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Artigo de periodico
Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure (2020)
Fontes, Milene Tavares ; Paula, Suliana Mesquita ; Lino, Caroline Antunes ; Senger, Nathalia ; Couto, Gisele Kruger; Chaves, Maria Luiza Morais Barreto ; Mill, José Geraldo ; Rossoni, Luciana Venturini
Source: Clinical Science. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA, SISTEMA RENINA-ANGIOTENSINA, TECIDO ADIPOSO, INSUFICIÊNCIA CARDÍACA ANIMAL, DOENÇAS CARDIOVASCULARES EM ANIMAL, ENDOTÉLIO VASCULAR, INFARTO DO MIOCÁRDIO, RATOS, ANGIOTENSINAS, AORTA DE ANIMAL, ESTRESSE OXIDATIVO, ÓXIDO NÍTRICO, AORTA TORÁCICA
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ABNT
FONTES, Milene Tavares; PAULA, Suliana Mesquita; LINO, Caroline Antunes; et al. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure. Clinical Science, London, 2020. Disponível em: < https://doi.org/10.1042/CS20201099 > DOI: 10.1042/CS20201099.
APA
Fontes, M. T., Paula, S. M., Lino, C. A., Senger, N., Couto, G. K., Chaves, M. L. M. B., et al. (2020). Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure. Clinical Science. doi:10.1042/CS20201099
NLM
Fontes MT, Paula SM, Lino CA, Senger N, Couto GK, Chaves MLMB, Mill JG, Rossoni LV. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure [Internet]. Clinical Science. 2020 ;Available from: https://doi.org/10.1042/CS20201099
Vancouver
Fontes MT, Paula SM, Lino CA, Senger N, Couto GK, Chaves MLMB, Mill JG, Rossoni LV. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure [Internet]. Clinical Science. 2020 ;Available from: https://doi.org/10.1042/CS20201099
Artigo de periodico
NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis (2019)
Braga, Tárcio Teodoro ; Brandão, Wesley Nogueira ; Azevedo, Hatylas ; Terra, Fernanda Fernandes; Feitosa, Amanda Campelo Lima Melo; Pereira, Felipe Valença; Oliveira, Vinicius de Andrade ; Hiyane, Meire Ioshie; Peron, Jean Pierre Schatzmann ; Câmara, Niels Olsen Saraiva
Source: Clinical Science. Unidade: ICB
Subjects: IMUNOLOGIA, AUTOIMUNIDADE, NUCLEOTÍDEOS, CITOCINAS, DOENÇAS AUTOIMUNES, LINFÓCITOS T, ENCEFALOMIELITE ANIMAL, MODELOS ANIMAIS, FATORES DE TRANSCRIÇÃO
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BRAGA, Tárcio Teodoro; BRANDÃO, Wesley Nogueira; AZEVEDO, Hatylas; et al. NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis. Clinical Science, London, v. 133, n. 17, p. 1901-1916, 2019. Disponível em: < https://doi.org/10.1042/CS20190506 > DOI: 10.1042/CS20190506.
APA
Braga, T. T., Brandão, W. N., Azevedo, H., Terra, F. F., Feitosa, A. C. L. M., Pereira, F. V., et al. (2019). NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis. Clinical Science, 133( 17), 1901-1916. doi:10.1042/CS20190506
NLM
Braga TT, Brandão WN, Azevedo H, Terra FF, Feitosa ACLM, Pereira FV, Oliveira V de A, Hiyane MI, Peron JPS, Câmara NOS. NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis [Internet]. Clinical Science. 2019 ; 133( 17): 1901-1916.Available from: https://doi.org/10.1042/CS20190506
Vancouver
Braga TT, Brandão WN, Azevedo H, Terra FF, Feitosa ACLM, Pereira FV, Oliveira V de A, Hiyane MI, Peron JPS, Câmara NOS. NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis [Internet]. Clinical Science. 2019 ; 133( 17): 1901-1916.Available from: https://doi.org/10.1042/CS20190506
Artigo de periodico
Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure (2019)
Andrade, David C.; Álvarez, Alexis Arce; Santos, Luiz Marcelo Oliveira ; Moreira, Ana Carolina Takakura ; Moreira, Thiago dos Santos ; Schultz, Harold D.; Marcus, Noah J. ; Alcayaga, Julio ; Rio, Rodrigo Del ; Toledo, Camilo; Díaz, Hugo S.; Lucero, Claudia
Source: Clinical Science. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, SISTEMA NERVOSO SIMPÁTICO, INSUFICIÊNCIA CARDÍACA ANIMAL, NEURÔNIOS, IMUNOTOXINAS, TOXINAS EM ANIMAL, RATOS, ARRITMIA, TRONCO DE ANIMAL, CATECOLAMINAS
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ABNT
ANDRADE, David C.; ÁLVAREZ, Alexis Arce; SANTOS, Luiz Marcelo Oliveira; et al. Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure. Clinical Science, London, v. 133, n. 3, p. 393-405, 2019. Disponível em: < https://doi.org/10.1042/CS20180589 > DOI: 10.1042/CS20180589.
