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Artigo de periodico
Promotion of neutralizing antibody-independent immunity to wild-type and SARS-CoV-2 variants of concern using an RBD-Nucleocapsid fusion protein (2022)
Castro, Julia T.; Azevedo, Patrick; Fumagalli, Marcílio Jorge; Hojo-Souza, Natalia S.; Salazar, Natalia; Almeida, Gregório G.; Oliveira, Livia I.; Faustino, Lídia; Antonelli, Lis R.; Marçal, Tomas G.; Augusto, Marconi; Valiate, Bruno; Fiorini, Alex; Rattis, Bruna Amanda da Cruz; Ramos, Simone Gusmão; Piccin, Mariela Pires Cabral; Nonato, Osvaldo Campos dos Santos; Benevides, Luciana; Magalhães, Rubens; Cassaro, Bruno; Burle, Gabriela; Doro, Daniel; Kalil, Jorge; Durigon, Edison Luiz ; Salazar, Andrés; Caballero, Otávia; Santiago, Helton; Machado, Alexandre; Silva, João Santana da ; Fonseca, Flávio da; Fernandes, Ana Paula; Teixeira, Santuza R.; Gazzinelli, Ricardo Tostes
Source: Nature Communications. Unidades: FMRP, ICB
Subjects: ANTICORPOS, LINFÓCITOS T, VACINAS, COVID-19, GLICOPROTEÍNAS, CORONAVIRUS
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ABNT
CASTRO, Julia T. et al. Promotion of neutralizing antibody-independent immunity to wild-type and SARS-CoV-2 variants of concern using an RBD-Nucleocapsid fusion protein. Nature Communications, v. 13, n. 1, p. 1-16, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41467-022-32547-y. Acesso em: 28 jul. 2024.
APA
Castro, J. T., Azevedo, P., Fumagalli, M. J., Hojo-Souza, N. S., Salazar, N., Almeida, G. G., et al. (2022). Promotion of neutralizing antibody-independent immunity to wild-type and SARS-CoV-2 variants of concern using an RBD-Nucleocapsid fusion protein. Nature Communications, 13( 1), 1-16. doi:10.1038/s41467-022-32547-y
NLM
Castro JT, Azevedo P, Fumagalli MJ, Hojo-Souza NS, Salazar N, Almeida GG, Oliveira LI, Faustino L, Antonelli LR, Marçal TG, Augusto M, Valiate B, Fiorini A, Rattis BA da C, Ramos SG, Piccin MPC, Nonato OC dos S, Benevides L, Magalhães R, Cassaro B, Burle G, Doro D, Kalil J, Durigon EL, Salazar A, Caballero O, Santiago H, Machado A, Silva JS da, Fonseca F da, Fernandes AP, Teixeira SR, Gazzinelli RT. Promotion of neutralizing antibody-independent immunity to wild-type and SARS-CoV-2 variants of concern using an RBD-Nucleocapsid fusion protein [Internet]. Nature Communications. 2022 ; 13( 1): 1-16.[citado 2024 jul. 28 ] Available from: https://doi.org/10.1038/s41467-022-32547-y
Vancouver
Castro JT, Azevedo P, Fumagalli MJ, Hojo-Souza NS, Salazar N, Almeida GG, Oliveira LI, Faustino L, Antonelli LR, Marçal TG, Augusto M, Valiate B, Fiorini A, Rattis BA da C, Ramos SG, Piccin MPC, Nonato OC dos S, Benevides L, Magalhães R, Cassaro B, Burle G, Doro D, Kalil J, Durigon EL, Salazar A, Caballero O, Santiago H, Machado A, Silva JS da, Fonseca F da, Fernandes AP, Teixeira SR, Gazzinelli RT. Promotion of neutralizing antibody-independent immunity to wild-type and SARS-CoV-2 variants of concern using an RBD-Nucleocapsid fusion protein [Internet]. Nature Communications. 2022 ; 13( 1): 1-16.[citado 2024 jul. 28 ] Available from: https://doi.org/10.1038/s41467-022-32547-y
Artigo de periodico
Curcumin as a Potential Treatment for COVID-19 (2021)
Rattis, Bruna Amanda da Cruz; Ramos, Simone Gusmão ; Celes, Mara Rubia Nunes
Source: Frontiers in Pharmacology. Unidade: FMRP
Subjects: COVID-19, DOENÇAS INFECCIOSAS, AÇAFRÃO
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ABNT
RATTIS, Bruna Amanda da Cruz e RAMOS, Simone Gusmão e CELES, Mara Rubia Nunes. Curcumin as a Potential Treatment for COVID-19. Frontiers in Pharmacology, v. 12, p. 1-14, 2021Tradução . . Disponível em: https://doi.org/10.3389/fphar.2021.675287. Acesso em: 28 jul. 2024.
