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Artigo de periodico
Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology (2025)
Ferreira, Ana Flávia Fernandes ; Ulrich, Henning ; Mori, Yasuo; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Fonte: Molecular Neurobiology. Unidades: IQ, ICB
Assuntos: DELEÇÃO DE GENES, DOENÇA DE PARKINSON, RATOS
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FERREIRA, Ana Flávia Fernandes et al. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology. Molecular Neurobiology, v. 62, p. 5333-5346, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s12035-024-04611-9. Acesso em: 07 dez. 2025.
APA
Ferreira, A. F. F., Ulrich, H., Mori, Y., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2025). Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology. Molecular Neurobiology, 62, 5333-5346. doi:10.1007/s12035-024-04611-9
NLM
Ferreira AFF, Ulrich H, Mori Y, Feng Z-P, Sun H-S, Britto LRG de. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology [Internet]. Molecular Neurobiology. 2025 ; 62 5333-5346.[citado 2025 dez. 07 ] Available from: https://dx.doi.org/10.1007/s12035-024-04611-9
Vancouver
Ferreira AFF, Ulrich H, Mori Y, Feng Z-P, Sun H-S, Britto LRG de. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology [Internet]. Molecular Neurobiology. 2025 ; 62 5333-5346.[citado 2025 dez. 07 ] Available from: https://dx.doi.org/10.1007/s12035-024-04611-9
Artigo de periodico
Benfotiamine ameliorates streptozotocin-induced Alzheimer’s disease in rats by modulating neuroinflammation, oxidative stress, and microglia (2025)
Cardinali, Camila Aparecida Errerias Fernandes ; Martins, Yandara Akamine ; Moraes, Ruan Carlos Macêdo de ; Costa, Andressa Pereira ; Torrão, Andréa da Silva
Fonte: Molecular Neurobiology. Unidades: ICB, FM
Assuntos: FISIOLOGIA, ASTRÓCITOS, NEURÓGLIA, VITAMINAS, RATOS WISTAR, DOENÇA DE ALZHEIMER, ANTIOXIDANTES, HIPOCAMPO, INFLAMAÇÃO, ESTRESSE OXIDATIVO
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CARDINALI, Camila Aparecida Errerias Fernandes et al. Benfotiamine ameliorates streptozotocin-induced Alzheimer’s disease in rats by modulating neuroinflammation, oxidative stress, and microglia. Molecular Neurobiology, v. 62, p. 8695-8717, 2025Tradução . . Disponível em: https://doi.org/10.1007/s12035-025-04811-x. Acesso em: 07 dez. 2025.
APA
Cardinali, C. A. E. F., Martins, Y. A., Moraes, R. C. M. de, Costa, A. P., & Torrão, A. da S. (2025). Benfotiamine ameliorates streptozotocin-induced Alzheimer’s disease in rats by modulating neuroinflammation, oxidative stress, and microglia. Molecular Neurobiology, 62, 8695-8717. doi:10.1007/s12035-025-04811-x
NLM
Cardinali CAEF, Martins YA, Moraes RCM de, Costa AP, Torrão A da S. Benfotiamine ameliorates streptozotocin-induced Alzheimer’s disease in rats by modulating neuroinflammation, oxidative stress, and microglia [Internet]. Molecular Neurobiology. 2025 ; 62 8695-8717.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-025-04811-x
Vancouver
Cardinali CAEF, Martins YA, Moraes RCM de, Costa AP, Torrão A da S. Benfotiamine ameliorates streptozotocin-induced Alzheimer’s disease in rats by modulating neuroinflammation, oxidative stress, and microglia [Internet]. Molecular Neurobiology. 2025 ; 62 8695-8717.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-025-04811-x
Artigo de periodico
Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology (2025)
Ferreira, Ana Flávia Fernandes ; Mori, Yasuo ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Ulrich, Henning ; Britto, Luiz Roberto Giorgetti de
Fonte: Molecular Neurobiology. Unidades: ICB, IQ
Assuntos: FISIOLOGIA, DOENÇA DE PARKINSON, PROTEÍNAS DA MEMBRANA, APOPTOSE, ATIVIDADE MOTORA, RECEPTORES DE DOPAMINA, ESTRESSE OXIDATIVO, CAMUNDONGOS
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FERREIRA, Ana Flávia Fernandes et al. Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology. Molecular Neurobiology, v. 62, n. 4, p. 5333-5346, 2025Tradução . . Disponível em: https://doi.org/10.1007/s12035-024-04611-9. Acesso em: 07 dez. 2025.
APA
Ferreira, A. F. F., Mori, Y., Feng, Z. -P., Sun, H. -S., Ulrich, H., & Britto, L. R. G. de. (2025). Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology. Molecular Neurobiology, 62( 4), 5333-5346. doi:10.1007/s12035-024-04611-9
NLM
Ferreira AFF, Mori Y, Feng Z-P, Sun H-S, Ulrich H, Britto LRG de. Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology [Internet]. Molecular Neurobiology. 2025 ; 62( 4): 5333-5346.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-024-04611-9
Vancouver
Ferreira AFF, Mori Y, Feng Z-P, Sun H-S, Ulrich H, Britto LRG de. Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology [Internet]. Molecular Neurobiology. 2025 ; 62( 4): 5333-5346.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-024-04611-9
Artigo de periodico
EHMT2 as a Candidate Gene for an Autosomal Recessive Neurodevelopmental Syndrome (2025)
Carvalho, Laura Machado Lara ; Jorge, Alexander Augusto de Lima ; Branco, Elisa Varella ; Oliveira, Danyllo Felipe de ; Haddad, Luciana Amaral ; Bertola, Débora Romeo ; Rosenberg, Carla ; Krepischi, Ana Cristina Victorino
Fonte: Molecular Neurobiology. Unidades: FM, IB
Assuntos: GENÉTICA MÉDICA, MUTAÇÃO GENÉTICA, GENES
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CARVALHO, Laura Machado Lara et al. EHMT2 as a Candidate Gene for an Autosomal Recessive Neurodevelopmental Syndrome. Molecular Neurobiology, v. 62, p. 5977–5989, 2025Tradução . . Disponível em: https://doi.org/10.1007/s12035-024-04655-x. Acesso em: 07 dez. 2025.
