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Artigo de periodico
Uncovering the role of DNA repair impairment in UVA-induced mutagenesis in human Xeroderma Pigmentosum variant cells (2025)
Corradi, Camila ; Moreno, Natália Cestari ; Ruiz, Nathalia Quintero ; Leandro, Giovana da Silva ; Latância, Marcela Teatin ; Souza, Tiago Antonio de ; Munford, Veridiana ; Menck, Carlos Frederico Martins
Source: Molecular Carcinogenesis. Unidades: ICB, Interunidades em Bioinformática
Assunto: MICROBIOLOGIA
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CORRADI, Camila et al. Uncovering the role of DNA repair impairment in UVA-induced mutagenesis in human Xeroderma Pigmentosum variant cells. Molecular Carcinogenesis, v. 64, p. 1823-1837, 2025Tradução . . Disponível em: https://doi.org/10.1002/mc.70028. Acesso em: 23 abr. 2026.
APA
Corradi, C., Moreno, N. C., Ruiz, N. Q., Leandro, G. da S., Latância, M. T., Souza, T. A. de, et al. (2025). Uncovering the role of DNA repair impairment in UVA-induced mutagenesis in human Xeroderma Pigmentosum variant cells. Molecular Carcinogenesis, 64, 1823-1837. doi:10.1002/mc.70028
NLM
Corradi C, Moreno NC, Ruiz NQ, Leandro G da S, Latância MT, Souza TA de, Munford V, Menck CFM. Uncovering the role of DNA repair impairment in UVA-induced mutagenesis in human Xeroderma Pigmentosum variant cells [Internet]. Molecular Carcinogenesis. 2025 ; 64 1823-1837.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.70028
Vancouver
Corradi C, Moreno NC, Ruiz NQ, Leandro G da S, Latância MT, Souza TA de, Munford V, Menck CFM. Uncovering the role of DNA repair impairment in UVA-induced mutagenesis in human Xeroderma Pigmentosum variant cells [Internet]. Molecular Carcinogenesis. 2025 ; 64 1823-1837.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.70028
Artigo de periodico
Inhibition of mTOR in head and neck cancer cells alters endothelial cell morphology in a paracrine fashion (2019)
Duarte, Andressa ; André-Grégoire, Gwennan; Trillet, Kilian; Thys, An; Bidère, Nicolas; Ribeiro-Silva, Alfredo ; Gavard, Julie
Source: Molecular Carcinogenesis. Unidade: FMRP
Subjects: NEOPLASIAS DE CABEÇA E PESCOÇO, CÉLULAS ENDOTELIAIS
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DUARTE, Andressa et al. Inhibition of mTOR in head and neck cancer cells alters endothelial cell morphology in a paracrine fashion. Molecular Carcinogenesis, v. 58, n. 1, p. 161-168, 2019Tradução . . Disponível em: https://doi.org/10.1002/mc.22911. Acesso em: 23 abr. 2026.
