ReP USP - Resultado da busca

Artigo de periodico
Acetate coordinates neutrophil and ILC3 responses against C. difficile through FFAR2 (2020)
Fachi, José Luís ; Silva, Cristiane Sécca da ; Rodrigues, Patrícia Brito; Mato, Felipe Cézar Pinheiro de ; Luccia, Blanda Di ; Felipe, Jaqueline de Souza; Pral, Laís Passariello ; Oliveira, Marcella Rungue ; Rocha, Victor de Melo; Sato, Fabio Takeo ; Sampaio, Ulliana ; Clerici, Maria Teresa Pedrosa Silva ; Rodrigues, Hosana Gomes ; Câmara, Niels Olsen Saraiva ; Consonni, Sílvio Roberto ; Vieira, Angélica Thomaz ; Oliveira, Sergio Costa; Mackay, Charles Reay ; Layden, Brian T. ; Bortoluci, Karina Ramalho ; Colonna, Marco ; Vinolo, Marco Aurélio Ramirez
Fonte: Journal of Experimental Medicine. Unidade: ICB
Assuntos: IMUNOLOGIA, NEUTRÓFILOS, ANTIBIÓTICOS, CLOSTRIDIUM, DIARREIA, COLITE, METABÓLITOS, ÁCIDOS GRAXOS, RATOS, MODELOS ANIMAIS
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ABNT
FACHI, José Luís et al. Acetate coordinates neutrophil and ILC3 responses against C. difficile through FFAR2. Journal of Experimental Medicine, v. 217, n. 3, p. 29 , 2020Tradução . . Disponível em: https://doi.org/10.1084/jem.20190489. Acesso em: 22 out. 2025.
APA
Fachi, J. L., Silva, C. S. da, Rodrigues, P. B., Mato, F. C. P. de, Luccia, B. D., Felipe, J. de S., et al. (2020). Acetate coordinates neutrophil and ILC3 responses against C. difficile through FFAR2. Journal of Experimental Medicine, 217( 3), 29 . doi:10.1084/jem.20190489
NLM
Fachi JL, Silva CS da, Rodrigues PB, Mato FCP de, Luccia BD, Felipe J de S, Pral LP, Oliveira MR, Rocha V de M, Sato FT, Sampaio U, Clerici MTPS, Rodrigues HG, Câmara NOS, Consonni SR, Vieira AT, Oliveira SC, Mackay CR, Layden BT, Bortoluci KR, Colonna M, Vinolo MAR. Acetate coordinates neutrophil and ILC3 responses against C. difficile through FFAR2 [Internet]. Journal of Experimental Medicine. 2020 ; 217( 3): 29 .[citado 2025 out. 22 ] Available from: https://doi.org/10.1084/jem.20190489
Vancouver
Fachi JL, Silva CS da, Rodrigues PB, Mato FCP de, Luccia BD, Felipe J de S, Pral LP, Oliveira MR, Rocha V de M, Sato FT, Sampaio U, Clerici MTPS, Rodrigues HG, Câmara NOS, Consonni SR, Vieira AT, Oliveira SC, Mackay CR, Layden BT, Bortoluci KR, Colonna M, Vinolo MAR. Acetate coordinates neutrophil and ILC3 responses against C. difficile through FFAR2 [Internet]. Journal of Experimental Medicine. 2020 ; 217( 3): 29 .[citado 2025 out. 22 ] Available from: https://doi.org/10.1084/jem.20190489
Artigo de periodico
PKM2 promotes Th17 cell differentiation and autoimmune inflammation by fine-tuning STAT3 activation (2020)
Damasceno, Luis Eduardo Alves; Prado, Douglas da Silva; Veras, Flávio Protásio; Fonseca, Mirian das Dores; Toller-kawahisa, Juliana Escher; Rosa, Marcos Henrique; Públio, Gabriel Azevedo ; Martins, Timna Varela ; Ramalho, Fernando Silva ; Waisman, Ari; Cunha, Fernando de Queiroz ; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assuntos: AUTOIMUNIDADE, ANIMAIS, DIFERENCIAÇÃO CELULAR, ENCEFALOMIELITE ANIMAL, REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
DAMASCENO, Luis Eduardo Alves et al. PKM2 promotes Th17 cell differentiation and autoimmune inflammation by fine-tuning STAT3 activation. Journal of Experimental Medicine, v. 217, n. 10, 2020Tradução . . Disponível em: https://doi.org/10.1084/jem.20190613. Acesso em: 22 out. 2025.
