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Trabalho de evento-resumo periodico
Adult human brain slice cultures in the investigation of SARS-CoV-2 CNS infection (2021)
Almeida, Glaucia Maria ; Mendes, Niele Dias ; Santos, Marcelo Volpon ; Gondim, Guilherme Gozzoli Podolsky ; Oliveira, Ricardo Santos de ; Cunha, Thiago Mattar ; Arruda, Eurico ; Sebollela, Adriano Silva ; Neder, Luciano
Source: Journal of Neuropathology and Experimental Neurology. Conference titles: Annual Meeting of the American Association of Neuropathologists. Unidade: FMRP
Subjects: COVID-19, RECEPTORES CELULARES, DOENÇAS DO SISTEMA NERVOSO CENTRAL, BIOMARCADORES, EXPRESSÃO GÊNICA, CITOCININAS
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ABNT
ALMEIDA, Glaucia Maria et al. Adult human brain slice cultures in the investigation of SARS-CoV-2 CNS infection. Journal of Neuropathology and Experimental Neurology. Cary: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1093/jnen/nlab042. Acesso em: 04 nov. 2024. , 2021
APA
Almeida, G. M., Mendes, N. D., Santos, M. V., Gondim, G. G. P., Oliveira, R. S. de, Cunha, T. M., et al. (2021). Adult human brain slice cultures in the investigation of SARS-CoV-2 CNS infection. Journal of Neuropathology and Experimental Neurology. Cary: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.1093/jnen/nlab042
NLM
Almeida GM, Mendes ND, Santos MV, Gondim GGP, Oliveira RS de, Cunha TM, Arruda E, Sebollela AS, Neder L. Adult human brain slice cultures in the investigation of SARS-CoV-2 CNS infection [Internet]. Journal of Neuropathology and Experimental Neurology. 2021 ; 80( 6): 586-587.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1093/jnen/nlab042
Vancouver
Almeida GM, Mendes ND, Santos MV, Gondim GGP, Oliveira RS de, Cunha TM, Arruda E, Sebollela AS, Neder L. Adult human brain slice cultures in the investigation of SARS-CoV-2 CNS infection [Internet]. Journal of Neuropathology and Experimental Neurology. 2021 ; 80( 6): 586-587.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1093/jnen/nlab042
Artigo de periodico
Lack of KBTBD4 mutations in molecularly classified brazilian medulloblastomas (2019)
Leal, Letícia Ferro ; Cavagna, Rodrigo de Oliveira; Campanella, Nathalia Cristina; Mançano, Bruna; Almeida, Gisele Caravina; Matsushita, Marcus; Almeida Junior, Carlos Roberto; Saggioro, Fabiano Pinto ; Stavale, João Norberto; Malheiros, Suzana M. F.; Lima, Matheus; Hajj, Glaucia N. M.; Neder, Luciano ; Reis, Rui Manuel
Source: Journal of Neuropathology and Experimental Neurology. Unidades: SVOI, FMRP
Subjects: NEOPLASIAS CEREBRAIS, MEDULOBLASTOMA, MUTAÇÃO
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ABNT
LEAL, Letícia Ferro et al. Lack of KBTBD4 mutations in molecularly classified brazilian medulloblastomas. Journal of Neuropathology and Experimental Neurology, v. 78, n. 9, p. 788-790, 2019Tradução . . Disponível em: https://doi.org/10.1093/jnen/nlz066. Acesso em: 04 nov. 2024.
