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Artigo de periodico
Selective regulation of hepatic lipid metabolism by the AMP-activated protein kinase pathway in late-pregnant rats (2014)
Rodrigues, Sandra C.; Pantaleão, Lucas Carminatti; Nogueira, Tatiane Cristina de Araujo; Gomes, Patrícia R.; Albuquerque, Gabriela G.; Nachbar, Renato Tadeu; Torres-Leal, Francisco Leonardo; Caperuto, Luciana Chagas; Lellis-Santos, Camilo; Anhê, Gabriel F0rato; Bordin, Silvana
Source: American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. Unidade: ICB
Subjects: FISIOLOGIA, METABOLISMO ANIMAL, PROTEINAS QUINASES, RATOS
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ABNT
RODRIGUES, Sandra C. et al. Selective regulation of hepatic lipid metabolism by the AMP-activated protein kinase pathway in late-pregnant rats. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, v. 307, n. 9, p. R1146-R1156, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00513.2013. Acesso em: 20 nov. 2024.
APA
Rodrigues, S. C., Pantaleão, L. C., Nogueira, T. C. de A., Gomes, P. R., Albuquerque, G. G., Nachbar, R. T., et al. (2014). Selective regulation of hepatic lipid metabolism by the AMP-activated protein kinase pathway in late-pregnant rats. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, 307( 9), R1146-R1156. doi:10.1152/ajpregu.00513.2013
NLM
Rodrigues SC, Pantaleão LC, Nogueira TC de A, Gomes PR, Albuquerque GG, Nachbar RT, Torres-Leal FL, Caperuto LC, Lellis-Santos C, Anhê GF0rato, Bordin S. Selective regulation of hepatic lipid metabolism by the AMP-activated protein kinase pathway in late-pregnant rats [Internet]. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 2014 ; 307( 9): R1146-R1156.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1152/ajpregu.00513.2013
Vancouver
Rodrigues SC, Pantaleão LC, Nogueira TC de A, Gomes PR, Albuquerque GG, Nachbar RT, Torres-Leal FL, Caperuto LC, Lellis-Santos C, Anhê GF0rato, Bordin S. Selective regulation of hepatic lipid metabolism by the AMP-activated protein kinase pathway in late-pregnant rats [Internet]. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 2014 ; 307( 9): R1146-R1156.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1152/ajpregu.00513.2013
Artigo de periodico
Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation (2014)
Gomes, Patrícia R.; Graciano, Maria Fernanda; Pantaleão, Lucas Carminatti; Rennó, André L.; Rodrigues, Sandra Campos; Velloso, Lício Augusto; Latorraca, Márcia Q.; Carpinelli, Angelo Rafael ; Anhê, Gabriel F.; Bordin, Silvana
Source: American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology. Unidade: ICB
Subjects: FISIOLOGIA, GLICOSE, ESTERÓIDES
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ABNT
GOMES, Patrícia R. et al. Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation. American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology, v. 306, n. 1, p. E109-E120, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00364.2013. Acesso em: 20 nov. 2024.
APA
Gomes, P. R., Graciano, M. F., Pantaleão, L. C., Rennó, A. L., Rodrigues, S. C., Velloso, L. A., et al. (2014). Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation. American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology, 306( 1), E109-E120. doi:10.1152/ajpendo.00364.2013
NLM
Gomes PR, Graciano MF, Pantaleão LC, Rennó AL, Rodrigues SC, Velloso LA, Latorraca MQ, Carpinelli AR, Anhê GF, Bordin S. Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation [Internet]. American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology. 2014 ; 306( 1): E109-E120.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1152/ajpendo.00364.2013
Vancouver
Gomes PR, Graciano MF, Pantaleão LC, Rennó AL, Rodrigues SC, Velloso LA, Latorraca MQ, Carpinelli AR, Anhê GF, Bordin S. Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation [Internet]. American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology. 2014 ; 306( 1): E109-E120.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1152/ajpendo.00364.2013
Artigo de periodico
Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats (2013)
Faria, Juliana A.; Kinote, Andrezza; Ignacio-Souza, Letícia M.; Araújo, Thiago M. de; Razolli, Daniela S.; Doneda, Diego L.; Paschoal, Lívia B.; Lellis-Santos, Camilo; Bertolini, Gisele L.; Velloso, Lício A.; Bordin, Silvana ; Anhê, Gabriel F.
