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Artigo de periodico
Immunoregulatory macrophages induced by mycobacterial nonpolar lipids (2025)
Petrilli, Jéssica Dias; Estevão, Paulo ; Araújo, Luana Evangelista De; Muller, Igor; Yoshinaga, Marcos Yukio ; Ramos, Pablo Ivan P. ; Chaves Filho, Adriano de Britto ; Horta, Thainá; Sorgi, Carlos Arterio ; Miyamoto, Sayuri ; Riley, Lee; Arruda, Sérgio; Queiroz, Adriano
Source: Journal of Immunology. Unidades: FFCLRP, IQ
Subjects: MACRÓFAGOS, MYCOBACTERIUM TUBERCULOSIS, LIPÍDEOS
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PETRILLI, Jéssica Dias et al. Immunoregulatory macrophages induced by mycobacterial nonpolar lipids. Journal of Immunology, v. 214, n. 5, p. 1059–1070, 2025Tradução . . Disponível em: https://dx.doi.org/10.1093/jimmun/vkae058. Acesso em: 15 jul. 2025.
APA
Petrilli, J. D., Estevão, P., Araújo, L. E. D., Muller, I., Yoshinaga, M. Y., Ramos, P. I. P., et al. (2025). Immunoregulatory macrophages induced by mycobacterial nonpolar lipids. Journal of Immunology, 214( 5), 1059–1070. doi:10.1093/jimmun/vkae058
NLM
Petrilli JD, Estevão P, Araújo LED, Muller I, Yoshinaga MY, Ramos PIP, Chaves Filho A de B, Horta T, Sorgi CA, Miyamoto S, Riley L, Arruda S, Queiroz A. Immunoregulatory macrophages induced by mycobacterial nonpolar lipids [Internet]. Journal of Immunology. 2025 ; 214( 5): 1059–1070.[citado 2025 jul. 15 ] Available from: https://dx.doi.org/10.1093/jimmun/vkae058
Vancouver
Petrilli JD, Estevão P, Araújo LED, Muller I, Yoshinaga MY, Ramos PIP, Chaves Filho A de B, Horta T, Sorgi CA, Miyamoto S, Riley L, Arruda S, Queiroz A. Immunoregulatory macrophages induced by mycobacterial nonpolar lipids [Internet]. Journal of Immunology. 2025 ; 214( 5): 1059–1070.[citado 2025 jul. 15 ] Available from: https://dx.doi.org/10.1093/jimmun/vkae058
Trabalho de evento-resumo periodico
T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections (2024)
Santiago-Carvalho, Igor ; Almeida-Santos, Gislane ; Macêdo, Bruna de Gois ; Barbosa-Bomfim, Caio César ; Almeida, Fabricio Moreira ; Cione, Marcos Vinícios Pinheiro ; Lassounskaia, Elena ; Silva, Henrique Borges da ; Lima, Maria Regina D'Império
Source: Journal of Immunology. Conference titles: IMMUNOLOGY2024™ Meeting Abstracts. The Annual Meeting of the American Association of Immunologists. Emerging Concepts in Regulation of Autoimmune T Cells. Unidade: ICB
Subjects: IMUNOLOGIA, IMUNOLOGIA, PNEUMOPATIAS, TUBERCULOSE PULMONAR, MODELOS ANIMAIS DE DOENÇAS, QUIMIOCINAS, PULMÃO, POLIMORFISMO, CAMUNDONGOS
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SANTIAGO-CARVALHO, Igor et al. T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections. Journal of Immunology. Cary, NC: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.4049/jimmunol.212.supp.0361.5171. Acesso em: 15 jul. 2025. , 2024
APA
Santiago-Carvalho, I., Almeida-Santos, G., Macêdo, B. de G., Barbosa-Bomfim, C. C., Almeida, F. M., Cione, M. V. P., et al. (2024). T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections. Journal of Immunology. Cary, NC: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.4049/jimmunol.212.supp.0361.5171
NLM
Santiago-Carvalho I, Almeida-Santos G, Macêdo B de G, Barbosa-Bomfim CC, Almeida FM, Cione MVP, Lassounskaia E, Silva HB da, Lima MRD'I. T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections [Internet]. Journal of Immunology. 2024 ; 212( 1): 0361_5171.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.212.supp.0361.5171
Vancouver
Santiago-Carvalho I, Almeida-Santos G, Macêdo B de G, Barbosa-Bomfim CC, Almeida FM, Cione MVP, Lassounskaia E, Silva HB da, Lima MRD'I. T cell-specific P2RX7 favors lung parenchymal CD4+ T cell accumulation in response to severe lung infections [Internet]. Journal of Immunology. 2024 ; 212( 1): 0361_5171.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.212.supp.0361.5171
Artigo de periodico
Bacillus Calmette-Guérin-Trained macrophages elicit a protective inflammatory response against the pathogenic bacteria Brucella abortus (2023)
Araujo, Ana Carolina V. S. C. de ; Queiroz, Nina M. G. P. de ; Marinho, Fábio V. ; Oliveira, Sergio C.