APA
Damasceno, L. E. A., Prado, D. da S., Veras, F. P., Fonseca, M. das D., Toller-kawahisa, J. E., Rosa, M. H., et al. (2020). PKM2 promotes Th17 cell differentiation and autoimmune inflammation by fine-tuning STAT3 activation. Journal of Experimental Medicine, 217( 10). doi:10.1084/jem.20190613
NLM
Damasceno LEA, Prado D da S, Veras FP, Fonseca M das D, Toller-kawahisa JE, Rosa MH, Públio GA, Martins TV, Ramalho FS, Waisman A, Cunha F de Q, Cunha TM, Alves Filho JCF. PKM2 promotes Th17 cell differentiation and autoimmune inflammation by fine-tuning STAT3 activation [Internet]. Journal of Experimental Medicine. 2020 ; 217( 10):[citado 2025 out. 22 ] Available from: https://doi.org/10.1084/jem.20190613
Vancouver
Damasceno LEA, Prado D da S, Veras FP, Fonseca M das D, Toller-kawahisa JE, Rosa MH, Públio GA, Martins TV, Ramalho FS, Waisman A, Cunha F de Q, Cunha TM, Alves Filho JCF. PKM2 promotes Th17 cell differentiation and autoimmune inflammation by fine-tuning STAT3 activation [Internet]. Journal of Experimental Medicine. 2020 ; 217( 10):[citado 2025 out. 22 ] Available from: https://doi.org/10.1084/jem.20190613
Artigo de periodico
Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset (2016)
Costa, Frederico R. C.; Françozo, Marcela C. S.; Oliveira, Gabriela G. de; Ignacio, Aline; Castoldi, Angela; Zamboni, Dario Simões; Ramos, Simone Gusmão; Câmara, Niels Olsen Saraiva ; Zoete, Marcel R. de; Palm, Noah W.; Flavell, Richard A.; Silva, João Santana da; Carlos, Daniela
Fonte: Journal of Experimental Medicine. Unidades: FMRP, ICB
Assuntos: DIABETES MELLITUS, INTESTINOS, BACTÉRIAS, PROTEÍNAS, DOENÇAS AUTOIMUNES, SISTEMA IMUNE, PÂNCREAS
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ABNT
COSTA, Frederico R. C. et al. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset. Journal of Experimental Medicine, v. 213, n. 7, p. 1223-1239, 2016Tradução . . Disponível em: https://doi.org/10.1084/jem.20150744. Acesso em: 22 out. 2025.
APA
Costa, F. R. C., Françozo, M. C. S., Oliveira, G. G. de, Ignacio, A., Castoldi, A., Zamboni, D. S., et al. (2016). Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset. Journal of Experimental Medicine, 213( 7), 1223-1239. doi:10.1084/jem.20150744
NLM
Costa FRC, Françozo MCS, Oliveira GG de, Ignacio A, Castoldi A, Zamboni DS, Ramos SG, Câmara NOS, Zoete MR de, Palm NW, Flavell RA, Silva JS da, Carlos D. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset [Internet]. Journal of Experimental Medicine. 2016 ; 213( 7): 1223-1239.[citado 2025 out. 22 ] Available from: https://doi.org/10.1084/jem.20150744
Vancouver
Costa FRC, Françozo MCS, Oliveira GG de, Ignacio A, Castoldi A, Zamboni DS, Ramos SG, Câmara NOS, Zoete MR de, Palm NW, Flavell RA, Silva JS da, Carlos D. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset [Internet]. Journal of Experimental Medicine. 2016 ; 213( 7): 1223-1239.[citado 2025 out. 22 ] Available from: https://doi.org/10.1084/jem.20150744
Artigo de periodico
Ticks produce highly selective chemokine binding proteins with antiinflammatory activity (2008)
Déruaz, Maud; Frauenschuh, Achim; Alessandri, Ana L.; Dias, João M.; Coelho, Fernanda M.; Russo, Remo C.; Ferreira, Beatriz Rossetti ; Graham, Gerard J.; Shaw, Jeffrey P.; Wells, Timothy N. C.; Teixeira, Mauro M.; Power, Christine A.; Proudfoot, Amanda E. I.