APA
Andrade, D. C., Álvarez, A. A., Santos, L. M. O., Moreira, A. C. T., Moreira, T. dos S., Schultz, H. D., et al. (2019). Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure. Clinical Science, 133( 3), 393-405. doi:10.1042/CS20180589
NLM
Andrade DC, Álvarez AA, Santos LMO, Moreira ACT, Moreira T dos S, Schultz HD, Marcus NJ, Alcayaga J, Rio RD, Toledo C, Díaz HS, Lucero C. Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure [Internet]. Clinical Science. 2019 ; 133( 3): 393-405.Available from: https://doi.org/10.1042/CS20180589
Vancouver
Andrade DC, Álvarez AA, Santos LMO, Moreira ACT, Moreira T dos S, Schultz HD, Marcus NJ, Alcayaga J, Rio RD, Toledo C, Díaz HS, Lucero C. Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure [Internet]. Clinical Science. 2019 ; 133( 3): 393-405.Available from: https://doi.org/10.1042/CS20180589
Artigo de periodico
Hyperfiltration in ubiquitin C-terminal hydrolase L1-deleted mice (2018)
Boisvert, Naomi C.; Holterman, Chet E.; Thibodeau, Jean-François; Nasrallah, Rania; Kamto, Eldjonai; Comin, Cesar H.; Costa, Luciano da Fontoura; Carter, Anthony; Hébert, Richard L.; Gutsol, Alex; Cron, Gregory O.; Lacoste, Baptiste; Gray, Douglas A.; Kennedy, Chris R.
Source: Clinical Science. Unidade: IFSC
Subjects: REDES COMPLEXAS, AUTOR, MINERAÇÃO DE DADOS, VISÃO COMPUTACIONAL
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ABNT
BOISVERT, Naomi C.; HOLTERMAN, Chet E.; THIBODEAU, Jean-François; et al. Hyperfiltration in ubiquitin C-terminal hydrolase L1-deleted mice. Clinical Science, Oxford, v. 132, n. 13, p. 1453-1470, 2018. Disponível em: < http://dx.doi.org/10.1042/CS20180085 > DOI: 10.1042/CS20180085.
APA
Boisvert, N. C., Holterman, C. E., Thibodeau, J. -F., Nasrallah, R., Kamto, E., Comin, C. H., et al. (2018). Hyperfiltration in ubiquitin C-terminal hydrolase L1-deleted mice. Clinical Science, 132( 13), 1453-1470. doi:10.1042/CS20180085
NLM
Boisvert NC, Holterman CE, Thibodeau J-F, Nasrallah R, Kamto E, Comin CH, Costa L da F, Carter A, Hébert RL, Gutsol A, Cron GO, Lacoste B, Gray DA, Kennedy CR. Hyperfiltration in ubiquitin C-terminal hydrolase L1-deleted mice [Internet]. Clinical Science. 2018 ; 132( 13): 1453-1470.Available from: http://dx.doi.org/10.1042/CS20180085
Vancouver
Boisvert NC, Holterman CE, Thibodeau J-F, Nasrallah R, Kamto E, Comin CH, Costa L da F, Carter A, Hébert RL, Gutsol A, Cron GO, Lacoste B, Gray DA, Kennedy CR. Hyperfiltration in ubiquitin C-terminal hydrolase L1-deleted mice [Internet]. Clinical Science. 2018 ; 132( 13): 1453-1470.Available from: http://dx.doi.org/10.1042/CS20180085
Artigo de periodico
Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway (2018)
Senger, Nathalia; Melo, Marcos Barrouin; Diniz, Gabriela Placoná; Santos, Maria José Campagnole dos; Santos, Robson Augusto Souza dos; Chaves, Maria Luiza Morais Barreto de
Source: Clinical Science. Unidade: ICB
Subjects: ANGIOTENSINAS, CORAÇÃO, HIPERTIREOIDISMO, SISTEMA RENINA-ANGIOTENSINA, HORMÔNIOS TIREOIDIANOS, ANGIOTENSINA II
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ABNT
SENGER, Nathalia; MELO, Marcos Barrouin; DINIZ, Gabriela Placoná; et al. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, London, v. 132, n. 11, p. 1117-1133, 2018. Disponível em: < http://dx.doi.org/ 10.1042/CS20171606 > DOI: 10.1042/CS20171606.