Source: American Journal of Physiology, Endocrinology and Methabolism. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
FARIA, Juliana A. et al. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats. American Journal of Physiology, Endocrinology and Methabolism, v. 305, n. 2, p. E230-E242, 2013Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00094.2013. Acesso em: 20 nov. 2024.
APA
Faria, J. A., Kinote, A., Ignacio-Souza, L. M., Araújo, T. M. de, Razolli, D. S., Doneda, D. L., et al. (2013). Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats. American Journal of Physiology, Endocrinology and Methabolism, 305( 2), E230-E242. doi:10.1152/ajpendo.00094.2013
NLM
Faria JA, Kinote A, Ignacio-Souza LM, Araújo TM de, Razolli DS, Doneda DL, Paschoal LB, Lellis-Santos C, Bertolini GL, Velloso LA, Bordin S, Anhê GF. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats [Internet]. American Journal of Physiology, Endocrinology and Methabolism. 2013 ; 305( 2): E230-E242.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1152/ajpendo.00094.2013
Vancouver
Faria JA, Kinote A, Ignacio-Souza LM, Araújo TM de, Razolli DS, Doneda DL, Paschoal LB, Lellis-Santos C, Bertolini GL, Velloso LA, Bordin S, Anhê GF. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats [Internet]. American Journal of Physiology, Endocrinology and Methabolism. 2013 ; 305( 2): E230-E242.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1152/ajpendo.00094.2013
Artigo de periodico
Metformin attenuates the exacerbation of the allergic eosinophilic inflammation in high fat-diet-induced obesity in mice (2013)
Calixto, Marina Ciarallo; Lintomen, Letícia; André, Diana Majoli; Leiria, Luiz Osório; Ferreira, Danilo; Lellis-Santos, Camilo; Anhê, Gabriel Forato; Bordin, Silvana ; Landgraf, Richardt Gama; Antunes, Edson
Source: PLOS ONE. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
CALIXTO, Marina Ciarallo et al. Metformin attenuates the exacerbation of the allergic eosinophilic inflammation in high fat-diet-induced obesity in mice. PLOS ONE, v. 8, n. 10, p. 01-13, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0076786. Acesso em: 20 nov. 2024.
APA
Calixto, M. C., Lintomen, L., André, D. M., Leiria, L. O., Ferreira, D., Lellis-Santos, C., et al. (2013). Metformin attenuates the exacerbation of the allergic eosinophilic inflammation in high fat-diet-induced obesity in mice. PLOS ONE, 8( 10), 01-13. doi:10.1371/journal.pone.0076786
NLM
Calixto MC, Lintomen L, André DM, Leiria LO, Ferreira D, Lellis-Santos C, Anhê GF, Bordin S, Landgraf RG, Antunes E. Metformin attenuates the exacerbation of the allergic eosinophilic inflammation in high fat-diet-induced obesity in mice [Internet]. PLOS ONE. 2013 ; 8( 10): 01-13.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1371/journal.pone.0076786
Vancouver
Calixto MC, Lintomen L, André DM, Leiria LO, Ferreira D, Lellis-Santos C, Anhê GF, Bordin S, Landgraf RG, Antunes E. Metformin attenuates the exacerbation of the allergic eosinophilic inflammation in high fat-diet-induced obesity in mice [Internet]. PLOS ONE. 2013 ; 8( 10): 01-13.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1371/journal.pone.0076786
Artigo de periodico
The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion (2012)
Lellis-Santos, Camilo; Sakamoto, Luciano H.; Bromati, Carla Rodrigues; Nogueira, Tatiane Cristina de Araújo; Leite, Adriana R.; Yamanaka, Tatiana S.; Kinote, Andrezza; Anhê, Gabriel F.; Bordin, Silvana
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
LELLIS-SANTOS, Camilo et al. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion. Endocrinology, v. 153, n. 8, p. 3668-3678, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1135. Acesso em: 20 nov. 2024.
