with congenital lipoatrophic diabetes: a study with emulsion models of chylomicrons [Internet]. Clinical Endocrinology. 2004 ; 61( 3): 347-352.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1111/j.1365-2265.2004.02103.x
  • Source: Clinical Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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      JORGE, Alexander A. L. et al. The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration. Clinical Endocrinology, v. 60, p. 36-40, 2004Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2004.01930.x. Acesso em: 29 mar. 2024.
    • APA

      Jorge, A. A. L., Souza, S. C. A. L., Arnhold, I. J. P., & Mendonça, B. B. (2004). The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration. Clinical Endocrinology, 60, 36-40. doi:10.1111/j.1365-2265.2004.01930.x
    • NLM

      Jorge AAL, Souza SCAL, Arnhold IJP, Mendonça BB. The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration [Internet]. Clinical Endocrinology. 2004 ; 60 36-40.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1111/j.1365-2265.2004.01930.x
    • Vancouver

      Jorge AAL, Souza SCAL, Arnhold IJP, Mendonça BB. The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration [Internet]. Clinical Endocrinology. 2004 ; 60 36-40.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1111/j.1365-2265.2004.01930.x
  • Source: Clinical Endocrinology. Unidade: ICB

    Assunto: HISTOLOGIA

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    • ABNT

      KIMURA, Edna Teruko et al. Autonomously functioning thyroid nodules are not associated with BRAF mutations. Clinical Endocrinology. Oxford: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 29 mar. 2024. , 2004
    • APA

      Kimura, E. T., Vanvooren, V., Sande, J. van, Nikiforov, Y. E., & Fagin, J. A. (2004). Autonomously functioning thyroid nodules are not associated with BRAF mutations. Clinical Endocrinology. Oxford: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Kimura ET, Vanvooren V, Sande J van, Nikiforov YE, Fagin JA. Autonomously functioning thyroid nodules are not associated with BRAF mutations. Clinical Endocrinology. 2004 ; 60 394-396.[citado 2024 mar. 29 ]
    • Vancouver

      Kimura ET, Vanvooren V, Sande J van, Nikiforov YE, Fagin JA. Autonomously functioning thyroid nodules are not associated with BRAF mutations. Clinical Endocrinology. 2004 ; 60 394-396.[citado 2024 mar. 29 ]
  • Source: Clinical Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      CARVALHO, Luciani R. et al. Acromegalic features in growth hormore (GH)-deficient patients after long-term GH therapy. Clinical Endocrinology, v. 59, p. 788-792, 2003Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2003.01899.x. Acesso em: 29 mar. 2024.
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      Carvalho, L. R., Faria, M. E. J. de, Osorio, M. G. F., Estefan, V., Jorge, A. A. L., Arnhold, I. J. P., & Mendonça, B. B. (2003). Acromegalic features in growth hormore (GH)-deficient patients after long-term GH therapy. Clinical Endocrinology, 59, 788-792. doi:10.1046/j.1365-2265.2003.01899.x
    • NLM

      Carvalho LR, Faria MEJ de, Osorio MGF, Estefan V, Jorge AAL, Arnhold IJP, Mendonça BB. Acromegalic features in growth hormore (GH)-deficient patients after long-term GH therapy [Internet]. Clinical Endocrinology. 2003 ; 59 788-792.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01899.x
    • Vancouver

      Carvalho LR, Faria MEJ de, Osorio MGF, Estefan V, Jorge AAL, Arnhold IJP, Mendonça BB. Acromegalic features in growth hormore (GH)-deficient patients after long-term GH therapy [Internet]. Clinical Endocrinology. 2003 ; 59 788-792.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01899.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Assunto: ENDOCRINOLOGIA

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      CASTRO, Margaret et al. A dose-response study of salivary cortisol after dexamethasone suppression test in Cushing's disease and its potential use in the differential diagnosis of Cushing's syndrome. Clinical Endocrinology, v. 59, p. 800-805, 2003Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2003.01908.x. Acesso em: 29 mar. 2024.
    • APA

      Castro, M., Elias, L. L. K., Elias, P. C. L., & Moreira, A. C. (2003). A dose-response study of salivary cortisol after dexamethasone suppression test in Cushing's disease and its potential use in the differential diagnosis of Cushing's syndrome. Clinical Endocrinology, 59, 800-805. doi:10.1046/j.1365-2265.2003.01908.x
    • NLM

