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Artigo de periodico
Local sustained delivery of anti IL-17A antibodies limits inflammatory bone loss in murine experimental periodontitis (2021)
Pacheco, Cinthia Mara da Fonseca ; Maltos, Katia L. M ; Shehabeldin, Mostafa S ; Thomas, Laura L ; Zhuang, Zhe ; Yoshizawa, Sayuri; Verdelis, Konstantinos; Gaffen, Sarah L ; Garlet, Gustavo Pompermaier ; Little, Steven ; Sfeir, Charles
Source: The Journal of Immunology. Unidade: FOB
Subjects: ANTICORPOS, DOENÇAS PERIODONTAIS, PERDA DE DENTE, CITOCINAS, OSSO E OSSOS
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ABNT
PACHECO, Cinthia Mara da Fonseca et al. Local sustained delivery of anti IL-17A antibodies limits inflammatory bone loss in murine experimental periodontitis. The Journal of Immunology, v. 206, n. 10, p. 2386-2392, 2021Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.2001432. Acesso em: 05 jul. 2022.
APA
Pacheco, C. M. da F., Maltos, K. L. M., Shehabeldin, M. S., Thomas, L. L., Zhuang, Z., Yoshizawa, S., et al. (2021). Local sustained delivery of anti IL-17A antibodies limits inflammatory bone loss in murine experimental periodontitis. The Journal of Immunology, 206( 10), 2386-2392. doi:10.4049/jimmunol.2001432
NLM
Pacheco CM da F, Maltos KLM, Shehabeldin MS, Thomas LL, Zhuang Z, Yoshizawa S, Verdelis K, Gaffen SL, Garlet GP, Little S, Sfeir C. Local sustained delivery of anti IL-17A antibodies limits inflammatory bone loss in murine experimental periodontitis [Internet]. The Journal of Immunology. 2021 ; 206( 10): 2386-2392.[citado 2022 jul. 05 ] Available from: https://doi.org/10.4049/jimmunol.2001432
Vancouver
Pacheco CM da F, Maltos KLM, Shehabeldin MS, Thomas LL, Zhuang Z, Yoshizawa S, Verdelis K, Gaffen SL, Garlet GP, Little S, Sfeir C. Local sustained delivery of anti IL-17A antibodies limits inflammatory bone loss in murine experimental periodontitis [Internet]. The Journal of Immunology. 2021 ; 206( 10): 2386-2392.[citado 2022 jul. 05 ] Available from: https://doi.org/10.4049/jimmunol.2001432
Artigo de periodico
Immune cell dynamics in rhesus macaques infected with a Brazilian strain of Zika virus (2017)
Silveira, Eduardo Lani Volpe da ; Rogers, Kenneth A; Gumber, Sanjeev; Amancha, Praveen; Xiao, Peng; Woollard, Shawna M; Byrareddy, Siddappa N; Teixeira, Mauro Martins; Villinger, Francois
Source: The Journal of Immunology. Unidade: FCF
Subjects: ZIKA VÍRUS, MACACOS
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ABNT
SILVEIRA, Eduardo Lani Volpe da et al. Immune cell dynamics in rhesus macaques infected with a Brazilian strain of Zika virus. The Journal of Immunology, v. 199, n. 3, p. 1003-1011, 2017Tradução . . Disponível em: https://doi-org.ez67.periodicos.capes.gov.br/10.4049/jimmunol.1700256. Acesso em: 05 jul. 2022.