APA
Lellis-Santos, C., Sakamoto, L. H., Bromati, C. R., Nogueira, T. C. de A., Leite, A. R., Yamanaka, T. S., et al. (2012). The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion. Endocrinology, 153( 8), 3668-3678. doi:10.1210/en.2012-1135
NLM
Lellis-Santos C, Sakamoto LH, Bromati CR, Nogueira TC de A, Leite AR, Yamanaka TS, Kinote A, Anhê GF, Bordin S. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion [Internet]. Endocrinology. 2012 ; 153( 8): 3668-3678.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1210/en.2012-1135
Vancouver
Lellis-Santos C, Sakamoto LH, Bromati CR, Nogueira TC de A, Leite AR, Yamanaka TS, Kinote A, Anhê GF, Bordin S. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion [Internet]. Endocrinology. 2012 ; 153( 8): 3668-3678.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1210/en.2012-1135
Artigo de periodico
Maternal melatonin programs the daily pattern of energy metabolism in adult offspring (2012)
Ferreira, Danilo S.; Amaral, Fernanda Gaspar do; Mesquita, Caroline C.; Barbosa, Ana Paula L.; Lellis-Santos, Camilo; Turati, Ariane de Oliveira; Santos, Laila Romagueira Bichara; Sollon. Carolina S.; Gomes, Patricia Rodrigues Lourenço; Faria, Juliana A.; Cipolla-Neto, José; Bordin, Silvana ; Anhê, Gabriel F.
Source: PLOS ONE. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
FERREIRA, Danilo S. et al. Maternal melatonin programs the daily pattern of energy metabolism in adult offspring. PLOS ONE, v. 7, n. 6, p. 1-10, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0038795. Acesso em: 20 nov. 2024.
APA
Ferreira, D. S., Amaral, F. G. do, Mesquita, C. C., Barbosa, A. P. L., Lellis-Santos, C., Turati, A. de O., et al. (2012). Maternal melatonin programs the daily pattern of energy metabolism in adult offspring. PLOS ONE, 7( 6), 1-10. doi:10.1371/journal.pone.0038795
NLM
Ferreira DS, Amaral FG do, Mesquita CC, Barbosa APL, Lellis-Santos C, Turati A de O, Santos LRB, Sollon. Carolina S., Gomes PRL, Faria JA, Cipolla-Neto J, Bordin S, Anhê GF. Maternal melatonin programs the daily pattern of energy metabolism in adult offspring [Internet]. PLOS ONE. 2012 ; 7( 6): 1-10.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1371/journal.pone.0038795
Vancouver
Ferreira DS, Amaral FG do, Mesquita CC, Barbosa APL, Lellis-Santos C, Turati A de O, Santos LRB, Sollon. Carolina S., Gomes PRL, Faria JA, Cipolla-Neto J, Bordin S, Anhê GF. Maternal melatonin programs the daily pattern of energy metabolism in adult offspring [Internet]. PLOS ONE. 2012 ; 7( 6): 1-10.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1371/journal.pone.0038795
Artigo de periodico
Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels (2012)
Kinote, Andrezza; Faria, Juliana A.; Roman, Erika A.; Solon, Carina; Razolli, Daniela S.; Ignacio-Souza, Letícia M.; Sollon, Carolina S.; Nascimento, Lucas F.; Araújo, Thiago M. de; Barbosa, Ana Paula L.; Lellis-Santos, Camilo; Velloso, Licio A.; Bordin, Silvana ; Anhê, Gabriel F.
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
KINOTE, Andrezza et al. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels. Endocrinology, v. 153, n. 8, p. 3633-3645, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1341. Acesso em: 20 nov. 2024.
