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Artigo de periodico
Influence of high altitude on the expression of HIF-1 and on the prognosis of Ecuadorian patients with gastric adenocarcinoma (2022)
Barrera, Edwin Cevallos; Martinez, Edson Zangiacomi ; Brunaldi, Mariângela Ottoboni; Donadi, Eduardo Antônio; Sankarankutty, Ajith Kumar ; Kemp, Rafael; Santos, José Sebastião dos
Source: Oncotarget. Unidade: FMRP
Subjects: ADENOCARCINOMA, ESTÔMAGO, NEOPLASIAS, ANÓXIA, ESTUDOS RETROSPECTIVOS, PROGNÓSTICO
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ABNT
BARRERA, Edwin Cevallos et al. Influence of high altitude on the expression of HIF-1 and on the prognosis of Ecuadorian patients with gastric adenocarcinoma. Oncotarget, v. 13, p. 1043-1053, 2022Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.28275. Acesso em: 18 nov. 2024.
APA
Barrera, E. C., Martinez, E. Z., Brunaldi, M. O., Donadi, E. A., Sankarankutty, A. K., Kemp, R., & Santos, J. S. dos. (2022). Influence of high altitude on the expression of HIF-1 and on the prognosis of Ecuadorian patients with gastric adenocarcinoma. Oncotarget, 13, 1043-1053. doi:10.18632/oncotarget.28275
NLM
Barrera EC, Martinez EZ, Brunaldi MO, Donadi EA, Sankarankutty AK, Kemp R, Santos JS dos. Influence of high altitude on the expression of HIF-1 and on the prognosis of Ecuadorian patients with gastric adenocarcinoma [Internet]. Oncotarget. 2022 ; 13 1043-1053.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.28275
Vancouver
Barrera EC, Martinez EZ, Brunaldi MO, Donadi EA, Sankarankutty AK, Kemp R, Santos JS dos. Influence of high altitude on the expression of HIF-1 and on the prognosis of Ecuadorian patients with gastric adenocarcinoma [Internet]. Oncotarget. 2022 ; 13 1043-1053.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.28275
Artigo de periodico
Distinct pattern of one-carbon metabolism, a nutrient-sensitive pathway, in invasive breast cancer: a metabolomic study (2020)
Santos, Jéssica Reis; Waitzberg, Dan Linetzky ; Silva, Ismael Dale Cotrim Guerreiro da; Tortelli Junior, Tharcisio Citrangulo; Barros, Luciana Rodrigues Carvalho; Canuto, Gisele André Baptista; Faccio, Andrea Tedesco; Yamaguchi, Lydia Fumiko; Kato, Massuo Jorge ; Tavares, Marina Franco Maggi ; Martinez, Ana Cristina; Logullo, Angela Flavia; Torrinhas, Raquel Suzana M. M; Ravacci, Graziela
Source: Oncotarget. Unidades: FM, IQ
Subjects: NEOPLASIAS MAMÁRIAS, CARBONO
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ABNT
SANTOS, Jéssica Reis et al. Distinct pattern of one-carbon metabolism, a nutrient-sensitive pathway, in invasive breast cancer: a metabolomic study. Oncotarget, v. 11, n. 18 p. 1637-1652, 2020Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.27575. Acesso em: 18 nov. 2024.
APA
Santos, J. R., Waitzberg, D. L., Silva, I. D. C. G. da, Tortelli Junior, T. C., Barros, L. R. C., Canuto, G. A. B., et al. (2020). Distinct pattern of one-carbon metabolism, a nutrient-sensitive pathway, in invasive breast cancer: a metabolomic study. Oncotarget, 11( 18 p. 1637-1652). doi:10.18632/oncotarget.27575
NLM
Santos JR, Waitzberg DL, Silva IDCG da, Tortelli Junior TC, Barros LRC, Canuto GAB, Faccio AT, Yamaguchi LF, Kato MJ, Tavares MFM, Martinez AC, Logullo AF, Torrinhas RSMM, Ravacci G. Distinct pattern of one-carbon metabolism, a nutrient-sensitive pathway, in invasive breast cancer: a metabolomic study [Internet]. Oncotarget. 2020 ; 11( 18 p. 1637-1652):[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.27575
Vancouver
Santos JR, Waitzberg DL, Silva IDCG da, Tortelli Junior TC, Barros LRC, Canuto GAB, Faccio AT, Yamaguchi LF, Kato MJ, Tavares MFM, Martinez AC, Logullo AF, Torrinhas RSMM, Ravacci G. Distinct pattern of one-carbon metabolism, a nutrient-sensitive pathway, in invasive breast cancer: a metabolomic study [Internet]. Oncotarget. 2020 ; 11( 18 p. 1637-1652):[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.27575
Artigo de periodico
Chronic activation profile of circulating CD8+T cells in Sezary syndrome (2018)
Torrealba, Marina Passos; Pereira, Juliana; Duarte, Alberto J. S.; Sato, Maria N.
