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Artigo de periodico
LPS-induced low-grade inflammation increases hypothalamic JNK expression and causes central insulin resistance irrespective of body weight changes (2017)
Rorato, Rodrigo; Borges, Beatriz de Carvalho; Uchoa, Ernane Torres; Antunes-Rodrigues, José; Elias, Carol Fuzeti; Elias, Lucila Leico Kagohara
Source: International Journal of Molecular Sciences. Unidade: FMRP
Subjects: MODELOS ANIMAIS DE DOENÇAS, HIPOTÁLAMO, INFLAMAÇÃO, RESISTÊNCIA À INSULINA, LIPOPOLISSACARÍDEOS, PESOS E MEDIDAS CORPORAIS
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ABNT
RORATO, Rodrigo et al. LPS-induced low-grade inflammation increases hypothalamic JNK expression and causes central insulin resistance irrespective of body weight changes. International Journal of Molecular Sciences, v. 18, n. 7, 2017Tradução . . Disponível em: https://doi.org/10.3390/ijms18071431. Acesso em: 13 nov. 2024.
APA
Rorato, R., Borges, B. de C., Uchoa, E. T., Antunes-Rodrigues, J., Elias, C. F., & Elias, L. L. K. (2017). LPS-induced low-grade inflammation increases hypothalamic JNK expression and causes central insulin resistance irrespective of body weight changes. International Journal of Molecular Sciences, 18( 7). doi:10.3390/ijms18071431
NLM
Rorato R, Borges B de C, Uchoa ET, Antunes-Rodrigues J, Elias CF, Elias LLK. LPS-induced low-grade inflammation increases hypothalamic JNK expression and causes central insulin resistance irrespective of body weight changes [Internet]. International Journal of Molecular Sciences. 2017 ; 18( 7):[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms18071431
Vancouver
Rorato R, Borges B de C, Uchoa ET, Antunes-Rodrigues J, Elias CF, Elias LLK. LPS-induced low-grade inflammation increases hypothalamic JNK expression and causes central insulin resistance irrespective of body weight changes [Internet]. International Journal of Molecular Sciences. 2017 ; 18( 7):[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms18071431
Artigo de periodico
PI3K p110β subunit in leptin receptor expressing cells is required for the acute hypophagia induced by endotoxemia (2016)
Borges, Beatriz de Carvalho; Garcia-Galiano, David; Rorato, Rodrigo; Elias, Lucila Leico Kagohara; Elias, Carol Fuzeti
Source: Molecular Metabolism. Unidade: FMRP
Subjects: HIPOTÁLAMO, INFLAMAÇÃO, LEPTINA, METABOLISMO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORGES, Beatriz de Carvalho et al. PI3K p110β subunit in leptin receptor expressing cells is required for the acute hypophagia induced by endotoxemia. Molecular Metabolism, v. 5, n. 6, p. 379-391, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2016.03.003. Acesso em: 13 nov. 2024.
APA
Borges, B. de C., Garcia-Galiano, D., Rorato, R., Elias, L. L. K., & Elias, C. F. (2016). PI3K p110β subunit in leptin receptor expressing cells is required for the acute hypophagia induced by endotoxemia. Molecular Metabolism, 5( 6), 379-391. doi:10.1016/j.molmet.2016.03.003
NLM
Borges B de C, Garcia-Galiano D, Rorato R, Elias LLK, Elias CF. PI3K p110β subunit in leptin receptor expressing cells is required for the acute hypophagia induced by endotoxemia [Internet]. Molecular Metabolism. 2016 ; 5( 6): 379-391.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.molmet.2016.03.003
Vancouver
Borges B de C, Garcia-Galiano D, Rorato R, Elias LLK, Elias CF. PI3K p110β subunit in leptin receptor expressing cells is required for the acute hypophagia induced by endotoxemia [Internet]. Molecular Metabolism. 2016 ; 5( 6): 379-391.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.molmet.2016.03.003
Artigo de periodico
PI3K signaling: a molecular pathway associated with acute hypophagic response during inflammatory challenges (2016)
Borges, Beatriz de Carvalho; Elias, Carol Fuzeti; Elias, Lucila Leico Kagohara
Source: Molecular and Cellular Endocrinology. Unidade: FMRP
Subjects: HIPOTÁLAMO, INFLAMAÇÃO, METABOLISMO ENERGÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORGES, Beatriz de Carvalho e ELIAS, Carol Fuzeti e ELIAS, Lucila Leico Kagohara. PI3K signaling: a molecular pathway associated with acute hypophagic response during inflammatory challenges. Molecular and Cellular Endocrinology, v. 438, p. 36-41, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.07.005. Acesso em: 13 nov. 2024.