Fonte: Journal of Experimental Medicine. Unidade: EERP
Assuntos: ANTI-INFLAMATÓRIOS, ADJUVANTES IMUNOLÓGICOS
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ABNT
DÉRUAZ, Maud et al. Ticks produce highly selective chemokine binding proteins with antiinflammatory activity. Journal of Experimental Medicine, v. 205, n. 9, p. 2019-2031, 2008Tradução . . Disponível em: https://doi.org/10.1084/jem.20072689. Acesso em: 22 out. 2025.
APA
Déruaz, M., Frauenschuh, A., Alessandri, A. L., Dias, J. M., Coelho, F. M., Russo, R. C., et al. (2008). Ticks produce highly selective chemokine binding proteins with antiinflammatory activity. Journal of Experimental Medicine, 205( 9), 2019-2031. doi:10.1084/jem.20072689
NLM
Déruaz M, Frauenschuh A, Alessandri AL, Dias JM, Coelho FM, Russo RC, Ferreira BR, Graham GJ, Shaw JP, Wells TNC, Teixeira MM, Power CA, Proudfoot AEI. Ticks produce highly selective chemokine binding proteins with antiinflammatory activity [Internet]. Journal of Experimental Medicine. 2008 ; 205( 9): 2019-2031.[citado 2025 out. 22 ] Available from: https://doi.org/10.1084/jem.20072689
Vancouver
Déruaz M, Frauenschuh A, Alessandri AL, Dias JM, Coelho FM, Russo RC, Ferreira BR, Graham GJ, Shaw JP, Wells TNC, Teixeira MM, Power CA, Proudfoot AEI. Ticks produce highly selective chemokine binding proteins with antiinflammatory activity [Internet]. Journal of Experimental Medicine. 2008 ; 205( 9): 2019-2031.[citado 2025 out. 22 ] Available from: https://doi.org/10.1084/jem.20072689
Artigo de periodico
Role od promyelocytic leukemia (PML) protein in tumor suppression (2005)
Rego, Eduardo Magalhães; Wang, Zhu-Gang; Peruzzi, Daniela; He, Li-Zhen; Pandolfi, Pier Paolo
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assuntos: LEUCEMIA (GENÉTICA), GENÉTICA, APOPTOSE
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ABNT
REGO, Eduardo Magalhães et al. Role od promyelocytic leukemia (PML) protein in tumor suppression. Journal of Experimental Medicine, v. 193, n. 4, p. 521-529, 2005Tradução . . Acesso em: 22 out. 2025.
APA
Rego, E. M., Wang, Z. -G., Peruzzi, D., He, L. -Z., & Pandolfi, P. P. (2005). Role od promyelocytic leukemia (PML) protein in tumor suppression. Journal of Experimental Medicine, 193( 4), 521-529.
