Filtros : "Nunes, Maria Tereza" "Indexado na Base de Dados Excerpta Medica" Removido: "Indexado na Base de Dados SCOPUS" Limpar

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  • Source: American Journal of Physiology. Cell Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      CALIL-SILVEIRA, Jamile et al. Iodide excess regulates its own efflux: a possible involvement of pendrin. American Journal of Physiology. Cell Physiology, v. 310, n. 7, p. C576-C582, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajpcell.00210.2015. Acesso em: 01 out. 2024.
    • APA

      Calil-Silveira, J., Serrano-Nascimento, C., Kopp, P. A., & Nunes, M. T. (2016). Iodide excess regulates its own efflux: a possible involvement of pendrin. American Journal of Physiology. Cell Physiology, 310( 7), C576-C582. doi:10.1152/ajpcell.00210.2015
    • NLM

      Calil-Silveira J, Serrano-Nascimento C, Kopp PA, Nunes MT. Iodide excess regulates its own efflux: a possible involvement of pendrin [Internet]. American Journal of Physiology. Cell Physiology. 2016 ; 310( 7): C576-C582.[citado 2024 out. 01 ] Available from: https://doi.org/10.1152/ajpcell.00210.2015
    • Vancouver

      Calil-Silveira J, Serrano-Nascimento C, Kopp PA, Nunes MT. Iodide excess regulates its own efflux: a possible involvement of pendrin [Internet]. American Journal of Physiology. Cell Physiology. 2016 ; 310( 7): C576-C582.[citado 2024 out. 01 ] Available from: https://doi.org/10.1152/ajpcell.00210.2015
  • Source: Cell Calcium. Unidade: ICB

    Subjects: FISIOLOGIA, HORMÔNIO TIREOTROFICO, CAMUNDONGOS, CALCIO

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      TOMIC, Melanija et al. Calcium signaling properties of a thyrotroph cell line, mouse TαT1 cells. Cell Calcium, v. 58, p. 598-605, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.ceca.2015.09.003. Acesso em: 01 out. 2024.
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      Tomic, M., Bargi-Souza, P., Leiva-Salcedo, E., Nunes, M. T., & Stojilkovic, S. S. (2015). Calcium signaling properties of a thyrotroph cell line, mouse TαT1 cells. Cell Calcium, 58, 598-605. doi:10.1016/j.ceca.2015.09.003
    • NLM

      Tomic M, Bargi-Souza P, Leiva-Salcedo E, Nunes MT, Stojilkovic SS. Calcium signaling properties of a thyrotroph cell line, mouse TαT1 cells [Internet]. Cell Calcium. 2015 ; 58 598-605.[citado 2024 out. 01 ] Available from: https://doi.org/10.1016/j.ceca.2015.09.003
    • Vancouver

      Tomic M, Bargi-Souza P, Leiva-Salcedo E, Nunes MT, Stojilkovic SS. Calcium signaling properties of a thyrotroph cell line, mouse TαT1 cells [Internet]. Cell Calcium. 2015 ; 58 598-605.[citado 2024 out. 01 ] Available from: https://doi.org/10.1016/j.ceca.2015.09.003
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Subjects: FISIOLOGIA, GLICOPROTEINAS, GLÂNDULA PITUITÁRIA, RATOS, SECREÇÃO ANIMAL

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      SOUZA, Paula Bargi de et al. T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion. Molecular and Cellular Endocrinology, v. 409, p. 73-81, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2015.04.002. Acesso em: 01 out. 2024.
    • APA

      Souza, P. B. de, Romano, R. M., Silva, F. G. da, Brunetto, E. L., & Nunes, M. T. (2015). T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion. Molecular and Cellular Endocrinology, 409, 73-81. doi:10.1016/j.mce.2015.04.002
    • NLM

      Souza PB de, Romano RM, Silva FG da, Brunetto EL, Nunes MT. T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion [Internet]. Molecular and Cellular Endocrinology. 2015 ; 409 73-81.[citado 2024 out. 01 ] Available from: https://doi.org/10.1016/j.mce.2015.04.002
    • Vancouver

      Souza PB de, Romano RM, Silva FG da, Brunetto EL, Nunes MT. T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion [Internet]. Molecular and Cellular Endocrinology. 2015 ; 409 73-81.[citado 2024 out. 01 ] Available from: https://doi.org/10.1016/j.mce.2015.04.002
  • Source: Thyroid. Unidades: FMVZ, ICB

