Filtros : "Indexado no: Science Direct" "Indexado no Chemical Abstracts" Removido: "2017" Limpar

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  • Source: Experimental Diabetes Research. Unidade: EACH

    Subjects: CHÁ (APLICAÇÕES TERAPÊUTICAS), DIABETES MELLITUS

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    • ABNT

      FIORINO, Patricia et al. The effects of green tea consumption on cardiometabolic alterations induced by experimental diabetes. Experimental Diabetes Research, v. 2012, p. 7 , 2012Tradução . . Disponível em: https://doi.org/10.1155/2012/309231. Acesso em: 16 nov. 2024.
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      Fiorino, P., Evangelista, F. S. ´A., Santos, F., Magri, F. M. M., Delorenzi, J. C. M. O. B., Ginoza, M., & Farah, V. (2012). The effects of green tea consumption on cardiometabolic alterations induced by experimental diabetes. Experimental Diabetes Research, 2012, 7 . doi:10.1155/2012/309231
    • NLM

      Fiorino P, Evangelista FS´A, Santos F, Magri FMM, Delorenzi JCMOB, Ginoza M, Farah V. The effects of green tea consumption on cardiometabolic alterations induced by experimental diabetes [Internet]. Experimental Diabetes Research. 2012 ; 2012 7 .[citado 2024 nov. 16 ] Available from: https://doi.org/10.1155/2012/309231
    • Vancouver

      Fiorino P, Evangelista FS´A, Santos F, Magri FMM, Delorenzi JCMOB, Ginoza M, Farah V. The effects of green tea consumption on cardiometabolic alterations induced by experimental diabetes [Internet]. Experimental Diabetes Research. 2012 ; 2012 7 .[citado 2024 nov. 16 ] Available from: https://doi.org/10.1155/2012/309231
  • Source: Chemical Research in Toxicology. Unidade: IQ

    Subjects: BIOQUÍMICA, REAÇÕES QUÍMICAS

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      ROYER, Leandro de Oliveira et al. Succinylacetone oxidation by oxygen/peroxynitrite: a possible source of reactive intermediates in hereditary tyrosinemia type I. Chemical Research in Toxicology, v. 17, n. 5, p. 598-604, 2004Tradução . . Acesso em: 16 nov. 2024.
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      Royer, L. de O., Knudsen, F. da S., Oliveira, M. A. L. de, Tavares, M. F. M., & Bechara, E. J. H. (2004). Succinylacetone oxidation by oxygen/peroxynitrite: a possible source of reactive intermediates in hereditary tyrosinemia type I. Chemical Research in Toxicology, 17( 5), 598-604.
    • NLM

      Royer L de O, Knudsen F da S, Oliveira MAL de, Tavares MFM, Bechara EJH. Succinylacetone oxidation by oxygen/peroxynitrite: a possible source of reactive intermediates in hereditary tyrosinemia type I. Chemical Research in Toxicology. 2004 ; 17( 5): 598-604.[citado 2024 nov. 16 ]
    • Vancouver

      Royer L de O, Knudsen F da S, Oliveira MAL de, Tavares MFM, Bechara EJH. Succinylacetone oxidation by oxygen/peroxynitrite: a possible source of reactive intermediates in hereditary tyrosinemia type I. Chemical Research in Toxicology. 2004 ; 17( 5): 598-604.[citado 2024 nov. 16 ]
  • Source: Preventive Medicine. Unidade: EERP

    Subjects: ENFERMAGEM EM SAÚDE PÚBLICA, ARTERIOSCLEROSE, HIPERTENSÃO

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      MORAES, Suzana Alves de et al. The relationship between temporal changes in blood pressure and changes in cognitive function: atherosclerosis risk in communities (ARIC) study. Preventive Medicine, v. 35, n. 3, p. 258-263, 2002Tradução . . Acesso em: 16 nov. 2024.
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      Moraes, S. A. de, Szklo, M., Knopman, D., & Sato, R. (2002). The relationship between temporal changes in blood pressure and changes in cognitive function: atherosclerosis risk in communities (ARIC) study. Preventive Medicine, 35( 3), 258-263.
    • NLM

      Moraes SA de, Szklo M, Knopman D, Sato R. The relationship between temporal changes in blood pressure and changes in cognitive function: atherosclerosis risk in communities (ARIC) study. Preventive Medicine. 2002 ; 35( 3): 258-263.[citado 2024 nov. 16 ]
    • Vancouver

      Moraes SA de, Szklo M, Knopman D, Sato R. The relationship between temporal changes in blood pressure and changes in cognitive function: atherosclerosis risk in communities (ARIC) study. Preventive Medicine. 2002 ; 35( 3): 258-263.[citado 2024 nov. 16 ]
  • Source: Journal of Comparative Physiology A. Unidade: FFCLRP

    Assunto: FISIOLOGIA

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      SANTANA, U. J. et al. Interference with the GABAergic system in the dorsolateral telencephalon and modulation of the electric organ discharge frequency in the weakly electric fish Gymnotus carapo. Journal of Comparative Physiology A, v. 187, p. 925-933, 2001Tradução . . Disponível em: https://doi.org/10.1007/s00359-001-0266-6. Acesso em: 16 nov. 2024.
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      Santana, U. J., Roque-da-Silva, A. C., Duarte, T. T., & Corrêa, S. A. L. (2001). Interference with the GABAergic system in the dorsolateral telencephalon and modulation of the electric organ discharge frequency in the weakly electric fish Gymnotus carapo. Journal of Comparative Physiology A, 187, 925-933. doi:10.1007/s00359-001-0266-6
    • NLM