Source: Journal of Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, CAMUNDONGOS, INFLAMAÇÃO, MACRÓFAGOS, BRUCELOSE, INFECÇÕES BACTERIANAS GRAM-NEGATIVAS, MEDULA ÓSSEA DE ANIMAL, MODELOS ANIMAIS DE DOENÇAS
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ARAUJO, Ana Carolina V. S. C. de et al. Bacillus Calmette-Guérin-Trained macrophages elicit a protective inflammatory response against the pathogenic bacteria Brucella abortus. Journal of Immunology, v. 211, n. 5, p. 791-803, 2023Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.2200642. Acesso em: 15 jul. 2025.
APA
Araujo, A. C. V. S. C. de, Queiroz, N. M. G. P. de, Marinho, F. V., & Oliveira, S. C. (2023). Bacillus Calmette-Guérin-Trained macrophages elicit a protective inflammatory response against the pathogenic bacteria Brucella abortus. Journal of Immunology, 211( 5), 791-803. doi:10.4049/jimmunol.2200642
NLM
Araujo ACVSC de, Queiroz NMGP de, Marinho FV, Oliveira SC. Bacillus Calmette-Guérin-Trained macrophages elicit a protective inflammatory response against the pathogenic bacteria Brucella abortus [Internet]. Journal of Immunology. 2023 ; 211( 5): 791-803.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.2200642
Vancouver
Araujo ACVSC de, Queiroz NMGP de, Marinho FV, Oliveira SC. Bacillus Calmette-Guérin-Trained macrophages elicit a protective inflammatory response against the pathogenic bacteria Brucella abortus [Internet]. Journal of Immunology. 2023 ; 211( 5): 791-803.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.2200642
Artigo de periodico
Oral exposure to Escherichia colitoxin protects against lung allergen-induced inflammation (2023)
Salgado, Caio Loureiro ; Ayupe, Marina Caçador ; Rodrigues, Juliana Falcão ; Silva, Luisa Menezes ; Oliveira, Bernardo de Castro ; Moreira, Francielly ; Gonçalves, Leonardo Mandu ; Silva, Guilherme William da ; Ramirez, Jofer Andree Zamame ; Silva, Henrique Borges da ; Ferreira, Luis Carlos de Souza ; Lima, Maria Regina D'Império ; Fonseca, Denise Morais da
Source: Journal of Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, IMUNOLOGIA CELULAR, CÉLULAS SANGUÍNEAS, MUCOSA ORAL, CAMUNDONGOS, HIPERSENSIBILIDADE RESPIRATÓRIA, ALÉRGENOS, PULMÃO, ESCHERICHIA COLI, INFLAMAÇÃO
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ABNT
SALGADO, Caio Loureiro et al. Oral exposure to Escherichia colitoxin protects against lung allergen-induced inflammation. Journal of Immunology, v. 210, n. Supl. 1, p. 67.29, 2023Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.210.Supp.67.29. Acesso em: 15 jul. 2025.
APA
Salgado, C. L., Ayupe, M. C., Rodrigues, J. F., Silva, L. M., Oliveira, B. de C., Moreira, F., et al. (2023). Oral exposure to Escherichia colitoxin protects against lung allergen-induced inflammation. Journal of Immunology, 210( Supl. 1), 67.29. doi:10.4049/jimmunol.210.Supp.67.29
NLM
Salgado CL, Ayupe MC, Rodrigues JF, Silva LM, Oliveira B de C, Moreira F, Gonçalves LM, Silva GW da, Ramirez JAZ, Silva HB da, Ferreira LC de S, Lima MRD'I, Fonseca DM da. Oral exposure to Escherichia colitoxin protects against lung allergen-induced inflammation [Internet]. Journal of Immunology. 2023 ; 210( Supl. 1): 67.29.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.210.Supp.67.29.
Vancouver
Salgado CL, Ayupe MC, Rodrigues JF, Silva LM, Oliveira B de C, Moreira F, Gonçalves LM, Silva GW da, Ramirez JAZ, Silva HB da, Ferreira LC de S, Lima MRD'I, Fonseca DM da. Oral exposure to Escherichia colitoxin protects against lung allergen-induced inflammation [Internet]. Journal of Immunology. 2023 ; 210( Supl. 1): 67.29.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.210.Supp.67.29.
Artigo de periodico
IFNs reset the differential capacity of human monocyte subsets to produce IL-12 in response to microbial stimulation (2021)
Amancio, Alice Muglia; Mittereder, Lara; Carletti, Alexie; Tosh, Kevin W.; Green, Daniel; Antonelli, Lis R.; Gazzinelli, Ricardo Tostes; Sher, Alan; Jankovic, Dragana
Source: Journal of Immunology. Unidade: FMRP
Subjects: DIFERENCIAÇÃO CELULAR, CÉLULAS CULTIVADAS, INFLAMAÇÃO, INTERFERONS, RECEPTORES, LIPOPOLISSACARÍDEOS, MONÓCITOS, CULTURA DE CÉLULAS, TOXOPLASMOSE
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ABNT
AMANCIO, Alice Muglia et al. IFNs reset the differential capacity of human monocyte subsets to produce IL-12 in response to microbial stimulation. Journal of Immunology, v. 206, n. 7, p. 1642-1652, 2021Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.2001194. Acesso em: 15 jul. 2025.
