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Artigo de periodico
Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients (2021)
Rodrigues, Tamara Silva ; Sá, Keyla Santos Guedes de ; Ishimoto, Adriene Yumi; Becerra, Amanda; Oliveira, Samuel; Almeida, Leticia; Gonçalves, Augusto V.; Perucello, Debora B.; Andrade, Warrison A. ; Castro, Ricardo; Veras, Flavio P.; Toller-Kawahisa, Juliana E.; Nascimento, Daniele C.; Lima, Mikhael H. F. de; Silva, Camila M. S.; Caetite, Diego B.; Martins, Ronaldo Bragança ; Castro, Italo A.; Pontelli, Marjorie C.; Barros, Fabio C. de; Amaral, Natália B. do; Giannini, Marcela C.; Bonjorno, Letícia P.; Lopes, Maria Isabel F.; Santana, Rodrigo de Carvalho ; Vilar, Fernando C.; Auxiliadora-Martins, Maria ; Luppino-Assad, Rodrigo; Almeida, Sergio C. L. de; Oliveira, Fabiola R. de; Batah, Sabrina Setembre ; Siyuan, Li; Benatti, Maira N.; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz ; Cunha, Larissa D.; Frantz, Fabiani Gai ; Kohlsdorf, Tiana ; Fabro, Alexandre Todorovic ; Arruda, Eurico ; Oliveira, Renê D. R. de; Louzada Júnior, Paulo ; Zamboni, Dario Simões
Fonte: Journal of Experimental Medicine. Unidades: FMRP, FCFRP, FFCLRP
Assuntos: COVID-19, INFLAMAÇÃO
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ABNT
RODRIGUES, Tamara Silva et al. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients. Journal of Experimental Medicine, v. 218, n. 3, 2021Tradução . . Disponível em: https://doi.org/10.1084/jem.20201707. Acesso em: 24 out. 2025.
APA
Rodrigues, T. S., Sá, K. S. G. de, Ishimoto, A. Y., Becerra, A., Oliveira, S., Almeida, L., et al. (2021). Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients. Journal of Experimental Medicine, 218( 3). doi:10.1084/jem.20201707
NLM
Rodrigues TS, Sá KSG de, Ishimoto AY, Becerra A, Oliveira S, Almeida L, Gonçalves AV, Perucello DB, Andrade WA, Castro R, Veras FP, Toller-Kawahisa JE, Nascimento DC, Lima MHF de, Silva CMS, Caetite DB, Martins RB, Castro IA, Pontelli MC, Barros FC de, Amaral NB do, Giannini MC, Bonjorno LP, Lopes MIF, Santana R de C, Vilar FC, Auxiliadora-Martins M, Luppino-Assad R, Almeida SCL de, Oliveira FR de, Batah SS, Siyuan L, Benatti MN, Cunha TM, Alves Filho JCF, Cunha F de Q, Cunha LD, Frantz FG, Kohlsdorf T, Fabro AT, Arruda E, Oliveira RDR de, Louzada Júnior P, Zamboni DS. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients [Internet]. Journal of Experimental Medicine. 2021 ; 218( 3):[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.20201707
Vancouver
Rodrigues TS, Sá KSG de, Ishimoto AY, Becerra A, Oliveira S, Almeida L, Gonçalves AV, Perucello DB, Andrade WA, Castro R, Veras FP, Toller-Kawahisa JE, Nascimento DC, Lima MHF de, Silva CMS, Caetite DB, Martins RB, Castro IA, Pontelli MC, Barros FC de, Amaral NB do, Giannini MC, Bonjorno LP, Lopes MIF, Santana R de C, Vilar FC, Auxiliadora-Martins M, Luppino-Assad R, Almeida SCL de, Oliveira FR de, Batah SS, Siyuan L, Benatti MN, Cunha TM, Alves Filho JCF, Cunha F de Q, Cunha LD, Frantz FG, Kohlsdorf T, Fabro AT, Arruda E, Oliveira RDR de, Louzada Júnior P, Zamboni DS. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients [Internet]. Journal of Experimental Medicine. 2021 ; 218( 3):[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.20201707
Artigo de periodico
SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology (2020)
