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Artigo de periodico
Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology (2025)
Ferreira, Ana Flávia Fernandes ; Ulrich, Henning ; Mori, Yasuo; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Molecular Neurobiology. Unidades: IQ, ICB
Subjects: GENES, DOENÇA DE PARKINSON
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FERREIRA, Ana Flávia Fernandes et al. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology. Molecular Neurobiology, v. 62, p. 5333-5346, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s12035-024-04611-9. Acesso em: 16 mar. 2025.
APA
Ferreira, A. F. F., Ulrich, H., Mori, Y., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2025). Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology. Molecular Neurobiology, 62, 5333-5346. doi:10.1007/s12035-024-04611-9
NLM
Ferreira AFF, Ulrich H, Mori Y, Feng Z-P, Sun H-S, Britto LRG de. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology [Internet]. Molecular Neurobiology. 2025 ; 62 5333-5346.[citado 2025 mar. 16 ] Available from: https://dx.doi.org/10.1007/s12035-024-04611-9
Vancouver
Ferreira AFF, Ulrich H, Mori Y, Feng Z-P, Sun H-S, Britto LRG de. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology [Internet]. Molecular Neurobiology. 2025 ; 62 5333-5346.[citado 2025 mar. 16 ] Available from: https://dx.doi.org/10.1007/s12035-024-04611-9
Artigo de periodico
Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model (2024)
Bianchetti, Maria Eduada Goulart ; Ferreira, Ana Flávia Fernandes ; Britto, Luiz Roberto Giorgetti de
Source: Frontiers in Neuroanatomy. Unidade: ICB
Subjects: FISIOLOGIA, DOENÇA DE PARKINSON, ATROFIA DE MÚLTIPLOS SISTEMAS, CITOCINAS, DOENÇAS NEURODEGENERATIVAS, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, IMUNOHISTOQUÍMICA, ELISA EM ANIMAL
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BIANCHETTI, Maria Eduada Goulart e FERREIRA, Ana Flávia Fernandes e BRITTO, Luiz Roberto Giorgetti de. Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model. Frontiers in Neuroanatomy, v. 18, p. 12 , 2024Tradução . . Disponível em: https://doi.org/10.3389/fnana.2024.1511951. Acesso em: 16 mar. 2025.
APA
Bianchetti, M. E. G., Ferreira, A. F. F., & Britto, L. R. G. de. (2024). Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model. Frontiers in Neuroanatomy, 18, 12 . doi:10.3389/fnana.2024.1511951
NLM
Bianchetti MEG, Ferreira AFF, Britto LRG de. Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model [Internet]. Frontiers in Neuroanatomy. 2024 ; 18 12 .[citado 2025 mar. 16 ] Available from: https://doi.org/10.3389/fnana.2024.1511951
Vancouver
Bianchetti MEG, Ferreira AFF, Britto LRG de. Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model [Internet]. Frontiers in Neuroanatomy. 2024 ; 18 12 .[citado 2025 mar. 16 ] Available from: https://doi.org/10.3389/fnana.2024.1511951
Artigo de periodico
Most utilized rodent models for Alzheimer’s and Parkinson’s disease: a critical review of the past 5 years (2024)
Ferreira, Ana Flávia Fernandes ; Meira Neta, Marina da Silva ; Lemuchi, Livia M. ; Bianchetti, Maria E. ; Kamidai, Nicole M. ; Kilinsky, Livia Marques ; Britto, Luiz Roberto Giorgetti de
Source: Advances in Neurology. Unidade: ICB
Subjects: FISIOLOGIA, MODELOS ANIMAIS DE DOENÇAS, ROEDORES, DOENÇAS NEURODEGENERATIVAS, DOENÇA DE PARKINSON
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FERREIRA, Ana Flávia Fernandes et al. Most utilized rodent models for Alzheimer’s and Parkinson’s disease: a critical review of the past 5 years. Advances in Neurology, v. 3, n. 3, p. 19 , 2024Tradução . . Disponível em: https://doi.org/10.36922/an.2903. Acesso em: 16 mar. 2025.
