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Trabalho de evento
Exploring identifiability in hybrid models of cell signaling pathways (2023)
Sousa, Ronaldo Nogueira de ; Campos, Cristiano G. S ; Wang, Willian ; Hashimoto, Ronaldo Fumio ; Armelin, Hugo Aguirre ; Reis, Marcelo S.
Nome do evento: Brazilian Symposium on Bioinformatics - BSB. Unidades: IME, IQ, BIOINFORMÁTICA
Assuntos: PROBLEMAS INVERSOS, REDES NEURAIS, APRENDIZADO COMPUTACIONAL
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ABNT
SOUSA, Ronaldo Nogueira de et al. Exploring identifiability in hybrid models of cell signaling pathways. 2023, Anais.. Cham: Springer, 2023. Disponível em: https://doi.org/10.1007/978-3-031-42715-2_14. Acesso em: 17 nov. 2024.
APA
Sousa, R. N. de, Campos, C. G. S., Wang, W., Hashimoto, R. F., Armelin, H. A., & Reis, M. S. (2023). Exploring identifiability in hybrid models of cell signaling pathways. In . Cham: Springer. doi:10.1007/978-3-031-42715-2_14
NLM
Sousa RN de, Campos CGS, Wang W, Hashimoto RF, Armelin HA, Reis MS. Exploring identifiability in hybrid models of cell signaling pathways [Internet]. 2023 ;[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/978-3-031-42715-2_14
Vancouver
Sousa RN de, Campos CGS, Wang W, Hashimoto RF, Armelin HA, Reis MS. Exploring identifiability in hybrid models of cell signaling pathways [Internet]. 2023 ;[citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/978-3-031-42715-2_14
Artigo de periodico
The antiproliferative effect of FGF2 in K-Ras-driven tumor cells involves modulation of rRNA and the nucleolus (2023)
Vitorino, Francisca Nathalia de Luna ; Levy, Michaella J; Wailemann, Rosangela A. Mansano; Lopes, Mariana; Silva, Mariana Loterio; Sardiu, Mihaela E; Garcia, Benjamin A; Motta, Maria Cristina Machado ; Oliveira, Carla Columbano de ; Armelin, Hugo Aguirre ; Florens, Laurence A; Washburn, Michael P; Cunha, Julia Pinheiro Chagas da
Fonte: Journal of Cell Science. Unidade: IQ
Assuntos: PROTEÍNAS, NUCLÉOLO
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ABNT
VITORINO, Francisca Nathalia de Luna et al. The antiproliferative effect of FGF2 in K-Ras-driven tumor cells involves modulation of rRNA and the nucleolus. Journal of Cell Science, v. 136, p. 1-13 art. 260989, 2023Tradução . . Disponível em: https://dx.doi.org/10.1242/jcs.260989. Acesso em: 17 nov. 2024.
APA
Vitorino, F. N. de L., Levy, M. J., Wailemann, R. A. M., Lopes, M., Silva, M. L., Sardiu, M. E., et al. (2023). The antiproliferative effect of FGF2 in K-Ras-driven tumor cells involves modulation of rRNA and the nucleolus. Journal of Cell Science, 136, 1-13 art. 260989. doi:10.1242/jcs.260989
NLM
Vitorino FN de L, Levy MJ, Wailemann RAM, Lopes M, Silva ML, Sardiu ME, Garcia BA, Motta MCM, Oliveira CC de, Armelin HA, Florens LA, Washburn MP, Cunha JPC da. The antiproliferative effect of FGF2 in K-Ras-driven tumor cells involves modulation of rRNA and the nucleolus [Internet]. Journal of Cell Science. 2023 ; 136 1-13 art. 260989.[citado 2024 nov. 17 ] Available from: https://dx.doi.org/10.1242/jcs.260989
Vancouver
Vitorino FN de L, Levy MJ, Wailemann RAM, Lopes M, Silva ML, Sardiu ME, Garcia BA, Motta MCM, Oliveira CC de, Armelin HA, Florens LA, Washburn MP, Cunha JPC da. The antiproliferative effect of FGF2 in K-Ras-driven tumor cells involves modulation of rRNA and the nucleolus [Internet]. Journal of Cell Science. 2023 ; 136 1-13 art. 260989.[citado 2024 nov. 17 ] Available from: https://dx.doi.org/10.1242/jcs.260989
Trabalho de evento-resumo
Anguix: cell signaling modeling improvement through Sabio-RK association to reactome (2022)
Montoni, Fabio ; Sousa, Ronaldo Nogueira de ; Lima Junior, Marcelo Batista de; Campos, Cristiano G. S; Wang, William; Constantino, Vivian Marinelli; Santos, Cássia S; Armelin, Hugo Aguirre ; Reis, Marcelo S
Fonte: Abstracts. Nome do evento: International Conference On e-Science (e-Science). Unidades: IQ, BIOINFORMÁTICA
Assuntos: CINÉTICA, REAÇÕES QUÍMICAS
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MONTONI, Fabio et al. Anguix: cell signaling modeling improvement through Sabio-RK association to reactome. 2022, Anais.. Salt Lake City: IEEE, 2022. Disponível em: https://ieeexplore.ieee.org/stamp/stamp.jsp?tp=&arnumber=9973605. Acesso em: 17 nov. 2024.