Source: Oncotarget. Unidade: FM
Subjects: NEOPLASIAS CUTÂNEAS, LINFÓCITOS T, SOBREVIDA
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ABNT
TORREALBA, Marina Passos et al. Chronic activation profile of circulating CD8+T cells in Sezary syndrome. Oncotarget, v. 9, n. 3, p. 3497-3506, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.23334. Acesso em: 18 nov. 2024.
APA
Torrealba, M. P., Pereira, J., Duarte, A. J. S., & Sato, M. N. (2018). Chronic activation profile of circulating CD8+T cells in Sezary syndrome. Oncotarget, 9( 3), 3497-3506. doi:10.18632/oncotarget.23334
NLM
Torrealba MP, Pereira J, Duarte AJS, Sato MN. Chronic activation profile of circulating CD8+T cells in Sezary syndrome [Internet]. Oncotarget. 2018 ; 9( 3): 3497-3506.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.23334
Vancouver
Torrealba MP, Pereira J, Duarte AJS, Sato MN. Chronic activation profile of circulating CD8+T cells in Sezary syndrome [Internet]. Oncotarget. 2018 ; 9( 3): 3497-3506.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.23334
Artigo de periodico
Strong SOD2 expression and HPV-16/18 positivity are independent events in cervical cancer (2018)
Santos, Silvia Helena Rabelo ; Termini, Lara ; Boccardo, Enrique ; Derchain, Sophie Françoise Mauricette ; Longatto Filho, Adhemar ; Andreoli, Maria Antonieta Avilla; Costa, Maria Cecília; Nunes, Rafaella Almeida Lima ; Andrade, Liliana Aparecida Lucci de Ângelo ; Villa, Luisa Lina ; Zeferino, Luiz Carlos
Source: Oncotarget. Unidades: ICB, FM
Subjects: INFECÇÕES POR PAPILLOMAVIRUS, NEOPLASIAS DO COLO UTERINO, HPV, SUPERÓXIDO DISMUTASE
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ABNT
SANTOS, Silvia Helena Rabelo et al. Strong SOD2 expression and HPV-16/18 positivity are independent events in cervical cancer. Oncotarget, v. 9, n. 31, p. 21630-21640, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.24850. Acesso em: 18 nov. 2024.
APA
Santos, S. H. R., Termini, L., Boccardo, E., Derchain, S. F. M., Longatto Filho, A., Andreoli, M. A. A., et al. (2018). Strong SOD2 expression and HPV-16/18 positivity are independent events in cervical cancer. Oncotarget, 9( 31), 21630-21640. doi:10.18632/oncotarget.24850
NLM
Santos SHR, Termini L, Boccardo E, Derchain SFM, Longatto Filho A, Andreoli MAA, Costa MC, Nunes RAL, Andrade LAL de Â, Villa LL, Zeferino LC. Strong SOD2 expression and HPV-16/18 positivity are independent events in cervical cancer [Internet]. Oncotarget. 2018 ; 9( 31): 21630-21640.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.24850
Vancouver
Santos SHR, Termini L, Boccardo E, Derchain SFM, Longatto Filho A, Andreoli MAA, Costa MC, Nunes RAL, Andrade LAL de Â, Villa LL, Zeferino LC. Strong SOD2 expression and HPV-16/18 positivity are independent events in cervical cancer [Internet]. Oncotarget. 2018 ; 9( 31): 21630-21640.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.24850
Artigo de periodico
Aptamers: novelty tools for cancer biology (2018)
Pereira, Ricardo L; Nascimento, Isis C; Santos, Ana P; Ogusuku, Isabella E. Y; Lameu, Claudiana ; Mayer, Günter; Ulrich, Henning
Source: Oncotarget. Unidade: IQ
Subjects: NEOPLASIAS, ANTICORPOS MONOCLONAIS
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ABNT
PEREIRA, Ricardo L et al. Aptamers: novelty tools for cancer biology. Oncotarget, v. 9, n. 42, p. 26934-26953, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.25260. Acesso em: 18 nov. 2024.
APA
Pereira, R. L., Nascimento, I. C., Santos, A. P., Ogusuku, I. E. Y., Lameu, C., Mayer, G., & Ulrich, H. (2018). Aptamers: novelty tools for cancer biology. Oncotarget, 9( 42), 26934-26953. doi:10.18632/oncotarget.25260
NLM
Pereira RL, Nascimento IC, Santos AP, Ogusuku IEY, Lameu C, Mayer G, Ulrich H. Aptamers: novelty tools for cancer biology [Internet]. Oncotarget. 2018 ; 9( 42): 26934-26953.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25260
Vancouver
Pereira RL, Nascimento IC, Santos AP, Ogusuku IEY, Lameu C, Mayer G, Ulrich H. Aptamers: novelty tools for cancer biology [Internet]. Oncotarget. 2018 ; 9( 42): 26934-26953.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25260
Artigo de periodico
The role of E-cadherin and β-catenin in laryngeal cancer (2018)
Nardi, C. E; Dedivitis, R. A; Almeida, R. C; Matos, L. L. de; Cernea, Claudio Roberto
Source: Oncotarget. Unidade: FM
Subjects: NEOPLASIAS LARÍNGEAS, CADERINAS, CITOESQUELETO, IMUNOHISTOLOGIA
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ABNT
NARDI, C. E et al. The role of E-cadherin and β-catenin in laryngeal cancer. Oncotarget, v. 9, n. 53, p. 30199-30209, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.25680. Acesso em: 18 nov. 2024.
