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Artigo de periodico
High extracellular glucose concentration drives palmitate-induced toxicity and metabolic dysfunction in BV2 Microglia cells (2026)
Vilela, Wembley Rodrigues ; Platt, Nicole; Bechara, Luiz Roberto Grassmann ; Paula, Gabriela Cristina de ; Ferreira, Julio Cesar Batista ; Oliveira, Jade de ; Duarte, João Miguel das Neves ; Bem, Andreza Fabro de
Source: Molecular Neurobiology. Unidade: ICB
Subjects: ANATOMIA, NEURÓGLIA, ÁCIDOS GRAXOS, INFLAMAÇÃO, MITOCÔNDRIAS
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VILELA, Wembley Rodrigues et al. High extracellular glucose concentration drives palmitate-induced toxicity and metabolic dysfunction in BV2 Microglia cells. Molecular Neurobiology, v. 63, 2026Tradução . . Disponível em: https://doi.org/10.1007/s12035-025-05455-7. Acesso em: 03 maio 2026.
APA
Vilela, W. R., Platt, N., Bechara, L. R. G., Paula, G. C. de, Ferreira, J. C. B., Oliveira, J. de, et al. (2026). High extracellular glucose concentration drives palmitate-induced toxicity and metabolic dysfunction in BV2 Microglia cells. Molecular Neurobiology, 63. doi:10.1007/s12035-025-05455-7
NLM
Vilela WR, Platt N, Bechara LRG, Paula GC de, Ferreira JCB, Oliveira J de, Duarte JM das N, Bem AF de. High extracellular glucose concentration drives palmitate-induced toxicity and metabolic dysfunction in BV2 Microglia cells [Internet]. Molecular Neurobiology. 2026 ; 63[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-025-05455-7
Vancouver
Vilela WR, Platt N, Bechara LRG, Paula GC de, Ferreira JCB, Oliveira J de, Duarte JM das N, Bem AF de. High extracellular glucose concentration drives palmitate-induced toxicity and metabolic dysfunction in BV2 Microglia cells [Internet]. Molecular Neurobiology. 2026 ; 63[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-025-05455-7
Artigo de periodico
Chronic unpredictable stress impairs social behaviour in adult male rats: correlations with dopamine and serotonin levels (2026)
Mograbi, Karla De Michelis; Okeowo, Oritoke Modupe ; Sumarán, Paula ; Carvalho, Milene Cristina de ; Covolan, Luciene ; Hamani, Clement ; Suchecki, Deborah ; Dale, Camila Squarzoni
Source: Molecular Neurobiology. Unidades: ICB, FMRP
Subjects: ANATOMIA, ESTRESSE, HIPOCAMPU DE ANIMAL, COMPORTAMENTO ANIMAL, NEUROTRANSMISSORES, RECEPTORES DE DOPAMINA, RECEPTORES DE SEROTONINA, AMÍGDALA DO CEREBELO, CAMUNDONGOS, CITOCINAS
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MOGRABI, Karla De Michelis et al. Chronic unpredictable stress impairs social behaviour in adult male rats: correlations with dopamine and serotonin levels. Molecular Neurobiology, v. 63, p. m 15 , 2026Tradução . . Disponível em: https://doi.org/10.1007/s12035-025-05375-6. Acesso em: 03 maio 2026.
APA
Mograbi, K. D. M., Okeowo, O. M., Sumarán, P., Carvalho, M. C. de, Covolan, L., Hamani, C., et al. (2026). Chronic unpredictable stress impairs social behaviour in adult male rats: correlations with dopamine and serotonin levels. Molecular Neurobiology, 63, m 15 . doi:10.1007/s12035-025-05375-6
NLM
Mograbi KDM, Okeowo OM, Sumarán P, Carvalho MC de, Covolan L, Hamani C, Suchecki D, Dale CS. Chronic unpredictable stress impairs social behaviour in adult male rats: correlations with dopamine and serotonin levels [Internet]. Molecular Neurobiology. 2026 ; 63m 15 .[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-025-05375-6
Vancouver
Mograbi KDM, Okeowo OM, Sumarán P, Carvalho MC de, Covolan L, Hamani C, Suchecki D, Dale CS. Chronic unpredictable stress impairs social behaviour in adult male rats: correlations with dopamine and serotonin levels [Internet]. Molecular Neurobiology. 2026 ; 63m 15 .[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-025-05375-6
Artigo de periodico
Ayahuasca pretreatment prevents Sepsis-induced anxiety-ike behavior, Neuroinflammation, and oxidative stress, and increases brain-derived neurotrophic factor (2025)
Camargo, Rick Wilhiam de; Joaquim, Larissa; Machado, Richard Simon; Ramos, Suelen de Souza; Rosa, Lara Rodrigues da; Novais Junior, Linério Ribeiro de; Mathias, Khiany ; Maximiano, Lara; Strickert, Yasmin Ribeiro; Nord, Rafael; Gava, Maria Laura; Scarpari, Eduarda; Martins, Helena Mafra; Lins, Elisa Mitkus Flores; Chaves, Jéssica Schaefer; Silva, Larissa Espindola da; Oliveira, Mariana Pacheco de; Silva, Mariella Reinol da; Fernandes, Bruna Barros; Tiscoski, Anita dal Bó; Piacentini, Natália ; Santos, Fabiana Pereira; Yonamine, Maurício ; Petronilho, Fabrícia; Bitencourt, Rafael Mariano de
Source: Molecular Neurobiology. Unidade: FCF
Subjects: SEPSE, ALUCINOGÊNICOS
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CAMARGO, Rick Wilhiam de et al. Ayahuasca pretreatment prevents Sepsis-induced anxiety-ike behavior, Neuroinflammation, and oxidative stress, and increases brain-derived neurotrophic factor. Molecular Neurobiology, v. 62, n. 5, p. 5695-5719, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s12035-024-04597-4. Acesso em: 03 maio 2026.