APA
Duarte, A., André-Grégoire, G., Trillet, K., Thys, A., Bidère, N., Ribeiro-Silva, A., & Gavard, J. (2019). Inhibition of mTOR in head and neck cancer cells alters endothelial cell morphology in a paracrine fashion. Molecular Carcinogenesis, 58( 1), 161-168. doi:10.1002/mc.22911
NLM
Duarte A, André-Grégoire G, Trillet K, Thys A, Bidère N, Ribeiro-Silva A, Gavard J. Inhibition of mTOR in head and neck cancer cells alters endothelial cell morphology in a paracrine fashion [Internet]. Molecular Carcinogenesis. 2019 ; 58( 1): 161-168.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.22911
Vancouver
Duarte A, André-Grégoire G, Trillet K, Thys A, Bidère N, Ribeiro-Silva A, Gavard J. Inhibition of mTOR in head and neck cancer cells alters endothelial cell morphology in a paracrine fashion [Internet]. Molecular Carcinogenesis. 2019 ; 58( 1): 161-168.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.22911
Artigo de periodico
β-ionone modulates the expression of miRNAs and genes involved in the metastatic phenotype of microdissected persistent preneoplastic lesions in rats submitted to hepatocarcinogenesis (2017)
Furtado, kelly Silva; Andrade, Fábia de Oliveira; Campos, Adriana; Rosim, Mariana Papaléo; Vargas Mendez, Ernesto; Henriques, Aline; De Conti, Aline; Scolastici, Clarissa; Barbisan, Luis Fernando; Carvalho, Robson Francisco; Moreno, Fernando Salvador
Source: Molecular Carcinogenesis. Unidade: FCF
Subjects: FENÓTIPOS, CARCINOMA HEPATOCELULAR
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FURTADO, kelly Silva et al. β-ionone modulates the expression of miRNAs and genes involved in the metastatic phenotype of microdissected persistent preneoplastic lesions in rats submitted to hepatocarcinogenesis. Molecular Carcinogenesis, v. 56, n. 1, p. 184-196, 2017Tradução . . Disponível em: https://doi.org/10.1002/mc.22483. Acesso em: 23 abr. 2026.
APA
Furtado, kelly S., Andrade, F. de O., Campos, A., Rosim, M. P., Vargas Mendez, E., Henriques, A., et al. (2017). β-ionone modulates the expression of miRNAs and genes involved in the metastatic phenotype of microdissected persistent preneoplastic lesions in rats submitted to hepatocarcinogenesis. Molecular Carcinogenesis, 56( 1), 184-196. doi:10.1002/mc.22483
NLM
Furtado kelly S, Andrade F de O, Campos A, Rosim MP, Vargas Mendez E, Henriques A, De Conti A, Scolastici C, Barbisan LF, Carvalho RF, Moreno FS. β-ionone modulates the expression of miRNAs and genes involved in the metastatic phenotype of microdissected persistent preneoplastic lesions in rats submitted to hepatocarcinogenesis [Internet]. Molecular Carcinogenesis. 2017 ; 56( 1): 184-196.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.22483
Vancouver
Furtado kelly S, Andrade F de O, Campos A, Rosim MP, Vargas Mendez E, Henriques A, De Conti A, Scolastici C, Barbisan LF, Carvalho RF, Moreno FS. β-ionone modulates the expression of miRNAs and genes involved in the metastatic phenotype of microdissected persistent preneoplastic lesions in rats submitted to hepatocarcinogenesis [Internet]. Molecular Carcinogenesis. 2017 ; 56( 1): 184-196.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.22483
Artigo de periodico
Chronic exposure of lung alveolar epithelial type II cells to tobacco-specific carcinogen nnk results in malignant transformation: a new in vitro lung carcinogenesis model (2014)
Mennecier, Gregory; Torres, Luciana Neves; Cogliati, Bruno ; Sanches, Daniel Soares; Mori, Claudia Madalena Cabrera ; Latorre, Andréia Oliveira; Chaible, Lucas Martins; Mackowiak, Ivone Isabel; Nagamine, Márcia Kazumi; Silva, Tereza C. da; Fukumasu, Heidge; Dagli, Maria Lúcia Zaidan
Source: Molecular Carcinogenesis. Unidades: FMVZ, FZEA
Subjects: FUMO, NEOPLASIAS PULMONARES, ONCOLOGIA
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MENNECIER, Gregory et al. Chronic exposure of lung alveolar epithelial type II cells to tobacco-specific carcinogen nnk results in malignant transformation: a new in vitro lung carcinogenesis model. Molecular Carcinogenesis, v. 53, n. 5, p. 392-402, 2014Tradução . . Disponível em: https://doi.org/10.1002/mc.21987. Acesso em: 23 abr. 2026.
