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Artigo de periodico
Multifaceted effect of caffeic acid against Streptococcus mutans infection: microbicidal and immunomodulatory agent in macrophages (2021)
Sorgi, Carlos Arterio ; Lamarque, Giuliana Campos Chaves ; Verri, Maraísa Palhão; Nelson Filho, Paulo ; Faccioli, Lucia Helena ; Paula-Silva, Francisco Wanderley Garcia de
Source: Archives of Microbiology. Unidades: FFCLRP, FCFRP, FORP
Subjects: STREPTOCOCCUS MUTANS, MACRÓFAGOS, ANTI-INFLAMATÓRIOS
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ABNT
SORGI, Carlos Arterio et al. Multifaceted effect of caffeic acid against Streptococcus mutans infection: microbicidal and immunomodulatory agent in macrophages. Archives of Microbiology, v. 203, n. 6, p. 2979-2987, 2021Tradução . . Disponível em: https://doi.org/10.1007/s00203-021-02290-x. Acesso em: 11 nov. 2024.
APA
Sorgi, C. A., Lamarque, G. C. C., Verri, M. P., Nelson Filho, P., Faccioli, L. H., & Paula-Silva, F. W. G. de. (2021). Multifaceted effect of caffeic acid against Streptococcus mutans infection: microbicidal and immunomodulatory agent in macrophages. Archives of Microbiology, 203( 6), 2979-2987. doi:10.1007/s00203-021-02290-x
NLM
Sorgi CA, Lamarque GCC, Verri MP, Nelson Filho P, Faccioli LH, Paula-Silva FWG de. Multifaceted effect of caffeic acid against Streptococcus mutans infection: microbicidal and immunomodulatory agent in macrophages [Internet]. Archives of Microbiology. 2021 ; 203( 6): 2979-2987.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1007/s00203-021-02290-x
Vancouver
Sorgi CA, Lamarque GCC, Verri MP, Nelson Filho P, Faccioli LH, Paula-Silva FWG de. Multifaceted effect of caffeic acid against Streptococcus mutans infection: microbicidal and immunomodulatory agent in macrophages [Internet]. Archives of Microbiology. 2021 ; 203( 6): 2979-2987.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1007/s00203-021-02290-x
Artigo de periodico
β2 integrin-mediated susceptibility to Paracoccidioides brasiliensis experimental infection in mice (2021)
Oliveira, Stephan Alberto Machado de; Reis, Janayna Nunes; Catão, Elisa; Amaral, Andre Correa; Souza, Ana Camila Oliveira; Ribeiro, Alice Melo; Faccioli, Lucia Helena ; Carneiro, Fabiana Pirani; Marina, Clara Luna Freitas; Bürgel, Pedro Henrique; Fernandes, Larissa; Tavares, Aldo Henrique; Bocca, Anamelia Lorenzetti
Source: Frontiers in Cellular and Infection Microbiology. Unidade: FCFRP
Subjects: ÓXIDO NÍTRICO, PARACOCCIDIOIDES BRASILIENSIS, ANTÍGENOS, PULMÃO, MACRÓFAGOS
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OLIVEIRA, Stephan Alberto Machado de et al. β2 integrin-mediated susceptibility to Paracoccidioides brasiliensis experimental infection in mice. Frontiers in Cellular and Infection Microbiology, v. 11, p. 1-11, 2021Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2021.622899. Acesso em: 11 nov. 2024.
