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Filtros : "Nunes, Maria Tereza" "Universidad Nacional de Córdoba (UNC)" Removido: "IONS" Limpar

Filtros

Artigo de periodico
Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway (2016)
Serrano-Nascimento, Caroline; Nicola, Juan Pablo; Teixeira, Silvania da Silva; Poyares, Leonice Lourenço; Lellis-Santos, Camilo; Bordin, Silvana ; Masini-Repiso, Ana Maria; Nunes, Maria Tereza
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SERRANO-NASCIMENTO, Caroline et al. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway. Molecular and Cellular Endocrinology, v. 426, p. 73-90, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.02.006. Acesso em: 03 nov. 2024.
APA
Serrano-Nascimento, C., Nicola, J. P., Teixeira, S. da S., Poyares, L. L., Lellis-Santos, C., Bordin, S., et al. (2016). Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway. Molecular and Cellular Endocrinology, 426, 73-90. doi:10.1016/j.mce.2016.02.006
NLM
Serrano-Nascimento C, Nicola JP, Teixeira S da S, Poyares LL, Lellis-Santos C, Bordin S, Masini-Repiso AM, Nunes MT. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway [Internet]. Molecular and Cellular Endocrinology. 2016 ; 426 73-90.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.mce.2016.02.006
Vancouver
Serrano-Nascimento C, Nicola JP, Teixeira S da S, Poyares LL, Lellis-Santos C, Bordin S, Masini-Repiso AM, Nunes MT. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway [Internet]. Molecular and Cellular Endocrinology. 2016 ; 426 73-90.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.mce.2016.02.006
Artigo de periodico
Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism (2011)
Nicola, Juan Pablo; Nazar, Magalí; Serrano-Nascimento, Caroline; Silva, Francemilson Goulart da ; Sobrero, Gabriela; Testa, Graciela; Nunes, Maria Tereza ; Muñoz, Liliana; Miras, Mirta; Masini-Repiso, Ana María
Source: The Journal of Clinical Endocrinology and Metabolism. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NICOLA, Juan Pablo et al. Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism. The Journal of Clinical Endocrinology and Metabolism, v. 96, n. 7, p. E1100-E1107, 2011Tradução . . Disponível em: https://doi.org/10.1210/jc.2011-0349. Acesso em: 03 nov. 2024.
APA
Nicola, J. P., Nazar, M., Serrano-Nascimento, C., Silva, F. G. da, Sobrero, G., Testa, G., et al. (2011). Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism. The Journal of Clinical Endocrinology and Metabolism, 96( 7), E1100-E1107. doi:10.1210/jc.2011-0349
NLM
Nicola JP, Nazar M, Serrano-Nascimento C, Silva FG da, Sobrero G, Testa G, Nunes MT, Muñoz L, Miras M, Masini-Repiso AM. Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism [Internet]. The Journal of Clinical Endocrinology and Metabolism. 2011 ; 96( 7): E1100-E1107.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1210/jc.2011-0349
Vancouver
Nicola JP, Nazar M, Serrano-Nascimento C, Silva FG da, Sobrero G, Testa G, Nunes MT, Muñoz L, Miras M, Masini-Repiso AM. Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism [Internet]. The Journal of Clinical Endocrinology and Metabolism. 2011 ; 96( 7): E1100-E1107.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1210/jc.2011-0349

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