APA
Senger, N., Melo, M. B., Diniz, G. P., Santos, M. J. C. dos, Santos, R. A. S. dos, & Chaves, M. L. M. B. de. (2018). Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, 132( 11), 1117-1133. doi:10.1042/CS20171606
NLM
Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Chaves MLMB de. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.Available from: http://dx.doi.org/ 10.1042/CS20171606
Vancouver
Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Chaves MLMB de. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.Available from: http://dx.doi.org/ 10.1042/CS20171606
Artigo de periodico
Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease (2017)
Festuccia, William T.
Source: Clinical Science. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
FESTUCCIA, William T. Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease. Clinical Science, London, v. 131, n. 4, p. 327-328, 2017. Disponível em: < http://dx.doi.org/10.1042/CS20160855 > DOI: 10.1042/CS20160855.
APA
Festuccia, W. T. (2017). Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease. Clinical Science, 131( 4), 327-328. doi:10.1042/CS20160855
NLM
Festuccia WT. Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease [Internet]. Clinical Science. 2017 ; 131( 4): 327-328.Available from: http://dx.doi.org/10.1042/CS20160855
Vancouver
Festuccia WT. Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease [Internet]. Clinical Science. 2017 ; 131( 4): 327-328.Available from: http://dx.doi.org/10.1042/CS20160855
Artigo de periodico
Sympathetic nervous activity in patients with acute coronary syndrome: a comparative study of inflammatory biomarkers (2017)
Moreira, Humberto G; Lage, Rony L; Martinez, Daniel G; Ferreira-Santos, Larissa; Rondon, M. U.; Negrão, C. E.; Nicolau, José Carlos
Source: Clinical Science. Unidades: EEFE, FM
Subjects: SISTEMA NERVOSO SIMPÁTICO, SÍNDROME CORONARIANA AGUDA, BIOMARCADORES
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ABNT
MOREIRA, Humberto G; LAGE, Rony L; MARTINEZ, Daniel G; et al. Sympathetic nervous activity in patients with acute coronary syndrome: a comparative study of inflammatory biomarkers. Clinical Science, London, v. 131, n. 9, p. 883-895, 2017. DOI: 10.1042/CS20170049.
APA
Moreira, H. G., Lage, R. L., Martinez, D. G., Ferreira-Santos, L., Rondon, M. U., Negrão, C. E., & Nicolau, J. C. (2017). Sympathetic nervous activity in patients with acute coronary syndrome: a comparative study of inflammatory biomarkers. Clinical Science, 131( 9), 883-895. doi:10.1042/CS20170049
NLM
Moreira HG, Lage RL, Martinez DG, Ferreira-Santos L, Rondon MU, Negrão CE, Nicolau JC. Sympathetic nervous activity in patients with acute coronary syndrome: a comparative study of inflammatory biomarkers. Clinical Science. 2017 ; 131( 9): 883-895.
Vancouver
Moreira HG, Lage RL, Martinez DG, Ferreira-Santos L, Rondon MU, Negrão CE, Nicolau JC. Sympathetic nervous activity in patients with acute coronary syndrome: a comparative study of inflammatory biomarkers. Clinical Science. 2017 ; 131( 9): 883-895.
Artigo de periodico
Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy (2017)
Diniz, Gabriela Placoná; Wang, Da-Zhi; Hu, Xiaoyun; Huang, Zhan-Peng; Liu, Jianming; Chen, Jinghai; Ding, Jian; Fonseca, Renata Inzinna; Barreto-Chaves, Maria Luiza; Donato Jr, Jose
Source: Clinical Science. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
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ABNT
DINIZ, Gabriela Placoná; WANG, Da-Zhi; HU, Xiaoyun; et al. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, London, v. 131, n. 24, p. 2885-2900, 2017. Disponível em: < http://dx.doi.org/10.1042/CS20171368 > DOI: 10.1042/CS20171368.
APA
Diniz, G. P., Wang, D. -Z., Hu, X., Huang, Z. -P., Liu, J., Chen, J., et al. (2017). Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, 131( 24), 2885-2900. doi:10.1042/CS20171368
NLM
Diniz GP, Wang D-Z, Hu X, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves ML, Donato Jr J. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.Available from: http://dx.doi.org/10.1042/CS20171368
Vancouver
Diniz GP, Wang D-Z, Hu X, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves ML, Donato Jr J. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.Available from: http://dx.doi.org/10.1042/CS20171368
Artigo de periodico
PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis (2016)
Filgueiras, Luciano Ribeiro; Koga, Marianna Mainardi; Quaresma, Paula G.; Ishizuka, Edson Kiyotaka; Montes, Marlise B. A.; Prada, Patricia O.; Saad, Mario J.; Jancar, Sonia; Rios, Francisco J.
Source: Clinical Science. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
FILGUEIRAS, Luciano Ribeiro; KOGA, Marianna Mainardi; QUARESMA, Paula G.; et al. PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis. Clinical Science, London, v. 130, n. 8, p. 601-612, 2016. Disponível em: < http://dx.doi.org/10.1042/CS20150538 > DOI: 10.1042/CS20150538.
