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Artigo de periodico
New germline GATA1 variant in females with anemia and thrombocytopenia (2021)
Svidnicki, Maria Carolina Costa Melo; Ferreira Filho, Moisés Alves; Brandão, Marcelo Mendes; Santos, Marielza dos; Dias, Renata de Oliveira; Tavares, Renato Sampaio; Mendonça, Guilherme Rossi Assis; Traina, Fabíola ; Saad, Sara Teresinha Olalla
Source: Blood Cells, Molecules, and Diseases. Unidades: FMRP, FM
Subjects: MIELITE, MUTAÇÃO, ANEMIA, HEMATOPOESE, TROMBOCITOPENIA
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ABNT
SVIDNICKI, Maria Carolina Costa Melo et al. New germline GATA1 variant in females with anemia and thrombocytopenia. Blood Cells, Molecules, and Diseases, v. 88, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.bcmd.2021.102545. Acesso em: 17 mar. 2026.
APA
Svidnicki, M. C. C. M., Ferreira Filho, M. A., Brandão, M. M., Santos, M. dos, Dias, R. de O., Tavares, R. S., et al. (2021). New germline GATA1 variant in females with anemia and thrombocytopenia. Blood Cells, Molecules, and Diseases, 88. doi:10.1016/j.bcmd.2021.102545
NLM
Svidnicki MCCM, Ferreira Filho MA, Brandão MM, Santos M dos, Dias R de O, Tavares RS, Mendonça GRA, Traina F, Saad STO. New germline GATA1 variant in females with anemia and thrombocytopenia [Internet]. Blood Cells, Molecules, and Diseases. 2021 ; 88[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2021.102545
Vancouver
Svidnicki MCCM, Ferreira Filho MA, Brandão MM, Santos M dos, Dias R de O, Tavares RS, Mendonça GRA, Traina F, Saad STO. New germline GATA1 variant in females with anemia and thrombocytopenia [Internet]. Blood Cells, Molecules, and Diseases. 2021 ; 88[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2021.102545
Artigo de periodico
RHD and RHCE genotyping by next-generation sequencing is an effective strategy to identify molecular variants within sickle cell disease patients (2017)
Dezana, Marcia R.; Ribeiro, Ingrid Helena; Oliveira, Valéria B.; Vieira, Juliana B.; Gomes, Francisco C.; Franco, Lucas Augusto Moysés ; Varuzza, Leonardo; Ribeiro, Roberto; Chinoca, Karen Ziza; Levi, José Eduardo; Krieger, Jose Eduardo ; Pereira, Alexandre Costa; Gualandro, Sandra Fátima Menosi ; Rocha, Vanderson Geraldo ; Mendrone Júnior, Alfredo ; Sabino, Ester Cerdeira ; Dinardo, Carla Luana
Source: Blood Cells, Molecules, and Diseases. Unidade: FM
Subjects: GENÓTIPOS, TÉCNICAS CITOLÓGICAS, REAÇÃO EM CADEIA POR POLIMERASE, TRANSFUSÃO DE SANGUE, SEQUENCIAMENTO GENÉTICO
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DEZANA, Marcia R. et al. RHD and RHCE genotyping by next-generation sequencing is an effective strategy to identify molecular variants within sickle cell disease patients. Blood Cells, Molecules, and Diseases, v. 65, n. 1, p. 8-15, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.bcmd.2017.03.014. Acesso em: 17 mar. 2026.