APA
Camargo, R. W. de, Joaquim, L., Machado, R. S., Ramos, S. de S., Rosa, L. R. da, Novais Junior, L. R. de, et al. (2025). Ayahuasca pretreatment prevents Sepsis-induced anxiety-ike behavior, Neuroinflammation, and oxidative stress, and increases brain-derived neurotrophic factor. Molecular Neurobiology, 62( 5), 5695-5719. doi:10.1007/s12035-024-04597-4
NLM
Camargo RW de, Joaquim L, Machado RS, Ramos S de S, Rosa LR da, Novais Junior LR de, Mathias K, Maximiano L, Strickert YR, Nord R, Gava ML, Scarpari E, Martins HM, Lins EMF, Chaves JS, Silva LE da, Oliveira MP de, Silva MR da, Fernandes BB, Tiscoski A dal B, Piacentini N, Santos FP, Yonamine M, Petronilho F, Bitencourt RM de. Ayahuasca pretreatment prevents Sepsis-induced anxiety-ike behavior, Neuroinflammation, and oxidative stress, and increases brain-derived neurotrophic factor [Internet]. Molecular Neurobiology. 2025 ; 62( 5): 5695-5719.[citado 2026 maio 03 ] Available from: https://dx.doi.org/10.1007/s12035-024-04597-4
Vancouver
Camargo RW de, Joaquim L, Machado RS, Ramos S de S, Rosa LR da, Novais Junior LR de, Mathias K, Maximiano L, Strickert YR, Nord R, Gava ML, Scarpari E, Martins HM, Lins EMF, Chaves JS, Silva LE da, Oliveira MP de, Silva MR da, Fernandes BB, Tiscoski A dal B, Piacentini N, Santos FP, Yonamine M, Petronilho F, Bitencourt RM de. Ayahuasca pretreatment prevents Sepsis-induced anxiety-ike behavior, Neuroinflammation, and oxidative stress, and increases brain-derived neurotrophic factor [Internet]. Molecular Neurobiology. 2025 ; 62( 5): 5695-5719.[citado 2026 maio 03 ] Available from: https://dx.doi.org/10.1007/s12035-024-04597-4
Artigo de periodico
Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology (2025)
Ferreira, Ana Flávia Fernandes ; Mori, Yasuo ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Ulrich, Henning ; Britto, Luiz Roberto Giorgetti de
Source: Molecular Neurobiology. Unidades: ICB, IQ
Subjects: FISIOLOGIA, DOENÇA DE PARKINSON, PROTEÍNAS DA MEMBRANA, APOPTOSE, ATIVIDADE MOTORA, RECEPTORES DE DOPAMINA, ESTRESSE OXIDATIVO, CAMUNDONGOS
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FERREIRA, Ana Flávia Fernandes et al. Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology. Molecular Neurobiology, v. 62, n. 4, p. 5333-5346, 2025Tradução . . Disponível em: https://doi.org/10.1007/s12035-024-04611-9. Acesso em: 03 maio 2026.
APA
Ferreira, A. F. F., Mori, Y., Feng, Z. -P., Sun, H. -S., Ulrich, H., & Britto, L. R. G. de. (2025). Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology. Molecular Neurobiology, 62( 4), 5333-5346. doi:10.1007/s12035-024-04611-9
NLM
Ferreira AFF, Mori Y, Feng Z-P, Sun H-S, Ulrich H, Britto LRG de. Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology [Internet]. Molecular Neurobiology. 2025 ; 62( 4): 5333-5346.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-024-04611-9
Vancouver
Ferreira AFF, Mori Y, Feng Z-P, Sun H-S, Ulrich H, Britto LRG de. Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology [Internet]. Molecular Neurobiology. 2025 ; 62( 4): 5333-5346.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-024-04611-9
Artigo de periodico
Benfotiamine ameliorates streptozotocin-induced Alzheimer’s disease in rats by modulating neuroinflammation, oxidative stress, and microglia (2025)
Cardinali, Camila Aparecida Errerias Fernandes ; Martins, Yandara Akamine ; Moraes, Ruan Carlos Macêdo de ; Costa, Andressa Pereira ; Torrão, Andréa da Silva
Source: Molecular Neurobiology. Unidades: ICB, FM
Subjects: FISIOLOGIA, ASTRÓCITOS, NEURÓGLIA, VITAMINAS, RATOS WISTAR, DOENÇA DE ALZHEIMER, ANTIOXIDANTES, HIPOCAMPO, INFLAMAÇÃO, ESTRESSE OXIDATIVO
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CARDINALI, Camila Aparecida Errerias Fernandes et al. Benfotiamine ameliorates streptozotocin-induced Alzheimer’s disease in rats by modulating neuroinflammation, oxidative stress, and microglia. Molecular Neurobiology, v. 62, p. 8695-8717, 2025Tradução . . Disponível em: https://doi.org/10.1007/s12035-025-04811-x. Acesso em: 03 maio 2026.