APA
Filgueiras, L. R., Koga, M. M., Quaresma, P. G., Ishizuka, E. K., Montes, M. B. A., Prada, P. O., et al. (2016). PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis. Clinical Science, 130( 8), 601-612. doi:10.1042/CS20150538
NLM
Filgueiras LR, Koga MM, Quaresma PG, Ishizuka EK, Montes MBA, Prada PO, Saad MJ, Jancar S, Rios FJ. PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis [Internet]. Clinical Science. 2016 ; 130( 8): 601-612.Available from: http://dx.doi.org/10.1042/CS20150538
Vancouver
Filgueiras LR, Koga MM, Quaresma PG, Ishizuka EK, Montes MBA, Prada PO, Saad MJ, Jancar S, Rios FJ. PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis [Internet]. Clinical Science. 2016 ; 130( 8): 601-612.Available from: http://dx.doi.org/10.1042/CS20150538
Artigo de periodico
High-fat diet increases O-GlcNAc levels in cerebral arteries: a link to vascular dysfunction associated with hyperlipidaemia/obesity ? (2016)
Lima, Victor L.; Giachini, Fernanda R.; Matsumoto, Takayuki; Li, Weiguo; Bressan, Alecsander F. M.; Chawla, Dhruv; Webb, R. Clinton; Ergul, Adviye; Tostes, Rita de Cássia Aleixo
Source: Clinical Science. Unidade: FMRP
Subjects: ARTÉRIAS CEREBRAIS, HIPERLIPIDEMIA, PROTEÍNAS QUINASES, OBESIDADE
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ABNT
LIMA, Victor L.; GIACHINI, Fernanda R.; MATSUMOTO, Takayuki; et al. High-fat diet increases O-GlcNAc levels in cerebral arteries: a link to vascular dysfunction associated with hyperlipidaemia/obesity ? Clinical Science, London, v. 130, n. 11, p. 871-881, 2016. Disponível em: < http://dx.doi.org/10.1042/CS20150777 > DOI: 10.1042/CS20150777.
APA
Lima, V. L., Giachini, F. R., Matsumoto, T., Li, W., Bressan, A. F. M., Chawla, D., et al. (2016). High-fat diet increases O-GlcNAc levels in cerebral arteries: a link to vascular dysfunction associated with hyperlipidaemia/obesity ? Clinical Science, 130( 11), 871-881. doi:10.1042/CS20150777
NLM
Lima VL, Giachini FR, Matsumoto T, Li W, Bressan AFM, Chawla D, Webb RC, Ergul A, Tostes R de CA. High-fat diet increases O-GlcNAc levels in cerebral arteries: a link to vascular dysfunction associated with hyperlipidaemia/obesity ? [Internet]. Clinical Science. 2016 ; 130( 11): 871-881.Available from: http://dx.doi.org/10.1042/CS20150777
Vancouver
Lima VL, Giachini FR, Matsumoto T, Li W, Bressan AFM, Chawla D, Webb RC, Ergul A, Tostes R de CA. High-fat diet increases O-GlcNAc levels in cerebral arteries: a link to vascular dysfunction associated with hyperlipidaemia/obesity ? [Internet]. Clinical Science. 2016 ; 130( 11): 871-881.Available from: http://dx.doi.org/10.1042/CS20150777
Artigo de periodico
EEF1D modulates proliferation and epithelial–mesenchymal transition in oral squamous cell carcinoma (2016)
Flores, Isadora L; Kawahara, Rebeca; Miguel, Márcia C. C; Granato, Daniela Campos; Domingues, Romênia R; Macedo, Carolina C. S; Carnielli, Carolina M; Yokoo, Sami; Rodrigues, Priscila C; Monteiro, Bárbara V. B; Oliveira, Carine E; Salmon, Cristiane Ribeiro; Nociti, Francisco H; Lopes, Márcio A; Silva, Alan Santos; Winck, Flavia Vischi ; Coletta, Ricardo D; Leme, Adriana Franco Paes
Source: Clinical Science. Unidade: IQ
Subjects: CARCINOMA, CÉLULAS EPITELIAIS, EXPRESSÃO GÊNICA
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ABNT
FLORES, Isadora L; KAWAHARA, Rebeca; MIGUEL, Márcia C. C; et al. EEF1D modulates proliferation and epithelial–mesenchymal transition in oral squamous cell carcinoma. Clinical Science, London, v. 130, p. 785-799, 2016. Disponível em: < http://dx.doi.org/10.1042/CS20150646 > DOI: 10.1042/CS20150646.