Veras, Flavio Protasio; Pontelli, Marjorie Cornejo ; Silva, Camila Meirelles de Souza; Toller-Kawahisa, Juliana E.; Lima, Mikhael de; Nascimento, Daniele Carvalho; Schneider, Ayda Henriques; Caetité, Diego; Tavares, Lucas Alves ; Paiva, Isadora M.; Rosales, Roberta Ribeiro Costa; morelo, David Fernando Colon ; Martins, Ronaldo Bragança ; Castro, Ítalo Araújo de ; Almeida, Glaucia Maria ; Lopes, Maria Isabel Fernandes; Benatti, Maíra Nilson; Bonjorno, Letícia Pastorelli; Giannini, Marcela Cavichioli; Luppino-Assad, Rodrigo; Almeida, Sérgio Luna; Vilar, Fernando; Santana, Rodrigo de Carvalho ; Bollela, Valdes Roberto ; Auxiliadora-Martins, Maria ; Borges, Marcos de Carvalho ; Miranda, Carlos Henrique ; Pazin Filho, Antônio ; Silva, Luis Lamberti Pinto da ; Cunha, Larissa Dias; Zamboni, Dario Simões ; Dal-Pizzol, Felipe; Leiria, Luiz Osório Silveira ; Siyuan, Li; Batah, Sabrina Setembre ; Fabro, Alexandre Todorovic ; Mauad, Thais ; Dolhnikoff, Marisa ; Duarte Neto, Amaro Nunes ; Saldiva, Paulo Hilário Nascimento ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Arruda, Eurico ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti; Cunha, Fernando de Queiroz
Fonte: Journal of Experimental Medicine. Unidades: FMRP, FM
Assuntos: COVID-19, CÉLULAS EPITELIAIS, PATOLOGIA, VIROLOGIA, PNEUMONIA, NEUTRÓFILOS, ANGIOTENSINA II
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ABNT
VERAS, Flavio Protasio et al. SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology. Journal of Experimental Medicine, v. 217, n. 12, 2020Tradução . . Disponível em: https://doi.org/10.1084/jem.20201129. Acesso em: 24 out. 2025.
APA
Veras, F. P., Pontelli, M. C., Silva, C. M. de S., Toller-Kawahisa, J. E., Lima, M. de, Nascimento, D. C., et al. (2020). SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology. Journal of Experimental Medicine, 217( 12). doi:10.1084/jem.20201129
NLM
Veras FP, Pontelli MC, Silva CM de S, Toller-Kawahisa JE, Lima M de, Nascimento DC, Schneider AH, Caetité D, Tavares LA, Paiva IM, Rosales RRC, morelo DFC, Martins RB, Castro ÍA de, Almeida GM, Lopes MIF, Benatti MN, Bonjorno LP, Giannini MC, Luppino-Assad R, Almeida SL, Vilar F, Santana R de C, Bollela VR, Auxiliadora-Martins M, Borges M de C, Miranda CH, Pazin Filho A, Silva LLP da, Cunha LD, Zamboni DS, Dal-Pizzol F, Leiria LOS, Siyuan L, Batah SS, Fabro AT, Mauad T, Dolhnikoff M, Duarte Neto AN, Saldiva PHN, Cunha TM, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RD, Cunha F de Q. SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology [Internet]. Journal of Experimental Medicine. 2020 ; 217( 12):[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.20201129
Vancouver
Veras FP, Pontelli MC, Silva CM de S, Toller-Kawahisa JE, Lima M de, Nascimento DC, Schneider AH, Caetité D, Tavares LA, Paiva IM, Rosales RRC, morelo DFC, Martins RB, Castro ÍA de, Almeida GM, Lopes MIF, Benatti MN, Bonjorno LP, Giannini MC, Luppino-Assad R, Almeida SL, Vilar F, Santana R de C, Bollela VR, Auxiliadora-Martins M, Borges M de C, Miranda CH, Pazin Filho A, Silva LLP da, Cunha LD, Zamboni DS, Dal-Pizzol F, Leiria LOS, Siyuan L, Batah SS, Fabro AT, Mauad T, Dolhnikoff M, Duarte Neto AN, Saldiva PHN, Cunha TM, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RD, Cunha F de Q. SARS-CoV-2–triggered neutrophil extracellular traps mediate COVID-19 pathology [Internet]. Journal of Experimental Medicine. 2020 ; 217( 12):[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.20201129
