Filtros : "MENCK, CARLOS FREDERICO MARTINS" "Indexado na Base de Dados Index Medicus" Removidos: "PAULA, EDISON JOSE DE" "CDCC" Limpar

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  • Source: Journal of Cell Science. Unidade: ICB

    Subjects: MICROBIOLOGIA, APOPTOSE

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      ANDRADE-LIMA, Leonardo Carmo de e ANDRADE, Luciana N. e MENCK, Carlos Frederico Martins. ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways. Journal of Cell Science, v. 28, n. 1, p. 150-159, 2015Tradução . . Disponível em: https://doi.org/10.1242/jcs.161596. Acesso em: 04 jul. 2024.
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      Andrade-Lima, L. C. de, Andrade, L. N., & Menck, C. F. M. (2015). ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways. Journal of Cell Science, 28( 1), 150-159. doi:10.1242/jcs.161596
    • NLM

      Andrade-Lima LC de, Andrade LN, Menck CFM. ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways [Internet]. Journal of Cell Science. 2015 ; 28( 1): 150-159.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1242/jcs.161596
    • Vancouver

      Andrade-Lima LC de, Andrade LN, Menck CFM. ATR suppresses apoptosis after UVB irradiation by controlling both translesion synthesis and alternative tolerance pathways [Internet]. Journal of Cell Science. 2015 ; 28( 1): 150-159.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1242/jcs.161596
  • Source: DNA Repair. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      QUINET, Annabel et al. Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome. DNA Repair, v. 14, p. 27-38, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.dnarep.2013.12.005. Acesso em: 04 jul. 2024.
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      Quinet, A., Vessoni, A. T., Rocha, C. R. R., Gottifredi, V., Biard, D., Sarasin, A., et al. (2014). Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome. DNA Repair, 14, 27-38. doi:10.1016/j.dnarep.2013.12.005
    • NLM

      Quinet A, Vessoni AT, Rocha CRR, Gottifredi V, Biard D, Sarasin A, Menck CFM, Stary A. Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome [Internet]. DNA Repair. 2014 ; 14 27-38.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.dnarep.2013.12.005
    • Vancouver

      Quinet A, Vessoni AT, Rocha CRR, Gottifredi V, Biard D, Sarasin A, Menck CFM, Stary A. Gap-filling and bypass at the replication fork are both activemechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome [Internet]. DNA Repair. 2014 ; 14 27-38.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.dnarep.2013.12.005
  • Source: Pharmaceutical Biology. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      VERNHES, Marioly et al. Protective effect of a Phyllanthus orbicularis aqueous extract against UVB light in human cells. Pharmaceutical Biology, v. 51, n. 1, p. 1-7, 2013Tradução . . Disponível em: https://doi.org/10.3109/13880209.2012.695800. Acesso em: 04 jul. 2024.
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      Vernhes, M., González-Pumariega, M., Andrade, L., Schuch, A. P., Lima-Bessa, K. M. de, Menck, C. F. M., & Sánchez-Lamar, A. (2013). Protective effect of a Phyllanthus orbicularis aqueous extract against UVB light in human cells. Pharmaceutical Biology, 51( 1), 1-7. doi:10.3109/13880209.2012.695800
    • NLM

      Vernhes M, González-Pumariega M, Andrade L, Schuch AP, Lima-Bessa KM de, Menck CFM, Sánchez-Lamar A. Protective effect of a Phyllanthus orbicularis aqueous extract against UVB light in human cells [Internet]. Pharmaceutical Biology. 2013 ; 51( 1): 1-7.[citado 2024 jul. 04 ] Available from: https://doi.org/10.3109/13880209.2012.695800
    • Vancouver

      Vernhes M, González-Pumariega M, Andrade L, Schuch AP, Lima-Bessa KM de, Menck CFM, Sánchez-Lamar A. Protective effect of a Phyllanthus orbicularis aqueous extract against UVB light in human cells [Internet]. Pharmaceutical Biology. 2013 ; 51( 1): 1-7.[citado 2024 jul. 04 ] Available from: https://doi.org/10.3109/13880209.2012.695800
  • Source: Human Mutation. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      SOLTYS, Daniela Tathiana et al. Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress. Human Mutation, v. 34, n. 3, p. 481-489, 2013Tradução . . Disponível em: https://doi.org/10.1002/humu.22259. Acesso em: 04 jul. 2024.
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      Soltys, D. T., Rocha, C. R. R., Lerner, L. K., Souza, T. A. de, Munford, V., Cabral, F., et al. (2013). Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress. Human Mutation, 34( 3), 481-489. doi:10.1002/humu.22259
    • NLM

