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Artigo de periodico
Molecular degree of perturbation of plasma infammatory markers associated with tuberculosis reveals distinct disease profles between Indian and Chinese populations (2019)
Souza, Deivide Oliveira de; Vinhaes, Caian L; Arriaga, María B; Kumar, Nathella Pavan; Angulo, Juan M. Cubillos; Shi, Ruiru; Wei, Wang; Yuan, Xing; Zhang, Guolong; Cai, Ying; Barry, Clifton E., III; Via, Laura E ; Sher, Alan; Babu, Subash; Barber, Katrin D. Mayer; Nakaya, Helder Takashi Imoto ; Fukutani, Kiyoshi F; Andrade, Bruno B
Source: Scientific Reports. Unidade: FCF
Subjects: INTERLEUCINAS, TUBERCULOSE, BIOMARCADORES
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ABNT
SOUZA, Deivide Oliveira de et al. Molecular degree of perturbation of plasma infammatory markers associated with tuberculosis reveals distinct disease profles between Indian and Chinese populations. Scientific Reports, v. 9, n. 1, p. 1-10 art. 8002, 2019Tradução . . Disponível em: https://doi.org/10.1038/s41598-019-44513-8. Acesso em: 18 nov. 2024.
APA
Souza, D. O. de, Vinhaes, C. L., Arriaga, M. B., Kumar, N. P., Angulo, J. M. C., Shi, R., et al. (2019). Molecular degree of perturbation of plasma infammatory markers associated with tuberculosis reveals distinct disease profles between Indian and Chinese populations. Scientific Reports, 9( 1), 1-10 art. 8002. doi:10.1038/s41598-019-44513-8
NLM
Souza DO de, Vinhaes CL, Arriaga MB, Kumar NP, Angulo JMC, Shi R, Wei W, Yuan X, Zhang G, Cai Y, Barry CE, Via LE, Sher A, Babu S, Barber KDM, Nakaya HTI, Fukutani KF, Andrade BB. Molecular degree of perturbation of plasma infammatory markers associated with tuberculosis reveals distinct disease profles between Indian and Chinese populations [Internet]. Scientific Reports. 2019 ; 9( 1): 1-10 art. 8002.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41598-019-44513-8
Vancouver
Souza DO de, Vinhaes CL, Arriaga MB, Kumar NP, Angulo JMC, Shi R, Wei W, Yuan X, Zhang G, Cai Y, Barry CE, Via LE, Sher A, Babu S, Barber KDM, Nakaya HTI, Fukutani KF, Andrade BB. Molecular degree of perturbation of plasma infammatory markers associated with tuberculosis reveals distinct disease profles between Indian and Chinese populations [Internet]. Scientific Reports. 2019 ; 9( 1): 1-10 art. 8002.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41598-019-44513-8
Artigo de periodico
Building capacity for advances in tuberculosis research; proceedings of the third RePORT international meeting (2018)
Heijden, Yuri F. van der; Abdullah, Fareed; Andrade, Bruno B; Andrews, Jason R; Christopher, Devasahayam J; Croda, Julio; Ewing, Heather; Haas, David W; Hatherill, Mark; C. Jr, Robert Horsburgh; Mave, Vidya; Nakaya, Helder Takashi Imoto ; Rolla, Valeria; Srinivasan, Sudha; Sugiyono, Retna Indah; Gil, Cesar Ugarte; Hamilton, Carol
Source: Tuberculosis. Unidade: FCF
Subjects: TUBERCULOSE, HIV
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ABNT
HEIJDEN, Yuri F. van der et al. Building capacity for advances in tuberculosis research; proceedings of the third RePORT international meeting. Tuberculosis, v. 113, p. 153-162, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.tube.2018.09.009. Acesso em: 18 nov. 2024.