APA
Amancio, A. M., Mittereder, L., Carletti, A., Tosh, K. W., Green, D., Antonelli, L. R., et al. (2021). IFNs reset the differential capacity of human monocyte subsets to produce IL-12 in response to microbial stimulation. Journal of Immunology, 206( 7), 1642-1652. doi:10.4049/jimmunol.2001194
NLM
Amancio AM, Mittereder L, Carletti A, Tosh KW, Green D, Antonelli LR, Gazzinelli RT, Sher A, Jankovic D. IFNs reset the differential capacity of human monocyte subsets to produce IL-12 in response to microbial stimulation [Internet]. Journal of Immunology. 2021 ; 206( 7): 1642-1652.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.2001194
Vancouver
Amancio AM, Mittereder L, Carletti A, Tosh KW, Green D, Antonelli LR, Gazzinelli RT, Sher A, Jankovic D. IFNs reset the differential capacity of human monocyte subsets to produce IL-12 in response to microbial stimulation [Internet]. Journal of Immunology. 2021 ; 206( 7): 1642-1652.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.2001194
Artigo de periodico-divulgacao
Characterization of notch and toll-like receptor interactions in macrophages against Paracoccidioides brasiliensis (2016)
Romera, Lavínia Maria Dal'Mas; Jannuzzi, Grasielle Pereira; Almeida, Sandro Rogério de
Source: Journal of Immunology. Unidade: FCF
Subjects: MACRÓFAGOS, PARACOCCIDIOIDES BRASILIENSIS
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ABNT
ROMERA, Lavínia Maria Dal'Mas e JANNUZZI, Grasielle Pereira e ALMEIDA, Sandro Rogério de. Characterization of notch and toll-like receptor interactions in macrophages against Paracoccidioides brasiliensis. Journal of Immunology. Bethesda: Faculdade de Ciências Farmacêuticas, Universidade de São Paulo. Disponível em: http://www.jimmunol.org/content/196/1_Supplement/205.16. Acesso em: 15 jul. 2025. , 2016
APA
Romera, L. M. D. 'M., Jannuzzi, G. P., & Almeida, S. R. de. (2016). Characterization of notch and toll-like receptor interactions in macrophages against Paracoccidioides brasiliensis. Journal of Immunology. Bethesda: Faculdade de Ciências Farmacêuticas, Universidade de São Paulo. Recuperado de http://www.jimmunol.org/content/196/1_Supplement/205.16
NLM
Romera LMD'M, Jannuzzi GP, Almeida SR de. Characterization of notch and toll-like receptor interactions in macrophages against Paracoccidioides brasiliensis [Internet]. Journal of Immunology. 2016 ; 196( 1): 205.16.[citado 2025 jul. 15 ] Available from: http://www.jimmunol.org/content/196/1_Supplement/205.16
Vancouver
Romera LMD'M, Jannuzzi GP, Almeida SR de. Characterization of notch and toll-like receptor interactions in macrophages against Paracoccidioides brasiliensis [Internet]. Journal of Immunology. 2016 ; 196( 1): 205.16.[citado 2025 jul. 15 ] Available from: http://www.jimmunol.org/content/196/1_Supplement/205.16
Artigo de periodico
Dendritic cells and their multiple roles during malaria infection (2016)
Amorim, Kelly N. S.; Chagas, Daniele C. G.; Sulczewski, Fernando B.; Boscardin, Silvia Beatriz
Source: Journal of Immunology. Unidade: ICB
Assunto: PARASITOLOGIA
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AMORIM, Kelly N. S. et al. Dendritic cells and their multiple roles during malaria infection. Journal of Immunology, v. 2016, p. 1-11, 2016Tradução . . Disponível em: https://doi.org/10.1155/2016/2926436. Acesso em: 15 jul. 2025.
APA
Amorim, K. N. S., Chagas, D. C. G., Sulczewski, F. B., & Boscardin, S. B. (2016). Dendritic cells and their multiple roles during malaria infection. Journal of Immunology, 2016, 1-11. doi:10.1155/2016/2926436
NLM
Amorim KNS, Chagas DCG, Sulczewski FB, Boscardin SB. Dendritic cells and their multiple roles during malaria infection [Internet]. Journal of Immunology. 2016 ; 2016 1-11.[citado 2025 jul. 15 ] Available from: https://doi.org/10.1155/2016/2926436
Vancouver
Amorim KNS, Chagas DCG, Sulczewski FB, Boscardin SB. Dendritic cells and their multiple roles during malaria infection [Internet]. Journal of Immunology. 2016 ; 2016 1-11.[citado 2025 jul. 15 ] Available from: https://doi.org/10.1155/2016/2926436
Artigo de periodico
Adjuvants: classification, Modus Operandi, and licensing. (2016)
Apostólico, Juliana de Souza; Lunardelli, Victória Alves Santos; Coirada, Fernanda Caroline; Boscardin, Silvia Beatriz ; Rosa, Daniela Santoro
Source: Journal of Immunology. Unidade: ICB
Assunto: PARASITOLOGIA
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ABNT
APOSTÓLICO, Juliana de Souza et al. Adjuvants: classification, Modus Operandi, and licensing. Journal of Immunology, v. 2016, p. 1-16, 2016Tradução . . Disponível em: https://doi.org/10.1155/2016/1459394. Acesso em: 15 jul. 2025.