APA
Carvalho, L. M. L., Jorge, A. A. de L., Branco, E. V., Oliveira, D. F. de, Haddad, L. A., Bertola, D. R., et al. (2025). EHMT2 as a Candidate Gene for an Autosomal Recessive Neurodevelopmental Syndrome. Molecular Neurobiology, 62, 5977–5989. doi:10.1007/s12035-024-04655-x
NLM
Carvalho LML, Jorge AA de L, Branco EV, Oliveira DF de, Haddad LA, Bertola DR, Rosenberg C, Krepischi ACV. EHMT2 as a Candidate Gene for an Autosomal Recessive Neurodevelopmental Syndrome [Internet]. Molecular Neurobiology. 2025 ; 62 5977–5989.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-024-04655-x
Vancouver
Carvalho LML, Jorge AA de L, Branco EV, Oliveira DF de, Haddad LA, Bertola DR, Rosenberg C, Krepischi ACV. EHMT2 as a Candidate Gene for an Autosomal Recessive Neurodevelopmental Syndrome [Internet]. Molecular Neurobiology. 2025 ; 62 5977–5989.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-024-04655-x
Artigo de periodico
Clinical characterization and underlying genetic findings in brazilian patients with syndromic microcephaly associated with neurodevelopmental disorders (2024)
Tolezano, Giovanna Cantini; Bastos, Giovanna Civitate; Costa, Silvia Souza da; Scliar, Marília de Oliveira; Souza, Carolina Fischinger Moura de; Linden Jr, Hélio Van Der; Fernandes, Walter Luiz Magalhães; Otto, Paulo A ; Vianna-Morgante, Angela M ; Haddad, Luciana Amaral ; Honjo, Rachel Sayuri; Yamamoto, Guilherme Lopes; Kim, Chong Ae; Rosenberg, Carla ; Jorge, Alexander Augusto de Lima ; Bertola, Débora Romeo ; Krepischi, Ana Cristina Victorino
Fonte: Molecular Neurobiology. Unidades: IB, Interunidades em Bioinformática, FM
Assuntos: MICROCEFALIA, NEUROLOGIA, GENÉTICA
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TOLEZANO, Giovanna Cantini et al. Clinical characterization and underlying genetic findings in brazilian patients with syndromic microcephaly associated with neurodevelopmental disorders. Molecular Neurobiology, 2024Tradução . . Disponível em: https://doi.org/10.1007/s12035-023-03894-8. Acesso em: 07 dez. 2025.
APA
Tolezano, G. C., Bastos, G. C., Costa, S. S. da, Scliar, M. de O., Souza, C. F. M. de, Linden Jr, H. V. D., et al. (2024). Clinical characterization and underlying genetic findings in brazilian patients with syndromic microcephaly associated with neurodevelopmental disorders. Molecular Neurobiology. doi:10.1007/s12035-023-03894-8
NLM
Tolezano GC, Bastos GC, Costa SS da, Scliar M de O, Souza CFM de, Linden Jr HVD, Fernandes WLM, Otto PA, Vianna-Morgante AM, Haddad LA, Honjo RS, Yamamoto GL, Kim CA, Rosenberg C, Jorge AA de L, Bertola DR, Krepischi ACV. Clinical characterization and underlying genetic findings in brazilian patients with syndromic microcephaly associated with neurodevelopmental disorders [Internet]. Molecular Neurobiology. 2024 ;[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-023-03894-8
Vancouver
Tolezano GC, Bastos GC, Costa SS da, Scliar M de O, Souza CFM de, Linden Jr HVD, Fernandes WLM, Otto PA, Vianna-Morgante AM, Haddad LA, Honjo RS, Yamamoto GL, Kim CA, Rosenberg C, Jorge AA de L, Bertola DR, Krepischi ACV. Clinical characterization and underlying genetic findings in brazilian patients with syndromic microcephaly associated with neurodevelopmental disorders [Internet]. Molecular Neurobiology. 2024 ;[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-023-03894-8
Artigo de periodico
A Purine derivative containing an organoselenium group protects against memory impairment, sensitivity to nociception, oxidative damage, and neuroinflammation in a mouse model of Alzheimer’s disease (2023)
Pinz, Mikaela Peglow ; Oliveira, Renata Leivas de ; Fonseca, Caren Aline Ramson da ; Voss, Guilherme Teixeira ; Silva, Beatriz Pereira da ; Duarte, Luis Fernando Barbosa ; Domingues, William Borges; Ortiz, Hadassa Gabriela; Savall, Anne Suély Pinto ; Meotti, Flavia Carla ; Alves, Diego ; Campos, Vinicius Farias ; Pinton, Simone ; Wilhelm, Ethel Antunes ; Luchese, Cristiane
Fonte: Molecular Neurobiology. Unidade: IQ
Assuntos: DOENÇA DE ALZHEIMER, ESTRESSE OXIDATIVO
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PINZ, Mikaela Peglow et al. A Purine derivative containing an organoselenium group protects against memory impairment, sensitivity to nociception, oxidative damage, and neuroinflammation in a mouse model of Alzheimer’s disease. Molecular Neurobiology, v. 60, p. 1214–1231, 2023Tradução . . Disponível em: https://doi.org/10.1007/s12035-022-03110-z. Acesso em: 07 dez. 2025.
