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  • Source: Neuroscience Letters. Unidade: IQ

    Subjects: DOENÇA DE PARKINSON, PROCESSOS COGNITIVOS

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      RIBEIRO, Raul Izidoro et al. Glycine transporter-1 inhibition by NFPS promotes neuroprotection against striatal damage models. Neuroscience Letters, v. 826, n. 15, p. 1-7 art. 137715, 2024Tradução . . Disponível em: https://dx.doi.org/10.1016/j.neulet.2024.137715. Acesso em: 29 set. 2024.
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      Ribeiro, R. I., Carvalho, G. A. de, Chiareli, R. A., Lima, I. V. de A., Bellozi, P. M. Q., Lima, O. C. O., et al. (2024). Glycine transporter-1 inhibition by NFPS promotes neuroprotection against striatal damage models. Neuroscience Letters, 826( 15), 1-7 art. 137715. doi:10.1016/j.neulet.2024.137715
    • NLM

      Ribeiro RI, Carvalho GA de, Chiareli RA, Lima IV de A, Bellozi PMQ, Lima OCO, Giacomelli ÁO, Birbrair A, Gomez RS, Oliveira ACP de, Ulrich H, Pinto MCX. Glycine transporter-1 inhibition by NFPS promotes neuroprotection against striatal damage models [Internet]. Neuroscience Letters. 2024 ; 826( 15): 1-7 art. 137715.[citado 2024 set. 29 ] Available from: https://dx.doi.org/10.1016/j.neulet.2024.137715
    • Vancouver

      Ribeiro RI, Carvalho GA de, Chiareli RA, Lima IV de A, Bellozi PMQ, Lima OCO, Giacomelli ÁO, Birbrair A, Gomez RS, Oliveira ACP de, Ulrich H, Pinto MCX. Glycine transporter-1 inhibition by NFPS promotes neuroprotection against striatal damage models [Internet]. Neuroscience Letters. 2024 ; 826( 15): 1-7 art. 137715.[citado 2024 set. 29 ] Available from: https://dx.doi.org/10.1016/j.neulet.2024.137715
  • Source: Mechanisms of Ageing and Development. Unidade: IQ

    Subjects: MITOCÔNDRIAS, DIETA

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      ABREU, Phablo et al. Calorie restriction changes muscle satellite cell proliferation in a manner independent of metabolic modulation. Mechanisms of Ageing and Development, v. 192, p. 1-4 art. 111362, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.mad.2020.111362. Acesso em: 29 set. 2024.
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      Abreu, P., Serna, J. D. C., Munhoz, A. C., & Kowaltowski, A. J. (2020). Calorie restriction changes muscle satellite cell proliferation in a manner independent of metabolic modulation. Mechanisms of Ageing and Development, 192, 1-4 art. 111362. doi:10.1016/j.mad.2020.111362
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      Abreu P, Serna JDC, Munhoz AC, Kowaltowski AJ. Calorie restriction changes muscle satellite cell proliferation in a manner independent of metabolic modulation [Internet]. Mechanisms of Ageing and Development. 2020 ; 192 1-4 art. 111362.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.mad.2020.111362
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      Abreu P, Serna JDC, Munhoz AC, Kowaltowski AJ. Calorie restriction changes muscle satellite cell proliferation in a manner independent of metabolic modulation [Internet]. Mechanisms of Ageing and Development. 2020 ; 192 1-4 art. 111362.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.mad.2020.111362
  • Source: Molecular and Cellular Endocrinology. Unidade: IQ

