Source: Journal of Neural Transmission. Unidade: FM
Subjects: ESQUIZOFRENIA (FISIOPATOLOGIA), PROLIFERAÇÃO CELULAR, COMPLICAÇÕES DO TRABALHO DE PARTO, IMUNOHISTOQUÍMICA, ANÓXIA FETAL, RATOS, MODELOS ANIMAIS
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SCHAEFFER, Evelin L. et al. Increased cell proliferation in the rat anterior cingulate cortex following neonatal hypoxia: relevance to schizophrenia. Journal of Neural Transmission, v. 120, n. 1, p. 187-195, 2013Tradução . . Disponível em: http://link.springer.com/article/10.1007%2Fs00702-012-0859-y. Acesso em: 10 nov. 2024.APA
Schaeffer, E. L., Kühn, F., Schmitt, A., Gattaz, W. F., Gruber, O., Schneider-Axmann, T., et al. (2013). Increased cell proliferation in the rat anterior cingulate cortex following neonatal hypoxia: relevance to schizophrenia. Journal of Neural Transmission, 120( 1), 187-195. doi:10.1007/s00702-012-0859-yNLM
Schaeffer EL, Kühn F, Schmitt A, Gattaz WF, Gruber O, Schneider-Axmann T, Falkai P, Schmitt A. Increased cell proliferation in the rat anterior cingulate cortex following neonatal hypoxia: relevance to schizophrenia [Internet]. Journal of Neural Transmission. 2013 ; 120( 1): 187-195.[citado 2024 nov. 10 ] Available from: http://link.springer.com/article/10.1007%2Fs00702-012-0859-yVancouver
Schaeffer EL, Kühn F, Schmitt A, Gattaz WF, Gruber O, Schneider-Axmann T, Falkai P, Schmitt A. Increased cell proliferation in the rat anterior cingulate cortex following neonatal hypoxia: relevance to schizophrenia [Internet]. Journal of Neural Transmission. 2013 ; 120( 1): 187-195.[citado 2024 nov. 10 ] Available from: http://link.springer.com/article/10.1007%2Fs00702-012-0859-y