APA
Apostólico, J. de S., Lunardelli, V. A. S., Coirada, F. C., Boscardin, S. B., & Rosa, D. S. (2016). Adjuvants: classification, Modus Operandi, and licensing. Journal of Immunology, 2016, 1-16. doi:10.1155/2016/1459394
NLM
Apostólico J de S, Lunardelli VAS, Coirada FC, Boscardin SB, Rosa DS. Adjuvants: classification, Modus Operandi, and licensing. [Internet]. Journal of Immunology. 2016 ; 2016 1-16.[citado 2025 jul. 15 ] Available from: https://doi.org/10.1155/2016/1459394
Vancouver
Apostólico J de S, Lunardelli VAS, Coirada FC, Boscardin SB, Rosa DS. Adjuvants: classification, Modus Operandi, and licensing. [Internet]. Journal of Immunology. 2016 ; 2016 1-16.[citado 2025 jul. 15 ] Available from: https://doi.org/10.1155/2016/1459394
Artigo de periodico
Inhibiting STAT5 by the BET bromodomain inhibitor JQ1 disrupts human dendritic cell maturation (2015)
Toniolo, Patricia Argenta.; Liu, Suhu; Yeh, Jennifer E.; Moraes-Vieira; Walker, Sarah R.; Vafaizadeh, Vida; Barbuto, José Alexandre Marzagão ; Frank, David A.
Source: Journal of Immunology. Unidade: ICB
Assunto: IMUNOLOGIA
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TONIOLO, Patricia Argenta. et al. Inhibiting STAT5 by the BET bromodomain inhibitor JQ1 disrupts human dendritic cell maturation. Journal of Immunology, v. 194, n. 7, p. 3180-3190, 2015Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1401635. Acesso em: 15 jul. 2025.
APA
Toniolo, P. A., Liu, S., Yeh, J. E., Moraes-Vieira,, Walker, S. R., Vafaizadeh, V., et al. (2015). Inhibiting STAT5 by the BET bromodomain inhibitor JQ1 disrupts human dendritic cell maturation. Journal of Immunology, 194( 7), 3180-3190. doi:10.4049/jimmunol.1401635
NLM
Toniolo PA, Liu S, Yeh JE, Moraes-Vieira, Walker SR, Vafaizadeh V, Barbuto JAM, Frank DA. Inhibiting STAT5 by the BET bromodomain inhibitor JQ1 disrupts human dendritic cell maturation [Internet]. Journal of Immunology. 2015 ; 194( 7): 3180-3190.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1401635
Vancouver
Toniolo PA, Liu S, Yeh JE, Moraes-Vieira, Walker SR, Vafaizadeh V, Barbuto JAM, Frank DA. Inhibiting STAT5 by the BET bromodomain inhibitor JQ1 disrupts human dendritic cell maturation [Internet]. Journal of Immunology. 2015 ; 194( 7): 3180-3190.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1401635
Artigo de periodico
Integrated innate mechanisms involved in airway allergic inflammation to the serine protease subtilisin (2015)
Florsheim, Esther; Yu, Shuang; Bargatto, Ivan; Faustino, Lucas; Gomes, Eliane; Ramos, Rodrigo Nalio; Barbuto, José Alexandre Marzagão ; Medzhitov, Ruslan; Russo, Momtchilo
Source: Journal of Immunology. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
FLORSHEIM, Esther et al. Integrated innate mechanisms involved in airway allergic inflammation to the serine protease subtilisin. Journal of Immunology, v. 194, n. 10, p. 4621-4630, 2015Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1402493. Acesso em: 15 jul. 2025.