APA
Flores, I. L., Kawahara, R., Miguel, M. C. C., Granato, D. C., Domingues, R. R., Macedo, C. C. S., et al. (2016). EEF1D modulates proliferation and epithelial–mesenchymal transition in oral squamous cell carcinoma. Clinical Science, 130, 785-799. doi:10.1042/CS20150646
NLM
Flores IL, Kawahara R, Miguel MCC, Granato DC, Domingues RR, Macedo CCS, Carnielli CM, Yokoo S, Rodrigues PC, Monteiro BVB, Oliveira CE, Salmon CR, Nociti FH, Lopes MA, Silva AS, Winck FV, Coletta RD, Leme AFP. EEF1D modulates proliferation and epithelial–mesenchymal transition in oral squamous cell carcinoma [Internet]. Clinical Science. 2016 ; 130 785-799.Available from: http://dx.doi.org/10.1042/CS20150646
Vancouver
Flores IL, Kawahara R, Miguel MCC, Granato DC, Domingues RR, Macedo CCS, Carnielli CM, Yokoo S, Rodrigues PC, Monteiro BVB, Oliveira CE, Salmon CR, Nociti FH, Lopes MA, Silva AS, Winck FV, Coletta RD, Leme AFP. EEF1D modulates proliferation and epithelial–mesenchymal transition in oral squamous cell carcinoma [Internet]. Clinical Science. 2016 ; 130 785-799.Available from: http://dx.doi.org/10.1042/CS20150646
Artigo de periodico
Nox1 in cardiovascular diseases: regulation and pathophysiology (2016)
Gimenez, Marcela; Schickling, Brandon M.; Lopes, Lucia R.; Miller, Francis J.
Source: Clinical Science. Unidade: ICB
Assunto: FARMACOLOGIA
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ABNT
GIMENEZ, Marcela; SCHICKLING, Brandon M.; LOPES, Lucia R.; MILLER, Francis J. Nox1 in cardiovascular diseases: regulation and pathophysiology. Clinical Science, London, v. 130, n. 3, p. 151-165, 2016. Disponível em: < http://dx.doi.org/10.1042/CS20150404 > DOI: 10.1042/CS20150404.
APA
Gimenez, M., Schickling, B. M., Lopes, L. R., & Miller, F. J. (2016). Nox1 in cardiovascular diseases: regulation and pathophysiology. Clinical Science, 130( 3), 151-165. doi:10.1042/CS20150404
NLM
Gimenez M, Schickling BM, Lopes LR, Miller FJ. Nox1 in cardiovascular diseases: regulation and pathophysiology [Internet]. Clinical Science. 2016 ; 130( 3): 151-165.Available from: http://dx.doi.org/10.1042/CS20150404
Vancouver
Gimenez M, Schickling BM, Lopes LR, Miller FJ. Nox1 in cardiovascular diseases: regulation and pathophysiology [Internet]. Clinical Science. 2016 ; 130( 3): 151-165.Available from: http://dx.doi.org/10.1042/CS20150404
Artigo de periodico
Autologous haematopoietic stem cell transplantation reduces abnormalities in the expression of immune genes in multiple sclerosis (2015)
Sousa, Alessandra de Paula; Malmegrim, Kelen C.; Panepucci, Rodrigo A.; Brum, Doralina S.; Barreira, Amilton Antunes; Santos, Antônio Carlos dos; Araújo, Amélia Goes; Covas, Dimas Tadeu; Oliveira, Maria C.; Moraes, Daniela A.; Pieroni, Fabiano; Barros, George M.; Simões, Belinda Pinto; Nicholas, Richard; Burt, Richard K.; Voltarelli, Julio Cesar; Muraro, Paolo A.
Source: Clinical Science. Unidade: FMRP
Subjects: EXPRESSÃO GÊNICA, REGULAÇÃO GÊNICA, CÉLULAS-TRONCO (TRANSPLANTE), ESCLEROSE MÚLTIPLA
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ABNT
SOUSA, Alessandra de Paula; MALMEGRIM, Kelen C.; PANEPUCCI, Rodrigo A.; et al. Autologous haematopoietic stem cell transplantation reduces abnormalities in the expression of immune genes in multiple sclerosis. Clinical Science, London, v. 128, n. 2, p. 111-120, 2015. Disponível em: < http://dx.doi.org/10.1042/CS20140095 > DOI: 10.1042/CS20140095.