APA
Ferreira, A. F. F., Meira Neta, M. da S., Lemuchi, L. M., Bianchetti, M. E., Kamidai, N. M., Kilinsky, L. M., & Britto, L. R. G. de. (2024). Most utilized rodent models for Alzheimer’s and Parkinson’s disease: a critical review of the past 5 years. Advances in Neurology, 3( 3), 19 . doi:10.36922/an.2903
NLM
Ferreira AFF, Meira Neta M da S, Lemuchi LM, Bianchetti ME, Kamidai NM, Kilinsky LM, Britto LRG de. Most utilized rodent models for Alzheimer’s and Parkinson’s disease: a critical review of the past 5 years [Internet]. Advances in Neurology. 2024 ; 3( 3): 19 .[citado 2025 mar. 16 ] Available from: https://doi.org/10.36922/an.2903
Vancouver
Ferreira AFF, Meira Neta M da S, Lemuchi LM, Bianchetti ME, Kamidai NM, Kilinsky LM, Britto LRG de. Most utilized rodent models for Alzheimer’s and Parkinson’s disease: a critical review of the past 5 years [Internet]. Advances in Neurology. 2024 ; 3( 3): 19 .[citado 2025 mar. 16 ] Available from: https://doi.org/10.36922/an.2903
Artigo de periodico
Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease (2024)
Juvenal, Guilherme Araújo ; Higa, Guilherme Shigueto Vilar ; Marques, Lucas Bonfim; Zampieri, Thais Tessari; Viana, Felipe José Costa ; Britto, Luiz Roberto Giorgetti de ; Tang, Yong; Illes, Peter ; Virgilio, Francesco Di ; Ulrich, Henning ; Pasquale, Roberto de
Source: Purinergic Signalling. Unidades: ICB, IQ
Subjects: NEUROTRANSMISSORES, RECEPTORES
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JUVENAL, Guilherme Araújo et al. Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease. Purinergic Signalling, 2024Tradução . . Disponível em: https://dx.doi.org/10.1007/s11302-024-10034-x. Acesso em: 16 mar. 2025.
APA
Juvenal, G. A., Higa, G. S. V., Marques, L. B., Zampieri, T. T., Viana, F. J. C., Britto, L. R. G. de, et al. (2024). Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease. Purinergic Signalling. doi:10.1007/s11302-024-10034-x
NLM
Juvenal GA, Higa GSV, Marques LB, Zampieri TT, Viana FJC, Britto LRG de, Tang Y, Illes P, Virgilio FD, Ulrich H, Pasquale R de. Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease [Internet]. Purinergic Signalling. 2024 ;[citado 2025 mar. 16 ] Available from: https://dx.doi.org/10.1007/s11302-024-10034-x
Vancouver
Juvenal GA, Higa GSV, Marques LB, Zampieri TT, Viana FJC, Britto LRG de, Tang Y, Illes P, Virgilio FD, Ulrich H, Pasquale R de. Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease [Internet]. Purinergic Signalling. 2024 ;[citado 2025 mar. 16 ] Available from: https://dx.doi.org/10.1007/s11302-024-10034-x
Artigo de periodico
Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model (2024)
Ferreira, Ana Flávia Fernandes ; Ulrich, Henning ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Experimental Neurology. Unidades: ICB, IQ
Subjects: FISIOLOGIA, DOENÇAS NEURODEGENERATIVAS, DOENÇA DE PARKINSON, MODELOS ANIMAIS DE DOENÇAS, CÉLULAS MORTAS, ASTRÓCITOS, NEURÓGLIA, CAMUNDONGOS, RATOS, TÉCNICAS ESTEREOTÁXICAS, IMUNOHISTOQUÍMICA, REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
FERREIRA, Ana Flávia Fernandes et al. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, v. 377, p. 15 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2024.114780. Acesso em: 16 mar. 2025.
APA
Ferreira, A. F. F., Ulrich, H., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2024). Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, 377, 15 . doi:10.1016/j.expneurol.2024.114780
NLM
Ferreira AFF, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2025 mar. 16 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Vancouver
Ferreira AFF, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2025 mar. 16 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Artigo de periodico
Lithium as a pharmacological approach to treat behavioral complications in Alzheimer’s disease: a preclinical study (2024)
Singulani, Monique Patricio ; Talib, Leda Leme ; Forlenza, Orestes Vicente ; Britto, Luiz Roberto Giorgetti de ; Camarini, Rosana
Source: Alzheimer's & Dementia. Unidades: FM, ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, DOENÇA DE ALZHEIMER, LÍTIO, TRANSTORNO BIPOLAR, CAMUNDONGOS, IMUNOHISTOQUÍMICA
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SINGULANI, Monique Patricio et al. Lithium as a pharmacological approach to treat behavioral complications in Alzheimer’s disease: a preclinical study. Alzheimer's & Dementia, v. 20, n. S1, p. 2 , 2024Tradução . . Disponível em: https://doi.org/10.1002/alz.090340. Acesso em: 16 mar. 2025.
