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Artigo de periodico
Phthalocyanine derivative attenuates TNF-α production in macrophage culture and prevents alveolar bone loss in experimental periodontitis (2024)
Breseghello, Isadora; Rosalen, Pedro Luiz; Bruzadelli, Rafaela Franco Dias; Araújo, Leonardo Pereira de; Abdalla, Henrique Ballassini; Lazarini, Josy Goldoni; Paiva, Isadora Marques; Bueno-Silva, Bruno; Cordeiro, Márcia Regina; Alencar, Severino Matias de; Vilhena, Fabiano Vieira; Cunha, Thiago Mattar; Fernandes, Leandro Araújo; Ikegaki, Masaharu; Marcelo Franchin, Marcelo
Source: Journal Periodontal Research. Unidades: ESALQ, FMRP
Subjects: EXPRESSÃO GÊNICA, GEL (FORMAS FARMACÊUTICAS), MACRÓFAGOS, PERIODONTITE, TERAPÊUTICA ODONTOLÓGICA
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ABNT
BRESEGHELLO, Isadora et al. Phthalocyanine derivative attenuates TNF-α production in macrophage culture and prevents alveolar bone loss in experimental periodontitis. Journal Periodontal Research, p. 1-7, 2024Tradução . . Disponível em: https://10.1111/jre.13341. Acesso em: 14 nov. 2024.
APA
Breseghello, I., Rosalen, P. L., Bruzadelli, R. F. D., Araújo, L. P. de, Abdalla, H. B., Lazarini, J. G., et al. (2024). Phthalocyanine derivative attenuates TNF-α production in macrophage culture and prevents alveolar bone loss in experimental periodontitis. Journal Periodontal Research, 1-7. doi:10.1111/jre.13341
NLM
Breseghello I, Rosalen PL, Bruzadelli RFD, Araújo LP de, Abdalla HB, Lazarini JG, Paiva IM, Bueno-Silva B, Cordeiro MR, Alencar SM de, Vilhena FV, Cunha TM, Fernandes LA, Ikegaki M, Marcelo Franchin M. Phthalocyanine derivative attenuates TNF-α production in macrophage culture and prevents alveolar bone loss in experimental periodontitis [Internet]. Journal Periodontal Research. 2024 ; 1-7.[citado 2024 nov. 14 ] Available from: https://10.1111/jre.13341
Vancouver
Breseghello I, Rosalen PL, Bruzadelli RFD, Araújo LP de, Abdalla HB, Lazarini JG, Paiva IM, Bueno-Silva B, Cordeiro MR, Alencar SM de, Vilhena FV, Cunha TM, Fernandes LA, Ikegaki M, Marcelo Franchin M. Phthalocyanine derivative attenuates TNF-α production in macrophage culture and prevents alveolar bone loss in experimental periodontitis [Internet]. Journal Periodontal Research. 2024 ; 1-7.[citado 2024 nov. 14 ] Available from: https://10.1111/jre.13341
Artigo de periodico
Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19 (2023)
Fraga-Silva, Thais Fernanda de Campos ; Rosa, Ualter Guilherme Cipriano; Fumagalli, Marcílio Jorge ; Corrêa, Giseli Furlan ; Fuzo, Carlos Alessandro ; Santos, Douglas dos; Mestriner, Fabíola Leslie Antunes Cardoso ; Ribeiro, Christiane Becari ; Teixeira-Carvalho, Andrea; Coelho-dos-Reis, Jordana; Menegueti, Mayra Gonçalves ; Figueiredo, Luiz Tadeu Moraes ; Cunha, Larissa Dias da; Martins-Filho, Olindo A.; Baruffi, Marcelo Dias ; Martins, Maria Auxiliadora ; Passaglia, Rita de Cassia Aleixo Tostes ; Bonato, Vânia Luiza Deperon
Source: iScience. Unidades: FMRP, EERP, FCFRP
Subjects: SISTEMA IMUNE, VIROLOGIA, MACRÓFAGOS
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FRAGA-SILVA, Thais Fernanda de Campos et al. Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19. iScience, v. 26, n. 12, p. 1-19, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2023.108366. Acesso em: 14 nov. 2024.
APA
Fraga-Silva, T. F. de C., Rosa, U. G. C., Fumagalli, M. J., Corrêa, G. F., Fuzo, C. A., Santos, D. dos, et al. (2023). Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19. iScience, 26( 12), 1-19. doi:10.1016/j.isci.2023.108366
NLM
Fraga-Silva TF de C, Rosa UGC, Fumagalli MJ, Corrêa GF, Fuzo CA, Santos D dos, Mestriner FLAC, Ribeiro CB, Teixeira-Carvalho A, Coelho-dos-Reis J, Menegueti MG, Figueiredo LTM, Cunha LD da, Martins-Filho OA, Baruffi MD, Martins MA, Passaglia R de CAT, Bonato VLD. Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19 [Internet]. iScience. 2023 ; 26( 12): 1-19.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.isci.2023.108366
Vancouver
Fraga-Silva TF de C, Rosa UGC, Fumagalli MJ, Corrêa GF, Fuzo CA, Santos D dos, Mestriner FLAC, Ribeiro CB, Teixeira-Carvalho A, Coelho-dos-Reis J, Menegueti MG, Figueiredo LTM, Cunha LD da, Martins-Filho OA, Baruffi MD, Martins MA, Passaglia R de CAT, Bonato VLD. Airway epithelial cells and macrophages trigger IL-6-CD95/CD95L axis and mediate initial immunopathology of COVID-19 [Internet]. iScience. 2023 ; 26( 12): 1-19.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.isci.2023.108366
Artigo de periodico
CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation (2023)
Rodrigues, Tamara Silva ; Caetano, Camila C. S.; Sá, Keyla Santos Guedes de ; Almeida, Leticia de; Becerra, Amanda de Matos; Gonçalves, Augusto Velozo ; Lopes, Letícia de Sousa; Oliveira, Samuel; Mascarenhas, Danielle Pini Alves; Batah, Sabrina Setembre ; Silva, Bruna Manuella Souza; Gomes, Giovanni Freitas; Castro, Ricardo de; Martins Júnior, Ronaldo Bragança; Avila, Jonathan; Frantz, Fabiani Gai ; Cunha, Thiago Mattar ; Arruda Neto, Eurico de; Cunha, Fernando Queiroz ; Helder Takashi Imoto Nakaya ; Cunha, Larissa Dias da; Fabro, Alexandre Todorovic ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de; Zamboni, Dario Simões
Source: The Journal of Infectious Diseases. Unidades: FMRP, FCFRP
Subjects: COVID-19, MACRÓFAGOS, CORONAVIRUS
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RODRIGUES, Tamara Silva et al. CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation. The Journal of Infectious Diseases, v. 227, n. 12, p. 1364-1375, 2023Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiad037. Acesso em: 14 nov. 2024.