APA
Rattis, B. A. da C., Ramos, S. G., & Celes, M. R. N. (2021). Curcumin as a Potential Treatment for COVID-19. Frontiers in Pharmacology, 12, 1-14. doi:10.3389/fphar.2021.675287
NLM
Rattis BA da C, Ramos SG, Celes MRN. Curcumin as a Potential Treatment for COVID-19 [Internet]. Frontiers in Pharmacology. 2021 ; 12 1-14.[citado 2024 jul. 28 ] Available from: https://doi.org/10.3389/fphar.2021.675287
Vancouver
Rattis BA da C, Ramos SG, Celes MRN. Curcumin as a Potential Treatment for COVID-19 [Internet]. Frontiers in Pharmacology. 2021 ; 12 1-14.[citado 2024 jul. 28 ] Available from: https://doi.org/10.3389/fphar.2021.675287
Artigo de periodico
ACE2 down-regulation may act as a transient molecular disease causing RAAS dysregulation and tissue damage in the microcirculatory environment among COVID-19 patients (2021)
Ramos, Simone Gusmão; Rattis, Bruna Amanda da Cruz; Ottaviani, Giulia; Celes, Mara Rubia Nunes ; Dias, Eliane Pedra
Source: The American Journal of Pathology. Unidade: FMRP
Subjects: ANGIOTENSINA II, COVID-19, MICROCIRCULAÇÃO, SISTEMA RENINA-ANGIOTENSINA
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ABNT
RAMOS, Simone Gusmão et al. ACE2 down-regulation may act as a transient molecular disease causing RAAS dysregulation and tissue damage in the microcirculatory environment among COVID-19 patients. The American Journal of Pathology, v. 191, n. 7, p. 1154-1164, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.ajpath.2021.04.010. Acesso em: 28 jul. 2024.
APA
Ramos, S. G., Rattis, B. A. da C., Ottaviani, G., Celes, M. R. N., & Dias, E. P. (2021). ACE2 down-regulation may act as a transient molecular disease causing RAAS dysregulation and tissue damage in the microcirculatory environment among COVID-19 patients. The American Journal of Pathology, 191( 7), 1154-1164. doi:10.1016/j.ajpath.2021.04.010
NLM
Ramos SG, Rattis BA da C, Ottaviani G, Celes MRN, Dias EP. ACE2 down-regulation may act as a transient molecular disease causing RAAS dysregulation and tissue damage in the microcirculatory environment among COVID-19 patients [Internet]. The American Journal of Pathology. 2021 ; 191( 7): 1154-1164.[citado 2024 jul. 28 ] Available from: https://doi.org/10.1016/j.ajpath.2021.04.010
Vancouver
Ramos SG, Rattis BA da C, Ottaviani G, Celes MRN, Dias EP. ACE2 down-regulation may act as a transient molecular disease causing RAAS dysregulation and tissue damage in the microcirculatory environment among COVID-19 patients [Internet]. The American Journal of Pathology. 2021 ; 191( 7): 1154-1164.[citado 2024 jul. 28 ] Available from: https://doi.org/10.1016/j.ajpath.2021.04.010
Artigo de periodico