APA
Cardinali, C. A. E. F., Martins, Y. A., Moraes, R. C. M. de, Costa, A. P., & Torrão, A. da S. (2025). Benfotiamine ameliorates streptozotocin-induced Alzheimer’s disease in rats by modulating neuroinflammation, oxidative stress, and microglia. Molecular Neurobiology, 62, 8695-8717. doi:10.1007/s12035-025-04811-x
NLM
Cardinali CAEF, Martins YA, Moraes RCM de, Costa AP, Torrão A da S. Benfotiamine ameliorates streptozotocin-induced Alzheimer’s disease in rats by modulating neuroinflammation, oxidative stress, and microglia [Internet]. Molecular Neurobiology. 2025 ; 62 8695-8717.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-025-04811-x
Vancouver
Cardinali CAEF, Martins YA, Moraes RCM de, Costa AP, Torrão A da S. Benfotiamine ameliorates streptozotocin-induced Alzheimer’s disease in rats by modulating neuroinflammation, oxidative stress, and microglia [Internet]. Molecular Neurobiology. 2025 ; 62 8695-8717.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-025-04811-x
Artigo de periodico
Metformin improves spatial memory and reduces seizure severity in a rat model of epilepsy and Alzheimer’s disease comorbidity via PI3K/Akt signaling pathway (2025)
Alves, Suélen Santos ; Rossi, Letícia; Oliveira, José Antonio Cortes de ; Servilha-Menezes, Gabriel ; Grigorio-de-Sant’Ana, Mariana ; Mazzei, Rodrigo Focosi ; Almeida, Sebastião Sousa ; Sebollela, Adriano Silva ; Silva Júnior, Rui Milton Patrício da ; Garcia-Cairasco, Norberto
Source: Molecular Neurobiology. Unidades: FMRP, FFCLRP
Subjects: CONVULSÕES, DOENÇA DE ALZHEIMER, COMORBIDADE, MODELOS ANIMAIS DE DOENÇAS, EPILEPSIA, HIPOCAMPO
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ALVES, Suélen Santos et al. Metformin improves spatial memory and reduces seizure severity in a rat model of epilepsy and Alzheimer’s disease comorbidity via PI3K/Akt signaling pathway. Molecular Neurobiology, v. 62, p. 9545-9572, 2025Tradução . . Disponível em: https://doi.org/10.1007/s12035-025-04844-2. Acesso em: 03 maio 2026.
APA
Alves, S. S., Rossi, L., Oliveira, J. A. C. de, Servilha-Menezes, G., Grigorio-de-Sant’Ana, M., Mazzei, R. F., et al. (2025). Metformin improves spatial memory and reduces seizure severity in a rat model of epilepsy and Alzheimer’s disease comorbidity via PI3K/Akt signaling pathway. Molecular Neurobiology, 62, 9545-9572. doi:10.1007/s12035-025-04844-2
NLM
Alves SS, Rossi L, Oliveira JAC de, Servilha-Menezes G, Grigorio-de-Sant’Ana M, Mazzei RF, Almeida SS, Sebollela AS, Silva Júnior RMP da, Garcia-Cairasco N. Metformin improves spatial memory and reduces seizure severity in a rat model of epilepsy and Alzheimer’s disease comorbidity via PI3K/Akt signaling pathway [Internet]. Molecular Neurobiology. 2025 ; 62 9545-9572.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-025-04844-2
Vancouver
Alves SS, Rossi L, Oliveira JAC de, Servilha-Menezes G, Grigorio-de-Sant’Ana M, Mazzei RF, Almeida SS, Sebollela AS, Silva Júnior RMP da, Garcia-Cairasco N. Metformin improves spatial memory and reduces seizure severity in a rat model of epilepsy and Alzheimer’s disease comorbidity via PI3K/Akt signaling pathway [Internet]. Molecular Neurobiology. 2025 ; 62 9545-9572.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-025-04844-2
Artigo de periodico
EHMT2 as a Candidate Gene for an Autosomal Recessive Neurodevelopmental Syndrome (2025)
Carvalho, Laura Machado Lara ; Jorge, Alexander Augusto de Lima ; Branco, Elisa Varella ; Oliveira, Danyllo Felipe de ; Haddad, Luciana Amaral ; Bertola, Débora Romeo ; Rosenberg, Carla ; Krepischi, Ana Cristina Victorino
Source: Molecular Neurobiology. Unidades: FM, IB
Subjects: GENÉTICA MÉDICA, MUTAÇÃO GENÉTICA, GENES
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CARVALHO, Laura Machado Lara et al. EHMT2 as a Candidate Gene for an Autosomal Recessive Neurodevelopmental Syndrome. Molecular Neurobiology, v. 62, p. 5977–5989, 2025Tradução . . Disponível em: https://doi.org/10.1007/s12035-024-04655-x. Acesso em: 03 maio 2026.
APA
Carvalho, L. M. L., Jorge, A. A. de L., Branco, E. V., Oliveira, D. F. de, Haddad, L. A., Bertola, D. R., et al. (2025). EHMT2 as a Candidate Gene for an Autosomal Recessive Neurodevelopmental Syndrome. Molecular Neurobiology, 62, 5977–5989. doi:10.1007/s12035-024-04655-x
NLM
Carvalho LML, Jorge AA de L, Branco EV, Oliveira DF de, Haddad LA, Bertola DR, Rosenberg C, Krepischi ACV. EHMT2 as a Candidate Gene for an Autosomal Recessive Neurodevelopmental Syndrome [Internet]. Molecular Neurobiology. 2025 ; 62 5977–5989.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-024-04655-x
Vancouver
Carvalho LML, Jorge AA de L, Branco EV, Oliveira DF de, Haddad LA, Bertola DR, Rosenberg C, Krepischi ACV. EHMT2 as a Candidate Gene for an Autosomal Recessive Neurodevelopmental Syndrome [Internet]. Molecular Neurobiology. 2025 ; 62 5977–5989.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-024-04655-x
Artigo de periodico
Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology (2025)
Ferreira, Ana Flávia Fernandes ; Ulrich, Henning ; Mori, Yasuo; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Molecular Neurobiology. Unidades: IQ, ICB
Subjects: DELEÇÃO DE GENES, DOENÇA DE PARKINSON, RATOS
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FERREIRA, Ana Flávia Fernandes et al. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology. Molecular Neurobiology, v. 62, p. 5333-5346, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s12035-024-04611-9. Acesso em: 03 maio 2026.
