Filtros : "British Journal of Pharmacology" "2008" Removidos: "IFSC005" "Department of Chemistry, Federal University of São Carlos, São Carlos, SP" "China" "Suécia" Limpar

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  • Source: British Journal of Pharmacology. Unidade: FMRP

    Subjects: ARTRITE, DOR, NEUTRÓFILOS

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    • ABNT

      CUNHA, T. M. et al. Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice. British Journal of Pharmacology, v. 154, n. 2, p. 460-470, 2008Tradução . . Disponível em: https://doi.org/10.1038/bjp.2008.94. Acesso em: 03 set. 2024.
    • APA

      Cunha, T. M., Barsante, M. M., Guerrero, A. T., Verri Júnior, W. A., Ferreira, S. H., Coelho, F. M., et al. (2008). Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice. British Journal of Pharmacology, 154( 2), 460-470. doi:10.1038/bjp.2008.94
    • NLM

      Cunha TM, Barsante MM, Guerrero AT, Verri Júnior WA, Ferreira SH, Coelho FM, Bertini R, Giacinto CD, Allegretti M, Cunha F de Q, Teixeira MM. Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice [Internet]. British Journal of Pharmacology. 2008 ; 154( 2): 460-470.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/bjp.2008.94
    • Vancouver

      Cunha TM, Barsante MM, Guerrero AT, Verri Júnior WA, Ferreira SH, Coelho FM, Bertini R, Giacinto CD, Allegretti M, Cunha F de Q, Teixeira MM. Treatment with DF 2162, a non-competitive allosteric inhibitor of CXCR1/2, diminishes neutrophil influx and inflammatory hypernociception in mice [Internet]. British Journal of Pharmacology. 2008 ; 154( 2): 460-470.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/bjp.2008.94
  • Source: British Journal of Pharmacology. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      MÓNICA, F. Z. T. et al. Long-term nitric oxide deficiency causes muscarinic supersensitivity and reduces ß3-adrenoceptor-mediated relaxation, causing rat detrusor overactivity. British Journal of Pharmacology, v. 153, n. 8, p. 1659-1668, 2008Tradução . . Disponível em: https://doi.org/10.1038/bjp.2008.39. Acesso em: 03 set. 2024.
    • APA

      Mónica, F. Z. T., Bricola, A. A. O., Báu, F. R., Lopes Freitas, L. L., Teixeira, S. A., Muscará, M. N., et al. (2008). Long-term nitric oxide deficiency causes muscarinic supersensitivity and reduces ß3-adrenoceptor-mediated relaxation, causing rat detrusor overactivity. British Journal of Pharmacology, 153( 8), 1659-1668. doi:10.1038/bjp.2008.39
    • NLM

      Mónica FZT, Bricola AAO, Báu FR, Lopes Freitas LL, Teixeira SA, Muscará MN, Abdalla FMF, Porto CS, De Nucci G, Zanesco A, Antunes E. Long-term nitric oxide deficiency causes muscarinic supersensitivity and reduces ß3-adrenoceptor-mediated relaxation, causing rat detrusor overactivity [Internet]. British Journal of Pharmacology. 2008 ; 153( 8): 1659-1668.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/bjp.2008.39
    • Vancouver

      Mónica FZT, Bricola AAO, Báu FR, Lopes Freitas LL, Teixeira SA, Muscará MN, Abdalla FMF, Porto CS, De Nucci G, Zanesco A, Antunes E. Long-term nitric oxide deficiency causes muscarinic supersensitivity and reduces ß3-adrenoceptor-mediated relaxation, causing rat detrusor overactivity [Internet]. British Journal of Pharmacology. 2008 ; 153( 8): 1659-1668.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/bjp.2008.39
  • Source: British Journal of Pharmacology. Unidade: FMRP

    Subjects: ADRENORECEPTORES, SISTEMA CARDIOVASCULAR, NORADRENALINA

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    • ABNT

      SCOPINHO, A. A. e RESSTEL, L. B. M. e CORRÊA, Fernando Morgan de Aguiar. 'alfa IND.1'-adrenoceptors in the lateral septal area modulate food intake behaviour in rats. British Journal of Pharmacology, p. 1-5, 2008Tradução . . Acesso em: 03 set. 2024.
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      Scopinho, A. A., Resstel, L. B. M., & Corrêa, F. M. de A. (2008). 'alfa IND.1'-adrenoceptors in the lateral septal area modulate food intake behaviour in rats. British Journal of Pharmacology, 1-5.
    • NLM