APA
Leal, L. F., Cavagna, R. de O., Campanella, N. C., Mançano, B., Almeida, G. C., Matsushita, M., et al. (2019). Lack of KBTBD4 mutations in molecularly classified brazilian medulloblastomas. Journal of Neuropathology and Experimental Neurology, 78( 9), 788-790. doi:10.1093/jnen/nlz066
NLM
Leal LF, Cavagna R de O, Campanella NC, Mançano B, Almeida GC, Matsushita M, Almeida Junior CR, Saggioro FP, Stavale JN, Malheiros SMF, Lima M, Hajj GNM, Neder L, Reis RM. Lack of KBTBD4 mutations in molecularly classified brazilian medulloblastomas [Internet]. Journal of Neuropathology and Experimental Neurology. 2019 ; 78( 9): 788-790.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1093/jnen/nlz066
Vancouver
Leal LF, Cavagna R de O, Campanella NC, Mançano B, Almeida GC, Matsushita M, Almeida Junior CR, Saggioro FP, Stavale JN, Malheiros SMF, Lima M, Hajj GNM, Neder L, Reis RM. Lack of KBTBD4 mutations in molecularly classified brazilian medulloblastomas [Internet]. Journal of Neuropathology and Experimental Neurology. 2019 ; 78( 9): 788-790.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1093/jnen/nlz066
Artigo de periodico
IAA1549:BRAF gene fusion and FGFR1 hotspot mutations are prognostic factors in pilocytic astrocytomas (2015)
Becker, Aline Paixão; Scapulatempo-Neto, Cristovam; Carloni, Adriana C; Paulino, Alessandra; Sheren, Jamie; Aisner, Dara L; Musselwhite, Evelyn; Clara, Carlos; Machado, Hélio Rubens; Oliveira, Ricardo S; Neder, Luciano; Varella-Garcia, Marileila; Reis, Rui M
Source: Journal of Neuropathology and Experimental Neurology. Unidade: FMRP
Subjects: ASTROCITOMA (GENÉTICA;METABOLISMO), NEOPLASIAS CEREBRAIS (DIAGNÓSTICO), PROTEÍNAS PROTO-ONCOGÊNICAS, MUTAÇÃO GENÉTICA
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ABNT
BECKER, Aline Paixão et al. IAA1549:BRAF gene fusion and FGFR1 hotspot mutations are prognostic factors in pilocytic astrocytomas. Journal of Neuropathology and Experimental Neurology, v. 74, n. 7, p. 743-754, 2015Tradução . . Disponível em: https://doi.org/10.1097/NEN.0000000000000213. Acesso em: 04 nov. 2024.
APA
Becker, A. P., Scapulatempo-Neto, C., Carloni, A. C., Paulino, A., Sheren, J., Aisner, D. L., et al. (2015). IAA1549:BRAF gene fusion and FGFR1 hotspot mutations are prognostic factors in pilocytic astrocytomas. Journal of Neuropathology and Experimental Neurology, 74( 7), 743-754. doi:10.1097/NEN.0000000000000213
NLM
Becker AP, Scapulatempo-Neto C, Carloni AC, Paulino A, Sheren J, Aisner DL, Musselwhite E, Clara C, Machado HR, Oliveira RS, Neder L, Varella-Garcia M, Reis RM. IAA1549:BRAF gene fusion and FGFR1 hotspot mutations are prognostic factors in pilocytic astrocytomas [Internet]. Journal of Neuropathology and Experimental Neurology. 2015 ; 74( 7): 743-754.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1097/NEN.0000000000000213
Vancouver
Becker AP, Scapulatempo-Neto C, Carloni AC, Paulino A, Sheren J, Aisner DL, Musselwhite E, Clara C, Machado HR, Oliveira RS, Neder L, Varella-Garcia M, Reis RM. IAA1549:BRAF gene fusion and FGFR1 hotspot mutations are prognostic factors in pilocytic astrocytomas [Internet]. Journal of Neuropathology and Experimental Neurology. 2015 ; 74( 7): 743-754.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1097/NEN.0000000000000213
Trabalho de evento-resumo periodico
Argyrophilic grain disease may delay cognitive decline in AD: an autopsy study (2015)
Grinberg, Lea; Rodriguez, Roberta; Suemoto, Claudia; Molina, Mariana; Nascimento, Camila; Leite, Renata; Ferretti-Rebustini, Renata Eloah de Lucena ; Farfel, Jose; Heinsen, Helmut; Nitrini, Ricardo; Pasquallucci, Carlos; Jacob-Filho, Wilson; Yaffe, Kristine
Source: Journal of Neuropathology and Experimental Neurology. Conference titles: Annual Meeting of the American Association of Neuropathologists. Unidade: FM