APA
Mennecier, G., Torres, L. N., Cogliati, B., Sanches, D. S., Mori, C. M. C., Latorre, A. O., et al. (2014). Chronic exposure of lung alveolar epithelial type II cells to tobacco-specific carcinogen nnk results in malignant transformation: a new in vitro lung carcinogenesis model. Molecular Carcinogenesis, 53( 5), 392-402. doi:10.1002/mc.21987
NLM
Mennecier G, Torres LN, Cogliati B, Sanches DS, Mori CMC, Latorre AO, Chaible LM, Mackowiak II, Nagamine MK, Silva TC da, Fukumasu H, Dagli MLZ. Chronic exposure of lung alveolar epithelial type II cells to tobacco-specific carcinogen nnk results in malignant transformation: a new in vitro lung carcinogenesis model [Internet]. Molecular Carcinogenesis. 2014 ; 53( 5): 392-402.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.21987
Vancouver
Mennecier G, Torres LN, Cogliati B, Sanches DS, Mori CMC, Latorre AO, Chaible LM, Mackowiak II, Nagamine MK, Silva TC da, Fukumasu H, Dagli MLZ. Chronic exposure of lung alveolar epithelial type II cells to tobacco-specific carcinogen nnk results in malignant transformation: a new in vitro lung carcinogenesis model [Internet]. Molecular Carcinogenesis. 2014 ; 53( 5): 392-402.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.21987
Artigo de periodico
Higher susceptibility of spontaneous and NNK-induced lung neoplasms in connexin 43 deficient CD1 x AJ F1 mice: paradoxical expression of connexin 43 during lung carcinogenesis (2013)
Fukumasu, Heidge; Avanzo, Jose L; Sanches, Daniel S; Mennecier, Gregory; Mori, Claudia Madalena Cabrera ; Dagli, Maria Lúcia Zaidan
Source: Molecular Carcinogenesis. Unidades: FZEA, FMVZ
Subjects: NEOPLASIAS PULMONARES, RATOS, PROTEÍNAS DA MEMBRANA, CARCINOGÊNESE ANIMAL
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FUKUMASU, Heidge et al. Higher susceptibility of spontaneous and NNK-induced lung neoplasms in connexin 43 deficient CD1 x AJ F1 mice: paradoxical expression of connexin 43 during lung carcinogenesis. Molecular Carcinogenesis, v. 52, n. 7, p. 497-506, 2013Tradução . . Disponível em: https://doi.org/10.1002/mc.21884. Acesso em: 23 abr. 2026.
APA
Fukumasu, H., Avanzo, J. L., Sanches, D. S., Mennecier, G., Mori, C. M. C., & Dagli, M. L. Z. (2013). Higher susceptibility of spontaneous and NNK-induced lung neoplasms in connexin 43 deficient CD1 x AJ F1 mice: paradoxical expression of connexin 43 during lung carcinogenesis. Molecular Carcinogenesis, 52( 7), 497-506. doi:10.1002/mc.21884
NLM
Fukumasu H, Avanzo JL, Sanches DS, Mennecier G, Mori CMC, Dagli MLZ. Higher susceptibility of spontaneous and NNK-induced lung neoplasms in connexin 43 deficient CD1 x AJ F1 mice: paradoxical expression of connexin 43 during lung carcinogenesis [Internet]. Molecular Carcinogenesis. 2013 ; 52( 7): 497-506.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.21884
Vancouver
Fukumasu H, Avanzo JL, Sanches DS, Mennecier G, Mori CMC, Dagli MLZ. Higher susceptibility of spontaneous and NNK-induced lung neoplasms in connexin 43 deficient CD1 x AJ F1 mice: paradoxical expression of connexin 43 during lung carcinogenesis [Internet]. Molecular Carcinogenesis. 2013 ; 52( 7): 497-506.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.21884
Artigo de periodico
Identification of protein-coding and intronic noncoding RNAs down-regulated in clear cell renal carcinoma (2008)
Brito, Glauber Costa; Fachel, Angela Aguirres; Vettore, André Luiz; Vignal, Giselle Maria; Gimba, Etel Rodrigues Pereira; Campos, Franz Santos de; Barcinski, Marcello André; Verjovski-Almeida, Sergio ; Reis, Eduardo Moraes
Source: Molecular Carcinogenesis. Unidade: IQ
Subjects: BIOQUÍMICA, EXPRESSÃO GÊNICA, RNA
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BRITO, Glauber Costa et al. Identification of protein-coding and intronic noncoding RNAs down-regulated in clear cell renal carcinoma. Molecular Carcinogenesis, v. 47, n. 10, p. 757-767, 2008Tradução . . Disponível em: https://doi.org/10.1002/mc.20433. Acesso em: 23 abr. 2026.