APA
Montoni, F., Sousa, R. N. de, Lima Junior, M. B. de, Campos, C. G. S., Wang, W., Constantino, V. M., et al. (2022). Anguix: cell signaling modeling improvement through Sabio-RK association to reactome. In Abstracts. Salt Lake City: IEEE. Recuperado de https://ieeexplore.ieee.org/stamp/stamp.jsp?tp=&arnumber=9973605
NLM
Montoni F, Sousa RN de, Lima Junior MB de, Campos CGS, Wang W, Constantino VM, Santos CS, Armelin HA, Reis MS. Anguix: cell signaling modeling improvement through Sabio-RK association to reactome [Internet]. Abstracts. 2022 ;[citado 2024 nov. 17 ] Available from: https://ieeexplore.ieee.org/stamp/stamp.jsp?tp=&arnumber=9973605
Vancouver
Montoni F, Sousa RN de, Lima Junior MB de, Campos CGS, Wang W, Constantino VM, Santos CS, Armelin HA, Reis MS. Anguix: cell signaling modeling improvement through Sabio-RK association to reactome [Internet]. Abstracts. 2022 ;[citado 2024 nov. 17 ] Available from: https://ieeexplore.ieee.org/stamp/stamp.jsp?tp=&arnumber=9973605
Artigo de periodico
Autophagy buffers Ras-induced genotoxic stress enabling malignant transformation in keratinocytes primed by human papillomavirus (2021)
Lopes, Eduardo Cararo ; Dias, Matheus H. ; Silva, Marcelo S. da ; Zeidler, Julianna Dias ; Vessoni, Alexandre T. ; Reis, Marcelo S. ; Boccardo, Enrique ; Armelin, Hugo Aguirre
Fonte: Cell Death & Disease. Unidades: ICB, IQ
Assuntos: MICROBIOLOGIA, PAPILLOMAVIRUS, INFECÇÕES POR PAPILLOMAVIRUS, REGENERAÇÃO (FENÔMENOS BIOLÓGICOS), ESTRESSE OXIDATIVO, CICLO CELULAR, ONCOPROTEÍNAS
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ABNT
LOPES, Eduardo Cararo et al. Autophagy buffers Ras-induced genotoxic stress enabling malignant transformation in keratinocytes primed by human papillomavirus. Cell Death & Disease, v. 12, n. 2, p. 16 , 2021Tradução . . Disponível em: https://doi.org/10.1038/s41419-021-03476-3. Acesso em: 17 nov. 2024.
APA
Lopes, E. C., Dias, M. H., Silva, M. S. da, Zeidler, J. D., Vessoni, A. T., Reis, M. S., et al. (2021). Autophagy buffers Ras-induced genotoxic stress enabling malignant transformation in keratinocytes primed by human papillomavirus. Cell Death & Disease, 12( 2), 16 . doi:10.1038/s41419-021-03476-3
NLM
Lopes EC, Dias MH, Silva MS da, Zeidler JD, Vessoni AT, Reis MS, Boccardo E, Armelin HA. Autophagy buffers Ras-induced genotoxic stress enabling malignant transformation in keratinocytes primed by human papillomavirus [Internet]. Cell Death & Disease. 2021 ; 12( 2): 16 .[citado 2024 nov. 17 ] Available from: https://doi.org/10.1038/s41419-021-03476-3
Vancouver
Lopes EC, Dias MH, Silva MS da, Zeidler JD, Vessoni AT, Reis MS, Boccardo E, Armelin HA. Autophagy buffers Ras-induced genotoxic stress enabling malignant transformation in keratinocytes primed by human papillomavirus [Internet]. Cell Death & Disease. 2021 ; 12( 2): 16 .[citado 2024 nov. 17 ] Available from: https://doi.org/10.1038/s41419-021-03476-3
Trabalho de evento-resumo
Mitogenic overstimulation causes cytotoxicity in RAS-driven cancer cells by overloading stress response pathways (2019)
Santos, Edmilson Ozorio dos; Silva, Juliana Galvão da; Dias, Matheus Henrique dos Santos; Armelin, Hugo Aguirre
Fonte: Abstracts. Nome do evento: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology/SBBq. Unidade: IQ
Assuntos: CÉLULAS CULTIVADAS DE TUMOR, ONCOGENES
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ABNT
SANTOS, Edmilson Ozorio dos et al. Mitogenic overstimulation causes cytotoxicity in RAS-driven cancer cells by overloading stress response pathways. 2019, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq, 2019. Disponível em: http://www.sbbq.org.br/reuniao/2019/images/Livro_Resumos_2019.pdf. Acesso em: 17 nov. 2024.
