Filtros : "ALMEIDA, SANDRO ROGERIO DE" "FCF" "Indexado na Base de dados Excerpta Medica" Limpar

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  • Source: The Journal of Infectious Diseases. Unidades: ICB, FCF

    Assunto: IMUNOLOGIA

    How to cite
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    • ABNT

      SPADARI, Karen et al. Interaction between paracoccidioides brasiliensis and pulmonary dendritic cells induces interleukin-10 production and toll-like receptor-2 expression: possible mechanisms of susceptibility. The Journal of Infectious Diseases, v. 196, n. 7, p. 1108-1115, 2007Tradução . . Acesso em: 05 ago. 2024.
    • APA

      Spadari, K., Bastos, K. R., Russo, M., & Almeida, S. R. de. (2007). Interaction between paracoccidioides brasiliensis and pulmonary dendritic cells induces interleukin-10 production and toll-like receptor-2 expression: possible mechanisms of susceptibility. The Journal of Infectious Diseases, 196( 7), 1108-1115.
    • NLM

      Spadari K, Bastos KR, Russo M, Almeida SR de. Interaction between paracoccidioides brasiliensis and pulmonary dendritic cells induces interleukin-10 production and toll-like receptor-2 expression: possible mechanisms of susceptibility. The Journal of Infectious Diseases. 2007 ; 196( 7): 1108-1115.[citado 2024 ago. 05 ]
    • Vancouver

      Spadari K, Bastos KR, Russo M, Almeida SR de. Interaction between paracoccidioides brasiliensis and pulmonary dendritic cells induces interleukin-10 production and toll-like receptor-2 expression: possible mechanisms of susceptibility. The Journal of Infectious Diseases. 2007 ; 196( 7): 1108-1115.[citado 2024 ago. 05 ]
  • Source: Toxicology. Unidades: ICB, FCF

    Assunto: FARMACOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      MACEDO, S. M. D. et al. In vivo hydroquinone exposure impairs allergic lung inflammation in rats. Toxicology, v. 241, n. 1-2, p. 47-57, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.tox.2007.08.085. Acesso em: 05 ago. 2024.
    • APA

      Macedo, S. M. D., Vaz, S. C. M., Lourenço, E. L. B., da Glória de Sousa, M., Ligeiro-Oliveira, A. P., Ferreira Jr., J. M. C., et al. (2007). In vivo hydroquinone exposure impairs allergic lung inflammation in rats. Toxicology, 241( 1-2), 47-57. doi:10.1016/j.tox.2007.08.085
    • NLM

      Macedo SMD, Vaz SCM, Lourenço ELB, da Glória de Sousa M, Ligeiro-Oliveira AP, Ferreira Jr. JMC, Almeida SR de, Lima WT de, Farsky SHP. In vivo hydroquinone exposure impairs allergic lung inflammation in rats [Internet]. Toxicology. 2007 ; 241( 1-2): 47-57.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.tox.2007.08.085
    • Vancouver

      Macedo SMD, Vaz SCM, Lourenço ELB, da Glória de Sousa M, Ligeiro-Oliveira AP, Ferreira Jr. JMC, Almeida SR de, Lima WT de, Farsky SHP. In vivo hydroquinone exposure impairs allergic lung inflammation in rats [Internet]. Toxicology. 2007 ; 241( 1-2): 47-57.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.tox.2007.08.085
  • Source: Journal of Leukocyte Biology. Unidades: ICB, FCF

    Assunto: IMUNOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      GHOSN, Eliver Eid Bou e RUSSO, Momtchilo e ALMEIDA, Sandro Rogério de. Nitric oxide-dependent killing of Cryptococcus neoformans by B-1-derived mononuclear phagocyte. Journal of Leukocyte Biology, v. 80, n. 1, p. 36-44, 2006Tradução . . Disponível em: https://doi.org/10.1189/jlb.1005603. Acesso em: 05 ago. 2024.
    • APA

      Ghosn, E. E. B., Russo, M., & Almeida, S. R. de. (2006). Nitric oxide-dependent killing of Cryptococcus neoformans by B-1-derived mononuclear phagocyte. Journal of Leukocyte Biology, 80( 1), 36-44. doi:10.1189/jlb.1005603
    • NLM

      Ghosn EEB, Russo M, Almeida SR de. Nitric oxide-dependent killing of Cryptococcus neoformans by B-1-derived mononuclear phagocyte [Internet]. Journal of Leukocyte Biology. 2006 ; 80( 1): 36-44.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1189/jlb.1005603
    • Vancouver

      Ghosn EEB, Russo M, Almeida SR de. Nitric oxide-dependent killing of Cryptococcus neoformans by B-1-derived mononuclear phagocyte [Internet]. Journal of Leukocyte Biology. 2006 ; 80( 1): 36-44.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1189/jlb.1005603

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