APA
Singulani, M. P., Talib, L. L., Forlenza, O. V., Britto, L. R. G. de, & Camarini, R. (2024). Lithium as a pharmacological approach to treat behavioral complications in Alzheimer’s disease: a preclinical study. Alzheimer's & Dementia, 20( S1), 2 . doi:10.1002/alz.090340
NLM
Singulani MP, Talib LL, Forlenza OV, Britto LRG de, Camarini R. Lithium as a pharmacological approach to treat behavioral complications in Alzheimer’s disease: a preclinical study [Internet]. Alzheimer's & Dementia. 2024 ; 20( S1): 2 .[citado 2025 mar. 16 ] Available from: https://doi.org/10.1002/alz.090340
Vancouver
Singulani MP, Talib LL, Forlenza OV, Britto LRG de, Camarini R. Lithium as a pharmacological approach to treat behavioral complications in Alzheimer’s disease: a preclinical study [Internet]. Alzheimer's & Dementia. 2024 ; 20( S1): 2 .[citado 2025 mar. 16 ] Available from: https://doi.org/10.1002/alz.090340
Artigo de periodico
‘Latent’ mild behavioral impairment as a potential diagnostic strategy for preclinical Alzheimer’s disease: an animal model study (2024)
Singulani, Monique Patricio ; Talib, Leda Leme ; Forlenza, Orestes Vicente ; Britto, Luiz Roberto Giorgetti de ; Camarini, Rosana
Source: Alzheimer's & Dementia. Unidades: FM, ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, DOENÇA DE ALZHEIMER, CAMUNDONGOS, IMUNOHISTOQUÍMICA, MODELOS ANIMAIS DE DOENÇAS, COMPORTAMENTO ANIMAL, ANSIEDADE
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ABNT
SINGULANI, Monique Patricio et al. ‘Latent’ mild behavioral impairment as a potential diagnostic strategy for preclinical Alzheimer’s disease: an animal model study. Alzheimer's & Dementia, v. 21, n. S1, p. 5 , 2024Tradução . . Disponível em: https://doi.org/10.1002/alz.087517. Acesso em: 16 mar. 2025.
APA
Singulani, M. P., Talib, L. L., Forlenza, O. V., Britto, L. R. G. de, & Camarini, R. (2024). ‘Latent’ mild behavioral impairment as a potential diagnostic strategy for preclinical Alzheimer’s disease: an animal model study. Alzheimer's & Dementia, 21( S1), 5 . doi:10.1002/alz.087517
NLM
Singulani MP, Talib LL, Forlenza OV, Britto LRG de, Camarini R. ‘Latent’ mild behavioral impairment as a potential diagnostic strategy for preclinical Alzheimer’s disease: an animal model study [Internet]. Alzheimer's & Dementia. 2024 ; 21( S1): 5 .[citado 2025 mar. 16 ] Available from: https://doi.org/10.1002/alz.087517
Vancouver
Singulani MP, Talib LL, Forlenza OV, Britto LRG de, Camarini R. ‘Latent’ mild behavioral impairment as a potential diagnostic strategy for preclinical Alzheimer’s disease: an animal model study [Internet]. Alzheimer's & Dementia. 2024 ; 21( S1): 5 .[citado 2025 mar. 16 ] Available from: https://doi.org/10.1002/alz.087517
Artigo de periodico
Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease (2024)
Singulani, Monique Patricio ; Ferreira, Ana Flávia Fernandes ; Figueroa, Paulina Belén Sepulveda ; Cuyul-Vásquez, Iván; Talib, Leda Leme ; Forlenza, Orestes Vicente ; Britto, Luiz Roberto Giorgetti de
Source: Ageing Research Reviews. Unidades: ICB, FM
Subjects: FISIOLOGIA, DOENÇA DE ALZHEIMER, DOENÇA DE PARKINSON, LÍTIO, DOENÇAS NEURODEGENERATIVAS, REVISÃO SISTEMÁTICA
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SINGULANI, Monique Patricio et al. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease. Ageing Research Reviews, v. 95, p. 20 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.arr.2024.102231. Acesso em: 16 mar. 2025.
APA
Singulani, M. P., Ferreira, A. F. F., Figueroa, P. B. S., Cuyul-Vásquez, I., Talib, L. L., Forlenza, O. V., & Britto, L. R. G. de. (2024). Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease. Ageing Research Reviews, 95, 20 . doi:10.1016/j.arr.2024.102231
NLM
Singulani MP, Ferreira AFF, Figueroa PBS, Cuyul-Vásquez I, Talib LL, Forlenza OV, Britto LRG de. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease [Internet]. Ageing Research Reviews. 2024 ; 95 20 .[citado 2025 mar. 16 ] Available from: https://doi.org/10.1016/j.arr.2024.102231
Vancouver
Singulani MP, Ferreira AFF, Figueroa PBS, Cuyul-Vásquez I, Talib LL, Forlenza OV, Britto LRG de. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease [Internet]. Ageing Research Reviews. 2024 ; 95 20 .[citado 2025 mar. 16 ] Available from: https://doi.org/10.1016/j.arr.2024.102231
Artigo de periodico
Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease (2023)
Pereira, Carolina Parga Martins; Oliveira, José Francis ; Singulani, Monique Patricio ; Ferreira, Ana Flávia Fernandes ; Britto, Luiz Roberto Giorgetti de
Source: Journal of Neuroimmunology. Unidades: ICB, FM
Subjects: FISIOLOGIA, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, DOENÇA DE PARKINSON, NEURÓGLIA, TOXINAS, COORDENAÇÃO MOTORA, RECEPTORES DOPAMINÉRGICOS, NEURÔNIOS MOTORES
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PEREIRA, Carolina Parga Martins et al. Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease. Journal of Neuroimmunology, v. 375, p. 1-11, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.jneuroim.2023.578019. Acesso em: 16 mar. 2025.
