Filtros : "Journal of Physiology" Removidos: "Canadá" "Moreira, Thiago dos Santos" Limpar

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  • Source: Journal of Physiology. Unidades: FMRP, ICB

    Subjects: FISIOLOGIA, FISIOLOGIA RESPIRATÓRIA E CIRCULATÓRIA, FISIOLOGIA CARDIOVASCULAR, TRONCO CEREBRAL, ANÓXIA, HIPERTENSÃO, CORAÇÃO, PULMÃO, CAMUNDONGOS, RATOS

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      PATON, Julian F. R. et al. Advancing respiratory–cardiovascular physiology with the working heart–brainstem preparation over 25 years. Journal of Physiology, v. 600, n. 9, p. 2049-2075, 2022Tradução . . Disponível em: https://doi.org/10.1113/JP281953. Acesso em: 30 set. 2024.
    • APA

      Paton, J. F. R., Machado, B. H., Moraes, D. J. de A., Zoccal, D. B., Sheikh, A. P. A., Smith, J. C., et al. (2022). Advancing respiratory–cardiovascular physiology with the working heart–brainstem preparation over 25 years. Journal of Physiology, 600( 9), 2049-2075. doi:10.1113/JP281953
    • NLM

      Paton JFR, Machado BH, Moraes DJ de A, Zoccal DB, Sheikh APA, Smith JC, Antunes VR, Murphy D, Dutschmann M. Advancing respiratory–cardiovascular physiology with the working heart–brainstem preparation over 25 years [Internet]. Journal of Physiology. 2022 ; 600( 9): 2049-2075.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP281953
    • Vancouver

      Paton JFR, Machado BH, Moraes DJ de A, Zoccal DB, Sheikh APA, Smith JC, Antunes VR, Murphy D, Dutschmann M. Advancing respiratory–cardiovascular physiology with the working heart–brainstem preparation over 25 years [Internet]. Journal of Physiology. 2022 ; 600( 9): 2049-2075.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP281953
  • Source: Journal of Physiology. Unidade: FMRP

    Subjects: POTENCIAIS DE AÇÃO, ADENOSINA TRIFOSFATO, NERVO COCLEAR

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      STRAZZA JÚNIOR, Paulo Sergio e SIQUEIRA, Daniela Vanessa Ferreira e LEÃO, Ricardo Maurício Xavier. ATP-sensitive K+ channels control the spontaneous firing of a glycinergic interneuron in the auditory brainstem. Journal of Physiology, v. 599, n. 5, p. 1611-1630, 2021Tradução . . Disponível em: https://doi.org/10.1113/JP280233. Acesso em: 30 set. 2024.
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      Strazza Júnior, P. S., Siqueira, D. V. F., & Leão, R. M. X. (2021). ATP-sensitive K+ channels control the spontaneous firing of a glycinergic interneuron in the auditory brainstem. Journal of Physiology, 599( 5), 1611-1630. doi:10.1113/JP280233
    • NLM

      Strazza Júnior PS, Siqueira DVF, Leão RMX. ATP-sensitive K+ channels control the spontaneous firing of a glycinergic interneuron in the auditory brainstem [Internet]. Journal of Physiology. 2021 ; 599( 5): 1611-1630.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP280233
    • Vancouver

      Strazza Júnior PS, Siqueira DVF, Leão RMX. ATP-sensitive K+ channels control the spontaneous firing of a glycinergic interneuron in the auditory brainstem [Internet]. Journal of Physiology. 2021 ; 599( 5): 1611-1630.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP280233
  • Source: Journal of Physiology. Unidade: ICB

    Subjects: FISIOLOGIA, AMÔNIA, HIPERTENSÃO, ACIDOSE, OÓCITOS, WESTERN BLOTTING, IMUNOFLUORESCÊNCIA EM ANIMAL, RATOS, ELETRÓLITOS

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      WANG, Jin-Lin et al. Multiple acid‐base and electrolyte disturbances upregulate NBCn1, NBCn2, IRBIT and L‐IRBIT in the mTAL. Journal of Physiology, v. 598, n. 16, p. 3395-3415, 2020Tradução . . Disponível em: https://doi.org/10.1113/JP279009. Acesso em: 30 set. 2024.
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      Wang, J. -L., Wang, X. ‐Y., Wang, D. ‐K., Parker, M. D., Musa-Aziz, R., Popple, J., et al. (2020). Multiple acid‐base and electrolyte disturbances upregulate NBCn1, NBCn2, IRBIT and L‐IRBIT in the mTAL. Journal of Physiology, 598( 16), 3395-3415. doi:10.1113/JP279009
    • NLM