APA
Pinz, M. P., Oliveira, R. L. de, Fonseca, C. A. R. da, Voss, G. T., Silva, B. P. da, Duarte, L. F. B., et al. (2023). A Purine derivative containing an organoselenium group protects against memory impairment, sensitivity to nociception, oxidative damage, and neuroinflammation in a mouse model of Alzheimer’s disease. Molecular Neurobiology, 60, 1214–1231. doi:10.1007/s12035-022-03110-z
NLM
Pinz MP, Oliveira RL de, Fonseca CAR da, Voss GT, Silva BP da, Duarte LFB, Domingues WB, Ortiz HG, Savall ASP, Meotti FC, Alves D, Campos VF, Pinton S, Wilhelm EA, Luchese C. A Purine derivative containing an organoselenium group protects against memory impairment, sensitivity to nociception, oxidative damage, and neuroinflammation in a mouse model of Alzheimer’s disease [Internet]. Molecular Neurobiology. 2023 ; 60 1214–1231.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-022-03110-z
Vancouver
Pinz MP, Oliveira RL de, Fonseca CAR da, Voss GT, Silva BP da, Duarte LFB, Domingues WB, Ortiz HG, Savall ASP, Meotti FC, Alves D, Campos VF, Pinton S, Wilhelm EA, Luchese C. A Purine derivative containing an organoselenium group protects against memory impairment, sensitivity to nociception, oxidative damage, and neuroinflammation in a mouse model of Alzheimer’s disease [Internet]. Molecular Neurobiology. 2023 ; 60 1214–1231.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-022-03110-z
Artigo de periodico
Skewed x-chromosome inactivation in women with idiopathic intellectual disability is indicative of pathogenic variants (2023)
Chaves, Luiza D; Carvalho, Laura M. L; Tolezano, Giovanna C; Pires, Sara F; Costa, Silvia S; Scliar, Marília O. de; Giuliani, Liane de R; Bertola, Débora Romeo ; Santos-Rebouças, Cíntia B; Seo, Go Hun; Otto, Paulo A ; Rosenberg, Carla ; Vianna-Morgante, Angela M ; Krepischi, Ana Cristina Victorino
Fonte: Molecular Neurobiology. Unidade: IB
Assuntos: MULHERES, CROMOSSOMO X, RETARDO MENTAL, GENÉTICA MÉDICA
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CHAVES, Luiza D et al. Skewed x-chromosome inactivation in women with idiopathic intellectual disability is indicative of pathogenic variants. Molecular Neurobiology, v. 60, p. 3758–3769, 2023Tradução . . Disponível em: https://doi.org/10.1007/s12035-023-03311-0. Acesso em: 07 dez. 2025.
APA
Chaves, L. D., Carvalho, L. M. L., Tolezano, G. C., Pires, S. F., Costa, S. S., Scliar, M. O. de, et al. (2023). Skewed x-chromosome inactivation in women with idiopathic intellectual disability is indicative of pathogenic variants. Molecular Neurobiology, 60, 3758–3769. doi:10.1007/s12035-023-03311-0
NLM
Chaves LD, Carvalho LML, Tolezano GC, Pires SF, Costa SS, Scliar MO de, Giuliani L de R, Bertola DR, Santos-Rebouças CB, Seo GH, Otto PA, Rosenberg C, Vianna-Morgante AM, Krepischi ACV. Skewed x-chromosome inactivation in women with idiopathic intellectual disability is indicative of pathogenic variants [Internet]. Molecular Neurobiology. 2023 ; 60 3758–3769.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-023-03311-0
Vancouver
Chaves LD, Carvalho LML, Tolezano GC, Pires SF, Costa SS, Scliar MO de, Giuliani L de R, Bertola DR, Santos-Rebouças CB, Seo GH, Otto PA, Rosenberg C, Vianna-Morgante AM, Krepischi ACV. Skewed x-chromosome inactivation in women with idiopathic intellectual disability is indicative of pathogenic variants [Internet]. Molecular Neurobiology. 2023 ; 60 3758–3769.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-023-03311-0
Artigo de periodico
A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance (2022)
Alves, Suélen Santos ; Silva Júnior, Rui Milton Patrício da; Delfino-Pereira, Polianna; Pereira, Marília Gabriella Alves Goulart; Vasconcelos, Israel Costa; Schwaemmle, Hanna; Mazzei, Rodrigo Focosi; Carlos, Maiko Luiz ; Espreáfico, Enilza Maria; Tedesco, Antônio Cláudio ; Sebollela, Adriano Silva ; Almeida, Sebastião de Sousa ; Oliveira, José Antonio Cortes de; Garcia-Cairasco, Norberto
Fonte: Molecular Neurobiology. Unidades: FFCLRP, FMRP
Assuntos: EPILEPSIA, DOENÇA DE ALZHEIMER, RESISTÊNCIA À INSULINA, MEMÓRIA
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ALVES, Suélen Santos et al. A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance. Molecular Neurobiology, v. 59, n. 6, p. 3721-3737, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-022-02810-w. Acesso em: 07 dez. 2025.