APA
Silveira, E. L. V. da, Rogers, K. A., Gumber, S., Amancha, P., Xiao, P., Woollard, S. M., et al. (2017). Immune cell dynamics in rhesus macaques infected with a Brazilian strain of Zika virus. The Journal of Immunology, 199( 3), 1003-1011. doi:10.4049/jimmunol.1700256
NLM
Silveira ELV da, Rogers KA, Gumber S, Amancha P, Xiao P, Woollard SM, Byrareddy SN, Teixeira MM, Villinger F. Immune cell dynamics in rhesus macaques infected with a Brazilian strain of Zika virus [Internet]. The Journal of Immunology. 2017 ; 199( 3): 1003-1011.[citado 2022 jul. 05 ] Available from: https://doi-org.ez67.periodicos.capes.gov.br/10.4049/jimmunol.1700256
Vancouver
Silveira ELV da, Rogers KA, Gumber S, Amancha P, Xiao P, Woollard SM, Byrareddy SN, Teixeira MM, Villinger F. Immune cell dynamics in rhesus macaques infected with a Brazilian strain of Zika virus [Internet]. The Journal of Immunology. 2017 ; 199( 3): 1003-1011.[citado 2022 jul. 05 ] Available from: https://doi-org.ez67.periodicos.capes.gov.br/10.4049/jimmunol.1700256
Artigo de periodico
Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication (2017)
Lima Júnior, Djalma de Souza; Mineo, Tiago Wilson Patriarca; Calich, Vera Lúcia Garcia; Zamboni, Dario Simões
Source: The Journal of Immunology. Unidades: ICB, FMRP
Subjects: LEISHMANIA, FAGOCITOSE, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
LIMA JÚNIOR, Djalma de Souza et al. Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication. The Journal of Immunology, v. 199, n. 6, p. 2055-2068, 2017Tradução . . Disponível em: https://www.researchgate.net/publication/318985410_Dectin-1_Activation_during_Leishmania_amazonensis_Phagocytosis_Prompts_Syk-Dependent_Reactive_Oxygen_Species_Production_To_Trigger_Inflammasome_Assembly_and_Restriction_of_Parasite_Replication. Acesso em: 05 jul. 2022.
APA
Lima Júnior, D. de S., Mineo, T. W. P., Calich, V. L. G., & Zamboni, D. S. (2017). Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication. The Journal of Immunology, 199( 6), 2055-2068. doi:10.4049/jimmunol.1700258
NLM
Lima Júnior D de S, Mineo TWP, Calich VLG, Zamboni DS. Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication [Internet]. The Journal of Immunology. 2017 ; 199( 6): 2055-2068.[citado 2022 jul. 05 ] Available from: https://www.researchgate.net/publication/318985410_Dectin-1_Activation_during_Leishmania_amazonensis_Phagocytosis_Prompts_Syk-Dependent_Reactive_Oxygen_Species_Production_To_Trigger_Inflammasome_Assembly_and_Restriction_of_Parasite_Replication
Vancouver
Lima Júnior D de S, Mineo TWP, Calich VLG, Zamboni DS. Dectin-1 activation during Leishmania amazonensis phagocytosis prompts syk-dependent reactive oxygen species production to trigger inflammasome assembly and restriction of parasite replication [Internet]. The Journal of Immunology. 2017 ; 199( 6): 2055-2068.[citado 2022 jul. 05 ] Available from: https://www.researchgate.net/publication/318985410_Dectin-1_Activation_during_Leishmania_amazonensis_Phagocytosis_Prompts_Syk-Dependent_Reactive_Oxygen_Species_Production_To_Trigger_Inflammasome_Assembly_and_Restriction_of_Parasite_Replication
Artigo de periodico
Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo (2015)
Cerqueira, Daiane M.; Pereira, Marcelo S. F.; Silva, Alexandre L. N.; Cunha, Larissa D.; Zamboni, Dario Simões
Source: The Journal of Immunology. Unidade: FMRP
Subjects: BACTÉRIAS GRAM-NEGATIVAS, PNEUMONIA, MACRÓFAGOS, PROTEÍNAS DE LIGAÇÃO DO CÁLCIO, APOPTOSE
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ABNT
CERQUEIRA, Daiane M. et al. Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo. The Journal of Immunology, v. 195, n. 5, p. 2303-2311, 2015Tradução . . Disponível em: http://dx.doi.org/10.4049/jimmunol.1501223. Acesso em: 05 jul. 2022.