APA
Santos, E. O. dos, Silva, J. G. da, Dias, M. H. dos S., & Armelin, H. A. (2019). Mitogenic overstimulation causes cytotoxicity in RAS-driven cancer cells by overloading stress response pathways. In Abstracts. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular/SBBq. Recuperado de http://www.sbbq.org.br/reuniao/2019/images/Livro_Resumos_2019.pdf
NLM
Santos EO dos, Silva JG da, Dias MH dos S, Armelin HA. Mitogenic overstimulation causes cytotoxicity in RAS-driven cancer cells by overloading stress response pathways [Internet]. Abstracts. 2019 ;[citado 2024 nov. 17 ] Available from: http://www.sbbq.org.br/reuniao/2019/images/Livro_Resumos_2019.pdf
Vancouver
Santos EO dos, Silva JG da, Dias MH dos S, Armelin HA. Mitogenic overstimulation causes cytotoxicity in RAS-driven cancer cells by overloading stress response pathways [Internet]. Abstracts. 2019 ;[citado 2024 nov. 17 ] Available from: http://www.sbbq.org.br/reuniao/2019/images/Livro_Resumos_2019.pdf
Artigo de periodico
Where do we aspire to publish? A position paper on scientific communication in biochemistry and molecular biology (2019)
Baptista, Mauricio da Silva ; Alves, Maria Júlia Manso ; Arantes, Guilherme Menegon ; Armelin, Hugo Aguirre ; Augusto, Ohara ; Baldini, Regina Lúcia ; Bassères, Daniela Sanchez ; Bechara, Etelvino José Henriques ; Bruni-Cardoso, Alexandre ; Chaimovich Guralnik, Hernan ; Colepicolo, Pio ; Colli, Walter ; Cuccovia, Iolanda Midea ; Da Silva, Aline Maria ; Di Mascio, Paolo ; Farah, Chuck Shaker ; Ferreira, Clélia ; Forti, Fábio Luís ; Giordano, Ricardo José ; Gomes, Suely Lopes ; Gueiros Filho, Frederico José ; Hoch, Nicolas Carlos ; Hotta, Carlos Takeshi ; Labriola, Leticia ; Lameu, Claudiana ; Machini, Maria Teresa ; Malnic, Bettina ; Marana, Sandro Roberto ; Medeiros, Marisa Helena Gennari de ; Meotti, Flávia Carla ; Miyamoto, Sayuri ; Oliveira, Carla Columbano de ; Souza-Pinto, Nadja Cristhina de ; Reis, Eduardo Moraes ; Ronsein, Graziella Eliza ; Salinas, Roberto Kopke ; Schechtman, Deborah ; Schreier, Shirley ; Setubal, João Carlos ; Sogayar, Mari Cleide ; Souza, Gláucia Mendes ; Terra, Walter Ribeiro ; Truzzi, Daniela Ramos ; Ulrich, Henning ; Verjovski-Almeida, Sergio ; Winck, Flavia Vischi ; Zingales, Bianca ; Kowaltowski, Alicia Juliana
Fonte: Brazilian Journal of Medical and Biological Research. Unidade: IQ
Assuntos: PERIÓDICOS CIENTÍFICOS, PRÉ-IMPRESSOS
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ABNT
BAPTISTA, Mauricio da Silva et al. Where do we aspire to publish? A position paper on scientific communication in biochemistry and molecular biology. Brazilian Journal of Medical and Biological Research, v. 52, n. 9, p. 1-5 art. e8935, 2019Tradução . . Disponível em: https://doi.org/10.1590/1414-431x20198935. Acesso em: 17 nov. 2024.
APA
Baptista, M. da S., Alves, M. J. M., Arantes, G. M., Armelin, H. A., Augusto, O., Baldini, R. L., et al. (2019). Where do we aspire to publish? A position paper on scientific communication in biochemistry and molecular biology. Brazilian Journal of Medical and Biological Research, 52( 9), 1-5 art. e8935. doi:10.1590/1414-431x20198935
NLM
Baptista M da S, Alves MJM, Arantes GM, Armelin HA, Augusto O, Baldini RL, Bassères DS, Bechara EJH, Bruni-Cardoso A, Chaimovich Guralnik H, Colepicolo P, Colli W, Cuccovia IM, Da Silva AM, Di Mascio P, Farah CS, Ferreira C, Forti FL, Giordano RJ, Gomes SL, Gueiros Filho FJ, Hoch NC, Hotta CT, Labriola L, Lameu C, Machini MT, Malnic B, Marana SR, Medeiros MHG de, Meotti FC, Miyamoto S, Oliveira CC de, Souza-Pinto NC de, Reis EM, Ronsein GE, Salinas RK, Schechtman D, Schreier S, Setubal JC, Sogayar MC, Souza GM, Terra WR, Truzzi DR, Ulrich H, Verjovski-Almeida S, Winck FV, Zingales B, Kowaltowski AJ. Where do we aspire to publish? A position paper on scientific communication in biochemistry and molecular biology [Internet]. Brazilian Journal of Medical and Biological Research. 2019 ; 52( 9): 1-5 art. e8935.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1590/1414-431x20198935
Vancouver
Baptista M da S, Alves MJM, Arantes GM, Armelin HA, Augusto O, Baldini RL, Bassères DS, Bechara EJH, Bruni-Cardoso A, Chaimovich Guralnik H, Colepicolo P, Colli W, Cuccovia IM, Da Silva AM, Di Mascio P, Farah CS, Ferreira C, Forti FL, Giordano RJ, Gomes SL, Gueiros Filho FJ, Hoch NC, Hotta CT, Labriola L, Lameu C, Machini MT, Malnic B, Marana SR, Medeiros MHG de, Meotti FC, Miyamoto S, Oliveira CC de, Souza-Pinto NC de, Reis EM, Ronsein GE, Salinas RK, Schechtman D, Schreier S, Setubal JC, Sogayar MC, Souza GM, Terra WR, Truzzi DR, Ulrich H, Verjovski-Almeida S, Winck FV, Zingales B, Kowaltowski AJ. Where do we aspire to publish? A position paper on scientific communication in biochemistry and molecular biology [Internet]. Brazilian Journal of Medical and Biological Research. 2019 ; 52( 9): 1-5 art. e8935.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1590/1414-431x20198935
Parte de monografia/livro
An interdisciplinary approach for designing kinetic models of the Ras/MAPK signaling pathway (2017)
Reis, Marcelo da Silva ; Noël, Vincent; Dias, Matheus H; Albuquerque, Layra Lucy; Guimarães, Amanda Sayuri; Wu, Lulu; Barrera, Junior; Armelin, Hugo Aguirre
Fonte: Kinase signaling networks. Unidades: IME, IQ
Assuntos: EQUAÇÕES DIFERENCIAIS ORDINÁRIAS, EQUAÇÕES ALGÉBRICAS DIFERENCIAIS, OTIMIZAÇÃO NÃO LINEAR
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ABNT
REIS, Marcelo da Silva et al. An interdisciplinary approach for designing kinetic models of the Ras/MAPK signaling pathway. Kinase signaling networks. Tradução . New York: Humana Press, 2017. . Disponível em: https://doi.org/10.1007/978-1-4939-7154-1_28. Acesso em: 17 nov. 2024.