APA
Alves, S. S., Silva Júnior, R. M. P. da, Delfino-Pereira, P., Pereira, M. G. A. G., Vasconcelos, I. C., Schwaemmle, H., et al. (2022). A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance. Molecular Neurobiology, 59( 6), 3721-3737. doi:10.1007/s12035-022-02810-w
NLM
Alves SS, Silva Júnior RMP da, Delfino-Pereira P, Pereira MGAG, Vasconcelos IC, Schwaemmle H, Mazzei RF, Carlos ML, Espreáfico EM, Tedesco AC, Sebollela AS, Almeida S de S, Oliveira JAC de, Garcia-Cairasco N. A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance [Internet]. Molecular Neurobiology. 2022 ; 59( 6): 3721-3737.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-022-02810-w
Vancouver
Alves SS, Silva Júnior RMP da, Delfino-Pereira P, Pereira MGAG, Vasconcelos IC, Schwaemmle H, Mazzei RF, Carlos ML, Espreáfico EM, Tedesco AC, Sebollela AS, Almeida S de S, Oliveira JAC de, Garcia-Cairasco N. A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance [Internet]. Molecular Neurobiology. 2022 ; 59( 6): 3721-3737.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-022-02810-w
Artigo de periodico
Neuroprotective effect of exogenous galectin-1 in status epilepticus (2022)
Pacheco, Amanda Larissa Dias; Melo, Igor Santana de; Costa, Maisa de Araujo; Amaral, Mariah Morais Celestino; Silva, Nívea Karla De Gusmão Taveiros ; Santos, Yngrid Mickaelli Oliveira; Gitaí, Daniel Leite Goes; Duzzioni, Marcelo; Borbely, Alexandre Urban; Silva, Robinson Sabino da; Donatti, Ana Luiza Ferreira; Mestriner, Luisa; Fuzo, Carlos Alessandro ; Cummings, Richard D.; Garcia-Cairasco, Norberto ; Dias-Baruffi, Marcelo ; Castro, Olagide Wagner de
Fonte: Molecular Neurobiology. Unidades: FMRP, FCFRP
Assuntos: EPILEPSIA, EPILEPSIA DO LOBO TEMPORAL
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ABNT
PACHECO, Amanda Larissa Dias et al. Neuroprotective effect of exogenous galectin-1 in status epilepticus. Molecular Neurobiology, v. 59, n. 12, p. 7354-7369, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-022-03038-4. Acesso em: 07 dez. 2025.
APA
Pacheco, A. L. D., Melo, I. S. de, Costa, M. de A., Amaral, M. M. C., Silva, N. K. D. G. T., Santos, Y. M. O., et al. (2022). Neuroprotective effect of exogenous galectin-1 in status epilepticus. Molecular Neurobiology, 59( 12), 7354-7369. doi:10.1007/s12035-022-03038-4
NLM
Pacheco ALD, Melo IS de, Costa M de A, Amaral MMC, Silva NKDGT, Santos YMO, Gitaí DLG, Duzzioni M, Borbely AU, Silva RS da, Donatti ALF, Mestriner L, Fuzo CA, Cummings RD, Garcia-Cairasco N, Dias-Baruffi M, Castro OW de. Neuroprotective effect of exogenous galectin-1 in status epilepticus [Internet]. Molecular Neurobiology. 2022 ; 59( 12): 7354-7369.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-022-03038-4
Vancouver
Pacheco ALD, Melo IS de, Costa M de A, Amaral MMC, Silva NKDGT, Santos YMO, Gitaí DLG, Duzzioni M, Borbely AU, Silva RS da, Donatti ALF, Mestriner L, Fuzo CA, Cummings RD, Garcia-Cairasco N, Dias-Baruffi M, Castro OW de. Neuroprotective effect of exogenous galectin-1 in status epilepticus [Internet]. Molecular Neurobiology. 2022 ; 59( 12): 7354-7369.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-022-03038-4
Artigo de periodico
Metabolic profile in plasma and CSF of LEVODOPA-induced dyskinesia in Parkinson’s disease: focus on neuroinflammation (2022)
Santos-Lobato, Bruno L.; Gardinassi, Luiz Gustavo; Bortolanza, Mariza; Peti, Ana Paula Ferranti; Pimentel, Ângela V.; Faccioli, Lucia Helena ; Del Bel, Elaine Aparecida ; Tumas, Vitor
Fonte: Molecular Neurobiology. Unidades: FMRP, FCFRP, FORP
Assuntos: METABOLÔMICA, DOENÇA DE PARKINSON, DISCINESIAS, FÁRMACOS (SISTEMA NERVOSO CENTRAL)
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ABNT
SANTOS-LOBATO, Bruno L. et al. Metabolic profile in plasma and CSF of LEVODOPA-induced dyskinesia in Parkinson’s disease: focus on neuroinflammation. Molecular Neurobiology, v. 59, n. 2, p. 1140-1150, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-021-02625-1. Acesso em: 07 dez. 2025.