APA
Borges, B. de C., Elias, C. F., & Elias, L. L. K. (2016). PI3K signaling: a molecular pathway associated with acute hypophagic response during inflammatory challenges. Molecular and Cellular Endocrinology, 438, 36-41. doi:10.1016/j.mce.2016.07.005
NLM
Borges B de C, Elias CF, Elias LLK. PI3K signaling: a molecular pathway associated with acute hypophagic response during inflammatory challenges [Internet]. Molecular and Cellular Endocrinology. 2016 ; 438 36-41.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.mce.2016.07.005
Vancouver
Borges B de C, Elias CF, Elias LLK. PI3K signaling: a molecular pathway associated with acute hypophagic response during inflammatory challenges [Internet]. Molecular and Cellular Endocrinology. 2016 ; 438 36-41.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.mce.2016.07.005
Artigo de periodico
Protein tyrosine phosphatase-1B contributes to LPS-induced leptin resistance in male rats (2015)
Borges, Beatriz de Carvalho; Rorato, Rodrigo César; Uchôa, Ernane Torres; Marangon, Paula Beatriz; Elias, Carol F.; Antunes-Rodrigues, José; Elias, Lucila Leico Kagohara
Source: American Journal of Physiology: Endocrinology and Metabolism. Unidade: FMRP
Subjects: LEPTINA, HIPOTÁLAMO, OBESIDADE, METABOLISMO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORGES, Beatriz de Carvalho et al. Protein tyrosine phosphatase-1B contributes to LPS-induced leptin resistance in male rats. American Journal of Physiology: Endocrinology and Metabolism, v. 308, n. 1, p. E40-E50, 2015Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00094.2014. Acesso em: 13 nov. 2024.
APA
Borges, B. de C., Rorato, R. C., Uchôa, E. T., Marangon, P. B., Elias, C. F., Antunes-Rodrigues, J., & Elias, L. L. K. (2015). Protein tyrosine phosphatase-1B contributes to LPS-induced leptin resistance in male rats. American Journal of Physiology: Endocrinology and Metabolism, 308( 1), E40-E50. doi:10.1152/ajpendo.00094.2014
NLM
Borges B de C, Rorato RC, Uchôa ET, Marangon PB, Elias CF, Antunes-Rodrigues J, Elias LLK. Protein tyrosine phosphatase-1B contributes to LPS-induced leptin resistance in male rats [Internet]. American Journal of Physiology: Endocrinology and Metabolism. 2015 ; 308( 1): E40-E50.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpendo.00094.2014
Vancouver
Borges B de C, Rorato RC, Uchôa ET, Marangon PB, Elias CF, Antunes-Rodrigues J, Elias LLK. Protein tyrosine phosphatase-1B contributes to LPS-induced leptin resistance in male rats [Internet]. American Journal of Physiology: Endocrinology and Metabolism. 2015 ; 308( 1): E40-E50.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpendo.00094.2014
Artigo de periodico
Oxytocin projections to the nucleus of the solitary tract contribute to the increased meal-related satiety responses in primary adrenal insufficiency (2013)
Uchoa, Ernane Torres; Zahm, Daniel S.; Borges, Beatriz de Carvalho; Rorato, Rodrigo; Antunes-Rodrigues, José; Elias, Lucila Leico Kagohara
Source: Experimental Physiology. Unidade: FMRP
Subjects: OCITOCINA, HIPOTÁLAMO, ADRENALECTOMIA ANIMAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
UCHOA, Ernane Torres et al. Oxytocin projections to the nucleus of the solitary tract contribute to the increased meal-related satiety responses in primary adrenal insufficiency. Experimental Physiology, v. 98, n. 10, p. 1495-1504, 2013Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2013.073726. Acesso em: 13 nov. 2024.
APA
Uchoa, E. T., Zahm, D. S., Borges, B. de C., Rorato, R., Antunes-Rodrigues, J., & Elias, L. L. K. (2013). Oxytocin projections to the nucleus of the solitary tract contribute to the increased meal-related satiety responses in primary adrenal insufficiency. Experimental Physiology, 98( 10), 1495-1504. doi:10.1113/expphysiol.2013.073726
NLM
Uchoa ET, Zahm DS, Borges B de C, Rorato R, Antunes-Rodrigues J, Elias LLK. Oxytocin projections to the nucleus of the solitary tract contribute to the increased meal-related satiety responses in primary adrenal insufficiency [Internet]. Experimental Physiology. 2013 ; 98( 10): 1495-1504.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1113/expphysiol.2013.073726
Vancouver
Uchoa ET, Zahm DS, Borges B de C, Rorato R, Antunes-Rodrigues J, Elias LLK. Oxytocin projections to the nucleus of the solitary tract contribute to the increased meal-related satiety responses in primary adrenal insufficiency [Internet]. Experimental Physiology. 2013 ; 98( 10): 1495-1504.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1113/expphysiol.2013.073726

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