NLM
Rego EM, Wang Z-G, Peruzzi D, He L-Z, Pandolfi PP. Role od promyelocytic leukemia (PML) protein in tumor suppression. Journal of Experimental Medicine. 2005 ; 193( 4): 521-529.[citado 2025 out. 22 ]
Vancouver
Rego EM, Wang Z-G, Peruzzi D, He L-Z, Pandolfi PP. Role od promyelocytic leukemia (PML) protein in tumor suppression. Journal of Experimental Medicine. 2005 ; 193( 4): 521-529.[citado 2025 out. 22 ]
Artigo de periodico
Low penetrance, broad resistance, and favorable outcome of interleukin 12 receptor 'beta'1 deficiency: medical and immunological implications (2003)
Fieschi, Claire; Dupuis, Stéphanie; Catherinot, Emilie; Feinberg, Jacqueline; Bustamente, Jacinta; Breiman, Adrien; Altare, Frédéric; Baretto, Richard; Le Deist, Françoise; Kayal, Samer; Koch, Hartmut; Richter, Darko; Brezina, Martin; Aksu, Guzide; Wood, Phil; Al-Jumaah, Suliman; Raspall, Miquel; Duarte, Alberto José da Silva; Tuerlinckx, David; Virelizier, Jean-Louis; Fischer, Alain; Enright, Andrea; Bernhöft, Jutta; Clearly, Aileen M.; Vermylen, Christiane; Rodriguez-Gallego, Carlos; Davies, Graham; Blütters-Sawatzki, Renate; Siegrist, Claire-Anne; Ehlayel, Mohammad; Novelli, Vas; Haas, Walther H.; Levy, Jacob; Freihorst, Joachim; Al-Hajjar, Sami; Nadal, David; Vasconcelos, Dewton de Moraes; Jeppsson, Olle; Kutukculer, Necil; Frecerova, Klara; Caragol, Isabel; Lammas, David; Kumararatne, Dinakantha; Abel, Laurent; Casanova, Jean-Laurent
Fonte: Journal of Experimental Medicine. Unidade: FM
Assuntos: MICOBACTERIOSE, SALMONELLA, INTERFERONS
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ABNT
FIESCHI, Claire et al. Low penetrance, broad resistance, and favorable outcome of interleukin 12 receptor 'beta'1 deficiency: medical and immunological implications. Journal of Experimental Medicine, v. 197, n. 4, p. 527-535, 2003Tradução . . Acesso em: 22 out. 2025.
APA
Fieschi, C., Dupuis, S., Catherinot, E., Feinberg, J., Bustamente, J., Breiman, A., et al. (2003). Low penetrance, broad resistance, and favorable outcome of interleukin 12 receptor 'beta'1 deficiency: medical and immunological implications. Journal of Experimental Medicine, 197( 4), 527-535.
NLM
Fieschi C, Dupuis S, Catherinot E, Feinberg J, Bustamente J, Breiman A, Altare F, Baretto R, Le Deist F, Kayal S, Koch H, Richter D, Brezina M, Aksu G, Wood P, Al-Jumaah S, Raspall M, Duarte AJ da S, Tuerlinckx D, Virelizier J-L, Fischer A, Enright A, Bernhöft J, Clearly AM, Vermylen C, Rodriguez-Gallego C, Davies G, Blütters-Sawatzki R, Siegrist C-A, Ehlayel M, Novelli V, Haas WH, Levy J, Freihorst J, Al-Hajjar S, Nadal D, Vasconcelos D de M, Jeppsson O, Kutukculer N, Frecerova K, Caragol I, Lammas D, Kumararatne D, Abel L, Casanova J-L. Low penetrance, broad resistance, and favorable outcome of interleukin 12 receptor 'beta'1 deficiency: medical and immunological implications. Journal of Experimental Medicine. 2003 ; 197( 4): 527-535.[citado 2025 out. 22 ]
Vancouver
Fieschi C, Dupuis S, Catherinot E, Feinberg J, Bustamente J, Breiman A, Altare F, Baretto R, Le Deist F, Kayal S, Koch H, Richter D, Brezina M, Aksu G, Wood P, Al-Jumaah S, Raspall M, Duarte AJ da S, Tuerlinckx D, Virelizier J-L, Fischer A, Enright A, Bernhöft J, Clearly AM, Vermylen C, Rodriguez-Gallego C, Davies G, Blütters-Sawatzki R, Siegrist C-A, Ehlayel M, Novelli V, Haas WH, Levy J, Freihorst J, Al-Hajjar S, Nadal D, Vasconcelos D de M, Jeppsson O, Kutukculer N, Frecerova K, Caragol I, Lammas D, Kumararatne D, Abel L, Casanova J-L. Low penetrance, broad resistance, and favorable outcome of interleukin 12 receptor 'beta'1 deficiency: medical and immunological implications. Journal of Experimental Medicine. 2003 ; 197( 4): 527-535.[citado 2025 out. 22 ]
Artigo de periodico
Cloning and expression of a leishmania donavani gene instructed by a peptide isolated from major histocompatibility complex closs in molecules of infected macrophages (1995)
Campos Neto, A; Cordova, J; Santogelo, D; Ruddle, N H; Janeway, C Y; Mcmahon-Pratt, D
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assunto: GENÉTICA
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ABNT
CAMPOS NETO, A et al. Cloning and expression of a leishmania donavani gene instructed by a peptide isolated from major histocompatibility complex closs in molecules of infected macrophages. Journal of Experimental Medicine, v. 182, p. 1423, 1995Tradução . . Disponível em: https://doi.org/10.1084/jem.182.5.1423. Acesso em: 22 out. 2025.