APA
Nardi, C. E., Dedivitis, R. A., Almeida, R. C., Matos, L. L. de, & Cernea, C. R. (2018). The role of E-cadherin and β-catenin in laryngeal cancer. Oncotarget, 9( 53), 30199-30209. doi:10.18632/oncotarget.25680
NLM
Nardi CE, Dedivitis RA, Almeida RC, Matos LL de, Cernea CR. The role of E-cadherin and β-catenin in laryngeal cancer [Internet]. Oncotarget. 2018 ; 9( 53): 30199-30209.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25680
Vancouver
Nardi CE, Dedivitis RA, Almeida RC, Matos LL de, Cernea CR. The role of E-cadherin and β-catenin in laryngeal cancer [Internet]. Oncotarget. 2018 ; 9( 53): 30199-30209.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25680
Artigo de periodico
Highly expressed placental miRNAs control key biological processes in human cancer cell lines (2018)
Vidal, Daniel Onofre; Ramão, Anelisa ; Pinheiro, Daniel Guariz; Muys, Bruna Rodrigues ; Lorenzi, Julio Cesar Cetrulo; Alves, Cleidson de Pádua; Zanette, Dalila Luciola; Molfetta, Greice Andreotti de ; Duarte, Geraldo ; Silva Junior, Wilson Araújo
Source: Oncotarget. Unidade: FMRP
Subjects: PLACENTA, CARCINOGÊNESE, REGULAÇÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VIDAL, Daniel Onofre et al. Highly expressed placental miRNAs control key biological processes in human cancer cell lines. Oncotarget, v. 9, n. 34, p. 23554-23563, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.25264. Acesso em: 18 nov. 2024.
APA
Vidal, D. O., Ramão, A., Pinheiro, D. G., Muys, B. R., Lorenzi, J. C. C., Alves, C. de P., et al. (2018). Highly expressed placental miRNAs control key biological processes in human cancer cell lines. Oncotarget, 9( 34), 23554-23563. doi:10.18632/oncotarget.25264
NLM
Vidal DO, Ramão A, Pinheiro DG, Muys BR, Lorenzi JCC, Alves C de P, Zanette DL, Molfetta GA de, Duarte G, Silva Junior WA. Highly expressed placental miRNAs control key biological processes in human cancer cell lines [Internet]. Oncotarget. 2018 ; 9( 34): 23554-23563.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25264
Vancouver
Vidal DO, Ramão A, Pinheiro DG, Muys BR, Lorenzi JCC, Alves C de P, Zanette DL, Molfetta GA de, Duarte G, Silva Junior WA. Highly expressed placental miRNAs control key biological processes in human cancer cell lines [Internet]. Oncotarget. 2018 ; 9( 34): 23554-23563.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25264
Artigo de periodico
Mesenchymal stem cells enhance tumorigenic properties of human glioblastoma through independent cell-cell communication mechanisms (2018)
Rodini, Carolina Oliveira; Silva, Patrícia Benites Gonçalves da; Assoni, Amanda Faria ; Carvalho, Valdemir Melechco ; Okamoto, Oswaldo Keith
Source: Oncotarget. Unidade: IB
Subjects: CÉLULAS-TRONCO, TUMOR CARCINOIDE, NEOPLASIAS CEREBRAIS, GLICOPROTEÍNAS, EXPRESSÃO GÊNICA
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ABNT
RODINI, Carolina Oliveira et al. Mesenchymal stem cells enhance tumorigenic properties of human glioblastoma through independent cell-cell communication mechanisms. Oncotarget, v. 9, n. 37, p. 24766-24777, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.25346. Acesso em: 18 nov. 2024.
APA
Rodini, C. O., Silva, P. B. G. da, Assoni, A. F., Carvalho, V. M., & Okamoto, O. K. (2018). Mesenchymal stem cells enhance tumorigenic properties of human glioblastoma through independent cell-cell communication mechanisms. Oncotarget, 9( 37), 24766-24777. doi:10.18632/oncotarget.25346
NLM
Rodini CO, Silva PBG da, Assoni AF, Carvalho VM, Okamoto OK. Mesenchymal stem cells enhance tumorigenic properties of human glioblastoma through independent cell-cell communication mechanisms [Internet]. Oncotarget. 2018 ; 9( 37): 24766-24777.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25346
Vancouver
Rodini CO, Silva PBG da, Assoni AF, Carvalho VM, Okamoto OK. Mesenchymal stem cells enhance tumorigenic properties of human glioblastoma through independent cell-cell communication mechanisms [Internet]. Oncotarget. 2018 ; 9( 37): 24766-24777.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25346
Artigo de periodico
Establishment of primary cell culture and an intracranial xenograft model of pediatric ependymoma: a prospect for therapy development and understanding of tumor biology (2018)
Pavon, Lorena Favaro; Sibov, Tatiana Tais; Toledo, Silvia Regina Caminada de; Oliveira, Daniela Mara de; Cabral, Francisco Romero; Souza, Jean Gabriel de; Boufleur, Pamela; Marti, Luciana C.; Malheiros, Jackeline Moraes; Cruz, Edgar Ferreira da; Paiva, Fernando Fernandes ; Malheiros, Suzana M. F.; Paiva Neto, Manoel A.; Tannús, Alberto ; Mascarenhas, Sérgio; Silva, Nasjla Saba; Cappellano, Andrea Maria; Petrilli, Antonio Sérgio; Chudzinski-Tavassi, Ana Marisa; Cavalheiro, Sérgio
Source: Oncotarget. Unidade: IFSC
Subjects: NEOPLASIAS CEREBRAIS, MONITORAMENTO, CULTURA DE CÉLULAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PAVON, Lorena Favaro et al. Establishment of primary cell culture and an intracranial xenograft model of pediatric ependymoma: a prospect for therapy development and understanding of tumor biology. Oncotarget, v. 9, n. 31, p. 21731-21743, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.24932. Acesso em: 18 nov. 2024.