APA
Ferreira, A. F. F., Ulrich, H., Mori, Y., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2025). Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology. Molecular Neurobiology, 62, 5333-5346. doi:10.1007/s12035-024-04611-9
NLM
Ferreira AFF, Ulrich H, Mori Y, Feng Z-P, Sun H-S, Britto LRG de. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology [Internet]. Molecular Neurobiology. 2025 ; 62 5333-5346.[citado 2026 maio 03 ] Available from: https://dx.doi.org/10.1007/s12035-024-04611-9
Vancouver
Ferreira AFF, Ulrich H, Mori Y, Feng Z-P, Sun H-S, Britto LRG de. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology [Internet]. Molecular Neurobiology. 2025 ; 62 5333-5346.[citado 2026 maio 03 ] Available from: https://dx.doi.org/10.1007/s12035-024-04611-9
Artigo de periodico
Hippocampal interneurons shape spatial coding alterations in neurological disorders (2025)
Ikebara, Juliane Midori ; Jorge, Renata Silva ; Marinho, Luciana Simões Rafagnin; Higa, Guilherme Shigueto Vilar ; Adhikari, Avishek ; Reis, Fernando Midea Cuccovia Vasconcelos ; Borges, Fernando Silva ; Ulrich, Henning ; Takada, Silvia Honda ; Pasquale, Roberto De ; Kihara, Alexandre Hiroaki
Source: Molecular Neurobiology. Unidades: IQ, ICB
Subjects: FISIOLOGIA, HIPOCAMPO, DOENÇAS DO SISTEMA NERVOSO, NEURÔNIOS, DOENÇA DE ALZHEIMER, EPILEPSIA DO LOBO TEMPORAL
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IKEBARA, Juliane Midori et al. Hippocampal interneurons shape spatial coding alterations in neurological disorders. Molecular Neurobiology, v. 62, p. 14777-14800, 2025Tradução . . Disponível em: https://doi.org/10.1007/s12035-025-05020-2. Acesso em: 03 maio 2026.
APA
Ikebara, J. M., Jorge, R. S., Marinho, L. S. R., Higa, G. S. V., Adhikari, A., Reis, F. M. C. V., et al. (2025). Hippocampal interneurons shape spatial coding alterations in neurological disorders. Molecular Neurobiology, 62, 14777-14800. doi:10.1007/s12035-025-05020-2
NLM
Ikebara JM, Jorge RS, Marinho LSR, Higa GSV, Adhikari A, Reis FMCV, Borges FS, Ulrich H, Takada SH, Pasquale RD, Kihara AH. Hippocampal interneurons shape spatial coding alterations in neurological disorders [Internet]. Molecular Neurobiology. 2025 ; 62 14777-14800.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-025-05020-2
Vancouver
Ikebara JM, Jorge RS, Marinho LSR, Higa GSV, Adhikari A, Reis FMCV, Borges FS, Ulrich H, Takada SH, Pasquale RD, Kihara AH. Hippocampal interneurons shape spatial coding alterations in neurological disorders [Internet]. Molecular Neurobiology. 2025 ; 62 14777-14800.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-025-05020-2
Artigo de periodico
Clinical characterization and underlying genetic findings in brazilian patients with syndromic microcephaly associated with neurodevelopmental disorders (2024)
Tolezano, Giovanna Cantini; Bastos, Giovanna Civitate; Costa, Silvia Souza da; Scliar, Marília de Oliveira; Souza, Carolina Fischinger Moura de; Linden Jr, Hélio Van Der; Fernandes, Walter Luiz Magalhães; Otto, Paulo A ; Vianna-Morgante, Angela M ; Haddad, Luciana Amaral ; Honjo, Rachel Sayuri; Yamamoto, Guilherme Lopes; Kim, Chong Ae; Rosenberg, Carla ; Jorge, Alexander Augusto de Lima ; Bertola, Débora Romeo ; Krepischi, Ana Cristina Victorino
Source: Molecular Neurobiology. Unidades: IB, Interunidades em Bioinformática, FM
Subjects: MICROCEFALIA, NEUROLOGIA, GENÉTICA
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TOLEZANO, Giovanna Cantini et al. Clinical characterization and underlying genetic findings in brazilian patients with syndromic microcephaly associated with neurodevelopmental disorders. Molecular Neurobiology, 2024Tradução . . Disponível em: https://doi.org/10.1007/s12035-023-03894-8. Acesso em: 03 maio 2026.