IL-22 promotes IFN-γ-mediated immunity against histoplasma capsulatum Infection (2020)
Prado, Morgana Kelly Borges ; Fontanari, Caroline; Souza, Camila de Oliveira Silva e ; Gardinassi, Luiz Gustavo Araujo ; Zoccal, Karina Furlani ; Paula-Silva, Francisco Wanderley Garcia de ; Peti, Ana Paula Ferranti ; Sorgi, Carlos Arterio ; Meirelles, Alyne Fávero Galvão ; Ramos, Simone Gusmão ; Alves Filho, José Carlos Farias ; Faccioli, Lucia Helena
Source: Biomolecules. Unidades: FCFRP, FORP, FFCLRP, FMRP
Subjects: MICOSES, LINFÓCITOS T, CITOCINAS, ÓXIDO NÍTRICO
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ABNT
PRADO, Morgana Kelly Borges et al. IL-22 promotes IFN-γ-mediated immunity against histoplasma capsulatum Infection. Biomolecules, v. 10, n. 6, p. 1-18, 2020Tradução . . Disponível em: https://doi.org/10.3390/biom10060865. Acesso em: 28 jul. 2024.
APA
Prado, M. K. B., Fontanari, C., Souza, C. de O. S. e, Gardinassi, L. G. A., Zoccal, K. F., Paula-Silva, F. W. G. de, et al. (2020). IL-22 promotes IFN-γ-mediated immunity against histoplasma capsulatum Infection. Biomolecules, 10( 6), 1-18. doi:10.3390/biom10060865
NLM
Prado MKB, Fontanari C, Souza C de OS e, Gardinassi LGA, Zoccal KF, Paula-Silva FWG de, Peti APF, Sorgi CA, Meirelles AFG, Ramos SG, Alves Filho JCF, Faccioli LH. IL-22 promotes IFN-γ-mediated immunity against histoplasma capsulatum Infection [Internet]. Biomolecules. 2020 ;10( 6): 1-18.[citado 2024 jul. 28 ] Available from: https://doi.org/10.3390/biom10060865
Vancouver
Prado MKB, Fontanari C, Souza C de OS e, Gardinassi LGA, Zoccal KF, Paula-Silva FWG de, Peti APF, Sorgi CA, Meirelles AFG, Ramos SG, Alves Filho JCF, Faccioli LH. IL-22 promotes IFN-γ-mediated immunity against histoplasma capsulatum Infection [Internet]. Biomolecules. 2020 ;10( 6): 1-18.[citado 2024 jul. 28 ] Available from: https://doi.org/10.3390/biom10060865
Artigo de periodico
Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model (2019)
Pérez, Malena Martínez ; Martins, Larissa Marfori Sampaio; Dias, Murilo Solano; Pereira, Camila André; Leite, Jefferson Antonio; Gonçalves, Enrico Cerioni Spiropulos; Almeida, Paula Zaghetto de ; Freitas, Emanuelle Neivert de; Tostes, Rita de Cássia Aleixo ; Ramos, Simone Gusmão ; Zoete, Marcel R. de; Ryffel, Bernhard; Silva, João Santana da ; Sartori, Daniela Carlos
Source: Immunology. Unidade: FMRP
Subjects: DOENÇAS METABÓLICAS, INFLAMAÇÃO, OBESIDADE, IMUNOLOGIA, HOMEOSTASE, INTESTINOS
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ABNT
PÉREZ, Malena Martínez et al. Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model. Immunology, v. 156, n. 4, p. 339-355, 2019Tradução . . Disponível em: https://doi.org/10.1111/imm.13028. Acesso em: 28 jul. 2024.