APA
Dezana, M. R., Ribeiro, I. H., Oliveira, V. B., Vieira, J. B., Gomes, F. C., Franco, L. A. M., et al. (2017). RHD and RHCE genotyping by next-generation sequencing is an effective strategy to identify molecular variants within sickle cell disease patients. Blood Cells, Molecules, and Diseases, 65( 1), 8-15. doi:10.1016/j.bcmd.2017.03.014
NLM
Dezana MR, Ribeiro IH, Oliveira VB, Vieira JB, Gomes FC, Franco LAM, Varuzza L, Ribeiro R, Chinoca KZ, Levi JE, Krieger JE, Pereira AC, Gualandro SFM, Rocha VG, Mendrone Júnior A, Sabino EC, Dinardo CL. RHD and RHCE genotyping by next-generation sequencing is an effective strategy to identify molecular variants within sickle cell disease patients [Internet]. Blood Cells, Molecules, and Diseases. 2017 ; 65( 1): 8-15.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2017.03.014
Vancouver
Dezana MR, Ribeiro IH, Oliveira VB, Vieira JB, Gomes FC, Franco LAM, Varuzza L, Ribeiro R, Chinoca KZ, Levi JE, Krieger JE, Pereira AC, Gualandro SFM, Rocha VG, Mendrone Júnior A, Sabino EC, Dinardo CL. RHD and RHCE genotyping by next-generation sequencing is an effective strategy to identify molecular variants within sickle cell disease patients [Internet]. Blood Cells, Molecules, and Diseases. 2017 ; 65( 1): 8-15.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2017.03.014
Artigo de periodico
The expression of Death Inducer-Obliterator (DIDO) variants in Myeloproliferative Neoplasms (2016)
Berzoti-Coelho, Maria Gabriela; Ferreira, Aline Fernanda; Nunes, Natalia de Souza; Pinto, Mariana Tomazini; Rocha Júnior, Maurício Cristiano; Simões, Belinda Pinto; Martínez-A, Carlos; Souto, Elizabeth Xisto; Panepucci, Rodrigo Alexandre; Covas, Dimas Tadeu; Kashima, Simone; Castro, Fabíola Attié de
Source: Blood Cells, Molecules, and Diseases. Unidades: FMRP, FCFRP
Subjects: LEUCEMIA MIELOIDE, DOENÇA CRÔNICA, APOPTOSE, TROMBOCITOSE
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ABNT
BERZOTI-COELHO, Maria Gabriela et al. The expression of Death Inducer-Obliterator (DIDO) variants in Myeloproliferative Neoplasms. Blood Cells, Molecules, and Diseases, v. 59, p. 25-30, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.bcmd.2016.03.008. Acesso em: 17 mar. 2026.
APA
Berzoti-Coelho, M. G., Ferreira, A. F., Nunes, N. de S., Pinto, M. T., Rocha Júnior, M. C., Simões, B. P., et al. (2016). The expression of Death Inducer-Obliterator (DIDO) variants in Myeloproliferative Neoplasms. Blood Cells, Molecules, and Diseases, 59, 25-30. doi:10.1016/j.bcmd.2016.03.008
NLM
Berzoti-Coelho MG, Ferreira AF, Nunes N de S, Pinto MT, Rocha Júnior MC, Simões BP, Martínez-A C, Souto EX, Panepucci RA, Covas DT, Kashima S, Castro FA de. The expression of Death Inducer-Obliterator (DIDO) variants in Myeloproliferative Neoplasms [Internet]. Blood Cells, Molecules, and Diseases. 2016 ; 59 25-30.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2016.03.008
Vancouver
Berzoti-Coelho MG, Ferreira AF, Nunes N de S, Pinto MT, Rocha Júnior MC, Simões BP, Martínez-A C, Souto EX, Panepucci RA, Covas DT, Kashima S, Castro FA de. The expression of Death Inducer-Obliterator (DIDO) variants in Myeloproliferative Neoplasms [Internet]. Blood Cells, Molecules, and Diseases. 2016 ; 59 25-30.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2016.03.008
Artigo de periodico
CD18 deficiency evolving to megakaryocytic (M7) acute myeloid leukemia: Case report (2014)
Vasconcelos, Dewton de Moraes; Beitler, Beatriz; Martinez, Gracia A.; Pereira, Juliana; Amigo Filho, José Ulysses; Klautau, Giselle Burlamaqui; Lian, Yu Cheng; Della Negra, Marinella; Duarte, Alberto José da Silva
Source: Blood Cells, Molecules, and Diseases. Unidade: FM
Subjects: LEUCEMIA, ANTÉNOS, GLICOPROTEÍNAS
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ABNT
VASCONCELOS, Dewton de Moraes et al. CD18 deficiency evolving to megakaryocytic (M7) acute myeloid leukemia: Case report. Blood Cells, Molecules, and Diseases, v. 53, n. 4, p. 180-184, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.bcmd.2014.07.005. Acesso em: 17 mar. 2026.