Artigo de periodico
Acetate coordinates neutrophil and ILC3 responses against C. difficile through FFAR2 (2020)
Fachi, José Luís ; Silva, Cristiane Sécca da ; Rodrigues, Patrícia Brito; Mato, Felipe Cézar Pinheiro de ; Luccia, Blanda Di ; Felipe, Jaqueline de Souza; Pral, Laís Passariello ; Oliveira, Marcella Rungue ; Rocha, Victor de Melo; Sato, Fabio Takeo ; Sampaio, Ulliana ; Clerici, Maria Teresa Pedrosa Silva ; Rodrigues, Hosana Gomes ; Câmara, Niels Olsen Saraiva ; Consonni, Sílvio Roberto ; Vieira, Angélica Thomaz ; Oliveira, Sergio Costa; Mackay, Charles Reay ; Layden, Brian T. ; Bortoluci, Karina Ramalho ; Colonna, Marco ; Vinolo, Marco Aurélio Ramirez
Fonte: Journal of Experimental Medicine. Unidade: ICB
Assuntos: IMUNOLOGIA, NEUTRÓFILOS, ANTIBIÓTICOS, CLOSTRIDIUM, DIARREIA, COLITE, METABÓLITOS, ÁCIDOS GRAXOS, RATOS, MODELOS ANIMAIS
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ABNT
FACHI, José Luís et al. Acetate coordinates neutrophil and ILC3 responses against C. difficile through FFAR2. Journal of Experimental Medicine, v. 217, n. 3, p. 29 , 2020Tradução . . Disponível em: https://doi.org/10.1084/jem.20190489. Acesso em: 24 out. 2025.
APA
Fachi, J. L., Silva, C. S. da, Rodrigues, P. B., Mato, F. C. P. de, Luccia, B. D., Felipe, J. de S., et al. (2020). Acetate coordinates neutrophil and ILC3 responses against C. difficile through FFAR2. Journal of Experimental Medicine, 217( 3), 29 . doi:10.1084/jem.20190489
NLM
Fachi JL, Silva CS da, Rodrigues PB, Mato FCP de, Luccia BD, Felipe J de S, Pral LP, Oliveira MR, Rocha V de M, Sato FT, Sampaio U, Clerici MTPS, Rodrigues HG, Câmara NOS, Consonni SR, Vieira AT, Oliveira SC, Mackay CR, Layden BT, Bortoluci KR, Colonna M, Vinolo MAR. Acetate coordinates neutrophil and ILC3 responses against C. difficile through FFAR2 [Internet]. Journal of Experimental Medicine. 2020 ; 217( 3): 29 .[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.20190489
Vancouver
Fachi JL, Silva CS da, Rodrigues PB, Mato FCP de, Luccia BD, Felipe J de S, Pral LP, Oliveira MR, Rocha V de M, Sato FT, Sampaio U, Clerici MTPS, Rodrigues HG, Câmara NOS, Consonni SR, Vieira AT, Oliveira SC, Mackay CR, Layden BT, Bortoluci KR, Colonna M, Vinolo MAR. Acetate coordinates neutrophil and ILC3 responses against C. difficile through FFAR2 [Internet]. Journal of Experimental Medicine. 2020 ; 217( 3): 29 .[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.20190489
Artigo de periodico
PKM2 promotes Th17 cell differentiation and autoimmune inflammation by fine-tuning STAT3 activation (2020)
Damasceno, Luis Eduardo Alves; Prado, Douglas da Silva; Veras, Flávio Protásio; Fonseca, Mirian das Dores; Toller-kawahisa, Juliana Escher; Rosa, Marcos Henrique; Públio, Gabriel Azevedo ; Martins, Timna Varela ; Ramalho, Fernando Silva ; Waisman, Ari; Cunha, Fernando de Queiroz ; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assuntos: AUTOIMUNIDADE, ANIMAIS, DIFERENCIAÇÃO CELULAR, ENCEFALOMIELITE ANIMAL, REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
DAMASCENO, Luis Eduardo Alves et al. PKM2 promotes Th17 cell differentiation and autoimmune inflammation by fine-tuning STAT3 activation. Journal of Experimental Medicine, v. 217, n. 10, 2020Tradução . . Disponível em: https://doi.org/10.1084/jem.20190613. Acesso em: 24 out. 2025.