      Scopinho AA, Resstel LBM, Corrêa FM de A. 'alfa IND.1'-adrenoceptors in the lateral septal area modulate food intake behaviour in rats. British Journal of Pharmacology. 2008 ; 1-5.[citado 2024 set. 03 ]
    • Vancouver

      Scopinho AA, Resstel LBM, Corrêa FM de A. 'alfa IND.1'-adrenoceptors in the lateral septal area modulate food intake behaviour in rats. British Journal of Pharmacology. 2008 ; 1-5.[citado 2024 set. 03 ]
  • Source: British Journal of Pharmacology. Unidade: FMRP

    Subjects: ADRENORECEPTORES, SEPSE

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    • ABNT

      CRESTANI, C. C. et al. Both 'alfa IND.1' and 'alfa IND.2'-adrenoceptors mediate the cardiovascular responses to noradrenaline microinjected into the bed nucleus of the stria terminal of rats. British Journal of Pharmacology, v. 153, n. 3, p. 583-590, 2008Tradução . . Acesso em: 03 set. 2024.
    • APA

      Crestani, C. C., Alves, F. H. F., Resstel, L. B., & Corrêa, F. M. de A. (2008). Both 'alfa IND.1' and 'alfa IND.2'-adrenoceptors mediate the cardiovascular responses to noradrenaline microinjected into the bed nucleus of the stria terminal of rats. British Journal of Pharmacology, 153( 3), 583-590.
    • NLM

      Crestani CC, Alves FHF, Resstel LB, Corrêa FM de A. Both 'alfa IND.1' and 'alfa IND.2'-adrenoceptors mediate the cardiovascular responses to noradrenaline microinjected into the bed nucleus of the stria terminal of rats. British Journal of Pharmacology. 2008 ; 153( 3): 583-590.[citado 2024 set. 03 ]
    • Vancouver

      Crestani CC, Alves FHF, Resstel LB, Corrêa FM de A. Both 'alfa IND.1' and 'alfa IND.2'-adrenoceptors mediate the cardiovascular responses to noradrenaline microinjected into the bed nucleus of the stria terminal of rats. British Journal of Pharmacology. 2008 ; 153( 3): 583-590.[citado 2024 set. 03 ]
  • Source: British Journal of Pharmacology. Unidade: FMRP

    Subjects: INFLAMAÇÃO, DOR, CITOQUÍMICA

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    • ABNT

      TING, E. et al. Role of complement C5a in mechanical inflammatory hypernociception: potential use of C5a receptor antagonists to control inflammatory pain. British Journal of Pharmacology, v. 153, n. 5, p. 1043-1053, 2008Tradução . . Disponível em: https://doi.org/10.1038/sj.bjp.0707640. Acesso em: 03 set. 2024.
    • APA

      Ting, E., Guerrero, A. T. G., Cunha, T. M., Verri Júnior, W. A., Taylor, S. M., Woodruff, T. M., et al. (2008). Role of complement C5a in mechanical inflammatory hypernociception: potential use of C5a receptor antagonists to control inflammatory pain. British Journal of Pharmacology, 153( 5), 1043-1053. doi:10.1038/sj.bjp.0707640
    • NLM

      Ting E, Guerrero ATG, Cunha TM, Verri Júnior WA, Taylor SM, Woodruff TM, Cunha F de Q, Ferreira SH. Role of complement C5a in mechanical inflammatory hypernociception: potential use of C5a receptor antagonists to control inflammatory pain [Internet]. British Journal of Pharmacology. 2008 ; 153( 5): 1043-1053.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/sj.bjp.0707640
    • Vancouver

      Ting E, Guerrero ATG, Cunha TM, Verri Júnior WA, Taylor SM, Woodruff TM, Cunha F de Q, Ferreira SH. Role of complement C5a in mechanical inflammatory hypernociception: potential use of C5a receptor antagonists to control inflammatory pain [Internet]. British Journal of Pharmacology. 2008 ; 153( 5): 1043-1053.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/sj.bjp.0707640
  • Source: British Journal of Pharmacology. Unidade: ICB