    Assunto: FISIOLOGIA

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      ROMANO, Renata Marino et al. Hypothyroidism in adult male rats alters posttranscriptional mechanisms of luteinizing hormone biosynthesis. Thyroid, v. 23, n. 4, p. 497-505, 2013Tradução . . Disponível em: https://doi.org/10.1089/thy.2011.0514. Acesso em: 01 out. 2024.
    • APA

      Romano, R. M., Bargi-Souza, P., Brunetto, E. L., Silva, F. G. da, Avellar, M. C. W., Oliveira, C. A., & Nunes, M. T. (2013). Hypothyroidism in adult male rats alters posttranscriptional mechanisms of luteinizing hormone biosynthesis. Thyroid, 23( 4), 497-505. doi:10.1089/thy.2011.0514
    • NLM

      Romano RM, Bargi-Souza P, Brunetto EL, Silva FG da, Avellar MCW, Oliveira CA, Nunes MT. Hypothyroidism in adult male rats alters posttranscriptional mechanisms of luteinizing hormone biosynthesis [Internet]. Thyroid. 2013 ; 23( 4): 497-505.[citado 2024 out. 01 ] Available from: https://doi.org/10.1089/thy.2011.0514
    • Vancouver

      Romano RM, Bargi-Souza P, Brunetto EL, Silva FG da, Avellar MCW, Oliveira CA, Nunes MT. Hypothyroidism in adult male rats alters posttranscriptional mechanisms of luteinizing hormone biosynthesis [Internet]. Thyroid. 2013 ; 23( 4): 497-505.[citado 2024 out. 01 ] Available from: https://doi.org/10.1089/thy.2011.0514
  • Source: Experimental and Clinical Endocrinology Diabetes. Unidade: ICB

    Assunto: FISIOLOGIA

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      SILVA, Francemilson Goulart da et al. Triiodothyronine (T3) induces proinsulin gene expression by activating PI3K: possible roles for GSK-3β and the transcriptional factor PDX-1. Experimental and Clinical Endocrinology Diabetes, v. 121, n. 1, p. 14-19, 2013Tradução . . Acesso em: 01 out. 2024.
    • APA

      Silva, F. G. da, Serrano-Nascimento, C., Teixeira, S. S., & Nunes, M. T. (2013). Triiodothyronine (T3) induces proinsulin gene expression by activating PI3K: possible roles for GSK-3β and the transcriptional factor PDX-1. Experimental and Clinical Endocrinology Diabetes, 121( 1), 14-19.
    • NLM

      Silva FG da, Serrano-Nascimento C, Teixeira SS, Nunes MT. Triiodothyronine (T3) induces proinsulin gene expression by activating PI3K: possible roles for GSK-3β and the transcriptional factor PDX-1. Experimental and Clinical Endocrinology Diabetes. 2013 ; 121( 1): 14-19.[citado 2024 out. 01 ]
    • Vancouver

      Silva FG da, Serrano-Nascimento C, Teixeira SS, Nunes MT. Triiodothyronine (T3) induces proinsulin gene expression by activating PI3K: possible roles for GSK-3β and the transcriptional factor PDX-1. Experimental and Clinical Endocrinology Diabetes. 2013 ; 121( 1): 14-19.[citado 2024 out. 01 ]
  • Source: European Journal of Applied Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      PINHEIRO, Carlos Hermano da Justa et al. Metabolic and functional effects of beta-hydroxy-betamethylbutyrate (HMB) supplementation in skeletal muscle. European Journal of Applied Physiology, v. 112, n. 7, p. 2531-2537, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00421-011-2224-5. Acesso em: 01 out. 2024.
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      Pinheiro, C. H. da J., Gerlinger-Romero, F., Guimarães-Ferreira, L., Souza-Jr, A. L. de, Vitzel, K. F., Nachbar, R. T., et al. (2012). Metabolic and functional effects of beta-hydroxy-betamethylbutyrate (HMB) supplementation in skeletal muscle. European Journal of Applied Physiology, 112( 7), 2531-2537. doi:10.1007/s00421-011-2224-5
    • NLM