      Castro M, Elias LLK, Elias PCL, Moreira AC. A dose-response study of salivary cortisol after dexamethasone suppression test in Cushing's disease and its potential use in the differential diagnosis of Cushing's syndrome [Internet]. Clinical Endocrinology. 2003 ; 59 800-805.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01908.x
    • Vancouver

      Castro M, Elias LLK, Elias PCL, Moreira AC. A dose-response study of salivary cortisol after dexamethasone suppression test in Cushing's disease and its potential use in the differential diagnosis of Cushing's syndrome [Internet]. Clinical Endocrinology. 2003 ; 59 800-805.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01908.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      ELIAS, Paula Conde Lamparelli et al. Progressive decline of vasopressin secretion in familial autossomal dominant neurohypophyseal diabetes insipidus presenting a novel mutation in the vasopressin-neurophysin II gene. Clinical Endocrinology, v. 39, p. 511-518, 2003Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2003.01834.x. Acesso em: 29 mar. 2024.
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      Elias, P. C. L., Elias, L. L. K., Torres, N., Moreira, A. C., Antunes-Rodrigues, J., & Castro, M. de. (2003). Progressive decline of vasopressin secretion in familial autossomal dominant neurohypophyseal diabetes insipidus presenting a novel mutation in the vasopressin-neurophysin II gene. Clinical Endocrinology, 39, 511-518. doi:10.1046/j.1365-2265.2003.01834.x
    • NLM

      Elias PCL, Elias LLK, Torres N, Moreira AC, Antunes-Rodrigues J, Castro M de. Progressive decline of vasopressin secretion in familial autossomal dominant neurohypophyseal diabetes insipidus presenting a novel mutation in the vasopressin-neurophysin II gene [Internet]. Clinical Endocrinology. 2003 ; 39 511-518.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01834.x
    • Vancouver

      Elias PCL, Elias LLK, Torres N, Moreira AC, Antunes-Rodrigues J, Castro M de. Progressive decline of vasopressin secretion in familial autossomal dominant neurohypophyseal diabetes insipidus presenting a novel mutation in the vasopressin-neurophysin II gene [Internet]. Clinical Endocrinology. 2003 ; 39 511-518.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01834.x
  • Source: Clinical Endocrinology. Unidade: FM

    Subjects: HORMÔNIOS, PUBERDADE

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      XAVIER, Ana Claudia Latronico et al. Maternal isodisomy causing homozygosity for a dominant activating mutation of the luteinizing hormone receptor gene in a boy with familial male-limited precocius puberty. Clinical Endocrinology, v. 59, p. 533-534, 2003Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2003.01810.x. Acesso em: 29 mar. 2024.
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      Xavier, A. C. L., Billerbeck, A. E. C., Pinto, E. M., d'Alva, C. B., Arnhold, I. J. P., & Mendonça, B. B. (2003). Maternal isodisomy causing homozygosity for a dominant activating mutation of the luteinizing hormone receptor gene in a boy with familial male-limited precocius puberty. Clinical Endocrinology, 59, 533-534. doi:10.1046/j.1365-2265.2003.01810.x
    • NLM

      Xavier ACL, Billerbeck AEC, Pinto EM, d'Alva CB, Arnhold IJP, Mendonça BB. Maternal isodisomy causing homozygosity for a dominant activating mutation of the luteinizing hormone receptor gene in a boy with familial male-limited precocius puberty [Internet]. Clinical Endocrinology. 2003 ; 59 533-534.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01810.x
    • Vancouver

      Xavier ACL, Billerbeck AEC, Pinto EM, d'Alva CB, Arnhold IJP, Mendonça BB. Maternal isodisomy causing homozygosity for a dominant activating mutation of the luteinizing hormone receptor gene in a boy with familial male-limited precocius puberty [Internet]. Clinical Endocrinology. 2003 ; 59 533-534.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2003.01810.x
  • Source: Clinical Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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      ANTONINI, S. R. R. et al. Glucocorticoid receptor gene polymorphism in ACTH-secreting pituitary tumours. Clinical Endocrinology, v. 57, n. 05, p. 657-662, 2002Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2002.01639.x. Acesso em: 29 mar. 2024.
    • APA