    Subjects: CITOCINAS, ESTRESSE OXIDATIVO, HIPERTIREOIDISMO

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      BALDISSARELLI, Jucimara et al. Increased cytokines production and oxidative stress are related with purinergic signaling and cell survival in post-thyroidectomy hypothyroidism. Molecular and Cellular Endocrinology, v. 499, p. 1-8 art. 110594 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2019.110594. Acesso em: 29 set. 2024.
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      Baldissarelli, J., Mânica, A., Pillat, M. M., Bagatini, M. D., Leal, D. B. R., Abdala, F. H., et al. (2020). Increased cytokines production and oxidative stress are related with purinergic signaling and cell survival in post-thyroidectomy hypothyroidism. Molecular and Cellular Endocrinology, 499, 1-8 art. 110594 . doi:10.1016/j.mce.2019.110594
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      Baldissarelli J, Mânica A, Pillat MM, Bagatini MD, Leal DBR, Abdala FH, Morsch VM, Ulrich H, Bornemann CP, Schetinger MRC. Increased cytokines production and oxidative stress are related with purinergic signaling and cell survival in post-thyroidectomy hypothyroidism [Internet]. Molecular and Cellular Endocrinology. 2020 ; 499 1-8 art. 110594 .[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.mce.2019.110594
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      Baldissarelli J, Mânica A, Pillat MM, Bagatini MD, Leal DBR, Abdala FH, Morsch VM, Ulrich H, Bornemann CP, Schetinger MRC. Increased cytokines production and oxidative stress are related with purinergic signaling and cell survival in post-thyroidectomy hypothyroidism [Internet]. Molecular and Cellular Endocrinology. 2020 ; 499 1-8 art. 110594 .[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.mce.2019.110594
  • Source: Chemico-Biological Interactions. Unidade: IQ

    Subjects: QUIMIOTAXIA, FUMAÇA, DOENÇA PULMONAR OBSTRUTIVA CRÔNICA

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      SILVA, Danielba Almeida da et al. Tempol reduces inflammation and oxidative damage in cigarette smoke-exposed mice by decreasing neutrophil infiltration and activating the Nrf2 pathway. Chemico-Biological Interactions, v. 329, p. 1-11 art. 109210, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2020.109210. Acesso em: 29 set. 2024.
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      Silva, D. A. da, Correia, T. M. L., Pereira, R., Silva, R. A. A. da, Augusto, O., & Queiroz, R. F. (2020). Tempol reduces inflammation and oxidative damage in cigarette smoke-exposed mice by decreasing neutrophil infiltration and activating the Nrf2 pathway. Chemico-Biological Interactions, 329, 1-11 art. 109210. doi:10.1016/j.cbi.2020.109210
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      Silva DA da, Correia TML, Pereira R, Silva RAA da, Augusto O, Queiroz RF. Tempol reduces inflammation and oxidative damage in cigarette smoke-exposed mice by decreasing neutrophil infiltration and activating the Nrf2 pathway [Internet]. Chemico-Biological Interactions. 2020 ; 329 1-11 art. 109210.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.cbi.2020.109210
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      Silva DA da, Correia TML, Pereira R, Silva RAA da, Augusto O, Queiroz RF. Tempol reduces inflammation and oxidative damage in cigarette smoke-exposed mice by decreasing neutrophil infiltration and activating the Nrf2 pathway [Internet]. Chemico-Biological Interactions. 2020 ; 329 1-11 art. 109210.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.cbi.2020.109210
  • Source: Lung Cancer. Unidade: IQ

    Subjects: NEOPLASIAS PULMONARES, ANTINEOPLÁSICOS

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      LOBO, Tatiana Correa Carneiro et al. IKKβ targeting reduces KRAS-induced lung cancer angiogenesis in vitro and in vivo: a potential anti-angiogenic therapeutic target. Lung Cancer, v. 130, p. 169-178, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.lungcan.2019.02.027. Acesso em: 29 set. 2024.
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      Lobo, T. C. C., Scalabrini, L. C., Magalhães, L. da S., Cardeal, L. B., Rodrigues, F. S., Santos, E. O. dos, et al. (2019). IKKβ targeting reduces KRAS-induced lung cancer angiogenesis in vitro and in vivo: a potential anti-angiogenic therapeutic target. Lung Cancer, 130, 169-178. doi:10.1016/j.lungcan.2019.02.027
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      Lobo TCC, Scalabrini LC, Magalhães L da S, Cardeal LB, Rodrigues FS, Santos EO dos, Baldwin AS, Levantini E, Giordano RJ, Bassères DS. IKKβ targeting reduces KRAS-induced lung cancer angiogenesis in vitro and in vivo: a potential anti-angiogenic therapeutic target [Internet]. Lung Cancer. 2019 ; 130 169-178.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.lungcan.2019.02.027
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      Lobo TCC, Scalabrini LC, Magalhães L da S, Cardeal LB, Rodrigues FS, Santos EO dos, Baldwin AS, Levantini E, Giordano RJ, Bassères DS. IKKβ targeting reduces KRAS-induced lung cancer angiogenesis in vitro and in vivo: a potential anti-angiogenic therapeutic target [Internet]. Lung Cancer. 2019 ; 130 169-178.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.lungcan.2019.02.027
  • Source: Abstracts. Conference titles: International Symposium on Bioelectrochemistry and Bioenergetics of the Bioelectrochemical Society. Unidade: IQ

