Source: Program and Abstracts. Conference titles: The Endocrine Society's Annual Meeting. Unidade: FM
Subjects: HIPERTENSÃO (ETIOLOGIA), MUTAÇÃO GENÉTICA, BRASILEIROS, HORMÔNIOS SEXUAIS (ANORMALIDADES)
ABNT
MARTIN, Regina M. et al. A homozigous 25 Bp duplication (Nt 4157-4181) at exon 5 in the CYP17 resulting in a premature stop codon as a new cause of combined 17alpha-hydroxylase/17,20-lyase deficiency. 2005, Anais.. San Diego: Faculdade de Medicina, Universidade de São Paulo, 2005. . Acesso em: 14 set. 2024.APA
Martin, R. M., Costa, E. F., Arnhold, I. J. P., & Mendonça, B. B. (2005). A homozigous 25 Bp duplication (Nt 4157-4181) at exon 5 in the CYP17 resulting in a premature stop codon as a new cause of combined 17alpha-hydroxylase/17,20-lyase deficiency. In Program and Abstracts. San Diego: Faculdade de Medicina, Universidade de São Paulo.NLM
Martin RM, Costa EF, Arnhold IJP, Mendonça BB. A homozigous 25 Bp duplication (Nt 4157-4181) at exon 5 in the CYP17 resulting in a premature stop codon as a new cause of combined 17alpha-hydroxylase/17,20-lyase deficiency. Program and Abstracts. 2005 ;[citado 2024 set. 14 ]Vancouver
Martin RM, Costa EF, Arnhold IJP, Mendonça BB. A homozigous 25 Bp duplication (Nt 4157-4181) at exon 5 in the CYP17 resulting in a premature stop codon as a new cause of combined 17alpha-hydroxylase/17,20-lyase deficiency. Program and Abstracts. 2005 ;[citado 2024 set. 14 ]