Filtros : "Indexado na Base de Dados Current Contents" "ICB" "Davel, Ana Paula Couto" Limpar

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  • Source: Journal of Neuroinflammation. Unidade: ICB

    Subjects: FARMACOLOGIA, HIPOCAMPU DE ANIMAL, RATOS WISTAR, INFLAMAÇÃO

    Acesso à fonteDOIHow to cite
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    • ABNT

      KINOSHITA, Paula Fernanda et al. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, v. 11, n. 1, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1186/s12974-014-0218-z. Acesso em: 06 nov. 2024.
    • APA

      Kinoshita, P. F., Yshii, L. M., Vasconcelos, A. R., Orellana, A. M. M., Lima, L. de S., Davel, A. P. C., et al. (2014). Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, 11( 1), 1-12. doi:10.1186/s12974-014-0218-z
    • NLM

      Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
    • Vancouver

      Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
  • Source: Journal of Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      PACHECO, Bruna P. M. et al. Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats. Journal of Hypertension, v. 29, n. 3, p. 520-528, 2011Tradução . . Disponível em: https://doi.org/10.1097/HJH.0b013e32833ca68c. Acesso em: 06 nov. 2024.
    • APA

      Pacheco, B. P. M., Crajoinas, R. O., Couto, G. K., Davel, A. P. C., Lessa, L. M., Rossoni, L. V., & Girardi, A. C. C. (2011). Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats. Journal of Hypertension, 29( 3), 520-528. doi:10.1097/HJH.0b013e32833ca68c
    • NLM

      Pacheco BPM, Crajoinas RO, Couto GK, Davel APC, Lessa LM, Rossoni LV, Girardi ACC. Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2011 ; 29( 3): 520-528.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c
    • Vancouver

      Pacheco BPM, Crajoinas RO, Couto GK, Davel APC, Lessa LM, Rossoni LV, Girardi ACC. Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2011 ; 29( 3): 520-528.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c
  • Source: American Journal of Physiology. Renal Physiology. Unidade: ICB

    Assunto: FISIOLOGIA (HUMANO)

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    • ABNT

      CRAJOINAS, Renato O. et al. Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR. American Journal of Physiology. Renal Physiology, v. 299, n. 4, p. F872-F881, 2010Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00654.2009. Acesso em: 06 nov. 2024.
    • APA

      Crajoinas, R. O., Lessa, L. M. A., Carraro-Lacroix, L. R., Davel, A. P. C., Pacheco, B. P. M., Rossoni, L. V., et al. (2010). Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR. American Journal of Physiology. Renal Physiology, 299( 4), F872-F881. doi:10.1152/ajprenal.00654.2009
    • NLM

      Crajoinas RO, Lessa LMA, Carraro-Lacroix LR, Davel APC, Pacheco BPM, Rossoni LV, Malnic G, Girardi ACC. Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR [Internet]. American Journal of Physiology. Renal Physiology. 2010 ; 299( 4): F872-F881.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1152/ajprenal.00654.2009
    • Vancouver

      Crajoinas RO, Lessa LMA, Carraro-Lacroix LR, Davel APC, Pacheco BPM, Rossoni LV, Malnic G, Girardi ACC. Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR [Internet]. American Journal of Physiology. Renal Physiology. 2010 ; 299( 4): F872-F881.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1152/ajprenal.00654.2009
  • Source: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA (HUMANO)

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    • ABNT

      GALLO, Luana C. et al. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C. Life Sciences, v. 87, n. 9-10 p. 302-308, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2010.07.005. Acesso em: 06 nov. 2024.
    • APA

      Gallo, L. C., Davel, A. P. C., Xavier, F. E., & Rossoni, L. V. (2010). Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C. Life Sciences, 87( 9-10 p. 302-308). doi:10.1016/j.lfs.2010.07.005
    • NLM

      Gallo LC, Davel APC, Xavier FE, Rossoni LV. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C [Internet]. Life Sciences. 2010 ; 87( 9-10 p. 302-308):[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.lfs.2010.07.005
    • Vancouver

      Gallo LC, Davel APC, Xavier FE, Rossoni LV. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C [Internet]. Life Sciences. 2010 ; 87( 9-10 p. 302-308):[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.lfs.2010.07.005
  • Source: Journal of Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA (HUMANO)

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    • ABNT

      KOBAYASI, Renata et al. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice. Journal of Hypertension, v. 28, n. 10, p. 2111-2119, 2010Tradução . . Disponível em: https://doi.org/10.1097/HJH.0b013e32833ca68c. Acesso em: 06 nov. 2024.
    • APA

      Kobayasi, R., Akamine, E. H., Davel, A. P. C., Rodrigues, M. A. M., Carvalho, C. R. de O., & Rossoni, L. V. (2010). Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice. Journal of Hypertension, 28( 10), 2111-2119. doi:10.1097/HJH.0b013e32833ca68c
    • NLM

      Kobayasi R, Akamine EH, Davel APC, Rodrigues MAM, Carvalho CR de O, Rossoni LV. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice [Internet]. Journal of Hypertension. 2010 ; 28( 10): 2111-2119.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c
    • Vancouver

      Kobayasi R, Akamine EH, Davel APC, Rodrigues MAM, Carvalho CR de O, Rossoni LV. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice [Internet]. Journal of Hypertension. 2010 ; 28( 10): 2111-2119.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c

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