    Assunto: FISIOLOGIA

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      XAVIER, F. E. et al. Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin. British Journal of Pharmacology, v. 154, n. 6, p. 1225-1235, 2008Tradução . . Disponível em: https://doi.org/10.1038/bjp.2008.200. Acesso em: 03 set. 2024.
    • APA

      Xavier, F. E., Aras-López, R., Arroyo-Villa, I., del Campo, L., Salaices, M., Rossoni, L. V., et al. (2008). Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin. British Journal of Pharmacology, 154( 6), 1225-1235. doi:10.1038/bjp.2008.200
    • NLM

      Xavier FE, Aras-López R, Arroyo-Villa I, del Campo L, Salaices M, Rossoni LV, Ferrer M, Balfagón G. Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin [Internet]. British Journal of Pharmacology. 2008 ; 154( 6): 1225-1235.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/bjp.2008.200
    • Vancouver

      Xavier FE, Aras-López R, Arroyo-Villa I, del Campo L, Salaices M, Rossoni LV, Ferrer M, Balfagón G. Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin [Internet]. British Journal of Pharmacology. 2008 ; 154( 6): 1225-1235.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/bjp.2008.200
  • Source: British Journal of Pharmacology. Unidades: EERP, ICB, FCFRP, FMRP

    Subjects: SISTEMA CARDIOVASCULAR (EFEITOS DE DROGAS), ÓXIDO NÍTRICO (SÍNTESE)

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    • ABNT

      TIRAPELLI, Carlos Renato et al. Gender-specific vascular effects elicited by chronic ethanol consumption in rats: a role for inducible nitric oxide synthase. British Journal of Pharmacology, v. 53, n. 3, p. 468-479, 2008Tradução . . Disponível em: https://doi.org/10.1038/sj.bjp.0707589. Acesso em: 03 set. 2024.
    • APA

      Tirapelli, C. R., Fukada, S. Y., Chignalia, A. Z., Passaglia, R. de C. A. T., Bonaventura, D., Lanchote, V. L., et al. (2008). Gender-specific vascular effects elicited by chronic ethanol consumption in rats: a role for inducible nitric oxide synthase. British Journal of Pharmacology, 53( 3), 468-479. doi:10.1038/sj.bjp.0707589
    • NLM

      Tirapelli CR, Fukada SY, Chignalia AZ, Passaglia R de CAT, Bonaventura D, Lanchote VL, Cunha F de Q, Oliveira AM de. Gender-specific vascular effects elicited by chronic ethanol consumption in rats: a role for inducible nitric oxide synthase [Internet]. British Journal of Pharmacology. 2008 ; 53( 3): 468-479.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/sj.bjp.0707589
    • Vancouver

      Tirapelli CR, Fukada SY, Chignalia AZ, Passaglia R de CAT, Bonaventura D, Lanchote VL, Cunha F de Q, Oliveira AM de. Gender-specific vascular effects elicited by chronic ethanol consumption in rats: a role for inducible nitric oxide synthase [Internet]. British Journal of Pharmacology. 2008 ; 53( 3): 468-479.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/sj.bjp.0707589
  • Source: British Journal of Pharmacology. Unidade: FMRP

    Subjects: ARTRITE REUMATOIDE, NEUTRÓFILOS, RECEPTORES

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    • ABNT

      BARSANTE, M. M. et al. Blockade of the chemokine receptor CXCR2 ameliorates adjuvant-induced arthritis in rats. British Journal of Pharmacology, v. 153, n. 5, p. 992-1002, 2008Tradução . . Disponível em: https://doi.org/10.1038/sj.bjp.0707462. Acesso em: 03 set. 2024.
    • APA

      Barsante, M. M., Cunha, T. M., Allegretti, M., Cattani, F., Policani, F., Bizzarri, C., et al. (2008). Blockade of the chemokine receptor CXCR2 ameliorates adjuvant-induced arthritis in rats. British Journal of Pharmacology, 153( 5), 992-1002. doi:10.1038/sj.bjp.0707462
    • NLM

