ReP USP - Resultado da busca

Parte de monografia/livro
Myocardial structural changes in long-term human severe sepsis/septic shock may be responsible for cardiac dysfunction (2008)
Rossi, Marcos Antonio; Celes, Mara Rúbia Nunes; Prado, C. M.; Saggioro, Fabiano Pinto
Source: Recent advances in cardiovascular disease. Unidade: FMRP
Subjects: SEPSE, CARDIOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROSSI, Marcos Antonio et al. Myocardial structural changes in long-term human severe sepsis/septic shock may be responsible for cardiac dysfunction. Recent advances in cardiovascular disease. Tradução . Bologna: Medimond, 2008. . . Acesso em: 14 nov. 2024.
APA
Rossi, M. A., Celes, M. R. N., Prado, C. M., & Saggioro, F. P. (2008). Myocardial structural changes in long-term human severe sepsis/septic shock may be responsible for cardiac dysfunction. In Recent advances in cardiovascular disease. Bologna: Medimond.
NLM
Rossi MA, Celes MRN, Prado CM, Saggioro FP. Myocardial structural changes in long-term human severe sepsis/septic shock may be responsible for cardiac dysfunction. In: Recent advances in cardiovascular disease. Bologna: Medimond; 2008. [citado 2024 nov. 14 ]
Vancouver
Rossi MA, Celes MRN, Prado CM, Saggioro FP. Myocardial structural changes in long-term human severe sepsis/septic shock may be responsible for cardiac dysfunction. In: Recent advances in cardiovascular disease. Bologna: Medimond; 2008. [citado 2024 nov. 14 ]
Parte de monografia/livro
Reduction of gap and adherens junction proteins and intercalated disc structural remodeling in the hearts of mice submitted to severe clp sepsis (2008)
Celes, Mara Rúbia Nunes; Rossi, Marcos Antonio
Source: Recent advances in cardiovascular disease. Unidade: FMRP
Subjects: SEPSE, CORAÇÃO, DOENÇAS CARDIOVASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CELES, Mara Rúbia Nunes e ROSSI, Marcos Antonio. Reduction of gap and adherens junction proteins and intercalated disc structural remodeling in the hearts of mice submitted to severe clp sepsis. Recent advances in cardiovascular disease. Tradução . Bologna: Medimond, 2008. . . Acesso em: 14 nov. 2024.
APA
Celes, M. R. N., & Rossi, M. A. (2008). Reduction of gap and adherens junction proteins and intercalated disc structural remodeling in the hearts of mice submitted to severe clp sepsis. In Recent advances in cardiovascular disease. Bologna: Medimond.
NLM
Celes MRN, Rossi MA. Reduction of gap and adherens junction proteins and intercalated disc structural remodeling in the hearts of mice submitted to severe clp sepsis. In: Recent advances in cardiovascular disease. Bologna: Medimond; 2008. [citado 2024 nov. 14 ]
Vancouver
Celes MRN, Rossi MA. Reduction of gap and adherens junction proteins and intercalated disc structural remodeling in the hearts of mice submitted to severe clp sepsis. In: Recent advances in cardiovascular disease. Bologna: Medimond; 2008. [citado 2024 nov. 14 ]
Trabalho de evento
Circumferential wall tension due to hypertension plays a pivotal role in aorta remodeling (2007)
Prado, C. M.; Rossi, Marcos Antonio
Source: Proceedings. Conference titles: International Congress on Coronary Artery Disease(ICCAD). Unidade: FMRP
Subjects: CARDIOPATIAS, HIPERTENSÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PRADO, C. M. e ROSSI, Marcos Antonio. Circumferential wall tension due to hypertension plays a pivotal role in aorta remodeling. 2007, Anais.. Venice: Medimond, 2007. . Acesso em: 14 nov. 2024.
APA
Prado, C. M., & Rossi, M. A. (2007). Circumferential wall tension due to hypertension plays a pivotal role in aorta remodeling. In Proceedings. Venice: Medimond.