      Wang J-L, Wang X‐Y, Wang D‐K, Parker MD, Musa-Aziz R, Popple J, Guo Y‐M, Min T‐X, Xia T, Tan M, Liu Y, Boron WF, Chen L‐M. Multiple acid‐base and electrolyte disturbances upregulate NBCn1, NBCn2, IRBIT and L‐IRBIT in the mTAL [Internet]. Journal of Physiology. 2020 ; 598( 16): 3395-3415.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP279009
    • Vancouver

      Wang J-L, Wang X‐Y, Wang D‐K, Parker MD, Musa-Aziz R, Popple J, Guo Y‐M, Min T‐X, Xia T, Tan M, Liu Y, Boron WF, Chen L‐M. Multiple acid‐base and electrolyte disturbances upregulate NBCn1, NBCn2, IRBIT and L‐IRBIT in the mTAL [Internet]. Journal of Physiology. 2020 ; 598( 16): 3395-3415.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP279009
  • Source: Journal of Physiology. Unidade: IFSC

    Subjects: PROTEÍNAS, CRISTALOGRAFIA, PLANEJAMENTO DE FÁRMACOS

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      GUDLUR, Aparna et al. STIM calcium sensing and conformational change. Journal of Physiology, v. 598, n. 9, p. 1695-1705, 2020Tradução . . Disponível em: https://doi.org/10.1113/JP276524. Acesso em: 30 set. 2024.
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      Gudlur, A., Zeraik, A. E., Hirve, N., & Hogan, P. G. (2020). STIM calcium sensing and conformational change. Journal of Physiology, 598( 9), 1695-1705. doi:10.1113/JP276524
    • NLM

      Gudlur A, Zeraik AE, Hirve N, Hogan PG. STIM calcium sensing and conformational change [Internet]. Journal of Physiology. 2020 ; 598( 9): 1695-1705.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP276524
    • Vancouver

      Gudlur A, Zeraik AE, Hirve N, Hogan PG. STIM calcium sensing and conformational change [Internet]. Journal of Physiology. 2020 ; 598( 9): 1695-1705.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP276524
  • Source: Journal of Physiology. Unidades: FMRP, ICMC

    Subjects: BARORREFLEXO, PRESSÃO SANGUÍNEA, INSUFICIÊNCIA CARDÍACA, SISTEMA NERVOSO SIMPÁTICO, RIM

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      LATARO, Renata Maria et al. Baroreflex control of renal sympathetic nerve activity in early heart failure assessed by the sequence method. Journal of Physiology, v. 595, n. 11, p. Se 2017, 2017Tradução . . Disponível em: https://doi.org/10.1113/JP274065. Acesso em: 30 set. 2024.
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      Lataro, R. M., Silva, L. E. V. da, Silva, C. A. A., Salgado, H. C., & Fazan Júnior, R. (2017). Baroreflex control of renal sympathetic nerve activity in early heart failure assessed by the sequence method. Journal of Physiology, 595( 11), Se 2017. doi:10.1113/JP274065
    • NLM

      Lataro RM, Silva LEV da, Silva CAA, Salgado HC, Fazan Júnior R. Baroreflex control of renal sympathetic nerve activity in early heart failure assessed by the sequence method [Internet]. Journal of Physiology. 2017 ; 595( 11): Se 2017.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP274065
    • Vancouver

      Lataro RM, Silva LEV da, Silva CAA, Salgado HC, Fazan Júnior R. Baroreflex control of renal sympathetic nerve activity in early heart failure assessed by the sequence method [Internet]. Journal of Physiology. 2017 ; 595( 11): Se 2017.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP274065
  • Source: Journal of Physiology. Unidade: FMRP