APA
Pereira, C. P. M., Oliveira, J. F., Singulani, M. P., Ferreira, A. F. F., & Britto, L. R. G. de. (2023). Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease. Journal of Neuroimmunology, 375, 1-11. doi:10.1016/j.jneuroim.2023.578019
NLM
Pereira CPM, Oliveira JF, Singulani MP, Ferreira AFF, Britto LRG de. Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease [Internet]. Journal of Neuroimmunology. 2023 ; 375 1-11.[citado 2025 mar. 16 ] Available from: https://doi.org/10.1016/j.jneuroim.2023.578019
Vancouver
Pereira CPM, Oliveira JF, Singulani MP, Ferreira AFF, Britto LRG de. Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease [Internet]. Journal of Neuroimmunology. 2023 ; 375 1-11.[citado 2025 mar. 16 ] Available from: https://doi.org/10.1016/j.jneuroim.2023.578019
Artigo de periodico
Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions (2023)
Hamasaki, Mike Yoshio ; Mendes, Caroline; Batagello, Daniella Sabino ; Hirata, Mario Hiroyuki ; Britto, Luiz Roberto Giorgetti de ; Nogueira, Maria Inês
Source: NeuroReport. Unidades: FCF, ICB
Subjects: ANÓXIA, CÓRTEX CEREBRAL
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ABNT
HAMASAKI, Mike Yoshio et al. Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions. NeuroReport, v. 34, n. 11, p. 575-582, 2023Tradução . . Disponível em: https://doi.org/10.1097/WNR.0000000000001927. Acesso em: 16 mar. 2025.
APA
Hamasaki, M. Y., Mendes, C., Batagello, D. S., Hirata, M. H., Britto, L. R. G. de, & Nogueira, M. I. (2023). Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions. NeuroReport, 34( 11), 575-582. doi:10.1097/WNR.0000000000001927
NLM
Hamasaki MY, Mendes C, Batagello DS, Hirata MH, Britto LRG de, Nogueira MI. Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions [Internet]. NeuroReport. 2023 ; 34( 11): 575-582.[citado 2025 mar. 16 ] Available from: https://doi.org/10.1097/WNR.0000000000001927
Vancouver
Hamasaki MY, Mendes C, Batagello DS, Hirata MH, Britto LRG de, Nogueira MI. Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions [Internet]. NeuroReport. 2023 ; 34( 11): 575-582.[citado 2025 mar. 16 ] Available from: https://doi.org/10.1097/WNR.0000000000001927
Artigo de periodico
The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease? (2023)
Ferreira, Ana Flávia Fernandes ; Britto, Luiz Roberto Giorgetti de
Source: Neural Regeneration Research. Unidade: ICB
Subjects: FISIOLOGIA, CANAIS IÔNICOS, ESTRESSE OXIDATIVO, DOENÇA DE PARKINSON, PROTEÍNAS DA MEMBRANA, DOENÇAS NEURODEGENERATIVAS, DOENÇAS NEUROMUSCULARES
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ABNT
FERREIRA, Ana Flávia Fernandes e BRITTO, Luiz Roberto Giorgetti de. The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease?. Neural Regeneration Research, v. 18, n. 8, p. 1652-1656, 2023Tradução . . Disponível em: https://doi.org/10.4103/1673-5374.360343. Acesso em: 16 mar. 2025.
APA
Ferreira, A. F. F., & Britto, L. R. G. de. (2023). The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease? Neural Regeneration Research, 18( 8), 1652-1656. doi:10.4103/1673-5374.360343
NLM
Ferreira AFF, Britto LRG de. The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease? [Internet]. Neural Regeneration Research. 2023 ; 18( 8): 1652-1656.[citado 2025 mar. 16 ] Available from: https://doi.org/10.4103/1673-5374.360343
Vancouver
Ferreira AFF, Britto LRG de. The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease? [Internet]. Neural Regeneration Research. 2023 ; 18( 8): 1652-1656.[citado 2025 mar. 16 ] Available from: https://doi.org/10.4103/1673-5374.360343
Parte de monografia/livro
Purinergic signaling in Parkinson’s disease (2023)
Silva, Jean Bezerra ; Ferreira, Ana Flávia Fernandes ; Glaser, Talita ; Ulrich, Henning ; Britto, Luiz Roberto Giorgetti de
Source: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Unidades: IQ, ICB
Assunto: DOENÇA DE PARKINSON
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ABNT
SILVA, Jean Bezerra et al. Purinergic signaling in Parkinson’s disease. Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Tradução . Cham: Springer, 2023. . . Acesso em: 16 mar. 2025.