APA
Pavon, L. F., Sibov, T. T., Toledo, S. R. C. de, Oliveira, D. M. de, Cabral, F. R., Souza, J. G. de, et al. (2018). Establishment of primary cell culture and an intracranial xenograft model of pediatric ependymoma: a prospect for therapy development and understanding of tumor biology. Oncotarget, 9( 31), 21731-21743. doi:10.18632/oncotarget.24932
NLM
Pavon LF, Sibov TT, Toledo SRC de, Oliveira DM de, Cabral FR, Souza JG de, Boufleur P, Marti LC, Malheiros JM, Cruz EF da, Paiva FF, Malheiros SMF, Paiva Neto MA, Tannús A, Mascarenhas S, Silva NS, Cappellano AM, Petrilli AS, Chudzinski-Tavassi AM, Cavalheiro S. Establishment of primary cell culture and an intracranial xenograft model of pediatric ependymoma: a prospect for therapy development and understanding of tumor biology [Internet]. Oncotarget. 2018 ; 9( 31): 21731-21743.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.24932
Vancouver
Pavon LF, Sibov TT, Toledo SRC de, Oliveira DM de, Cabral FR, Souza JG de, Boufleur P, Marti LC, Malheiros JM, Cruz EF da, Paiva FF, Malheiros SMF, Paiva Neto MA, Tannús A, Mascarenhas S, Silva NS, Cappellano AM, Petrilli AS, Chudzinski-Tavassi AM, Cavalheiro S. Establishment of primary cell culture and an intracranial xenograft model of pediatric ependymoma: a prospect for therapy development and understanding of tumor biology [Internet]. Oncotarget. 2018 ; 9( 31): 21731-21743.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.24932
Artigo de periodico
Analysis of KIT gene mutations in patients with melanoma of the head and neck mucosa: a retrospective clinical report (2018)
Mendonça, U. B. T. de; Cernea, Claudio Roberto; Matos, L. L; Lima, R. R. M. A
Source: Oncotarget. Unidade: FM
Subjects: MELANOMA, NEOPLASIAS DE CABEÇA E PESCOÇO, MUTAÇÃO GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MENDONÇA, U. B. T. de et al. Analysis of KIT gene mutations in patients with melanoma of the head and neck mucosa: a retrospective clinical report. Oncotarget, v. 9, n. 33, p. 22886-22894, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.25094. Acesso em: 18 nov. 2024.
APA
Mendonça, U. B. T. de, Cernea, C. R., Matos, L. L., & Lima, R. R. M. A. (2018). Analysis of KIT gene mutations in patients with melanoma of the head and neck mucosa: a retrospective clinical report. Oncotarget, 9( 33), 22886-22894. doi:10.18632/oncotarget.25094
NLM
Mendonça UBT de, Cernea CR, Matos LL, Lima RRMA. Analysis of KIT gene mutations in patients with melanoma of the head and neck mucosa: a retrospective clinical report [Internet]. Oncotarget. 2018 ; 9( 33): 22886-22894.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25094
Vancouver
Mendonça UBT de, Cernea CR, Matos LL, Lima RRMA. Analysis of KIT gene mutations in patients with melanoma of the head and neck mucosa: a retrospective clinical report [Internet]. Oncotarget. 2018 ; 9( 33): 22886-22894.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25094
Artigo de periodico
Cell internalization of 7-ketocholesterol-containing nanoemulsion through LDL receptor reduces melanoma growth in vitro and in vivo: a preliminary report (2018)
Favero, Giovani Marino; Paz, Jessica Liliane; Otake, Andréia Hanada; Maria, Durvanei Augusto; Caldini, Elia Tamaso Espin Garcia; Medeiros, Raphael Salles Scortegagna de; Deus, Débora F; Chammas, Roger; Maranhão, Raul Cavalcante ; Bydlowski, Sérgio Paulo
Source: Oncotarget. Unidades: FM, FCF
Subjects: MELANOMA, COLESTEROL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FAVERO, Giovani Marino et al. Cell internalization of 7-ketocholesterol-containing nanoemulsion through LDL receptor reduces melanoma growth in vitro and in vivo: a preliminary report. Oncotarget, v. 9, n. 18, p. 14160-14174, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.24389. Acesso em: 18 nov. 2024.
