A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CUNHA, Fernanda Marques da et al. Mild mitochondrial uncoupling as a therapeutic strategy. Current Drug Targets, v. 12, n. 6, p. 783-789, 2011Tradução . . Acesso em: 04 out. 2024.
APA
Cunha, F. M. da, Caldeira da Silva, C. C., Cerqueira, F. M., & Kowaltowski, A. J. (2011). Mild mitochondrial uncoupling as a therapeutic strategy. Current Drug Targets, 12( 6), 783-789.
NLM
Cunha FM da, Caldeira da Silva CC, Cerqueira FM, Kowaltowski AJ. Mild mitochondrial uncoupling as a therapeutic strategy. Current Drug Targets. 2011 ; 12( 6): 783-789.[citado 2024 out. 04 ]
Vancouver
Cunha FM da, Caldeira da Silva CC, Cerqueira FM, Kowaltowski AJ. Mild mitochondrial uncoupling as a therapeutic strategy. Current Drug Targets. 2011 ; 12( 6): 783-789.[citado 2024 out. 04 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SARTORI, Adriano et al. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Free Radical Biology and Medicine. New York: Escola de Artes, Ciências e Humanidades, Universidade de São Paulo. . Acesso em: 04 out. 2024. , 2008
APA
Sartori, A., Garay-Malpartida, H. M., Forni, M. F. P. de A. D., Schumacher, R. I., Dutra, F., Sogayar, M. C., & Bechara, E. J. H. (2008). Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Free Radical Biology and Medicine. New York: Escola de Artes, Ciências e Humanidades, Universidade de São Paulo.
NLM
Sartori A, Garay-Malpartida HM, Forni MFP de AD, Schumacher RI, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Free Radical Biology and Medicine. 2008 ; 45 S157 res. 426.[citado 2024 out. 04 ]
Vancouver
Sartori A, Garay-Malpartida HM, Forni MFP de AD, Schumacher RI, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Free Radical Biology and Medicine. 2008 ; 45 S157 res. 426.[citado 2024 out. 04 ]
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SARTORI, Adriano et al. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Chemical Research in Toxicology, v. 21, n. 9, p. 1841-1850, 2008Tradução . . Disponível em: https://doi.org/10.1021/tx8001753. Acesso em: 04 out. 2024.
APA
Sartori, A., Garay-Malpartida, H. M., Forni, M. F., Schumacher, R. I., Dutra, F., Sogayar, M. C., & Bechara, E. J. H. (2008). Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Chemical Research in Toxicology, 21( 9), 1841-1850. doi:10.1021/tx8001753
NLM
Sartori A, Garay-Malpartida HM, Forni MF, Schumacher RI, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells [Internet]. Chemical Research in Toxicology. 2008 ; 21( 9): 1841-1850.[citado 2024 out. 04 ] Available from: https://doi.org/10.1021/tx8001753
Vancouver
Sartori A, Garay-Malpartida HM, Forni MF, Schumacher RI, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells [Internet]. Chemical Research in Toxicology. 2008 ; 21( 9): 1841-1850.[citado 2024 out. 04 ] Available from: https://doi.org/10.1021/tx8001753