Subjects: AUTÓPSIA (ESTUDO), COGNIÇÃO (DEFICIÊNCIA), DOENÇAS NEURODEGENERATIVAS, ENVELHECIMENTO
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ABNT
GRINBERG, Lea et al. Argyrophilic grain disease may delay cognitive decline in AD: an autopsy study. Journal of Neuropathology and Experimental Neurology. Baltimore: Faculdade de Medicina, Universidade de São Paulo. Disponível em: file:///C:/Users/DBDFMUSP/Downloads/American_Association_of_Neuropathologists,_Inc_.12.pdf. Acesso em: 04 nov. 2024. , 2015
APA
Grinberg, L., Rodriguez, R., Suemoto, C., Molina, M., Nascimento, C., Leite, R., et al. (2015). Argyrophilic grain disease may delay cognitive decline in AD: an autopsy study. Journal of Neuropathology and Experimental Neurology. Baltimore: Faculdade de Medicina, Universidade de São Paulo. Recuperado de file:///C:/Users/DBDFMUSP/Downloads/American_Association_of_Neuropathologists,_Inc_.12.pdf
NLM
Grinberg L, Rodriguez R, Suemoto C, Molina M, Nascimento C, Leite R, Ferretti-Rebustini RE de L, Farfel J, Heinsen H, Nitrini R, Pasquallucci C, Jacob-Filho W, Yaffe K. Argyrophilic grain disease may delay cognitive decline in AD: an autopsy study [Internet]. Journal of Neuropathology and Experimental Neurology. 2015 ; 74( 6): 618-619.[citado 2024 nov. 04 ] Available from: file:///C:/Users/DBDFMUSP/Downloads/American_Association_of_Neuropathologists,_Inc_.12.pdf
Vancouver
Grinberg L, Rodriguez R, Suemoto C, Molina M, Nascimento C, Leite R, Ferretti-Rebustini RE de L, Farfel J, Heinsen H, Nitrini R, Pasquallucci C, Jacob-Filho W, Yaffe K. Argyrophilic grain disease may delay cognitive decline in AD: an autopsy study [Internet]. Journal of Neuropathology and Experimental Neurology. 2015 ; 74( 6): 618-619.[citado 2024 nov. 04 ] Available from: file:///C:/Users/DBDFMUSP/Downloads/American_Association_of_Neuropathologists,_Inc_.12.pdf
Trabalho de evento-resumo periodico
A rare form of sporadic Jakob-Creutzfeldt disease presenting as PSP: A Public Health Issue (2015)
Grinberg, Lea; Fernandez-Fournier, Mireya; Perry, David; Tartaglia, Maria; DeMay, Mary; Boxer, Adam; Coppola, Giovanni; Christine, Chadwick; Gambetti, Perluigi; Cali, Ignazio; Seeley, William; Miller, Bruce; deArmond, Steven; Geschwind, Michael
Source: Journal of Neuropathology and Experimental Neurology. Conference titles: Annual Meeting of the American Association of Neuropathologists. Unidade: FM
Subjects: DEMÊNCIA, TRANSTORNOS DA MOTILIDADE OCULAR, DOENÇAS CEREBRAIS
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ABNT
GRINBERG, Lea et al. A rare form of sporadic Jakob-Creutzfeldt disease presenting as PSP: A Public Health Issue. Journal of Neuropathology and Experimental Neurology. Baltimore: Faculdade de Medicina, Universidade de São Paulo. Disponível em: file:///C:/Users/DBDFMUSP/Downloads/American_Association_of_Neuropathologists,_Inc_.12.pdf. Acesso em: 04 nov. 2024. , 2015
APA
Grinberg, L., Fernandez-Fournier, M., Perry, D., Tartaglia, M., DeMay, M., Boxer, A., et al. (2015). A rare form of sporadic Jakob-Creutzfeldt disease presenting as PSP: A Public Health Issue. Journal of Neuropathology and Experimental Neurology. Baltimore: Faculdade de Medicina, Universidade de São Paulo. Recuperado de file:///C:/Users/DBDFMUSP/Downloads/American_Association_of_Neuropathologists,_Inc_.12.pdf
NLM
Grinberg L, Fernandez-Fournier M, Perry D, Tartaglia M, DeMay M, Boxer A, Coppola G, Christine C, Gambetti P, Cali I, Seeley W, Miller B, deArmond S, Geschwind M. A rare form of sporadic Jakob-Creutzfeldt disease presenting as PSP: A Public Health Issue [Internet]. Journal of Neuropathology and Experimental Neurology. 2015 ; 74( 6): 601.[citado 2024 nov. 04 ] Available from: file:///C:/Users/DBDFMUSP/Downloads/American_Association_of_Neuropathologists,_Inc_.12.pdf
Vancouver