APA
Sousa, A. de P., Malmegrim, K. C., Panepucci, R. A., Brum, D. S., Barreira, A. A., Santos, A. C. dos, et al. (2015). Autologous haematopoietic stem cell transplantation reduces abnormalities in the expression of immune genes in multiple sclerosis. Clinical Science, 128( 2), 111-120. doi:10.1042/CS20140095
NLM
Sousa A de P, Malmegrim KC, Panepucci RA, Brum DS, Barreira AA, Santos AC dos, Araújo AG, Covas DT, Oliveira MC, Moraes DA, Pieroni F, Barros GM, Simões BP, Nicholas R, Burt RK, Voltarelli JC, Muraro PA. Autologous haematopoietic stem cell transplantation reduces abnormalities in the expression of immune genes in multiple sclerosis [Internet]. Clinical Science. 2015 ; 128( 2): 111-120.Available from: http://dx.doi.org/10.1042/CS20140095
Vancouver
Sousa A de P, Malmegrim KC, Panepucci RA, Brum DS, Barreira AA, Santos AC dos, Araújo AG, Covas DT, Oliveira MC, Moraes DA, Pieroni F, Barros GM, Simões BP, Nicholas R, Burt RK, Voltarelli JC, Muraro PA. Autologous haematopoietic stem cell transplantation reduces abnormalities in the expression of immune genes in multiple sclerosis [Internet]. Clinical Science. 2015 ; 128( 2): 111-120.Available from: http://dx.doi.org/10.1042/CS20140095
Artigo de periodico
Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways (2015)
Bruder-Nascimento, Thiago; Callera, Glaucia E; Montezano, Augusto C; He, Ying; Antunes, Tayze T; Cat, Aurelie Nguyen Dinh; Tostes, Rita de Cássia Aleixo; Touyz, Rhian M
Source: Clinical Science. Unidade: FMRP
Subjects: DIABETES MELLITUS (FISIOPATOLOGIA), LEPTINA, VASODILATAÇÃO (FARMACOLOGIA), PROTEÍNAS QUINASES, ARTÉRIAS (EFEITOS DE DROGAS)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRUDER-NASCIMENTO, Thiago; CALLERA, Glaucia E; MONTEZANO, Augusto C; et al. Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways. Clinical Science, London, v. 128, n. 7, p. 411-423, 2015. Disponível em: < http://dx.doi.org/10.1042/CS20140456 > DOI: 10.1042/CS20140456.
APA
Bruder-Nascimento, T., Callera, G. E., Montezano, A. C., He, Y., Antunes, T. T., Cat, A. N. D., et al. (2015). Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways. Clinical Science, 128( 7), 411-423. doi:10.1042/CS20140456
NLM
Bruder-Nascimento T, Callera GE, Montezano AC, He Y, Antunes TT, Cat AND, Tostes R de CA, Touyz RM. Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways [Internet]. Clinical Science. 2015 ; 128( 7): 411-423.Available from: http://dx.doi.org/10.1042/CS20140456
Vancouver
Bruder-Nascimento T, Callera GE, Montezano AC, He Y, Antunes TT, Cat AND, Tostes R de CA, Touyz RM. Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways [Internet]. Clinical Science. 2015 ; 128( 7): 411-423.Available from: http://dx.doi.org/10.1042/CS20140456
Artigo de periodico
Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms (2015)
Chignalia, Andreia Z; Oliveira, Maria Aparecida; Debbas, Victor; Dull, Randal O; Laurindo, Francisco R. M; Touyz, Rhian M; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno; Tostes, Rita de Cássia Aleixo
Source: Clinical Science. Unidades: FMRP, FM, ICB
Subjects: ANDRÓGENOS (FARMACOLOGIA), INIBIDORES DE ENZIMAS, LEUCÓCITOS (METABOLISMO), TRANSDUÇÃO DE SINAL CELULAR, TESTOSTERONA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHIGNALIA, Andreia Z; OLIVEIRA, Maria Aparecida; DEBBAS, Victor; et al. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, London, v. 129, n. 1, p. 39-48, 2015. Disponível em: < http://dx.doi.org/10.1042/CS20140548 > DOI: 10.1042/CS20140548.
APA
Chignalia, A. Z., Oliveira, M. A., Debbas, V., Dull, R. O., Laurindo, F. R. M., Touyz, R. M., et al. (2015). Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, 129( 1), 39-48. doi:10.1042/CS20140548
NLM
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.Available from: http://dx.doi.org/10.1042/CS20140548
Vancouver
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.Available from: http://dx.doi.org/10.1042/CS20140548
Artigo de periodico
The kinin B1 receptor regulates muscle-specific E3 ligases expression and is involved in skeletal muscle mass control (2014)
Parreiras‑e‑Silva, Lucas T; Reis, Rosana I; Santos, Geisa A; Pires‑Oliveira, Marcelo; Pesquero, João B; Gomes, Marcelo D; Godinho, Rosely O; Costa Neto, Cláudio Miguel
Source: Clinical Science. Unidade: FMRP
Subjects: CININAS (METABOLISMO), FIBRAS MUSCULARES (ENZIMOLOGIA), PROTEÍNAS MUSCULARES, ATROFIA MUSCULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PARREIRAS‑E‑SILVA, Lucas T; REIS, Rosana I; SANTOS, Geisa A; et al. The kinin B1 receptor regulates muscle-specific E3 ligases expression and is involved in skeletal muscle mass control. Clinical Science, London, v. 127, n. 3, p. 185-194, 2014. Disponível em: < http://dx.doi.org/10.1042/CS20130358 > DOI: 10.1042/CS20130358.