APA
Rodrigues, T. S., Caetano, C. C. S., Sá, K. S. G. de, Almeida, L. de, Becerra, A. de M., Gonçalves, A. V., et al. (2023). CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation. The Journal of Infectious Diseases, 227( 12), 1364-1375. doi:10.1093/infdis/jiad037
NLM
Rodrigues TS, Caetano CCS, Sá KSG de, Almeida L de, Becerra A de M, Gonçalves AV, Lopes L de S, Oliveira S, Mascarenhas DPA, Batah SS, Silva BMS, Gomes GF, Castro R de, Martins Júnior RB, Avila J, Frantz FG, Cunha TM, Arruda Neto E de, Cunha FQ, Helder Takashi Imoto Nakaya, Cunha LD da, Fabro AT, Louzada Júnior P, Oliveira RDR de, Zamboni DS. CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation [Internet]. The Journal of Infectious Diseases. 2023 ; 227( 12): 1364-1375.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/infdis/jiad037
Vancouver
Rodrigues TS, Caetano CCS, Sá KSG de, Almeida L de, Becerra A de M, Gonçalves AV, Lopes L de S, Oliveira S, Mascarenhas DPA, Batah SS, Silva BMS, Gomes GF, Castro R de, Martins Júnior RB, Avila J, Frantz FG, Cunha TM, Arruda Neto E de, Cunha FQ, Helder Takashi Imoto Nakaya, Cunha LD da, Fabro AT, Louzada Júnior P, Oliveira RDR de, Zamboni DS. CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation [Internet]. The Journal of Infectious Diseases. 2023 ; 227( 12): 1364-1375.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/infdis/jiad037
Artigo de periodico
A study on the anti-NF-κB, anti-candida, and antioxidant activities of two natural plant hormones: Gibberellin A4 and A7 (2022)
Nani, Bruno Dias; Rosalen, Pedro Luiz ; Lazarini, Josy Goldoni; Sardi, Janaína de Cássia Orlandi; Romário-Silva, Diego; Araújo, Leonardo Pereira de ; Reis, Mateus Silva Beker dos ; Breseghello, Isadora ; Cunha, Thiago Mattar ; Alencar, Severino Matias de ; Silveira, Nelson José Freitas da ; Franchin, Marcelo
Source: Pharmaceutics. Unidades: FMRP, ESALQ
Subjects: ANTIFÚNGICOS, ANTIOXIDANTES, CANDIDA ALBICANS, FATORES DE TRANSCRIÇÃO, HORMÔNIOS VEGETAIS, MACRÓFAGOS, PRODUTOS NATURAIS, TERPENOS, TRAÇAS
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NANI, Bruno Dias et al. A study on the anti-NF-κB, anti-candida, and antioxidant activities of two natural plant hormones: Gibberellin A4 and A7. Pharmaceutics, v. 14, p. 1-11, 2022Tradução . . Disponível em: https://doi.org/10.3390/pharmaceutics14071347. Acesso em: 14 nov. 2024.
APA
Nani, B. D., Rosalen, P. L., Lazarini, J. G., Sardi, J. de C. O., Romário-Silva, D., Araújo, L. P. de, et al. (2022). A study on the anti-NF-κB, anti-candida, and antioxidant activities of two natural plant hormones: Gibberellin A4 and A7. Pharmaceutics, 14, 1-11. doi:10.3390/pharmaceutics14071347
NLM
Nani BD, Rosalen PL, Lazarini JG, Sardi J de CO, Romário-Silva D, Araújo LP de, Reis MSB dos, Breseghello I, Cunha TM, Alencar SM de, Silveira NJF da, Franchin M. A study on the anti-NF-κB, anti-candida, and antioxidant activities of two natural plant hormones: Gibberellin A4 and A7 [Internet]. Pharmaceutics. 2022 ; 14 1-11.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3390/pharmaceutics14071347
Vancouver
Nani BD, Rosalen PL, Lazarini JG, Sardi J de CO, Romário-Silva D, Araújo LP de, Reis MSB dos, Breseghello I, Cunha TM, Alencar SM de, Silveira NJF da, Franchin M. A study on the anti-NF-κB, anti-candida, and antioxidant activities of two natural plant hormones: Gibberellin A4 and A7 [Internet]. Pharmaceutics. 2022 ; 14 1-11.[citado 2024 nov. 14 ] Available from: https://doi.org/10.3390/pharmaceutics14071347
Artigo de periodico
Aldosterone, inflammation, immune system, and hypertension (2021)
Ferreira, Nathanne dos Santos; Tostes, Rita de Cássia Aleixo ; Paradis, Pierre; Schiffrin, Ernesto L.
Source: American Journal of Hypertension. Unidade: FMRP
Subjects: PRESSÃO SANGUÍNEA, SISTEMA IMUNE, LINFÓCITOS, MONÓCITOS, MACRÓFAGOS
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FERREIRA, Nathanne dos Santos et al. Aldosterone, inflammation, immune system, and hypertension. American Journal of Hypertension, v. 34, n. 1, p. 15-27, 2021Tradução . . Disponível em: https://doi.org/10.1093/ajh/hpaa137. Acesso em: 14 nov. 2024.