      Antonini, S. R. R., Latronico, A. C., Elias, L. L. K., Cukier, A., Machado, H. R., Liberman, B., et al. (2002). Glucocorticoid receptor gene polymorphism in ACTH-secreting pituitary tumours. Clinical Endocrinology, 57( 05), 657-662. doi:10.1046/j.1365-2265.2002.01639.x
    • NLM

      Antonini SRR, Latronico AC, Elias LLK, Cukier A, Machado HR, Liberman B, Mendonça BB, Moreira AC, Castro M. Glucocorticoid receptor gene polymorphism in ACTH-secreting pituitary tumours [Internet]. Clinical Endocrinology. 2002 ; 57( 05): 657-662.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2002.01639.x
    • Vancouver

      Antonini SRR, Latronico AC, Elias LLK, Cukier A, Machado HR, Liberman B, Mendonça BB, Moreira AC, Castro M. Glucocorticoid receptor gene polymorphism in ACTH-secreting pituitary tumours [Internet]. Clinical Endocrinology. 2002 ; 57( 05): 657-662.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2002.01639.x
  • Source: Clinical Endocrinology. Unidade: FM

    Subjects: POLIMORFISMO (GENÉTICA), MUTAÇÃO GENÉTICA, RECEPTORES, DNA

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      RICHTER-UNRUH, A. et al. Leydig cell hypoplasia:: cases with new mutations, new polymorphisms and cases without mutations in the luteinizing hormone receptor gene. Clinical Endocrinology, v. 56, p. 103-112, 2002Tradução . . Disponível em: https://doi.org/10.1046/j.0300-0664.2001.01437.x. Acesso em: 29 mar. 2024.
    • APA

      Richter-Unruh, A., Martens, J. W. M., Verhoef-Post, M., Wessels, H. T., Kors, W. A., Sinnecker, G. H. G., et al. (2002). Leydig cell hypoplasia:: cases with new mutations, new polymorphisms and cases without mutations in the luteinizing hormone receptor gene. Clinical Endocrinology, 56, 103-112. doi:10.1046/j.0300-0664.2001.01437.x
    • NLM

      Richter-Unruh A, Martens JWM, Verhoef-Post M, Wessels HT, Kors WA, Sinnecker GHG, Boehmer A, Drop SLS, Toledo SPA, Brunner HG, Themmen APN. Leydig cell hypoplasia:: cases with new mutations, new polymorphisms and cases without mutations in the luteinizing hormone receptor gene [Internet]. Clinical Endocrinology. 2002 ; 56 103-112.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.0300-0664.2001.01437.x
    • Vancouver

      Richter-Unruh A, Martens JWM, Verhoef-Post M, Wessels HT, Kors WA, Sinnecker GHG, Boehmer A, Drop SLS, Toledo SPA, Brunner HG, Themmen APN. Leydig cell hypoplasia:: cases with new mutations, new polymorphisms and cases without mutations in the luteinizing hormone receptor gene [Internet]. Clinical Endocrinology. 2002 ; 56 103-112.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.0300-0664.2001.01437.x
  • Source: Clinical Endocrinology. Unidades: FMRP, FM

    Assunto: ENDOCRINOLOGIA

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      ANTONINI, S. R. R. et al. Glucocorticoid receptor gene polymorphysms in ACTH-secreting pituitary tumors. Clinical Endocrinology, v. 57, p. 657-662, 2002Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2002.01639.x. Acesso em: 29 mar. 2024.
    • APA

      Antonini, S. R. R., Latronicot, A. C., ELIAS, L. L. K., Machado, H. R., Liberman, B., Mendonça, B. B. de, et al. (2002). Glucocorticoid receptor gene polymorphysms in ACTH-secreting pituitary tumors. Clinical Endocrinology, 57, 657-662. doi:10.1046/j.1365-2265.2002.01639.x
    • NLM

      Antonini SRR, Latronicot AC, ELIAS LLK, Machado HR, Liberman B, Mendonça BB de, Moreira AC, Castro M de. Glucocorticoid receptor gene polymorphysms in ACTH-secreting pituitary tumors [Internet]. Clinical Endocrinology. 2002 ; 57 657-662.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2002.01639.x
    • Vancouver