APA
Pérez, M. M., Martins, L. M. S., Dias, M. S., Pereira, C. A., Leite, J. A., Gonçalves, E. C. S., et al. (2019). Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model. Immunology, 156( 4), 339-355. doi:10.1111/imm.13028
NLM
Pérez MM, Martins LMS, Dias MS, Pereira CA, Leite JA, Gonçalves ECS, Almeida PZ de, Freitas EN de, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Sartori DC. Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model [Internet]. Immunology. 2019 ; 156( 4): 339-355.[citado 2024 jul. 28 ] Available from: https://doi.org/10.1111/imm.13028
Vancouver
Pérez MM, Martins LMS, Dias MS, Pereira CA, Leite JA, Gonçalves ECS, Almeida PZ de, Freitas EN de, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Sartori DC. Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model [Internet]. Immunology. 2019 ; 156( 4): 339-355.[citado 2024 jul. 28 ] Available from: https://doi.org/10.1111/imm.13028
Artigo de periodico
Improvement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophils (2018)
Cavalcanti Neto, Marinaldo Pacífico; Prado, Rafael de Queiroz; Piñeros, Annie Rocio; Sérgio, Cássia Alves; Bertolini, Thaís Barboza ; Gembre, Ana Flávia; Ramos, Simone Gusmão ; Bonato, Vânia Luiza Deperon
Source: Tuberculosis. Unidade: FMRP
Subjects: TUBERCULOSE, SEQUENCIAMENTO GENÉTICO, TESTES DE SENSIBILIDADE MICROBIANA, MUTAÇÃO, MYCOBACTERIUM TUBERCULOSIS
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ABNT
CAVALCANTI NETO, Marinaldo Pacífico et al. Improvement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophils. Tuberculosis, v. 113, p. 1-9, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.tube.2018.08.009. Acesso em: 28 jul. 2024.
APA
Cavalcanti Neto, M. P., Prado, R. de Q., Piñeros, A. R., Sérgio, C. A., Bertolini, T. B., Gembre, A. F., et al. (2018). Improvement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophils. Tuberculosis, 113, 1-9. doi:10.1016/j.tube.2018.08.009
NLM
Cavalcanti Neto MP, Prado R de Q, Piñeros AR, Sérgio CA, Bertolini TB, Gembre AF, Ramos SG, Bonato VLD. Improvement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophils [Internet]. Tuberculosis. 2018 ; 113 1-9.[citado 2024 jul. 28 ] Available from: https://doi.org/10.1016/j.tube.2018.08.009
Vancouver
Cavalcanti Neto MP, Prado R de Q, Piñeros AR, Sérgio CA, Bertolini TB, Gembre AF, Ramos SG, Bonato VLD. Improvement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophils [Internet]. Tuberculosis. 2018 ; 113 1-9.[citado 2024 jul. 28 ] Available from: https://doi.org/10.1016/j.tube.2018.08.009
Artigo de periodico
Leukotriene B4 is essential for lung host defence and alpha-defensin-1 production during Achromobacter xylosoxidans infection (2017)
Prado, Morgana K. B.; Locachevic, Gisele A.; Zoccal, Karina F.; Paula-Silva, Francisco Wanderley Garcia de; Fontanari, Caroline; Ferreira, Joseane Cristina; Pereira, Priscilla A. T.; Gardinassi, Luiz G.; Ramos, Simone Gusmão; Sorgi, Carlos Arterio; Darini, Ana Lúcia da Costa; Faccioli, Lucia Helena
Source: Scientific Reports. Unidades: FORP, FCFRP, FMRP
Subjects: LEUCOTRIENOS B, PSEUDOMONAS, INFECÇÕES BACTERIANAS GRAM-NEGATIVAS
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ABNT
PRADO, Morgana K. B. et al. Leukotriene B4 is essential for lung host defence and alpha-defensin-1 production during Achromobacter xylosoxidans infection. Scientific Reports, v. 7, n. 1, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-17993-9. Acesso em: 28 jul. 2024.