    Assunto: ELETROQUÍMICA

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      LIMA, Alex da Silva et al. An electrochemical study of urate hydroperoxide using carbon microelectrodes. 2019, Anais.. Limerick: The Bioelectrochemical Society/BES, 2019. Disponível em: http://www.bes2019.ie/BES-PROGRAM.pdf. Acesso em: 29 set. 2024.
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      Lima, A. da S., Santos, C. S., Cruz, L. C. da, Meotti, F. C., & Bertotti, M. (2019). An electrochemical study of urate hydroperoxide using carbon microelectrodes. In Abstracts. Limerick: The Bioelectrochemical Society/BES. Recuperado de http://www.bes2019.ie/BES-PROGRAM.pdf
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      Lima A da S, Santos CS, Cruz LC da, Meotti FC, Bertotti M. An electrochemical study of urate hydroperoxide using carbon microelectrodes [Internet]. Abstracts. 2019 ;[citado 2024 set. 29 ] Available from: http://www.bes2019.ie/BES-PROGRAM.pdf
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      Lima A da S, Santos CS, Cruz LC da, Meotti FC, Bertotti M. An electrochemical study of urate hydroperoxide using carbon microelectrodes [Internet]. Abstracts. 2019 ;[citado 2024 set. 29 ] Available from: http://www.bes2019.ie/BES-PROGRAM.pdf
  • Source: Toxicology Letters. Conference titles: Congress of the European Societies of Toxicology/EUROTOX. Unidade: IQ

    Subjects: COLESTEROL, BIOQUÍMICA

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      MEDEIROS, Marisa Helena Gennari de e SANCHEZ, Angelica Bianchini e DI MASCIO, Paolo. DNA adducts mapping as possible urban pollution biomarker. Toxicology Letters. Clare: Instituto de Química, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.toxlet.2018.06.865. Acesso em: 29 set. 2024. , 2018
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      Medeiros, M. H. G. de, Sanchez, A. B., & Di Mascio, P. (2018). DNA adducts mapping as possible urban pollution biomarker. Toxicology Letters. Clare: Instituto de Química, Universidade de São Paulo. doi:10.1016/j.toxlet.2018.06.865
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      Medeiros MHG de, Sanchez AB, Di Mascio P. DNA adducts mapping as possible urban pollution biomarker [Internet]. Toxicology Letters. 2018 ; 295 S192 res. P16-19.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.toxlet.2018.06.865
    • Vancouver

      Medeiros MHG de, Sanchez AB, Di Mascio P. DNA adducts mapping as possible urban pollution biomarker [Internet]. Toxicology Letters. 2018 ; 295 S192 res. P16-19.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.toxlet.2018.06.865
  • Source: Molecular and Cellular Endocrinology. Unidade: IQ

    Subjects: APOPTOSE, PROLACTINA

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      WAILEMANN, Rosângela A. M et al. Heat shock protein B1 is required for the prolactin-induced cytoprotective effects on pancreatic islets. Molecular and Cellular Endocrinology, v. 477, p. 39-47, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2018.05.013. Acesso em: 29 set. 2024.
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      Wailemann, R. A. M., Terra, L. F., Oliveira, T. C., Santos, A. F. dos, Gomes, V. M., & Labriola, L. (2018). Heat shock protein B1 is required for the prolactin-induced cytoprotective effects on pancreatic islets. Molecular and Cellular Endocrinology, 477, 39-47. doi:10.1016/j.mce.2018.05.013
    • NLM

      Wailemann RAM, Terra LF, Oliveira TC, Santos AF dos, Gomes VM, Labriola L. Heat shock protein B1 is required for the prolactin-induced cytoprotective effects on pancreatic islets [Internet]. Molecular and Cellular Endocrinology. 2018 ; 477 39-47.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.mce.2018.05.013
    • Vancouver

      Wailemann RAM, Terra LF, Oliveira TC, Santos AF dos, Gomes VM, Labriola L. Heat shock protein B1 is required for the prolactin-induced cytoprotective effects on pancreatic islets [Internet]. Molecular and Cellular Endocrinology. 2018 ; 477 39-47.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.mce.2018.05.013
  • Source: Toxicology Letters. Conference titles: Congress of the European Societies of Toxicology/EUROTOX. Unidade: IQ