NLM
Prado CM, Rossi MA. Circumferential wall tension due to hypertension plays a pivotal role in aorta remodeling. Proceedings. 2007 ;[citado 2024 nov. 14 ]
Vancouver
Prado CM, Rossi MA. Circumferential wall tension due to hypertension plays a pivotal role in aorta remodeling. Proceedings. 2007 ;[citado 2024 nov. 14 ]
Trabalho de evento
Turbulent blood flow plays an essential localizing role in the development of atherosclerotic lesions in experimentally-induced hypercholesterolemia in rats (2007)
Prado, C. M.; Ramos, Simone Gusmão; Campos, E. C.; Rossi, Marcos Antonio
Source: Proceedings. Conference titles: International Congress on Coronary Artery Disease(ICCAD). Unidade: FMRP
Subjects: ARTERIOSCLEROSE, HIPERCOLESTEROLEMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PRADO, C. M. et al. Turbulent blood flow plays an essential localizing role in the development of atherosclerotic lesions in experimentally-induced hypercholesterolemia in rats. 2007, Anais.. Venice: Medimond, 2007. . Acesso em: 14 nov. 2024.
APA
Prado, C. M., Ramos, S. G., Campos, E. C., & Rossi, M. A. (2007). Turbulent blood flow plays an essential localizing role in the development of atherosclerotic lesions in experimentally-induced hypercholesterolemia in rats. In Proceedings. Venice: Medimond.
NLM
Prado CM, Ramos SG, Campos EC, Rossi MA. Turbulent blood flow plays an essential localizing role in the development of atherosclerotic lesions in experimentally-induced hypercholesterolemia in rats. Proceedings. 2007 ;[citado 2024 nov. 14 ]
Vancouver
Prado CM, Ramos SG, Campos EC, Rossi MA. Turbulent blood flow plays an essential localizing role in the development of atherosclerotic lesions in experimentally-induced hypercholesterolemia in rats. Proceedings. 2007 ;[citado 2024 nov. 14 ]
Trabalho de evento
Hypertension, laminar flow and normal shear stress or normotension, turbulent flow and low shear stress cause distinct aorta remodeling (2005)
Prado, Cibele M.; Ramos, Simone Gusmão; Alves-Filho, José C. F.; Elias Júnior, Jorge; Cunha, Fernando de Queiroz; Rossi, Marcos Antonio
Source: Proceedings. Progress in Coronary Artery Disease. Conference titles: International Congress on Coronary Artery Disease. Unidade: FMRP
Subjects: HIPERTENSÃO, ARTERIOSCLEROSE CORONÁRIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PRADO, Cibele M. et al. Hypertension, laminar flow and normal shear stress or normotension, turbulent flow and low shear stress cause distinct aorta remodeling. 2005, Anais.. Bologna: Medimond, 2005. . Acesso em: 14 nov. 2024.
APA
Prado, C. M., Ramos, S. G., Alves-Filho, J. C. F., Elias Júnior, J., Cunha, F. de Q., & Rossi, M. A. (2005). Hypertension, laminar flow and normal shear stress or normotension, turbulent flow and low shear stress cause distinct aorta remodeling. In Proceedings. Progress in Coronary Artery Disease. Bologna: Medimond.
NLM
Prado CM, Ramos SG, Alves-Filho JCF, Elias Júnior J, Cunha F de Q, Rossi MA. Hypertension, laminar flow and normal shear stress or normotension, turbulent flow and low shear stress cause distinct aorta remodeling. Proceedings. Progress in Coronary Artery Disease. 2005 ;[citado 2024 nov. 14 ]
Vancouver
Prado CM, Ramos SG, Alves-Filho JCF, Elias Júnior J, Cunha F de Q, Rossi MA. Hypertension, laminar flow and normal shear stress or normotension, turbulent flow and low shear stress cause distinct aorta remodeling. Proceedings. Progress in Coronary Artery Disease. 2005 ;[citado 2024 nov. 14 ]

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