APA
Vasconcelos, D. de M., Beitler, B., Martinez, G. A., Pereira, J., Amigo Filho, J. U., Klautau, G. B., et al. (2014). CD18 deficiency evolving to megakaryocytic (M7) acute myeloid leukemia: Case report. Blood Cells, Molecules, and Diseases, 53( 4), 180-184. doi:10.1016/j.bcmd.2014.07.005
NLM
Vasconcelos D de M, Beitler B, Martinez GA, Pereira J, Amigo Filho JU, Klautau GB, Lian YC, Della Negra M, Duarte AJ da S. CD18 deficiency evolving to megakaryocytic (M7) acute myeloid leukemia: Case report [Internet]. Blood Cells, Molecules, and Diseases. 2014 ; 53( 4): 180-184.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2014.07.005
Vancouver
Vasconcelos D de M, Beitler B, Martinez GA, Pereira J, Amigo Filho JU, Klautau GB, Lian YC, Della Negra M, Duarte AJ da S. CD18 deficiency evolving to megakaryocytic (M7) acute myeloid leukemia: Case report [Internet]. Blood Cells, Molecules, and Diseases. 2014 ; 53( 4): 180-184.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2014.07.005
Artigo de periodico
ABCB1 haplotype is associated with major molecular response in chronic myeloid leukemia patients treated with standard-dose of imatinib (2012)
Vivona, Douglas; Bueno, Carolina Tosin; Lima, Luciene Terezinha de; Hirata, Rosario Dominguez Crespo ; Hirata, Mario Hiroyuki ; Luchessi, André Ducati; Zanichelli, Maria Aparecida; Chiattone, Carlos Sérgio; Guerra-Shinohara, Elvira Maria
Source: Blood Cells, Molecules, and Diseases. Unidade: FCF
Subjects: LEUCEMIA MIELOIDE AGUDA, POLIMORFISMO
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ABNT
VIVONA, Douglas et al. ABCB1 haplotype is associated with major molecular response in chronic myeloid leukemia patients treated with standard-dose of imatinib. Blood Cells, Molecules, and Diseases, v. 48, n. 2, p. 132-136, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.bcmd.2011.11.001. Acesso em: 17 mar. 2026.
APA
Vivona, D., Bueno, C. T., Lima, L. T. de, Hirata, R. D. C., Hirata, M. H., Luchessi, A. D., et al. (2012). ABCB1 haplotype is associated with major molecular response in chronic myeloid leukemia patients treated with standard-dose of imatinib. Blood Cells, Molecules, and Diseases, 48( 2), 132-136. doi:10.1016/j.bcmd.2011.11.001
NLM
Vivona D, Bueno CT, Lima LT de, Hirata RDC, Hirata MH, Luchessi AD, Zanichelli MA, Chiattone CS, Guerra-Shinohara EM. ABCB1 haplotype is associated with major molecular response in chronic myeloid leukemia patients treated with standard-dose of imatinib [Internet]. Blood Cells, Molecules, and Diseases. 2012 ; 48( 2): 132-136.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2011.11.001
Vancouver
Vivona D, Bueno CT, Lima LT de, Hirata RDC, Hirata MH, Luchessi AD, Zanichelli MA, Chiattone CS, Guerra-Shinohara EM. ABCB1 haplotype is associated with major molecular response in chronic myeloid leukemia patients treated with standard-dose of imatinib [Internet]. Blood Cells, Molecules, and Diseases. 2012 ; 48( 2): 132-136.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2011.11.001
Artigo de periodico
Hereditary hemochromatosis: mutations in genes involved in iron homeostasis in Brazilian patients (2011)
Santos, Paulo Caleb Junior de Lima; Cançado, Rodolfo Delfini; Pereira, Alexandre C; Schettert, Isolmar Tadeu; Soares, Renata A. G; Pagliusi, Regina Alexandre; Hirata, Rosario Dominguez Crespo ; Hirata, Mario Hiroyuki ; Teixiera, Ana C; Figueiredo, Maria Stella; Chiattone, Carlos Sérgio; Krieger, José Eduardo; Guerra-Shinohara, Elvira Maria
Source: Blood Cells, Molecules, and Diseases. Unidades: FCF, FM
Subjects: HEMOCROMATOSE, SEQUENCIAMENTO GENÉTICO
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SANTOS, Paulo Caleb Junior de Lima et al. Hereditary hemochromatosis: mutations in genes involved in iron homeostasis in Brazilian patients. Blood Cells, Molecules, and Diseases, v. 46, n. 4, p. 302-307, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.bcmd.2011.02.008. Acesso em: 17 mar. 2026.