APA
Florsheim, E., Yu, S., Bargatto, I., Faustino, L., Gomes, E., Ramos, R. N., et al. (2015). Integrated innate mechanisms involved in airway allergic inflammation to the serine protease subtilisin. Journal of Immunology, 194( 10), 4621-4630. doi:10.4049/jimmunol.1402493
NLM
Florsheim E, Yu S, Bargatto I, Faustino L, Gomes E, Ramos RN, Barbuto JAM, Medzhitov R, Russo M. Integrated innate mechanisms involved in airway allergic inflammation to the serine protease subtilisin [Internet]. Journal of Immunology. 2015 ; 194( 10): 4621-4630.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1402493
Vancouver
Florsheim E, Yu S, Bargatto I, Faustino L, Gomes E, Ramos RN, Barbuto JAM, Medzhitov R, Russo M. Integrated innate mechanisms involved in airway allergic inflammation to the serine protease subtilisin [Internet]. Journal of Immunology. 2015 ; 194( 10): 4621-4630.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1402493
Artigo de periodico
The role and effects of glucocorticoid-induced leucine zipper in the context of inflammation resolution (2015)
Vago, Juliana P; Tavares, Luciana P; Garcia, Cristiana C; Lima, Katia M; Perucci, Luiza O; Vieira, Erica L; Nogueira, Camila R. C; Soriani, Frederico Marianetti; Martins, Joilson de Oliveira ; Silva, Patricia M. R; Gomes, Karina B; Pinho, Vanessa; Bruscoli, Stefano; Riccardi, Carlo; Beaulieu, Elaine; Morand, Eric F; Teixeira, Mauro M; Sousa, Lirlandia P
Source: Journal of Immunology. Unidade: FCF
Subjects: INFLAMAÇÃO, MACRÓFAGOS
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ABNT
VAGO, Juliana P et al. The role and effects of glucocorticoid-induced leucine zipper in the context of inflammation resolution. Journal of Immunology, v. 194, n. 10, p. 4940-4950 : + Supplementary materials (S1-S4), 2015Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1401722. Acesso em: 15 jul. 2025.
APA
Vago, J. P., Tavares, L. P., Garcia, C. C., Lima, K. M., Perucci, L. O., Vieira, E. L., et al. (2015). The role and effects of glucocorticoid-induced leucine zipper in the context of inflammation resolution. Journal of Immunology, 194( 10), 4940-4950 : + Supplementary materials (S1-S4). doi:10.4049/jimmunol.1401722
NLM
Vago JP, Tavares LP, Garcia CC, Lima KM, Perucci LO, Vieira EL, Nogueira CRC, Soriani FM, Martins J de O, Silva PMR, Gomes KB, Pinho V, Bruscoli S, Riccardi C, Beaulieu E, Morand EF, Teixeira MM, Sousa LP. The role and effects of glucocorticoid-induced leucine zipper in the context of inflammation resolution [Internet]. Journal of Immunology. 2015 ; 194( 10): 4940-4950 : + Supplementary materials (S1-S4).[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1401722
Vancouver
Vago JP, Tavares LP, Garcia CC, Lima KM, Perucci LO, Vieira EL, Nogueira CRC, Soriani FM, Martins J de O, Silva PMR, Gomes KB, Pinho V, Bruscoli S, Riccardi C, Beaulieu E, Morand EF, Teixeira MM, Sousa LP. The role and effects of glucocorticoid-induced leucine zipper in the context of inflammation resolution [Internet]. Journal of Immunology. 2015 ; 194( 10): 4940-4950 : + Supplementary materials (S1-S4).[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1401722
Artigo de periodico
PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis (2014)
Ferreira, Ana Elisa; Sisti, Flavia; Sônego, Fabiane; Wang, Suojuan; Filgueiras, Luciano Ribeiro; Brandt, Stephanie; Serezani, Ana Paula Moreira; Du, Hong; Cunha, Fernando de Queiroz; Alves Filho, José Carlos Farias; Serezani, Carlos Henrique
Source: Journal of Immunology. Unidade: FMRP
Subjects: EXPRESSÃO GÊNICA, REGULAÇÃO GÊNICA, INTERLEUCINA 10, SEPSE (IMUNOLOGIA), MODELOS ANIMAIS DE DOENÇAS
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ABNT
FERREIRA, Ana Elisa et al. PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis. Journal of Immunology, v. 192, n. 5, p. 2357-2365, 2014Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1302375. Acesso em: 15 jul. 2025.
APA
Ferreira, A. E., Sisti, F., Sônego, F., Wang, S., Filgueiras, L. R., Brandt, S., et al. (2014). PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis. Journal of Immunology, 192( 5), 2357-2365. doi:10.4049/jimmunol.1302375
NLM
Ferreira AE, Sisti F, Sônego F, Wang S, Filgueiras LR, Brandt S, Serezani APM, Du H, Cunha F de Q, Alves Filho JCF, Serezani CH. PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis [Internet]. Journal of Immunology. 2014 ; 192( 5): 2357-2365.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1302375
Vancouver
Ferreira AE, Sisti F, Sônego F, Wang S, Filgueiras LR, Brandt S, Serezani APM, Du H, Cunha F de Q, Alves Filho JCF, Serezani CH. PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis [Internet]. Journal of Immunology. 2014 ; 192( 5): 2357-2365.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1302375
Artigo de periodico
Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response (2014)
Aquino, Sabrina G. de; Abdollahi-Roodsaz, Shahla; Koenders, Marije I.; van de Loo, Fons A. J.; Pruijn, Ger J. M.; Marijnissen, Renoud J.; Walgreen, Birgitte; Helsen, Monique M.; van den Bersselaar, Liduine A.; Molon, Rafael S. de; Avila-Campos, Mario Julio; Cunha, Fernando de Queiroz; Cirelli, Joni A.; van den Berg, Wim B.