APA
Ferreira, N. dos S., Tostes, R. de C. A., Paradis, P., & Schiffrin, E. L. (2021). Aldosterone, inflammation, immune system, and hypertension. American Journal of Hypertension, 34( 1), 15-27. doi:10.1093/ajh/hpaa137
NLM
Ferreira N dos S, Tostes R de CA, Paradis P, Schiffrin EL. Aldosterone, inflammation, immune system, and hypertension [Internet]. American Journal of Hypertension. 2021 ; 34( 1): 15-27.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/ajh/hpaa137
Vancouver
Ferreira N dos S, Tostes R de CA, Paradis P, Schiffrin EL. Aldosterone, inflammation, immune system, and hypertension [Internet]. American Journal of Hypertension. 2021 ; 34( 1): 15-27.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/ajh/hpaa137
Artigo de periodico
Inflammatory effect of Bothropstoxin-I from Bothrops jararacussu venom mediated by NLRP3 inflammasome involves ATP and P2X7 receptor (2021)
Silva, Priscila de Andrade Ranéia e; Lima, Dhêmerson Souza de ; Luiz, João Paulo Mesquita ; Câmara, Niels Olsen Saraiva ; Alves Filho, José Carlos Farias ; Pontillo, Alessandra ; Bortoluci, Karina Ramalho ; Mauro, Eliana Faquim de Lima
Source: Clinical Science. Unidades: ICB, FMRP
Subjects: IMUNOLOGIA, MACRÓFAGOS, INFLAMAÇÃO, VENENOS DE ORIGEM ANIMAL, SERPENTES, FOSFOLIPASES A, ATIVAÇÃO ENZIMÁTICA, CATALASE, INTERLEUCINAS
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SILVA, Priscila de Andrade Ranéia e et al. Inflammatory effect of Bothropstoxin-I from Bothrops jararacussu venom mediated by NLRP3 inflammasome involves ATP and P2X7 receptor. Clinical Science, v. 135, n. 5, p. 687–701, 2021Tradução . . Disponível em: https://doi.org/10.1042/CS20201419. Acesso em: 14 nov. 2024.
APA
Silva, P. de A. R. e, Lima, D. S. de, Luiz, J. P. M., Câmara, N. O. S., Alves Filho, J. C. F., Pontillo, A., et al. (2021). Inflammatory effect of Bothropstoxin-I from Bothrops jararacussu venom mediated by NLRP3 inflammasome involves ATP and P2X7 receptor. Clinical Science, 135( 5), 687–701. doi:10.1042/CS20201419
NLM
Silva P de AR e, Lima DS de, Luiz JPM, Câmara NOS, Alves Filho JCF, Pontillo A, Bortoluci KR, Mauro EF de L. Inflammatory effect of Bothropstoxin-I from Bothrops jararacussu venom mediated by NLRP3 inflammasome involves ATP and P2X7 receptor [Internet]. Clinical Science. 2021 ; 135( 5): 687–701.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1042/CS20201419
Vancouver
Silva P de AR e, Lima DS de, Luiz JPM, Câmara NOS, Alves Filho JCF, Pontillo A, Bortoluci KR, Mauro EF de L. Inflammatory effect of Bothropstoxin-I from Bothrops jararacussu venom mediated by NLRP3 inflammasome involves ATP and P2X7 receptor [Internet]. Clinical Science. 2021 ; 135( 5): 687–701.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1042/CS20201419
Artigo de periodico
STING regulates metabolic reprogramming in macrophages via HIF-1α during Brucella infection (2021)
Gomes, Marco Tulio R.; Guimarães, Erika S.; Marinho, Fabio V.; Macedo, Isabella; Aguiar, Eric R. G. R.; Barber, Glen N.; Moraes-Vieira, Pedro M. M.; Alves Filho, José Carlos Farias ; Oliveira, Sergio C.
Source: PLOS Pathogens. Unidade: FMRP
Subjects: MACRÓFAGOS, INFLAMAÇÃO, INFECÇÕES RESPIRATÓRIAS, BRUCELOSE, MITOCÔNDRIAS, REAÇÃO EM CADEIA POR POLIMERASE
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GOMES, Marco Tulio R. et al. STING regulates metabolic reprogramming in macrophages via HIF-1α during Brucella infection. PLOS Pathogens, 2021Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1009597. Acesso em: 14 nov. 2024.
APA
Gomes, M. T. R., Guimarães, E. S., Marinho, F. V., Macedo, I., Aguiar, E. R. G. R., Barber, G. N., et al. (2021). STING regulates metabolic reprogramming in macrophages via HIF-1α during Brucella infection. PLOS Pathogens. doi:10.1371/journal.ppat.1009597
NLM
Gomes MTR, Guimarães ES, Marinho FV, Macedo I, Aguiar ERGR, Barber GN, Moraes-Vieira PMM, Alves Filho JCF, Oliveira SC. STING regulates metabolic reprogramming in macrophages via HIF-1α during Brucella infection [Internet]. PLOS Pathogens. 2021 ;[citado 2024 nov. 14 ] Available from: https://doi.org/10.1371/journal.ppat.1009597
Vancouver
Gomes MTR, Guimarães ES, Marinho FV, Macedo I, Aguiar ERGR, Barber GN, Moraes-Vieira PMM, Alves Filho JCF, Oliveira SC. STING regulates metabolic reprogramming in macrophages via HIF-1α during Brucella infection [Internet]. PLOS Pathogens. 2021 ;[citado 2024 nov. 14 ] Available from: https://doi.org/10.1371/journal.ppat.1009597
Artigo de periodico
Molecular basis of carrageenan-induced cytokines production in macrophages (2020)
Lopes, Alexandre Hashimoto Pereira; Silva, Rangel Leal; Fonseca, Miriam das Dores Mendes; Gomes, Francisco Isaac Fernandes; Maganin, Alexandre Gomes de Macedo; Ribeiro, Lucas S.; Marques, Lucas Maciel Mauriz; Cunha, Fernando de Queiroz ; Alves Filho, José Carlos Farias ; Zamboni, Dario Simões; Lopes, Norberto Peporine ; Franklin, Bernardo S.; Gombault, Aurélie; Ramalho, Fernando Silva ; Quesniaux, Valérie F. J.; Couillin, Isabelle; Ryffel, Bernhard; Cunha, Thiago Mattar
Source: Cell Communication and Signaling. Unidades: FCFRP, FMRP
Subjects: CARRAGENINA, MACRÓFAGOS, POLISSACARÍDEOS, CITOCINAS, INFLAMAÇÃO
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LOPES, Alexandre Hashimoto Pereira et al. Molecular basis of carrageenan-induced cytokines production in macrophages. Cell Communication and Signaling, v. 18, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.1186/s12964-020-00621-x. Acesso em: 14 nov. 2024.