    Subjects: POLUIÇÃO, BIOMARCADORES

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      DI MASCIO, Paolo et al. Singlet molecular oxygen generated by cholesterol, oleic, linoleic hydroperoxides with nitronium ion. Toxicology Letters. Clare: Instituto de Química, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.toxlet.2018.06.864. Acesso em: 29 set. 2024. , 2018
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      Di Mascio, P., Scalfo, A. C., Miyamoto, S., Prado, F. M., & Medeiros, M. H. G. de. (2018). Singlet molecular oxygen generated by cholesterol, oleic, linoleic hydroperoxides with nitronium ion. Toxicology Letters. Clare: Instituto de Química, Universidade de São Paulo. doi:10.1016/j.toxlet.2018.06.864
    • NLM

      Di Mascio P, Scalfo AC, Miyamoto S, Prado FM, Medeiros MHG de. Singlet molecular oxygen generated by cholesterol, oleic, linoleic hydroperoxides with nitronium ion [Internet]. Toxicology Letters. 2018 ; 295 S192 res. P16-18.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.toxlet.2018.06.864
    • Vancouver

      Di Mascio P, Scalfo AC, Miyamoto S, Prado FM, Medeiros MHG de. Singlet molecular oxygen generated by cholesterol, oleic, linoleic hydroperoxides with nitronium ion [Internet]. Toxicology Letters. 2018 ; 295 S192 res. P16-18.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.toxlet.2018.06.864
  • Source: Toxicology. Unidade: IQ

    Subjects: DANO AO DNA, MITOCÔNDRIAS, CONTAMINAÇÃO

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      ROUBICEK, Deborah Arnsdorff e SOUZA-PINTO, Nadja Cristhina de. Mitochondria and mitochondrial DNA as relevant targets for environmental contaminants. Toxicology, v. 391, p. 100-108, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.tox.2017.06.012. Acesso em: 29 set. 2024.
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      Roubicek, D. A., & Souza-Pinto, N. C. de. (2017). Mitochondria and mitochondrial DNA as relevant targets for environmental contaminants. Toxicology, 391, 100-108. doi:10.1016/j.tox.2017.06.012
    • NLM

      Roubicek DA, Souza-Pinto NC de. Mitochondria and mitochondrial DNA as relevant targets for environmental contaminants [Internet]. Toxicology. 2017 ; 391 100-108.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.tox.2017.06.012
    • Vancouver

      Roubicek DA, Souza-Pinto NC de. Mitochondria and mitochondrial DNA as relevant targets for environmental contaminants [Internet]. Toxicology. 2017 ; 391 100-108.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.tox.2017.06.012
  • Source: Chemico Biological Interactions. Unidade: IQ

    Subjects: MITOCÔNDRIAS, ESTRESSE OXIDATIVO, RADICAIS LIVRES, CÁLCIO

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      LUCAS, Aline M et al. Diazoxide prevents reactive oxygen species and mitochondrial damage, leading to anti-hypertrophic effects. Chemico Biological Interactions, v. 261, p. 50-55, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2016.11.012. Acesso em: 29 set. 2024.
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      Lucas, A. M., Caldas, F. R., Silva, A. P. da, Ventura, M. M., Leite, I. M., Filgueiras, A. B., et al. (2017). Diazoxide prevents reactive oxygen species and mitochondrial damage, leading to anti-hypertrophic effects. Chemico Biological Interactions, 261, 50-55. doi:10.1016/j.cbi.2016.11.012
    • NLM

      Lucas AM, Caldas FR, Silva AP da, Ventura MM, Leite IM, Filgueiras AB, Silva CGL, Kowaltowski AJ, Facundo HT. Diazoxide prevents reactive oxygen species and mitochondrial damage, leading to anti-hypertrophic effects [Internet]. Chemico Biological Interactions. 2017 ; 261 50-55.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.cbi.2016.11.012
    • Vancouver

      Lucas AM, Caldas FR, Silva AP da, Ventura MM, Leite IM, Filgueiras AB, Silva CGL, Kowaltowski AJ, Facundo HT. Diazoxide prevents reactive oxygen species and mitochondrial damage, leading to anti-hypertrophic effects [Internet]. Chemico Biological Interactions. 2017 ; 261 50-55.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.cbi.2016.11.012
  • Source: Mechanisms of Ageing and Development. Unidades: ICB, IQ

