Filtros : "Nunes, Maria Tereza" "Universidad Nacional de Córdoba (UNC)" Limpar

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  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      SERRANO-NASCIMENTO, Caroline et al. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway. Molecular and Cellular Endocrinology, v. 426, p. 73-90, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.02.006. Acesso em: 21 jul. 2024.
    • APA

      Serrano-Nascimento, C., Nicola, J. P., Teixeira, S. da S., Poyares, L. L., Lellis-Santos, C., Bordin, S., et al. (2016). Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway. Molecular and Cellular Endocrinology, 426, 73-90. doi:10.1016/j.mce.2016.02.006
    • NLM

      Serrano-Nascimento C, Nicola JP, Teixeira S da S, Poyares LL, Lellis-Santos C, Bordin S, Masini-Repiso AM, Nunes MT. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway [Internet]. Molecular and Cellular Endocrinology. 2016 ; 426 73-90.[citado 2024 jul. 21 ] Available from: https://doi.org/10.1016/j.mce.2016.02.006
    • Vancouver

      Serrano-Nascimento C, Nicola JP, Teixeira S da S, Poyares LL, Lellis-Santos C, Bordin S, Masini-Repiso AM, Nunes MT. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway [Internet]. Molecular and Cellular Endocrinology. 2016 ; 426 73-90.[citado 2024 jul. 21 ] Available from: https://doi.org/10.1016/j.mce.2016.02.006
  • Source: Endocrinology. Unidade: ICB

    Subjects: FISIOLOGIA, SODIO, IONS, HORMÔNIOS TIREOIDIANOS, IODO

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      SERRANO-NASCIMENTO, Caroline et al. The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway. Endocrinology, v. 155, n. 3, p. 1145-1156, 2014Tradução . . Disponível em: https://doi.org/10.1210/en.2013-1665. Acesso em: 21 jul. 2024.
    • APA

      Serrano-Nascimento, C., Teixeira, S. da S., Nachbar, R. T., Nicola, J. P., Masini-Repiso, A. M., & Nunes, M. T. (2014). The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway. Endocrinology, 155( 3), 1145-1156. doi:10.1210/en.2013-1665
    • NLM

      Serrano-Nascimento C, Teixeira S da S, Nachbar RT, Nicola JP, Masini-Repiso AM, Nunes MT. The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway [Internet]. Endocrinology. 2014 ; 155( 3): 1145-1156.[citado 2024 jul. 21 ] Available from: https://doi.org/10.1210/en.2013-1665
    • Vancouver

      Serrano-Nascimento C, Teixeira S da S, Nachbar RT, Nicola JP, Masini-Repiso AM, Nunes MT. The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway [Internet]. Endocrinology. 2014 ; 155( 3): 1145-1156.[citado 2024 jul. 21 ] Available from: https://doi.org/10.1210/en.2013-1665
  • Source: The Journal of Clinical Endocrinology and Metabolism. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      NICOLA, Juan Pablo et al. Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism. The Journal of Clinical Endocrinology and Metabolism, v. 96, n. 7, p. E1100-E1107, 2011Tradução . . Disponível em: https://doi.org/10.1210/jc.2011-0349. Acesso em: 21 jul. 2024.
    • APA

      Nicola, J. P., Nazar, M., Serrano-Nascimento, C., Silva, F. G. da, Sobrero, G., Testa, G., et al. (2011). Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism. The Journal of Clinical Endocrinology and Metabolism, 96( 7), E1100-E1107. doi:10.1210/jc.2011-0349
    • NLM

      Nicola JP, Nazar M, Serrano-Nascimento C, Silva FG da, Sobrero G, Testa G, Nunes MT, Muñoz L, Miras M, Masini-Repiso AM. Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism [Internet]. The Journal of Clinical Endocrinology and Metabolism. 2011 ; 96( 7): E1100-E1107.[citado 2024 jul. 21 ] Available from: https://doi.org/10.1210/jc.2011-0349
    • Vancouver

      Nicola JP, Nazar M, Serrano-Nascimento C, Silva FG da, Sobrero G, Testa G, Nunes MT, Muñoz L, Miras M, Masini-Repiso AM. Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism [Internet]. The Journal of Clinical Endocrinology and Metabolism. 2011 ; 96( 7): E1100-E1107.[citado 2024 jul. 21 ] Available from: https://doi.org/10.1210/jc.2011-0349

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