APA
Silva, J. B., Ferreira, A. F. F., Glaser, T., Ulrich, H., & Britto, L. R. G. de. (2023). Purinergic signaling in Parkinson’s disease. In Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer.
NLM
Silva JB, Ferreira AFF, Glaser T, Ulrich H, Britto LRG de. Purinergic signaling in Parkinson’s disease. In: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer; 2023. [citado 2025 mar. 16 ]
Vancouver
Silva JB, Ferreira AFF, Glaser T, Ulrich H, Britto LRG de. Purinergic signaling in Parkinson’s disease. In: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer; 2023. [citado 2025 mar. 16 ]
Artigo de periodico
Triiodothyronine treatment reverses Depression-Like behavior in a triple-transgenic animal model of Alzheimer’s Disease (2022)
Maglione, Andréa V. ; Nascimento, Bruna P. P. do ; Ribeiro, Miriam O. ; Souza, Talytha J. L. de ; Silva, Renata E. C. da; Sato, Monica A. ; Penatti, Carlos A. A.; Britto, Luiz Roberto Giorgetti de ; Souza, Janaina Sena de ; Maciel, Rui M. B. ; Conceição, Rodrigo Rodrigues da ; Laureano-Melo, Roberto ; Giannocco, Gisele
Source: Metabolic Brain Disease. Unidade: ICB
Subjects: FISIOLOGIA, DOENÇA DE ALZHEIMER, DOENÇAS NEURODEGENERATIVAS, HIPOCAMPU DE ANIMAL, DOENÇAS DO SISTEMA NERVOSO CENTRAL EM ANIMAL, MODELOS ANIMAIS DE DOENÇAS, TRIIODOTIRONINA, DEPRESSÃO, CAMUNDONGOS, COMPORTAMENTO ANIMAL, SUPERÓXIDO DISMUTASE
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ABNT
MAGLIONE, Andréa V. et al. Triiodothyronine treatment reverses Depression-Like behavior in a triple-transgenic animal model of Alzheimer’s Disease. Metabolic Brain Disease, p. 1-16, 2022Tradução . . Disponível em: https://doi.org/10.1007/s11011-022-01055-9. Acesso em: 16 mar. 2025.
APA
Maglione, A. V., Nascimento, B. P. P. do, Ribeiro, M. O., Souza, T. J. L. de, Silva, R. E. C. da, Sato, M. A., et al. (2022). Triiodothyronine treatment reverses Depression-Like behavior in a triple-transgenic animal model of Alzheimer’s Disease. Metabolic Brain Disease, 1-16. doi:10.1007/s11011-022-01055-9
NLM
Maglione AV, Nascimento BPP do, Ribeiro MO, Souza TJL de, Silva REC da, Sato MA, Penatti CAA, Britto LRG de, Souza JS de, Maciel RMB, Conceição RR da, Laureano-Melo R, Giannocco G. Triiodothyronine treatment reverses Depression-Like behavior in a triple-transgenic animal model of Alzheimer’s Disease [Internet]. Metabolic Brain Disease. 2022 ; 1-16.[citado 2025 mar. 16 ] Available from: https://doi.org/10.1007/s11011-022-01055-9
Vancouver
Maglione AV, Nascimento BPP do, Ribeiro MO, Souza TJL de, Silva REC da, Sato MA, Penatti CAA, Britto LRG de, Souza JS de, Maciel RMB, Conceição RR da, Laureano-Melo R, Giannocco G. Triiodothyronine treatment reverses Depression-Like behavior in a triple-transgenic animal model of Alzheimer’s Disease [Internet]. Metabolic Brain Disease. 2022 ; 1-16.[citado 2025 mar. 16 ] Available from: https://doi.org/10.1007/s11011-022-01055-9
Artigo de periodico
The symbiotic effect of a new nutraceutical with Yeast β-Glucan, Prebiotics, Minerals, and Silybum marianum (Silymarin) for recovering metabolic homeostasis via Pgc-1α, Il-6, and Il-10 gene expression in a Type-2 Diabetes Obesity model (2022)
Santamarina, Aline Boveto ; Moraes, Ruan Carlos Macêdo ; Nehmi Filho, Victor; Murata, Gilson Masahiro ; Freitas, Jéssica Alves de ; Miranda, Danielle Araujo de ; Cerqueira, Anderson Romério Azevedo ; Costa, Soraia Katia Pereira ; Ferreira, Ana Flávia Fernandes ; Britto, Luiz Roberto Giorgetti de
Source: Antioxidants. Unidades: FM, ICB
Subjects: FARMACOLOGIA, FISIOLOGIA, PRODUTOS NATURAIS, INTERLEUCINA 6, OBESIDADE, SUPLEMENTAÇÃO ALIMENTAR, DIABETES MELLITUS, MINERAIS NA DIETA, ENZIMAS OXIRREDUTORAS, ANTIOXIDANTES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTAMARINA, Aline Boveto et al. The symbiotic effect of a new nutraceutical with Yeast β-Glucan, Prebiotics, Minerals, and Silybum marianum (Silymarin) for recovering metabolic homeostasis via Pgc-1α, Il-6, and Il-10 gene expression in a Type-2 Diabetes Obesity model. Antioxidants, v. 11, p. 1-24, 2022Tradução . . Disponível em: https://doi.org/10.3390/antiox11030447. Acesso em: 16 mar. 2025.