APA
Tolezano, G. C., Bastos, G. C., Costa, S. S. da, Scliar, M. de O., Souza, C. F. M. de, Linden Jr, H. V. D., et al. (2024). Clinical characterization and underlying genetic findings in brazilian patients with syndromic microcephaly associated with neurodevelopmental disorders. Molecular Neurobiology. doi:10.1007/s12035-023-03894-8
NLM
Tolezano GC, Bastos GC, Costa SS da, Scliar M de O, Souza CFM de, Linden Jr HVD, Fernandes WLM, Otto PA, Vianna-Morgante AM, Haddad LA, Honjo RS, Yamamoto GL, Kim CA, Rosenberg C, Jorge AA de L, Bertola DR, Krepischi ACV. Clinical characterization and underlying genetic findings in brazilian patients with syndromic microcephaly associated with neurodevelopmental disorders [Internet]. Molecular Neurobiology. 2024 ;[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-023-03894-8
Vancouver
Tolezano GC, Bastos GC, Costa SS da, Scliar M de O, Souza CFM de, Linden Jr HVD, Fernandes WLM, Otto PA, Vianna-Morgante AM, Haddad LA, Honjo RS, Yamamoto GL, Kim CA, Rosenberg C, Jorge AA de L, Bertola DR, Krepischi ACV. Clinical characterization and underlying genetic findings in brazilian patients with syndromic microcephaly associated with neurodevelopmental disorders [Internet]. Molecular Neurobiology. 2024 ;[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-023-03894-8
Artigo de periodico
A Purine derivative containing an organoselenium group protects against memory impairment, sensitivity to nociception, oxidative damage, and neuroinflammation in a mouse model of Alzheimer’s disease (2023)
Pinz, Mikaela Peglow ; Oliveira, Renata Leivas de ; Fonseca, Caren Aline Ramson da ; Voss, Guilherme Teixeira ; Silva, Beatriz Pereira da ; Duarte, Luis Fernando Barbosa ; Domingues, William Borges; Ortiz, Hadassa Gabriela; Savall, Anne Suély Pinto ; Meotti, Flavia Carla ; Alves, Diego ; Campos, Vinicius Farias ; Pinton, Simone ; Wilhelm, Ethel Antunes ; Luchese, Cristiane
Source: Molecular Neurobiology. Unidade: IQ
Subjects: DOENÇA DE ALZHEIMER, ESTRESSE OXIDATIVO
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ABNT
PINZ, Mikaela Peglow et al. A Purine derivative containing an organoselenium group protects against memory impairment, sensitivity to nociception, oxidative damage, and neuroinflammation in a mouse model of Alzheimer’s disease. Molecular Neurobiology, v. 60, p. 1214–1231, 2023Tradução . . Disponível em: https://doi.org/10.1007/s12035-022-03110-z. Acesso em: 03 maio 2026.
APA
Pinz, M. P., Oliveira, R. L. de, Fonseca, C. A. R. da, Voss, G. T., Silva, B. P. da, Duarte, L. F. B., et al. (2023). A Purine derivative containing an organoselenium group protects against memory impairment, sensitivity to nociception, oxidative damage, and neuroinflammation in a mouse model of Alzheimer’s disease. Molecular Neurobiology, 60, 1214–1231. doi:10.1007/s12035-022-03110-z
NLM
Pinz MP, Oliveira RL de, Fonseca CAR da, Voss GT, Silva BP da, Duarte LFB, Domingues WB, Ortiz HG, Savall ASP, Meotti FC, Alves D, Campos VF, Pinton S, Wilhelm EA, Luchese C. A Purine derivative containing an organoselenium group protects against memory impairment, sensitivity to nociception, oxidative damage, and neuroinflammation in a mouse model of Alzheimer’s disease [Internet]. Molecular Neurobiology. 2023 ; 60 1214–1231.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-022-03110-z
Vancouver
Pinz MP, Oliveira RL de, Fonseca CAR da, Voss GT, Silva BP da, Duarte LFB, Domingues WB, Ortiz HG, Savall ASP, Meotti FC, Alves D, Campos VF, Pinton S, Wilhelm EA, Luchese C. A Purine derivative containing an organoselenium group protects against memory impairment, sensitivity to nociception, oxidative damage, and neuroinflammation in a mouse model of Alzheimer’s disease [Internet]. Molecular Neurobiology. 2023 ; 60 1214–1231.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-022-03110-z
Artigo de periodico
Skewed x-chromosome inactivation in women with idiopathic intellectual disability is indicative of pathogenic variants (2023)
Chaves, Luiza D; Carvalho, Laura M. L; Tolezano, Giovanna C; Pires, Sara F; Costa, Silvia S; Scliar, Marília O. de; Giuliani, Liane de R; Bertola, Débora Romeo ; Santos-Rebouças, Cíntia B; Seo, Go Hun; Otto, Paulo A ; Rosenberg, Carla ; Vianna-Morgante, Angela M ; Krepischi, Ana Cristina Victorino
Source: Molecular Neurobiology. Unidade: IB
Subjects: MULHERES, CROMOSSOMO X, RETARDO MENTAL, GENÉTICA MÉDICA
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ABNT
CHAVES, Luiza D et al. Skewed x-chromosome inactivation in women with idiopathic intellectual disability is indicative of pathogenic variants. Molecular Neurobiology, v. 60, p. 3758–3769, 2023Tradução . . Disponível em: https://doi.org/10.1007/s12035-023-03311-0. Acesso em: 03 maio 2026.
APA
Chaves, L. D., Carvalho, L. M. L., Tolezano, G. C., Pires, S. F., Costa, S. S., Scliar, M. O. de, et al. (2023). Skewed x-chromosome inactivation in women with idiopathic intellectual disability is indicative of pathogenic variants. Molecular Neurobiology, 60, 3758–3769. doi:10.1007/s12035-023-03311-0
NLM
Chaves LD, Carvalho LML, Tolezano GC, Pires SF, Costa SS, Scliar MO de, Giuliani L de R, Bertola DR, Santos-Rebouças CB, Seo GH, Otto PA, Rosenberg C, Vianna-Morgante AM, Krepischi ACV. Skewed x-chromosome inactivation in women with idiopathic intellectual disability is indicative of pathogenic variants [Internet]. Molecular Neurobiology. 2023 ; 60 3758–3769.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-023-03311-0
Vancouver
Chaves LD, Carvalho LML, Tolezano GC, Pires SF, Costa SS, Scliar MO de, Giuliani L de R, Bertola DR, Santos-Rebouças CB, Seo GH, Otto PA, Rosenberg C, Vianna-Morgante AM, Krepischi ACV. Skewed x-chromosome inactivation in women with idiopathic intellectual disability is indicative of pathogenic variants [Internet]. Molecular Neurobiology. 2023 ; 60 3758–3769.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-023-03311-0
Artigo de periodico
Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model (2022)
Ferreira, Ana Flávia Fernandes ; Singulani, Monique Patricio ; Ulrich, Henning ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Molecular Neurobiology. Unidades: IQ, ICB, FM
Subjects: FISIOLOGIA, NEUROBIOLOGIA, MODELOS ANIMAIS DE DOENÇAS, DOENÇA DE PARKINSON, MESENCÉFALO, GÂNGLIOS, RECEPTORES DE DOPAMINA, CAMUNDONGOS, IMUNOHISTOQUÍMICA, IMMUNOBLOTTING
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ABNT
FERREIRA, Ana Flávia Fernandes et al. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model. Molecular Neurobiology, v. 59, p. 1543–1559, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-022-02723-8. Acesso em: 03 maio 2026.