APA
Campos Neto, A., Cordova, J., Santogelo, D., Ruddle, N. H., Janeway, C. Y., & Mcmahon-Pratt, D. (1995). Cloning and expression of a leishmania donavani gene instructed by a peptide isolated from major histocompatibility complex closs in molecules of infected macrophages. Journal of Experimental Medicine, 182, 1423. doi:10.1084/jem.182.5.1423
NLM
Campos Neto A, Cordova J, Santogelo D, Ruddle NH, Janeway CY, Mcmahon-Pratt D. Cloning and expression of a leishmania donavani gene instructed by a peptide isolated from major histocompatibility complex closs in molecules of infected macrophages [Internet]. Journal of Experimental Medicine. 1995 ;182 1423.[citado 2025 out. 22 ] Available from: https://doi.org/10.1084/jem.182.5.1423
Vancouver
Campos Neto A, Cordova J, Santogelo D, Ruddle NH, Janeway CY, Mcmahon-Pratt D. Cloning and expression of a leishmania donavani gene instructed by a peptide isolated from major histocompatibility complex closs in molecules of infected macrophages [Internet]. Journal of Experimental Medicine. 1995 ;182 1423.[citado 2025 out. 22 ] Available from: https://doi.org/10.1084/jem.182.5.1423
Artigo de periodico
Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi (1992)
Santos, R R; Rossi, Marcos Antonio; Laus, J L; Savino, J S S W; Mengele Junior, J O; Silva, João Santana da; Savino, W; Mengel, J.
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assunto: PATOLOGIA
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ABNT
SANTOS, R R et al. Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi. Journal of Experimental Medicine, v. 175, p. 29-39, 1992Tradução . . Acesso em: 22 out. 2025.
APA
Santos, R. R., Rossi, M. A., Laus, J. L., Savino, J. S. S. W., Mengele Junior, J. O., Silva, J. S. da, et al. (1992). Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi. Journal of Experimental Medicine, 175, 29-39.
NLM
Santos RR, Rossi MA, Laus JL, Savino JSSW, Mengele Junior JO, Silva JS da, Savino W, Mengel J. Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi. Journal of Experimental Medicine. 1992 ;175 29-39.[citado 2025 out. 22 ]
Vancouver
Santos RR, Rossi MA, Laus JL, Savino JSSW, Mengele Junior JO, Silva JS da, Savino W, Mengel J. Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi. Journal of Experimental Medicine. 1992 ;175 29-39.[citado 2025 out. 22 ]
Artigo de periodico
Il-10 and tnf-gama regulation of experimental trypanosoma cruzi infection (1992)
Silva, João Santana da; Morrissey, P J; Garbstein, K M; Anderson, D; Reed, S G
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assunto: MEDICINA INTERNA
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ABNT
SILVA, João Santana da et al. Il-10 and tnf-gama regulation of experimental trypanosoma cruzi infection. Journal of Experimental Medicine, v. 175, n. 1 , p. 169-74, 1992Tradução . . Acesso em: 22 out. 2025.