APA
Oliveira, S. A. M. de, Reis, J. N., Catão, E., Amaral, A. C., Souza, A. C. O., Ribeiro, A. M., et al. (2021). β2 integrin-mediated susceptibility to Paracoccidioides brasiliensis experimental infection in mice. Frontiers in Cellular and Infection Microbiology, 11, 1-11. doi:10.3389/fcimb.2021.622899
NLM
Oliveira SAM de, Reis JN, Catão E, Amaral AC, Souza ACO, Ribeiro AM, Faccioli LH, Carneiro FP, Marina CLF, Bürgel PH, Fernandes L, Tavares AH, Bocca AL. β2 integrin-mediated susceptibility to Paracoccidioides brasiliensis experimental infection in mice [Internet]. Frontiers in Cellular and Infection Microbiology. 2021 ; 11 1-11.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3389/fcimb.2021.622899
Vancouver
Oliveira SAM de, Reis JN, Catão E, Amaral AC, Souza ACO, Ribeiro AM, Faccioli LH, Carneiro FP, Marina CLF, Bürgel PH, Fernandes L, Tavares AH, Bocca AL. β2 integrin-mediated susceptibility to Paracoccidioides brasiliensis experimental infection in mice [Internet]. Frontiers in Cellular and Infection Microbiology. 2021 ; 11 1-11.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3389/fcimb.2021.622899
Artigo de periodico
A leukotriene-dependent spleen-liver axis drives TNF production in systemic inflammation (2021)
Fonseca, Monique Thaís Costa ; Moretti, Eduardo Hermógenes ; Marques, Lucas Marciel Mauriz ; Machado, Bianca de Freitas ; Brito, Camila de Fátima Carvalho; Silva, Jady Tamaio Guedes da ; Komegae, Evilin Naname ; Vieira, Thayna dos Santos ; Festuccia, William Tadeu Lara ; Lopes, Norberto Peporine ; Steiner, Alexandre Alarcon
Source: Science Signaling. Unidades: ICB, FCFRP
Subjects: IMUNOLOGIA, FISIOLOGIA, LEUCOTRIENOS, CITOCINAS, LIPOPOLISSACARÍDEOS, ESPLENECTOMIA, BAÇO, FÍGADO, ESPECTROMETRIA DE MASSAS, MACRÓFAGOS
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FONSECA, Monique Thaís Costa et al. A leukotriene-dependent spleen-liver axis drives TNF production in systemic inflammation. Science Signaling, v. 14, n. 679, p. 1-13, 2021Tradução . . Disponível em: https://doi.org/10.1126/scisignal.abb0969. Acesso em: 11 nov. 2024.
APA
Fonseca, M. T. C., Moretti, E. H., Marques, L. M. M., Machado, B. de F., Brito, C. de F. C., Silva, J. T. G. da, et al. (2021). A leukotriene-dependent spleen-liver axis drives TNF production in systemic inflammation. Science Signaling, 14( 679), 1-13. doi:10.1126/scisignal.abb0969
NLM
Fonseca MTC, Moretti EH, Marques LMM, Machado B de F, Brito C de FC, Silva JTG da, Komegae EN, Vieira T dos S, Festuccia WTL, Lopes NP, Steiner AA. A leukotriene-dependent spleen-liver axis drives TNF production in systemic inflammation [Internet]. Science Signaling. 2021 ; 14( 679): 1-13.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1126/scisignal.abb0969
Vancouver
Fonseca MTC, Moretti EH, Marques LMM, Machado B de F, Brito C de FC, Silva JTG da, Komegae EN, Vieira T dos S, Festuccia WTL, Lopes NP, Steiner AA. A leukotriene-dependent spleen-liver axis drives TNF production in systemic inflammation [Internet]. Science Signaling. 2021 ; 14( 679): 1-13.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1126/scisignal.abb0969
Artigo de periodico
Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity (2021)
Nascimento, Daniele Carvalho ; Viacava, Paula Ramos; Ferreira, Raphael Gomes; Damaceno, Marina Alves; Piñeros, Annie Rocío Alvarez ; Melo, Paulo Henrique de; Donate, Paula Barbim ; Toller-Kawahisa, Juliana Escher; Zoppi, Daniel ; Veras, Flávio Protásio ; Peres, Raphael S. ; Silva, Luisa Menezes ; Caetité, Diego ; Oliveira, Antonio Edson Rocha; Castro, Icaro Maia Santos ; Kauffenstein, Gilles ; Nakaya, Helder Takashi Imoto ; Borges, Marcos de Carvalho ; Zamboni, Dario Simões ; Fonseca, Denise Morais da ; Paschoal, Jonas Augusto Rizzato ; Cunha, Thiago Mattar ; Quesniaux, Valerie ; Linden, Joel ; Cunha, Fernando Queiróz ; Ryffe, Bernhard; Alves-Filho, José Carlos
Source: Immunity. Unidades: FMRP, FCFRP, FCF, ICB, BIOINFORMÁTICA
Subjects: ADENOSINA, FÁRMACOS IMUNOSSUPRESSORES, SEPSE, CAMUNDONGOS, INTERLEUCINA 10, MACRÓFAGOS
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ABNT
NASCIMENTO, Daniele Carvalho et al. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity. Immunity, v. 54, n. 9, p. 2024-2041, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.immuni.2021.08.005. Acesso em: 11 nov. 2024.