APA
Santamarina, A. B., Moraes, R. C. M., Nehmi Filho, V., Murata, G. M., Freitas, J. A. de, Miranda, D. A. de, et al. (2022). The symbiotic effect of a new nutraceutical with Yeast β-Glucan, Prebiotics, Minerals, and Silybum marianum (Silymarin) for recovering metabolic homeostasis via Pgc-1α, Il-6, and Il-10 gene expression in a Type-2 Diabetes Obesity model. Antioxidants, 11, 1-24. doi:10.3390/antiox11030447
NLM
Santamarina AB, Moraes RCM, Nehmi Filho V, Murata GM, Freitas JA de, Miranda DA de, Cerqueira ARA, Costa SKP, Ferreira AFF, Britto LRG de. The symbiotic effect of a new nutraceutical with Yeast β-Glucan, Prebiotics, Minerals, and Silybum marianum (Silymarin) for recovering metabolic homeostasis via Pgc-1α, Il-6, and Il-10 gene expression in a Type-2 Diabetes Obesity model [Internet]. Antioxidants. 2022 ; 11 1-24.[citado 2025 mar. 16 ] Available from: https://doi.org/10.3390/antiox11030447
Vancouver
Santamarina AB, Moraes RCM, Nehmi Filho V, Murata GM, Freitas JA de, Miranda DA de, Cerqueira ARA, Costa SKP, Ferreira AFF, Britto LRG de. The symbiotic effect of a new nutraceutical with Yeast β-Glucan, Prebiotics, Minerals, and Silybum marianum (Silymarin) for recovering metabolic homeostasis via Pgc-1α, Il-6, and Il-10 gene expression in a Type-2 Diabetes Obesity model [Internet]. Antioxidants. 2022 ; 11 1-24.[citado 2025 mar. 16 ] Available from: https://doi.org/10.3390/antiox11030447
Artigo de periodico
Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study (2022)
Faria, Daniele de Paula ; Souza, Larissa Estessi de ; Duran, Fabio Luis de Souza ; Buchpiguel, Carlos Alberto ; Britto, Luiz Roberto Giorgetti de ; Crippa, José Alexandre de Souza ; Busatto Filho, Geraldo ; Gregório, Caroline Cristiano Real
Source: International Journal of Molecular Sciences. Unidades: FM, ICB, FMRP
Subjects: FISIOLOGIA, CANABINOIDES, DOENÇA DE ALZHEIMER, MODELOS ANIMAIS DE DOENÇAS, RATOS WISTAR, ANTINEOPLÁSICOS, MEMÓRIA ANIMAL, GLICOSE
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ABNT
FARIA, Daniele de Paula et al. Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study. International Journal of Molecular Sciences, v. 23. n. 3, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.3390/ijms23031076. Acesso em: 16 mar. 2025.
APA
Faria, D. de P., Souza, L. E. de, Duran, F. L. de S., Buchpiguel, C. A., Britto, L. R. G. de, Crippa, J. A. de S., et al. (2022). Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study. International Journal of Molecular Sciences, 23. n. 3, 1-10. doi:10.3390/ijms23031076
NLM
Faria D de P, Souza LE de, Duran FL de S, Buchpiguel CA, Britto LRG de, Crippa JA de S, Busatto Filho G, Gregório CCR. Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study [Internet]. International Journal of Molecular Sciences. 2022 ; 23. n. 3 1-10.[citado 2025 mar. 16 ] Available from: https://doi.org/10.3390/ijms23031076
Vancouver
Faria D de P, Souza LE de, Duran FL de S, Buchpiguel CA, Britto LRG de, Crippa JA de S, Busatto Filho G, Gregório CCR. Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study [Internet]. International Journal of Molecular Sciences. 2022 ; 23. n. 3 1-10.[citado 2025 mar. 16 ] Available from: https://doi.org/10.3390/ijms23031076
Artigo de periodico
Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model (2022)
Ferreira, Ana Flávia Fernandes ; Singulani, Monique Patricio ; Ulrich, Henning ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Molecular Neurobiology. Unidades: IQ, ICB, FM
Subjects: FISIOLOGIA, NEUROBIOLOGIA, MODELOS ANIMAIS DE DOENÇAS, DOENÇA DE PARKINSON, MESENCÉFALO, GÂNGLIOS, RECEPTORES DE DOPAMINA, CAMUNDONGOS, IMUNOHISTOQUÍMICA, IMMUNOBLOTTING
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ABNT
FERREIRA, Ana Flávia Fernandes et al. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model. Molecular Neurobiology, v. 59, p. 1543–1559, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-022-02723-8. Acesso em: 16 mar. 2025.