APA
Reis, M. da S., Noël, V., Dias, M. H., Albuquerque, L. L., Guimarães, A. S., Wu, L., et al. (2017). An interdisciplinary approach for designing kinetic models of the Ras/MAPK signaling pathway. In Kinase signaling networks. New York: Humana Press. doi:10.1007/978-1-4939-7154-1_28
NLM
Reis M da S, Noël V, Dias MH, Albuquerque LL, Guimarães AS, Wu L, Barrera J, Armelin HA. An interdisciplinary approach for designing kinetic models of the Ras/MAPK signaling pathway [Internet]. In: Kinase signaling networks. New York: Humana Press; 2017. [citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/978-1-4939-7154-1_28
Vancouver
Reis M da S, Noël V, Dias MH, Albuquerque LL, Guimarães AS, Wu L, Barrera J, Armelin HA. An interdisciplinary approach for designing kinetic models of the Ras/MAPK signaling pathway [Internet]. In: Kinase signaling networks. New York: Humana Press; 2017. [citado 2024 nov. 17 ] Available from: https://doi.org/10.1007/978-1-4939-7154-1_28
Artigo de periodico
Protein disulfide isomerase externalization in endothelial cells follows classical and unconventional routes (2017)
Araujo, Thaís L. S; Zeidler, Julianna Dias; Oliveira, Percíllia V. S; Dias, Matheus H; Armelin, Hugo Aguirre ; Laurindo, Francisco R. M
Fonte: Free Radical Biology and Medicine. Unidade: IQ
Assuntos: CÉLULAS ENDOTELIAIS, PROTEÍNAS
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ABNT
ARAUJO, Thaís L. S et al. Protein disulfide isomerase externalization in endothelial cells follows classical and unconventional routes. Free Radical Biology and Medicine, v. 103, p. 199-208, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2016.12.021. Acesso em: 17 nov. 2024.
APA
Araujo, T. L. S., Zeidler, J. D., Oliveira, P. V. S., Dias, M. H., Armelin, H. A., & Laurindo, F. R. M. (2017). Protein disulfide isomerase externalization in endothelial cells follows classical and unconventional routes. Free Radical Biology and Medicine, 103, 199-208. doi:10.1016/j.freeradbiomed.2016.12.021
NLM
Araujo TLS, Zeidler JD, Oliveira PVS, Dias MH, Armelin HA, Laurindo FRM. Protein disulfide isomerase externalization in endothelial cells follows classical and unconventional routes [Internet]. Free Radical Biology and Medicine. 2017 ; 103 199-208.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2016.12.021
Vancouver
Araujo TLS, Zeidler JD, Oliveira PVS, Dias MH, Armelin HA, Laurindo FRM. Protein disulfide isomerase externalization in endothelial cells follows classical and unconventional routes [Internet]. Free Radical Biology and Medicine. 2017 ; 103 199-208.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2016.12.021
Trabalho de evento-resumo
SigNetSim: a web-based framework for designing kinetic models of molecular signaling networks (2016)
Noel, Vincent; Reis, Marcelo da Silva ; Dias, Matheus Henrique dos Santos; Wu, Lulu; Guimarães, Amanda S; Reverbel, Daniel F; Barrera, Júnior; Armelin, Hugo Aguirre
Fonte: Abstracts. Nome do evento: Computational Modeling in BIology NEtwork - COMBINE. Unidades: IME, IQ
Assuntos: COMPUTAÇÃO APLICADA, BIOINFORMÁTICA
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ABNT
NOEL, Vincent et al. SigNetSim: a web-based framework for designing kinetic models of molecular signaling networks. 2016, Anais.. [S.l.]: COMBINE, 2016. Disponível em: http://co.mbine.org/events/COMBINE_2016/abstracts. Acesso em: 17 nov. 2024.
APA
Noel, V., Reis, M. da S., Dias, M. H. dos S., Wu, L., Guimarães, A. S., Reverbel, D. F., et al. (2016). SigNetSim: a web-based framework for designing kinetic models of molecular signaling networks. In Abstracts. [S.l.]: COMBINE. Recuperado de http://co.mbine.org/events/COMBINE_2016/abstracts
NLM
Noel V, Reis M da S, Dias MH dos S, Wu L, Guimarães AS, Reverbel DF, Barrera J, Armelin HA. SigNetSim: a web-based framework for designing kinetic models of molecular signaling networks [Internet]. Abstracts. 2016 ;[citado 2024 nov. 17 ] Available from: http://co.mbine.org/events/COMBINE_2016/abstracts
Vancouver
Noel V, Reis M da S, Dias MH dos S, Wu L, Guimarães AS, Reverbel DF, Barrera J, Armelin HA. SigNetSim: a web-based framework for designing kinetic models of molecular signaling networks [Internet]. Abstracts. 2016 ;[citado 2024 nov. 17 ] Available from: http://co.mbine.org/events/COMBINE_2016/abstracts
Trabalho de evento-resumo
In E6E7-immortalized human keratinocytes, [HRASG12V-GTP] high levels block autophagic flux leading to cell death (2016)
Silva, Eduardo Lopes da; Dias, Matheus Santos; Silva, Marcelo Santos da ; Boccardo, Enrique ; Armelin, Hugo Aguirre
Fonte: Abstracts. Nome do evento: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology (SBBq). Unidade: IQ
Assuntos: ONCOGENES, CÉLULAS EPITELIAIS
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ABNT
SILVA, Eduardo Lopes da et al. In E6E7-immortalized human keratinocytes, [HRASG12V-GTP] high levels block autophagic flux leading to cell death. 2016, Anais.. Natal: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2016. . Acesso em: 17 nov. 2024.
APA
Silva, E. L. da, Dias, M. S., Silva, M. S. da, Boccardo, E., & Armelin, H. A. (2016). In E6E7-immortalized human keratinocytes, [HRASG12V-GTP] high levels block autophagic flux leading to cell death. In Abstracts. Natal: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq).