APA
Santos, P. C. J. de L., Cançado, R. D., Pereira, A. C., Schettert, I. T., Soares, R. A. G., Pagliusi, R. A., et al. (2011). Hereditary hemochromatosis: mutations in genes involved in iron homeostasis in Brazilian patients. Blood Cells, Molecules, and Diseases, 46( 4), 302-307. doi:10.1016/j.bcmd.2011.02.008
NLM
Santos PCJ de L, Cançado RD, Pereira AC, Schettert IT, Soares RAG, Pagliusi RA, Hirata RDC, Hirata MH, Teixiera AC, Figueiredo MS, Chiattone CS, Krieger JE, Guerra-Shinohara EM. Hereditary hemochromatosis: mutations in genes involved in iron homeostasis in Brazilian patients [Internet]. Blood Cells, Molecules, and Diseases. 2011 ; 46( 4): 302-307.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2011.02.008
Vancouver
Santos PCJ de L, Cançado RD, Pereira AC, Schettert IT, Soares RAG, Pagliusi RA, Hirata RDC, Hirata MH, Teixiera AC, Figueiredo MS, Chiattone CS, Krieger JE, Guerra-Shinohara EM. Hereditary hemochromatosis: mutations in genes involved in iron homeostasis in Brazilian patients [Internet]. Blood Cells, Molecules, and Diseases. 2011 ; 46( 4): 302-307.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2011.02.008
Artigo de periodico
Increased expression of protease-activated receptor 1 (PAR-1) in human leukemias (2011)
Veiga, Camilla de S. B.; Carneiro-Lobo, Tatiana C.; Coelho, Cláudia J. B. P.; Carvalho, Silvia M. F.; Maia, Raquel C.; Vasconcelos, Flávia C.; Abdelhay, Eliana; Mencalha, André L.; Ferreira, Aline F.; Castro, Fabíola Attié de; Monteiro, Robson Q.
Source: Blood Cells, Molecules, and Diseases. Unidade: FCFRP
Subjects: LEUCEMIA MIELOIDE AGUDA, RECEPTORES
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ABNT
VEIGA, Camilla de S. B. et al. Increased expression of protease-activated receptor 1 (PAR-1) in human leukemias. Blood Cells, Molecules, and Diseases, v. 46, n. 3, p. 230-234, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.bcmd.2010.12.005. Acesso em: 17 mar. 2026.
APA
Veiga, C. de S. B., Carneiro-Lobo, T. C., Coelho, C. J. B. P., Carvalho, S. M. F., Maia, R. C., Vasconcelos, F. C., et al. (2011). Increased expression of protease-activated receptor 1 (PAR-1) in human leukemias. Blood Cells, Molecules, and Diseases, 46( 3), 230-234. doi:10.1016/j.bcmd.2010.12.005
NLM
Veiga C de SB, Carneiro-Lobo TC, Coelho CJBP, Carvalho SMF, Maia RC, Vasconcelos FC, Abdelhay E, Mencalha AL, Ferreira AF, Castro FA de, Monteiro RQ. Increased expression of protease-activated receptor 1 (PAR-1) in human leukemias [Internet]. Blood Cells, Molecules, and Diseases. 2011 ; 46( 3): 230-234.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2010.12.005
Vancouver
Veiga C de SB, Carneiro-Lobo TC, Coelho CJBP, Carvalho SMF, Maia RC, Vasconcelos FC, Abdelhay E, Mencalha AL, Ferreira AF, Castro FA de, Monteiro RQ. Increased expression of protease-activated receptor 1 (PAR-1) in human leukemias [Internet]. Blood Cells, Molecules, and Diseases. 2011 ; 46( 3): 230-234.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2010.12.005
Artigo de periodico
HFE gene mutations in patients with primary iron overload: is there a significant improvement in molecular diagnosis yield with HFE sequencing (2010)
Santos, Paulo Caleb Junior de Lima; Pereira, Alexandre C; Cançado, Rodolfo Delfini; Schttert, Isolmar T; Sobreira, Tiago J. Paschoal; Oliveira, Paulo S. L; Hirata, Rosario Dominguez Crespo ; Hirata, Mario Hiroyuki ; Figueiredo, Maria Stella; Chiattone, Carlos Sérgio; Krieger, José Eduardo; Guerra-Shinohara, Elvira Maria
Source: Blood Cells, Molecules, and Diseases. Unidades: FCF, FM
Subjects: MUTAÇÃO GENÉTICA, HEMOCROMATOSE, MODELAGEM MOLECULAR
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ABNT
SANTOS, Paulo Caleb Junior de Lima et al. HFE gene mutations in patients with primary iron overload: is there a significant improvement in molecular diagnosis yield with HFE sequencing. Blood Cells, Molecules, and Diseases, v. 45, n. 4, p. 302-307, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.bcmd.2010.08.008. Acesso em: 17 mar. 2026.