APA
Nascimento, D. C., Viacava, P. R., Ferreira, R. G., Damaceno, M. A., Piñeros, A. R. A., Melo, P. H. de, et al. (2021). Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity. Immunity, 54( 9), 2024-2041. doi:10.1016/j.immuni.2021.08.005
NLM
Nascimento DC, Viacava PR, Ferreira RG, Damaceno MA, Piñeros ARA, Melo PH de, Donate PB, Toller-Kawahisa JE, Zoppi D, Veras FP, Peres RS, Silva LM, Caetité D, Oliveira AER, Castro IMS, Kauffenstein G, Nakaya HTI, Borges M de C, Zamboni DS, Fonseca DM da, Paschoal JAR, Cunha TM, Quesniaux V, Linden J, Cunha FQ, Ryffe B, Alves-Filho JC. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity [Internet]. Immunity. 2021 ; 54( 9): 2024-2041.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.immuni.2021.08.005
Vancouver
Nascimento DC, Viacava PR, Ferreira RG, Damaceno MA, Piñeros ARA, Melo PH de, Donate PB, Toller-Kawahisa JE, Zoppi D, Veras FP, Peres RS, Silva LM, Caetité D, Oliveira AER, Castro IMS, Kauffenstein G, Nakaya HTI, Borges M de C, Zamboni DS, Fonseca DM da, Paschoal JAR, Cunha TM, Quesniaux V, Linden J, Cunha FQ, Ryffe B, Alves-Filho JC. Sepsis expands a CD39 + plasmablast population that promotes immunosuppression via adenosine-mediated inhibition of macrophage antimicrobial activity [Internet]. Immunity. 2021 ; 54( 9): 2024-2041.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.immuni.2021.08.005
Artigo de periodico
Decitabine promotes modulation in phenotype and function of monocytes and macrophages that drive immune response regulation (2021)
Zambuzi, Fabiana Albani ; Silva, Priscilla Mariane Cardoso; Castro, Ricardo Cardoso ; Fontanari, Caroline ; Emery, Flavio da Silva ; Frantz, Fabiani Gai
Source: Cells. Unidades: FCFRP, FMRP
Subjects: FAGOCITOSE, MONÓCITOS, MACRÓFAGOS, ADJUVANTES IMUNOLÓGICOS, SISTEMA IMUNE, IMUNOLOGIA, FENÓTIPOS, DOENÇAS INFECCIOSAS
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ABNT
ZAMBUZI, Fabiana Albani et al. Decitabine promotes modulation in phenotype and function of monocytes and macrophages that drive immune response regulation. Cells, v. 10, n. 4, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10040868. Acesso em: 11 nov. 2024.
APA
Zambuzi, F. A., Silva, P. M. C., Castro, R. C., Fontanari, C., Emery, F. da S., & Frantz, F. G. (2021). Decitabine promotes modulation in phenotype and function of monocytes and macrophages that drive immune response regulation. Cells, 10( 4), 1-16. doi:10.3390/cells10040868
NLM
Zambuzi FA, Silva PMC, Castro RC, Fontanari C, Emery F da S, Frantz FG. Decitabine promotes modulation in phenotype and function of monocytes and macrophages that drive immune response regulation [Internet]. Cells. 2021 ; 10( 4): 1-16.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3390/cells10040868
Vancouver
Zambuzi FA, Silva PMC, Castro RC, Fontanari C, Emery F da S, Frantz FG. Decitabine promotes modulation in phenotype and function of monocytes and macrophages that drive immune response regulation [Internet]. Cells. 2021 ; 10( 4): 1-16.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3390/cells10040868
Artigo de periodico
Cytotoxicity and inflammatory mediators release by macrophages exposed to real seal XT and sealapex xpress (2021)
Silva, Léa Assed Bezerra da ; Hidalgo, Lidia Regina da Costa; Sousa Neto, Manoel Damião de ; Arnez, Maya Fernanda Manfrin; Barnett, Frederic; Hernández, Patricia María Gatón; Faccioli, Lucia Helena ; Paula-Silva, Francisco Wanderley Garcia de
Source: Brazilian Dental Journal. Unidades: FORP, FCFRP
Subjects: CITOTOXICIDADE IMUNOLÓGICA, MACRÓFAGOS, CITOCINAS
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ABNT
SILVA, Léa Assed Bezerra da et al. Cytotoxicity and inflammatory mediators release by macrophages exposed to real seal XT and sealapex xpress. Brazilian Dental Journal, v. 32, n. 1, p. 48-52, 2021Tradução . . Disponível em: https://doi.org/10.1590/0103-6440202103330. Acesso em: 11 nov. 2024.
