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Artigo de periodico
Tracheal smooth muscle cells stimulated by stem cell factor-c-kit coordinate the production of transforming growth factor-ß1 and fibroblast growth factor-2 mediated by chemokine (C-C motif) ligand 3 (2016)
Oliveira, Luis Cezar Farias de; Danilucci, Taís Marolato; Chaves-Neto, Antonio Hernandes; Campanelli, Ana Paula; Silva, Tereza Cristina Cardoso da; Oliveira, Sandra Helena Penha
Fonte: Journal of Interferon and Cytokine Research. Unidade: FOB
Assuntos: CÉLULAS-TRONCO, FIBROBLASTOS, QUIMIOCINAS, TRAQUEIA
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ABNT
OLIVEIRA, Luis Cezar Farias de et al. Tracheal smooth muscle cells stimulated by stem cell factor-c-kit coordinate the production of transforming growth factor-ß1 and fibroblast growth factor-2 mediated by chemokine (C-C motif) ligand 3. Journal of Interferon and Cytokine Research, v. 36, n. 6, p. 401-411, 2016Tradução . . Disponível em: https://doi.org/10.1089/jir.2015.0102. Acesso em: 26 out. 2025.
APA
Oliveira, L. C. F. de, Danilucci, T. M., Chaves-Neto, A. H., Campanelli, A. P., Silva, T. C. C. da, & Oliveira, S. H. P. (2016). Tracheal smooth muscle cells stimulated by stem cell factor-c-kit coordinate the production of transforming growth factor-ß1 and fibroblast growth factor-2 mediated by chemokine (C-C motif) ligand 3. Journal of Interferon and Cytokine Research, 36( 6), 401-411. doi:10.1089/jir.2015.0102
NLM
Oliveira LCF de, Danilucci TM, Chaves-Neto AH, Campanelli AP, Silva TCC da, Oliveira SHP. Tracheal smooth muscle cells stimulated by stem cell factor-c-kit coordinate the production of transforming growth factor-ß1 and fibroblast growth factor-2 mediated by chemokine (C-C motif) ligand 3 [Internet]. Journal of Interferon and Cytokine Research. 2016 ; 36( 6): 401-411.[citado 2025 out. 26 ] Available from: https://doi.org/10.1089/jir.2015.0102
Vancouver
Oliveira LCF de, Danilucci TM, Chaves-Neto AH, Campanelli AP, Silva TCC da, Oliveira SHP. Tracheal smooth muscle cells stimulated by stem cell factor-c-kit coordinate the production of transforming growth factor-ß1 and fibroblast growth factor-2 mediated by chemokine (C-C motif) ligand 3 [Internet]. Journal of Interferon and Cytokine Research. 2016 ; 36( 6): 401-411.[citado 2025 out. 26 ] Available from: https://doi.org/10.1089/jir.2015.0102
Artigo de periodico
Comparative analysis of activation phenotype proliferation, and IFN-γ production by spleen 'NK1.1 POT. +' and 'NK1.1 POT.-' T-cells during Plasmodium chabaudi AS Malaria (2010)
Muxel, Sandra Marcia; Rosário, Ana Paula Freitas do; Sardinha, Luiz Roberto; Castillo-Méndez, Sheyla Inés; Zago, Cláudia Augusta; Málaga, Sérgio Marcelo Rodriguez; Alvarez, José Maria ; Lima, Maria Regina D'Império
Fonte: Journal of Interferon and Cytokine Research. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
MUXEL, Sandra Marcia et al. Comparative analysis of activation phenotype proliferation, and IFN-γ production by spleen 'NK1.1 POT. +' and 'NK1.1 POT.-' T-cells during Plasmodium chabaudi AS Malaria. Journal of Interferon and Cytokine Research, v. 30, n. 6, p. 417-426, 2010Tradução . . Disponível em: https://doi.org/10.1089/jir.2009.0095. Acesso em: 26 out. 2025.