APA
Cerqueira, D. M., Pereira, M. S. F., Silva, A. L. N., Cunha, L. D., & Zamboni, D. S. (2015). Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo. The Journal of Immunology, 195( 5), 2303-2311. doi:10.4049/jimmunol.1501223
NLM
Cerqueira DM, Pereira MSF, Silva ALN, Cunha LD, Zamboni DS. Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo [Internet]. The Journal of Immunology. 2015 ; 195( 5): 2303-2311.[citado 2022 jul. 05 ] Available from: http://dx.doi.org/10.4049/jimmunol.1501223
Vancouver
Cerqueira DM, Pereira MSF, Silva ALN, Cunha LD, Zamboni DS. Caspase-1 but not caspase-11 is required for NLRC4-mediated pyroptosis and restriction of infection by flagellated Legionella species in mouse macrophages and in vivo [Internet]. The Journal of Immunology. 2015 ; 195( 5): 2303-2311.[citado 2022 jul. 05 ] Available from: http://dx.doi.org/10.4049/jimmunol.1501223
Artigo de periodico
IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection (2015)
Ketelut-Carneiro, Natália; Silva, Grace Kelly; Rocha, Fernanda Agostini; Milanezi, Cristiane Maria; Cavalcanti-Neto, Florêncio Figueiredo; Zamboni, Dario Simões; Silva, João Santana da
Source: The Journal of Immunology. Unidade: FMRP
Subjects: SISTEMA IMUNE, FUNGOS, INFLAMAÇÃO, MICOSES, PARACOCCIDIOIDES BRASILIENSIS
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ABNT
KETELUT-CARNEIRO, Natália et al. IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection. The Journal of Immunology, v. 194, n. 9, p. 4507-4517, 2015Tradução . . Disponível em: http://dx.doi.org/10.4049/jimmunol.1402321. Acesso em: 05 jul. 2022.
APA
Ketelut-Carneiro, N., Silva, G. K., Rocha, F. A., Milanezi, C. M., Cavalcanti-Neto, F. F., Zamboni, D. S., & Silva, J. S. da. (2015). IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection. The Journal of Immunology, 194( 9), 4507-4517. doi:10.4049/jimmunol.1402321
NLM
Ketelut-Carneiro N, Silva GK, Rocha FA, Milanezi CM, Cavalcanti-Neto FF, Zamboni DS, Silva JS da. IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection [Internet]. The Journal of Immunology. 2015 ; 194( 9): 4507-4517.[citado 2022 jul. 05 ] Available from: http://dx.doi.org/10.4049/jimmunol.1402321
Vancouver
Ketelut-Carneiro N, Silva GK, Rocha FA, Milanezi CM, Cavalcanti-Neto FF, Zamboni DS, Silva JS da. IL-18 triggered by the Nlrp3 inflammasome induces host innate resistance in a pulmonary model of fungal infection [Internet]. The Journal of Immunology. 2015 ; 194( 9): 4507-4517.[citado 2022 jul. 05 ] Available from: http://dx.doi.org/10.4049/jimmunol.1402321
Artigo de periodico-carta/editorial
Comment on "Impaired priming and activation of the neutrophil NADPH oxidase in patients with IRAK4 or NEMO deficiency" [Carta/Editorial] (2009)
Errante, Paolo Ruggero; Prando, Carolina; Bustamante, Jacinta; Aragão Filho, Walmir Cutrim; Pereira, Paulo Vitor Soelio; Rehder, Jussara; Casanova, Jean Laurent; Newburger, Peter; Condino Neto, Antonio
Source: The Journal of Immunology. Unidade: ICB
Subject: MICROBIOLOGIA
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ABNT
ERRANTE, Paolo Ruggero et al. Comment on "Impaired priming and activation of the neutrophil NADPH oxidase in patients with IRAK4 or NEMO deficiency" [Carta/Editorial]. The Journal of Immunology. Baltimore: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 05 jul. 2022. , 2009
APA
Errante, P. R., Prando, C., Bustamante, J., Aragão Filho, W. C., Pereira, P. V. S., Rehder, J., et al. (2009). Comment on "Impaired priming and activation of the neutrophil NADPH oxidase in patients with IRAK4 or NEMO deficiency" [Carta/Editorial]. The Journal of Immunology. Baltimore: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Errante PR, Prando C, Bustamante J, Aragão Filho WC, Pereira PVS, Rehder J, Casanova JL, Newburger P, Condino Neto A. Comment on "Impaired priming and activation of the neutrophil NADPH oxidase in patients with IRAK4 or NEMO deficiency" [Carta/Editorial]. The Journal of Immunology. 2009 ; 183( 6): 3559.[citado 2022 jul. 05 ]
Vancouver
Errante PR, Prando C, Bustamante J, Aragão Filho WC, Pereira PVS, Rehder J, Casanova JL, Newburger P, Condino Neto A. Comment on "Impaired priming and activation of the neutrophil NADPH oxidase in patients with IRAK4 or NEMO deficiency" [Carta/Editorial]. The Journal of Immunology. 2009 ; 183( 6): 3559.[citado 2022 jul. 05 ]
Artigo de periodico
Sialic acid residues are essential for the anaphylactic activity of murine IgG1 antibodies (2008)
Silva, Sandriana Ramos; Casabuono, Adriana; Jacysyn, Jacqueline F.; Favoretto, Bruna Cristina; Fernandes, Irene; Macedo, Mahasti Sahihi de; Couto, Alicia S.; Faquim-Mauro, Eliana L.