APA
Favero, G. M., Paz, J. L., Otake, A. H., Maria, D. A., Caldini, E. T. E. G., Medeiros, R. S. S. de, et al. (2018). Cell internalization of 7-ketocholesterol-containing nanoemulsion through LDL receptor reduces melanoma growth in vitro and in vivo: a preliminary report. Oncotarget, 9( 18), 14160-14174. doi:10.18632/oncotarget.24389
NLM
Favero GM, Paz JL, Otake AH, Maria DA, Caldini ETEG, Medeiros RSS de, Deus DF, Chammas R, Maranhão RC, Bydlowski SP. Cell internalization of 7-ketocholesterol-containing nanoemulsion through LDL receptor reduces melanoma growth in vitro and in vivo: a preliminary report [Internet]. Oncotarget. 2018 ; 9( 18): 14160-14174.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.24389
Vancouver
Favero GM, Paz JL, Otake AH, Maria DA, Caldini ETEG, Medeiros RSS de, Deus DF, Chammas R, Maranhão RC, Bydlowski SP. Cell internalization of 7-ketocholesterol-containing nanoemulsion through LDL receptor reduces melanoma growth in vitro and in vivo: a preliminary report [Internet]. Oncotarget. 2018 ; 9( 18): 14160-14174.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.24389
Artigo de periodico
Somatic mutations in early onset luminal breast cancer (2018)
Encinas, G.; Sabelnykova, V. Y.; Lyra, E. C. de; Katayama, Maria Lúcia Hirata; Maistro, S; Valle, P. W. M. V; Pereira, G. F. L; Rodrigues, L. M; Serio, P. A. M. P; Gouvêa, A. C. R. C. de; Pasini, Fatima Solange; Brentani, Maria Mitzi; Chammas, Roger; Folgueira, Maria Aparecida Azevedo Koike
Source: Oncotarget. Unidade: FM
Subjects: NEOPLASIAS MAMÁRIAS, MUTAÇÃO GENÉTICA, JOVENS, GENÔMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ENCINAS, G. et al. Somatic mutations in early onset luminal breast cancer. Oncotarget, v. 9, n. 32, p. 22460-22479, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.25123. Acesso em: 18 nov. 2024.
APA
Encinas, G., Sabelnykova, V. Y., Lyra, E. C. de, Katayama, M. L. H., Maistro, S., Valle, P. W. M. V., et al. (2018). Somatic mutations in early onset luminal breast cancer. Oncotarget, 9( 32), 22460-22479. doi:10.18632/oncotarget.25123
NLM
Encinas G, Sabelnykova VY, Lyra EC de, Katayama MLH, Maistro S, Valle PWMV, Pereira GFL, Rodrigues LM, Serio PAMP, Gouvêa ACRC de, Pasini FS, Brentani MM, Chammas R, Folgueira MAAK. Somatic mutations in early onset luminal breast cancer [Internet]. Oncotarget. 2018 ; 9( 32): 22460-22479.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25123
Vancouver
Encinas G, Sabelnykova VY, Lyra EC de, Katayama MLH, Maistro S, Valle PWMV, Pereira GFL, Rodrigues LM, Serio PAMP, Gouvêa ACRC de, Pasini FS, Brentani MM, Chammas R, Folgueira MAAK. Somatic mutations in early onset luminal breast cancer [Internet]. Oncotarget. 2018 ; 9( 32): 22460-22479.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25123
Artigo de periodico
Galectin-3 sensitized melanoma cell lines to vemurafenib (PLX4032) induced cell death through prevention of autophagy (2018)
Bustos, S. O; Pereira, G. J. S; Saito, R. F; Gil, C. D; Zanatta, D. B; Smaili, S. S; Chammas, Roger
Source: Oncotarget. Unidade: FM
Subjects: MELANOMA, LECTINAS, INIBIDORES DE ENZIMAS, PRIVAÇÃO DE ALIMENTOS, FAGOCITOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BUSTOS, S. O et al. Galectin-3 sensitized melanoma cell lines to vemurafenib (PLX4032) induced cell death through prevention of autophagy. Oncotarget, v. 9, n. 18, p. 14567-14579, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.24516. Acesso em: 18 nov. 2024.