    Subjects: NERVO HIPOGLOSSO, NERVOS LARÍNGEOS, NEURÔNIOS, NERVO FRÊNICO, RESPIRAÇÃO, MEDULA ESPINHAL

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      BRITTO, Alan A. de e MORAES, Davi José de Almeida. Non‐chemosensitive parafacial neurons simultaneously regulate active expiration and airway patency under hypercapnia in rats. Journal of Physiology, v. 595, n. 6, p. 2043-2064, 2017Tradução . . Disponível em: https://doi.org/10.1113/JP273335. Acesso em: 30 set. 2024.
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      Britto, A. A. de, & Moraes, D. J. de A. (2017). Non‐chemosensitive parafacial neurons simultaneously regulate active expiration and airway patency under hypercapnia in rats. Journal of Physiology, 595( 6), 2043-2064. doi:10.1113/JP273335
    • NLM

      Britto AA de, Moraes DJ de A. Non‐chemosensitive parafacial neurons simultaneously regulate active expiration and airway patency under hypercapnia in rats [Internet]. Journal of Physiology. 2017 ; 595( 6): 2043-2064.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP273335
    • Vancouver

      Britto AA de, Moraes DJ de A. Non‐chemosensitive parafacial neurons simultaneously regulate active expiration and airway patency under hypercapnia in rats [Internet]. Journal of Physiology. 2017 ; 595( 6): 2043-2064.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP273335
  • Source: Journal of Physiology. Unidade: ICMC

    Subjects: PROCESSAMENTO DE SINAIS BIOMÉDICOS, SISTEMA NERVOSO SIMPÁTICO, BARORREFLEXO

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      SILVA, Luiz Eduardo Virgilio da e KATAYAMA, Pedro Lourenço. Baroreflex-mediated sympathetic overactivation induced by mental stress in post-traumatic stress disorder depends on the type of stressor [Carta]. Journal of Physiology. London: Instituto de Ciências Matemáticas e de Computação, Universidade de São Paulo. Disponível em: https://doi.org/10.1113/JP274859. Acesso em: 30 set. 2024. , 2017
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      Silva, L. E. V. da, & Katayama, P. L. (2017). Baroreflex-mediated sympathetic overactivation induced by mental stress in post-traumatic stress disorder depends on the type of stressor [Carta]. Journal of Physiology. London: Instituto de Ciências Matemáticas e de Computação, Universidade de São Paulo. doi:10.1113/JP274859
    • NLM

      Silva LEV da, Katayama PL. Baroreflex-mediated sympathetic overactivation induced by mental stress in post-traumatic stress disorder depends on the type of stressor [Carta] [Internet]. Journal of Physiology. 2017 ; 595( 17): Se 2017.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP274859
    • Vancouver

      Silva LEV da, Katayama PL. Baroreflex-mediated sympathetic overactivation induced by mental stress in post-traumatic stress disorder depends on the type of stressor [Carta] [Internet]. Journal of Physiology. 2017 ; 595( 17): Se 2017.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP274859
  • Source: Journal of Physiology. Unidades: ICB, EEFE

    Subjects: INSUFICIÊNCIA CARDÍACA, SISTEMA NERVOSO PARASSIMPÁTICO, SISTEMA NERVOSO AUTÔNOMO, TREINAMENTO FÍSICO, FISIOLOGIA

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      ICHIGE, Marcelo Hiro Akiyoshi et al. Exercise training preserves vagal preganglionic neurones and restores parasympathetic tonus in heart failure. Journal of Physiology, v. no 2016, n. 21, p. 6241-6254, 2016Tradução . . Disponível em: https://doi.org/10.1113/JP272730. Acesso em: 30 set. 2024.
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      Ichige, M. H. A., Santos, C. R. dos, Jordão, C. P., Ceroni, A., Negrão, C. E., & Michelini, L. C. (2016). Exercise training preserves vagal preganglionic neurones and restores parasympathetic tonus in heart failure. Journal of Physiology, no 2016( 21), 6241-6254. doi:10.1113/JP272730
    • NLM

      Ichige MHA, Santos CR dos, Jordão CP, Ceroni A, Negrão CE, Michelini LC. Exercise training preserves vagal preganglionic neurones and restores parasympathetic tonus in heart failure [Internet]. Journal of Physiology. 2016 ; no 2016( 21): 6241-6254.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP272730
    • Vancouver

      Ichige MHA, Santos CR dos, Jordão CP, Ceroni A, Negrão CE, Michelini LC. Exercise training preserves vagal preganglionic neurones and restores parasympathetic tonus in heart failure [Internet]. Journal of Physiology. 2016 ; no 2016( 21): 6241-6254.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/JP272730
  • Source: Journal of Physiology. Unidade: IFSC