APA
Silva, L. A. B. da, Hidalgo, L. R. da C., Sousa Neto, M. D. de, Arnez, M. F. M., Barnett, F., Hernández, P. M. G., et al. (2021). Cytotoxicity and inflammatory mediators release by macrophages exposed to real seal XT and sealapex xpress. Brazilian Dental Journal, 32( 1), 48-52. doi:10.1590/0103-6440202103330
NLM
Silva LAB da, Hidalgo LR da C, Sousa Neto MD de, Arnez MFM, Barnett F, Hernández PMG, Faccioli LH, Paula-Silva FWG de. Cytotoxicity and inflammatory mediators release by macrophages exposed to real seal XT and sealapex xpress [Internet]. Brazilian Dental Journal. 2021 ; 32( 1): 48-52.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1590/0103-6440202103330
Vancouver
Silva LAB da, Hidalgo LR da C, Sousa Neto MD de, Arnez MFM, Barnett F, Hernández PMG, Faccioli LH, Paula-Silva FWG de. Cytotoxicity and inflammatory mediators release by macrophages exposed to real seal XT and sealapex xpress [Internet]. Brazilian Dental Journal. 2021 ; 32( 1): 48-52.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1590/0103-6440202103330
Artigo de periodico
AeMOPE-1, a novel salivary peptide from Aedes aegypti, selectively modulates activation of murine macrophages and ameliorates experimental colitis (2021)
Lara, Priscila Guirão; Esteves, Eliane Virgínia da Silva ; Campos, Helioswilton Sales de; Assis, Josiane Betim de ; Henrique, Maressa de Oliveira ; Barros, Michele Silva de; Santos Neto, Leila; Silva Junior, Pedro I. ; Martins, Joilson de Oliveira ; Cardoso, Cristina Ribeiro de Barros; Ribeiro, José M. C.; Nunes, Anderson de Sá
Source: Frontiers in Immunology. Unidades: ICB, FCF, FCFRP
Subjects: IMUNOLOGIA, PEPTÍDEOS, AEDES, GLÂNDULAS SALIVARES, MACRÓFAGOS, COLITE, ADJUVANTES IMUNOLÓGICOS, SALIVA, ÓXIDO NÍTRICO, CITOCINAS
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ABNT
LARA, Priscila Guirão et al. AeMOPE-1, a novel salivary peptide from Aedes aegypti, selectively modulates activation of murine macrophages and ameliorates experimental colitis. Frontiers in Immunology, v. 12, p. 1-15, 2021Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2021.681671. Acesso em: 11 nov. 2024.
APA
Lara, P. G., Esteves, E. V. da S., Campos, H. S. de, Assis, J. B. de, Henrique, M. de O., Barros, M. S. de, et al. (2021). AeMOPE-1, a novel salivary peptide from Aedes aegypti, selectively modulates activation of murine macrophages and ameliorates experimental colitis. Frontiers in Immunology, 12, 1-15. doi:10.3389/fimmu.2021.681671
NLM
Lara PG, Esteves EV da S, Campos HS de, Assis JB de, Henrique M de O, Barros MS de, Santos Neto L, Silva Junior PI, Martins J de O, Cardoso CR de B, Ribeiro JMC, Nunes A de S. AeMOPE-1, a novel salivary peptide from Aedes aegypti, selectively modulates activation of murine macrophages and ameliorates experimental colitis [Internet]. Frontiers in Immunology. 2021 ; 12 1-15.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3389/fimmu.2021.681671
Vancouver
Lara PG, Esteves EV da S, Campos HS de, Assis JB de, Henrique M de O, Barros MS de, Santos Neto L, Silva Junior PI, Martins J de O, Cardoso CR de B, Ribeiro JMC, Nunes A de S. AeMOPE-1, a novel salivary peptide from Aedes aegypti, selectively modulates activation of murine macrophages and ameliorates experimental colitis [Internet]. Frontiers in Immunology. 2021 ; 12 1-15.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3389/fimmu.2021.681671
Trabalho de evento-resumo periodico
CD18 regulates monocyte-macrophage DC progenitors and the differentiation into alternatively activated macrophages during schistosomiasis (2021)
Souza, Camila O. S.; Oliveira, Jefferson Elias ; Fontanari, Caroline ; Rodriguez, Vanderlei; Gardinassi, Luiz; Faccioli, Lucia Helena