APA
Bustos, S. O., Pereira, G. J. S., Saito, R. F., Gil, C. D., Zanatta, D. B., Smaili, S. S., & Chammas, R. (2018). Galectin-3 sensitized melanoma cell lines to vemurafenib (PLX4032) induced cell death through prevention of autophagy. Oncotarget, 9( 18), 14567-14579. doi:10.18632/oncotarget.24516
NLM
Bustos SO, Pereira GJS, Saito RF, Gil CD, Zanatta DB, Smaili SS, Chammas R. Galectin-3 sensitized melanoma cell lines to vemurafenib (PLX4032) induced cell death through prevention of autophagy [Internet]. Oncotarget. 2018 ; 9( 18): 14567-14579.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.24516
Vancouver
Bustos SO, Pereira GJS, Saito RF, Gil CD, Zanatta DB, Smaili SS, Chammas R. Galectin-3 sensitized melanoma cell lines to vemurafenib (PLX4032) induced cell death through prevention of autophagy [Internet]. Oncotarget. 2018 ; 9( 18): 14567-14579.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.24516
Artigo de periodico
Inborn-like errors of metabolism are determinants of breast cancer risk, clinical response and survival: a study of human biochemical individuality (2018)
Silva, I. da; Vieira, R. C; Stella, C; Loturco, E; Carvalho, A. L; Veo, C; Neto, C; Silva, S. M; D'amora, P; Salzgeber, M; Maciel, Gustavo Arantes Rosa; Baracat, Edmund Chada; Silva, Clovis Artur Almeida da
Source: Oncotarget. Unidade: FM
Subjects: ERROS INATOS DO METABOLISMO, NEOPLASIAS MAMÁRIAS, SOBREVIDA, PROGNÓSTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, I. da et al. Inborn-like errors of metabolism are determinants of breast cancer risk, clinical response and survival: a study of human biochemical individuality. Oncotarget, v. 9, n. 60, p. 31664-31681, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.25839. Acesso em: 18 nov. 2024.
APA
Silva, I. da, Vieira, R. C., Stella, C., Loturco, E., Carvalho, A. L., Veo, C., et al. (2018). Inborn-like errors of metabolism are determinants of breast cancer risk, clinical response and survival: a study of human biochemical individuality. Oncotarget, 9( 60), 31664-31681. doi:10.18632/oncotarget.25839
NLM
Silva I da, Vieira RC, Stella C, Loturco E, Carvalho AL, Veo C, Neto C, Silva SM, D'amora P, Salzgeber M, Maciel GAR, Baracat EC, Silva CAA da. Inborn-like errors of metabolism are determinants of breast cancer risk, clinical response and survival: a study of human biochemical individuality [Internet]. Oncotarget. 2018 ; 9( 60): 31664-31681.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25839
Vancouver
Silva I da, Vieira RC, Stella C, Loturco E, Carvalho AL, Veo C, Neto C, Silva SM, D'amora P, Salzgeber M, Maciel GAR, Baracat EC, Silva CAA da. Inborn-like errors of metabolism are determinants of breast cancer risk, clinical response and survival: a study of human biochemical individuality [Internet]. Oncotarget. 2018 ; 9( 60): 31664-31681.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.25839
Artigo de periodico
ST2/IL-33 signaling promotes malignant development of experimental squamous cell carcinoma by decreasing NK cells cytotoxicity and modulating the intratumoral cell infiltrate (2018)
Amôr, Nádia Ghinelli ; Oliveira, Carine Ervolino de; Gasparoto, Thaís Helena ; Vilas Boas, Vanessa Garcia ; Perri, Graziela ; Kaneno, Ramon; Lara, Vanessa Soares ; Garlet, Gustavo Pompermaier ; Silva, João Santana da ; Martins, Gislaine Aparecida; Hogaboam, Cory ; Cavassani, Karen Angelica; Campanelli, Ana Paula
Source: Oncotarget. Unidades: FOB, FMRP
Subjects: CARCINOMA DE CÉLULAS ESCAMOSAS, CAMUNDONGOS, INTERLEUCINAS, CARCINÓGENOS QUÍMICOS
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ABNT
AMÔR, Nádia Ghinelli et al. ST2/IL-33 signaling promotes malignant development of experimental squamous cell carcinoma by decreasing NK cells cytotoxicity and modulating the intratumoral cell infiltrate. Oncotarget, v. 9, n. 56, p. 30894-30904, 2018Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.30894. Acesso em: 18 nov. 2024.
APA
Amôr, N. G., Oliveira, C. E. de, Gasparoto, T. H., Vilas Boas, V. G., Perri, G., Kaneno, R., et al. (2018). ST2/IL-33 signaling promotes malignant development of experimental squamous cell carcinoma by decreasing NK cells cytotoxicity and modulating the intratumoral cell infiltrate. Oncotarget, 9( 56), 30894-30904. doi:10.18632/oncotarget.30894
NLM
Amôr NG, Oliveira CE de, Gasparoto TH, Vilas Boas VG, Perri G, Kaneno R, Lara VS, Garlet GP, Silva JS da, Martins GA, Hogaboam C, Cavassani KA, Campanelli AP. ST2/IL-33 signaling promotes malignant development of experimental squamous cell carcinoma by decreasing NK cells cytotoxicity and modulating the intratumoral cell infiltrate [Internet]. Oncotarget. 2018 ; 9( 56): 30894-30904.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.30894
Vancouver
Amôr NG, Oliveira CE de, Gasparoto TH, Vilas Boas VG, Perri G, Kaneno R, Lara VS, Garlet GP, Silva JS da, Martins GA, Hogaboam C, Cavassani KA, Campanelli AP. ST2/IL-33 signaling promotes malignant development of experimental squamous cell carcinoma by decreasing NK cells cytotoxicity and modulating the intratumoral cell infiltrate [Internet]. Oncotarget. 2018 ; 9( 56): 30894-30904.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.30894
Artigo de periodico
Relevance of CCL3/CCR5 axis in oral carcinogenesis (2017)
Silva, Janine Mayra da; Santos, Tálita Pollyanna Moreira dos; Sobral, Lays Martin; Queiroz-Junior, Celso Martins; Rachid, Milene Alvarenga; Proudfoot, Amanda E. I.; Garlet, Gustavo Pompermaier; Batista, Aline Carvalho; Teixeira, Mauro Martins; Leopoldino, Andréia Machado; Russo, Remo Castro; Silva, Tarcília Aparecida
Source: Oncotarget. Unidades: FCFRP, FOB
Subjects: CARCINOGÊNESE, QUIMIOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Janine Mayra da et al. Relevance of CCL3/CCR5 axis in oral carcinogenesis. Oncotarget, v. 8, n. 31, p. 51024-51036, 2017Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.16882. Acesso em: 18 nov. 2024.