NLM
Silva EL da, Dias MS, Silva MS da, Boccardo E, Armelin HA. In E6E7-immortalized human keratinocytes, [HRASG12V-GTP] high levels block autophagic flux leading to cell death. Abstracts. 2016 ;[citado 2024 nov. 17 ]
Vancouver
Silva EL da, Dias MS, Silva MS da, Boccardo E, Armelin HA. In E6E7-immortalized human keratinocytes, [HRASG12V-GTP] high levels block autophagic flux leading to cell death. Abstracts. 2016 ;[citado 2024 nov. 17 ]
Trabalho de evento-resumo
FGF2 enhances replicative stress and leads to permanent DDR and G2 cell cycle block selectively in K-Ras transformed cells (2016)
Fonseca, Cecilia S; Dias, Matheus Henrique dos Santos; Armelin, Hugo Aguirre
Fonte: Abstracts. Nome do evento: International Conference of Biochemistry and Molecular Biology. Unidade: IQ
Assuntos: CICLO CELULAR, BIOQUÍMICA
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ABNT
FONSECA, Cecilia S e DIAS, Matheus Henrique dos Santos e ARMELIN, Hugo Aguirre. FGF2 enhances replicative stress and leads to permanent DDR and G2 cell cycle block selectively in K-Ras transformed cells. 2016, Anais.. Vancouver: International Union on Biochemistry and Molecular Biology (IUBMB), 2016. . Acesso em: 17 nov. 2024.
APA
Fonseca, C. S., Dias, M. H. dos S., & Armelin, H. A. (2016). FGF2 enhances replicative stress and leads to permanent DDR and G2 cell cycle block selectively in K-Ras transformed cells. In Abstracts. Vancouver: International Union on Biochemistry and Molecular Biology (IUBMB).
NLM
Fonseca CS, Dias MH dos S, Armelin HA. FGF2 enhances replicative stress and leads to permanent DDR and G2 cell cycle block selectively in K-Ras transformed cells. Abstracts. 2016 ;[citado 2024 nov. 17 ]
Vancouver
Fonseca CS, Dias MH dos S, Armelin HA. FGF2 enhances replicative stress and leads to permanent DDR and G2 cell cycle block selectively in K-Ras transformed cells. Abstracts. 2016 ;[citado 2024 nov. 17 ]
Artigo de periodico
Intratumoral heterogeneity of ADAM23 promotes tumor growth and metastasis through LGI4 and nitric oxide signals (2015)
Costa, E. T; Barnabé, G. F; Li, M; Dias, A. A. M; Machado, T. R; Asprino, P. F; Cavalher, F. P; Ferreira, E. N; Inda, M. del Mar; Nagai, M. A; Malnic, Bettina ; Duarte, M L; Leite, K R M; Barros, A C S D; Carraro, D M; Chammas, Roger; Armelin, Hugo Aguirre ; Cavenee, W; Furnari, F; Camargo, A A
Fonte: Oncogene. Unidades: IQ, FM
Assuntos: METÁSTASE NEOPLÁSICA, ÓXIDO NÍTRICO
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ABNT
COSTA, E. T et al. Intratumoral heterogeneity of ADAM23 promotes tumor growth and metastasis through LGI4 and nitric oxide signals. Oncogene, v. 34, n. 10, p. 1270-1279, 2015Tradução . . Disponível em: https://doi.org/10.1038/onc.2014.70. Acesso em: 17 nov. 2024.
APA
Costa, E. T., Barnabé, G. F., Li, M., Dias, A. A. M., Machado, T. R., Asprino, P. F., et al. (2015). Intratumoral heterogeneity of ADAM23 promotes tumor growth and metastasis through LGI4 and nitric oxide signals. Oncogene, 34( 10), 1270-1279. doi:10.1038/onc.2014.70
NLM
Costa ET, Barnabé GF, Li M, Dias AAM, Machado TR, Asprino PF, Cavalher FP, Ferreira EN, Inda M del M, Nagai MA, Malnic B, Duarte ML, Leite KRM, Barros ACSD, Carraro DM, Chammas R, Armelin HA, Cavenee W, Furnari F, Camargo AA. Intratumoral heterogeneity of ADAM23 promotes tumor growth and metastasis through LGI4 and nitric oxide signals [Internet]. Oncogene. 2015 ; 34( 10): 1270-1279.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1038/onc.2014.70
Vancouver
Costa ET, Barnabé GF, Li M, Dias AAM, Machado TR, Asprino PF, Cavalher FP, Ferreira EN, Inda M del M, Nagai MA, Malnic B, Duarte ML, Leite KRM, Barros ACSD, Carraro DM, Chammas R, Armelin HA, Cavenee W, Furnari F, Camargo AA. Intratumoral heterogeneity of ADAM23 promotes tumor growth and metastasis through LGI4 and nitric oxide signals [Internet]. Oncogene. 2015 ; 34( 10): 1270-1279.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1038/onc.2014.70
Artigo de periodico
Glyceraldehyde 3-Phosphate Dehydrogenase-Telomere association correlates with redox status in Trypanosoma cruzi (2015)
Pariona-Llanos, Ricardo; Pavani, Raphael Souza; Reis, Marcelo da Silva ; Noël, Vincent; Silber, Ariel Mariano ; Armelin, Hugo Aguirre ; Cano, Maria Isabel Nogueira; Elias, Maria Carolina
Fonte: PLOS ONE. Unidades: ICB, IQ
Assuntos: PARASITOLOGIA, ENZIMAS (METABOLISMO), CATALISE, CATALISADORES, TRYPANOSOMA CRUZI, PROTEINAS RECOMBINANTES
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ABNT
PARIONA-LLANOS, Ricardo et al. Glyceraldehyde 3-Phosphate Dehydrogenase-Telomere association correlates with redox status in Trypanosoma cruzi. PLOS ONE, v. 10, n. 3, p. 1-17, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0120896. Acesso em: 17 nov. 2024.