Grinberg L, Fernandez-Fournier M, Perry D, Tartaglia M, DeMay M, Boxer A, Coppola G, Christine C, Gambetti P, Cali I, Seeley W, Miller B, deArmond S, Geschwind M. A rare form of sporadic Jakob-Creutzfeldt disease presenting as PSP: A Public Health Issue [Internet]. Journal of Neuropathology and Experimental Neurology. 2015 ; 74( 6): 601.[citado 2024 nov. 04 ] Available from: file:///C:/Users/DBDFMUSP/Downloads/American_Association_of_Neuropathologists,_Inc_.12.pdf
Artigo de periodico
Neurotrophins in mesial temporal lobe epilepsy with and without psychiatric comorbidities (2013)
Kandratavicius, Ludmyla; Monteiro, Mariana Raquel; Assirati Júnior, João Alberto; Carlotti Júnior, Carlos Gilberto; Hallak, Jaime Eduardo Cecílio; Leite, João Pereira
Source: Journal of Neuropathology and Experimental Neurology. Unidade: FMRP
Subjects: DEPRESSÃO, EPILEPSIA DO LOBO TEMPORAL, TRANSTORNOS PSICÓTICOS, NEUROLOGIA
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ABNT
KANDRATAVICIUS, Ludmyla et al. Neurotrophins in mesial temporal lobe epilepsy with and without psychiatric comorbidities. Journal of Neuropathology and Experimental Neurology, v. 72, n. 11, p. 1029-1042, 2013Tradução . . Disponível em: https://doi.org/10.1097/NEN.0000000000000002. Acesso em: 04 nov. 2024.
APA
Kandratavicius, L., Monteiro, M. R., Assirati Júnior, J. A., Carlotti Júnior, C. G., Hallak, J. E. C., & Leite, J. P. (2013). Neurotrophins in mesial temporal lobe epilepsy with and without psychiatric comorbidities. Journal of Neuropathology and Experimental Neurology, 72( 11), 1029-1042. doi:10.1097/NEN.0000000000000002
NLM
Kandratavicius L, Monteiro MR, Assirati Júnior JA, Carlotti Júnior CG, Hallak JEC, Leite JP. Neurotrophins in mesial temporal lobe epilepsy with and without psychiatric comorbidities [Internet]. Journal of Neuropathology and Experimental Neurology. 2013 ; 72( 11): 1029-1042.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1097/NEN.0000000000000002
Vancouver
Kandratavicius L, Monteiro MR, Assirati Júnior JA, Carlotti Júnior CG, Hallak JEC, Leite JP. Neurotrophins in mesial temporal lobe epilepsy with and without psychiatric comorbidities [Internet]. Journal of Neuropathology and Experimental Neurology. 2013 ; 72( 11): 1029-1042.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1097/NEN.0000000000000002
Artigo de periodico
Cerebral white matter oxidation and nitrosylation in young rodents with kaolin-induced hydrocephalus (2012)
Bigio, Marc R. Del; Khan, Osaama H.; Lopes, Luiza da Silva; Juliet, Packiasamy A. R.
Source: Journal of Neuropathology and Experimental Neurology. Unidade: FMRP
Subjects: CÉREBRO (LESÕES;DANOS;INVESTIGAÇÃO), ÓXIDO NÍTRICO, VENTRÍCULOS CEREBRAIS, DILATAÇÃO
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ABNT
BIGIO, Marc R. Del et al. Cerebral white matter oxidation and nitrosylation in young rodents with kaolin-induced hydrocephalus. Journal of Neuropathology and Experimental Neurology, v. 71, n. 4, p. 274-288, 2012Tradução . . Acesso em: 04 nov. 2024.
APA
Bigio, M. R. D., Khan, O. H., Lopes, L. da S., & Juliet, P. A. R. (2012). Cerebral white matter oxidation and nitrosylation in young rodents with kaolin-induced hydrocephalus. Journal of Neuropathology and Experimental Neurology, 71( 4), 274-288.
NLM
Bigio MRD, Khan OH, Lopes L da S, Juliet PAR. Cerebral white matter oxidation and nitrosylation in young rodents with kaolin-induced hydrocephalus. Journal of Neuropathology and Experimental Neurology. 2012 ; 71( 4): 274-288.[citado 2024 nov. 04 ]
Vancouver
Bigio MRD, Khan OH, Lopes L da S, Juliet PAR. Cerebral white matter oxidation and nitrosylation in young rodents with kaolin-induced hydrocephalus. Journal of Neuropathology and Experimental Neurology. 2012 ; 71( 4): 274-288.[citado 2024 nov. 04 ]

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