ReP USP - Resultado da busca

Artigo de periodico
A comprehensive review on phytochemicals for fatty liver: are they potential adjuvants? (2022)
Moinho, Taís Menezes do ; Matos, Sandro Leão; Carvalho, Carla Roberta de Oliveira
Fonte: Journal of Molecular Medicine. Unidade: ICB
Assuntos: FISIOLOGIA, FITOQUÍMICA, FÍGADO GORDUROSO, OBESIDADE, HEPATOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MOINHO, Taís Menezes do e MATOS, Sandro Leão e CARVALHO, Carla Roberta de Oliveira. A comprehensive review on phytochemicals for fatty liver: are they potential adjuvants?. Journal of Molecular Medicine, p. 1-15, 2022Tradução . . Disponível em: https://doi.org/10.1007/s00109-021-02170-3. Acesso em: 01 nov. 2024.
APA
Moinho, T. M. do, Matos, S. L., & Carvalho, C. R. de O. (2022). A comprehensive review on phytochemicals for fatty liver: are they potential adjuvants? Journal of Molecular Medicine, 1-15. doi:10.1007/s00109-021-02170-3
NLM
Moinho TM do, Matos SL, Carvalho CR de O. A comprehensive review on phytochemicals for fatty liver: are they potential adjuvants? [Internet]. Journal of Molecular Medicine. 2022 ; 1-15.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-021-02170-3
Vancouver
Moinho TM do, Matos SL, Carvalho CR de O. A comprehensive review on phytochemicals for fatty liver: are they potential adjuvants? [Internet]. Journal of Molecular Medicine. 2022 ; 1-15.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-021-02170-3
Artigo de periodico
MT1 and MT2 melatonin receptors play opposite roles in brain cancer progression (2021)
Kinker, Gabriela Sarti ; Ostrowski, Luiza Henriques; Ribeiro, Paula do Amaral Costa; Chanoch, Rony; Muxel, Sandra Márcia; Tirosh, Itay; Spadoni, Gilberto; Rivara, Silvia; Martins, Vilma Regina; Santos, Tiago Góss dos; Markus, Regina P ; Fernandes, Pedro Augusto Carlos Magno
Fonte: Journal of Molecular Medicine. Unidade: IB
Assuntos: NEOPLASIAS CEREBRAIS, MELATONINA, GLIOMA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KINKER, Gabriela Sarti et al. MT1 and MT2 melatonin receptors play opposite roles in brain cancer progression. Journal of Molecular Medicine, v. 99, p. 289–301, 2021Tradução . . Disponível em: https://doi.org/10.1007/s00109-020-02023-5. Acesso em: 01 nov. 2024.
APA
Kinker, G. S., Ostrowski, L. H., Ribeiro, P. do A. C., Chanoch, R., Muxel, S. M., Tirosh, I., et al. (2021). MT1 and MT2 melatonin receptors play opposite roles in brain cancer progression. Journal of Molecular Medicine, 99, 289–301. doi:10.1007/s00109-020-02023-5
NLM
Kinker GS, Ostrowski LH, Ribeiro P do AC, Chanoch R, Muxel SM, Tirosh I, Spadoni G, Rivara S, Martins VR, Santos TG dos, Markus RP, Fernandes PACM. MT1 and MT2 melatonin receptors play opposite roles in brain cancer progression [Internet]. Journal of Molecular Medicine. 2021 ; 99 289–301.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-020-02023-5
Vancouver
Kinker GS, Ostrowski LH, Ribeiro P do AC, Chanoch R, Muxel SM, Tirosh I, Spadoni G, Rivara S, Martins VR, Santos TG dos, Markus RP, Fernandes PACM. MT1 and MT2 melatonin receptors play opposite roles in brain cancer progression [Internet]. Journal of Molecular Medicine. 2021 ; 99 289–301.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-020-02023-5
Artigo de periodico
A coordinated approach for the assessment of molecular subgroups in pediatric ependymomas using low-cost methods (2021)