APA
Lopes, A. H. P., Silva, R. L., Fonseca, M. das D. M., Gomes, F. I. F., Maganin, A. G. de M., Ribeiro, L. S., et al. (2020). Molecular basis of carrageenan-induced cytokines production in macrophages. Cell Communication and Signaling, 18( 1). doi:10.1186/s12964-020-00621-x
NLM
Lopes AHP, Silva RL, Fonseca M das DM, Gomes FIF, Maganin AG de M, Ribeiro LS, Marques LMM, Cunha F de Q, Alves Filho JCF, Zamboni DS, Lopes NP, Franklin BS, Gombault A, Ramalho FS, Quesniaux VFJ, Couillin I, Ryffel B, Cunha TM. Molecular basis of carrageenan-induced cytokines production in macrophages [Internet]. Cell Communication and Signaling. 2020 ; 18( 1):[citado 2024 nov. 14 ] Available from: https://doi.org/10.1186/s12964-020-00621-x
Vancouver
Lopes AHP, Silva RL, Fonseca M das DM, Gomes FIF, Maganin AG de M, Ribeiro LS, Marques LMM, Cunha F de Q, Alves Filho JCF, Zamboni DS, Lopes NP, Franklin BS, Gombault A, Ramalho FS, Quesniaux VFJ, Couillin I, Ryffel B, Cunha TM. Molecular basis of carrageenan-induced cytokines production in macrophages [Internet]. Cell Communication and Signaling. 2020 ; 18( 1):[citado 2024 nov. 14 ] Available from: https://doi.org/10.1186/s12964-020-00621-x
Artigo de periodico
IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis (2020)
Fattori, Victor; Staurengo-Ferrari, Larissa; Zaninelli, Tiago H.; Casagrande, Rúbia; Oliveira, Renê Donizeti Ribeiro de; Louzada Júnior, Paulo; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias ; Teixeira, Mauro Martins; Cunha, Fernando de Queiroz ; Amaral, Flávio Almeida; Verri Júnior, Waldiceu Aparecido
Source: Inflammation Research. Unidade: FMRP
Subjects: ARTRITE, INFLAMAÇÃO, INTERLEUCINA 1, MACRÓFAGOS, DOR, PEROXIDASE, MEMBRANA SINOVIAL, ÁCIDO ÚRICO
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ABNT
FATTORI, Victor et al. IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis. Inflammation Research, v. 69, n. 12, p. 1271-1282, 2020Tradução . . Disponível em: https://doi.org/10.1007/s00011-020-01399-x. Acesso em: 14 nov. 2024.
APA
Fattori, V., Staurengo-Ferrari, L., Zaninelli, T. H., Casagrande, R., Oliveira, R. D. R. de, Louzada Júnior, P., et al. (2020). IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis. Inflammation Research, 69( 12), 1271-1282. doi:10.1007/s00011-020-01399-x
NLM
Fattori V, Staurengo-Ferrari L, Zaninelli TH, Casagrande R, Oliveira RDR de, Louzada Júnior P, Cunha TM, Alves Filho JCF, Teixeira MM, Cunha F de Q, Amaral FA, Verri Júnior WA. IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis [Internet]. Inflammation Research. 2020 ; 69( 12): 1271-1282.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s00011-020-01399-x
Vancouver
Fattori V, Staurengo-Ferrari L, Zaninelli TH, Casagrande R, Oliveira RDR de, Louzada Júnior P, Cunha TM, Alves Filho JCF, Teixeira MM, Cunha F de Q, Amaral FA, Verri Júnior WA. IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis [Internet]. Inflammation Research. 2020 ; 69( 12): 1271-1282.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s00011-020-01399-x
Artigo de periodico
Activation of PPAR-γ induces macrophage polarization and reduces neutrophil migration mediated by heme oxygenase 1 (2020)
Abdalla, Henrique Ballassini; Napimoga, Marcelo Henrique; Lopes, Alexandre Hashimoto Pereira; Maganin, Alexandre Gomes de Macedo; Cunha, Thiago Mattar; Van Dyke, Thomas E.; Napimoga, Juliana T. Clemente
Source: International Immunopharmacology. Unidade: FMRP
Subjects: CÁLCIO, CALMODULINA, IMUNOLOGIA, CARRAGENINA, CITOCINAS, LIPOPOLISSACARÍDEOS, NEUTRÓFILOS, ÓXIDO NÍTRICO, PROSTAGLANDINAS, ARTICULAÇÃO TEMPOROMANDIBULAR, MACRÓFAGOS
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ABNT
ABDALLA, Henrique Ballassini et al. Activation of PPAR-γ induces macrophage polarization and reduces neutrophil migration mediated by heme oxygenase 1. International Immunopharmacology, v. 84, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2020.106565. Acesso em: 14 nov. 2024.