APA
Damasceno, L. E. A., Prado, D. da S., Veras, F. P., Fonseca, M. das D., Toller-kawahisa, J. E., Rosa, M. H., et al. (2020). PKM2 promotes Th17 cell differentiation and autoimmune inflammation by fine-tuning STAT3 activation. Journal of Experimental Medicine, 217( 10). doi:10.1084/jem.20190613
NLM
Damasceno LEA, Prado D da S, Veras FP, Fonseca M das D, Toller-kawahisa JE, Rosa MH, Públio GA, Martins TV, Ramalho FS, Waisman A, Cunha F de Q, Cunha TM, Alves Filho JCF. PKM2 promotes Th17 cell differentiation and autoimmune inflammation by fine-tuning STAT3 activation [Internet]. Journal of Experimental Medicine. 2020 ; 217( 10):[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.20190613
Vancouver
Damasceno LEA, Prado D da S, Veras FP, Fonseca M das D, Toller-kawahisa JE, Rosa MH, Públio GA, Martins TV, Ramalho FS, Waisman A, Cunha F de Q, Cunha TM, Alves Filho JCF. PKM2 promotes Th17 cell differentiation and autoimmune inflammation by fine-tuning STAT3 activation [Internet]. Journal of Experimental Medicine. 2020 ; 217( 10):[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.20190613
Artigo de periodico
Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset (2016)
Costa, Frederico R. C.; Françozo, Marcela C. S.; Oliveira, Gabriela G. de; Ignacio, Aline; Castoldi, Angela; Zamboni, Dario Simões; Ramos, Simone Gusmão; Câmara, Niels Olsen Saraiva ; Zoete, Marcel R. de; Palm, Noah W.; Flavell, Richard A.; Silva, João Santana da; Carlos, Daniela
Fonte: Journal of Experimental Medicine. Unidades: FMRP, ICB
Assuntos: DIABETES MELLITUS, INTESTINOS, BACTÉRIAS, PROTEÍNAS, DOENÇAS AUTOIMUNES, SISTEMA IMUNE, PÂNCREAS
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ABNT
COSTA, Frederico R. C. et al. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset. Journal of Experimental Medicine, v. 213, n. 7, p. 1223-1239, 2016Tradução . . Disponível em: https://doi.org/10.1084/jem.20150744. Acesso em: 24 out. 2025.
APA
Costa, F. R. C., Françozo, M. C. S., Oliveira, G. G. de, Ignacio, A., Castoldi, A., Zamboni, D. S., et al. (2016). Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset. Journal of Experimental Medicine, 213( 7), 1223-1239. doi:10.1084/jem.20150744
NLM
Costa FRC, Françozo MCS, Oliveira GG de, Ignacio A, Castoldi A, Zamboni DS, Ramos SG, Câmara NOS, Zoete MR de, Palm NW, Flavell RA, Silva JS da, Carlos D. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset [Internet]. Journal of Experimental Medicine. 2016 ; 213( 7): 1223-1239.[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.20150744
Vancouver
Costa FRC, Françozo MCS, Oliveira GG de, Ignacio A, Castoldi A, Zamboni DS, Ramos SG, Câmara NOS, Zoete MR de, Palm NW, Flavell RA, Silva JS da, Carlos D. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset [Internet]. Journal of Experimental Medicine. 2016 ; 213( 7): 1223-1239.[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.20150744
Artigo de periodico
Ticks produce highly selective chemokine binding proteins with antiinflammatory activity (2008)
Déruaz, Maud; Frauenschuh, Achim; Alessandri, Ana L.; Dias, João M.; Coelho, Fernanda M.; Russo, Remo C.; Ferreira, Beatriz Rossetti ; Graham, Gerard J.; Shaw, Jeffrey P.; Wells, Timothy N. C.; Teixeira, Mauro M.; Power, Christine A.; Proudfoot, Amanda E. I.
Fonte: Journal of Experimental Medicine. Unidade: EERP
Assuntos: ANTI-INFLAMATÓRIOS, ADJUVANTES IMUNOLÓGICOS
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ABNT
DÉRUAZ, Maud et al. Ticks produce highly selective chemokine binding proteins with antiinflammatory activity. Journal of Experimental Medicine, v. 205, n. 9, p. 2019-2031, 2008Tradução . . Disponível em: https://doi.org/10.1084/jem.20072689. Acesso em: 24 out. 2025.