    Subjects: PEIXES ELÉTRICOS, DESCARGA ELÉTRICA

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      GUARIENTO, Rafael T. et al. Automated pulse discrimination of two freely-swimming weakly electric fish and analysis of their electrical behavior during dominance contest. Journal of Physiology, v. 110, n. 3, p. 216-223, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.jphysparis.2017.02.001. Acesso em: 30 set. 2024.
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      Guariento, R. T., Mosqueiro, T. S., Matias, P., Cesarino, V. B., Almeida, L. O. B. de, Slaets, J. F. W., et al. (2016). Automated pulse discrimination of two freely-swimming weakly electric fish and analysis of their electrical behavior during dominance contest. Journal of Physiology, 110( 3), 216-223. doi:10.1016/j.jphysparis.2017.02.001
    • NLM

      Guariento RT, Mosqueiro TS, Matias P, Cesarino VB, Almeida LOB de, Slaets JFW, Maia LP, Pinto RD. Automated pulse discrimination of two freely-swimming weakly electric fish and analysis of their electrical behavior during dominance contest [Internet]. Journal of Physiology. 2016 ; 110( 3): 216-223.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.jphysparis.2017.02.001
    • Vancouver

      Guariento RT, Mosqueiro TS, Matias P, Cesarino VB, Almeida LOB de, Slaets JFW, Maia LP, Pinto RD. Automated pulse discrimination of two freely-swimming weakly electric fish and analysis of their electrical behavior during dominance contest [Internet]. Journal of Physiology. 2016 ; 110( 3): 216-223.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.jphysparis.2017.02.001
  • Source: Journal of Physiology. Unidade: FMRP

    Subjects: ANÓXIA (FISIOPATOLOGIA), PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA, RESPIRAÇÃO (FISIOLOGIA)

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      MORAES, Davi J. A. et al. Short-term sustained hypoxia induces changes in the coupling of sympathetic and respiratory activities in rats. Journal of Physiology, v. 592, n. 9, p. 2013-2033, 2014Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2013.262212. Acesso em: 30 set. 2024.
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      Moraes, D. J. A., Bonagamba, L. G. H., Costa, K. M., Silva, J. H. da C., Zoccal, D. B., & Machado, B. H. (2014). Short-term sustained hypoxia induces changes in the coupling of sympathetic and respiratory activities in rats. Journal of Physiology, 592( 9), 2013-2033. doi:10.1113/jphysiol.2013.262212
    • NLM

      Moraes DJA, Bonagamba LGH, Costa KM, Silva JH da C, Zoccal DB, Machado BH. Short-term sustained hypoxia induces changes in the coupling of sympathetic and respiratory activities in rats [Internet]. Journal of Physiology. 2014 ; 592( 9): 2013-2033.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2013.262212
    • Vancouver

      Moraes DJA, Bonagamba LGH, Costa KM, Silva JH da C, Zoccal DB, Machado BH. Short-term sustained hypoxia induces changes in the coupling of sympathetic and respiratory activities in rats [Internet]. Journal of Physiology. 2014 ; 592( 9): 2013-2033.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2013.262212
  • Source: Journal of Physiology. Unidade: ICB

    Subjects: IMUNOLOGIA, HIPOTERMIA, METABOLISMO, MODELOS ANIMAIS

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      CORRIGAN, Joshua J. et al. Hypometabolism and hypothermia in the rat model of endotoxic shock: independence of circulatory hypoxia. Journal of Physiology, v. 592, p. 3901-3916, 2014Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2014.277277. Acesso em: 30 set. 2024.
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      Corrigan, J. J., Fonseca, M. T. C., Flatow, E. A., Lewis, K., & Steiner, A. A. (2014). Hypometabolism and hypothermia in the rat model of endotoxic shock: independence of circulatory hypoxia. Journal of Physiology, 592, 3901-3916. doi:10.1113/jphysiol.2014.277277
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      Corrigan JJ, Fonseca MTC, Flatow EA, Lewis K, Steiner AA. Hypometabolism and hypothermia in the rat model of endotoxic shock: independence of circulatory hypoxia [Internet]. Journal of Physiology. 2014 ; 592 3901-3916.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2014.277277
    • Vancouver