APA
Abdalla, H. B., Napimoga, M. H., Lopes, A. H. P., Maganin, A. G. de M., Cunha, T. M., Van Dyke, T. E., & Napimoga, J. T. C. (2020). Activation of PPAR-γ induces macrophage polarization and reduces neutrophil migration mediated by heme oxygenase 1. International Immunopharmacology, 84. doi:10.1016/j.intimp.2020.106565
NLM
Abdalla HB, Napimoga MH, Lopes AHP, Maganin AG de M, Cunha TM, Van Dyke TE, Napimoga JTC. Activation of PPAR-γ induces macrophage polarization and reduces neutrophil migration mediated by heme oxygenase 1 [Internet]. International Immunopharmacology. 2020 ; 84[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.intimp.2020.106565
Vancouver
Abdalla HB, Napimoga MH, Lopes AHP, Maganin AG de M, Cunha TM, Van Dyke TE, Napimoga JTC. Activation of PPAR-γ induces macrophage polarization and reduces neutrophil migration mediated by heme oxygenase 1 [Internet]. International Immunopharmacology. 2020 ; 84[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.intimp.2020.106565
Artigo de periodico
Monosodium urate crystal interleukin-1β release is dependent on Toll-like receptor 4 and transient receptor potential V1 activation (2020)
Rossato, Mateus Fortes; Hoffmeister, Carin; Trevisan, Gabriela; Bezerra, Fábio; Cunha, Thiago Mattar; Ferreira, Juliano; Silva, Cássia Regina
Source: Rheumatology. Unidade: FMRP
Subjects: NOCICEPTIVIDADE, INFLAMAÇÃO, MACRÓFAGOS, ARTRITE, CITOCINAS
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ROSSATO, Mateus Fortes et al. Monosodium urate crystal interleukin-1β release is dependent on Toll-like receptor 4 and transient receptor potential V1 activation. Rheumatology, v. 59, n. 1, p. 233-242, 2020Tradução . . Disponível em: https://doi.org/10.1093/rheumatology/kez259. Acesso em: 14 nov. 2024.
APA
Rossato, M. F., Hoffmeister, C., Trevisan, G., Bezerra, F., Cunha, T. M., Ferreira, J., & Silva, C. R. (2020). Monosodium urate crystal interleukin-1β release is dependent on Toll-like receptor 4 and transient receptor potential V1 activation. Rheumatology, 59( 1), 233-242. doi:10.1093/rheumatology/kez259
NLM
Rossato MF, Hoffmeister C, Trevisan G, Bezerra F, Cunha TM, Ferreira J, Silva CR. Monosodium urate crystal interleukin-1β release is dependent on Toll-like receptor 4 and transient receptor potential V1 activation [Internet]. Rheumatology. 2020 ; 59( 1): 233-242.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/rheumatology/kez259
Vancouver
Rossato MF, Hoffmeister C, Trevisan G, Bezerra F, Cunha TM, Ferreira J, Silva CR. Monosodium urate crystal interleukin-1β release is dependent on Toll-like receptor 4 and transient receptor potential V1 activation [Internet]. Rheumatology. 2020 ; 59( 1): 233-242.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/rheumatology/kez259
Artigo de periodico
CD163 overexpression using a macrophage-directed gene therapy approach improves wound healing in ex vivo and in vivo human skin models (2020)
Ferreira, David Wilson; Ulecia-Morón, Cristina; Alvarado-Vázquez, Perla Abigail; Cunnane, Katharine; Moracho-Vilriales, Carolina; Grosick, Rachel L.; Cunha, Thiago Mattar ; Romero-Sandoval, Edgar Alfonso
Source: Immunobiology. Unidade: FMRP
Subjects: NANOTECNOLOGIA, NANOPARTÍCULAS, INFLAMAÇÃO, FIBROBLASTOS, DOR, MACRÓFAGOS, CICATRIZAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, David Wilson et al. CD163 overexpression using a macrophage-directed gene therapy approach improves wound healing in ex vivo and in vivo human skin models. Immunobiology, v. 225, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2019.10.011. Acesso em: 14 nov. 2024.
APA
Ferreira, D. W., Ulecia-Morón, C., Alvarado-Vázquez, P. A., Cunnane, K., Moracho-Vilriales, C., Grosick, R. L., et al. (2020). CD163 overexpression using a macrophage-directed gene therapy approach improves wound healing in ex vivo and in vivo human skin models. Immunobiology, 225( 1). doi:10.1016/j.imbio.2019.10.011
NLM
Ferreira DW, Ulecia-Morón C, Alvarado-Vázquez PA, Cunnane K, Moracho-Vilriales C, Grosick RL, Cunha TM, Romero-Sandoval EA. CD163 overexpression using a macrophage-directed gene therapy approach improves wound healing in ex vivo and in vivo human skin models [Internet]. Immunobiology. 2020 ; 225( 1):[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.imbio.2019.10.011
Vancouver
Ferreira DW, Ulecia-Morón C, Alvarado-Vázquez PA, Cunnane K, Moracho-Vilriales C, Grosick RL, Cunha TM, Romero-Sandoval EA. CD163 overexpression using a macrophage-directed gene therapy approach improves wound healing in ex vivo and in vivo human skin models [Internet]. Immunobiology. 2020 ; 225( 1):[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.imbio.2019.10.011
Artigo de periodico
MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression (2020)
Luiz, João Paulo Mesquita ; Toller‐Kawahisa, Juliana E.; Viacava, Paula Ramos; Nascimento, Daniele C.; Pereira, Priscilla T.; Saraiva, André L.; Prado, Douglas da Silva ; Le Bert, Marc; Giurisato, Emanuele; Tournier, Cathy; Cunha, Thiago Mattar ; Cunha, Fernando Queiróz ; Quesniaux, Valerie; Ryffel, Bernhard; Alves Filho, José Carlos Farias
Source: Journal of Leukocyte Biology. Unidade: FMRP
Subjects: DIFERENCIAÇÃO CELULAR, ANTÍGENOS, IMUNOLOGIA, REGULAÇÃO GÊNICA, EXPRESSÃO GÊNICA, INTERLEUCINA 4, MACRÓFAGOS, PROTEÍNAS PROTO-ONCOGÊNICAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LUIZ, João Paulo Mesquita et al. MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression. Journal of Leukocyte Biology, v. 108, p. 1215-1223, 2020Tradução . . Disponível em: https://doi.org/10.1002/JLB.1MA0520-016R. Acesso em: 14 nov. 2024.