APA
Kinote, A., Faria, J. A., Roman, E. A., Solon, C., Razolli, D. S., Ignacio-Souza, L. M., et al. (2012). Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels. Endocrinology, 153( 8), 3633-3645. doi:10.1210/en.2012-1341
NLM
Kinote A, Faria JA, Roman EA, Solon C, Razolli DS, Ignacio-Souza LM, Sollon CS, Nascimento LF, Araújo TM de, Barbosa APL, Lellis-Santos C, Velloso LA, Bordin S, Anhê GF. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels [Internet]. Endocrinology. 2012 ; 153( 8): 3633-3645.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1210/en.2012-1341
Vancouver
Kinote A, Faria JA, Roman EA, Solon C, Razolli DS, Ignacio-Souza LM, Sollon CS, Nascimento LF, Araújo TM de, Barbosa APL, Lellis-Santos C, Velloso LA, Bordin S, Anhê GF. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels [Internet]. Endocrinology. 2012 ; 153( 8): 3633-3645.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1210/en.2012-1341
Artigo de periodico
UPR induces transient burst of apoptosis in islets of early lactating rats through reduced AKT phosphorylation via ATF4/CHOP stimulation of TRB3 expression (2011)
Bromati, Carla Rodrigues; Santos, Camilo de Lellis; Yamanaka, Tatiana S.; Nogueira, Tatiane Cristina de Araújo; Leonelli, Mauro; Caperuto, Luciana C.; Gorjão, Renata; Leite, Adriana R.; Anhê, Gabriel F.; Bordin, Silvana
Source: American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BROMATI, Carla Rodrigues et al. UPR induces transient burst of apoptosis in islets of early lactating rats through reduced AKT phosphorylation via ATF4/CHOP stimulation of TRB3 expression. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, v. 300, n. 1, p. R92-R100, 2011Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00169.2010. Acesso em: 20 nov. 2024.
APA
Bromati, C. R., Santos, C. de L., Yamanaka, T. S., Nogueira, T. C. de A., Leonelli, M., Caperuto, L. C., et al. (2011). UPR induces transient burst of apoptosis in islets of early lactating rats through reduced AKT phosphorylation via ATF4/CHOP stimulation of TRB3 expression. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 300( 1), R92-R100. doi:10.1152/ajpregu.00169.2010
NLM
Bromati CR, Santos C de L, Yamanaka TS, Nogueira TC de A, Leonelli M, Caperuto LC, Gorjão R, Leite AR, Anhê GF, Bordin S. UPR induces transient burst of apoptosis in islets of early lactating rats through reduced AKT phosphorylation via ATF4/CHOP stimulation of TRB3 expression [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2011 ; 300( 1): R92-R100.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1152/ajpregu.00169.2010
Vancouver
Bromati CR, Santos C de L, Yamanaka TS, Nogueira TC de A, Leonelli M, Caperuto LC, Gorjão R, Leite AR, Anhê GF, Bordin S. UPR induces transient burst of apoptosis in islets of early lactating rats through reduced AKT phosphorylation via ATF4/CHOP stimulation of TRB3 expression [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2011 ; 300( 1): R92-R100.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1152/ajpregu.00169.2010
Artigo de periodico
Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response (2011)
Nogueira, Tatiane Cristina de Araújo; Santos, Camilo de Lellis; Jesus, Daniel Simões de; Taneda, Marco; Rodrigues, Sandra Campos; Amaral, Fernanda Gaspar do; Lopes, Ana Maria Souza; Cipolla Neto, José; Bordin, Silvana ; Anhê, Gabriel F.
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
NOGUEIRA, Tatiane Cristina de Araújo et al. Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response. Endocrinology, v. 152, n. 4, p. 1253-1263, 2011Tradução . . Disponível em: https://doi.org/10.1210/en.2010-1088. Acesso em: 20 nov. 2024.
APA
Nogueira, T. C. de A., Santos, C. de L., Jesus, D. S. de, Taneda, M., Rodrigues, S. C., Amaral, F. G. do, et al. (2011). Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response. Endocrinology, 152( 4), 1253-1263. doi:10.1210/en.2010-1088
NLM
Nogueira TC de A, Santos C de L, Jesus DS de, Taneda M, Rodrigues SC, Amaral FG do, Lopes AMS, Cipolla Neto J, Bordin S, Anhê GF. Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response [Internet]. Endocrinology. 2011 ; 152( 4): 1253-1263.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1210/en.2010-1088
Vancouver
Nogueira TC de A, Santos C de L, Jesus DS de, Taneda M, Rodrigues SC, Amaral FG do, Lopes AMS, Cipolla Neto J, Bordin S, Anhê GF. Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response [Internet]. Endocrinology. 2011 ; 152( 4): 1253-1263.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1210/en.2010-1088
Artigo de periodico
Smad5 regulates Akt2 expression and insulin-induced glucose uptake in L6 myotubes (2010)
Anhê, Fernando Forato; Lellis-Santos, Camilo; Leite, Adriana R.; Hirabara, Sandro M.; Boschero, Antonio C.; Curi, Rui; Anhê, Gabriel Forato; Bordin, Silvana
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
ANHÊ, Fernando Forato et al. Smad5 regulates Akt2 expression and insulin-induced glucose uptake in L6 myotubes. Molecular and Cellular Endocrinology, v. 319, n. 1/2, p. 30-8, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2010.01.003. Acesso em: 20 nov. 2024.