Source: The Journal of Immunology. Unidade: ICB
Subject: IMUNOLOGIA
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ABNT
SILVA, Sandriana Ramos et al. Sialic acid residues are essential for the anaphylactic activity of murine IgG1 antibodies. The Journal of Immunology, v. 181, n. 12, p. 8308-8314, 2008Tradução . . Acesso em: 05 jul. 2022.
APA
Silva, S. R., Casabuono, A., Jacysyn, J. F., Favoretto, B. C., Fernandes, I., Macedo, M. S. de, et al. (2008). Sialic acid residues are essential for the anaphylactic activity of murine IgG1 antibodies. The Journal of Immunology, 181( 12), 8308-8314.
NLM
Silva SR, Casabuono A, Jacysyn JF, Favoretto BC, Fernandes I, Macedo MS de, Couto AS, Faquim-Mauro EL. Sialic acid residues are essential for the anaphylactic activity of murine IgG1 antibodies. The Journal of Immunology. 2008 ; 181( 12): 8308-8314.[citado 2022 jul. 05 ]
Vancouver
Silva SR, Casabuono A, Jacysyn JF, Favoretto BC, Fernandes I, Macedo MS de, Couto AS, Faquim-Mauro EL. Sialic acid residues are essential for the anaphylactic activity of murine IgG1 antibodies. The Journal of Immunology. 2008 ; 181( 12): 8308-8314.[citado 2022 jul. 05 ]
Artigo de periodico
Gradual decline in malaria-specific memory T cell responses leads to failure to maintain long-term protective immunity to Plasmodium chabaudi as despite persistence of B cell memory and circulating antibody (2008)
Rosário, Ana Paula Freitas do; Muxel, Sandra Márcia; Rodríguez-Málaga, Sérgio Marcelo; Sardinha, Luiz Roberto; Zago, Cláudia Augusta; Castillo-Méndez, Sheyla Inés; Mosig, José Maria Alvarez; D'Império Lima, Maria Regina
Source: The Journal of Immunology. Unidade: ICB
Subject: IMUNOLOGIA
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ABNT
ROSÁRIO, Ana Paula Freitas do et al. Gradual decline in malaria-specific memory T cell responses leads to failure to maintain long-term protective immunity to Plasmodium chabaudi as despite persistence of B cell memory and circulating antibody. The Journal of Immunology, v. 181, n. 12, p. 8344-8355, 2008Tradução . . Acesso em: 05 jul. 2022.
APA
Rosário, A. P. F. do, Muxel, S. M., Rodríguez-Málaga, S. M., Sardinha, L. R., Zago, C. A., Castillo-Méndez, S. I., et al. (2008). Gradual decline in malaria-specific memory T cell responses leads to failure to maintain long-term protective immunity to Plasmodium chabaudi as despite persistence of B cell memory and circulating antibody. The Journal of Immunology, 181( 12), 8344-8355.
NLM
Rosário APF do, Muxel SM, Rodríguez-Málaga SM, Sardinha LR, Zago CA, Castillo-Méndez SI, Mosig JMA, D'Império Lima MR. Gradual decline in malaria-specific memory T cell responses leads to failure to maintain long-term protective immunity to Plasmodium chabaudi as despite persistence of B cell memory and circulating antibody. The Journal of Immunology. 2008 ; 181( 12): 8344-8355.[citado 2022 jul. 05 ]
Vancouver
Rosário APF do, Muxel SM, Rodríguez-Málaga SM, Sardinha LR, Zago CA, Castillo-Méndez SI, Mosig JMA, D'Império Lima MR. Gradual decline in malaria-specific memory T cell responses leads to failure to maintain long-term protective immunity to Plasmodium chabaudi as despite persistence of B cell memory and circulating antibody. The Journal of Immunology. 2008 ; 181( 12): 8344-8355.[citado 2022 jul. 05 ]
Artigo de periodico
CCR5-Dependent Regulatory T cell migration mediates fungal survival and severe immunosuppression (2008)
Moreira, Ana Paula; Cavassani, Karen Angelica; Tristão, Fabrine Sales Massafera; Campanelli, Ana Paula; Martinez, Roberto; Rossi, Marcos Antonio; Silva, João S.