APA
Silva, J. M. da, Santos, T. P. M. dos, Sobral, L. M., Queiroz-Junior, C. M., Rachid, M. A., Proudfoot, A. E. I., et al. (2017). Relevance of CCL3/CCR5 axis in oral carcinogenesis. Oncotarget, 8( 31), 51024-51036. doi:10.18632/oncotarget.16882
NLM
Silva JM da, Santos TPM dos, Sobral LM, Queiroz-Junior CM, Rachid MA, Proudfoot AEI, Garlet GP, Batista AC, Teixeira MM, Leopoldino AM, Russo RC, Silva TA. Relevance of CCL3/CCR5 axis in oral carcinogenesis [Internet]. Oncotarget. 2017 ; 8( 31): 51024-51036.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.16882
Vancouver
Silva JM da, Santos TPM dos, Sobral LM, Queiroz-Junior CM, Rachid MA, Proudfoot AEI, Garlet GP, Batista AC, Teixeira MM, Leopoldino AM, Russo RC, Silva TA. Relevance of CCL3/CCR5 axis in oral carcinogenesis [Internet]. Oncotarget. 2017 ; 8( 31): 51024-51036.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.16882
Artigo de periodico
Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death (2017)
Tortelli Junior, Tharcisio Citrangulo; Godoy, Lyris Martins Franco de; Souza, Gustavo Antonio de; Bonatto, Diego; Otake, Andreia Hanada; Saito, Renata de Freitas; Rosa, Jose Cesar; Greene, Lewis Joel; Chammas, Roger
Source: Oncotarget. Unidades: FMRP, FM
Subjects: APOPTOSE, NEOPLASIAS CUTÂNEAS, MANIFESTAÇÕES CUTÂNEAS, MELANOMA
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ABNT
TORTELLI JUNIOR, Tharcisio Citrangulo et al. Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death. Oncotarget, v. 8, n. 26, p. 43114-43129, 2017Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.17810. Acesso em: 18 nov. 2024.
APA
Tortelli Junior, T. C., Godoy, L. M. F. de, Souza, G. A. de, Bonatto, D., Otake, A. H., Saito, R. de F., et al. (2017). Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death. Oncotarget, 8( 26), 43114-43129. doi:10.18632/oncotarget.17810
NLM
Tortelli Junior TC, Godoy LMF de, Souza GA de, Bonatto D, Otake AH, Saito R de F, Rosa JC, Greene LJ, Chammas R. Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death [Internet]. Oncotarget. 2017 ; 8( 26): 43114-43129.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.17810
Vancouver
Tortelli Junior TC, Godoy LMF de, Souza GA de, Bonatto D, Otake AH, Saito R de F, Rosa JC, Greene LJ, Chammas R. Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death [Internet]. Oncotarget. 2017 ; 8( 26): 43114-43129.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.17810
Artigo de periodico
GLUT1 expression in pediatric adrenocortical tumors: a promising candidate to predict clinical behavior (2017)
Pinheiro, Céline; Granja, Sara; Longatto-Filho, Adhemar; Faria, André M.; Fragoso, Maria C. B. V.; Lovisolo, Silvana M.; Bonatelli, Murilo; Costa, Ricardo F. A.; Lerário, Antonio M.; Almeida, Madson Q.; Baltazar, Fátima; Zerbini, Maria C. N.
Source: Oncotarget. Unidades: HU, FM
Subjects: CRIANÇAS, TERAPÊUTICA MÉDICA, NEOPLASIAS DAS GLÂNDULAS SUPRARRENAIS, IMUNOHISTOQUÍMICA
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ABNT
PINHEIRO, Céline et al. GLUT1 expression in pediatric adrenocortical tumors: a promising candidate to predict clinical behavior. Oncotarget, v. 8, n. 38, p. 63835-63845, 2017Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.19135. Acesso em: 18 nov. 2024.