APA
Déruaz, M., Frauenschuh, A., Alessandri, A. L., Dias, J. M., Coelho, F. M., Russo, R. C., et al. (2008). Ticks produce highly selective chemokine binding proteins with antiinflammatory activity. Journal of Experimental Medicine, 205( 9), 2019-2031. doi:10.1084/jem.20072689
NLM
Déruaz M, Frauenschuh A, Alessandri AL, Dias JM, Coelho FM, Russo RC, Ferreira BR, Graham GJ, Shaw JP, Wells TNC, Teixeira MM, Power CA, Proudfoot AEI. Ticks produce highly selective chemokine binding proteins with antiinflammatory activity [Internet]. Journal of Experimental Medicine. 2008 ; 205( 9): 2019-2031.[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.20072689
Vancouver
Déruaz M, Frauenschuh A, Alessandri AL, Dias JM, Coelho FM, Russo RC, Ferreira BR, Graham GJ, Shaw JP, Wells TNC, Teixeira MM, Power CA, Proudfoot AEI. Ticks produce highly selective chemokine binding proteins with antiinflammatory activity [Internet]. Journal of Experimental Medicine. 2008 ; 205( 9): 2019-2031.[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.20072689
Artigo de periodico
Low penetrance, broad resistance, and favorable outcome of interleukin 12 receptor 'beta'1 deficiency: medical and immunological implications (2003)
Fieschi, Claire; Dupuis, Stéphanie; Catherinot, Emilie; Feinberg, Jacqueline; Bustamente, Jacinta; Breiman, Adrien; Altare, Frédéric; Baretto, Richard; Le Deist, Françoise; Kayal, Samer; Koch, Hartmut; Richter, Darko; Brezina, Martin; Aksu, Guzide; Wood, Phil; Al-Jumaah, Suliman; Raspall, Miquel; Duarte, Alberto José da Silva; Tuerlinckx, David; Virelizier, Jean-Louis; Fischer, Alain; Enright, Andrea; Bernhöft, Jutta; Clearly, Aileen M.; Vermylen, Christiane; Rodriguez-Gallego, Carlos; Davies, Graham; Blütters-Sawatzki, Renate; Siegrist, Claire-Anne; Ehlayel, Mohammad; Novelli, Vas; Haas, Walther H.; Levy, Jacob; Freihorst, Joachim; Al-Hajjar, Sami; Nadal, David; Vasconcelos, Dewton de Moraes; Jeppsson, Olle; Kutukculer, Necil; Frecerova, Klara; Caragol, Isabel; Lammas, David; Kumararatne, Dinakantha; Abel, Laurent; Casanova, Jean-Laurent
Fonte: Journal of Experimental Medicine. Unidade: FM
Assuntos: MICOBACTERIOSE, SALMONELLA, INTERFERONS
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ABNT
FIESCHI, Claire et al. Low penetrance, broad resistance, and favorable outcome of interleukin 12 receptor 'beta'1 deficiency: medical and immunological implications. Journal of Experimental Medicine, v. 197, n. 4, p. 527-535, 2003Tradução . . Acesso em: 24 out. 2025.
APA
Fieschi, C., Dupuis, S., Catherinot, E., Feinberg, J., Bustamente, J., Breiman, A., et al. (2003). Low penetrance, broad resistance, and favorable outcome of interleukin 12 receptor 'beta'1 deficiency: medical and immunological implications. Journal of Experimental Medicine, 197( 4), 527-535.