Source: Journal of Immunology. Unidades: ICB, FMRP
Subjects: DOENÇAS AUTOIMUNES, ARTRITE (IMUNOLOGIA), PERIODONTO, MICROBIOLOGIA ORAL
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ABNT
AQUINO, Sabrina G. de et al. Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response. Journal of Immunology, v. 192, n. 9, p. 4103-4111, 2014Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1301970. Acesso em: 15 jul. 2025.
APA
Aquino, S. G. de, Abdollahi-Roodsaz, S., Koenders, M. I., van de Loo, F. A. J., Pruijn, G. J. M., Marijnissen, R. J., et al. (2014). Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response. Journal of Immunology, 192( 9), 4103-4111. doi:10.4049/jimmunol.1301970
NLM
Aquino SG de, Abdollahi-Roodsaz S, Koenders MI, van de Loo FAJ, Pruijn GJM, Marijnissen RJ, Walgreen B, Helsen MM, van den Bersselaar LA, Molon RS de, Avila-Campos MJ, Cunha F de Q, Cirelli JA, van den Berg WB. Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response [Internet]. Journal of Immunology. 2014 ; 192( 9): 4103-4111.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1301970
Vancouver
Aquino SG de, Abdollahi-Roodsaz S, Koenders MI, van de Loo FAJ, Pruijn GJM, Marijnissen RJ, Walgreen B, Helsen MM, van den Bersselaar LA, Molon RS de, Avila-Campos MJ, Cunha F de Q, Cirelli JA, van den Berg WB. Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response [Internet]. Journal of Immunology. 2014 ; 192( 9): 4103-4111.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1301970
Artigo de periodico
Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation (2014)
Wang, Zhuo; Filgueiras, Luciano Ribeiro; Wang, Soujuan; Serezani, Ana Paula Moreira; Peters-Golden, Marc; Jancar, Sonia ; Serezani, C. Henrique
Source: Journal of Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, LEUCOTRIENOS B, MACRÓFAGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
WANG, Zhuo et al. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation. Journal of Immunology, v. 192, n. 5, p. 2349-2356, 2014Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1302982. Acesso em: 15 jul. 2025.
APA
Wang, Z., Filgueiras, L. R., Wang, S., Serezani, A. P. M., Peters-Golden, M., Jancar, S., & Serezani, C. H. (2014). Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation. Journal of Immunology, 192( 5), 2349-2356. doi:10.4049/jimmunol.1302982
NLM
Wang Z, Filgueiras LR, Wang S, Serezani APM, Peters-Golden M, Jancar S, Serezani CH. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation [Internet]. Journal of Immunology. 2014 ; 192( 5): 2349-2356.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1302982
Vancouver
Wang Z, Filgueiras LR, Wang S, Serezani APM, Peters-Golden M, Jancar S, Serezani CH. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation [Internet]. Journal of Immunology. 2014 ; 192( 5): 2349-2356.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1302982
Artigo de periodico
Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection (2013)
Silva, Grace Kelly da; Costa, Renata Sesti; Silveira, Tatiana Nunes; Caetano, Braulia C.; Horta, Catarina Veltrini; Gutierrez, Fredy Roberto Salazar; Guedes, Paulo Marcos da Matta; Andrade, Warrison A.; De Niz, Mariana; Gazzinelli, Ricardo Tostes; Zamboni, Dario Simões; Silva, João Santana da
Source: Journal of Immunology. Unidade: FMRP
Subjects: TRYPANOSOMA CRUZI, IMUNOLOGIA
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ABNT
SILVA, Grace Kelly da et al. Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection. Journal of Immunology, v. 191, n. 6, p. 3373-3383, 2013Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1203293. Acesso em: 15 jul. 2025.
APA
Silva, G. K. da, Costa, R. S., Silveira, T. N., Caetano, B. C., Horta, C. V., Gutierrez, F. R. S., et al. (2013). Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection. Journal of Immunology, 191( 6), 3373-3383. doi:10.4049/jimmunol.1203293
NLM
Silva GK da, Costa RS, Silveira TN, Caetano BC, Horta CV, Gutierrez FRS, Guedes PM da M, Andrade WA, De Niz M, Gazzinelli RT, Zamboni DS, Silva JS da. Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection [Internet]. Journal of Immunology. 2013 ; 191( 6): 3373-3383.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1203293
Vancouver
Silva GK da, Costa RS, Silveira TN, Caetano BC, Horta CV, Gutierrez FRS, Guedes PM da M, Andrade WA, De Niz M, Gazzinelli RT, Zamboni DS, Silva JS da. Apoptosis-associated speck–like protein containing a caspase recruitment domain inflammasomes mediate IL-1β response and host resistance to Trypanosoma cruzi infection [Internet]. Journal of Immunology. 2013 ; 191( 6): 3373-3383.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1203293
Artigo de periodico
The IL-33/ST2 pathway controls coxsackievirus B5–induced experimental pancreatitis (2013)
Sesti-Costa, Renata; Silva, Grace Kelly da; Proença-Módena, José Luiz; Carlos, Daniela; Silva, Maria L.; Alves Filho, José Carlos Farias; Arruda Neto, Eurico de; Liew, Foo Y.; Silva, João Santana da
Source: Journal of Immunology. Unidade: FMRP
Subjects: INTERLEUCINAS, PANCREATITE (EXPERIMENTAÇÃO), TRANSDUÇÃO DE SINAL CELULAR, CARGA VIRAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SESTI-COSTA, Renata et al. The IL-33/ST2 pathway controls coxsackievirus B5–induced experimental pancreatitis. Journal of Immunology, v. 191, n. 1, p. 283-292, 2013Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1202806. Acesso em: 15 jul. 2025.