      Corrigan JJ, Fonseca MTC, Flatow EA, Lewis K, Steiner AA. Hypometabolism and hypothermia in the rat model of endotoxic shock: independence of circulatory hypoxia [Internet]. Journal of Physiology. 2014 ; 592 3901-3916.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2014.277277
  • Source: Journal of Physiology. Unidades: ICB, FMRP

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      CAMPOREZ, João Paulo Gabriel et al. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, v. 589, n. 10, p. 2585-2596, 2011Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2011.206078. Acesso em: 30 set. 2024.
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      Camporez, J. P. G., Akamine, E. H., Davel, A. P. C., Franci, C. R., Rossoni, L. V., & Carvalho, C. R. de O. (2011). Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, 589( 10), 2585-2596. doi:10.1113/jphysiol.2011.206078
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      Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
    • Vancouver

      Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
  • Source: Journal of Physiology. Unidade: EEFE

    Subjects: TREINAMENTO FÍSICO, INSUFICIÊNCIA CARDÍACA

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      OLIVEIRA, R. S. F. et al. Cardiac anti-remodelling effect of aerobic training is associated with a reduction in the calcineurin/NFAT signalling pathway in heart failure mice. Journal of Physiology, v. 587, n. 15, p. 3899-3910, 2009Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2009.173948. Acesso em: 30 set. 2024.
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      Oliveira, R. S. F., Ferreira, J. C. B., Gomes, E. R. M., Paixão, N. A., Rolim, N. P. L., Medeiros, A., et al. (2009). Cardiac anti-remodelling effect of aerobic training is associated with a reduction in the calcineurin/NFAT signalling pathway in heart failure mice. Journal of Physiology, 587( 15), 3899-3910. doi:10.1113/jphysiol.2009.173948
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      Oliveira RSF, Ferreira JCB, Gomes ERM, Paixão NA, Rolim NPL, Medeiros A, Guatimosim S, Brum PC. Cardiac anti-remodelling effect of aerobic training is associated with a reduction in the calcineurin/NFAT signalling pathway in heart failure mice [Internet]. Journal of Physiology. 2009 ; 587( 15): 3899-3910.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2009.173948
    • Vancouver

      Oliveira RSF, Ferreira JCB, Gomes ERM, Paixão NA, Rolim NPL, Medeiros A, Guatimosim S, Brum PC. Cardiac anti-remodelling effect of aerobic training is associated with a reduction in the calcineurin/NFAT signalling pathway in heart failure mice [Internet]. Journal of Physiology. 2009 ; 587( 15): 3899-3910.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2009.173948
  • Source: Journal of Physiology. Unidade: FMRP

    Subjects: PRESSÃO SANGUÍNEA, FISIOLOGIA

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      ZOCCAL, Daniel B. et al. Increased sympathetic outflow in juvenile rats submitted to chronic intermittent hypoxia correlates with enhanced expiratory activity. Journal of Physiology, v. 586, n. 13, p. 3253-3265, 2008Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2008.154187. Acesso em: 30 set. 2024.
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      Zoccal, D. B., Simms, A. E., Bonagamba, L. G. H., Braga, V. A., Pickering, A. E., Paton, J. F. R., & Machado, B. H. (2008). Increased sympathetic outflow in juvenile rats submitted to chronic intermittent hypoxia correlates with enhanced expiratory activity. Journal of Physiology, 586( 13), 3253-3265. doi:10.1113/jphysiol.2008.154187
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      Zoccal DB, Simms AE, Bonagamba LGH, Braga VA, Pickering AE, Paton JFR, Machado BH. Increased sympathetic outflow in juvenile rats submitted to chronic intermittent hypoxia correlates with enhanced expiratory activity [Internet]. Journal of Physiology. 2008 ; 586( 13): 3253-3265.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2008.154187
    • Vancouver