      Santana UJ, Roque-da-Silva AC, Duarte TT, Corrêa SAL. Interference with the GABAergic system in the dorsolateral telencephalon and modulation of the electric organ discharge frequency in the weakly electric fish Gymnotus carapo [Internet]. Journal of Comparative Physiology A. 2001 ; 187 925-933.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00359-001-0266-6
    • Vancouver

      Santana UJ, Roque-da-Silva AC, Duarte TT, Corrêa SAL. Interference with the GABAergic system in the dorsolateral telencephalon and modulation of the electric organ discharge frequency in the weakly electric fish Gymnotus carapo [Internet]. Journal of Comparative Physiology A. 2001 ; 187 925-933.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00359-001-0266-6
  • Source: Pharmacological Research. Unidade: FCFRP

    Assunto: BROMATOLOGIA

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      DE MARTINIS, Bruno Spinosa e BIANCHI, Maria de Lourdes Pires. Effect of vitamin C supplementation against cisplatin-induced toxicity and oxidative DNA damage in rats. Pharmacological Research, v. 44, n. 4, p. 317-320, 2001Tradução . . Disponível em: https://doi.org/10.1006/phrs.2001.0860. Acesso em: 16 nov. 2024.
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      De Martinis, B. S., & Bianchi, M. de L. P. (2001). Effect of vitamin C supplementation against cisplatin-induced toxicity and oxidative DNA damage in rats. Pharmacological Research, 44( 4), 317-320. doi:10.1006/phrs.2001.0860
    • NLM

      De Martinis BS, Bianchi M de LP. Effect of vitamin C supplementation against cisplatin-induced toxicity and oxidative DNA damage in rats [Internet]. Pharmacological Research. 2001 ; 44( 4): 317-320.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1006/phrs.2001.0860
    • Vancouver

      De Martinis BS, Bianchi M de LP. Effect of vitamin C supplementation against cisplatin-induced toxicity and oxidative DNA damage in rats [Internet]. Pharmacological Research. 2001 ; 44( 4): 317-320.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1006/phrs.2001.0860
  • Source: International Immunopharmacology. Unidades: FMRP, FCFRP

    Assunto: IMUNOLOGIA CLÍNICA

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      DELLALIBERA-JOVILIANO, Renata e REIS, Marina Lemos dos e DONADI, Eduardo Antônio. Kinin system in lupus nephritis. International Immunopharmacology, v. 1, p. 1889-1896, 2001Tradução . . Disponível em: https://doi.org/10.1016/s1567-5769(01)00109-6. Acesso em: 16 nov. 2024.
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      Dellalibera-Joviliano, R., Reis, M. L. dos, & Donadi, E. A. (2001). Kinin system in lupus nephritis. International Immunopharmacology, 1, 1889-1896. doi:10.1016/s1567-5769(01)00109-6
    • NLM

      Dellalibera-Joviliano R, Reis ML dos, Donadi EA. Kinin system in lupus nephritis [Internet]. International Immunopharmacology. 2001 ; 1 1889-1896.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/s1567-5769(01)00109-6
    • Vancouver

      Dellalibera-Joviliano R, Reis ML dos, Donadi EA. Kinin system in lupus nephritis [Internet]. International Immunopharmacology. 2001 ; 1 1889-1896.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/s1567-5769(01)00109-6
  • Source: Glycobiology. Unidade: FMRP

    Subjects: INTERLEUCINAS, RECEPTORES IMUNOLÓGICOS, LEISHMANIOSES ANIMAL

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      PANUNTO-CASTELO, Ademilson et al. KM+, a lectin from Artocarpus integrifolia, induces IL-12 p40 production by macrophages and switches from type 2 to type 1 cell-mediated immunity against Leishmania major antigens, resulting in BALB/c mice resistance to infection. Glycobiology, v. 11, n. 12, p. 1035-1042, 2001Tradução . . Disponível em: https://doi.org/10.1093/glycob/11.12.1035. Acesso em: 16 nov. 2024.
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      Panunto-Castelo, A., Souza, M. A., Roque-Barreira, M. C., & Silva, J. S. da. (2001). KM+, a lectin from Artocarpus integrifolia, induces IL-12 p40 production by macrophages and switches from type 2 to type 1 cell-mediated immunity against Leishmania major antigens, resulting in BALB/c mice resistance to infection. Glycobiology, 11( 12), 1035-1042. doi:10.1093/glycob/11.12.1035
    • NLM

      Panunto-Castelo A, Souza MA, Roque-Barreira MC, Silva JS da. KM+, a lectin from Artocarpus integrifolia, induces IL-12 p40 production by macrophages and switches from type 2 to type 1 cell-mediated immunity against Leishmania major antigens, resulting in BALB/c mice resistance to infection [Internet]. Glycobiology. 2001 ; 11( 12): 1035-1042.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1093/glycob/11.12.1035
    • Vancouver

      Panunto-Castelo A, Souza MA, Roque-Barreira MC, Silva JS da. KM+, a lectin from Artocarpus integrifolia, induces IL-12 p40 production by macrophages and switches from type 2 to type 1 cell-mediated immunity against Leishmania major antigens, resulting in BALB/c mice resistance to infection [Internet]. Glycobiology. 2001 ; 11( 12): 1035-1042.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1093/glycob/11.12.1035

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