APA
Santos-Lobato, B. L., Gardinassi, L. G., Bortolanza, M., Peti, A. P. F., Pimentel, Â. V., Faccioli, L. H., et al. (2022). Metabolic profile in plasma and CSF of LEVODOPA-induced dyskinesia in Parkinson’s disease: focus on neuroinflammation. Molecular Neurobiology, 59( 2), 1140-1150. doi:10.1007/s12035-021-02625-1
NLM
Santos-Lobato BL, Gardinassi LG, Bortolanza M, Peti APF, Pimentel ÂV, Faccioli LH, Del Bel EA, Tumas V. Metabolic profile in plasma and CSF of LEVODOPA-induced dyskinesia in Parkinson’s disease: focus on neuroinflammation [Internet]. Molecular Neurobiology. 2022 ; 59( 2): 1140-1150.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-021-02625-1
Vancouver
Santos-Lobato BL, Gardinassi LG, Bortolanza M, Peti APF, Pimentel ÂV, Faccioli LH, Del Bel EA, Tumas V. Metabolic profile in plasma and CSF of LEVODOPA-induced dyskinesia in Parkinson’s disease: focus on neuroinflammation [Internet]. Molecular Neurobiology. 2022 ; 59( 2): 1140-1150.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-021-02625-1
Artigo de periodico
Photobiomodulation improves the inflammatory response and intracellular signaling proteins linked to vascular function and cell survival in the brain of aged rats (2022)
Cardoso, Fabrízio dos Santos ; Mansur, Fernanda Cristina Borini; Araujo, Bruno Henrique Silva; Gonzalez-Lima, F.; Silva, Sérgio Gomes da
Fonte: Molecular Neurobiology. Unidade: FMRP
Assuntos: ENVOLVIMENTO, CÉREBRO, SOBREVIVÊNCIA CELULAR, CITOCINAS, INFLAMAÇÃO, TECIDO NERVOSO
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ABNT
CARDOSO, Fabrízio dos Santos et al. Photobiomodulation improves the inflammatory response and intracellular signaling proteins linked to vascular function and cell survival in the brain of aged rats. Molecular Neurobiology, v. 59, n. 1, p. 420-428, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-021-02606-4. Acesso em: 07 dez. 2025.
APA
Cardoso, F. dos S., Mansur, F. C. B., Araujo, B. H. S., Gonzalez-Lima, F., & Silva, S. G. da. (2022). Photobiomodulation improves the inflammatory response and intracellular signaling proteins linked to vascular function and cell survival in the brain of aged rats. Molecular Neurobiology, 59( 1), 420-428. doi:10.1007/s12035-021-02606-4
NLM
Cardoso F dos S, Mansur FCB, Araujo BHS, Gonzalez-Lima F, Silva SG da. Photobiomodulation improves the inflammatory response and intracellular signaling proteins linked to vascular function and cell survival in the brain of aged rats [Internet]. Molecular Neurobiology. 2022 ; 59( 1): 420-428.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-021-02606-4
Vancouver
Cardoso F dos S, Mansur FCB, Araujo BHS, Gonzalez-Lima F, Silva SG da. Photobiomodulation improves the inflammatory response and intracellular signaling proteins linked to vascular function and cell survival in the brain of aged rats [Internet]. Molecular Neurobiology. 2022 ; 59( 1): 420-428.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-021-02606-4
Artigo de periodico
Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model (2022)
Ferreira, Ana Flávia Fernandes ; Singulani, Monique Patricio ; Ulrich, Henning ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Fonte: Molecular Neurobiology. Unidades: IQ, ICB, FM
Assuntos: FISIOLOGIA, NEUROBIOLOGIA, MODELOS ANIMAIS DE DOENÇAS, DOENÇA DE PARKINSON, MESENCÉFALO, GÂNGLIOS, RECEPTORES DE DOPAMINA, CAMUNDONGOS, IMUNOHISTOQUÍMICA, IMMUNOBLOTTING
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ABNT
FERREIRA, Ana Flávia Fernandes et al. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model. Molecular Neurobiology, v. 59, p. 1543–1559, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-022-02723-8. Acesso em: 07 dez. 2025.
APA
Ferreira, A. F. F., Singulani, M. P., Ulrich, H., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2022). Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model. Molecular Neurobiology, 59, 1543–1559. doi:10.1007/s12035-022-02723-8
NLM
Ferreira AFF, Singulani MP, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model [Internet]. Molecular Neurobiology. 2022 ; 59 1543–1559.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-022-02723-8
Vancouver
Ferreira AFF, Singulani MP, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model [Internet]. Molecular Neurobiology. 2022 ; 59 1543–1559.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-022-02723-8
Artigo de periodico
Microglial activation modulates neuroendocrine secretion during experimental sepsis (2021)
Costa, Luís Henrique Angenendt da ; Santos Junior, Nilton Nascimento dos ; Catalão, Carlos Henrique Rocha ; Rocha, Maria José Alves da
Fonte: Molecular Neurobiology. Unidades: FMRP, FORP
Assuntos: SISTEMA NERVOSO CENTRAL, RATOS WISTAR, SEPSE, INFLAMAÇÃO, INIBIDORES DE ENZIMAS
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ABNT
COSTA, Luís Henrique Angenendt da et al. Microglial activation modulates neuroendocrine secretion during experimental sepsis. Molecular Neurobiology, v. 58, n. 5, p. 2133-2144, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12035-020-02241-5. Acesso em: 07 dez. 2025.