      Antonini SRR, Latronicot AC, ELIAS LLK, Machado HR, Liberman B, Mendonça BB de, Moreira AC, Castro M de. Glucocorticoid receptor gene polymorphysms in ACTH-secreting pituitary tumors [Internet]. Clinical Endocrinology. 2002 ; 57 657-662.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2002.01639.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOLOGIA, HORMÔNIOS (METABOLISMO), DESENVOLVIMENTO FÍSICO

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      MARUI, S. et al. Mutations in the type II 3'beta'-hydroxysteroid dehydrogenase (HSD3B2) gene can cause premature pubarche in girls. Clinical Endocrinology, v. 52, p. 67-75, 2000Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2000.00873.x. Acesso em: 29 mar. 2024.
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      Marui, S., Castro, M. de, Latronico, A. C., Elias, L. L. K., Arnhold, I. J. P., Moreira, A. C., & Mendonça, B. B. (2000). Mutations in the type II 3'beta'-hydroxysteroid dehydrogenase (HSD3B2) gene can cause premature pubarche in girls. Clinical Endocrinology, 52, 67-75. doi:10.1046/j.1365-2265.2000.00873.x
    • NLM

      Marui S, Castro M de, Latronico AC, Elias LLK, Arnhold IJP, Moreira AC, Mendonça BB. Mutations in the type II 3'beta'-hydroxysteroid dehydrogenase (HSD3B2) gene can cause premature pubarche in girls [Internet]. Clinical Endocrinology. 2000 ; 52 67-75.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2000.00873.x
    • Vancouver

      Marui S, Castro M de, Latronico AC, Elias LLK, Arnhold IJP, Moreira AC, Mendonça BB. Mutations in the type II 3'beta'-hydroxysteroid dehydrogenase (HSD3B2) gene can cause premature pubarche in girls [Internet]. Clinical Endocrinology. 2000 ; 52 67-75.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2000.00873.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOLOGIA, PEDIATRIA

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      ANTONINI, Sonir Roberto Rauber e JORGE, Salim Moysés e MOREIRA, Ayrton Custódio. The emergence of salivary cortisol circadin rhythm and its relationship to sleep activity in preterm infants. Clinical Endocrinology, v. 52, p. 423-426, 2000Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2000.tb00561.x. Acesso em: 29 mar. 2024.
    • APA

      Antonini, S. R. R., Jorge, S. M., & Moreira, A. C. (2000). The emergence of salivary cortisol circadin rhythm and its relationship to sleep activity in preterm infants. Clinical Endocrinology, 52, 423-426. doi:10.1111/j.1365-2265.2000.tb00561.x
    • NLM

      Antonini SRR, Jorge SM, Moreira AC. The emergence of salivary cortisol circadin rhythm and its relationship to sleep activity in preterm infants [Internet]. Clinical Endocrinology. 2000 ; 52 423-426.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1111/j.1365-2265.2000.tb00561.x
    • Vancouver

      Antonini SRR, Jorge SM, Moreira AC. The emergence of salivary cortisol circadin rhythm and its relationship to sleep activity in preterm infants [Internet]. Clinical Endocrinology. 2000 ; 52 423-426.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1111/j.1365-2265.2000.tb00561.x
  • Source: Clinical Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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      BARCA, Maria Fernanda et al. Prevalence and characteristics of postpartum thyroid dysfunction in São Paulo, Brazil. Clinical Endocrinology, v. 53, p. 21-31, 2000Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.2000.01034.x. Acesso em: 29 mar. 2024.
    • APA

      Barca, M. F., Knobel, M., Tomimori, E., Cardia, M. S., & Medeiros Neto, G. A. (2000). Prevalence and characteristics of postpartum thyroid dysfunction in São Paulo, Brazil. Clinical Endocrinology, 53, 21-31. doi:10.1046/j.1365-2265.2000.01034.x
    • NLM

      Barca MF, Knobel M, Tomimori E, Cardia MS, Medeiros Neto GA. Prevalence and characteristics of postpartum thyroid dysfunction in São Paulo, Brazil [Internet]. Clinical Endocrinology. 2000 ; 53 21-31.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2000.01034.x
    • Vancouver