    Subjects: MITOCÔNDRIAS, FOSFOLIPÍDEOS

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      MARTINEZ, Luis Alberto Luevano et al. Calorie restriction promotes cardiolipin biosynthesis and distribution between mitochondrial membranes. Mechanisms of Ageing and Development, v. 162, p. 9-17, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mad.2017.02.004. Acesso em: 29 set. 2024.
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      Martinez, L. A. L., Forni, M. F., Peloggia, J., Watanabe, I. -S., & Kowaltowski, A. J. (2017). Calorie restriction promotes cardiolipin biosynthesis and distribution between mitochondrial membranes. Mechanisms of Ageing and Development, 162, 9-17. doi:10.1016/j.mad.2017.02.004
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      Martinez LAL, Forni MF, Peloggia J, Watanabe I-S, Kowaltowski AJ. Calorie restriction promotes cardiolipin biosynthesis and distribution between mitochondrial membranes [Internet]. Mechanisms of Ageing and Development. 2017 ; 162 9-17.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.mad.2017.02.004
    • Vancouver

      Martinez LAL, Forni MF, Peloggia J, Watanabe I-S, Kowaltowski AJ. Calorie restriction promotes cardiolipin biosynthesis and distribution between mitochondrial membranes [Internet]. Mechanisms of Ageing and Development. 2017 ; 162 9-17.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.mad.2017.02.004
  • Source: International Journal of Cardiology. Unidades: IQ, ICB

    Subjects: INFARTO DO MIOCÁRDIO, ESTRESSE OXIDATIVO, ALDEÍDOS

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      GOMES, Katia Maria Sampaio et al. Aldehydic load and aldehyde dehydrogenase 2 profile during the progression of post-myocardial infarction cardiomyopathy: benefits of alda-1. International Journal of Cardiology, v. 179, p. 129-138, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.ijcard.2014.10.140. Acesso em: 29 set. 2024.
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      Gomes, K. M. S., Bechara, L. R. G., Lima, V. M., Ribeiro, M. A. C., Campos, J. C., Dourado, P. M. M., et al. (2014). Aldehydic load and aldehyde dehydrogenase 2 profile during the progression of post-myocardial infarction cardiomyopathy: benefits of alda-1. International Journal of Cardiology, 179, 129-138. doi:10.1016/j.ijcard.2014.10.140
    • NLM

      Gomes KMS, Bechara LRG, Lima VM, Ribeiro MAC, Campos JC, Dourado PMM, Kowaltowski AJ, Mochly-Rosen D, Ferreira JCB. Aldehydic load and aldehyde dehydrogenase 2 profile during the progression of post-myocardial infarction cardiomyopathy: benefits of alda-1 [Internet]. International Journal of Cardiology. 2014 ; 179 129-138.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.ijcard.2014.10.140
    • Vancouver

      Gomes KMS, Bechara LRG, Lima VM, Ribeiro MAC, Campos JC, Dourado PMM, Kowaltowski AJ, Mochly-Rosen D, Ferreira JCB. Aldehydic load and aldehyde dehydrogenase 2 profile during the progression of post-myocardial infarction cardiomyopathy: benefits of alda-1 [Internet]. International Journal of Cardiology. 2014 ; 179 129-138.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.ijcard.2014.10.140
  • Source: Abstracts. Conference titles: Annual Meeting of the European Radiation Research Society. Unidade: IQ

    Subjects: DANO AO DNA, RADIAÇÃO GAMA

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      OSAKI, Juliana Harumi e SILVA, Gisele Espinha Teixeira da e FORTI, Fabio Luis. The small GTPases RhoA Rac1 are involved in DNA damage repair induced by low doses of gamma radiation. 2013, Anais.. Dublin: European Radiation Research Society, 2013. . Acesso em: 29 set. 2024.
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      Osaki, J. H., Silva, G. E. T. da, & Forti, F. L. (2013). The small GTPases RhoA Rac1 are involved in DNA damage repair induced by low doses of gamma radiation. In Abstracts. Dublin: European Radiation Research Society.
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      Osaki JH, Silva GET da, Forti FL. The small GTPases RhoA Rac1 are involved in DNA damage repair induced by low doses of gamma radiation. Abstracts. 2013 ;[citado 2024 set. 29 ]
    • Vancouver

      Osaki JH, Silva GET da, Forti FL. The small GTPases RhoA Rac1 are involved in DNA damage repair induced by low doses of gamma radiation. Abstracts. 2013 ;[citado 2024 set. 29 ]
  • Source: Chemico-Biological Interactions. Unidade: IQ