APA
Costa, L. H. A. da, Santos Junior, N. N. dos, Catalão, C. H. R., & Rocha, M. J. A. da. (2021). Microglial activation modulates neuroendocrine secretion during experimental sepsis. Molecular Neurobiology, 58( 5), 2133-2144. doi:10.1007/s12035-020-02241-5
NLM
Costa LHA da, Santos Junior NN dos, Catalão CHR, Rocha MJA da. Microglial activation modulates neuroendocrine secretion during experimental sepsis [Internet]. Molecular Neurobiology. 2021 ; 58( 5): 2133-2144.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-020-02241-5
Vancouver
Costa LHA da, Santos Junior NN dos, Catalão CHR, Rocha MJA da. Microglial activation modulates neuroendocrine secretion during experimental sepsis [Internet]. Molecular Neurobiology. 2021 ; 58( 5): 2133-2144.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-020-02241-5
Artigo de periodico
The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth (2021)
Teixeira, Silvia Aparecida; Viapiano, Mariano S.; Andrade, Augusto F.; Nandhu, Mohan S.; Pezuk, Julia Alejandra; Bidinotto, Lucas T.; Sant'Ana, Veridiana Kiill Suazo de; Serafini, Luciano Neder; Carlotti Júnior, Carlos Gilberto ; Becker, Aline P.; Tone, Luiz Gonzaga ; Scrideli, Carlos Alberto
Fonte: Molecular Neurobiology. Unidade: FMRP
Assuntos: ANIDRASE CARBÔNICA, METABOLISMO CELULAR, NEOPLASIAS CEREBRAIS, FARMACOTERAPIA, LINHAGEM CELULAR, CICLO CELULAR, SULFONAMIDAS
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ABNT
TEIXEIRA, Silvia Aparecida et al. The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth. Molecular Neurobiology, v. 58, n. 9, p. 4520-4534, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12035-021-02437-3. Acesso em: 07 dez. 2025.
APA
Teixeira, S. A., Viapiano, M. S., Andrade, A. F., Nandhu, M. S., Pezuk, J. A., Bidinotto, L. T., et al. (2021). The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth. Molecular Neurobiology, 58( 9), 4520-4534. doi:10.1007/s12035-021-02437-3
NLM
Teixeira SA, Viapiano MS, Andrade AF, Nandhu MS, Pezuk JA, Bidinotto LT, Sant'Ana VKS de, Serafini LN, Carlotti Júnior CG, Becker AP, Tone LG, Scrideli CA. The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth [Internet]. Molecular Neurobiology. 2021 ; 58( 9): 4520-4534.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-021-02437-3
Vancouver
Teixeira SA, Viapiano MS, Andrade AF, Nandhu MS, Pezuk JA, Bidinotto LT, Sant'Ana VKS de, Serafini LN, Carlotti Júnior CG, Becker AP, Tone LG, Scrideli CA. The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth [Internet]. Molecular Neurobiology. 2021 ; 58( 9): 4520-4534.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-021-02437-3
Artigo de periodico
Cannabidiol confers neuroprotection in rats in a model of transient global cerebral ischemia: impact of hippocampal synaptic neuroplasticity (2021)
Meyer, Erika; Bonato, Jéssica Mendes; Mori, Marco Aurélio; Mattos, Bianca Andretto; Guimarães, Francisco Silveira ; Milani, Humberto; Campos, Alline Cristina de; Oliveira, Rúbia Maria Weffort de
Fonte: Molecular Neurobiology. Unidade: FMRP
Assuntos: CANABINOIDES, HIPOCAMPO, PLASTICIDADE NEURONAL, CULTURA DE ORGÃOS, SINAPSE
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ABNT
MEYER, Erika et al. Cannabidiol confers neuroprotection in rats in a model of transient global cerebral ischemia: impact of hippocampal synaptic neuroplasticity. Molecular Neurobiology, v. 58, n. 10, p. 5338-5355, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12035-021-02479-7. Acesso em: 07 dez. 2025.
APA
Meyer, E., Bonato, J. M., Mori, M. A., Mattos, B. A., Guimarães, F. S., Milani, H., et al. (2021). Cannabidiol confers neuroprotection in rats in a model of transient global cerebral ischemia: impact of hippocampal synaptic neuroplasticity. Molecular Neurobiology, 58( 10), 5338-5355. doi:10.1007/s12035-021-02479-7
NLM
Meyer E, Bonato JM, Mori MA, Mattos BA, Guimarães FS, Milani H, Campos AC de, Oliveira RMW de. Cannabidiol confers neuroprotection in rats in a model of transient global cerebral ischemia: impact of hippocampal synaptic neuroplasticity [Internet]. Molecular Neurobiology. 2021 ; 58( 10): 5338-5355.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-021-02479-7
Vancouver
Meyer E, Bonato JM, Mori MA, Mattos BA, Guimarães FS, Milani H, Campos AC de, Oliveira RMW de. Cannabidiol confers neuroprotection in rats in a model of transient global cerebral ischemia: impact of hippocampal synaptic neuroplasticity [Internet]. Molecular Neurobiology. 2021 ; 58( 10): 5338-5355.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-021-02479-7
Artigo de periodico
Norepinephrine and glucocorticoids modulate chronic unpredictable stress-induced increase in the Type 2 CRF and glucocorticoid receptors in brain structures related to the HPA Axis activation (2021)
Malta, Marilia Brinati; Silva, Joelcimar Martins da; Novaes, Leonardo Santana ; Santos, Nilton Barreto dos ; Sita, Luciane Valéria ; Camarini, Rosana ; Scavone, Cristoforo ; Bittencourt, Jackson Cioni ; Munhoz, Carolina Demarchi
Fonte: Molecular Neurobiology. Unidade: ICB
Assuntos: FARMACOLOGIA, ANATOMIA, NORADRENALINA, RECEPTORES DE GLICOCORTICOIDES, ESTRESSE, COMPORTAMENTO, TRANSTORNOS DA ANSIEDADE, HIPOTÁLAMO, HORMÔNIOS DO CÓRTEX ADRENAL, HORMÔNIOS GLICOCORTICOIDES, NOREPINEFRINA
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ABNT
MALTA, Marilia Brinati et al. Norepinephrine and glucocorticoids modulate chronic unpredictable stress-induced increase in the Type 2 CRF and glucocorticoid receptors in brain structures related to the HPA Axis activation. Molecular Neurobiology, p. 1-15, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12035-021-02470-2. Acesso em: 07 dez. 2025.