APA
Anhê, F. F., Lellis-Santos, C., Leite, A. R., Hirabara, S. M., Boschero, A. C., Curi, R., et al. (2010). Smad5 regulates Akt2 expression and insulin-induced glucose uptake in L6 myotubes. Molecular and Cellular Endocrinology, 319( 1/2), 30-8. doi:10.1016/j.mce.2010.01.003
NLM
Anhê FF, Lellis-Santos C, Leite AR, Hirabara SM, Boschero AC, Curi R, Anhê GF, Bordin S. Smad5 regulates Akt2 expression and insulin-induced glucose uptake in L6 myotubes [Internet]. Molecular and Cellular Endocrinology. 2010 ; 319( 1/2): 30-8.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1016/j.mce.2010.01.003
Vancouver
Anhê FF, Lellis-Santos C, Leite AR, Hirabara SM, Boschero AC, Curi R, Anhê GF, Bordin S. Smad5 regulates Akt2 expression and insulin-induced glucose uptake in L6 myotubes [Internet]. Molecular and Cellular Endocrinology. 2010 ; 319( 1/2): 30-8.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1016/j.mce.2010.01.003
Artigo de periodico
The role of proliferator-activated receptor \03B3 coactivator\20131\03B1 in the fatty-acid\2013dependent transcriptional control of interleukin-10 in hepatic cells of rodents (2010)
Morari, Joseane; Torsoni, Adriana S.; Anhê, Gabriel Forato; Roman, Erika A.; Cintra, Dennys E.; Ward, Laura S.; Bordin, Silvana ; Velloso, Lício A.
Source: Metabolism Clinical and Experimental. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MORARI, Joseane et al. The role of proliferator-activated receptor \03B3 coactivator\20131\03B1 in the fatty-acid\2013dependent transcriptional control of interleukin-10 in hepatic cells of rodents. Metabolism Clinical and Experimental, v. 59, n. 2, p. 215-223, 2010Tradução . . Acesso em: 20 nov. 2024.
APA
Morari, J., Torsoni, A. S., Anhê, G. F., Roman, E. A., Cintra, D. E., Ward, L. S., et al. (2010). The role of proliferator-activated receptor \03B3 coactivator\20131\03B1 in the fatty-acid\2013dependent transcriptional control of interleukin-10 in hepatic cells of rodents. Metabolism Clinical and Experimental, 59( 2), 215-223.
NLM
Morari J, Torsoni AS, Anhê GF, Roman EA, Cintra DE, Ward LS, Bordin S, Velloso LA. The role of proliferator-activated receptor \03B3 coactivator\20131\03B1 in the fatty-acid\2013dependent transcriptional control of interleukin-10 in hepatic cells of rodents. Metabolism Clinical and Experimental. 2010 ; 59( 2): 215-223.[citado 2024 nov. 20 ]
Vancouver
Morari J, Torsoni AS, Anhê GF, Roman EA, Cintra DE, Ward LS, Bordin S, Velloso LA. The role of proliferator-activated receptor \03B3 coactivator\20131\03B1 in the fatty-acid\2013dependent transcriptional control of interleukin-10 in hepatic cells of rodents. Metabolism Clinical and Experimental. 2010 ; 59( 2): 215-223.[citado 2024 nov. 20 ]
Artigo de periodico
MKP-1 mediates glucocorticoid-induced ERK1/2 dephosphorylation and reduction in pancreatic ß-cell proliferation in islets from early lactating mothers (2010)
Carvalho, José Edgar Nicoletti; Santos, Camilo de Lellis; Yamanaka, Tatiana S.; Nogueira, Tatiane Cristina de; Caperuto, Luciana C.; Leite, Adriana R.; Anhê, Gabriel F.; Bordin, Silvana
Source: American Journal of Physiology: Endocrinology and Metabolism. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO, José Edgar Nicoletti et al. MKP-1 mediates glucocorticoid-induced ERK1/2 dephosphorylation and reduction in pancreatic ß-cell proliferation in islets from early lactating mothers. American Journal of Physiology: Endocrinology and Metabolism, v. 299, n. 6, p. E1006-E1015, 2010Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00341.2010. Acesso em: 20 nov. 2024.