      Barsante MM, Cunha TM, Allegretti M, Cattani F, Policani F, Bizzarri C, Tafuri WL, Poole S, Cunha F de Q, Bertini R, Teixeira MM. Blockade of the chemokine receptor CXCR2 ameliorates adjuvant-induced arthritis in rats [Internet]. British Journal of Pharmacology. 2008 ; 153( 5): 992-1002.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/sj.bjp.0707462
    • Vancouver

      Barsante MM, Cunha TM, Allegretti M, Cattani F, Policani F, Bizzarri C, Tafuri WL, Poole S, Cunha F de Q, Bertini R, Teixeira MM. Blockade of the chemokine receptor CXCR2 ameliorates adjuvant-induced arthritis in rats [Internet]. British Journal of Pharmacology. 2008 ; 153( 5): 992-1002.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/sj.bjp.0707462
  • Source: British Journal of Pharmacology. Unidades: FCFRP, EERP

    Subjects: DOENÇAS CARDIOVASCULARES, PRESSÃO SANGUÍNEA

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    • ABNT

      TIRAPELLI, Carlos Renato et al. Chronic ethanol intake modulates vascular levels of endothelin-1 receptor and enhances the pressor response to endothelin-1 in anaesthetized rats. British Journal of Pharmacology, v. 154, n. 5, p. 971-981, 2008Tradução . . Disponível em: https://doi.org/10.1038/bjp.2008.157. Acesso em: 03 set. 2024.
    • APA

      Tirapelli, C. R., Legros, E., Brochu, I., Honoré, J. -C., Lanchote, V. L., Uyemura, S. A., et al. (2008). Chronic ethanol intake modulates vascular levels of endothelin-1 receptor and enhances the pressor response to endothelin-1 in anaesthetized rats. British Journal of Pharmacology, 154( 5), 971-981. doi:10.1038/bjp.2008.157
    • NLM

      Tirapelli CR, Legros E, Brochu I, Honoré J-C, Lanchote VL, Uyemura SA, Oliveira AM de, D'Orleans-Juste P. Chronic ethanol intake modulates vascular levels of endothelin-1 receptor and enhances the pressor response to endothelin-1 in anaesthetized rats [Internet]. British Journal of Pharmacology. 2008 ; 154( 5): 971-981.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/bjp.2008.157
    • Vancouver

      Tirapelli CR, Legros E, Brochu I, Honoré J-C, Lanchote VL, Uyemura SA, Oliveira AM de, D'Orleans-Juste P. Chronic ethanol intake modulates vascular levels of endothelin-1 receptor and enhances the pressor response to endothelin-1 in anaesthetized rats [Internet]. British Journal of Pharmacology. 2008 ; 154( 5): 971-981.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/bjp.2008.157
  • Source: British Journal of Pharmacology. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      HERRERA, B. S. et al. An endogenous regulator of inflammation, resolving E1, modulates osteoclast differentiation and bone resorption. British Journal of Pharmacology, v. 155, n. 8, p. 1214-1223, 2008Tradução . . Disponível em: https://doi.org/10.1038/bjp.2008.367. Acesso em: 03 set. 2024.
    • APA

      Herrera, B. S., Ohira, T., Gao, L., Omori, K., Yang, R., Zhu, M., et al. (2008). An endogenous regulator of inflammation, resolving E1, modulates osteoclast differentiation and bone resorption. British Journal of Pharmacology, 155( 8), 1214-1223. doi:10.1038/bjp.2008.367
    • NLM

      Herrera BS, Ohira T, Gao L, Omori K, Yang R, Zhu M, Muscará MN, Serhan CN, Van Dyke TE, Gyurko R. An endogenous regulator of inflammation, resolving E1, modulates osteoclast differentiation and bone resorption [Internet]. British Journal of Pharmacology. 2008 ; 155( 8): 1214-1223.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/bjp.2008.367
    • Vancouver

      Herrera BS, Ohira T, Gao L, Omori K, Yang R, Zhu M, Muscará MN, Serhan CN, Van Dyke TE, Gyurko R. An endogenous regulator of inflammation, resolving E1, modulates osteoclast differentiation and bone resorption [Internet]. British Journal of Pharmacology. 2008 ; 155( 8): 1214-1223.[citado 2024 set. 03 ] Available from: https://doi.org/10.1038/bjp.2008.367

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