APA
Malta, M. B., Silva, J. M. da, Novaes, L. S., Santos, N. B. dos, Sita, L. V., Camarini, R., et al. (2021). Norepinephrine and glucocorticoids modulate chronic unpredictable stress-induced increase in the Type 2 CRF and glucocorticoid receptors in brain structures related to the HPA Axis activation. Molecular Neurobiology, 1-15. doi:10.1007/s12035-021-02470-2
NLM
Malta MB, Silva JM da, Novaes LS, Santos NB dos, Sita LV, Camarini R, Scavone C, Bittencourt JC, Munhoz CD. Norepinephrine and glucocorticoids modulate chronic unpredictable stress-induced increase in the Type 2 CRF and glucocorticoid receptors in brain structures related to the HPA Axis activation [Internet]. Molecular Neurobiology. 2021 ; 1-15.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-021-02470-2
Vancouver
Malta MB, Silva JM da, Novaes LS, Santos NB dos, Sita LV, Camarini R, Scavone C, Bittencourt JC, Munhoz CD. Norepinephrine and glucocorticoids modulate chronic unpredictable stress-induced increase in the Type 2 CRF and glucocorticoid receptors in brain structures related to the HPA Axis activation [Internet]. Molecular Neurobiology. 2021 ; 1-15.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-021-02470-2
Artigo de periodico
Mechanistic insights of astrocyte-mediated hyperactive autophagy and loss of motor neuron function in SOD1L39R linked amyotrophic lateral sclerosis (2020)
Rajpurohit, Chetan Singh; Kumar, Vivek; Cheffer, Arquimedes; Oliveira, Danyllo; Ulrich, Henning ; Okamoto, Oswaldo Keith ; Zatz, Mayana ; Ansari, Uzair Ahmad; Khanna, Vinay Kumar; Pant, Aditya Bhushan
Fonte: Molecular Neurobiology. Unidades: IQ, IB
Assuntos: ESCLEROSE AMIOTRÓFICA LATERAL, ASTRÓCITOS
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ABNT
RAJPUROHIT, Chetan Singh et al. Mechanistic insights of astrocyte-mediated hyperactive autophagy and loss of motor neuron function in SOD1L39R linked amyotrophic lateral sclerosis. Molecular Neurobiology, v. 57, p. 4117–4133, 2020Tradução . . Disponível em: https://doi.org/10.1007/s12035-020-02006-0. Acesso em: 07 dez. 2025.
APA
Rajpurohit, C. S., Kumar, V., Cheffer, A., Oliveira, D., Ulrich, H., Okamoto, O. K., et al. (2020). Mechanistic insights of astrocyte-mediated hyperactive autophagy and loss of motor neuron function in SOD1L39R linked amyotrophic lateral sclerosis. Molecular Neurobiology, 57, 4117–4133. doi:10.1007/s12035-020-02006-0
NLM
Rajpurohit CS, Kumar V, Cheffer A, Oliveira D, Ulrich H, Okamoto OK, Zatz M, Ansari UA, Khanna VK, Pant AB. Mechanistic insights of astrocyte-mediated hyperactive autophagy and loss of motor neuron function in SOD1L39R linked amyotrophic lateral sclerosis [Internet]. Molecular Neurobiology. 2020 ; 57 4117–4133.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-020-02006-0
Vancouver
Rajpurohit CS, Kumar V, Cheffer A, Oliveira D, Ulrich H, Okamoto OK, Zatz M, Ansari UA, Khanna VK, Pant AB. Mechanistic insights of astrocyte-mediated hyperactive autophagy and loss of motor neuron function in SOD1L39R linked amyotrophic lateral sclerosis [Internet]. Molecular Neurobiology. 2020 ; 57 4117–4133.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-020-02006-0
Artigo de periodico
Modulation of DNA methylation and gene expression in rodent cortical neuroplasticity pathways exerts rapid antidepressant-like effects (2020)
Sales, Amanda Juliana ; Maciel, Izaque de Sousa; Suavinha, Angélica Caroline Dutra Romano; Wegener, Sâmia Regiane Lourenço Joca
Fonte: Molecular Neurobiology. Unidades: FCFRP, FMRP
Assuntos: ANIMAIS, ANTIDEPRESSIVOS, COMPORTAMENTO ANIMAL, DNA, METILAÇÃO DE DNA, EXPRESSÃO GÊNICA
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ABNT
SALES, Amanda Juliana et al. Modulation of DNA methylation and gene expression in rodent cortical neuroplasticity pathways exerts rapid antidepressant-like effects. Molecular Neurobiology, v. 58, n. 2, p. 777-794, 2020Tradução . . Disponível em: https://doi.org/10.1007/s12035-020-02145-4. Acesso em: 07 dez. 2025.