    Subjects: ANTAGONISMO DE DROGAS, BIOQUÍMICA

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      MEOTTI, Flavia Carla et al. Antagonism of the transient receptor potential ankyrin 1 (TRPA1) attenuates hyperalgesia and urinary bladder overactivity in cyclophosphamide-induced haemorrhagic cystitis. Chemico-Biological Interactions, v. 203, n. 2, p. 440-447, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2013.03.008. Acesso em: 29 set. 2024.
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      Meotti, F. C., Forner, S., Lima-Garcia, J. F., Viana, A. F., & Calixto, J. B. (2013). Antagonism of the transient receptor potential ankyrin 1 (TRPA1) attenuates hyperalgesia and urinary bladder overactivity in cyclophosphamide-induced haemorrhagic cystitis. Chemico-Biological Interactions, 203( 2), 440-447. doi:10.1016/j.cbi.2013.03.008
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      Meotti FC, Forner S, Lima-Garcia JF, Viana AF, Calixto JB. Antagonism of the transient receptor potential ankyrin 1 (TRPA1) attenuates hyperalgesia and urinary bladder overactivity in cyclophosphamide-induced haemorrhagic cystitis [Internet]. Chemico-Biological Interactions. 2013 ; 203( 2): 440-447.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.cbi.2013.03.008
    • Vancouver

      Meotti FC, Forner S, Lima-Garcia JF, Viana AF, Calixto JB. Antagonism of the transient receptor potential ankyrin 1 (TRPA1) attenuates hyperalgesia and urinary bladder overactivity in cyclophosphamide-induced haemorrhagic cystitis [Internet]. Chemico-Biological Interactions. 2013 ; 203( 2): 440-447.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.cbi.2013.03.008
  • Source: Molecular and Cellular Endocrinology. Unidades: IQ, ICB

    Subjects: CÓRTEX ADRENAL, HORMÔNIO ADRENOCORTICOTRÓFICO

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      MENDONÇA, Pedro Omori Ribeiro de et al. N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex. Molecular and Cellular Endocrinology, v. 37, n. 1-2, p. 166-173, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2012.11.017. Acesso em: 29 set. 2024.
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      Mendonça, P. O. R. de, Liria, C. W., Machini, M. T., & Lotfi, C. F. P. (2013). N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex. Molecular and Cellular Endocrinology, 37( 1-2), 166-173. doi:10.1016/j.mce.2012.11.017
    • NLM

      Mendonça POR de, Liria CW, Machini MT, Lotfi CFP. N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex [Internet]. Molecular and Cellular Endocrinology. 2013 ; 37( 1-2): 166-173.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.mce.2012.11.017
    • Vancouver

      Mendonça POR de, Liria CW, Machini MT, Lotfi CFP. N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex [Internet]. Molecular and Cellular Endocrinology. 2013 ; 37( 1-2): 166-173.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.mce.2012.11.017
  • Source: Lung Cancer. Unidade: IQ

    Subjects: NEOPLASIAS DO TRATO RESPIRATÓRIO, ADENOCARCINOMA, MUTAÇÃO

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      BASSÈRES, Daniela Sanchez et al. Frequent downregulation of the transcription factor Foxa2 in lung cancer through epigenetic silencing. Lung Cancer, v. 77, n. 1, p. 31-37, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.lungcan.2012.01.011. Acesso em: 29 set. 2024.
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      Bassères, D. S., D'Aló, F., Yeap, B. Y., Löwenberg, E. C., Gonzalez, D. A., Yasuda, H., et al. (2012). Frequent downregulation of the transcription factor Foxa2 in lung cancer through epigenetic silencing. Lung Cancer, 77( 1), 31-37. doi:10.1016/j.lungcan.2012.01.011
    • NLM

      Bassères DS, D'Aló F, Yeap BY, Löwenberg EC, Gonzalez DA, Yasuda H, Dayaram T, Kocher ON, Godleski JJ, Richards WG, Meyerson M, Kobayashi S, Tenen DG, Halmos B, Costa DB. Frequent downregulation of the transcription factor Foxa2 in lung cancer through epigenetic silencing [Internet]. Lung Cancer. 2012 ; 77( 1): 31-37.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.lungcan.2012.01.011
    • Vancouver