APA
Luiz, J. P. M., Toller‐Kawahisa, J. E., Viacava, P. R., Nascimento, D. C., Pereira, P. T., Saraiva, A. L., et al. (2020). MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression. Journal of Leukocyte Biology, 108, 1215-1223. doi:10.1002/JLB.1MA0520-016R
NLM
Luiz JPM, Toller‐Kawahisa JE, Viacava PR, Nascimento DC, Pereira PT, Saraiva AL, Prado D da S, Le Bert M, Giurisato E, Tournier C, Cunha TM, Cunha FQ, Quesniaux V, Ryffel B, Alves Filho JCF. MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression [Internet]. Journal of Leukocyte Biology. 2020 ; 108 1215-1223.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1002/JLB.1MA0520-016R
Vancouver
Luiz JPM, Toller‐Kawahisa JE, Viacava PR, Nascimento DC, Pereira PT, Saraiva AL, Prado D da S, Le Bert M, Giurisato E, Tournier C, Cunha TM, Cunha FQ, Quesniaux V, Ryffel B, Alves Filho JCF. MEK5/ERK5 signaling mediates IL‐4‐induced M2 macrophage differentiation through regulation of c‐Myc expression [Internet]. Journal of Leukocyte Biology. 2020 ; 108 1215-1223.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1002/JLB.1MA0520-016R
Artigo de periodico
Vagus nerve regulates the phagocytic and secretory activity of resident macrophages in the liver (2019)
Fonseca, Roberta Cristelli; Bassi, Gabriel Shimizu; Brito, Camila Carvalho; Rosa, Lorena Barreto; David, Bruna Araujo; Araujo, Alan Moreira de; Nóbrega, Natália; Diniz, Ariane Barros; Jesus, Itamar Couto Guedes; Barcelos, Lucíola Silva; Fontes, Marco Antonio Peliky; Bonaventura, Daniella; Kanashiro, Alexandre; Cunha, Thiago Mattar ; Guatimosim, Silvia; Cardoso, Valbert Nascimento; Fernandes, Simone Odília Antunes; Menezes, Gustavo Batista de; de Lartigue, Guillaume; Oliveira, André Gustavo
Source: Brain, Behavior, and Immunity. Unidade: FMRP
Subjects: NERVO VAGO, FÍGADO, FAGOCITOSE, RECEPTORES COLINÉRGICOS, MACRÓFAGOS
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ABNT
FONSECA, Roberta Cristelli et al. Vagus nerve regulates the phagocytic and secretory activity of resident macrophages in the liver. Brain, Behavior, and Immunity, v. 81, p. 444-454, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2019.06.041. Acesso em: 14 nov. 2024.
APA
Fonseca, R. C., Bassi, G. S., Brito, C. C., Rosa, L. B., David, B. A., Araujo, A. M. de, et al. (2019). Vagus nerve regulates the phagocytic and secretory activity of resident macrophages in the liver. Brain, Behavior, and Immunity, 81, 444-454. doi:10.1016/j.bbi.2019.06.041
NLM
Fonseca RC, Bassi GS, Brito CC, Rosa LB, David BA, Araujo AM de, Nóbrega N, Diniz AB, Jesus ICG, Barcelos LS, Fontes MAP, Bonaventura D, Kanashiro A, Cunha TM, Guatimosim S, Cardoso VN, Fernandes SOA, Menezes GB de, de Lartigue G, Oliveira AG. Vagus nerve regulates the phagocytic and secretory activity of resident macrophages in the liver [Internet]. Brain, Behavior, and Immunity. 2019 ; 81 444-454.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.bbi.2019.06.041
Vancouver
Fonseca RC, Bassi GS, Brito CC, Rosa LB, David BA, Araujo AM de, Nóbrega N, Diniz AB, Jesus ICG, Barcelos LS, Fontes MAP, Bonaventura D, Kanashiro A, Cunha TM, Guatimosim S, Cardoso VN, Fernandes SOA, Menezes GB de, de Lartigue G, Oliveira AG. Vagus nerve regulates the phagocytic and secretory activity of resident macrophages in the liver [Internet]. Brain, Behavior, and Immunity. 2019 ; 81 444-454.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.bbi.2019.06.041
Artigo de periodico
CCR2 plays a protective role in rocio virus–induced encephalitis by promoting macrophage infiltration into the brain (2019)
Ortiz, Alberto Anastacio Amarilla ; Santos Junior, Nilton Nascimento dos ; Figueiredo, Mário Luis Garcia de ; Luiz, João Paulo Mesquita; Fumagalli, Marcílio Jorge ; morelo, David Fernando Colon ; Silva, Veronica Lippi Oliveira da ; Castro, Helda Liz Alfonso; Lima Júnior, Djalma de Souza ; Trabuco, Amanda Cristina; Spinieli, Richard Leandro; Desiderá, Amanda de Carvalho ; Leite-Panissi, Christie Ramos Andrade ; Lauretti, Flávio ; Mendoza, Silvia Elena Sanchez ; Silva, Cleide Lúcia Araújo; Rego, Eduardo Magalhaes ; Lima, Leonardo Judson Galvão de; Bassi, Gabriel Shimizu ; Martins, Sandra Lucia Balero Penharvel; Manrique, Wilson Gomez; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz ; Peng, Nias Y. G.; Modhiran, Naphak; Setoh, Yin Xiang; Khromykh, Alexander A; Figueiredo, Luiz Tadeu Moraes ; Quintana, Victor Hugo Aquino
Source: The Journal of Infectious Diseases. Unidades: FCFRP, FFCLRP, FM, FMRP, FORP
Subjects: ENCEFALITE, MACRÓFAGOS, FLAVIVIRUS
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ABNT
ORTIZ, Alberto Anastacio Amarilla et al. CCR2 plays a protective role in rocio virus–induced encephalitis by promoting macrophage infiltration into the brain. The Journal of Infectious Diseases, v. 219, n. 12, p. 2015-2025, 2019Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiz029. Acesso em: 14 nov. 2024.