APA
Carvalho, J. E. N., Santos, C. de L., Yamanaka, T. S., Nogueira, T. C. de, Caperuto, L. C., Leite, A. R., et al. (2010). MKP-1 mediates glucocorticoid-induced ERK1/2 dephosphorylation and reduction in pancreatic ß-cell proliferation in islets from early lactating mothers. American Journal of Physiology: Endocrinology and Metabolism, 299( 6), E1006-E1015. doi:10.1152/ajpendo.00341.2010
NLM
Carvalho JEN, Santos C de L, Yamanaka TS, Nogueira TC de, Caperuto LC, Leite AR, Anhê GF, Bordin S. MKP-1 mediates glucocorticoid-induced ERK1/2 dephosphorylation and reduction in pancreatic ß-cell proliferation in islets from early lactating mothers [Internet]. American Journal of Physiology: Endocrinology and Metabolism. 2010 ; 299( 6): E1006-E1015.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1152/ajpendo.00341.2010
Vancouver
Carvalho JEN, Santos C de L, Yamanaka TS, Nogueira TC de, Caperuto LC, Leite AR, Anhê GF, Bordin S. MKP-1 mediates glucocorticoid-induced ERK1/2 dephosphorylation and reduction in pancreatic ß-cell proliferation in islets from early lactating mothers [Internet]. American Journal of Physiology: Endocrinology and Metabolism. 2010 ; 299( 6): E1006-E1015.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1152/ajpendo.00341.2010
Artigo de periodico
Saturated fatty acids produce an inflammatory response predominantly through the activation of TLR4 signaling in hypothalamus: implications for the pathogenesis of obesity (2009)
Milanski, Marciane; Degasperi, Giovanna; Coope, Andressa; Morari, Joseane; Denis, Raphael; Cintra, Dennys E.; Tsukumo, Daniela M. L.; Anhê, Gabriel Forato; Amaral, Maria E.; Takahashi, Hilton Kenji; Curi, Rui; Oliveira, Helena C.; Carvalheira, José B. C.; Bordin, Silvana ; Saad, Mário J.; Velloso, Lício A.
Source: Journal of Neuroscience. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MILANSKI, Marciane et al. Saturated fatty acids produce an inflammatory response predominantly through the activation of TLR4 signaling in hypothalamus: implications for the pathogenesis of obesity. Journal of Neuroscience, v. 29, n. 2, p. 359-370, 2009Tradução . . Acesso em: 20 nov. 2024.
APA
Milanski, M., Degasperi, G., Coope, A., Morari, J., Denis, R., Cintra, D. E., et al. (2009). Saturated fatty acids produce an inflammatory response predominantly through the activation of TLR4 signaling in hypothalamus: implications for the pathogenesis of obesity. Journal of Neuroscience, 29( 2), 359-370.
NLM
Milanski M, Degasperi G, Coope A, Morari J, Denis R, Cintra DE, Tsukumo DML, Anhê GF, Amaral ME, Takahashi HK, Curi R, Oliveira HC, Carvalheira JBC, Bordin S, Saad MJ, Velloso LA. Saturated fatty acids produce an inflammatory response predominantly through the activation of TLR4 signaling in hypothalamus: implications for the pathogenesis of obesity. Journal of Neuroscience. 2009 ; 29( 2): 359-370.[citado 2024 nov. 20 ]
Vancouver
Milanski M, Degasperi G, Coope A, Morari J, Denis R, Cintra DE, Tsukumo DML, Anhê GF, Amaral ME, Takahashi HK, Curi R, Oliveira HC, Carvalheira JBC, Bordin S, Saad MJ, Velloso LA. Saturated fatty acids produce an inflammatory response predominantly through the activation of TLR4 signaling in hypothalamus: implications for the pathogenesis of obesity. Journal of Neuroscience. 2009 ; 29( 2): 359-370.[citado 2024 nov. 20 ]
Artigo de periodico
Distinct subsets of hypothalamic genes are modulated by two different thermogenesis-inducing stimuli (2008)
De Souza, Cláudio T.; Pereira-da-Silva, Márcio; Araujo, Eliana P.; Morari, Joseane; Alvarez-Rojas, Fernanda; Bordin, Silvana ; Moreira-Filho, Djalma C.; Carvalheira, José B.; Saad, Mario J.; Velloso, Licio A.