      Barca MF, Knobel M, Tomimori E, Cardia MS, Medeiros Neto GA. Prevalence and characteristics of postpartum thyroid dysfunction in São Paulo, Brazil [Internet]. Clinical Endocrinology. 2000 ; 53 21-31.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.2000.01034.x
  • Source: Clinical Endocrinology. Unidade: FM

    Subjects: ENDOCRINOLOGIA, HORMÔNIOS

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      SANTOS, Cecilia L. S. et al. A novel mutation in the TPO gene in goitrous hypothyroid patients with iodide organification defect. Clinical Endocrinology, v. 51, n. 2, p. 165-172, 1999Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.1999.00746.x. Acesso em: 29 mar. 2024.
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      Santos, C. L. S., Bikker, H., Rego, K. G. M., Nascimento, A. C., Tambascia, M., Vijlder, J. J. M. de, & Medeiros Neto, G. A. (1999). A novel mutation in the TPO gene in goitrous hypothyroid patients with iodide organification defect. Clinical Endocrinology, 51( 2), 165-172. doi:10.1046/j.1365-2265.1999.00746.x
    • NLM

      Santos CLS, Bikker H, Rego KGM, Nascimento AC, Tambascia M, Vijlder JJM de, Medeiros Neto GA. A novel mutation in the TPO gene in goitrous hypothyroid patients with iodide organification defect [Internet]. Clinical Endocrinology. 1999 ; 51( 2): 165-172.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00746.x
    • Vancouver

      Santos CLS, Bikker H, Rego KGM, Nascimento AC, Tambascia M, Vijlder JJM de, Medeiros Neto GA. A novel mutation in the TPO gene in goitrous hypothyroid patients with iodide organification defect [Internet]. Clinical Endocrinology. 1999 ; 51( 2): 165-172.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00746.x
  • Source: Clinical Endocrinology. Unidade: FM

    Subjects: ENDOCRINOLOGIA, HORMÔNIOS

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    • ABNT

      ARNHOLD, Ivo J. P. et al. Clinical features of women with resistance to luteinizing hormone. Clinical Endocrinology, v. 51, n. 6, p. 701-707, 1999Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.1999.00863.x. Acesso em: 29 mar. 2024.
    • APA

      Arnhold, I. J. P., Latrônico, A. C., Batista, M. C., Izzo, C. R., & Mendonça, B. B. de. (1999). Clinical features of women with resistance to luteinizing hormone. Clinical Endocrinology, 51( 6), 701-707. doi:10.1046/j.1365-2265.1999.00863.x
    • NLM

      Arnhold IJP, Latrônico AC, Batista MC, Izzo CR, Mendonça BB de. Clinical features of women with resistance to luteinizing hormone [Internet]. Clinical Endocrinology. 1999 ; 51( 6): 701-707.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00863.x
    • Vancouver

      Arnhold IJP, Latrônico AC, Batista MC, Izzo CR, Mendonça BB de. Clinical features of women with resistance to luteinizing hormone [Internet]. Clinical Endocrinology. 1999 ; 51( 6): 701-707.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00863.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOPATIAS, ENDOCRINOLOGIA

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      MARTINELLI, Carlos Eduardo et al. Correlation between cortisol and insulin-like growth factor-binding proteins (IGFBPs) under physiological conditions in children. Clinical Endocrinology, v. 50, p. 767-774, 1999Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.1999.00724.x. Acesso em: 29 mar. 2024.
    • APA

      Martinelli, C. E., Yateman, M., Cotterill, A. M., Moreira, A. C., & Camacho-Hübsner, C. (1999). Correlation between cortisol and insulin-like growth factor-binding proteins (IGFBPs) under physiological conditions in children. Clinical Endocrinology, 50, 767-774. doi:10.1046/j.1365-2265.1999.00724.x
    • NLM

      Martinelli CE, Yateman M, Cotterill AM, Moreira AC, Camacho-Hübsner C. Correlation between cortisol and insulin-like growth factor-binding proteins (IGFBPs) under physiological conditions in children [Internet]. Clinical Endocrinology. 1999 ; 50 767-774.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00724.x
    • Vancouver

