ReP USP - Resultado da busca

Artigo de periodico
Induction of oxidants distinguishes susceptibility of prostate carcinoma cell lines to p53 gene transfer mediated by an improved adenoviral vector (2017)
Esaki, Tamura Rodrigo; Hunger, Aline; Fernandes, Denise C.; Laurindo, Francisco R.; Costanzi-Strauss, Eugenia ; Strauss, Bryan E.
Source: Human Gene Therapy. Unidade: ICB
Assunto: HISTOLOGIA
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ABNT
ESAKI, Tamura Rodrigo et al. Induction of oxidants distinguishes susceptibility of prostate carcinoma cell lines to p53 gene transfer mediated by an improved adenoviral vector. Human Gene Therapy, v. 28, n. 8, p. 639-653, 2017Tradução . . Disponível em: https://doi.org/10.1089/hum.2016.139. Acesso em: 23 ago. 2024.
APA
Esaki, T. R., Hunger, A., Fernandes, D. C., Laurindo, F. R., Costanzi-Strauss, E., & Strauss, B. E. (2017). Induction of oxidants distinguishes susceptibility of prostate carcinoma cell lines to p53 gene transfer mediated by an improved adenoviral vector. Human Gene Therapy, 28( 8), 639-653. doi:10.1089/hum.2016.139
NLM
Esaki TR, Hunger A, Fernandes DC, Laurindo FR, Costanzi-Strauss E, Strauss BE. Induction of oxidants distinguishes susceptibility of prostate carcinoma cell lines to p53 gene transfer mediated by an improved adenoviral vector [Internet]. Human Gene Therapy. 2017 ; 28( 8): 639-653.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1089/hum.2016.139
Vancouver
Esaki TR, Hunger A, Fernandes DC, Laurindo FR, Costanzi-Strauss E, Strauss BE. Induction of oxidants distinguishes susceptibility of prostate carcinoma cell lines to p53 gene transfer mediated by an improved adenoviral vector [Internet]. Human Gene Therapy. 2017 ; 28( 8): 639-653.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1089/hum.2016.139
Artigo de periodico
Intratumoral Immunization by p19Arf and Interferon-β Gene Transfer in a Heterotopic Mouse Model of Lung Carcinoma (2016)
Catani, João Paulo Portela; Medrano, Ruan Felipe Vieira; Ribeiro, Aline Hunger; Valle, Paulo Roberto Del; Adjemian, Sandy; Zanatta, Daniela Bertolini; Kroemer, Guido; Costanzi-Strauss, Eugenia ; Strauss, Bryan Eric
Source: Translational Oncology. Unidade: ICB
Subjects: CARCINOMA, TRANSFERÊNCIA DE GENES, IMUNIZAÇÃO, IMUNOTERAPIA
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ABNT
CATANI, João Paulo Portela et al. Intratumoral Immunization by p19Arf and Interferon-β Gene Transfer in a Heterotopic Mouse Model of Lung Carcinoma. Translational Oncology, v. 9, n. 6, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.tranon.2016.09.011. Acesso em: 23 ago. 2024.
APA
Catani, J. P. P., Medrano, R. F. V., Ribeiro, A. H., Valle, P. R. D., Adjemian, S., Zanatta, D. B., et al. (2016). Intratumoral Immunization by p19Arf and Interferon-β Gene Transfer in a Heterotopic Mouse Model of Lung Carcinoma. Translational Oncology, 9( 6). doi:10.1016/j.tranon.2016.09.011
NLM
Catani JPP, Medrano RFV, Ribeiro AH, Valle PRD, Adjemian S, Zanatta DB, Kroemer G, Costanzi-Strauss E, Strauss BE. Intratumoral Immunization by p19Arf and Interferon-β Gene Transfer in a Heterotopic Mouse Model of Lung Carcinoma [Internet]. Translational Oncology. 2016 ; 9( 6):[citado 2024 ago. 23 ] Available from: https://doi.org/10.1016/j.tranon.2016.09.011
Vancouver
Catani JPP, Medrano RFV, Ribeiro AH, Valle PRD, Adjemian S, Zanatta DB, Kroemer G, Costanzi-Strauss E, Strauss BE. Intratumoral Immunization by p19Arf and Interferon-β Gene Transfer in a Heterotopic Mouse Model of Lung Carcinoma [Internet]. Translational Oncology. 2016 ; 9( 6):[citado 2024 ago. 23 ] Available from: https://doi.org/10.1016/j.tranon.2016.09.011
Artigo de periodico
Autoregulated expression of p53 from an adenoviral vector confers superior tumor inhibition in a model of prostate carcinoma gene therapy (2016)
Tamura, Rodrigo Esaki; Soares, Rafael Bento da Silva; Costanzi-Strauss, Eugenia ; Strauss, Bryan Eric
Source: Cancer Biology & Therapy. Unidade: ICB
Subjects: ADENOVÍRUS, APOPTOSE, CARCINOMA, PRÓSTATA
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ABNT
TAMURA, Rodrigo Esaki et al. Autoregulated expression of p53 from an adenoviral vector confers superior tumor inhibition in a model of prostate carcinoma gene therapy. Cancer Biology & Therapy, v. 17, n. 12, p. 1221-1230, 2016Tradução . . Disponível em: https://doi.org/10.1080/15384047.2016.1235655. Acesso em: 23 ago. 2024.