APA
Sesti-Costa, R., Silva, G. K. da, Proença-Módena, J. L., Carlos, D., Silva, M. L., Alves Filho, J. C. F., et al. (2013). The IL-33/ST2 pathway controls coxsackievirus B5–induced experimental pancreatitis. Journal of Immunology, 191( 1), 283-292. doi:10.4049/jimmunol.1202806
NLM
Sesti-Costa R, Silva GK da, Proença-Módena JL, Carlos D, Silva ML, Alves Filho JCF, Arruda Neto E de, Liew FY, Silva JS da. The IL-33/ST2 pathway controls coxsackievirus B5–induced experimental pancreatitis [Internet]. Journal of Immunology. 2013 ; 191( 1): 283-292.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1202806
Vancouver
Sesti-Costa R, Silva GK da, Proença-Módena JL, Carlos D, Silva ML, Alves Filho JCF, Arruda Neto E de, Liew FY, Silva JS da. The IL-33/ST2 pathway controls coxsackievirus B5–induced experimental pancreatitis [Internet]. Journal of Immunology. 2013 ; 191( 1): 283-292.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1202806
Artigo de periodico
Histamine H2 receptor signaling in the pathogenesis of sepsis: studies in a murine Diabetes model (2013)
Carlos, Daniela; Spiller, Fernando; Souto, Fabrício O.; Trevelin, Silvia C.; Borges, Vanessa F.; Freitas, Andressa de; Alves Filho, José Carlos Farias; Silva, João Santana da; Ryffel, Bernhard; Cunha, Fernando de Queiroz
Source: Journal of Immunology. Unidade: FMRP
Subjects: DIABETES MELLITUS (EXPERIMENTAÇÃO), SEPSE, RECEPTORES (METABOLISMO), BACTEREMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARLOS, Daniela et al. Histamine H2 receptor signaling in the pathogenesis of sepsis: studies in a murine Diabetes model. Journal of Immunology, v. 191, n. 3, p. 1373-1382, 2013Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1202907. Acesso em: 15 jul. 2025.
APA
Carlos, D., Spiller, F., Souto, F. O., Trevelin, S. C., Borges, V. F., Freitas, A. de, et al. (2013). Histamine H2 receptor signaling in the pathogenesis of sepsis: studies in a murine Diabetes model. Journal of Immunology, 191( 3), 1373-1382. doi:10.4049/jimmunol.1202907
NLM
Carlos D, Spiller F, Souto FO, Trevelin SC, Borges VF, Freitas A de, Alves Filho JCF, Silva JS da, Ryffel B, Cunha F de Q. Histamine H2 receptor signaling in the pathogenesis of sepsis: studies in a murine Diabetes model [Internet]. Journal of Immunology. 2013 ; 191( 3): 1373-1382.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1202907
Vancouver
Carlos D, Spiller F, Souto FO, Trevelin SC, Borges VF, Freitas A de, Alves Filho JCF, Silva JS da, Ryffel B, Cunha F de Q. Histamine H2 receptor signaling in the pathogenesis of sepsis: studies in a murine Diabetes model [Internet]. Journal of Immunology. 2013 ; 191( 3): 1373-1382.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1202907
Artigo de periodico
Critical role of ASC inflammasomes and bacterial type IV secretion system in caspase-1 activation and host innate resistance to Brucella abortus infection (2013)
Gomes, Marco Tulio R.; Campos, Priscila C.; Oliveira, Fernanda S.; Corsetti, Patricia P.; Bortoluci, Karina R.; Cunha, Larissa Dias da; Zamboni, Dario Simões; Oliveira, Sergio C.
Source: Journal of Immunology. Unidade: FMRP
Subjects: APOPTOSE, BRUCELOSE, PROTEÍNAS DE TRANSPORTE, GRANULOMA, INTERLEUCINA 1
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ABNT
GOMES, Marco Tulio R. et al. Critical role of ASC inflammasomes and bacterial type IV secretion system in caspase-1 activation and host innate resistance to Brucella abortus infection. Journal of Immunology, v. 190, n. 7, p. 3629-3638, 2013Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1202817. Acesso em: 15 jul. 2025.