      Soltys DT, Rocha CRR, Lerner LK, Souza TA de, Munford V, Cabral F, Nardo T, Stefanini M, Sarasin A, Cabral-Neto JB, Menck CFM. Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress [Internet]. Human Mutation. 2013 ; 34( 3): 481-489.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1002/humu.22259
    • Vancouver

      Soltys DT, Rocha CRR, Lerner LK, Souza TA de, Munford V, Cabral F, Nardo T, Stefanini M, Sarasin A, Cabral-Neto JB, Menck CFM. Novel XPG (ERCC5) mutations affect DNA repair and cell survival after ultraviolet but not oxidative stress [Internet]. Human Mutation. 2013 ; 34( 3): 481-489.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1002/humu.22259
  • Source: PLOS ONE. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      ORTOLAN, Tatiana Grohmann e MENCK, Carlos Frederico Martins. UVB-induced cell death signaling is associated with G1-S progression and transcription inhibition in primary human fibroblasts. PLOS ONE, v. 8, n. 10, p. 1-82013, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0076936. Acesso em: 04 jul. 2024.
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      Ortolan, T. G., & Menck, C. F. M. (2013). UVB-induced cell death signaling is associated with G1-S progression and transcription inhibition in primary human fibroblasts. PLOS ONE, 8( 10), 1-82013. doi:10.1371/journal.pone.0076936
    • NLM

      Ortolan TG, Menck CFM. UVB-induced cell death signaling is associated with G1-S progression and transcription inhibition in primary human fibroblasts [Internet]. PLOS ONE. 2013 ; 8( 10): 1-82013.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1371/journal.pone.0076936
    • Vancouver

      Ortolan TG, Menck CFM. UVB-induced cell death signaling is associated with G1-S progression and transcription inhibition in primary human fibroblasts [Internet]. PLOS ONE. 2013 ; 8( 10): 1-82013.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1371/journal.pone.0076936
  • Source: Mutation Research. Unidades: ICB, IB

    Subjects: MICROBIOLOGIA, REPARAÇÃO DE DNA, CÉLULAS-TRONCO, GENOMAS

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      ROCHA, Clarissa Ribeiro Reily et al. The role of DNA repair in the pluripotency and differentiation of human stem cells. Mutation Research, v. 752, n. 1, p. 25-35, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mrrev.2012.09.001. Acesso em: 04 jul. 2024.
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      Rocha, C. R. R., Lerner, L. K., Okamoto, O. K., Marchetto, M. C., & Menck, C. F. M. (2013). The role of DNA repair in the pluripotency and differentiation of human stem cells. Mutation Research, 752( 1), 25-35. doi:10.1016/j.mrrev.2012.09.001
    • NLM

      Rocha CRR, Lerner LK, Okamoto OK, Marchetto MC, Menck CFM. The role of DNA repair in the pluripotency and differentiation of human stem cells [Internet]. Mutation Research. 2013 ; 752( 1): 25-35.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.mrrev.2012.09.001
    • Vancouver

      Rocha CRR, Lerner LK, Okamoto OK, Marchetto MC, Menck CFM. The role of DNA repair in the pluripotency and differentiation of human stem cells [Internet]. Mutation Research. 2013 ; 752( 1): 25-35.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1016/j.mrrev.2012.09.001
  • Source: PLOS Genetics. Unidades: IQ, ICB

    Subjects: PROLIFERAÇÃO CELULAR, EXPRESSÃO GÊNICA

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      BECKEDORFF, Felipe Cesar Ferrarezi et al. The intronic long noncoding RNA ANRASSF1 recruits PRC2 to the RASSF1A promoter, reducing the expression of RASSF1A and increasing cell proliferation. PLOS Genetics, v. 9, n. 8, p. 1-15, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pgen.1003705. Acesso em: 04 jul. 2024.
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      Beckedorff, F. C. F., Ayupe, A. C., Crocci-Souza, R., Amaral, M. S., Nakaya, H. T. I., Soltys, D. T., et al. (2013). The intronic long noncoding RNA ANRASSF1 recruits PRC2 to the RASSF1A promoter, reducing the expression of RASSF1A and increasing cell proliferation. PLOS Genetics, 9( 8), 1-15. doi:10.1371/journal.pgen.1003705
    • NLM

      Beckedorff FCF, Ayupe AC, Crocci-Souza R, Amaral MS, Nakaya HTI, Soltys DT, Menck CFM, Reis EM, Verjovski-Almeida S. The intronic long noncoding RNA ANRASSF1 recruits PRC2 to the RASSF1A promoter, reducing the expression of RASSF1A and increasing cell proliferation [Internet]. PLOS Genetics. 2013 ; 9( 8): 1-15.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1371/journal.pgen.1003705
    • Vancouver