NLM
Fieschi C, Dupuis S, Catherinot E, Feinberg J, Bustamente J, Breiman A, Altare F, Baretto R, Le Deist F, Kayal S, Koch H, Richter D, Brezina M, Aksu G, Wood P, Al-Jumaah S, Raspall M, Duarte AJ da S, Tuerlinckx D, Virelizier J-L, Fischer A, Enright A, Bernhöft J, Clearly AM, Vermylen C, Rodriguez-Gallego C, Davies G, Blütters-Sawatzki R, Siegrist C-A, Ehlayel M, Novelli V, Haas WH, Levy J, Freihorst J, Al-Hajjar S, Nadal D, Vasconcelos D de M, Jeppsson O, Kutukculer N, Frecerova K, Caragol I, Lammas D, Kumararatne D, Abel L, Casanova J-L. Low penetrance, broad resistance, and favorable outcome of interleukin 12 receptor 'beta'1 deficiency: medical and immunological implications. Journal of Experimental Medicine. 2003 ; 197( 4): 527-535.[citado 2025 out. 24 ]
Vancouver
Fieschi C, Dupuis S, Catherinot E, Feinberg J, Bustamente J, Breiman A, Altare F, Baretto R, Le Deist F, Kayal S, Koch H, Richter D, Brezina M, Aksu G, Wood P, Al-Jumaah S, Raspall M, Duarte AJ da S, Tuerlinckx D, Virelizier J-L, Fischer A, Enright A, Bernhöft J, Clearly AM, Vermylen C, Rodriguez-Gallego C, Davies G, Blütters-Sawatzki R, Siegrist C-A, Ehlayel M, Novelli V, Haas WH, Levy J, Freihorst J, Al-Hajjar S, Nadal D, Vasconcelos D de M, Jeppsson O, Kutukculer N, Frecerova K, Caragol I, Lammas D, Kumararatne D, Abel L, Casanova J-L. Low penetrance, broad resistance, and favorable outcome of interleukin 12 receptor 'beta'1 deficiency: medical and immunological implications. Journal of Experimental Medicine. 2003 ; 197( 4): 527-535.[citado 2025 out. 24 ]
Artigo de periodico
Cloning and expression of a leishmania donavani gene instructed by a peptide isolated from major histocompatibility complex closs in molecules of infected macrophages (1995)
Campos Neto, A; Cordova, J; Santogelo, D; Ruddle, N H; Janeway, C Y; Mcmahon-Pratt, D
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assunto: GENÉTICA
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ABNT
CAMPOS NETO, A et al. Cloning and expression of a leishmania donavani gene instructed by a peptide isolated from major histocompatibility complex closs in molecules of infected macrophages. Journal of Experimental Medicine, v. 182, p. 1423, 1995Tradução . . Disponível em: https://doi.org/10.1084/jem.182.5.1423. Acesso em: 24 out. 2025.
APA
Campos Neto, A., Cordova, J., Santogelo, D., Ruddle, N. H., Janeway, C. Y., & Mcmahon-Pratt, D. (1995). Cloning and expression of a leishmania donavani gene instructed by a peptide isolated from major histocompatibility complex closs in molecules of infected macrophages. Journal of Experimental Medicine, 182, 1423. doi:10.1084/jem.182.5.1423
NLM
Campos Neto A, Cordova J, Santogelo D, Ruddle NH, Janeway CY, Mcmahon-Pratt D. Cloning and expression of a leishmania donavani gene instructed by a peptide isolated from major histocompatibility complex closs in molecules of infected macrophages [Internet]. Journal of Experimental Medicine. 1995 ;182 1423.[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.182.5.1423
Vancouver
Campos Neto A, Cordova J, Santogelo D, Ruddle NH, Janeway CY, Mcmahon-Pratt D. Cloning and expression of a leishmania donavani gene instructed by a peptide isolated from major histocompatibility complex closs in molecules of infected macrophages [Internet]. Journal of Experimental Medicine. 1995 ;182 1423.[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.182.5.1423
Artigo de periodico
Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi (1992)
Santos, R R; Rossi, Marcos Antonio; Laus, J L; Savino, J S S W; Mengele Junior, J O; Silva, João Santana da; Savino, W; Mengel, J.
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assunto: PATOLOGIA
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ABNT
SANTOS, R R et al. Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi. Journal of Experimental Medicine, v. 175, p. 29-39, 1992Tradução . . Acesso em: 24 out. 2025.
APA
Santos, R. R., Rossi, M. A., Laus, J. L., Savino, J. S. S. W., Mengele Junior, J. O., Silva, J. S. da, et al. (1992). Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi. Journal of Experimental Medicine, 175, 29-39.
NLM
Santos RR, Rossi MA, Laus JL, Savino JSSW, Mengele Junior JO, Silva JS da, Savino W, Mengel J. Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi. Journal of Experimental Medicine. 1992 ;175 29-39.[citado 2025 out. 24 ]
Vancouver
Santos RR, Rossi MA, Laus JL, Savino JSSW, Mengele Junior JO, Silva JS da, Savino W, Mengel J. Anti- 'CD IND.4' abrogates rejection and restabliches long-term tolerance to sybgenetic newborn hearts grafted in mice chronically infected with trypanosoma cruzi. Journal of Experimental Medicine. 1992 ;175 29-39.[citado 2025 out. 24 ]
Artigo de periodico
Il-10 and tnf-gama regulation of experimental trypanosoma cruzi infection (1992)
Silva, João Santana da; Morrissey, P J; Garbstein, K M; Anderson, D; Reed, S G
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assunto: MEDICINA INTERNA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, João Santana da et al. Il-10 and tnf-gama regulation of experimental trypanosoma cruzi infection. Journal of Experimental Medicine, v. 175, n. 1 , p. 169-74, 1992Tradução . . Acesso em: 24 out. 2025.