APA
Ferreira, A. F. F., Singulani, M. P., Ulrich, H., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2022). Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model. Molecular Neurobiology, 59, 1543–1559. doi:10.1007/s12035-022-02723-8
NLM
Ferreira AFF, Singulani MP, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model [Internet]. Molecular Neurobiology. 2022 ; 59 1543–1559.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-022-02723-8
Vancouver
Ferreira AFF, Singulani MP, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Inhibition of TRPM2 by AG490 is neuroprotective in a Parkinson’s Disease animal model [Internet]. Molecular Neurobiology. 2022 ; 59 1543–1559.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-022-02723-8
Artigo de periodico
Photobiomodulation improves the inflammatory response and intracellular signaling proteins linked to vascular function and cell survival in the brain of aged rats (2022)
Cardoso, Fabrízio dos Santos ; Mansur, Fernanda Cristina Borini; Araujo, Bruno Henrique Silva; Gonzalez-Lima, F.; Silva, Sérgio Gomes da
Source: Molecular Neurobiology. Unidade: FMRP
Subjects: ENVOLVIMENTO, CÉREBRO, SOBREVIVÊNCIA CELULAR, CITOCINAS, INFLAMAÇÃO, TECIDO NERVOSO
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ABNT
CARDOSO, Fabrízio dos Santos et al. Photobiomodulation improves the inflammatory response and intracellular signaling proteins linked to vascular function and cell survival in the brain of aged rats. Molecular Neurobiology, v. 59, n. 1, p. 420-428, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-021-02606-4. Acesso em: 03 maio 2026.
APA
Cardoso, F. dos S., Mansur, F. C. B., Araujo, B. H. S., Gonzalez-Lima, F., & Silva, S. G. da. (2022). Photobiomodulation improves the inflammatory response and intracellular signaling proteins linked to vascular function and cell survival in the brain of aged rats. Molecular Neurobiology, 59( 1), 420-428. doi:10.1007/s12035-021-02606-4
NLM
Cardoso F dos S, Mansur FCB, Araujo BHS, Gonzalez-Lima F, Silva SG da. Photobiomodulation improves the inflammatory response and intracellular signaling proteins linked to vascular function and cell survival in the brain of aged rats [Internet]. Molecular Neurobiology. 2022 ; 59( 1): 420-428.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-021-02606-4
Vancouver
Cardoso F dos S, Mansur FCB, Araujo BHS, Gonzalez-Lima F, Silva SG da. Photobiomodulation improves the inflammatory response and intracellular signaling proteins linked to vascular function and cell survival in the brain of aged rats [Internet]. Molecular Neurobiology. 2022 ; 59( 1): 420-428.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-021-02606-4
Artigo de periodico
A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance (2022)
Alves, Suélen Santos ; Silva Júnior, Rui Milton Patrício da; Delfino-Pereira, Polianna; Pereira, Marília Gabriella Alves Goulart; Vasconcelos, Israel Costa; Schwaemmle, Hanna; Mazzei, Rodrigo Focosi; Carlos, Maiko Luiz ; Espreáfico, Enilza Maria; Tedesco, Antônio Cláudio ; Sebollela, Adriano Silva ; Almeida, Sebastião de Sousa ; Oliveira, José Antonio Cortes de; Garcia-Cairasco, Norberto
Source: Molecular Neurobiology. Unidades: FFCLRP, FMRP
Subjects: EPILEPSIA, DOENÇA DE ALZHEIMER, RESISTÊNCIA À INSULINA, MEMÓRIA
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ABNT
ALVES, Suélen Santos et al. A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance. Molecular Neurobiology, v. 59, n. 6, p. 3721-3737, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-022-02810-w. Acesso em: 03 maio 2026.
APA
Alves, S. S., Silva Júnior, R. M. P. da, Delfino-Pereira, P., Pereira, M. G. A. G., Vasconcelos, I. C., Schwaemmle, H., et al. (2022). A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance. Molecular Neurobiology, 59( 6), 3721-3737. doi:10.1007/s12035-022-02810-w
NLM
Alves SS, Silva Júnior RMP da, Delfino-Pereira P, Pereira MGAG, Vasconcelos IC, Schwaemmle H, Mazzei RF, Carlos ML, Espreáfico EM, Tedesco AC, Sebollela AS, Almeida S de S, Oliveira JAC de, Garcia-Cairasco N. A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance [Internet]. Molecular Neurobiology. 2022 ; 59( 6): 3721-3737.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-022-02810-w
Vancouver
Alves SS, Silva Júnior RMP da, Delfino-Pereira P, Pereira MGAG, Vasconcelos IC, Schwaemmle H, Mazzei RF, Carlos ML, Espreáfico EM, Tedesco AC, Sebollela AS, Almeida S de S, Oliveira JAC de, Garcia-Cairasco N. A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance [Internet]. Molecular Neurobiology. 2022 ; 59( 6): 3721-3737.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-022-02810-w
Artigo de periodico
Metabolic profile in plasma and CSF of LEVODOPA-induced dyskinesia in Parkinson’s disease: focus on neuroinflammation (2022)
Santos-Lobato, Bruno L.; Gardinassi, Luiz Gustavo; Bortolanza, Mariza; Peti, Ana Paula Ferranti; Pimentel, Ângela V.; Faccioli, Lucia Helena ; Del Bel, Elaine Aparecida ; Tumas, Vitor
Source: Molecular Neurobiology. Unidades: FMRP, FCFRP, FORP
Subjects: METABOLÔMICA, DOENÇA DE PARKINSON, DISCINESIAS, FÁRMACOS (SISTEMA NERVOSO CENTRAL)
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ABNT
SANTOS-LOBATO, Bruno L. et al. Metabolic profile in plasma and CSF of LEVODOPA-induced dyskinesia in Parkinson’s disease: focus on neuroinflammation. Molecular Neurobiology, v. 59, n. 2, p. 1140-1150, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-021-02625-1. Acesso em: 03 maio 2026.