APA
Muxel, S. M., Rosário, A. P. F. do, Sardinha, L. R., Castillo-Méndez, S. I., Zago, C. A., Málaga, S. M. R., et al. (2010). Comparative analysis of activation phenotype proliferation, and IFN-γ production by spleen 'NK1.1 POT. +' and 'NK1.1 POT.-' T-cells during Plasmodium chabaudi AS Malaria. Journal of Interferon and Cytokine Research, 30( 6), 417-426. doi:10.1089/jir.2009.0095
NLM
Muxel SM, Rosário APF do, Sardinha LR, Castillo-Méndez SI, Zago CA, Málaga SMR, Alvarez JM, Lima MRD'I. Comparative analysis of activation phenotype proliferation, and IFN-γ production by spleen 'NK1.1 POT. +' and 'NK1.1 POT.-' T-cells during Plasmodium chabaudi AS Malaria [Internet]. Journal of Interferon and Cytokine Research. 2010 ; 30( 6): 417-426.[citado 2025 out. 26 ] Available from: https://doi.org/10.1089/jir.2009.0095
Vancouver
Muxel SM, Rosário APF do, Sardinha LR, Castillo-Méndez SI, Zago CA, Málaga SMR, Alvarez JM, Lima MRD'I. Comparative analysis of activation phenotype proliferation, and IFN-γ production by spleen 'NK1.1 POT. +' and 'NK1.1 POT.-' T-cells during Plasmodium chabaudi AS Malaria [Internet]. Journal of Interferon and Cytokine Research. 2010 ; 30( 6): 417-426.[citado 2025 out. 26 ] Available from: https://doi.org/10.1089/jir.2009.0095
Artigo de periodico
Impaired macrophage responses may contribute to exacerbation of blood-stage Plasmodium chabaudi malaria in IL-12 deficient mice (2002)
Bastos, Karina Ramalho Bortoluci; Barboza, Renato; Elias, Rosa Maria; Sardinha, Luiz Roberto; Grisotto, Marcos Grigolin; Marinho, Claudio Romero Farias ; Amarante-Mendes, João Gustavo Pessini ; Alvarez, José Maria ; Lima, Maria Regina D'Império
Fonte: Journal of Interferon and Cytokine Research. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
BASTOS, Karina Ramalho Bortoluci et al. Impaired macrophage responses may contribute to exacerbation of blood-stage Plasmodium chabaudi malaria in IL-12 deficient mice. Journal of Interferon and Cytokine Research, v. 01, p. 1191-1199, 2002Tradução . . Acesso em: 26 out. 2025.
APA
Bastos, K. R. B., Barboza, R., Elias, R. M., Sardinha, L. R., Grisotto, M. G., Marinho, C. R. F., et al. (2002). Impaired macrophage responses may contribute to exacerbation of blood-stage Plasmodium chabaudi malaria in IL-12 deficient mice. Journal of Interferon and Cytokine Research, 01, 1191-1199.
NLM
Bastos KRB, Barboza R, Elias RM, Sardinha LR, Grisotto MG, Marinho CRF, Amarante-Mendes JGP, Alvarez JM, Lima MRD'I. Impaired macrophage responses may contribute to exacerbation of blood-stage Plasmodium chabaudi malaria in IL-12 deficient mice. Journal of Interferon and Cytokine Research. 2002 ; 01 1191-1199.[citado 2025 out. 26 ]
Vancouver
Bastos KRB, Barboza R, Elias RM, Sardinha LR, Grisotto MG, Marinho CRF, Amarante-Mendes JGP, Alvarez JM, Lima MRD'I. Impaired macrophage responses may contribute to exacerbation of blood-stage Plasmodium chabaudi malaria in IL-12 deficient mice. Journal of Interferon and Cytokine Research. 2002 ; 01 1191-1199.[citado 2025 out. 26 ]
Artigo de periodico
Resistance to paracoccidioides brasiliensis infection is linked to a proferential Th1 immune response, whereas susceptibility is associated with absence of IFN-'gama' production (2000)
Kashino, Suely Sanae; Fazioli, R. A.; Cafalli-Favati, C.; Meloni-Bruneri, Lucia Helena; Vaz, C. A. C.; Burger, Eva; Singer, L. M.; Calich, Vera Lúcia Garcia
Fonte: Journal of Interferon and Cytokine Research. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
KASHINO, Suely Sanae et al. Resistance to paracoccidioides brasiliensis infection is linked to a proferential Th1 immune response, whereas susceptibility is associated with absence of IFN-'gama' production. Journal of Interferon and Cytokine Research, v. 20, p. 89-97, 2000Tradução . . Acesso em: 26 out. 2025.