APA
Tamura, R. E., Soares, R. B. da S., Costanzi-Strauss, E., & Strauss, B. E. (2016). Autoregulated expression of p53 from an adenoviral vector confers superior tumor inhibition in a model of prostate carcinoma gene therapy. Cancer Biology & Therapy, 17( 12), 1221-1230. doi:10.1080/15384047.2016.1235655
NLM
Tamura RE, Soares RB da S, Costanzi-Strauss E, Strauss BE. Autoregulated expression of p53 from an adenoviral vector confers superior tumor inhibition in a model of prostate carcinoma gene therapy [Internet]. Cancer Biology & Therapy. 2016 ; 17( 12): 1221-1230.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1080/15384047.2016.1235655
Vancouver
Tamura RE, Soares RB da S, Costanzi-Strauss E, Strauss BE. Autoregulated expression of p53 from an adenoviral vector confers superior tumor inhibition in a model of prostate carcinoma gene therapy [Internet]. Cancer Biology & Therapy. 2016 ; 17( 12): 1221-1230.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1080/15384047.2016.1235655
Artigo de periodico
Activation of endogenous p53 by combined p19Arf gene transfer and nutlin-3 drug treatment modalities in the murine cell lines B16 and C6 (2010)
Merkel, Christian A.; Soares, Rafael Bento da Silva; Carvalho, Anna Carolina Pereira Vieira de; Zanatta, Daniela Bertolini; Bajgelman, Marcio C.; Fratini, Paula; Costanzi-Strauss, Eugenia ; Strauss, Bryan E.
Source: BMC Cancer. Unidade: ICB
Assunto: HISTOLOGIA
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ABNT
MERKEL, Christian A. et al. Activation of endogenous p53 by combined p19Arf gene transfer and nutlin-3 drug treatment modalities in the murine cell lines B16 and C6. BMC Cancer, v. 10, n. 316, 2010Tradução . . Disponível em: https://doi.org/10.1186/1471-2407-10-316. Acesso em: 23 ago. 2024.
APA
Merkel, C. A., Soares, R. B. da S., Carvalho, A. C. P. V. de, Zanatta, D. B., Bajgelman, M. C., Fratini, P., et al. (2010). Activation of endogenous p53 by combined p19Arf gene transfer and nutlin-3 drug treatment modalities in the murine cell lines B16 and C6. BMC Cancer, 10( 316). doi:10.1186/1471-2407-10-316
NLM
Merkel CA, Soares RB da S, Carvalho ACPV de, Zanatta DB, Bajgelman MC, Fratini P, Costanzi-Strauss E, Strauss BE. Activation of endogenous p53 by combined p19Arf gene transfer and nutlin-3 drug treatment modalities in the murine cell lines B16 and C6 [Internet]. BMC Cancer. 2010 ; 10( 316):[citado 2024 ago. 23 ] Available from: https://doi.org/10.1186/1471-2407-10-316
Vancouver
Merkel CA, Soares RB da S, Carvalho ACPV de, Zanatta DB, Bajgelman MC, Fratini P, Costanzi-Strauss E, Strauss BE. Activation of endogenous p53 by combined p19Arf gene transfer and nutlin-3 drug treatment modalities in the murine cell lines B16 and C6 [Internet]. BMC Cancer. 2010 ; 10( 316):[citado 2024 ago. 23 ] Available from: https://doi.org/10.1186/1471-2407-10-316
Artigo de periodico
Retinoic acid and VEGF delay smooth muscle relative to endothelial differentiation to coordinate inner and outer coronary vessel wall morphogenesis (2010)
Azambuja, Ana Paula; Portillo-Sánchez, Victor; Rodrigues, Mariliza V.; Omae, Samantha V.; Schechtman, Deborah ; Strauss, Bryan E.; Costanzi-Strauss, Eugenia ; Krieger, José Eduardo; Perez-Pomares, José M.; Xavier-Neto, José
Source: Circulation Research. Unidades: IQ, ICB, FM
Subjects: HISTOLOGIA, ÁCIDOS, MORFOGÊNESE, CORAÇÃO (CRESCIMENTO E DESENVOLVIMENTO), ENDOTÉLIO
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ABNT
AZAMBUJA, Ana Paula et al. Retinoic acid and VEGF delay smooth muscle relative to endothelial differentiation to coordinate inner and outer coronary vessel wall morphogenesis. Circulation Research, v. 107, n. 2, p. 204-216, 2010Tradução . . Disponível em: https://doi.org/10.1161/CIRCRESAHA.109.214650. Acesso em: 23 ago. 2024.