Source: Obesity. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DE SOUZA, Cláudio T. et al. Distinct subsets of hypothalamic genes are modulated by two different thermogenesis-inducing stimuli. Obesity, v. 16, n. 6, p. 1239-1247, 2008Tradução . . Acesso em: 20 nov. 2024.
APA
De Souza, C. T., Pereira-da-Silva, M., Araujo, E. P., Morari, J., Alvarez-Rojas, F., Bordin, S., et al. (2008). Distinct subsets of hypothalamic genes are modulated by two different thermogenesis-inducing stimuli. Obesity, 16( 6), 1239-1247.
NLM
De Souza CT, Pereira-da-Silva M, Araujo EP, Morari J, Alvarez-Rojas F, Bordin S, Moreira-Filho DC, Carvalheira JB, Saad MJ, Velloso LA. Distinct subsets of hypothalamic genes are modulated by two different thermogenesis-inducing stimuli. Obesity. 2008 ; 16( 6): 1239-1247.[citado 2024 nov. 20 ]
Vancouver
De Souza CT, Pereira-da-Silva M, Araujo EP, Morari J, Alvarez-Rojas F, Bordin S, Moreira-Filho DC, Carvalheira JB, Saad MJ, Velloso LA. Distinct subsets of hypothalamic genes are modulated by two different thermogenesis-inducing stimuli. Obesity. 2008 ; 16( 6): 1239-1247.[citado 2024 nov. 20 ]
Artigo de periodico
Distinct regulation of IRS proteins in adipose tissue from obese aged and dexamethasone-treated rats (2006)
Caperuto, Luciana C.; Anhê, Gabriel Forato; Amanso, Angélica M.; Ribeiro, Luciene M.; Medina, Mayrin Correa; Souza, Lilian C.; Carvalho, Olga M. F.; Bordin, Silvana ; Saad, Mário J. A.; Carvalho, Carla Roberta de Oliveira
Source: Endocrine. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CAPERUTO, Luciana C. et al. Distinct regulation of IRS proteins in adipose tissue from obese aged and dexamethasone-treated rats. Endocrine, v. 113, n. 3, p. 391-398, 2006Tradução . . Acesso em: 20 nov. 2024.
APA
Caperuto, L. C., Anhê, G. F., Amanso, A. M., Ribeiro, L. M., Medina, M. C., Souza, L. C., et al. (2006). Distinct regulation of IRS proteins in adipose tissue from obese aged and dexamethasone-treated rats. Endocrine, 113( 3), 391-398.
NLM
Caperuto LC, Anhê GF, Amanso AM, Ribeiro LM, Medina MC, Souza LC, Carvalho OMF, Bordin S, Saad MJA, Carvalho CR de O. Distinct regulation of IRS proteins in adipose tissue from obese aged and dexamethasone-treated rats. Endocrine. 2006 ; 113( 3): 391-398.[citado 2024 nov. 20 ]
Vancouver
Caperuto LC, Anhê GF, Amanso AM, Ribeiro LM, Medina MC, Souza LC, Carvalho OMF, Bordin S, Saad MJA, Carvalho CR de O. Distinct regulation of IRS proteins in adipose tissue from obese aged and dexamethasone-treated rats. Endocrine. 2006 ; 113( 3): 391-398.[citado 2024 nov. 20 ]
Artigo de periodico
Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus (2005)
Souza, Cláudio T. de; Araujo, Eliana P.; Bordin, Silvana ; Ashimine, Rika; Zollner, Ricardo L.; Boschero, Antonio C.; Saad, Mário J. A.; Velloso, Lício Augusto
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA, Cláudio T. de et al. Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus. Endocrinology, v. 146, n. 10, p. 4192-4199, 2005Tradução . . Acesso em: 20 nov. 2024.
APA
Souza, C. T. de, Araujo, E. P., Bordin, S., Ashimine, R., Zollner, R. L., Boschero, A. C., et al. (2005). Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus. Endocrinology, 146( 10), 4192-4199.