APA
Santos-Lobato, B. L., Gardinassi, L. G., Bortolanza, M., Peti, A. P. F., Pimentel, Â. V., Faccioli, L. H., et al. (2022). Metabolic profile in plasma and CSF of LEVODOPA-induced dyskinesia in Parkinson’s disease: focus on neuroinflammation. Molecular Neurobiology, 59( 2), 1140-1150. doi:10.1007/s12035-021-02625-1
NLM
Santos-Lobato BL, Gardinassi LG, Bortolanza M, Peti APF, Pimentel ÂV, Faccioli LH, Del Bel EA, Tumas V. Metabolic profile in plasma and CSF of LEVODOPA-induced dyskinesia in Parkinson’s disease: focus on neuroinflammation [Internet]. Molecular Neurobiology. 2022 ; 59( 2): 1140-1150.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-021-02625-1
Vancouver
Santos-Lobato BL, Gardinassi LG, Bortolanza M, Peti APF, Pimentel ÂV, Faccioli LH, Del Bel EA, Tumas V. Metabolic profile in plasma and CSF of LEVODOPA-induced dyskinesia in Parkinson’s disease: focus on neuroinflammation [Internet]. Molecular Neurobiology. 2022 ; 59( 2): 1140-1150.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-021-02625-1
Artigo de periodico
Neuroprotective effect of exogenous galectin-1 in status epilepticus (2022)
Pacheco, Amanda Larissa Dias; Melo, Igor Santana de; Costa, Maisa de Araujo; Amaral, Mariah Morais Celestino; Silva, Nívea Karla De Gusmão Taveiros ; Santos, Yngrid Mickaelli Oliveira; Gitaí, Daniel Leite Goes; Duzzioni, Marcelo; Borbely, Alexandre Urban; Silva, Robinson Sabino da; Donatti, Ana Luiza Ferreira; Mestriner, Luisa; Fuzo, Carlos Alessandro ; Cummings, Richard D.; Garcia-Cairasco, Norberto ; Dias-Baruffi, Marcelo ; Castro, Olagide Wagner de
Source: Molecular Neurobiology. Unidades: FMRP, FCFRP
Subjects: EPILEPSIA, EPILEPSIA DO LOBO TEMPORAL
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ABNT
PACHECO, Amanda Larissa Dias et al. Neuroprotective effect of exogenous galectin-1 in status epilepticus. Molecular Neurobiology, v. 59, n. 12, p. 7354-7369, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-022-03038-4. Acesso em: 03 maio 2026.
APA
Pacheco, A. L. D., Melo, I. S. de, Costa, M. de A., Amaral, M. M. C., Silva, N. K. D. G. T., Santos, Y. M. O., et al. (2022). Neuroprotective effect of exogenous galectin-1 in status epilepticus. Molecular Neurobiology, 59( 12), 7354-7369. doi:10.1007/s12035-022-03038-4
NLM
Pacheco ALD, Melo IS de, Costa M de A, Amaral MMC, Silva NKDGT, Santos YMO, Gitaí DLG, Duzzioni M, Borbely AU, Silva RS da, Donatti ALF, Mestriner L, Fuzo CA, Cummings RD, Garcia-Cairasco N, Dias-Baruffi M, Castro OW de. Neuroprotective effect of exogenous galectin-1 in status epilepticus [Internet]. Molecular Neurobiology. 2022 ; 59( 12): 7354-7369.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-022-03038-4
Vancouver
Pacheco ALD, Melo IS de, Costa M de A, Amaral MMC, Silva NKDGT, Santos YMO, Gitaí DLG, Duzzioni M, Borbely AU, Silva RS da, Donatti ALF, Mestriner L, Fuzo CA, Cummings RD, Garcia-Cairasco N, Dias-Baruffi M, Castro OW de. Neuroprotective effect of exogenous galectin-1 in status epilepticus [Internet]. Molecular Neurobiology. 2022 ; 59( 12): 7354-7369.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-022-03038-4
Artigo de periodico
Norepinephrine and glucocorticoids modulate chronic unpredictable stress-induced increase in the Type 2 CRF and glucocorticoid receptors in brain structures related to the HPA Axis activation (2021)
Malta, Marilia Brinati; Silva, Joelcimar Martins da; Novaes, Leonardo Santana ; Santos, Nilton Barreto dos ; Sita, Luciane Valéria ; Camarini, Rosana ; Scavone, Cristoforo ; Bittencourt, Jackson Cioni ; Munhoz, Carolina Demarchi
Source: Molecular Neurobiology. Unidade: ICB
Subjects: FARMACOLOGIA, ANATOMIA, NORADRENALINA, RECEPTORES DE GLICOCORTICOIDES, ESTRESSE, COMPORTAMENTO, TRANSTORNOS DA ANSIEDADE, HIPOTÁLAMO, HORMÔNIOS DO CÓRTEX ADRENAL, HORMÔNIOS GLICOCORTICOIDES, NOREPINEFRINA
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ABNT
MALTA, Marilia Brinati et al. Norepinephrine and glucocorticoids modulate chronic unpredictable stress-induced increase in the Type 2 CRF and glucocorticoid receptors in brain structures related to the HPA Axis activation. Molecular Neurobiology, p. 1-15, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12035-021-02470-2. Acesso em: 03 maio 2026.