APA
Gomes, M. T. R., Campos, P. C., Oliveira, F. S., Corsetti, P. P., Bortoluci, K. R., Cunha, L. D. da, et al. (2013). Critical role of ASC inflammasomes and bacterial type IV secretion system in caspase-1 activation and host innate resistance to Brucella abortus infection. Journal of Immunology, 190( 7), 3629-3638. doi:10.4049/jimmunol.1202817
NLM
Gomes MTR, Campos PC, Oliveira FS, Corsetti PP, Bortoluci KR, Cunha LD da, Zamboni DS, Oliveira SC. Critical role of ASC inflammasomes and bacterial type IV secretion system in caspase-1 activation and host innate resistance to Brucella abortus infection [Internet]. Journal of Immunology. 2013 ; 190( 7): 3629-3638.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1202817
Vancouver
Gomes MTR, Campos PC, Oliveira FS, Corsetti PP, Bortoluci KR, Cunha LD da, Zamboni DS, Oliveira SC. Critical role of ASC inflammasomes and bacterial type IV secretion system in caspase-1 activation and host innate resistance to Brucella abortus infection [Internet]. Journal of Immunology. 2013 ; 190( 7): 3629-3638.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1202817
Artigo de periodico
Nitric oxide-induced regulatory T cells inhibit Th 17 but not Th1 cell differentiation and function (2013)
Niedbala, Wanda; Besnard, Anne-Gaelle; Jiang, Hui R.; Alves-Filho, José Carlos; Fukada, Sandra Y.; Nascimento, Daniela; Mitani, Akio; Pushparaj, Peter; Alqahtani, Mohammed H.; Liew, Foo Y.
Source: Journal of Immunology. Unidade: FMRP
Subjects: ÓXIDO NÍTRICO, LINFÓCITOS, DIFERENCIAÇÃO CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NIEDBALA, Wanda et al. Nitric oxide-induced regulatory T cells inhibit Th 17 but not Th1 cell differentiation and function. Journal of Immunology, v. 191, n. 1, p. 164-170, 2013Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1202580. Acesso em: 15 jul. 2025.
APA
Niedbala, W., Besnard, A. -G., Jiang, H. R., Alves-Filho, J. C., Fukada, S. Y., Nascimento, D., et al. (2013). Nitric oxide-induced regulatory T cells inhibit Th 17 but not Th1 cell differentiation and function. Journal of Immunology, 191( 1), 164-170. doi:10.4049/jimmunol.1202580
NLM
Niedbala W, Besnard A-G, Jiang HR, Alves-Filho JC, Fukada SY, Nascimento D, Mitani A, Pushparaj P, Alqahtani MH, Liew FY. Nitric oxide-induced regulatory T cells inhibit Th 17 but not Th1 cell differentiation and function [Internet]. Journal of Immunology. 2013 ; 191( 1): 164-170.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1202580
Vancouver
Niedbala W, Besnard A-G, Jiang HR, Alves-Filho JC, Fukada SY, Nascimento D, Mitani A, Pushparaj P, Alqahtani MH, Liew FY. Nitric oxide-induced regulatory T cells inhibit Th 17 but not Th1 cell differentiation and function [Internet]. Journal of Immunology. 2013 ; 191( 1): 164-170.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1202580
Artigo de periodico
Anti-inflammatory mechanisms of the annexin A1 protein and its mimetic peptide Ac2-26 in models of ocular inflammation in vivo and in vitro (2013)
Girol, Ana Paula; Mimura, Kallyne K. O; Drewes, Carine Cristiane; Bolonheis, Simone Marques de; Solito, Egle; Farsky, Sandra Helena Poliselli ; Gil, Cristiane D; Oliani, Sonia Maria
Source: Journal of Immunology. Unidade: FCF
Subjects: ANTI-INFLAMATÓRIOS, PEPTÍDEOS
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ABNT
GIROL, Ana Paula et al. Anti-inflammatory mechanisms of the annexin A1 protein and its mimetic peptide Ac2-26 in models of ocular inflammation in vivo and in vitro. Journal of Immunology, v. 190, n. 11, p. 5689-5701, 2013Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1202030. Acesso em: 15 jul. 2025.
APA
Girol, A. P., Mimura, K. K. O., Drewes, C. C., Bolonheis, S. M. de, Solito, E., Farsky, S. H. P., et al. (2013). Anti-inflammatory mechanisms of the annexin A1 protein and its mimetic peptide Ac2-26 in models of ocular inflammation in vivo and in vitro. Journal of Immunology, 190( 11), 5689-5701. doi:10.4049/jimmunol.1202030
NLM
Girol AP, Mimura KKO, Drewes CC, Bolonheis SM de, Solito E, Farsky SHP, Gil CD, Oliani SM. Anti-inflammatory mechanisms of the annexin A1 protein and its mimetic peptide Ac2-26 in models of ocular inflammation in vivo and in vitro [Internet]. Journal of Immunology. 2013 ; 190( 11): 5689-5701.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1202030
Vancouver
Girol AP, Mimura KKO, Drewes CC, Bolonheis SM de, Solito E, Farsky SHP, Gil CD, Oliani SM. Anti-inflammatory mechanisms of the annexin A1 protein and its mimetic peptide Ac2-26 in models of ocular inflammation in vivo and in vitro [Internet]. Journal of Immunology. 2013 ; 190( 11): 5689-5701.[citado 2025 jul. 15 ] Available from: https://doi.org/10.4049/jimmunol.1202030

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