      Bassères DS, D'Aló F, Yeap BY, Löwenberg EC, Gonzalez DA, Yasuda H, Dayaram T, Kocher ON, Godleski JJ, Richards WG, Meyerson M, Kobayashi S, Tenen DG, Halmos B, Costa DB. Frequent downregulation of the transcription factor Foxa2 in lung cancer through epigenetic silencing [Internet]. Lung Cancer. 2012 ; 77( 1): 31-37.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.lungcan.2012.01.011
  • Source: Chemistry and Physics Lipids. Unidades: IQ, IF

    Subjects: BIOQUÍMICA, SOLUÇÕES AQUOSAS

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      BARROSO, Rafael Pianca et al. Aqueous dispersions of DMPG in low salt contain leaky vesicles. Chemistry and Physics Lipids, v. 165, n. 2, p. 169-177, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.chemphyslip.2011.12.007. Acesso em: 29 set. 2024.
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      Barroso, R. P., Perez, K. R., Cuccovia, I. M., & Lamy, M. T. M. (2012). Aqueous dispersions of DMPG in low salt contain leaky vesicles. Chemistry and Physics Lipids, 165( 2), 169-177. doi:10.1016/j.chemphyslip.2011.12.007
    • NLM

      Barroso RP, Perez KR, Cuccovia IM, Lamy MTM. Aqueous dispersions of DMPG in low salt contain leaky vesicles [Internet]. Chemistry and Physics Lipids. 2012 ; 165( 2): 169-177.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.chemphyslip.2011.12.007
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      Barroso RP, Perez KR, Cuccovia IM, Lamy MTM. Aqueous dispersions of DMPG in low salt contain leaky vesicles [Internet]. Chemistry and Physics Lipids. 2012 ; 165( 2): 169-177.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.chemphyslip.2011.12.007
  • Source: Ageing Research Reviews. Unidade: IQ

    Subjects: MITOCÔNDRIAS, BIOQUÍMICA

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      CERQUEIRA, Fernanda Menezes e KOWALTOWSKI, Alicia Juliana. Mitochondrial metabolism in aging: effect of dietary interventions. Ageing Research Reviews, v. 12, n. 1, p. 22-28, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.arr.2012.03.009. Acesso em: 29 set. 2024.
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      Cerqueira, F. M., & Kowaltowski, A. J. (2012). Mitochondrial metabolism in aging: effect of dietary interventions. Ageing Research Reviews, 12( 1), 22-28. doi:10.1016/j.arr.2012.03.009
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      Cerqueira FM, Kowaltowski AJ. Mitochondrial metabolism in aging: effect of dietary interventions [Internet]. Ageing Research Reviews. 2012 ; 12( 1): 22-28.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.arr.2012.03.009
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      Cerqueira FM, Kowaltowski AJ. Mitochondrial metabolism in aging: effect of dietary interventions [Internet]. Ageing Research Reviews. 2012 ; 12( 1): 22-28.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.arr.2012.03.009
  • Source: Chemistry and Physics of Lipids. Unidade: IQ

    Subjects: PEPTÍDEOS, BIOQUÍMICA

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      BOZELLI JUNIOR, José Carlos et al. Effect of head group and curvature on binding of the antimicrobial peptide tritrpticin to lipid membranes. Chemistry and Physics of Lipids, v. 165, n. 4, p. 365-373, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.chemphyslip.2011.12.005. Acesso em: 29 set. 2024.
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      Bozelli Junior, J. C., Sasahara, E. T., Pinto, M. R. S., Nakaie, C. R., & Schreier, S. (2012). Effect of head group and curvature on binding of the antimicrobial peptide tritrpticin to lipid membranes. Chemistry and Physics of Lipids, 165( 4), 365-373. doi:10.1016/j.chemphyslip.2011.12.005
    • NLM

      Bozelli Junior JC, Sasahara ET, Pinto MRS, Nakaie CR, Schreier S. Effect of head group and curvature on binding of the antimicrobial peptide tritrpticin to lipid membranes [Internet]. Chemistry and Physics of Lipids. 2012 ; 165( 4): 365-373.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.chemphyslip.2011.12.005
    • Vancouver

      Bozelli Junior JC, Sasahara ET, Pinto MRS, Nakaie CR, Schreier S. Effect of head group and curvature on binding of the antimicrobial peptide tritrpticin to lipid membranes [Internet]. Chemistry and Physics of Lipids. 2012 ; 165( 4): 365-373.[citado 2024 set. 29 ] Available from: https://doi.org/10.1016/j.chemphyslip.2011.12.005

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