APA
Malta, M. B., Silva, J. M. da, Novaes, L. S., Santos, N. B. dos, Sita, L. V., Camarini, R., et al. (2021). Norepinephrine and glucocorticoids modulate chronic unpredictable stress-induced increase in the Type 2 CRF and glucocorticoid receptors in brain structures related to the HPA Axis activation. Molecular Neurobiology, 1-15. doi:10.1007/s12035-021-02470-2
NLM
Malta MB, Silva JM da, Novaes LS, Santos NB dos, Sita LV, Camarini R, Scavone C, Bittencourt JC, Munhoz CD. Norepinephrine and glucocorticoids modulate chronic unpredictable stress-induced increase in the Type 2 CRF and glucocorticoid receptors in brain structures related to the HPA Axis activation [Internet]. Molecular Neurobiology. 2021 ; 1-15.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-021-02470-2
Vancouver
Malta MB, Silva JM da, Novaes LS, Santos NB dos, Sita LV, Camarini R, Scavone C, Bittencourt JC, Munhoz CD. Norepinephrine and glucocorticoids modulate chronic unpredictable stress-induced increase in the Type 2 CRF and glucocorticoid receptors in brain structures related to the HPA Axis activation [Internet]. Molecular Neurobiology. 2021 ; 1-15.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-021-02470-2
Artigo de periodico
Cannabidiol confers neuroprotection in rats in a model of transient global cerebral ischemia: impact of hippocampal synaptic neuroplasticity (2021)
Meyer, Erika; Bonato, Jéssica Mendes; Mori, Marco Aurélio; Mattos, Bianca Andretto; Guimarães, Francisco Silveira ; Milani, Humberto; Campos, Alline Cristina de; Oliveira, Rúbia Maria Weffort de
Source: Molecular Neurobiology. Unidade: FMRP
Subjects: CANABINOIDES, HIPOCAMPO, PLASTICIDADE NEURONAL, CULTURA DE ORGÃOS, SINAPSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MEYER, Erika et al. Cannabidiol confers neuroprotection in rats in a model of transient global cerebral ischemia: impact of hippocampal synaptic neuroplasticity. Molecular Neurobiology, v. 58, n. 10, p. 5338-5355, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12035-021-02479-7. Acesso em: 03 maio 2026.
APA
Meyer, E., Bonato, J. M., Mori, M. A., Mattos, B. A., Guimarães, F. S., Milani, H., et al. (2021). Cannabidiol confers neuroprotection in rats in a model of transient global cerebral ischemia: impact of hippocampal synaptic neuroplasticity. Molecular Neurobiology, 58( 10), 5338-5355. doi:10.1007/s12035-021-02479-7
NLM
Meyer E, Bonato JM, Mori MA, Mattos BA, Guimarães FS, Milani H, Campos AC de, Oliveira RMW de. Cannabidiol confers neuroprotection in rats in a model of transient global cerebral ischemia: impact of hippocampal synaptic neuroplasticity [Internet]. Molecular Neurobiology. 2021 ; 58( 10): 5338-5355.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-021-02479-7
Vancouver
Meyer E, Bonato JM, Mori MA, Mattos BA, Guimarães FS, Milani H, Campos AC de, Oliveira RMW de. Cannabidiol confers neuroprotection in rats in a model of transient global cerebral ischemia: impact of hippocampal synaptic neuroplasticity [Internet]. Molecular Neurobiology. 2021 ; 58( 10): 5338-5355.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-021-02479-7
Artigo de periodico
The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth (2021)
Teixeira, Silvia Aparecida; Viapiano, Mariano S.; Andrade, Augusto F.; Nandhu, Mohan S.; Pezuk, Julia Alejandra; Bidinotto, Lucas T.; Sant'Ana, Veridiana Kiill Suazo de; Serafini, Luciano Neder; Carlotti Júnior, Carlos Gilberto ; Becker, Aline P.; Tone, Luiz Gonzaga ; Scrideli, Carlos Alberto
Source: Molecular Neurobiology. Unidade: FMRP
Subjects: ANIDRASE CARBÔNICA, METABOLISMO CELULAR, NEOPLASIAS CEREBRAIS, FARMACOTERAPIA, LINHAGEM CELULAR, CICLO CELULAR, SULFONAMIDAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TEIXEIRA, Silvia Aparecida et al. The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth. Molecular Neurobiology, v. 58, n. 9, p. 4520-4534, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12035-021-02437-3. Acesso em: 03 maio 2026.
APA
Teixeira, S. A., Viapiano, M. S., Andrade, A. F., Nandhu, M. S., Pezuk, J. A., Bidinotto, L. T., et al. (2021). The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth. Molecular Neurobiology, 58( 9), 4520-4534. doi:10.1007/s12035-021-02437-3
NLM
Teixeira SA, Viapiano MS, Andrade AF, Nandhu MS, Pezuk JA, Bidinotto LT, Sant'Ana VKS de, Serafini LN, Carlotti Júnior CG, Becker AP, Tone LG, Scrideli CA. The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth [Internet]. Molecular Neurobiology. 2021 ; 58( 9): 4520-4534.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-021-02437-3
Vancouver
Teixeira SA, Viapiano MS, Andrade AF, Nandhu MS, Pezuk JA, Bidinotto LT, Sant'Ana VKS de, Serafini LN, Carlotti Júnior CG, Becker AP, Tone LG, Scrideli CA. The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth [Internet]. Molecular Neurobiology. 2021 ; 58( 9): 4520-4534.[citado 2026 maio 03 ] Available from: https://doi.org/10.1007/s12035-021-02437-3

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