APA
Pariona-Llanos, R., Pavani, R. S., Reis, M. da S., Noël, V., Silber, A. M., Armelin, H. A., et al. (2015). Glyceraldehyde 3-Phosphate Dehydrogenase-Telomere association correlates with redox status in Trypanosoma cruzi. PLOS ONE, 10( 3), 1-17. doi:10.1371/journal.pone.0120896
NLM
Pariona-Llanos R, Pavani RS, Reis M da S, Noël V, Silber AM, Armelin HA, Cano MIN, Elias MC. Glyceraldehyde 3-Phosphate Dehydrogenase-Telomere association correlates with redox status in Trypanosoma cruzi [Internet]. PLOS ONE. 2015 ; 10( 3): 1-17.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.pone.0120896
Vancouver
Pariona-Llanos R, Pavani RS, Reis M da S, Noël V, Silber AM, Armelin HA, Cano MIN, Elias MC. Glyceraldehyde 3-Phosphate Dehydrogenase-Telomere association correlates with redox status in Trypanosoma cruzi [Internet]. PLOS ONE. 2015 ; 10( 3): 1-17.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1371/journal.pone.0120896
Trabalho de evento-resumo
Design and analysis of dynamical models of biochemical reaction networks (2015)
Noel, Vincent; Reis, Marcelo da Silva ; Dias, Matheus Henrique dos Santos; Albuquerque, Layra Lucy; Pariona-Llanos, Ricardo; Pavani, Raphael Souza; Silber, Ariel Mariano ; Cano, Maria Isabel Nogueira; Elias, Maria Carolina; Nakano, Fabio; Barrera, Júnior; Armelin, Hugo Aguirre
Fonte: Abstracts Book. Nome do evento: Congress of the International Union for Biochemistry and Molecular Biology - IUBMB. Unidades: ICB, IME, IQ
Assuntos: BIOINFORMÁTICA, MODELOS MATEMÁTICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NOEL, Vincent et al. Design and analysis of dynamical models of biochemical reaction networks. 2015, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2015. Disponível em: http://www.sbbq.org.br/iubmb2015/cdrom/resumos/R08662-1.pdf. Acesso em: 17 nov. 2024.
APA
Noel, V., Reis, M. da S., Dias, M. H. dos S., Albuquerque, L. L., Pariona-Llanos, R., Pavani, R. S., et al. (2015). Design and analysis of dynamical models of biochemical reaction networks. In Abstracts Book. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq). Recuperado de http://www.sbbq.org.br/iubmb2015/cdrom/resumos/R08662-1.pdf
NLM
Noel V, Reis M da S, Dias MH dos S, Albuquerque LL, Pariona-Llanos R, Pavani RS, Silber AM, Cano MIN, Elias MC, Nakano F, Barrera J, Armelin HA. Design and analysis of dynamical models of biochemical reaction networks [Internet]. Abstracts Book. 2015 ;[citado 2024 nov. 17 ] Available from: http://www.sbbq.org.br/iubmb2015/cdrom/resumos/R08662-1.pdf
Vancouver
Noel V, Reis M da S, Dias MH dos S, Albuquerque LL, Pariona-Llanos R, Pavani RS, Silber AM, Cano MIN, Elias MC, Nakano F, Barrera J, Armelin HA. Design and analysis of dynamical models of biochemical reaction networks [Internet]. Abstracts Book. 2015 ;[citado 2024 nov. 17 ] Available from: http://www.sbbq.org.br/iubmb2015/cdrom/resumos/R08662-1.pdf
Trabalho de evento-resumo
CETICSDB: a platform for integration and analysis of high-throughput-omics data and mathematical modeling of biochemical reactions (2015)
Nishiyama Junior, Milton Yutaka; Reis, M S; Silva, D. F; Junqueira-de-Azevedo, Inácio de Loiola Meirelles; Cunha, J. P. C. da; Barrera, Júnior; Iwai, L. K; Serrano, S. M. T; Armelin, Hugo Aguirre
Fonte: Abstracts Book. Nome do evento: Congress of the International Union for Biochemistry and Molecular Biology - IUBMB. Unidades: IME, IQ
Assuntos: BIOQUÍMICA, BIOINFORMÁTICA, MODELOS MATEMÁTICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NISHIYAMA JUNIOR, Milton Yutaka et al. CETICSDB: a platform for integration and analysis of high-throughput-omics data and mathematical modeling of biochemical reactions. 2015, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2015. Disponível em: http://www.sbbq.org.br/iubmb2015/cdrom/resumos/R09084-1.pdf. Acesso em: 17 nov. 2024.