APA
Sales, A. J., Maciel, I. de S., Suavinha, A. C. D. R., & Wegener, S. R. L. J. (2020). Modulation of DNA methylation and gene expression in rodent cortical neuroplasticity pathways exerts rapid antidepressant-like effects. Molecular Neurobiology, 58( 2), 777-794. doi:10.1007/s12035-020-02145-4
NLM
Sales AJ, Maciel I de S, Suavinha ACDR, Wegener SRLJ. Modulation of DNA methylation and gene expression in rodent cortical neuroplasticity pathways exerts rapid antidepressant-like effects [Internet]. Molecular Neurobiology. 2020 ; 58( 2): 777-794.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-020-02145-4
Vancouver
Sales AJ, Maciel I de S, Suavinha ACDR, Wegener SRLJ. Modulation of DNA methylation and gene expression in rodent cortical neuroplasticity pathways exerts rapid antidepressant-like effects [Internet]. Molecular Neurobiology. 2020 ; 58( 2): 777-794.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-020-02145-4
Artigo de periodico
Selective regulation of 5-HT1B serotonin receptor expression in the striatum by dopamine depletion and repeated L-DOPA treatment: relationship to L-DOPA-induced dyskinesias (2020)
Padovan Neto, Fernando Eduardo ; Patterson, Santanna; Voelkner, Nivea M. F.; Altwal, Feras; Beverley, Joel A.; West, Anthony R.; Steiner, Heinz
Fonte: Molecular Neurobiology. Unidade: FFCLRP
Assuntos: RECEPTORES DE DOPAMINA, RECEPTORES DE SEROTONINA, EXPRESSÃO GÊNICA, DOENÇA DE PARKINSON, NEUROLOGIA
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ABNT
PADOVAN NETO, Fernando Eduardo et al. Selective regulation of 5-HT1B serotonin receptor expression in the striatum by dopamine depletion and repeated L-DOPA treatment: relationship to L-DOPA-induced dyskinesias. Molecular Neurobiology, v. 57, n. 2, p. 736-751, 2020Tradução . . Disponível em: https://doi.org/10.1007/s12035-019-01739-x. Acesso em: 07 dez. 2025.
APA
Padovan Neto, F. E., Patterson, S., Voelkner, N. M. F., Altwal, F., Beverley, J. A., West, A. R., & Steiner, H. (2020). Selective regulation of 5-HT1B serotonin receptor expression in the striatum by dopamine depletion and repeated L-DOPA treatment: relationship to L-DOPA-induced dyskinesias. Molecular Neurobiology, 57( 2), 736-751. doi:10.1007/s12035-019-01739-x
NLM
Padovan Neto FE, Patterson S, Voelkner NMF, Altwal F, Beverley JA, West AR, Steiner H. Selective regulation of 5-HT1B serotonin receptor expression in the striatum by dopamine depletion and repeated L-DOPA treatment: relationship to L-DOPA-induced dyskinesias [Internet]. Molecular Neurobiology. 2020 ; 57( 2): 736-751.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-019-01739-x
Vancouver
Padovan Neto FE, Patterson S, Voelkner NMF, Altwal F, Beverley JA, West AR, Steiner H. Selective regulation of 5-HT1B serotonin receptor expression in the striatum by dopamine depletion and repeated L-DOPA treatment: relationship to L-DOPA-induced dyskinesias [Internet]. Molecular Neurobiology. 2020 ; 57( 2): 736-751.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-019-01739-x
Artigo de periodico
Non-peptidergic nociceptive neurons are essential for mechanical inflammatory hypersensitivity in mice (2019)
Pinto, Larissa G.; Souza, Guilherme R.; Kusuda, Ricardo; Lopes, Alexandre Hashimoto Pereira; Sant’Anna, Morena Brazil; Cunha, Fernando de Queiroz ; Ferreira, Sérgio Henrique; Cunha, Thiago Mattar
Fonte: Molecular Neurobiology. Unidade: FMRP
Assuntos: DOR, INFLAMAÇÃO, HIPERSENSIBILIDADE, NOCICEPTORES
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ABNT
PINTO, Larissa G. et al. Non-peptidergic nociceptive neurons are essential for mechanical inflammatory hypersensitivity in mice. Molecular Neurobiology, v. 56, n. 8, p. 5715-5728, 2019Tradução . . Disponível em: https://doi.org/10.1007/s12035-019-1494-5. Acesso em: 07 dez. 2025.
APA
Pinto, L. G., Souza, G. R., Kusuda, R., Lopes, A. H. P., Sant’Anna, M. B., Cunha, F. de Q., et al. (2019). Non-peptidergic nociceptive neurons are essential for mechanical inflammatory hypersensitivity in mice. Molecular Neurobiology, 56( 8), 5715-5728. doi:10.1007/s12035-019-1494-5
NLM
Pinto LG, Souza GR, Kusuda R, Lopes AHP, Sant’Anna MB, Cunha F de Q, Ferreira SH, Cunha TM. Non-peptidergic nociceptive neurons are essential for mechanical inflammatory hypersensitivity in mice [Internet]. Molecular Neurobiology. 2019 ; 56( 8): 5715-5728.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-019-1494-5
Vancouver
Pinto LG, Souza GR, Kusuda R, Lopes AHP, Sant’Anna MB, Cunha F de Q, Ferreira SH, Cunha TM. Non-peptidergic nociceptive neurons are essential for mechanical inflammatory hypersensitivity in mice [Internet]. Molecular Neurobiology. 2019 ; 56( 8): 5715-5728.[citado 2025 dez. 07 ] Available from: https://doi.org/10.1007/s12035-019-1494-5

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