APA
Heijden, Y. F. van der, Abdullah, F., Andrade, B. B., Andrews, J. R., Christopher, D. J., Croda, J., et al. (2018). Building capacity for advances in tuberculosis research; proceedings of the third RePORT international meeting. Tuberculosis, 113, 153-162. doi:10.1016/j.tube.2018.09.009
NLM
Heijden YF van der, Abdullah F, Andrade BB, Andrews JR, Christopher DJ, Croda J, Ewing H, Haas DW, Hatherill M, C. Jr RH, Mave V, Nakaya HTI, Rolla V, Srinivasan S, Sugiyono RI, Gil CU, Hamilton C. Building capacity for advances in tuberculosis research; proceedings of the third RePORT international meeting [Internet]. Tuberculosis. 2018 ; 113 153-162.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.tube.2018.09.009
Vancouver
Heijden YF van der, Abdullah F, Andrade BB, Andrews JR, Christopher DJ, Croda J, Ewing H, Haas DW, Hatherill M, C. Jr RH, Mave V, Nakaya HTI, Rolla V, Srinivasan S, Sugiyono RI, Gil CU, Hamilton C. Building capacity for advances in tuberculosis research; proceedings of the third RePORT international meeting [Internet]. Tuberculosis. 2018 ; 113 153-162.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.tube.2018.09.009
Artigo de periodico
Immune priming and Long-term persistence of memory B cells after inactivated poliovirus vaccine in macaque models: support for at least 2 doses (2018)
Bhaumik, Siddhartha Kumar; Kulkarni, Raveendra R; Weldon, William C; Silveira, Eduardo Lani Volpe da ; Ahmed, Hasan; Gunisetty, Sivaram; Chandele, Anmol; Antia, Rustom; Verma, Harish; Sutter, Roland; Pallansch, Mark A; Oberste, M. Steven; Villinger, Francois; Orenstein, Walter; Krishna, Kaja Murali
Source: Clinical Infectious Diseases. Unidade: FCF
Subjects: POLIOMIELITE, ANTICORPOS
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ABNT
BHAUMIK, Siddhartha Kumar et al. Immune priming and Long-term persistence of memory B cells after inactivated poliovirus vaccine in macaque models: support for at least 2 doses. Clinical Infectious Diseases, v. 67, n. SUPL. 1, p. S66-S77, 2018Tradução . . Disponível em: https://doi.org/10.1093/cid/ciy634. Acesso em: 18 nov. 2024.
APA
Bhaumik, S. K., Kulkarni, R. R., Weldon, W. C., Silveira, E. L. V. da, Ahmed, H., Gunisetty, S., et al. (2018). Immune priming and Long-term persistence of memory B cells after inactivated poliovirus vaccine in macaque models: support for at least 2 doses. Clinical Infectious Diseases, 67( SUPL. 1), S66-S77. doi:10.1093/cid/ciy634
NLM
Bhaumik SK, Kulkarni RR, Weldon WC, Silveira ELV da, Ahmed H, Gunisetty S, Chandele A, Antia R, Verma H, Sutter R, Pallansch MA, Oberste MS, Villinger F, Orenstein W, Krishna KM. Immune priming and Long-term persistence of memory B cells after inactivated poliovirus vaccine in macaque models: support for at least 2 doses [Internet]. Clinical Infectious Diseases. 2018 ; 67( SUPL. 1): S66-S77.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1093/cid/ciy634
Vancouver
Bhaumik SK, Kulkarni RR, Weldon WC, Silveira ELV da, Ahmed H, Gunisetty S, Chandele A, Antia R, Verma H, Sutter R, Pallansch MA, Oberste MS, Villinger F, Orenstein W, Krishna KM. Immune priming and Long-term persistence of memory B cells after inactivated poliovirus vaccine in macaque models: support for at least 2 doses [Internet]. Clinical Infectious Diseases. 2018 ; 67( SUPL. 1): S66-S77.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1093/cid/ciy634
Artigo de periodico
Systems immunology of Diabetes-Tuberculosis comorbidity reveals signatures of disease complications (2017)
Prada-Medina, Cesar A.; Fukutani, Kiyoshi F; Kumar, Nathella Pavan; Gil-Santana, Leonardo; Babu, Subash; Lichtenstein, Flávio; West, Kim; Sivakumar, Shanmugam; Menon, Pradeep A; Viswanathan, Vijay; Andrade, Bruno B; Nakaya, Helder Takashi Imoto ; Kornfeld, Hardy
Source: Scientific Reports. Unidade: FCF
Subjects: COMORBIDADE, DIABETES MELLITUS, TUBERCULOSE
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ABNT
PRADA-MEDINA, Cesar A. et al. Systems immunology of Diabetes-Tuberculosis comorbidity reveals signatures of disease complications. Scientific Reports, v. 7, n. 1, p. 1-16, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-01767-4. Acesso em: 18 nov. 2024.