      Beckedorff FCF, Ayupe AC, Crocci-Souza R, Amaral MS, Nakaya HTI, Soltys DT, Menck CFM, Reis EM, Verjovski-Almeida S. The intronic long noncoding RNA ANRASSF1 recruits PRC2 to the RASSF1A promoter, reducing the expression of RASSF1A and increasing cell proliferation [Internet]. PLOS Genetics. 2013 ; 9( 8): 1-15.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1371/journal.pgen.1003705
  • Source: Photochemical Photobiological Sciences. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      SCHUCH, André Passaglia et al. DNA damage as a biological sensor for environmental sunlight. Photochemical Photobiological Sciences, v. 12, n. 8, p. 1259-1272, 2013Tradução . . Disponível em: https://doi.org/10.1039/c3pp00004d. Acesso em: 04 jul. 2024.
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      Schuch, A. P., Garcia, C. C. M., Makita, K., & Menck, C. F. M. (2013). DNA damage as a biological sensor for environmental sunlight. Photochemical Photobiological Sciences, 12( 8), 1259-1272. doi:10.1039/c3pp00004d
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      Schuch AP, Garcia CCM, Makita K, Menck CFM. DNA damage as a biological sensor for environmental sunlight [Internet]. Photochemical Photobiological Sciences. 2013 ; 12( 8): 1259-1272.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1039/c3pp00004d
    • Vancouver

      Schuch AP, Garcia CCM, Makita K, Menck CFM. DNA damage as a biological sensor for environmental sunlight [Internet]. Photochemical Photobiological Sciences. 2013 ; 12( 8): 1259-1272.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1039/c3pp00004d
  • Source: Photochemical & Photobiological Sciences. Unidade: ICB

    Assunto: MICROBIOLOGIA

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      CORTAT, Barbara et al. The relative roles of DNA damage induced by UVA irradiation in human cells. Photochemical & Photobiological Sciences, v. 12, n. 8, p. 1483-1495, 2013Tradução . . Disponível em: https://doi.org/10.1039/c3pp50023c. Acesso em: 04 jul. 2024.
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      Cortat, B., Garcia, C. C. M., Quinet, A., Schuch, A. P., Lima-Bessa, K. M. de, & Menck, C. F. M. (2013). The relative roles of DNA damage induced by UVA irradiation in human cells. Photochemical & Photobiological Sciences, 12( 8), 1483-1495. doi:10.1039/c3pp50023c
    • NLM

      Cortat B, Garcia CCM, Quinet A, Schuch AP, Lima-Bessa KM de, Menck CFM. The relative roles of DNA damage induced by UVA irradiation in human cells [Internet]. Photochemical & Photobiological Sciences. 2013 ; 12( 8): 1483-1495.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1039/c3pp50023c
    • Vancouver

      Cortat B, Garcia CCM, Quinet A, Schuch AP, Lima-Bessa KM de, Menck CFM. The relative roles of DNA damage induced by UVA irradiation in human cells [Internet]. Photochemical & Photobiological Sciences. 2013 ; 12( 8): 1483-1495.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1039/c3pp50023c
  • Source: PLOS ONE. Unidades: ICB, IB

    Assunto: MICROBIOLOGIA

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      KOBAYASHI, Gerson Shigeru et al. Susceptibility to DNA damage as a molecular mechanism for non-syndromic cleft lip and palate. PLOS ONE, v. 8, n. 6, p. 1-11, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0065677. Acesso em: 04 jul. 2024.
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      Kobayashi, G. S., Alvizi, L., Sunaga, D. Y., Francis-West, P., Kuta, A., Almada, B. V. P., et al. (2013). Susceptibility to DNA damage as a molecular mechanism for non-syndromic cleft lip and palate. PLOS ONE, 8( 6), 1-11. doi:10.1371/journal.pone.0065677
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      Kobayashi GS, Alvizi L, Sunaga DY, Francis-West P, Kuta A, Almada BVP, Ferreira SG, Andrade-Lima LC de, Bueno DF, Raposo-Amaral CE, Menck CFM, Passos-Bueno MR. Susceptibility to DNA damage as a molecular mechanism for non-syndromic cleft lip and palate [Internet]. PLOS ONE. 2013 ; 8( 6): 1-11.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1371/journal.pone.0065677
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      Kobayashi GS, Alvizi L, Sunaga DY, Francis-West P, Kuta A, Almada BVP, Ferreira SG, Andrade-Lima LC de, Bueno DF, Raposo-Amaral CE, Menck CFM, Passos-Bueno MR. Susceptibility to DNA damage as a molecular mechanism for non-syndromic cleft lip and palate [Internet]. PLOS ONE. 2013 ; 8( 6): 1-11.[citado 2024 jul. 04 ] Available from: https://doi.org/10.1371/journal.pone.0065677

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