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Artigo de periodico
Purinergic signaling in cognitive impairment and neuropsychiatric symptoms of Alzheimer’s disease (2023)
Ribeiro, Deidiane Elisa ; Petiz, Lyvia Lintzmaier ; Glaser, Talita ; Giacomelli, Ágatha Oliveira ; Andrejew, Roberta ; Saab, Fernando de Azevedo Ribeiro; Milanis, Milena da Silva ; Campos, Henrique Correia; Sampaio, Vanessa Fernandes Arnaud ; La Banca, Sophia; Longo, Beatriz Monteiro ; Lameu, Claudiana ; Tang, Yong; Resende, Rodrigo Ribeiro ; Ferreira, Sergio T ; Ulrich, Henning
Source: Neuropharmacology. Unidade: IQ
Subjects: NEUROPSIQUIATRIA, DOENÇA DE ALZHEIMER, DEPRESSÃO, TRANSTORNOS DO SONO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RIBEIRO, Deidiane Elisa et al. Purinergic signaling in cognitive impairment and neuropsychiatric symptoms of Alzheimer’s disease. Neuropharmacology, v. 226, p. 1-16 art. 109371, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2022.109371. Acesso em: 03 nov. 2024.
APA
Ribeiro, D. E., Petiz, L. L., Glaser, T., Giacomelli, Á. O., Andrejew, R., Saab, F. de A. R., et al. (2023). Purinergic signaling in cognitive impairment and neuropsychiatric symptoms of Alzheimer’s disease. Neuropharmacology, 226, 1-16 art. 109371. doi:10.1016/j.neuropharm.2022.109371
NLM
Ribeiro DE, Petiz LL, Glaser T, Giacomelli ÁO, Andrejew R, Saab F de AR, Milanis M da S, Campos HC, Sampaio VFA, La Banca S, Longo BM, Lameu C, Tang Y, Resende RR, Ferreira ST, Ulrich H. Purinergic signaling in cognitive impairment and neuropsychiatric symptoms of Alzheimer’s disease [Internet]. Neuropharmacology. 2023 ; 226 1-16 art. 109371.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.neuropharm.2022.109371
Vancouver
Ribeiro DE, Petiz LL, Glaser T, Giacomelli ÁO, Andrejew R, Saab F de AR, Milanis M da S, Campos HC, Sampaio VFA, La Banca S, Longo BM, Lameu C, Tang Y, Resende RR, Ferreira ST, Ulrich H. Purinergic signaling in cognitive impairment and neuropsychiatric symptoms of Alzheimer’s disease [Internet]. Neuropharmacology. 2023 ; 226 1-16 art. 109371.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.neuropharm.2022.109371
Artigo de periodico
ATP and spontaneous calcium oscillations control neural stem cell fate determination in Huntington’s disease: a novel approach for cell clock research (2021)
Glaser, Talita ; Shimojo, Hiromi; Ribeiro, Deidiane Elisa ; Martins, Patrícia Pereira Lopes; Beco, Renata Pereira; Kosinski, Michal; Sampaio, Vanessa Fernandes Arnaud ; Velloso, Juliana Correa ; Giacomelli, Ágatha Oliveira ; Lameu, Claudiana ; Santos, Ana Paula de Jesus; Souza, Hellio Danny Nobrega de ; Teng, Yang D; Kageyama, Ryoichiro; Ulrich, Henning
Source: Molecular Psychiatry. Unidade: IQ
Subjects: EMBRIOGÊNESE, CÉLULAS-TRONCO, NEUROCIÊNCIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GLASER, Talita et al. ATP and spontaneous calcium oscillations control neural stem cell fate determination in Huntington’s disease: a novel approach for cell clock research. Molecular Psychiatry, v. 26, p. 2633–2650, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41380-020-0717-5. Acesso em: 03 nov. 2024.