Sousa, Graziella Ribeiro de ; Lira, Régia Caroline Peixoto ; Magalhães, Taciani de Almeida ; Silva, Keteryne Rodrigues da ; Nagano, Luís Fernando Peinado ; Saggioro, Fabiano Pinto ; Baroni, Mirella; Marie, Suely Kazue Nagahashi ; Oba-Shinjo, Sueli Mieko; Brandelise, Silvia; Queiroz, Rosane Gomes de Paula; Annichini, María Sol Brassesco ; Scrideli, Carlos Alberto ; Tone, Luiz Gonzaga ; Valera, Elvis Terci
Fonte: Journal of Molecular Medicine. Unidades: FMRP, FM, FFCLRP
Assuntos: MOLÉCULA, PEDIATRIA, SISTEMA NERVOSO CENTRAL, CÉREBRO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUSA, Graziella Ribeiro de et al. A coordinated approach for the assessment of molecular subgroups in pediatric ependymomas using low-cost methods. Journal of Molecular Medicine, v. 99, n. 8, p. 1101-1113, 2021Tradução . . Disponível em: https://doi.org/10.1007/s00109-021-02074-2. Acesso em: 01 nov. 2024.
APA
Sousa, G. R. de, Lira, R. C. P., Magalhães, T. de A., Silva, K. R. da, Nagano, L. F. P., Saggioro, F. P., et al. (2021). A coordinated approach for the assessment of molecular subgroups in pediatric ependymomas using low-cost methods. Journal of Molecular Medicine, 99( 8), 1101-1113. doi:10.1007/s00109-021-02074-2
NLM
Sousa GR de, Lira RCP, Magalhães T de A, Silva KR da, Nagano LFP, Saggioro FP, Baroni M, Marie SKN, Oba-Shinjo SM, Brandelise S, Queiroz RG de P, Annichini MSB, Scrideli CA, Tone LG, Valera ET. A coordinated approach for the assessment of molecular subgroups in pediatric ependymomas using low-cost methods [Internet]. Journal of Molecular Medicine. 2021 ; 99( 8): 1101-1113.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-021-02074-2
Vancouver
Sousa GR de, Lira RCP, Magalhães T de A, Silva KR da, Nagano LFP, Saggioro FP, Baroni M, Marie SKN, Oba-Shinjo SM, Brandelise S, Queiroz RG de P, Annichini MSB, Scrideli CA, Tone LG, Valera ET. A coordinated approach for the assessment of molecular subgroups in pediatric ependymomas using low-cost methods [Internet]. Journal of Molecular Medicine. 2021 ; 99( 8): 1101-1113.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-021-02074-2
Artigo de periodico
Biophysical and pharmacological characterization of a full-length synthetic analog of the antitumor polypeptide crotamine (2020)
Porta, Lucas de Carvalho; Fadel, Valmir; Campeiro, Joana D’Arc; Oliveira, Eduardo Brandt de; Godinho, Rosely Oliveira; Hayashi, Mirian Akemi Furuie
Fonte: Journal of Molecular Medicine. Unidade: FMRP
Assuntos: BIOFÍSICA, VENENOS DE ORIGEM ANIMAL, PEPTÍDEOS, EVENTO ADVERSO A MEDICAMENTO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PORTA, Lucas de Carvalho et al. Biophysical and pharmacological characterization of a full-length synthetic analog of the antitumor polypeptide crotamine. Journal of Molecular Medicine, v. 98, n. 11, p. 1561-1571, 2020Tradução . . Disponível em: https://doi.org/10.1007/s00109-020-01975-y. Acesso em: 01 nov. 2024.
APA
Porta, L. de C., Fadel, V., Campeiro, J. D. ’A., Oliveira, E. B. de, Godinho, R. O., & Hayashi, M. A. F. (2020). Biophysical and pharmacological characterization of a full-length synthetic analog of the antitumor polypeptide crotamine. Journal of Molecular Medicine, 98( 11), 1561-1571. doi:10.1007/s00109-020-01975-y
NLM
Porta L de C, Fadel V, Campeiro JD’A, Oliveira EB de, Godinho RO, Hayashi MAF. Biophysical and pharmacological characterization of a full-length synthetic analog of the antitumor polypeptide crotamine [Internet]. Journal of Molecular Medicine. 2020 ; 98( 11): 1561-1571.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-020-01975-y
Vancouver