APA
Ortiz, A. A. A., Santos Junior, N. N. dos, Figueiredo, M. L. G. de, Luiz, J. P. M., Fumagalli, M. J., morelo, D. F. C., et al. (2019). CCR2 plays a protective role in rocio virus–induced encephalitis by promoting macrophage infiltration into the brain. The Journal of Infectious Diseases, 219( 12), 2015-2025. doi:10.1093/infdis/jiz029
NLM
Ortiz AAA, Santos Junior NN dos, Figueiredo MLG de, Luiz JPM, Fumagalli MJ, morelo DFC, Silva VLO da, Castro HLA, Lima Júnior D de S, Trabuco AC, Spinieli RL, Desiderá A de C, Leite-Panissi CRA, Lauretti F, Mendoza SES, Silva CLA, Rego EM, Lima LJG de, Bassi GS, Martins SLBP, Manrique WG, Alves Filho JCF, Cunha F de Q, Peng NYG, Modhiran N, Setoh YX, Khromykh AA, Figueiredo LTM, Quintana VHA. CCR2 plays a protective role in rocio virus–induced encephalitis by promoting macrophage infiltration into the brain [Internet]. The Journal of Infectious Diseases. 2019 ; 219( 12): 2015-2025.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/infdis/jiz029
Vancouver
Ortiz AAA, Santos Junior NN dos, Figueiredo MLG de, Luiz JPM, Fumagalli MJ, morelo DFC, Silva VLO da, Castro HLA, Lima Júnior D de S, Trabuco AC, Spinieli RL, Desiderá A de C, Leite-Panissi CRA, Lauretti F, Mendoza SES, Silva CLA, Rego EM, Lima LJG de, Bassi GS, Martins SLBP, Manrique WG, Alves Filho JCF, Cunha F de Q, Peng NYG, Modhiran N, Setoh YX, Khromykh AA, Figueiredo LTM, Quintana VHA. CCR2 plays a protective role in rocio virus–induced encephalitis by promoting macrophage infiltration into the brain [Internet]. The Journal of Infectious Diseases. 2019 ; 219( 12): 2015-2025.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/infdis/jiz029
Artigo de periodico
The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β (2018)
Ruiz-Miyazawa, Kenji W.; Borghi, Sergio M.; Ribeiro, Felipe A. Pinho; Ferrari, Larissa Staurengo; Fattori, Victor; Fernandes, Glaura S. A.; Casella, Antonio M.; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Casagrande, Rubia; Verri, Waldiceu A.
Source: Journal of Functional Foods. Unidade: FMRP
Subjects: ARTRITE, DOR, INFLAMAÇÃO, RATOS, MACRÓFAGOS
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ABNT
RUIZ-MIYAZAWA, Kenji W. et al. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β. Journal of Functional Foods, v. 48, p. 106-116, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jff.2018.06.025. Acesso em: 14 nov. 2024.
APA
Ruiz-Miyazawa, K. W., Borghi, S. M., Ribeiro, F. A. P., Ferrari, L. S., Fattori, V., Fernandes, G. S. A., et al. (2018). The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β. Journal of Functional Foods, 48, 106-116. doi:10.1016/j.jff.2018.06.025
NLM
Ruiz-Miyazawa KW, Borghi SM, Ribeiro FAP, Ferrari LS, Fattori V, Fernandes GSA, Casella AM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri WA. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β [Internet]. Journal of Functional Foods. 2018 ; 48 106-116.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.jff.2018.06.025
Vancouver
Ruiz-Miyazawa KW, Borghi SM, Ribeiro FAP, Ferrari LS, Fattori V, Fernandes GSA, Casella AM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri WA. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β [Internet]. Journal of Functional Foods. 2018 ; 48 106-116.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.jff.2018.06.025
Artigo de periodico
Probucol attenuates lipopolysaccharide-induced leukocyte recruitment and inflammatory hyperalgesia: effect on NF-кB activation and cytokine production (2017)
Zucoloto, Amanda Z.; Manchope, Marília F.; Staurengo-Ferrari, Larissa; Pinho-Ribeiro, Felipe A.; Zarpelon, Ana C.; Saraiva, André L.L.; Cecílio, Nerry Tatiana; Alves-Filho, José Carlos; Cunha, Thiago Mattar; Menezes, Gustavo B.; Cunha, Fernando de Queiroz; Casagrande, Rubia; Verri, Waldiceu A.
Source: European Journal of Pharmacology. Unidade: FMRP
Subjects: INFLAMAÇÃO, NOCICEPTORES, DOR, LEUCÓCITOS, LIPOPOLISSACARÍDEOS, MACRÓFAGOS
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ABNT
ZUCOLOTO, Amanda Z. et al. Probucol attenuates lipopolysaccharide-induced leukocyte recruitment and inflammatory hyperalgesia: effect on NF-кB activation and cytokine production. European Journal of Pharmacology, v. 809, p. 52-63, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2017.05.016. Acesso em: 14 nov. 2024.
APA
Zucoloto, A. Z., Manchope, M. F., Staurengo-Ferrari, L., Pinho-Ribeiro, F. A., Zarpelon, A. C., Saraiva, A. L. L., et al. (2017). Probucol attenuates lipopolysaccharide-induced leukocyte recruitment and inflammatory hyperalgesia: effect on NF-кB activation and cytokine production. European Journal of Pharmacology, 809, 52-63. doi:10.1016/j.ejphar.2017.05.016
NLM
Zucoloto AZ, Manchope MF, Staurengo-Ferrari L, Pinho-Ribeiro FA, Zarpelon AC, Saraiva ALL, Cecílio NT, Alves-Filho JC, Cunha TM, Menezes GB, Cunha F de Q, Casagrande R, Verri WA. Probucol attenuates lipopolysaccharide-induced leukocyte recruitment and inflammatory hyperalgesia: effect on NF-кB activation and cytokine production [Internet]. European Journal of Pharmacology. 2017 ; 809 52-63.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.ejphar.2017.05.016
Vancouver
Zucoloto AZ, Manchope MF, Staurengo-Ferrari L, Pinho-Ribeiro FA, Zarpelon AC, Saraiva ALL, Cecílio NT, Alves-Filho JC, Cunha TM, Menezes GB, Cunha F de Q, Casagrande R, Verri WA. Probucol attenuates lipopolysaccharide-induced leukocyte recruitment and inflammatory hyperalgesia: effect on NF-кB activation and cytokine production [Internet]. European Journal of Pharmacology. 2017 ; 809 52-63.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.ejphar.2017.05.016
Artigo de periodico
M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection (2017)
Piñeros, A. R.; Campos, L. W.; Fonseca, Denise Morais da ; Bertolini, T. B.; Gembre, A. F.; Prado, R. Q.; Alves Filho, José Carlos Farias; Ramos, Simone Gusmão; Russo, Momtchilo; Bonato, Vânia Luiza Deperon
Source: Scientific Reports. Unidades: ICB, FMRP
Subjects: IMUNOLOGIA, ALÉRGENOS, INTERLEUCINAS, MACRÓFAGOS, MYCOBACTERIUM TUBERCULOSIS, TUBERCULOSE PULMONAR
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ABNT
PIÑEROS, A. R. et al. M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection. Scientific Reports, v. 7, n. 41240, p. 1-12, 2017Tradução . . Disponível em: https://doi.org/10.1038/srep41240. Acesso em: 14 nov. 2024.