APA
Nishiyama Junior, M. Y., Reis, M. S., Silva, D. F., Junqueira-de-Azevedo, I. de L. M., Cunha, J. P. C. da, Barrera, J., et al. (2015). CETICSDB: a platform for integration and analysis of high-throughput-omics data and mathematical modeling of biochemical reactions. In Abstracts Book. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq). Recuperado de http://www.sbbq.org.br/iubmb2015/cdrom/resumos/R09084-1.pdf
NLM
Nishiyama Junior MY, Reis MS, Silva DF, Junqueira-de-Azevedo I de LM, Cunha JPC da, Barrera J, Iwai LK, Serrano SMT, Armelin HA. CETICSDB: a platform for integration and analysis of high-throughput-omics data and mathematical modeling of biochemical reactions [Internet]. Abstracts Book. 2015 ;[citado 2024 nov. 17 ] Available from: http://www.sbbq.org.br/iubmb2015/cdrom/resumos/R09084-1.pdf
Vancouver
Nishiyama Junior MY, Reis MS, Silva DF, Junqueira-de-Azevedo I de LM, Cunha JPC da, Barrera J, Iwai LK, Serrano SMT, Armelin HA. CETICSDB: a platform for integration and analysis of high-throughput-omics data and mathematical modeling of biochemical reactions [Internet]. Abstracts Book. 2015 ;[citado 2024 nov. 17 ] Available from: http://www.sbbq.org.br/iubmb2015/cdrom/resumos/R09084-1.pdf
Trabalho de evento-resumo
CeTICSdb: a platform for interdisciplinary research that allows quantitative and qualitative -omics analyses and mathematical modeling of signaling networks (2015)
Reis, Marcelo da Silva ; Nishiyama Junior, Milton Yutaka; Silva, Daniel F; Junqueira-de-Azevedo, Inácio de Loiola Meirelles; Cunha, Julia Pinheiro Chagas da; Barrera, Júnior; Iwai, Leo Kei; Serrano, Solange Maria de Toledo; Armelin, Hugo Aguirre
Fonte: Abstracts. Nome do evento: Annual International Conference on Research in Computational Molecular Biology - RECOMB. Unidades: IME, IQ
Assuntos: COMPUTAÇÃO APLICADA, BIOINFORMÁTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
REIS, Marcelo da Silva et al. CeTICSdb: a platform for interdisciplinary research that allows quantitative and qualitative -omics analyses and mathematical modeling of signaling networks. 2015, Anais.. München: Max-Planck-Gesellschaft, 2015. Disponível em: https://f1000research.com/posters/1095384. Acesso em: 17 nov. 2024.
APA
Reis, M. da S., Nishiyama Junior, M. Y., Silva, D. F., Junqueira-de-Azevedo, I. de L. M., Cunha, J. P. C. da, Barrera, J., et al. (2015). CeTICSdb: a platform for interdisciplinary research that allows quantitative and qualitative -omics analyses and mathematical modeling of signaling networks. In Abstracts. München: Max-Planck-Gesellschaft. Recuperado de https://f1000research.com/posters/1095384
NLM
Reis M da S, Nishiyama Junior MY, Silva DF, Junqueira-de-Azevedo I de LM, Cunha JPC da, Barrera J, Iwai LK, Serrano SM de T, Armelin HA. CeTICSdb: a platform for interdisciplinary research that allows quantitative and qualitative -omics analyses and mathematical modeling of signaling networks [Internet]. Abstracts. 2015 ;[citado 2024 nov. 17 ] Available from: https://f1000research.com/posters/1095384
Vancouver
Reis M da S, Nishiyama Junior MY, Silva DF, Junqueira-de-Azevedo I de LM, Cunha JPC da, Barrera J, Iwai LK, Serrano SM de T, Armelin HA. CeTICSdb: a platform for interdisciplinary research that allows quantitative and qualitative -omics analyses and mathematical modeling of signaling networks [Internet]. Abstracts. 2015 ;[citado 2024 nov. 17 ] Available from: https://f1000research.com/posters/1095384
Trabalho de evento-resumo
Acute exposition and high levels of [HRasG12V-GTP] in E6E7-immortalized keratinocytes do not promote transformation (2015)
Lopes, E; Boccardo, Enrique ; Armelin, Hugo Aguirre
Fonte: Abstracts Book. Nome do evento: Congress of the International Union for Biochemistry and Molecular Biology - IUBMB. Unidade: IQ
Assuntos: NEOPLASIAS DO COLO UTERINO, BIOQUÍMICA
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ABNT
LOPES, E e BOCCARDO, Enrique e ARMELIN, Hugo Aguirre. Acute exposition and high levels of [HRasG12V-GTP] in E6E7-immortalized keratinocytes do not promote transformation. 2015, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2015. . Acesso em: 17 nov. 2024.
APA
Lopes, E., Boccardo, E., & Armelin, H. A. (2015). Acute exposition and high levels of [HRasG12V-GTP] in E6E7-immortalized keratinocytes do not promote transformation. In Abstracts Book. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq).
NLM
Lopes E, Boccardo E, Armelin HA. Acute exposition and high levels of [HRasG12V-GTP] in E6E7-immortalized keratinocytes do not promote transformation. Abstracts Book. 2015 ;[citado 2024 nov. 17 ]
Vancouver
Lopes E, Boccardo E, Armelin HA. Acute exposition and high levels of [HRasG12V-GTP] in E6E7-immortalized keratinocytes do not promote transformation. Abstracts Book. 2015 ;[citado 2024 nov. 17 ]
Trabalho de evento-resumo
FGF2-citotoxic mechanisms block mitotic entry in a Ras-dependent cell line by activation of G2-M checkpoint and inhibition of CDK1 activity (2015)
Fonseca, C. S; Dias, M. H. S; Armelin, Hugo Aguirre
Fonte: Abstracts Book. Nome do evento: Congress of the International Union for Biochemistry and Molecular Biology - IUBMB. Unidade: IQ
Assuntos: CICLO CELULAR, CÉLULAS CULTIVADAS DE TUMOR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FONSECA, C. S e DIAS, M. H. S e ARMELIN, Hugo Aguirre. FGF2-citotoxic mechanisms block mitotic entry in a Ras-dependent cell line by activation of G2-M checkpoint and inhibition of CDK1 activity. 2015, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2015. . Acesso em: 17 nov. 2024.
APA
Fonseca, C. S., Dias, M. H. S., & Armelin, H. A. (2015). FGF2-citotoxic mechanisms block mitotic entry in a Ras-dependent cell line by activation of G2-M checkpoint and inhibition of CDK1 activity. In Abstracts Book. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq).