Porta L de C, Fadel V, Campeiro JD’A, Oliveira EB de, Godinho RO, Hayashi MAF. Biophysical and pharmacological characterization of a full-length synthetic analog of the antitumor polypeptide crotamine [Internet]. Journal of Molecular Medicine. 2020 ; 98( 11): 1561-1571.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-020-01975-y
Artigo de periodico
The therapeutic potential of Aurora kinases targeting in glioblastoma: from preclinical research to translational oncology (2020)
Magalhães, Taciani de Almeida; Sousa, Graziella Ribeiro de ; Cruzeiro, Gustavo Alencastro Veiga; Tone, Luiz Gonzaga ; Valera, Elvis Terci; Borges, Kleiton Silva
Fonte: Journal of Molecular Medicine. Unidade: FMRP
Assuntos: TUMOR CARCINOIDE, SISTEMA NERVOSO CENTRAL, GLIOMA, ASTROCITOMA, PROTEÍNAS QUINASES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MAGALHÃES, Taciani de Almeida et al. The therapeutic potential of Aurora kinases targeting in glioblastoma: from preclinical research to translational oncology. Journal of Molecular Medicine, v. 98, n. 4, p. 495-512, 2020Tradução . . Disponível em: https://doi.org/10.1007/s00109-020-01895-x. Acesso em: 01 nov. 2024.
APA
Magalhães, T. de A., Sousa, G. R. de, Cruzeiro, G. A. V., Tone, L. G., Valera, E. T., & Borges, K. S. (2020). The therapeutic potential of Aurora kinases targeting in glioblastoma: from preclinical research to translational oncology. Journal of Molecular Medicine, 98( 4), 495-512. doi:10.1007/s00109-020-01895-x
NLM
Magalhães T de A, Sousa GR de, Cruzeiro GAV, Tone LG, Valera ET, Borges KS. The therapeutic potential of Aurora kinases targeting in glioblastoma: from preclinical research to translational oncology [Internet]. Journal of Molecular Medicine. 2020 ; 98( 4): 495-512.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-020-01895-x
Vancouver
Magalhães T de A, Sousa GR de, Cruzeiro GAV, Tone LG, Valera ET, Borges KS. The therapeutic potential of Aurora kinases targeting in glioblastoma: from preclinical research to translational oncology [Internet]. Journal of Molecular Medicine. 2020 ; 98( 4): 495-512.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-020-01895-x
Artigo de periodico
S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone (2017)
Takano, Ana Paula Cremasco ; Munhoz, Carolina Demarchi ; Moriscot, Anselmo Sigari ; Gupta, Sudhiranjan; Barreto-Chaves, Maria Luiza Morais
Fonte: Journal of Molecular Medicine. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TAKANO, Ana Paula Cremasco et al. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone. Journal of Molecular Medicine, v. 95, n. 6, p. 671-682, 2017Tradução . . Disponível em: https://doi.org/10.1007/s00109-017-1511-y. Acesso em: 01 nov. 2024.
APA
Takano, A. P. C., Munhoz, C. D., Moriscot, A. S., Gupta, S., & Barreto-Chaves, M. L. M. (2017). S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone. Journal of Molecular Medicine, 95( 6), 671-682. doi:10.1007/s00109-017-1511-y
NLM
Takano APC, Munhoz CD, Moriscot AS, Gupta S, Barreto-Chaves MLM. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone [Internet]. Journal of Molecular Medicine. 2017 ; 95( 6): 671-682.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-017-1511-y
Vancouver
Takano APC, Munhoz CD, Moriscot AS, Gupta S, Barreto-Chaves MLM. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone [Internet]. Journal of Molecular Medicine. 2017 ; 95( 6): 671-682.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-017-1511-y
Artigo de periodico
New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases (2015)