APA
Prado, M. K. B., Locachevic, G. A., Zoccal, K. F., Paula-Silva, F. W. G. de, Fontanari, C., Ferreira, J. C., et al. (2017). Leukotriene B4 is essential for lung host defence and alpha-defensin-1 production during Achromobacter xylosoxidans infection. Scientific Reports, 7( 1). doi:10.1038/s41598-017-17993-9
NLM
Prado MKB, Locachevic GA, Zoccal KF, Paula-Silva FWG de, Fontanari C, Ferreira JC, Pereira PAT, Gardinassi LG, Ramos SG, Sorgi CA, Darini AL da C, Faccioli LH. Leukotriene B4 is essential for lung host defence and alpha-defensin-1 production during Achromobacter xylosoxidans infection [Internet]. Scientific Reports. 2017 ; 7( 1):[citado 2024 jul. 28 ] Available from: https://doi.org/10.1038/s41598-017-17993-9
Vancouver
Prado MKB, Locachevic GA, Zoccal KF, Paula-Silva FWG de, Fontanari C, Ferreira JC, Pereira PAT, Gardinassi LG, Ramos SG, Sorgi CA, Darini AL da C, Faccioli LH. Leukotriene B4 is essential for lung host defence and alpha-defensin-1 production during Achromobacter xylosoxidans infection [Internet]. Scientific Reports. 2017 ; 7( 1):[citado 2024 jul. 28 ] Available from: https://doi.org/10.1038/s41598-017-17993-9
Artigo de periodico
Oral administration of Saccharomyces cerevisiae UFMG A‐905 prevents allergic asthma in mice (2017)
Fonseca, Vanessa M. B; Milani, Thamires M. S; Prado, Rafael; Bonato, Vânia Luiza Deperon; Ramos, Simone G; Martins, Flaviano S; Vianna, Elcio dos Santos Oliveira; Borges, Marcos de Carvalho
Source: Respirology. Unidade: FMRP
Subjects: SACCHAROMYCES, PROBIÓTICOS, MODELOS ANIMAIS, ASMA
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ABNT
FONSECA, Vanessa M. B et al. Oral administration of Saccharomyces cerevisiae UFMG A‐905 prevents allergic asthma in mice. Respirology, v. 22, n. 5, p. 905-912, 2017Tradução . . Disponível em: https://doi.org/10.1111/resp.12990. Acesso em: 28 jul. 2024.
APA
Fonseca, V. M. B., Milani, T. M. S., Prado, R., Bonato, V. L. D., Ramos, S. G., Martins, F. S., et al. (2017). Oral administration of Saccharomyces cerevisiae UFMG A‐905 prevents allergic asthma in mice. Respirology, 22( 5), 905-912. doi:10.1111/resp.12990
NLM
Fonseca VMB, Milani TMS, Prado R, Bonato VLD, Ramos SG, Martins FS, Vianna E dos SO, Borges M de C. Oral administration of Saccharomyces cerevisiae UFMG A‐905 prevents allergic asthma in mice [Internet]. Respirology. 2017 ; 22( 5): 905-912.[citado 2024 jul. 28 ] Available from: https://doi.org/10.1111/resp.12990
Vancouver
Fonseca VMB, Milani TMS, Prado R, Bonato VLD, Ramos SG, Martins FS, Vianna E dos SO, Borges M de C. Oral administration of Saccharomyces cerevisiae UFMG A‐905 prevents allergic asthma in mice [Internet]. Respirology. 2017 ; 22( 5): 905-912.[citado 2024 jul. 28 ] Available from: https://doi.org/10.1111/resp.12990
Artigo de periodico
Combining two potential causes of metalloproteinase secretion causes abnominal aortic aneurysms in rats: a new experimental model (2011)
Mata, Karina M.; Prudente, Paula Stramandinoli; Rocha, Fábio S.; Prado, Cibele Maria; Floriano, Elaine Medeiros; Elias Júnior, Jorge; Rizzi, Elen; Gerlach, Raquel Fernanda; Rossi, Marcos Antonio; Ramos, Simone Gusmão
Source: International Journal of Experimental Pathology. Unidades: FMRP, FORP
Subjects: ANEURISMA AÓRTICO, METALOPROTEINASES
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ABNT
MATA, Karina M. et al. Combining two potential causes of metalloproteinase secretion causes abnominal aortic aneurysms in rats: a new experimental model. International Journal of Experimental Pathology, v. 92, n. 1, p. 26-39, 2011Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2613.2010.00746.x. Acesso em: 28 jul. 2024.