APA
Ferreira, A. F. F., Singulani, M. P., Ulrich, H., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2022). Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model. Molecular Neurobiology, 59, 1543–1559. doi:10.1007/s12035-022-02723-8
NLM
Ferreira AFF, Singulani MP, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model [Internet]. Molecular Neurobiology. 2022 ; 59 1543–1559.[citado 2025 mar. 16 ] Available from: https://doi.org/10.1007/s12035-022-02723-8
Vancouver
Ferreira AFF, Singulani MP, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model [Internet]. Molecular Neurobiology. 2022 ; 59 1543–1559.[citado 2025 mar. 16 ] Available from: https://doi.org/10.1007/s12035-022-02723-8
Artigo de periodico
Electrical stimulation of the posterior insula induces mechanical analgesia in a rodent model of neuropathic pain by modulating GABAergic signaling and activity in the pain circuitry (2021)
Matielo, Heloísa Alonso ; Gonçalves, Elizamara Santos; Campos, Mariana; Oliveira, Victória Regina da Silva; Toniolo, Elaine F.; Alves, Adilson da Silva; Lebrun, Ivo ; Andrade, Daniel Ciampi Araujo de ; Teixeira, Manoel jacobsen ; Britto, Luiz Roberto Giorgetti de ; Hamani, Clement ; Dale, Camila Squarzoni
Source: Brain Research. Unidades: ICB, FM
Subjects: FISIOLOGIA, ANATOMIA, RATOS, DOR CRÔNICA, GABA, ESTIMULAÇÃO CEREBRAL, ESTIMULAÇÃO ELÉTRICA, CÓRTEX CEREBRAL DE ANIMAL, MODELOS ANIMAIS DE DOENÇAS, HIPOCAMPU DE ANIMAL, GLUTAMATOS
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ABNT
MATIELO, Heloísa Alonso et al. Electrical stimulation of the posterior insula induces mechanical analgesia in a rodent model of neuropathic pain by modulating GABAergic signaling and activity in the pain circuitry. Brain Research, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2020.147237. Acesso em: 16 mar. 2025.
APA
Matielo, H. A., Gonçalves, E. S., Campos, M., Oliveira, V. R. da S., Toniolo, E. F., Alves, A. da S., et al. (2021). Electrical stimulation of the posterior insula induces mechanical analgesia in a rodent model of neuropathic pain by modulating GABAergic signaling and activity in the pain circuitry. Brain Research. doi:10.1016/j.brainres.2020.147237
NLM
Matielo HA, Gonçalves ES, Campos M, Oliveira VR da S, Toniolo EF, Alves A da S, Lebrun I, Andrade DCA de, Teixeira M jacobsen, Britto LRG de, Hamani C, Dale CS. Electrical stimulation of the posterior insula induces mechanical analgesia in a rodent model of neuropathic pain by modulating GABAergic signaling and activity in the pain circuitry [Internet]. Brain Research. 2021 ;[citado 2025 mar. 16 ] Available from: https://doi.org/10.1016/j.brainres.2020.147237
Vancouver
Matielo HA, Gonçalves ES, Campos M, Oliveira VR da S, Toniolo EF, Alves A da S, Lebrun I, Andrade DCA de, Teixeira M jacobsen, Britto LRG de, Hamani C, Dale CS. Electrical stimulation of the posterior insula induces mechanical analgesia in a rodent model of neuropathic pain by modulating GABAergic signaling and activity in the pain circuitry [Internet]. Brain Research. 2021 ;[citado 2025 mar. 16 ] Available from: https://doi.org/10.1016/j.brainres.2020.147237
Artigo de periodico
Association between thyroid function and Alzheimer’s disease: a systematic review (2021)
Figueroa, Paulina Belén Sepulveda ; Ferreira, Ana Flávia Fernandes ; Britto, Luiz Roberto Giorgetti de ; Doussoulin, Arlette Patricia ; Torrão, Andréa da Silva
Source: Metabolic Brain Disease. Unidade: ICB
Assunto: FISIOLOGIA
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FIGUEROA, Paulina Belén Sepulveda et al. Association between thyroid function and Alzheimer’s disease: a systematic review. Metabolic Brain Disease, p. 1-21, 2021Tradução . . Disponível em: https://doi.org/10.1007/s11011-021-00760-1. Acesso em: 16 mar. 2025.