Disatnik, Marie-Hélène; Hwang, Sunhee; Ferreira, Julio Cesar Batista ; Mochly-Rosen, Daria
Fonte: Journal of Molecular Medicine. Unidade: ICB
Assuntos: ANATOMIA, DOENÇAS CARDIOVASCULARES, MITOCÔNDRIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DISATNIK, Marie-Hélène et al. New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases. Journal of Molecular Medicine, v. 93, n. 3, p. 279-287, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00109-015-1256-4. Acesso em: 01 nov. 2024.
APA
Disatnik, M. -H., Hwang, S., Ferreira, J. C. B., & Mochly-Rosen, D. (2015). New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases. Journal of Molecular Medicine, 93( 3), 279-287. doi:10.1007/s00109-015-1256-4
NLM
Disatnik M-H, Hwang S, Ferreira JCB, Mochly-Rosen D. New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases [Internet]. Journal of Molecular Medicine. 2015 ; 93( 3): 279-287.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-015-1256-4
Vancouver
Disatnik M-H, Hwang S, Ferreira JCB, Mochly-Rosen D. New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases [Internet]. Journal of Molecular Medicine. 2015 ; 93( 3): 279-287.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-015-1256-4
Artigo de periodico
Intragenic motifs regulate the transcriptional complexity of Pkhd1/PKHD1 (2014)
Boddu, Ravindra; Yang, Chaozhe; O'Connor, Amber K. O.; Hendrickson, Robert Curtis; Boone, Braden; Cui, Xiagqin; Garcia-Gonzalez, Miguel; Igarashi, Peter; Onuchic, Luiz F.; Germino, Gregory G.; Guay-Woodford, Lisa M.
Fonte: Journal of Molecular Medicine. Unidade: FM
Assuntos: NEFROPATIAS, HERANÇA GENÉTICA, EXAMES MÉDICOS, RNA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BODDU, Ravindra et al. Intragenic motifs regulate the transcriptional complexity of Pkhd1/PKHD1. Journal of Molecular Medicine, v. 92, p. 1045-1056, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00109-014-1185-7. Acesso em: 01 nov. 2024.
APA
Boddu, R., Yang, C., O'Connor, A. K. O., Hendrickson, R. C., Boone, B., Cui, X., et al. (2014). Intragenic motifs regulate the transcriptional complexity of Pkhd1/PKHD1. Journal of Molecular Medicine, 92, 1045-1056. doi:10.1007/s00109-014-1185-7
NLM
Boddu R, Yang C, O'Connor AKO, Hendrickson RC, Boone B, Cui X, Garcia-Gonzalez M, Igarashi P, Onuchic LF, Germino GG, Guay-Woodford LM. Intragenic motifs regulate the transcriptional complexity of Pkhd1/PKHD1 [Internet]. Journal of Molecular Medicine. 2014 ; 92 1045-1056.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-014-1185-7
Vancouver
Boddu R, Yang C, O'Connor AKO, Hendrickson RC, Boone B, Cui X, Garcia-Gonzalez M, Igarashi P, Onuchic LF, Germino GG, Guay-Woodford LM. Intragenic motifs regulate the transcriptional complexity of Pkhd1/PKHD1 [Internet]. Journal of Molecular Medicine. 2014 ; 92 1045-1056.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-014-1185-7
Artigo de periodico
Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells (2014)
Martín-Rufián, Mercedes; Nascimento-Gomes, Renata; Higuero, Ana; Crisma, Amanda R.; Campos-Sandoval, José A.; Gómez-Garcia, María C.; Cardona, Carolina; Cheng, Tzuling; Lobo, Carolina; Segura, Juan A.; Alonso, Francisco J.; Szeliga, Monika; Albrecht, Jan; Curi, Rui; Márquez, Javier; Colquhoun, Alison ; DeBerardinis, Ralph J.; Matés, José M.
Fonte: Journal of Molecular Medicine. Unidade: ICB
Assuntos: FISIOLOGIA, HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARTÍN-RUFIÁN, Mercedes et al. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, v. 92, n. 3, p. 277-290, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00109-013-1105-2. Acesso em: 01 nov. 2024.