APA
Mata, K. M., Prudente, P. S., Rocha, F. S., Prado, C. M., Floriano, E. M., Elias Júnior, J., et al. (2011). Combining two potential causes of metalloproteinase secretion causes abnominal aortic aneurysms in rats: a new experimental model. International Journal of Experimental Pathology, 92( 1), 26-39. doi:10.1111/j.1365-2613.2010.00746.x
NLM
Mata KM, Prudente PS, Rocha FS, Prado CM, Floriano EM, Elias Júnior J, Rizzi E, Gerlach RF, Rossi MA, Ramos SG. Combining two potential causes of metalloproteinase secretion causes abnominal aortic aneurysms in rats: a new experimental model [Internet]. International Journal of Experimental Pathology. 2011 ; 92( 1): 26-39.[citado 2024 jul. 28 ] Available from: https://doi.org/10.1111/j.1365-2613.2010.00746.x
Vancouver
Mata KM, Prudente PS, Rocha FS, Prado CM, Floriano EM, Elias Júnior J, Rizzi E, Gerlach RF, Rossi MA, Ramos SG. Combining two potential causes of metalloproteinase secretion causes abnominal aortic aneurysms in rats: a new experimental model [Internet]. International Journal of Experimental Pathology. 2011 ; 92( 1): 26-39.[citado 2024 jul. 28 ] Available from: https://doi.org/10.1111/j.1365-2613.2010.00746.x
Artigo de periodico
Dexamethasone effects in the strongyloides venezuelensis infection in a murine model (2011)
Machado, Eleuza Rodrigues; Carlos, Daniela; Sorgi, Carlos Arterio; Ramos, Simone Gusmão; Souza, Daniela Isabel; Soares, Edson Garcia; Cruz, Julia M. Costa; Ueta, Marlene Tiduko; Aronoff, David Michael; Faccioli, Lucia Helena
Source: American Journal of Tropical Medicine and Hygiene. Unidades: FCFRP, FMRP
Subjects: INFECÇÕES POR STRONGYLIDA, NEUTRÓFILOS, FÁRMACOS
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ABNT
MACHADO, Eleuza Rodrigues et al. Dexamethasone effects in the strongyloides venezuelensis infection in a murine model. American Journal of Tropical Medicine and Hygiene, v. 84, n. 6, p. 957-966, 2011Tradução . . Acesso em: 28 jul. 2024.
APA
Machado, E. R., Carlos, D., Sorgi, C. A., Ramos, S. G., Souza, D. I., Soares, E. G., et al. (2011). Dexamethasone effects in the strongyloides venezuelensis infection in a murine model. American Journal of Tropical Medicine and Hygiene, 84( 6), 957-966.
NLM
Machado ER, Carlos D, Sorgi CA, Ramos SG, Souza DI, Soares EG, Cruz JMC, Ueta MT, Aronoff DM, Faccioli LH. Dexamethasone effects in the strongyloides venezuelensis infection in a murine model. American Journal of Tropical Medicine and Hygiene. 2011 ; 84( 6): 957-966.[citado 2024 jul. 28 ]
Vancouver
Machado ER, Carlos D, Sorgi CA, Ramos SG, Souza DI, Soares EG, Cruz JMC, Ueta MT, Aronoff DM, Faccioli LH. Dexamethasone effects in the strongyloides venezuelensis infection in a murine model. American Journal of Tropical Medicine and Hygiene. 2011 ; 84( 6): 957-966.[citado 2024 jul. 28 ]

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