APA
Silva, J. S. da, Morrissey, P. J., Garbstein, K. M., Anderson, D., & Reed, S. G. (1992). Il-10 and tnf-gama regulation of experimental trypanosoma cruzi infection. Journal of Experimental Medicine, 175( 1 ), 169-74.
NLM
Silva JS da, Morrissey PJ, Garbstein KM, Anderson D, Reed SG. Il-10 and tnf-gama regulation of experimental trypanosoma cruzi infection. Journal of Experimental Medicine. 1992 ;175( 1 ): 169-74.[citado 2025 out. 22 ]
Vancouver
Silva JS da, Morrissey PJ, Garbstein KM, Anderson D, Reed SG. Il-10 and tnf-gama regulation of experimental trypanosoma cruzi infection. Journal of Experimental Medicine. 1992 ;175( 1 ): 169-74.[citado 2025 out. 22 ]
Artigo de periodico
Regulation of t. Cruzi infection in vitro and in vivo by transforming growth factor b (1991)
Silva, João Santana da; Twardik, D; Reed, S
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assunto: PARASITOLOGIA
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ABNT
SILVA, João Santana da e TWARDIK, D e REED, S. Regulation of t. Cruzi infection in vitro and in vivo by transforming growth factor b. Journal of Experimental Medicine, v. 174, p. 539-45, 1991Tradução . . Acesso em: 22 out. 2025.
APA
Silva, J. S. da, Twardik, D., & Reed, S. (1991). Regulation of t. Cruzi infection in vitro and in vivo by transforming growth factor b. Journal of Experimental Medicine, 174, 539-45.
NLM
Silva JS da, Twardik D, Reed S. Regulation of t. Cruzi infection in vitro and in vivo by transforming growth factor b. Journal of Experimental Medicine. 1991 ;174 539-45.[citado 2025 out. 22 ]
Vancouver
Silva JS da, Twardik D, Reed S. Regulation of t. Cruzi infection in vitro and in vivo by transforming growth factor b. Journal of Experimental Medicine. 1991 ;174 539-45.[citado 2025 out. 22 ]
Artigo de periodico
Translocation t (14 ; 18) in b cell lymphomas as a cause for defective immunoglobulin production (1989)
De Jong, David ; Voerdijk, B M H; Van Ommen, G J B; Kluin-Nelemans, J C; Beverstock, G C; Kluin, M
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assunto: GENÉTICA
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ABNT
DE JONG, David et al. Translocation t (14 ; 18) in b cell lymphomas as a cause for defective immunoglobulin production. Journal of Experimental Medicine, v. 169, n. 3 , p. 613-24, 1989Tradução . . Disponível em: https://doi.org/10.1084/jem.169.3.613. Acesso em: 22 out. 2025.
APA
De Jong, D., Voerdijk, B. M. H., Van Ommen, G. J. B., Kluin-Nelemans, J. C., Beverstock, G. C., & Kluin, M. (1989). Translocation t (14 ; 18) in b cell lymphomas as a cause for defective immunoglobulin production. Journal of Experimental Medicine, 169( 3 ), 613-24. doi:10.1084/jem.169.3.613
NLM
De Jong D, Voerdijk BMH, Van Ommen GJB, Kluin-Nelemans JC, Beverstock GC, Kluin M. Translocation t (14 ; 18) in b cell lymphomas as a cause for defective immunoglobulin production [Internet]. Journal of Experimental Medicine. 1989 ;169( 3 ): 613-24.[citado 2025 out. 22 ] Available from: https://doi.org/10.1084/jem.169.3.613
Vancouver
De Jong D, Voerdijk BMH, Van Ommen GJB, Kluin-Nelemans JC, Beverstock GC, Kluin M. Translocation t (14 ; 18) in b cell lymphomas as a cause for defective immunoglobulin production [Internet]. Journal of Experimental Medicine. 1989 ;169( 3 ): 613-24.[citado 2025 out. 22 ] Available from: https://doi.org/10.1084/jem.169.3.613

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