APA
Brito, G. C., Fachel, A. A., Vettore, A. L., Vignal, G. M., Gimba, E. R. P., Campos, F. S. de, et al. (2008). Identification of protein-coding and intronic noncoding RNAs down-regulated in clear cell renal carcinoma. Molecular Carcinogenesis, 47( 10), 757-767. doi:10.1002/mc.20433
NLM
Brito GC, Fachel AA, Vettore AL, Vignal GM, Gimba ERP, Campos FS de, Barcinski MA, Verjovski-Almeida S, Reis EM. Identification of protein-coding and intronic noncoding RNAs down-regulated in clear cell renal carcinoma [Internet]. Molecular Carcinogenesis. 2008 ; 47( 10): 757-767.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.20433
Vancouver
Brito GC, Fachel AA, Vettore AL, Vignal GM, Gimba ERP, Campos FS de, Barcinski MA, Verjovski-Almeida S, Reis EM. Identification of protein-coding and intronic noncoding RNAs down-regulated in clear cell renal carcinoma [Internet]. Molecular Carcinogenesis. 2008 ; 47( 10): 757-767.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.20433
Artigo de periodico
CpG hypermethylation of promoter region and inactivation of E-cadherin gene in human bladder cancer (2002)
Ribeiro Filho, Leopoldo Alves; Franks, Joseph; Sasaki, Masahiro; Shiina, Hiroaki; Li, Long Cheng; Nojima, Dana; Arap, Sami; Carroll, Peter; Enokida, Hideki; Nakagawa, Masayuki; Yonezawa, Suguru; Dahiya, Rajvir
Source: Molecular Carcinogenesis. Unidade: FM
Subjects: NEOPLASIAS DA BEXIGA (IMUNOLOGIA), GENES, REAÇÃO EM CADEIA POR POLIMERASE (MÉTODOS)
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RIBEIRO FILHO, Leopoldo Alves et al. CpG hypermethylation of promoter region and inactivation of E-cadherin gene in human bladder cancer. Molecular Carcinogenesis, v. 34, p. 187-198, 2002Tradução . . Disponível em: https://doi.org/10.1002/mc.10064. Acesso em: 23 abr. 2026.
APA
Ribeiro Filho, L. A., Franks, J., Sasaki, M., Shiina, H., Li, L. C., Nojima, D., et al. (2002). CpG hypermethylation of promoter region and inactivation of E-cadherin gene in human bladder cancer. Molecular Carcinogenesis, 34, 187-198. doi:10.1002/mc.10064
NLM
Ribeiro Filho LA, Franks J, Sasaki M, Shiina H, Li LC, Nojima D, Arap S, Carroll P, Enokida H, Nakagawa M, Yonezawa S, Dahiya R. CpG hypermethylation of promoter region and inactivation of E-cadherin gene in human bladder cancer [Internet]. Molecular Carcinogenesis. 2002 ; 34 187-198.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.10064
Vancouver
Ribeiro Filho LA, Franks J, Sasaki M, Shiina H, Li LC, Nojima D, Arap S, Carroll P, Enokida H, Nakagawa M, Yonezawa S, Dahiya R. CpG hypermethylation of promoter region and inactivation of E-cadherin gene in human bladder cancer [Internet]. Molecular Carcinogenesis. 2002 ; 34 187-198.[citado 2026 abr. 23 ] Available from: https://doi.org/10.1002/mc.10064

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