NLM
Souza CT de, Araujo EP, Bordin S, Ashimine R, Zollner RL, Boschero AC, Saad MJA, Velloso LA. Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus. Endocrinology. 2005 ; 146( 10): 4192-4199.[citado 2024 nov. 20 ]
Vancouver
Souza CT de, Araujo EP, Bordin S, Ashimine R, Zollner RL, Boschero AC, Saad MJA, Velloso LA. Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus. Endocrinology. 2005 ; 146( 10): 4192-4199.[citado 2024 nov. 20 ]
Artigo de periodico
Prolactin-modulated gene expression profiles in pancreatic islets from adult female rats (2004)
Bordin, Silvana ; Amaral, Maria E. C.; Anhê, Gabriel Forato; Delghingaro-Augusto, Viviane; Cunha, Daniel A.; Nicoletti-Carvalho, José E.; Boschero, Antonio C.
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORDIN, Silvana et al. Prolactin-modulated gene expression profiles in pancreatic islets from adult female rats. Molecular and Cellular Endocrinology, v. 220, p. 41-50, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2004.04.001. Acesso em: 20 nov. 2024.
APA
Bordin, S., Amaral, M. E. C., Anhê, G. F., Delghingaro-Augusto, V., Cunha, D. A., Nicoletti-Carvalho, J. E., & Boschero, A. C. (2004). Prolactin-modulated gene expression profiles in pancreatic islets from adult female rats. Molecular and Cellular Endocrinology, 220, 41-50. doi:10.1016/j.mce.2004.04.001
NLM
Bordin S, Amaral MEC, Anhê GF, Delghingaro-Augusto V, Cunha DA, Nicoletti-Carvalho JE, Boschero AC. Prolactin-modulated gene expression profiles in pancreatic islets from adult female rats [Internet]. Molecular and Cellular Endocrinology. 2004 ; 220 41-50.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1016/j.mce.2004.04.001
Vancouver
Bordin S, Amaral MEC, Anhê GF, Delghingaro-Augusto V, Cunha DA, Nicoletti-Carvalho JE, Boschero AC. Prolactin-modulated gene expression profiles in pancreatic islets from adult female rats [Internet]. Molecular and Cellular Endocrinology. 2004 ; 220 41-50.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1016/j.mce.2004.04.001
Artigo de periodico
Blockade of IRS1 in isolated rat pancreatic islets improves glucose-induced insulin secretion (2002)
Araujo, Eliana P.; Amaral, Maria E C.; Souza, Claudio T.; Bordin, Silvana ; Ferreira, Fabiano; Saad, Mario J A.; Boschero, Antonio C.; Magalhães, Everardo C.; Velloso, Lício Augusto
Source: FEBS Letters. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARAUJO, Eliana P. et al. Blockade of IRS1 in isolated rat pancreatic islets improves glucose-induced insulin secretion. FEBS Letters, v. 531, p. 437-442, 2002Tradução . . Disponível em: https://doi.org/10.1016/s0014-5793(02)03580-9. Acesso em: 20 nov. 2024.
APA
Araujo, E. P., Amaral, M. E. C., Souza, C. T., Bordin, S., Ferreira, F., Saad, M. J. A., et al. (2002). Blockade of IRS1 in isolated rat pancreatic islets improves glucose-induced insulin secretion. FEBS Letters, 531, 437-442. doi:10.1016/s0014-5793(02)03580-9
NLM
Araujo EP, Amaral MEC, Souza CT, Bordin S, Ferreira F, Saad MJA, Boschero AC, Magalhães EC, Velloso LA. Blockade of IRS1 in isolated rat pancreatic islets improves glucose-induced insulin secretion [Internet]. FEBS Letters. 2002 ;531 437-442.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1016/s0014-5793(02)03580-9
Vancouver
Araujo EP, Amaral MEC, Souza CT, Bordin S, Ferreira F, Saad MJA, Boschero AC, Magalhães EC, Velloso LA. Blockade of IRS1 in isolated rat pancreatic islets improves glucose-induced insulin secretion [Internet]. FEBS Letters. 2002 ;531 437-442.[citado 2024 nov. 20 ] Available from: https://doi.org/10.1016/s0014-5793(02)03580-9

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