      Zoccal DB, Simms AE, Bonagamba LGH, Braga VA, Pickering AE, Paton JFR, Machado BH. Increased sympathetic outflow in juvenile rats submitted to chronic intermittent hypoxia correlates with enhanced expiratory activity [Internet]. Journal of Physiology. 2008 ; 586( 13): 3253-3265.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2008.154187
  • Source: Journal of Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      ELIASSON, Lena et al. Novel aspects of the molecular mechanisms controlling insulin secretion. Journal of Physiology, v. 586, n. 14, p. 3313-3324, 2008Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2008.155317. Acesso em: 30 set. 2024.
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      Eliasson, L., Abdulkader, F. R. de M., Braun, M., Galvanovskis, J., Hoppa, M. B., & Rorsman, P. (2008). Novel aspects of the molecular mechanisms controlling insulin secretion. Journal of Physiology, 586( 14), 3313-3324. doi:10.1113/jphysiol.2008.155317
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      Eliasson L, Abdulkader FR de M, Braun M, Galvanovskis J, Hoppa MB, Rorsman P. Novel aspects of the molecular mechanisms controlling insulin secretion [Internet]. Journal of Physiology. 2008 ; 586( 14): 3313-3324.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2008.155317
    • Vancouver

      Eliasson L, Abdulkader FR de M, Braun M, Galvanovskis J, Hoppa MB, Rorsman P. Novel aspects of the molecular mechanisms controlling insulin secretion [Internet]. Journal of Physiology. 2008 ; 586( 14): 3313-3324.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2008.155317
  • Source: Journal of Physiology. Unidade: FMRP

    Subjects: GLUTAMATOS, NEUROTRANSMISSORES

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      BRAGA, Valdir A. et al. Involvement of L-glutamate and ATP in the neurotransmission of the sympathoexcitatory component of the chemoreflex in the commissural nucleus tractus solitarii of awake rats and in the working heart-brainstem preparation. Journal of Physiology, v. 581, n. 3, p. 1129-1145, 2007Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2007.129031. Acesso em: 30 set. 2024.
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      Braga, V. A., Soriano, R. N., Braccialli, A. L., Paula, P. M., Bonagamba, L. G. H., Paton, J. F. R., & Machado, B. H. (2007). Involvement of L-glutamate and ATP in the neurotransmission of the sympathoexcitatory component of the chemoreflex in the commissural nucleus tractus solitarii of awake rats and in the working heart-brainstem preparation. Journal of Physiology, 581( 3), 1129-1145. doi:10.1113/jphysiol.2007.129031
    • NLM

      Braga VA, Soriano RN, Braccialli AL, Paula PM, Bonagamba LGH, Paton JFR, Machado BH. Involvement of L-glutamate and ATP in the neurotransmission of the sympathoexcitatory component of the chemoreflex in the commissural nucleus tractus solitarii of awake rats and in the working heart-brainstem preparation [Internet]. Journal of Physiology. 2007 ; 581( 3): 1129-1145.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2007.129031
    • Vancouver

      Braga VA, Soriano RN, Braccialli AL, Paula PM, Bonagamba LGH, Paton JFR, Machado BH. Involvement of L-glutamate and ATP in the neurotransmission of the sympathoexcitatory component of the chemoreflex in the commissural nucleus tractus solitarii of awake rats and in the working heart-brainstem preparation [Internet]. Journal of Physiology. 2007 ; 581( 3): 1129-1145.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2007.129031
  • Source: Journal of Physiology. Unidade: FMRP

    Subjects: FISIOLOGIA, CÉLULAS, CÁLCIO

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      COSTA, Roberta Ribeiro e VARANDA, Wamberto Antônio. Intracellular calcium changes in mice Leydig cells are dependent on calcium entry through T-type calcium channels. Journal of Physiology, v. 585, n. 2, p. 339-349, 2007Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2007.137950. Acesso em: 30 set. 2024.
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      Costa, R. R., & Varanda, W. A. (2007). Intracellular calcium changes in mice Leydig cells are dependent on calcium entry through T-type calcium channels. Journal of Physiology, 585( 2), 339-349. doi:10.1113/jphysiol.2007.137950
    • NLM

      Costa RR, Varanda WA. Intracellular calcium changes in mice Leydig cells are dependent on calcium entry through T-type calcium channels [Internet]. Journal of Physiology. 2007 ; 585( 2): 339-349.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2007.137950
    • Vancouver

      Costa RR, Varanda WA. Intracellular calcium changes in mice Leydig cells are dependent on calcium entry through T-type calcium channels [Internet]. Journal of Physiology. 2007 ; 585( 2): 339-349.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2007.137950
  • Source: Journal of Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      NEWSHOLLME, P. et al. Diabetes associated cell stress and dysfunction: role f mitochondrial and non-mitochondrial ROS production and activity. Journal of Physiology, v. 583, n. 1, p. 9-24, 2007Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2007.135871. Acesso em: 30 set. 2024.
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      Newshollme, P., Haber, E. P., Hirabara, S. M., Rebelato, E. L. O., Procópio, J., Morgan, D., et al. (2007). Diabetes associated cell stress and dysfunction: role f mitochondrial and non-mitochondrial ROS production and activity. Journal of Physiology, 583( 1), 9-24. doi:10.1113/jphysiol.2007.135871
    • NLM