APA
Silva, J. S. da, Morrissey, P. J., Garbstein, K. M., Anderson, D., & Reed, S. G. (1992). Il-10 and tnf-gama regulation of experimental trypanosoma cruzi infection. Journal of Experimental Medicine, 175( 1 ), 169-74.
NLM
Silva JS da, Morrissey PJ, Garbstein KM, Anderson D, Reed SG. Il-10 and tnf-gama regulation of experimental trypanosoma cruzi infection. Journal of Experimental Medicine. 1992 ;175( 1 ): 169-74.[citado 2025 out. 24 ]
Vancouver
Silva JS da, Morrissey PJ, Garbstein KM, Anderson D, Reed SG. Il-10 and tnf-gama regulation of experimental trypanosoma cruzi infection. Journal of Experimental Medicine. 1992 ;175( 1 ): 169-74.[citado 2025 out. 24 ]
Artigo de periodico
Regulation of t. Cruzi infection in vitro and in vivo by transforming growth factor b (1991)
Silva, João Santana da; Twardik, D; Reed, S
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assunto: PARASITOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, João Santana da e TWARDIK, D e REED, S. Regulation of t. Cruzi infection in vitro and in vivo by transforming growth factor b. Journal of Experimental Medicine, v. 174, p. 539-45, 1991Tradução . . Acesso em: 24 out. 2025.
APA
Silva, J. S. da, Twardik, D., & Reed, S. (1991). Regulation of t. Cruzi infection in vitro and in vivo by transforming growth factor b. Journal of Experimental Medicine, 174, 539-45.
NLM
Silva JS da, Twardik D, Reed S. Regulation of t. Cruzi infection in vitro and in vivo by transforming growth factor b. Journal of Experimental Medicine. 1991 ;174 539-45.[citado 2025 out. 24 ]
Vancouver
Silva JS da, Twardik D, Reed S. Regulation of t. Cruzi infection in vitro and in vivo by transforming growth factor b. Journal of Experimental Medicine. 1991 ;174 539-45.[citado 2025 out. 24 ]
Artigo de periodico
Translocation t (14 ; 18) in b cell lymphomas as a cause for defective immunoglobulin production (1989)
De Jong, David ; Voerdijk, B M H; Van Ommen, G J B; Kluin-Nelemans, J C; Beverstock, G C; Kluin, M
Fonte: Journal of Experimental Medicine. Unidade: FMRP
Assunto: GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DE JONG, David et al. Translocation t (14 ; 18) in b cell lymphomas as a cause for defective immunoglobulin production. Journal of Experimental Medicine, v. 169, n. 3 , p. 613-24, 1989Tradução . . Disponível em: https://doi.org/10.1084/jem.169.3.613. Acesso em: 24 out. 2025.
APA
De Jong, D., Voerdijk, B. M. H., Van Ommen, G. J. B., Kluin-Nelemans, J. C., Beverstock, G. C., & Kluin, M. (1989). Translocation t (14 ; 18) in b cell lymphomas as a cause for defective immunoglobulin production. Journal of Experimental Medicine, 169( 3 ), 613-24. doi:10.1084/jem.169.3.613
NLM
De Jong D, Voerdijk BMH, Van Ommen GJB, Kluin-Nelemans JC, Beverstock GC, Kluin M. Translocation t (14 ; 18) in b cell lymphomas as a cause for defective immunoglobulin production [Internet]. Journal of Experimental Medicine. 1989 ;169( 3 ): 613-24.[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.169.3.613
Vancouver
De Jong D, Voerdijk BMH, Van Ommen GJB, Kluin-Nelemans JC, Beverstock GC, Kluin M. Translocation t (14 ; 18) in b cell lymphomas as a cause for defective immunoglobulin production [Internet]. Journal of Experimental Medicine. 1989 ;169( 3 ): 613-24.[citado 2025 out. 24 ] Available from: https://doi.org/10.1084/jem.169.3.613

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