Fonte: Journal of Clinical Immunology. Unidade: ICB
Assuntos: IMUNOLOGIA, DOENÇAS GENÉTICAS, MUTAÇÃO GENÉTICA, NEUTRÓFILOS, EXONS, OXIDAÇÃO
ABNT
HERNANDEZ, Marco Antonio Venancio et al. Duplication of exons 8–9 in NCF2 leading to incomplete clinical penetrance in Chronic Granulomatous Disease. Journal of Clinical Immunology. New York: Springer New York LLC. Disponível em: https://doi.org/10.1007/s10875-023-01624-2. Acesso em: 23 ago. 2024. , 2024APA
Hernandez, M. A. V., Flores, C. S., Juárez, M. J., Galicia, L. B., Espinosa-Padilla, S., & Condino Neto, A. (2024). Duplication of exons 8–9 in NCF2 leading to incomplete clinical penetrance in Chronic Granulomatous Disease. Journal of Clinical Immunology. New York: Springer New York LLC. doi:10.1007/s10875-023-01624-2NLM
Hernandez MAV, Flores CS, Juárez MJ, Galicia LB, Espinosa-Padilla S, Condino Neto A. Duplication of exons 8–9 in NCF2 leading to incomplete clinical penetrance in Chronic Granulomatous Disease [Internet]. Journal of Clinical Immunology. 2024 ; 44( 1): 4 .[citado 2024 ago. 23 ] Available from: https://doi.org/10.1007/s10875-023-01624-2Vancouver
Hernandez MAV, Flores CS, Juárez MJ, Galicia LB, Espinosa-Padilla S, Condino Neto A. Duplication of exons 8–9 in NCF2 leading to incomplete clinical penetrance in Chronic Granulomatous Disease [Internet]. Journal of Clinical Immunology. 2024 ; 44( 1): 4 .[citado 2024 ago. 23 ] Available from: https://doi.org/10.1007/s10875-023-01624-2