Source: The Journal of Immunology. Unidades: FOB, FMRP
Subjects: IMUNOSSUPRESSÃO, FUNGOS, PARACOCCIDIOIDES BRASILIENSIS, LEUCÓCITOS
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ABNT
MOREIRA, Ana Paula et al. CCR5-Dependent Regulatory T cell migration mediates fungal survival and severe immunosuppression. The Journal of Immunology, v. 180, n. 5, p. 3049-3056, 2008Tradução . . Disponível em: http://www.jimmunol.org/cgi/content/full/180/5/3049. Acesso em: 05 jul. 2022.
APA
Moreira, A. P., Cavassani, K. A., Tristão, F. S. M., Campanelli, A. P., Martinez, R., Rossi, M. A., & Silva, J. S. (2008). CCR5-Dependent Regulatory T cell migration mediates fungal survival and severe immunosuppression. The Journal of Immunology, 180( 5), 3049-3056. Recuperado de http://www.jimmunol.org/cgi/content/full/180/5/3049
NLM
Moreira AP, Cavassani KA, Tristão FSM, Campanelli AP, Martinez R, Rossi MA, Silva JS. CCR5-Dependent Regulatory T cell migration mediates fungal survival and severe immunosuppression [Internet]. The Journal of Immunology. 2008 ; 180( 5): 3049-3056.[citado 2022 jul. 05 ] Available from: http://www.jimmunol.org/cgi/content/full/180/5/3049
Vancouver
Moreira AP, Cavassani KA, Tristão FSM, Campanelli AP, Martinez R, Rossi MA, Silva JS. CCR5-Dependent Regulatory T cell migration mediates fungal survival and severe immunosuppression [Internet]. The Journal of Immunology. 2008 ; 180( 5): 3049-3056.[citado 2022 jul. 05 ] Available from: http://www.jimmunol.org/cgi/content/full/180/5/3049
Artigo de periodico
Mimicry in recognition of cardiac myosin peptides by heart-intralesional Tcell clones from rheumatic heart disease (2006)
Faé, Kellen C.; Silva, Danielle D. da; Oshiro, Sandra E.; Tanaka, Ana C.; Pomerantzeff, Pablo; Douay, Cornine; Charron, Dominique; Toubert, Antoine; Cunningham, Madeleine; Kalil Filho, J.; Guilherme, Ana L.
Source: The Journal of Immunology. Unidade: FM
Subjects: MIOSINA, PEPTÍDEOS
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ABNT
FAÉ, Kellen C. et al. Mimicry in recognition of cardiac myosin peptides by heart-intralesional Tcell clones from rheumatic heart disease. The Journal of Immunology, v. 176, p. 5662-5670, 2006Tradução . . Acesso em: 05 jul. 2022.
APA
Faé, K. C., Silva, D. D. da, Oshiro, S. E., Tanaka, A. C., Pomerantzeff, P., Douay, C., et al. (2006). Mimicry in recognition of cardiac myosin peptides by heart-intralesional Tcell clones from rheumatic heart disease. The Journal of Immunology, 176, 5662-5670.
NLM
Faé KC, Silva DD da, Oshiro SE, Tanaka AC, Pomerantzeff P, Douay C, Charron D, Toubert A, Cunningham M, Kalil Filho J, Guilherme AL. Mimicry in recognition of cardiac myosin peptides by heart-intralesional Tcell clones from rheumatic heart disease. The Journal of Immunology. 2006 ; 176 5662-5670.[citado 2022 jul. 05 ]
Vancouver
Faé KC, Silva DD da, Oshiro SE, Tanaka AC, Pomerantzeff P, Douay C, Charron D, Toubert A, Cunningham M, Kalil Filho J, Guilherme AL. Mimicry in recognition of cardiac myosin peptides by heart-intralesional Tcell clones from rheumatic heart disease. The Journal of Immunology. 2006 ; 176 5662-5670.[citado 2022 jul. 05 ]
Artigo de periodico
Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis (2006)
Moreno, Susana E.; Alves-Filho, José C.; Rios-Santos, Fabrício; Silva, João Santana da; Ferreira, Sérgio Henrique; Cunha, Fernando de Queiroz; Teixeira, Mauro M.