APA
Piñeros, A. R., Campos, L. W., Fonseca, D. M. da, Bertolini, T. B., Gembre, A. F., Prado, R. Q., et al. (2017). M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection. Scientific Reports, 7( 41240), 1-12. doi:10.1038/srep41240
NLM
Piñeros AR, Campos LW, Fonseca DM da, Bertolini TB, Gembre AF, Prado RQ, Alves Filho JCF, Ramos SG, Russo M, Bonato VLD. M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection [Internet]. Scientific Reports. 2017 ; 7( 41240): 1-12.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1038/srep41240
Vancouver
Piñeros AR, Campos LW, Fonseca DM da, Bertolini TB, Gembre AF, Prado RQ, Alves Filho JCF, Ramos SG, Russo M, Bonato VLD. M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection [Internet]. Scientific Reports. 2017 ; 7( 41240): 1-12.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1038/srep41240
Artigo de periodico
Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB (2016)
Franchin, Marcelo; Rosalen, Pedro Luiz; Cunha, Marcos Guilherme da; Silva, Rangel Leal; Colón, David F.; Bassi, Gabriel Shimizu; Alencar, Severino Matias de; Ikegaki, Masaharu; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Beutler, John A.; Cunha, Thiago Mattar
Source: Journal of Natural Products. Unidades: FMRP, ESALQ
Subjects: ANTI-INFLAMATÓRIOS, LIPOPOLISSACARÍDEOS, ÓXIDO NÍTRICO, MACRÓFAGOS, TRANSDUÇÃO DE SINAL CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FRANCHIN, Marcelo et al. Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB. Journal of Natural Products, v. 79, n. 7, p. 1828-1833, 2016Tradução . . Disponível em: https://doi.org/10.1021/acs.jnatprod.6b00263. Acesso em: 14 nov. 2024.
APA
Franchin, M., Rosalen, P. L., Cunha, M. G. da, Silva, R. L., Colón, D. F., Bassi, G. S., et al. (2016). Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB. Journal of Natural Products, 79( 7), 1828-1833. doi:10.1021/acs.jnatprod.6b00263
NLM
Franchin M, Rosalen PL, Cunha MG da, Silva RL, Colón DF, Bassi GS, Alencar SM de, Ikegaki M, Alves Filho JCF, Cunha F de Q, Beutler JA, Cunha TM. Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB [Internet]. Journal of Natural Products. 2016 ; 79( 7): 1828-1833.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1021/acs.jnatprod.6b00263
Vancouver
Franchin M, Rosalen PL, Cunha MG da, Silva RL, Colón DF, Bassi GS, Alencar SM de, Ikegaki M, Alves Filho JCF, Cunha F de Q, Beutler JA, Cunha TM. Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB [Internet]. Journal of Natural Products. 2016 ; 79( 7): 1828-1833.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1021/acs.jnatprod.6b00263
Artigo de periodico
Post-sepsis state induces tumor-associated macrophage accumulation through CXCR4/CXCL12 and favors tumor progression in mice (2016)
Mota, José M.; Leite, Caio A.; Souza, Lucas E.; Melo, Paulo H.; Nascimento, Daniele C.; Wagatsuma, Virginia M. de Deus; Temporal, Jessica; Figueiredo, Florêncio; Noushmehr, Houtan; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Rego, Eduardo Magalhães
Source: Cancer Immunology Research. Unidade: FMRP
Subjects: MACRÓFAGOS, SEPSE, INFECÇÕES OPORTUNISTAS, IMUNOSSUPRESSÃO, MORTALIDADE, NEOPLASIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MOTA, José M. et al. Post-sepsis state induces tumor-associated macrophage accumulation through CXCR4/CXCL12 and favors tumor progression in mice. Cancer Immunology Research, v. 4, n. 4, p. 312-322, 2016Tradução . . Disponível em: https://doi.org/10.1158/2326-6066.CIR-15-0170. Acesso em: 14 nov. 2024.
APA
Mota, J. M., Leite, C. A., Souza, L. E., Melo, P. H., Nascimento, D. C., Wagatsuma, V. M. de D., et al. (2016). Post-sepsis state induces tumor-associated macrophage accumulation through CXCR4/CXCL12 and favors tumor progression in mice. Cancer Immunology Research, 4( 4), 312-322. doi:10.1158/2326-6066.CIR-15-0170
NLM
Mota JM, Leite CA, Souza LE, Melo PH, Nascimento DC, Wagatsuma VM de D, Temporal J, Figueiredo F, Noushmehr H, Alves Filho JCF, Cunha F de Q, Rego EM. Post-sepsis state induces tumor-associated macrophage accumulation through CXCR4/CXCL12 and favors tumor progression in mice [Internet]. Cancer Immunology Research. 2016 ; 4( 4): 312-322.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1158/2326-6066.CIR-15-0170
Vancouver
Mota JM, Leite CA, Souza LE, Melo PH, Nascimento DC, Wagatsuma VM de D, Temporal J, Figueiredo F, Noushmehr H, Alves Filho JCF, Cunha F de Q, Rego EM. Post-sepsis state induces tumor-associated macrophage accumulation through CXCR4/CXCL12 and favors tumor progression in mice [Internet]. Cancer Immunology Research. 2016 ; 4( 4): 312-322.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1158/2326-6066.CIR-15-0170

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