NLM
Fonseca CS, Dias MHS, Armelin HA. FGF2-citotoxic mechanisms block mitotic entry in a Ras-dependent cell line by activation of G2-M checkpoint and inhibition of CDK1 activity. Abstracts Book. 2015 ;[citado 2024 nov. 17 ]
Vancouver
Fonseca CS, Dias MHS, Armelin HA. FGF2-citotoxic mechanisms block mitotic entry in a Ras-dependent cell line by activation of G2-M checkpoint and inhibition of CDK1 activity. Abstracts Book. 2015 ;[citado 2024 nov. 17 ]
Trabalho de evento-resumo
Mathematical modeling of RAs/MAPK signalling network kinetics provides insights into mechanisms of FGF2-induced cell cycle arrest in the K-K-ras-driven mouse Y1 adrenocortical tumor cells (2014)
Reis, Marcelo da Silva ; Dias, Matheus Henrique dos Santos; Nakano, Fábio ; Barrera, Júnior; Armelin, Hugo Aguirre
Fonte: Abstracts. Nome do evento: Annual International Conference on Research in Computational Molecular Biology - RECOMB. Unidades: EACH, IME, IQ
Assunto: COMPUTAÇÃO APLICADA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
REIS, Marcelo da Silva et al. Mathematical modeling of RAs/MAPK signalling network kinetics provides insights into mechanisms of FGF2-induced cell cycle arrest in the K-K-ras-driven mouse Y1 adrenocortical tumor cells. 2014, Anais.. München: Max-Planck-Gesellschaft, 2014. Disponível em: https://f1000research.com/posters/1095383. Acesso em: 17 nov. 2024.
APA
Reis, M. da S., Dias, M. H. dos S., Nakano, F., Barrera, J., & Armelin, H. A. (2014). Mathematical modeling of RAs/MAPK signalling network kinetics provides insights into mechanisms of FGF2-induced cell cycle arrest in the K-K-ras-driven mouse Y1 adrenocortical tumor cells. In Abstracts. München: Max-Planck-Gesellschaft. Recuperado de https://f1000research.com/posters/1095383
NLM
Reis M da S, Dias MH dos S, Nakano F, Barrera J, Armelin HA. Mathematical modeling of RAs/MAPK signalling network kinetics provides insights into mechanisms of FGF2-induced cell cycle arrest in the K-K-ras-driven mouse Y1 adrenocortical tumor cells [Internet]. Abstracts. 2014 ;[citado 2024 nov. 17 ] Available from: https://f1000research.com/posters/1095383
Vancouver
Reis M da S, Dias MH dos S, Nakano F, Barrera J, Armelin HA. Mathematical modeling of RAs/MAPK signalling network kinetics provides insights into mechanisms of FGF2-induced cell cycle arrest in the K-K-ras-driven mouse Y1 adrenocortical tumor cells [Internet]. Abstracts. 2014 ;[citado 2024 nov. 17 ] Available from: https://f1000research.com/posters/1095383
Trabalho de evento-resumo
CeTICSdb: an integrated platform for analysis of heterogeneous, high-throughput-omics and mathematical modeling of biochemical reactions (2014)
Nishiyama Junior, Milton Yutaka; Reis, Marcelo da Silva ; Silva, Daniel F; Kitano, Eduardo Shigueo; Souza, Gláucia Mendes ; Junqueira-de-Azevedo, Inácio de Loiola Meirelles; Cunha, Julia Pinheiro Chagas da; Barrera, Júnior; Iwai, Leo Kei; Serrano, Solange Maria de Toledo; Armelin, Hugo Aguirre
Fonte: Abstracts. Nome do evento: Annual International Conference on Intelligent Systems for Molecular Biology - ISMB 2014 Satellite Meeting - HiTSeq SIG: High Throughput Sequencing Algorithms & Applications. Unidades: IQ, IME
Assuntos: BIOINFORMÁTICA, COMPUTAÇÃO APLICADA, MODELOS MATEMÁTICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NISHIYAMA JUNIOR, Milton Yutaka et al. CeTICSdb: an integrated platform for analysis of heterogeneous, high-throughput-omics and mathematical modeling of biochemical reactions. 2014, Anais.. Bethesda: Instituto de Química, Universidade de São Paulo, 2014. Disponível em: https://f1000research.com/posters/1096242. Acesso em: 17 nov. 2024.
APA
Nishiyama Junior, M. Y., Reis, M. da S., Silva, D. F., Kitano, E. S., Souza, G. M., Junqueira-de-Azevedo, I. de L. M., et al. (2014). CeTICSdb: an integrated platform for analysis of heterogeneous, high-throughput-omics and mathematical modeling of biochemical reactions. In Abstracts. Bethesda: Instituto de Química, Universidade de São Paulo. Recuperado de https://f1000research.com/posters/1096242
NLM
Nishiyama Junior MY, Reis M da S, Silva DF, Kitano ES, Souza GM, Junqueira-de-Azevedo I de LM, Cunha JPC da, Barrera J, Iwai LK, Serrano SM de T, Armelin HA. CeTICSdb: an integrated platform for analysis of heterogeneous, high-throughput-omics and mathematical modeling of biochemical reactions [Internet]. Abstracts. 2014 ;[citado 2024 nov. 17 ] Available from: https://f1000research.com/posters/1096242
Vancouver
Nishiyama Junior MY, Reis M da S, Silva DF, Kitano ES, Souza GM, Junqueira-de-Azevedo I de LM, Cunha JPC da, Barrera J, Iwai LK, Serrano SM de T, Armelin HA. CeTICSdb: an integrated platform for analysis of heterogeneous, high-throughput-omics and mathematical modeling of biochemical reactions [Internet]. Abstracts. 2014 ;[citado 2024 nov. 17 ] Available from: https://f1000research.com/posters/1096242

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