Source: The Journal of Immunology. Conference titles: Immunology 2021™ Meeting Abstracts. Unidades: FCFRP, FMRP
Subjects: SCHISTOSOMA MANSONI, DOENÇAS PARASITÁRIAS, MACRÓFAGOS, ESQUISTOSSOMOSE
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ABNT
SOUZA, Camila O. S. et al. CD18 regulates monocyte-macrophage DC progenitors and the differentiation into alternatively activated macrophages during schistosomiasis. The Journal of Immunology. Rockville: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.4049/jimmunol.206.Supp.99.29. Acesso em: 11 nov. 2024. , 2021
APA
Souza, C. O. S., Oliveira, J. E., Fontanari, C., Rodriguez, V., Gardinassi, L., & Faccioli, L. H. (2021). CD18 regulates monocyte-macrophage DC progenitors and the differentiation into alternatively activated macrophages during schistosomiasis. The Journal of Immunology. Rockville: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. doi:10.4049/jimmunol.206.Supp.99.29
NLM
Souza COS, Oliveira JE, Fontanari C, Rodriguez V, Gardinassi L, Faccioli LH. CD18 regulates monocyte-macrophage DC progenitors and the differentiation into alternatively activated macrophages during schistosomiasis [Internet]. The Journal of Immunology. 2021 ; 206[citado 2024 nov. 11 ] Available from: https://doi.org/10.4049/jimmunol.206.Supp.99.29
Vancouver
Souza COS, Oliveira JE, Fontanari C, Rodriguez V, Gardinassi L, Faccioli LH. CD18 regulates monocyte-macrophage DC progenitors and the differentiation into alternatively activated macrophages during schistosomiasis [Internet]. The Journal of Immunology. 2021 ; 206[citado 2024 nov. 11 ] Available from: https://doi.org/10.4049/jimmunol.206.Supp.99.29
Artigo de periodico
M1 and M2 macrophages phenotypes modulation after stimuli with materials used in endodontic treatment (2021)
Segato, Raquel Assed Bezerra ; Silva, Léa Assed Bezerra da ; Gabriel-Junior, Edson Alves; Sorgi, Carlos Arterio ; Faccioli, Lucia Helena ; Massoni, Vivian Vicentin; Nelson Filho, Paulo ; Pucinelli, Carolina Maschietto
Source: Brazilian Dental Journal. Unidades: FORP, FFCLRP, FCFRP
Subjects: RESINAS EPOXI, MACRÓFAGOS, FENÓTIPOS, MATERIAIS DENTÁRIOS, OBTURAÇÃO DO CANAL RADICULAR
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ABNT
SEGATO, Raquel Assed Bezerra et al. M1 and M2 macrophages phenotypes modulation after stimuli with materials used in endodontic treatment. Brazilian Dental Journal, v. 32, n. 3, p. 32-43, 2021Tradução . . Disponível em: https://doi.org/10.1590/0103-6440202104038. Acesso em: 11 nov. 2024.
APA
Segato, R. A. B., Silva, L. A. B. da, Gabriel-Junior, E. A., Sorgi, C. A., Faccioli, L. H., Massoni, V. V., et al. (2021). M1 and M2 macrophages phenotypes modulation after stimuli with materials used in endodontic treatment. Brazilian Dental Journal, 32( 3), 32-43. doi:10.1590/0103-6440202104038
NLM
Segato RAB, Silva LAB da, Gabriel-Junior EA, Sorgi CA, Faccioli LH, Massoni VV, Nelson Filho P, Pucinelli CM. M1 and M2 macrophages phenotypes modulation after stimuli with materials used in endodontic treatment [Internet]. Brazilian Dental Journal. 2021 ; 32( 3): 32-43.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1590/0103-6440202104038
Vancouver
Segato RAB, Silva LAB da, Gabriel-Junior EA, Sorgi CA, Faccioli LH, Massoni VV, Nelson Filho P, Pucinelli CM. M1 and M2 macrophages phenotypes modulation after stimuli with materials used in endodontic treatment [Internet]. Brazilian Dental Journal. 2021 ; 32( 3): 32-43.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1590/0103-6440202104038

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