APA
Pinheiro, C., Granja, S., Longatto-Filho, A., Faria, A. M., Fragoso, M. C. B. V., Lovisolo, S. M., et al. (2017). GLUT1 expression in pediatric adrenocortical tumors: a promising candidate to predict clinical behavior. Oncotarget, 8( 38), 63835-63845. doi:10.18632/oncotarget.19135
NLM
Pinheiro C, Granja S, Longatto-Filho A, Faria AM, Fragoso MCBV, Lovisolo SM, Bonatelli M, Costa RFA, Lerário AM, Almeida MQ, Baltazar F, Zerbini MCN. GLUT1 expression in pediatric adrenocortical tumors: a promising candidate to predict clinical behavior [Internet]. Oncotarget. 2017 ; 8( 38): 63835-63845.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.19135
Vancouver
Pinheiro C, Granja S, Longatto-Filho A, Faria AM, Fragoso MCBV, Lovisolo SM, Bonatelli M, Costa RFA, Lerário AM, Almeida MQ, Baltazar F, Zerbini MCN. GLUT1 expression in pediatric adrenocortical tumors: a promising candidate to predict clinical behavior [Internet]. Oncotarget. 2017 ; 8( 38): 63835-63845.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.19135
Artigo de periodico
ΔNp73 overexpression promotes resistance to apoptosis but does not cooperate with PML/RARA in the induction of an APL-leukemic phenotype (2017)
Lucena-Araújo, Antonio Roberto; Coelho-Silva, Juan L.; Pereira-Martins, Diego A.; Thomé, Carolina; Scheucher, Priscila S.; Lange, Ana P.; Paiva, Helder H.; Hemmelgarn, Benjamin T.; Morais-Sobral, Mariana C.; Azevedo, Elisa A.; Franca-Neto, Pedro L.; Franca, Rafael F.; Silva, Cleide L.; Krause, Alexandre; Rego, Eduardo Magalhães
Source: Oncotarget. Unidade: FMRP
Subjects: LEUCEMIA, APOPTOSE, TRANSPLANTE DE MEDULA ÓSSEA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LUCENA-ARAÚJO, Antonio Roberto et al. ΔNp73 overexpression promotes resistance to apoptosis but does not cooperate with PML/RARA in the induction of an APL-leukemic phenotype. Oncotarget, v. 8, n. 5, p. 8475-8483, 2017Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.14295. Acesso em: 18 nov. 2024.
APA
Lucena-Araújo, A. R., Coelho-Silva, J. L., Pereira-Martins, D. A., Thomé, C., Scheucher, P. S., Lange, A. P., et al. (2017). ΔNp73 overexpression promotes resistance to apoptosis but does not cooperate with PML/RARA in the induction of an APL-leukemic phenotype. Oncotarget, 8( 5), 8475-8483. doi:10.18632/oncotarget.14295
NLM
Lucena-Araújo AR, Coelho-Silva JL, Pereira-Martins DA, Thomé C, Scheucher PS, Lange AP, Paiva HH, Hemmelgarn BT, Morais-Sobral MC, Azevedo EA, Franca-Neto PL, Franca RF, Silva CL, Krause A, Rego EM. ΔNp73 overexpression promotes resistance to apoptosis but does not cooperate with PML/RARA in the induction of an APL-leukemic phenotype [Internet]. Oncotarget. 2017 ; 8( 5): 8475-8483.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.14295
Vancouver
Lucena-Araújo AR, Coelho-Silva JL, Pereira-Martins DA, Thomé C, Scheucher PS, Lange AP, Paiva HH, Hemmelgarn BT, Morais-Sobral MC, Azevedo EA, Franca-Neto PL, Franca RF, Silva CL, Krause A, Rego EM. ΔNp73 overexpression promotes resistance to apoptosis but does not cooperate with PML/RARA in the induction of an APL-leukemic phenotype [Internet]. Oncotarget. 2017 ; 8( 5): 8475-8483.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.14295
Artigo de periodico
Down-regulation of 14q32-encoded miRNAs and tumor suppressorrole for miR-654-3p in papillary thyroid cancer (2017)
Geraldo, Murilo Vieira; Nakaya, Helder Takashi Imoto ; Kimura, Edna Teruko
Source: Oncotarget. Unidades: FCF, ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GERALDO, Murilo Vieira e NAKAYA, Helder Takashi Imoto e KIMURA, Edna Teruko. Down-regulation of 14q32-encoded miRNAs and tumor suppressorrole for miR-654-3p in papillary thyroid cancer. Oncotarget, v. 8, n. 6, p. 9597-9607, 2017Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.14162. Acesso em: 18 nov. 2024.
APA
Geraldo, M. V., Nakaya, H. T. I., & Kimura, E. T. (2017). Down-regulation of 14q32-encoded miRNAs and tumor suppressorrole for miR-654-3p in papillary thyroid cancer. Oncotarget, 8( 6), 9597-9607. doi:10.18632/oncotarget.14162
NLM
Geraldo MV, Nakaya HTI, Kimura ET. Down-regulation of 14q32-encoded miRNAs and tumor suppressorrole for miR-654-3p in papillary thyroid cancer [Internet]. Oncotarget. 2017 ; 8( 6): 9597-9607.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.14162
Vancouver
Geraldo MV, Nakaya HTI, Kimura ET. Down-regulation of 14q32-encoded miRNAs and tumor suppressorrole for miR-654-3p in papillary thyroid cancer [Internet]. Oncotarget. 2017 ; 8( 6): 9597-9607.[citado 2024 nov. 18 ] Available from: https://doi.org/10.18632/oncotarget.14162

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