APA
Figueroa, P. B. S., Ferreira, A. F. F., Britto, L. R. G. de, Doussoulin, A. P., & Torrão, A. da S. (2021). Association between thyroid function and Alzheimer’s disease: a systematic review. Metabolic Brain Disease, 1-21. doi:10.1007/s11011-021-00760-1
NLM
Figueroa PBS, Ferreira AFF, Britto LRG de, Doussoulin AP, Torrão A da S. Association between thyroid function and Alzheimer’s disease: a systematic review [Internet]. Metabolic Brain Disease. 2021 ; 1-21.[citado 2025 mar. 16 ] Available from: https://doi.org/10.1007/s11011-021-00760-1
Vancouver
Figueroa PBS, Ferreira AFF, Britto LRG de, Doussoulin AP, Torrão A da S. Association between thyroid function and Alzheimer’s disease: a systematic review [Internet]. Metabolic Brain Disease. 2021 ; 1-21.[citado 2025 mar. 16 ] Available from: https://doi.org/10.1007/s11011-021-00760-1
Artigo de periodico
Active lifestyle enhances protein expression profile in subjects with Lewy body pathology (2021)
Real, Caroline Cristiano; Suemoto, Cláudia Kimie; Binda, Karina Henrique; Grinberg, Lea Tenenholz; Pasqualucci, Carlos Augusto; Jacob-Filho, Wilson; Ferretti-Rebustini, Renata Eloah de Lucena ; Nitrini, Ricardo; Leite, Renata Elaine Paraizo; Britto, Luiz Roberto Giorgetti de
Source: Dementia & Neuropsychologia. Unidades: FM, EE, ICB
Subjects: MODO DE VIDA, ENVELHECIMENTO, AUTÓPSIA, DOENÇA DE PARKINSON, AGENTES DOPAMINÉRGICOS, ASTRÓCITOS
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REAL, Caroline Cristiano et al. Active lifestyle enhances protein expression profile in subjects with Lewy body pathology. Dementia & Neuropsychologia, v. 15, n. 1, p. 41-50, 2021Tradução . . Disponível em: https://doi.org/10.1590/1980-57642021dn15-010004. Acesso em: 16 mar. 2025.
APA
Real, C. C., Suemoto, C. K., Binda, K. H., Grinberg, L. T., Pasqualucci, C. A., Jacob-Filho, W., et al. (2021). Active lifestyle enhances protein expression profile in subjects with Lewy body pathology. Dementia & Neuropsychologia, 15( 1), 41-50. doi:10.1590/1980-57642021dn15-010004
NLM
Real CC, Suemoto CK, Binda KH, Grinberg LT, Pasqualucci CA, Jacob-Filho W, Ferretti-Rebustini RE de L, Nitrini R, Leite REP, Britto LRG de. Active lifestyle enhances protein expression profile in subjects with Lewy body pathology [Internet]. Dementia & Neuropsychologia. 2021 ; 15( 1): 41-50.[citado 2025 mar. 16 ] Available from: https://doi.org/10.1590/1980-57642021dn15-010004
Vancouver
Real CC, Suemoto CK, Binda KH, Grinberg LT, Pasqualucci CA, Jacob-Filho W, Ferretti-Rebustini RE de L, Nitrini R, Leite REP, Britto LRG de. Active lifestyle enhances protein expression profile in subjects with Lewy body pathology [Internet]. Dementia & Neuropsychologia. 2021 ; 15( 1): 41-50.[citado 2025 mar. 16 ] Available from: https://doi.org/10.1590/1980-57642021dn15-010004
Dissertação (Mestrado)
Efeito do cloreto de lítio no modelo de Doença de Parkinson por 6-OHDA (2021)
Silva, Ana Flavia Tostes da ; Britto, Luiz Roberto Giorgetti de (Orientador)
Unidade: ICB
Subjects: DOENÇA DE PARKINSON, CLORETO, LÍTIO, RECEPTORES DE DOPAMINA, EXPRESSÃO GÊNICA, REAÇÃO EM CADEIA POR POLIMERASE, IMMUNOBLOTTING
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ABNT
SILVA, Ana Flavia Tostes da. Efeito do cloreto de lítio no modelo de Doença de Parkinson por 6-OHDA. 2021. Dissertação (Mestrado) – Universidade de São Paulo, São Paulo, 2021. Disponível em: https://www.teses.usp.br/teses/disponiveis/42/42137/tde-14012022-232835/. Acesso em: 16 mar. 2025.
APA
Silva, A. F. T. da. (2021). Efeito do cloreto de lítio no modelo de Doença de Parkinson por 6-OHDA (Dissertação (Mestrado). Universidade de São Paulo, São Paulo. Recuperado de https://www.teses.usp.br/teses/disponiveis/42/42137/tde-14012022-232835/
NLM
Silva AFT da. Efeito do cloreto de lítio no modelo de Doença de Parkinson por 6-OHDA [Internet]. 2021 ;[citado 2025 mar. 16 ] Available from: https://www.teses.usp.br/teses/disponiveis/42/42137/tde-14012022-232835/
Vancouver
Silva AFT da. Efeito do cloreto de lítio no modelo de Doença de Parkinson por 6-OHDA [Internet]. 2021 ;[citado 2025 mar. 16 ] Available from: https://www.teses.usp.br/teses/disponiveis/42/42137/tde-14012022-232835/

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