      Newshollme P, Haber EP, Hirabara SM, Rebelato ELO, Procópio J, Morgan D, Oliveira-Emilio HC, Carpinelli AR, Curi R. Diabetes associated cell stress and dysfunction: role f mitochondrial and non-mitochondrial ROS production and activity [Internet]. Journal of Physiology. 2007 ; 583( 1): 9-24.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2007.135871
    • Vancouver

      Newshollme P, Haber EP, Hirabara SM, Rebelato ELO, Procópio J, Morgan D, Oliveira-Emilio HC, Carpinelli AR, Curi R. Diabetes associated cell stress and dysfunction: role f mitochondrial and non-mitochondrial ROS production and activity [Internet]. Journal of Physiology. 2007 ; 583( 1): 9-24.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2007.135871
  • Source: Journal of Physiology. Unidade: ICB

    Assunto: ANATOMIA

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      CARNEIRO-RAMOS, M. S. et al. Cardiac angiotensin II type I and type II receptors are increased in rats submitted to experimental hypothyroidism. Journal of Physiology, v. 583, n. 1, p. 213-223, 2007Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2007.134080. Acesso em: 30 set. 2024.
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      Carneiro-Ramos, M. S., Diniz, G. P., Almeida, J., Vieira, R. L. P., Pinheiro, S. V. B., Santos, R. A., & Barreto-Chaves, M. L. M. (2007). Cardiac angiotensin II type I and type II receptors are increased in rats submitted to experimental hypothyroidism. Journal of Physiology, 583( 1), 213-223. doi:10.1113/jphysiol.2007.134080
    • NLM

      Carneiro-Ramos MS, Diniz GP, Almeida J, Vieira RLP, Pinheiro SVB, Santos RA, Barreto-Chaves MLM. Cardiac angiotensin II type I and type II receptors are increased in rats submitted to experimental hypothyroidism [Internet]. Journal of Physiology. 2007 ; 583( 1): 213-223.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2007.134080
    • Vancouver

      Carneiro-Ramos MS, Diniz GP, Almeida J, Vieira RLP, Pinheiro SVB, Santos RA, Barreto-Chaves MLM. Cardiac angiotensin II type I and type II receptors are increased in rats submitted to experimental hypothyroidism [Internet]. Journal of Physiology. 2007 ; 583( 1): 213-223.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2007.134080
  • Source: Journal of Physiology. Unidade: FMRP

    Subjects: MIOSINA, SINAPSE

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    • ABNT

      TAKAGISHI, Yoshiko et al. Localization of myosin II and V isoforms in cultured rat sympathetic neurones and their potential involvement in presynaptic function. Journal of Physiology, v. 569, n. 1, p. 195-208, 2005Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2005.095943. Acesso em: 30 set. 2024.
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      Takagishi, Y., Futaki, S., Itoh, K., Espreafico, E. M., Murakami, N., Murata, Y., & Mochida, S. (2005). Localization of myosin II and V isoforms in cultured rat sympathetic neurones and their potential involvement in presynaptic function. Journal of Physiology, 569( 1), 195-208. doi:10.1113/jphysiol.2005.095943
    • NLM

      Takagishi Y, Futaki S, Itoh K, Espreafico EM, Murakami N, Murata Y, Mochida S. Localization of myosin II and V isoforms in cultured rat sympathetic neurones and their potential involvement in presynaptic function [Internet]. Journal of Physiology. 2005 ; 569( 1): 195-208.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2005.095943
    • Vancouver

      Takagishi Y, Futaki S, Itoh K, Espreafico EM, Murakami N, Murata Y, Mochida S. Localization of myosin II and V isoforms in cultured rat sympathetic neurones and their potential involvement in presynaptic function [Internet]. Journal of Physiology. 2005 ; 569( 1): 195-208.[citado 2024 set. 30 ] Available from: https://doi.org/10.1113/jphysiol.2005.095943

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