APA
Parreiras‑e‑Silva, L. T., Reis, R. I., Santos, G. A., Pires‑Oliveira, M., Pesquero, J. B., Gomes, M. D., et al. (2014). The kinin B1 receptor regulates muscle-specific E3 ligases expression and is involved in skeletal muscle mass control. Clinical Science, 127( 3), 185-194. doi:10.1042/CS20130358
NLM
Parreiras‑e‑Silva LT, Reis RI, Santos GA, Pires‑Oliveira M, Pesquero JB, Gomes MD, Godinho RO, Costa Neto CM. The kinin B1 receptor regulates muscle-specific E3 ligases expression and is involved in skeletal muscle mass control [Internet]. Clinical Science. 2014 ; 127( 3): 185-194.Available from: http://dx.doi.org/10.1042/CS20130358
Vancouver
Parreiras‑e‑Silva LT, Reis RI, Santos GA, Pires‑Oliveira M, Pesquero JB, Gomes MD, Godinho RO, Costa Neto CM. The kinin B1 receptor regulates muscle-specific E3 ligases expression and is involved in skeletal muscle mass control [Internet]. Clinical Science. 2014 ; 127( 3): 185-194.Available from: http://dx.doi.org/10.1042/CS20130358
Artigo de periodico
Chemerin reduces vascular nitric oxide/cGMP signalling in rat aorta: a link to vascular dysfunction in obesity? (2014)
Neves, Karla Bianca; Lobato, Núbia S; Lopes, Rhéure Alves Moreira; Filgueira, Fernando P; Zanotto, Camila Ziliotto; Oliveira, Ana Maria de; Tostes, Rita de Cassia Aleixo
Source: Clinical Science. Unidades: FCFRP, FMRP
Subjects: ACETILCOLINA (FARMACOLOGIA), AORTA TORÁCICA (EFEITOS DE DROGAS), ENDOTÉLIO VASCULAR, ÓXIDO NÍTRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NEVES, Karla Bianca; LOBATO, Núbia S; LOPES, Rhéure Alves Moreira; et al. Chemerin reduces vascular nitric oxide/cGMP signalling in rat aorta: a link to vascular dysfunction in obesity? Clinical Science, London, v. 127, n. 2, p. 111-122, 2014. Disponível em: < http://dx.doi.org/10.1042/CS20130286 > DOI: 10.1042/CS20130286.
APA
Neves, K. B., Lobato, N. S., Lopes, R. A. M., Filgueira, F. P., Zanotto, C. Z., Oliveira, A. M. de, & Tostes, R. de C. A. (2014). Chemerin reduces vascular nitric oxide/cGMP signalling in rat aorta: a link to vascular dysfunction in obesity? Clinical Science, 127( 2), 111-122. doi:10.1042/CS20130286
NLM
Neves KB, Lobato NS, Lopes RAM, Filgueira FP, Zanotto CZ, Oliveira AM de, Tostes R de CA. Chemerin reduces vascular nitric oxide/cGMP signalling in rat aorta: a link to vascular dysfunction in obesity? [Internet]. Clinical Science. 2014 ; 127( 2): 111-122.Available from: http://dx.doi.org/10.1042/CS20130286
Vancouver
Neves KB, Lobato NS, Lopes RAM, Filgueira FP, Zanotto CZ, Oliveira AM de, Tostes R de CA. Chemerin reduces vascular nitric oxide/cGMP signalling in rat aorta: a link to vascular dysfunction in obesity? [Internet]. Clinical Science. 2014 ; 127( 2): 111-122.Available from: http://dx.doi.org/10.1042/CS20130286
Artigo de periodico
Non-canonical signalling and roles of the vasoactive peptides angiotensins and kinins (2014)
Costa Neto, Cláudio Miguel; Duarte, Diego A; Lima, Vanessa; Maria, Andrea G; Prando, Érika C; Rodríguez, Deisy Y; Santos, Geisa A; Souza, Pedro P. C; Parreiras‑e‑Silva, Lucas T
Source: Clinical Science. Unidade: FMRP
Subjects: ANGIOTENSINA II (METABOLISMO), REABSORÇÃO ÓSSEA ALVEOLAR, DOENÇAS DO SISTEMA NERVOSO CENTRAL, REGULAÇÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA NETO, Cláudio Miguel; DUARTE, Diego A; LIMA, Vanessa; et al. Non-canonical signalling and roles of the vasoactive peptides angiotensins and kinins. Clinical Science, London, v. 126, n. 11, p. 753-774, 2014. Disponível em: < http://dx.doi.org/10.1042/CS20130414 > DOI: 10.1042/CS20130414.