APA
Azambuja, A. P., Portillo-Sánchez, V., Rodrigues, M. V., Omae, S. V., Schechtman, D., Strauss, B. E., et al. (2010). Retinoic acid and VEGF delay smooth muscle relative to endothelial differentiation to coordinate inner and outer coronary vessel wall morphogenesis. Circulation Research, 107( 2), 204-216. doi:10.1161/CIRCRESAHA.109.214650
NLM
Azambuja AP, Portillo-Sánchez V, Rodrigues MV, Omae SV, Schechtman D, Strauss BE, Costanzi-Strauss E, Krieger JE, Perez-Pomares JM, Xavier-Neto J. Retinoic acid and VEGF delay smooth muscle relative to endothelial differentiation to coordinate inner and outer coronary vessel wall morphogenesis [Internet]. Circulation Research. 2010 ; 107( 2): 204-216.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1161/CIRCRESAHA.109.214650
Vancouver
Azambuja AP, Portillo-Sánchez V, Rodrigues MV, Omae SV, Schechtman D, Strauss BE, Costanzi-Strauss E, Krieger JE, Perez-Pomares JM, Xavier-Neto J. Retinoic acid and VEGF delay smooth muscle relative to endothelial differentiation to coordinate inner and outer coronary vessel wall morphogenesis [Internet]. Circulation Research. 2010 ; 107( 2): 204-216.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1161/CIRCRESAHA.109.214650
Artigo de periodico
A novel gene transfer strategy that combines promoter and transgene activities for improved tumor cell inhibition (2005)
Strauss, Bryan Eric; Bajgelman, Márcio Chaim; Costanzi-Strauss, Eugenia
Source: Cancer Gene Therapy. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
STRAUSS, Bryan Eric e BAJGELMAN, Márcio Chaim e COSTANZI-STRAUSS, Eugenia. A novel gene transfer strategy that combines promoter and transgene activities for improved tumor cell inhibition. Cancer Gene Therapy, v. 12, p. 935-946, 2005Tradução . . Disponível em: https://doi.org/10.1038/sj.cgt.7700846. Acesso em: 23 ago. 2024.
APA
Strauss, B. E., Bajgelman, M. C., & Costanzi-Strauss, E. (2005). A novel gene transfer strategy that combines promoter and transgene activities for improved tumor cell inhibition. Cancer Gene Therapy, 12, 935-946. doi:10.1038/sj.cgt.7700846
NLM
Strauss BE, Bajgelman MC, Costanzi-Strauss E. A novel gene transfer strategy that combines promoter and transgene activities for improved tumor cell inhibition [Internet]. Cancer Gene Therapy. 2005 ; 12 935-946.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1038/sj.cgt.7700846
Vancouver
Strauss BE, Bajgelman MC, Costanzi-Strauss E. A novel gene transfer strategy that combines promoter and transgene activities for improved tumor cell inhibition [Internet]. Cancer Gene Therapy. 2005 ; 12 935-946.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1038/sj.cgt.7700846
Trabalho de evento-resumo periodico
High-level expression of wild-type p53 from the novel p53-responsive pCLPG retrovirus halts the proliferation of tumor cells regardless of their p53 status (2004)
Strauss, Bryan E.; Bajgelman, Marcio C.; Costanzi-Strauss, Eugenia
Source: Molecular Therapy. Conference titles: Annual Meeting the American Society of Gene Therapy's. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
STRAUSS, Bryan E. e BAJGELMAN, Marcio C. e COSTANZI-STRAUSS, Eugenia. High-level expression of wild-type p53 from the novel p53-responsive pCLPG retrovirus halts the proliferation of tumor cells regardless of their p53 status. Molecular Therapy. San Diego: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 23 ago. 2024. , 2004
APA
Strauss, B. E., Bajgelman, M. C., & Costanzi-Strauss, E. (2004). High-level expression of wild-type p53 from the novel p53-responsive pCLPG retrovirus halts the proliferation of tumor cells regardless of their p53 status. Molecular Therapy. San Diego: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Strauss BE, Bajgelman MC, Costanzi-Strauss E. High-level expression of wild-type p53 from the novel p53-responsive pCLPG retrovirus halts the proliferation of tumor cells regardless of their p53 status. Molecular Therapy. 2004 ; 9 S34 res. 85.[citado 2024 ago. 23 ]
Vancouver
Strauss BE, Bajgelman MC, Costanzi-Strauss E. High-level expression of wild-type p53 from the novel p53-responsive pCLPG retrovirus halts the proliferation of tumor cells regardless of their p53 status. Molecular Therapy. 2004 ; 9 S34 res. 85.[citado 2024 ago. 23 ]

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