APA
Prada-Medina, C. A., Fukutani, K. F., Kumar, N. P., Gil-Santana, L., Babu, S., Lichtenstein, F., et al. (2017). Systems immunology of Diabetes-Tuberculosis comorbidity reveals signatures of disease complications. Scientific Reports, 7( 1), 1-16. doi:10.1038/s41598-017-01767-4
NLM
Prada-Medina CA, Fukutani KF, Kumar NP, Gil-Santana L, Babu S, Lichtenstein F, West K, Sivakumar S, Menon PA, Viswanathan V, Andrade BB, Nakaya HTI, Kornfeld H. Systems immunology of Diabetes-Tuberculosis comorbidity reveals signatures of disease complications [Internet]. Scientific Reports. 2017 ; 7( 1): 1-16.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41598-017-01767-4
Vancouver
Prada-Medina CA, Fukutani KF, Kumar NP, Gil-Santana L, Babu S, Lichtenstein F, West K, Sivakumar S, Menon PA, Viswanathan V, Andrade BB, Nakaya HTI, Kornfeld H. Systems immunology of Diabetes-Tuberculosis comorbidity reveals signatures of disease complications [Internet]. Scientific Reports. 2017 ; 7( 1): 1-16.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41598-017-01767-4
Artigo de periodico
The amino acid sensor GCN2 controls gut inflammation by inhibiting inflammasome activation (2016)
Ravindran, Rajesh; Loebbermann, Jens; Nakaya, Helder Takashi Imoto ; Khan, Nooruddin; Ma, Hualing; Gama, Leonardo; Machiah, Deepa K; Lawson, Benton; Hakimpour, Paul; Wang, Yi-chong; Li, Shuzhao; Sharma, Prachi; Kaufman, Randal J; Martinez, Jennifer; Pulendran, Bali
Source: Nature. Unidade: FCF
Subjects: IMUNOLOGIA, AMINOÁCIDOS
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ABNT
RAVINDRAN, Rajesh et al. The amino acid sensor GCN2 controls gut inflammation by inhibiting inflammasome activation. Nature, v. 531, n. 7595, p. 523-527, 2016Tradução . . Disponível em: https://doi.org/10.1038/nature17186. Acesso em: 18 nov. 2024.
APA
Ravindran, R., Loebbermann, J., Nakaya, H. T. I., Khan, N., Ma, H., Gama, L., et al. (2016). The amino acid sensor GCN2 controls gut inflammation by inhibiting inflammasome activation. Nature, 531( 7595), 523-527. doi:10.1038/nature17186
NLM
Ravindran R, Loebbermann J, Nakaya HTI, Khan N, Ma H, Gama L, Machiah DK, Lawson B, Hakimpour P, Wang Y-chong, Li S, Sharma P, Kaufman RJ, Martinez J, Pulendran B. The amino acid sensor GCN2 controls gut inflammation by inhibiting inflammasome activation [Internet]. Nature. 2016 ; 531( 7595): 523-527.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/nature17186
Vancouver
Ravindran R, Loebbermann J, Nakaya HTI, Khan N, Ma H, Gama L, Machiah DK, Lawson B, Hakimpour P, Wang Y-chong, Li S, Sharma P, Kaufman RJ, Martinez J, Pulendran B. The amino acid sensor GCN2 controls gut inflammation by inhibiting inflammasome activation [Internet]. Nature. 2016 ; 531( 7595): 523-527.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/nature17186

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