Source: The Journal of Immunology. Unidade: FMRP
Subjects: IMUNOLOGIA, SISTEMA IMUNE, SEPSE
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ABNT
MORENO, Susana E. et al. Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis. The Journal of Immunology, v. 177, p. 1264-1271, 2006Tradução . . Acesso em: 05 jul. 2022.
APA
Moreno, S. E., Alves-Filho, J. C., Rios-Santos, F., Silva, J. S. da, Ferreira, S. H., Cunha, F. de Q., & Teixeira, M. M. (2006). Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis. The Journal of Immunology, 177, 1264-1271.
NLM
Moreno SE, Alves-Filho JC, Rios-Santos F, Silva JS da, Ferreira SH, Cunha F de Q, Teixeira MM. Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis. The Journal of Immunology. 2006 ; 177 1264-1271.[citado 2022 jul. 05 ]
Vancouver
Moreno SE, Alves-Filho JC, Rios-Santos F, Silva JS da, Ferreira SH, Cunha F de Q, Teixeira MM. Signaling via platelet-activating factor receptors accounts for the impairment of neutrophil migration in polymicrobial sepsis. The Journal of Immunology. 2006 ; 177 1264-1271.[citado 2022 jul. 05 ]
Artigo de periodico
Leukotrienes play a role in the control of parasite burden in murine strongyloidiasis (2005)
Machado, Eleuza Rodrigues; Ueta, Marlene T.; Lourenço, Elaine Vicente; Anibal, Fernanda F.; Sorgi, Carlos Artério; Soares, Edson Garcia; Roque-Barreira, Maria Cristina; Medeiros, Alexandra Ivo; Faccioli, Lúcia Helena
Source: The Journal of Immunology. Unidades: FCFRP, FMRP
Subject: IMUNOLOGIA
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ABNT
MACHADO, Eleuza Rodrigues et al. Leukotrienes play a role in the control of parasite burden in murine strongyloidiasis. The Journal of Immunology, v. 175, p. 3892-3899, 2005Tradução . . Acesso em: 05 jul. 2022.
APA
Machado, E. R., Ueta, M. T., Lourenço, E. V., Anibal, F. F., Sorgi, C. A., Soares, E. G., et al. (2005). Leukotrienes play a role in the control of parasite burden in murine strongyloidiasis. The Journal of Immunology, 175, 3892-3899.
NLM
Machado ER, Ueta MT, Lourenço EV, Anibal FF, Sorgi CA, Soares EG, Roque-Barreira MC, Medeiros AI, Faccioli LH. Leukotrienes play a role in the control of parasite burden in murine strongyloidiasis. The Journal of Immunology. 2005 ; 175 3892-3899.[citado 2022 jul. 05 ]
Vancouver
Machado ER, Ueta MT, Lourenço EV, Anibal FF, Sorgi CA, Soares EG, Roque-Barreira MC, Medeiros AI, Faccioli LH. Leukotrienes play a role in the control of parasite burden in murine strongyloidiasis. The Journal of Immunology. 2005 ; 175 3892-3899.[citado 2022 jul. 05 ]
Artigo de periodico
Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi (2004)
Michailowsky, Vladimir; Celes, Mara Rúbia Nunes; Marino, Ana P.; Silva, Andréa A.; Vieira, Leda Q.; Rossi, Marcos Antonio; Gazzinelli, Ricardo T.; Lannes-Vieira, Joseli; Silva, João Santana da
Source: The Journal of Immunology. Unidade: FMRP
Subject: TRYPANOSOMA CRUZI (IMUNOLOGIA)
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ABNT
MICHAILOWSKY, Vladimir et al. Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi. The Journal of Immunology, v. 173, p. 463-470, 2004Tradução . . Acesso em: 05 jul. 2022.
APA
Michailowsky, V., Celes, M. R. N., Marino, A. P., Silva, A. A., Vieira, L. Q., Rossi, M. A., et al. (2004). Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi. The Journal of Immunology, 173, 463-470.