      Martinelli CE, Yateman M, Cotterill AM, Moreira AC, Camacho-Hübsner C. Correlation between cortisol and insulin-like growth factor-binding proteins (IGFBPs) under physiological conditions in children [Internet]. Clinical Endocrinology. 1999 ; 50 767-774.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00724.x
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOPATIAS, ENDOCRINOLOGIA

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      MARTINELLI JÚNIOR, Carlos Eduardo et al. Salivary cortisol for screening of Cushing's syndrome in children. Clinical Endocrinology, v. 51, p. 67-71, 1999Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.1999.00749.x. Acesso em: 29 mar. 2024.
    • APA

      Martinelli Júnior, C. E., Sader, S. L., Oliveira, E. B. de, Daneluzzi, J. C., & Moreira, A. C. (1999). Salivary cortisol for screening of Cushing's syndrome in children. Clinical Endocrinology, 51, 67-71. doi:10.1046/j.1365-2265.1999.00749.x
    • NLM

      Martinelli Júnior CE, Sader SL, Oliveira EB de, Daneluzzi JC, Moreira AC. Salivary cortisol for screening of Cushing's syndrome in children [Internet]. Clinical Endocrinology. 1999 ; 51 67-71.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00749.x
    • Vancouver

      Martinelli Júnior CE, Sader SL, Oliveira EB de, Daneluzzi JC, Moreira AC. Salivary cortisol for screening of Cushing's syndrome in children [Internet]. Clinical Endocrinology. 1999 ; 51 67-71.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.1999.00749.x
  • Source: Clinical Endocrinology. Unidade: FM

    Assunto: MEDICINA INTERNA

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      BORGES, Maria F et al. Calcitonin deficiency in early stages of chronic autoimmune thryroiditis. Clinical Endocrinology, v. 49, n. 1, p. 69-75, 1998Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.1998.00478.x. Acesso em: 29 mar. 2024.
    • APA

      Borges, M. F., Abelin, N. M. A., Menezes, F. O. M., Dahia, P. L. M., & Toledo, S. P. A. (1998). Calcitonin deficiency in early stages of chronic autoimmune thryroiditis. Clinical Endocrinology, 49( 1), 69-75. doi:10.1046/j.1365-2265.1998.00478.x
    • NLM

      Borges MF, Abelin NMA, Menezes FOM, Dahia PLM, Toledo SPA. Calcitonin deficiency in early stages of chronic autoimmune thryroiditis [Internet]. Clinical Endocrinology. 1998 ; 49( 1): 69-75.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.1998.00478.x
    • Vancouver

      Borges MF, Abelin NMA, Menezes FOM, Dahia PLM, Toledo SPA. Calcitonin deficiency in early stages of chronic autoimmune thryroiditis [Internet]. Clinical Endocrinology. 1998 ; 49( 1): 69-75.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.1998.00478.x
  • Source: Clinical Endocrinology. Unidade: FM

    Assunto: MEDICINA

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      PEREIRA, Maria A Albergaria et al. A study of patients with Nelson`s syndrome. Clinical Endocrinology, v. 49, n. 4, p. 533-539, 1998Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2265.1998.00578.x. Acesso em: 29 mar. 2024.
    • APA

      Pereira, M. A. A., Halpern, A., Salgado, L. R., Mendonça, B. B. de, Nery, M., Liberman, B., et al. (1998). A study of patients with Nelson`s syndrome. Clinical Endocrinology, 49( 4), 533-539. doi:10.1046/j.1365-2265.1998.00578.x
    • NLM

      Pereira MAA, Halpern A, Salgado LR, Mendonça BB de, Nery M, Liberman B, Streetent HPD, Wajchenberg BL. A study of patients with Nelson`s syndrome [Internet]. Clinical Endocrinology. 1998 ; 49( 4): 533-539.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.1998.00578.x
    • Vancouver

      Pereira MAA, Halpern A, Salgado LR, Mendonça BB de, Nery M, Liberman B, Streetent HPD, Wajchenberg BL. A study of patients with Nelson`s syndrome [Internet]. Clinical Endocrinology. 1998 ; 49( 4): 533-539.[citado 2024 mar. 29 ] Available from: https://doi.org/10.1046/j.1365-2265.1998.00578.x

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