APA
Martín-Rufián, M., Nascimento-Gomes, R., Higuero, A., Crisma, A. R., Campos-Sandoval, J. A., Gómez-Garcia, M. C., et al. (2014). Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, 92( 3), 277-290. doi:10.1007/s00109-013-1105-2
NLM
Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
Vancouver
Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
Artigo de periodico
Brain renin angiotensin in disease (2008)
Phillips, M. Ian; Oliveira, E. M.
Fonte: Journal of Molecular Medicine. Unidade: EEFE
Assuntos: SISTEMA RENINA-ANGIOTENSINA, HIPERTENSÃO, DEPRESSÃO, ALCOOLISMO, DOENÇA DE ALZHEIMER
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PHILLIPS, M. Ian e OLIVEIRA, E. M. Brain renin angiotensin in disease. Journal of Molecular Medicine, v. 86, 2008Tradução . . Disponível em: https://doi.org/10.1007/s00109-008-0331-5. Acesso em: 01 nov. 2024.
APA
Phillips, M. I., & Oliveira, E. M. (2008). Brain renin angiotensin in disease. Journal of Molecular Medicine, 86. doi:10.1007/s00109-008-0331-5
NLM
Phillips MI, Oliveira EM. Brain renin angiotensin in disease [Internet]. Journal of Molecular Medicine. 2008 ; 86[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-008-0331-5
Vancouver
Phillips MI, Oliveira EM. Brain renin angiotensin in disease [Internet]. Journal of Molecular Medicine. 2008 ; 86[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-008-0331-5
Artigo de periodico
A new evidence for the maintenance of the sarcoglycan complex in muscle sarcolemma in spite of the primary absence of 'delta'-SG protein (2007)
Gouveia, Telma Luciana Furtado; Kossugue, Patricia Mayumi; Paim, Julia F.; Zatz, Mayana ; Anderson, Louise V. B.; Nigro, Vicenzo; Vainzof, Mariz
Fonte: Journal of Molecular Medicine. Unidade: IB
Assuntos: DISTROFIA MUSCULAR, MUTAÇÃO GENÉTICA, PROTEÍNAS MUSCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GOUVEIA, Telma Luciana Furtado et al. A new evidence for the maintenance of the sarcoglycan complex in muscle sarcolemma in spite of the primary absence of 'delta'-SG protein. Journal of Molecular Medicine, v. 85, n. 4, p. 415-420, 2007Tradução . . Disponível em: https://doi.org/10.1007/s00109-007-0163-8. Acesso em: 01 nov. 2024.
APA
Gouveia, T. L. F., Kossugue, P. M., Paim, J. F., Zatz, M., Anderson, L. V. B., Nigro, V., & Vainzof, M. (2007). A new evidence for the maintenance of the sarcoglycan complex in muscle sarcolemma in spite of the primary absence of 'delta'-SG protein. Journal of Molecular Medicine, 85( 4), 415-420. doi:10.1007/s00109-007-0163-8
NLM
Gouveia TLF, Kossugue PM, Paim JF, Zatz M, Anderson LVB, Nigro V, Vainzof M. A new evidence for the maintenance of the sarcoglycan complex in muscle sarcolemma in spite of the primary absence of 'delta'-SG protein [Internet]. Journal of Molecular Medicine. 2007 ; 85( 4): 415-420.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-007-0163-8
Vancouver
Gouveia TLF, Kossugue PM, Paim JF, Zatz M, Anderson LVB, Nigro V, Vainzof M. A new evidence for the maintenance of the sarcoglycan complex in muscle sarcolemma in spite of the primary absence of 'delta'-SG protein [Internet]. Journal of Molecular Medicine. 2007 ; 85( 4): 415-420.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s00109-007-0163-8

Unidades USP

ReP

Filtros

Unidades USP

Departamento

Autores

Autores USP

Ano de publicação

Assuntos

Agência de fomento

Indexado em

Afiliação dos autores externos não normalizada