      Pinheiro CH da J, Gerlinger-Romero F, Guimarães-Ferreira L, Souza-Jr AL de, Vitzel KF, Nachbar RT, Nunes MT, Curi R. Metabolic and functional effects of beta-hydroxy-betamethylbutyrate (HMB) supplementation in skeletal muscle [Internet]. European Journal of Applied Physiology. 2012 ; 112( 7): 2531-2537.[citado 2024 out. 01 ] Available from: https://doi.org/10.1007/s00421-011-2224-5
    • Vancouver

      Pinheiro CH da J, Gerlinger-Romero F, Guimarães-Ferreira L, Souza-Jr AL de, Vitzel KF, Nachbar RT, Nunes MT, Curi R. Metabolic and functional effects of beta-hydroxy-betamethylbutyrate (HMB) supplementation in skeletal muscle [Internet]. European Journal of Applied Physiology. 2012 ; 112( 7): 2531-2537.[citado 2024 out. 01 ] Available from: https://doi.org/10.1007/s00421-011-2224-5
  • Source: European Journal of Applied Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      GUIMARÃES-FERREIRA, Lucas et al. Short-term creatine supplementation decreases reactive oxygen species content with no changes in expression and activity of antioxidant enzymes in skeletal muscle. European Journal of Applied Physiology, v. 112, n. 11, p. 3905-3911, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00421-012-2378-9. Acesso em: 01 out. 2024.
    • APA

      Guimarães-Ferreira, L., Pinheiro, C. H. da J., Gerlinger-Romero, F., Vitzel, K. F., Nachbar, R. T., Nunes, M. T., & Curi, R. (2012). Short-term creatine supplementation decreases reactive oxygen species content with no changes in expression and activity of antioxidant enzymes in skeletal muscle. European Journal of Applied Physiology, 112( 11), 3905-3911. doi:10.1007/s00421-012-2378-9
    • NLM

      Guimarães-Ferreira L, Pinheiro CH da J, Gerlinger-Romero F, Vitzel KF, Nachbar RT, Nunes MT, Curi R. Short-term creatine supplementation decreases reactive oxygen species content with no changes in expression and activity of antioxidant enzymes in skeletal muscle [Internet]. European Journal of Applied Physiology. 2012 ; 112( 11): 3905-3911.[citado 2024 out. 01 ] Available from: https://doi.org/10.1007/s00421-012-2378-9
    • Vancouver

      Guimarães-Ferreira L, Pinheiro CH da J, Gerlinger-Romero F, Vitzel KF, Nachbar RT, Nunes MT, Curi R. Short-term creatine supplementation decreases reactive oxygen species content with no changes in expression and activity of antioxidant enzymes in skeletal muscle [Internet]. European Journal of Applied Physiology. 2012 ; 112( 11): 3905-3911.[citado 2024 out. 01 ] Available from: https://doi.org/10.1007/s00421-012-2378-9
  • Source: American Journal of Physiology Cell Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      SERRANO-NASCIMENTO, Caroline e SILVEIRA, Jamile Calil e NUNES, Maria Tereza. Posttranscriptional regulation of sodium-iodide symporter mRNA expression in the rat thyroid gland by acute iodide administration. American Journal of Physiology Cell Physiology, v. 298, p. C893-C899, 2010Tradução . . Disponível em: https://doi.org/10.1152/ajpcell.00224.2009. Acesso em: 01 out. 2024.
    • APA

      Serrano-Nascimento, C., Silveira, J. C., & Nunes, M. T. (2010). Posttranscriptional regulation of sodium-iodide symporter mRNA expression in the rat thyroid gland by acute iodide administration. American Journal of Physiology Cell Physiology, 298, C893-C899. doi:10.1152/ajpcell.00224.2009
    • NLM

      Serrano-Nascimento C, Silveira JC, Nunes MT. Posttranscriptional regulation of sodium-iodide symporter mRNA expression in the rat thyroid gland by acute iodide administration [Internet]. American Journal of Physiology Cell Physiology. 2010 ; 298 C893-C899.[citado 2024 out. 01 ] Available from: https://doi.org/10.1152/ajpcell.00224.2009
    • Vancouver

      Serrano-Nascimento C, Silveira JC, Nunes MT. Posttranscriptional regulation of sodium-iodide symporter mRNA expression in the rat thyroid gland by acute iodide administration [Internet]. American Journal of Physiology Cell Physiology. 2010 ; 298 C893-C899.[citado 2024 out. 01 ] Available from: https://doi.org/10.1152/ajpcell.00224.2009

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