APA
Costa Neto, C. M., Duarte, D. A., Lima, V., Maria, A. G., Prando, É. C., Rodríguez, D. Y., et al. (2014). Non-canonical signalling and roles of the vasoactive peptides angiotensins and kinins. Clinical Science, 126( 11), 753-774. doi:10.1042/CS20130414
NLM
Costa Neto CM, Duarte DA, Lima V, Maria AG, Prando ÉC, Rodríguez DY, Santos GA, Souza PPC, Parreiras‑e‑Silva LT. Non-canonical signalling and roles of the vasoactive peptides angiotensins and kinins [Internet]. Clinical Science. 2014 ; 126( 11): 753-774.Available from: http://dx.doi.org/10.1042/CS20130414
Vancouver
Costa Neto CM, Duarte DA, Lima V, Maria AG, Prando ÉC, Rodríguez DY, Santos GA, Souza PPC, Parreiras‑e‑Silva LT. Non-canonical signalling and roles of the vasoactive peptides angiotensins and kinins [Internet]. Clinical Science. 2014 ; 126( 11): 753-774.Available from: http://dx.doi.org/10.1042/CS20130414
Artigo de periodico
P2X7 receptor activation contributes to an initial upstream mechanism of lipopolysaccharide-induced vascular dysfunction (2013)
Chiao, Chin-Wei; Santos, José Eduardo da Silva; Giachini, Fernanda R.; Tostes, Rita de Cássia Aleixo; Su, Ming‑Jai; Webb, R. Clinton
Source: Clinical Science. Unidade: FMRP
Subjects: OXIGENASE, INTERLEUCINAS, LIPOPOLISSACARÍDEOS, ÓXIDO NÍTRICO, SEPSE, CITOCINAS, DOENÇAS VASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHIAO, Chin-Wei; SANTOS, José Eduardo da Silva; GIACHINI, Fernanda R.; et al. P2X7 receptor activation contributes to an initial upstream mechanism of lipopolysaccharide-induced vascular dysfunction. Clinical Science, London, v. 125, n. 3, p. 131-141, 2013. Disponível em: < http://dx.doi.org/10.1042/CS20120479 > DOI: 10.1042/cs20120479.
APA
Chiao, C. -W., Santos, J. E. da S., Giachini, F. R., Tostes, R. de C. A., Su, M. ‑J., & Webb, R. C. (2013). P2X7 receptor activation contributes to an initial upstream mechanism of lipopolysaccharide-induced vascular dysfunction. Clinical Science, 125( 3), 131-141. doi:10.1042/cs20120479
NLM
Chiao C-W, Santos JE da S, Giachini FR, Tostes R de CA, Su M‑J, Webb RC. P2X7 receptor activation contributes to an initial upstream mechanism of lipopolysaccharide-induced vascular dysfunction [Internet]. Clinical Science. 2013 ; 125( 3): 131-141.Available from: http://dx.doi.org/10.1042/CS20120479
Vancouver
Chiao C-W, Santos JE da S, Giachini FR, Tostes R de CA, Su M‑J, Webb RC. P2X7 receptor activation contributes to an initial upstream mechanism of lipopolysaccharide-induced vascular dysfunction [Internet]. Clinical Science. 2013 ; 125( 3): 131-141.Available from: http://dx.doi.org/10.1042/CS20120479
Artigo de periodico
O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter? (2012)
Lima, Victor V.; Spitler, Kathryn; Choi, Hyehun; Webb, R. Clinton; Tostes, Rita de Cassia Aleixo
Source: Clinical Science. Unidade: FMRP
Subjects: SISTEMA CARDIOVASCULAR (METABOLISMO), DIABETES MELLITUS, ESTRESSE OXIDATIVO, PROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Victor V.; SPITLER, Kathryn; CHOI, Hyehun; WEBB, R. Clinton; TOSTES, Rita de Cassia Aleixo. O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter? Clinical Science, London, v. 12, n. 8, p. 473-486, 2012. Disponível em: < http://dx.doi.org/10.1042/CS20110638 > DOI: 10.1042/cs20110638.
APA
Lima, V. V., Spitler, K., Choi, H., Webb, R. C., & Tostes, R. de C. A. (2012). O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter? Clinical Science, 12( 8), 473-486. doi:10.1042/cs20110638
NLM
Lima VV, Spitler K, Choi H, Webb RC, Tostes R de CA. O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter? [Internet]. Clinical Science. 2012 ; 12( 8): 473-486.Available from: http://dx.doi.org/10.1042/CS20110638
Vancouver
Lima VV, Spitler K, Choi H, Webb RC, Tostes R de CA. O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter? [Internet]. Clinical Science. 2012 ; 12( 8): 473-486.Available from: http://dx.doi.org/10.1042/CS20110638

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