Source: Neuroendocrinology. Unidades: FORP, FMRP
Subjects: HORMÔNIOS GLICOCORTICOIDES, SUPLEMENTOS DIETÉTICOS, PROGESTERONA, PROTEÍNAS PROTO-ONCOGÊNICAS
ABNT
RODRIGUES-SANTOS, Isabelle e KALIL-CUTTI, Bruna e ANSELMO-FRANCI, Janete Aparecida. Low corticosterone response to stress in a perimenopausal rat model is associated with the hypoactivation of pamp region of the paraventricular nucleus and can be corrected by exogenous progesterone supplementation. Neuroendocrinology, v. 112, n. 5, p. 467-480, 2022Tradução . . Disponível em: https://doi.org/10.1159/000518336. Acesso em: 08 out. 2025.APA
Rodrigues-Santos, I., Kalil-Cutti, B., & Anselmo-Franci, J. A. (2022). Low corticosterone response to stress in a perimenopausal rat model is associated with the hypoactivation of pamp region of the paraventricular nucleus and can be corrected by exogenous progesterone supplementation. Neuroendocrinology, 112( 5), 467-480. doi:10.1159/000518336NLM
Rodrigues-Santos I, Kalil-Cutti B, Anselmo-Franci JA. Low corticosterone response to stress in a perimenopausal rat model is associated with the hypoactivation of pamp region of the paraventricular nucleus and can be corrected by exogenous progesterone supplementation [Internet]. Neuroendocrinology. 2022 ; 112( 5): 467-480.[citado 2025 out. 08 ] Available from: https://doi.org/10.1159/000518336Vancouver
Rodrigues-Santos I, Kalil-Cutti B, Anselmo-Franci JA. Low corticosterone response to stress in a perimenopausal rat model is associated with the hypoactivation of pamp region of the paraventricular nucleus and can be corrected by exogenous progesterone supplementation [Internet]. Neuroendocrinology. 2022 ; 112( 5): 467-480.[citado 2025 out. 08 ] Available from: https://doi.org/10.1159/000518336