APA
Santos, P. C. J. de L., Pereira, A. C., Cançado, R. D., Schttert, I. T., Sobreira, T. J. P., Oliveira, P. S. L., et al. (2010). HFE gene mutations in patients with primary iron overload: is there a significant improvement in molecular diagnosis yield with HFE sequencing. Blood Cells, Molecules, and Diseases, 45( 4), 302-307. doi:10.1016/j.bcmd.2010.08.008
NLM
Santos PCJ de L, Pereira AC, Cançado RD, Schttert IT, Sobreira TJP, Oliveira PSL, Hirata RDC, Hirata MH, Figueiredo MS, Chiattone CS, Krieger JE, Guerra-Shinohara EM. HFE gene mutations in patients with primary iron overload: is there a significant improvement in molecular diagnosis yield with HFE sequencing [Internet]. Blood Cells, Molecules, and Diseases. 2010 ; 45( 4): 302-307.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2010.08.008
Vancouver
Santos PCJ de L, Pereira AC, Cançado RD, Schttert IT, Sobreira TJP, Oliveira PSL, Hirata RDC, Hirata MH, Figueiredo MS, Chiattone CS, Krieger JE, Guerra-Shinohara EM. HFE gene mutations in patients with primary iron overload: is there a significant improvement in molecular diagnosis yield with HFE sequencing [Internet]. Blood Cells, Molecules, and Diseases. 2010 ; 45( 4): 302-307.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2010.08.008
Artigo de periodico
A point mutation in the RNA-binding domain i results in decrease of PKR activation in acute lymphoblastic leukemia (2005)
Murad, Joana Maria; Tone, Luiz Gonzaga; Souza, Liliana Rodrigues de; De Lucca, Fernando Luiz
Source: Blood Cells, Molecules, and Diseases. Unidade: FMRP
Subjects: LEUCEMIA, PROTEÍNAS
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ABNT
MURAD, Joana Maria et al. A point mutation in the RNA-binding domain i results in decrease of PKR activation in acute lymphoblastic leukemia. Blood Cells, Molecules, and Diseases, v. 34, p. 1-5, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.bcmd.2004.08.025. Acesso em: 17 mar. 2026.
APA
Murad, J. M., Tone, L. G., Souza, L. R. de, & De Lucca, F. L. (2005). A point mutation in the RNA-binding domain i results in decrease of PKR activation in acute lymphoblastic leukemia. Blood Cells, Molecules, and Diseases, 34, 1-5. doi:10.1016/j.bcmd.2004.08.025
NLM
Murad JM, Tone LG, Souza LR de, De Lucca FL. A point mutation in the RNA-binding domain i results in decrease of PKR activation in acute lymphoblastic leukemia [Internet]. Blood Cells, Molecules, and Diseases. 2005 ; 34 1-5.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2004.08.025
Vancouver
Murad JM, Tone LG, Souza LR de, De Lucca FL. A point mutation in the RNA-binding domain i results in decrease of PKR activation in acute lymphoblastic leukemia [Internet]. Blood Cells, Molecules, and Diseases. 2005 ; 34 1-5.[citado 2026 mar. 17 ] Available from: https://doi.org/10.1016/j.bcmd.2004.08.025

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