APA
Kashino, S. S., Fazioli, R. A., Cafalli-Favati, C., Meloni-Bruneri, L. H., Vaz, C. A. C., Burger, E., et al. (2000). Resistance to paracoccidioides brasiliensis infection is linked to a proferential Th1 immune response, whereas susceptibility is associated with absence of IFN-'gama' production. Journal of Interferon and Cytokine Research, 20, 89-97.
NLM
Kashino SS, Fazioli RA, Cafalli-Favati C, Meloni-Bruneri LH, Vaz CAC, Burger E, Singer LM, Calich VLG. Resistance to paracoccidioides brasiliensis infection is linked to a proferential Th1 immune response, whereas susceptibility is associated with absence of IFN-'gama' production. Journal of Interferon and Cytokine Research. 2000 ; 20 89-97.[citado 2025 out. 26 ]
Vancouver
Kashino SS, Fazioli RA, Cafalli-Favati C, Meloni-Bruneri LH, Vaz CAC, Burger E, Singer LM, Calich VLG. Resistance to paracoccidioides brasiliensis infection is linked to a proferential Th1 immune response, whereas susceptibility is associated with absence of IFN-'gama' production. Journal of Interferon and Cytokine Research. 2000 ; 20 89-97.[citado 2025 out. 26 ]
Artigo de periodico
Effect of therapy with recombinant human interferon - 'GAMA' on the release of nitric oxide by neutrophils and mononuclear cells from patients with chronic granulomatous disease (1996)
Condino-Neto, A; Muscará, Marcelo Nicolás ; Bellinati-Pires, R; Carneiro-Sampaio, M. M. S.; Brandao, A C; Grumach, A S; De Nucci, Gilberto
Fonte: Journal of Interferon and Cytokine Research. Unidade: ICB
Assuntos: FARMACOLOGIA, IMUNOLOGIA
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ABNT
CONDINO-NETO, A et al. Effect of therapy with recombinant human interferon - 'GAMA' on the release of nitric oxide by neutrophils and mononuclear cells from patients with chronic granulomatous disease. Journal of Interferon and Cytokine Research, v. 16, p. 357-64, 1996Tradução . . Acesso em: 26 out. 2025.
APA
Condino-Neto, A., Muscará, M. N., Bellinati-Pires, R., Carneiro-Sampaio, M. M. S., Brandao, A. C., Grumach, A. S., & De Nucci, G. (1996). Effect of therapy with recombinant human interferon - 'GAMA' on the release of nitric oxide by neutrophils and mononuclear cells from patients with chronic granulomatous disease. Journal of Interferon and Cytokine Research, 16, 357-64.
NLM
Condino-Neto A, Muscará MN, Bellinati-Pires R, Carneiro-Sampaio MMS, Brandao AC, Grumach AS, De Nucci G. Effect of therapy with recombinant human interferon - 'GAMA' on the release of nitric oxide by neutrophils and mononuclear cells from patients with chronic granulomatous disease. Journal of Interferon and Cytokine Research. 1996 ;16 357-64.[citado 2025 out. 26 ]
Vancouver
Condino-Neto A, Muscará MN, Bellinati-Pires R, Carneiro-Sampaio MMS, Brandao AC, Grumach AS, De Nucci G. Effect of therapy with recombinant human interferon - 'GAMA' on the release of nitric oxide by neutrophils and mononuclear cells from patients with chronic granulomatous disease. Journal of Interferon and Cytokine Research. 1996 ;16 357-64.[citado 2025 out. 26 ]

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