NLM
Michailowsky V, Celes MRN, Marino AP, Silva AA, Vieira LQ, Rossi MA, Gazzinelli RT, Lannes-Vieira J, Silva JS da. Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi. The Journal of Immunology. 2004 ; 173 463-470.[citado 2022 jul. 05 ]
Vancouver
Michailowsky V, Celes MRN, Marino AP, Silva AA, Vieira LQ, Rossi MA, Gazzinelli RT, Lannes-Vieira J, Silva JS da. Intercellular adhesion molecule 1 deficieney leads to impaired recruitment of T lymphocytes and enhanced host susceptibility to infection with Trypanosoma cruzi. The Journal of Immunology. 2004 ; 173 463-470.[citado 2022 jul. 05 ]
Artigo de periodico
Bacterial lipopolysaccharide signaling through toll-like receptor 4 suprresses asthma-like responses via nitric oxide synthase 2 activity (2003)
Rodríguez, Dunia; Keller, Alexandre de Castro; Faquim-Mauro, Eliana Lima; Macedo, Mahasti Sahihi de; Cunha, Fernando de Queiroz; Lefort, Jean; Vargaftig, B. Boris; Russo, Momtchilo
Source: The Journal of Immunology. Unidades: FMRP, ICB
Subject: ASMA (IMUNOLOGIA)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRÍGUEZ, Dunia et al. Bacterial lipopolysaccharide signaling through toll-like receptor 4 suprresses asthma-like responses via nitric oxide synthase 2 activity. The Journal of Immunology, v. 171, n. 2, p. 1001-1008, 2003Tradução . . Acesso em: 05 jul. 2022.
APA
Rodríguez, D., Keller, A. de C., Faquim-Mauro, E. L., Macedo, M. S. de, Cunha, F. de Q., Lefort, J., et al. (2003). Bacterial lipopolysaccharide signaling through toll-like receptor 4 suprresses asthma-like responses via nitric oxide synthase 2 activity. The Journal of Immunology, 171( 2), 1001-1008.
NLM
Rodríguez D, Keller A de C, Faquim-Mauro EL, Macedo MS de, Cunha F de Q, Lefort J, Vargaftig BB, Russo M. Bacterial lipopolysaccharide signaling through toll-like receptor 4 suprresses asthma-like responses via nitric oxide synthase 2 activity. The Journal of Immunology. 2003 ; 171( 2): 1001-1008.[citado 2022 jul. 05 ]
Vancouver
Rodríguez D, Keller A de C, Faquim-Mauro EL, Macedo MS de, Cunha F de Q, Lefort J, Vargaftig BB, Russo M. Bacterial lipopolysaccharide signaling through toll-like receptor 4 suprresses asthma-like responses via nitric oxide synthase 2 activity. The Journal of Immunology. 2003 ; 171( 2): 1001-1008.[citado 2022 jul. 05 ]
Artigo de periodico
IL-18 enhances collagen-induced arthritis by recruiting neutrophils via TNF-'alfa' and leukotriene 'B IND.4 POT.1' (2003)
Cannetti, Claudio A.; Leung, Bernard P.; Culshaw, Shauna; McInnes, Iain B.; Cunha, Fernando de Queiroz; Liew, Foo Y.
Source: The Journal of Immunology. Unidade: FMRP
Subjects: IMUNOLOGIA, FARMACOLOGIA EXPERIMENTAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CANNETTI, Claudio A. et al. IL-18 enhances collagen-induced arthritis by recruiting neutrophils via TNF-'alfa' and leukotriene 'B IND.4 POT.1'. The Journal of Immunology, v. 171, n. 2, p. 1009-1015, 2003Tradução . . Acesso em: 05 jul. 2022.
APA
Cannetti, C. A., Leung, B. P., Culshaw, S., McInnes, I. B., Cunha, F. de Q., & Liew, F. Y. (2003). IL-18 enhances collagen-induced arthritis by recruiting neutrophils via TNF-'alfa' and leukotriene 'B IND.4 POT.1'. The Journal of Immunology, 171( 2), 1009-1015.