APA
Glaser, T., Shimojo, H., Ribeiro, D. E., Martins, P. P. L., Beco, R. P., Kosinski, M., et al. (2021). ATP and spontaneous calcium oscillations control neural stem cell fate determination in Huntington’s disease: a novel approach for cell clock research. Molecular Psychiatry, 26, 2633–2650. doi:10.1038/s41380-020-0717-5
NLM
Glaser T, Shimojo H, Ribeiro DE, Martins PPL, Beco RP, Kosinski M, Sampaio VFA, Velloso JC, Giacomelli ÁO, Lameu C, Santos AP de J, Souza HDN de, Teng YD, Kageyama R, Ulrich H. ATP and spontaneous calcium oscillations control neural stem cell fate determination in Huntington’s disease: a novel approach for cell clock research [Internet]. Molecular Psychiatry. 2021 ; 26 2633–2650.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1038/s41380-020-0717-5
Vancouver
Glaser T, Shimojo H, Ribeiro DE, Martins PPL, Beco RP, Kosinski M, Sampaio VFA, Velloso JC, Giacomelli ÁO, Lameu C, Santos AP de J, Souza HDN de, Teng YD, Kageyama R, Ulrich H. ATP and spontaneous calcium oscillations control neural stem cell fate determination in Huntington’s disease: a novel approach for cell clock research [Internet]. Molecular Psychiatry. 2021 ; 26 2633–2650.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1038/s41380-020-0717-5
Artigo de periodico
Hyperactivation of P2X7 receptors as a culprit of COVID-19 neuropathology (2021)
Ribeiro, Deidiane Elisa ; Giacomelli, Ágatha Oliveira ; Glaser, Talita ; Sampaio, Vanessa Fernandes Arnaud ; Andrejew, Roberta ; Dieckmann, Luiz; Baranova, Juliana; Lameu, Claudiana ; Ratajczak, Mariusz Z; Ulrich, Henning
Source: Molecular Psychiatry. Unidade: IQ
Subjects: COVID-19, RECEPTORES, SURTOS DE DOENÇAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RIBEIRO, Deidiane Elisa et al. Hyperactivation of P2X7 receptors as a culprit of COVID-19 neuropathology. Molecular Psychiatry, v. 26, p. 1044–1059, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41380-020-00965-3. Acesso em: 03 nov. 2024.
APA
Ribeiro, D. E., Giacomelli, Á. O., Glaser, T., Sampaio, V. F. A., Andrejew, R., Dieckmann, L., et al. (2021). Hyperactivation of P2X7 receptors as a culprit of COVID-19 neuropathology. Molecular Psychiatry, 26, 1044–1059. doi:10.1038/s41380-020-00965-3
NLM
Ribeiro DE, Giacomelli ÁO, Glaser T, Sampaio VFA, Andrejew R, Dieckmann L, Baranova J, Lameu C, Ratajczak MZ, Ulrich H. Hyperactivation of P2X7 receptors as a culprit of COVID-19 neuropathology [Internet]. Molecular Psychiatry. 2021 ; 26 1044–1059.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1038/s41380-020-00965-3
Vancouver
Ribeiro DE, Giacomelli ÁO, Glaser T, Sampaio VFA, Andrejew R, Dieckmann L, Baranova J, Lameu C, Ratajczak MZ, Ulrich H. Hyperactivation of P2X7 receptors as a culprit of COVID-19 neuropathology [Internet]. Molecular Psychiatry. 2021 ; 26 1044–1059.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1038/s41380-020-00965-3
Artigo de periodico
The P2X7 receptor: central hub of brain diseases (2020)
Andrejew, Roberta ; Giacomelli, Ágatha Oliveira ; Ribeiro, Deidiane Elisa ; Glaser, Talita ; Sampaio, Vanessa Fernandes Arnaud ; Lameu, Claudiana ; Ulrich, Henning
Source: Frontiers in Molecular Neuroscience. Unidade: IQ
Subjects: DOENÇAS CEREBRAIS, RECEPTORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANDREJEW, Roberta et al. The P2X7 receptor: central hub of brain diseases. Frontiers in Molecular Neuroscience, v. 13, p. 1-27 art. 124, 2020Tradução . . Disponível em: https://doi.org/10.3389/fnmol.2020.00124. Acesso em: 03 nov. 2024.
APA
Andrejew, R., Giacomelli, Á. O., Ribeiro, D. E., Glaser, T., Sampaio, V. F. A., Lameu, C., & Ulrich, H. (2020). The P2X7 receptor: central hub of brain diseases. Frontiers in Molecular Neuroscience, 13, 1-27 art. 124. doi:10.3389/fnmol.2020.00124
NLM
Andrejew R, Giacomelli ÁO, Ribeiro DE, Glaser T, Sampaio VFA, Lameu C, Ulrich H. The P2X7 receptor: central hub of brain diseases [Internet]. Frontiers in Molecular Neuroscience. 2020 ; 13 1-27 art. 124.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fnmol.2020.00124
Vancouver
Andrejew R, Giacomelli ÁO, Ribeiro DE, Glaser T, Sampaio VFA, Lameu C, Ulrich H. The P2X7 receptor: central hub of brain diseases [Internet]. Frontiers in Molecular Neuroscience. 2020 ; 13 1-27 art. 124.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fnmol.2020.00124

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