NLM
Cannetti CA, Leung BP, Culshaw S, McInnes IB, Cunha F de Q, Liew FY. IL-18 enhances collagen-induced arthritis by recruiting neutrophils via TNF-'alfa' and leukotriene 'B IND.4 POT.1'. The Journal of Immunology. 2003 ; 171( 2): 1009-1015.[citado 2022 jul. 05 ]
Vancouver
Cannetti CA, Leung BP, Culshaw S, McInnes IB, Cunha F de Q, Liew FY. IL-18 enhances collagen-induced arthritis by recruiting neutrophils via TNF-'alfa' and leukotriene 'B IND.4 POT.1'. The Journal of Immunology. 2003 ; 171( 2): 1009-1015.[citado 2022 jul. 05 ]
Artigo de periodico
A Role for IL-18 un neutrophil activation (2001)
Leung, Bernard P.; Culshaw, Shauna; Gracie, J. Alastair; Hunter, David; Canetti, Cláudio de Azevedo; Campbell, Carol; Cunha, Fernando de Queiroz; Liew, Foo Y.; McInnes, Iain B.
Source: The Journal of Immunology. Unidade: FMRP
Subjects: FARMACOLOGIA, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEUNG, Bernard P. et al. A Role for IL-18 un neutrophil activation. The Journal of Immunology, v. 167, p. 2879-2886, 2001Tradução . . Acesso em: 05 jul. 2022.
APA
Leung, B. P., Culshaw, S., Gracie, J. A., Hunter, D., Canetti, C. de A., Campbell, C., et al. (2001). A Role for IL-18 un neutrophil activation. The Journal of Immunology, 167, 2879-2886.
NLM
Leung BP, Culshaw S, Gracie JA, Hunter D, Canetti C de A, Campbell C, Cunha F de Q, Liew FY, McInnes IB. A Role for IL-18 un neutrophil activation. The Journal of Immunology. 2001 ; 167 2879-2886.[citado 2022 jul. 05 ]
Vancouver
Leung BP, Culshaw S, Gracie JA, Hunter D, Canetti C de A, Campbell C, Cunha F de Q, Liew FY, McInnes IB. A Role for IL-18 un neutrophil activation. The Journal of Immunology. 2001 ; 167 2879-2886.[citado 2022 jul. 05 ]
Artigo de periodico
Flagellin, a novel mediator of Salmonella - induced epithelial activation and systemic inflammation: IkB'alfa' degradation, induction of Nitric Oxide synthase, induction of proinflammatory mediators, and cardiovarcular dysfunction (2001)
Eaves-Pyles, Tonyia; Soriano, Francisco Garcia; Murthy, Kanneganti; Liaudet, Lucas; Virág, László; Ross, Gary; Szabó, Csaba; Salzman, Andrew
Source: The Journal of Immunology. Unidade: FM
Subjects: ÓXIDO NÍTRICO, INFLAMAÇÃO, SALMONELLA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
EAVES-PYLES, Tonyia et al. Flagellin, a novel mediator of Salmonella - induced epithelial activation and systemic inflammation: IkB'alfa' degradation, induction of Nitric Oxide synthase, induction of proinflammatory mediators, and cardiovarcular dysfunction. The Journal of Immunology, p. 1248-1259, 2001Tradução . . Acesso em: 05 jul. 2022.
APA
Eaves-Pyles, T., Soriano, F. G., Murthy, K., Liaudet, L., Virág, L., Ross, G., et al. (2001). Flagellin, a novel mediator of Salmonella - induced epithelial activation and systemic inflammation: IkB'alfa' degradation, induction of Nitric Oxide synthase, induction of proinflammatory mediators, and cardiovarcular dysfunction. The Journal of Immunology, 1248-1259.
NLM
Eaves-Pyles T, Soriano FG, Murthy K, Liaudet L, Virág L, Ross G, Szabó C, Salzman A. Flagellin, a novel mediator of Salmonella - induced epithelial activation and systemic inflammation: IkB'alfa' degradation, induction of Nitric Oxide synthase, induction of proinflammatory mediators, and cardiovarcular dysfunction. The Journal of Immunology. 2001 ; 1248-1259.[citado 2022 jul. 05 ]
Vancouver
Eaves-Pyles T, Soriano FG, Murthy K, Liaudet L, Virág L, Ross G, Szabó C, Salzman A. Flagellin, a novel mediator of Salmonella - induced epithelial activation and systemic inflammation: IkB'alfa' degradation, induction of Nitric Oxide synthase, induction of proinflammatory mediators, and cardiovarcular dysfunction. The Journal of Immunology. 2001 ; 1248-1259.[citado 2022 jul. 05 ]

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