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Artigo de periodico
Deciphering the IgG idiotype network through proteomic analysis of potential targets in SARS-CoV-2-induced immune responses (2025)
Machado, Nicolle Rakanidis; Fagundes, Beatriz Oliveira; Nascimento, Lais Alves do; Bergamasco, Isabella Siuffi; Sgnotto, Fabio da Ressureicao; Fernandes, Iara Grigoletto; Fernandes, Juliana Ruiz; Pinto, Thalyta Nery Carvalho; Benard, Gil ; Sato, Maria Notomi
Source: Immunology. Unidade: FM
Subjects: COVID-19, SISTEMA IMUNE, ANTICORPOS
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MACHADO, Nicolle Rakanidis et al. Deciphering the IgG idiotype network through proteomic analysis of potential targets in SARS-CoV-2-induced immune responses. Immunology, v. 175, n. 2, p. 226-239, 2025Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/87159. Acesso em: 24 abr. 2026.
APA
Machado, N. R., Fagundes, B. O., Nascimento, L. A. do, Bergamasco, I. S., Sgnotto, F. da R., Fernandes, I. G., et al. (2025). Deciphering the IgG idiotype network through proteomic analysis of potential targets in SARS-CoV-2-induced immune responses. Immunology, 175( 2), 226-239. doi:10.1111/imm.13919
NLM
Machado NR, Fagundes BO, Nascimento LA do, Bergamasco IS, Sgnotto F da R, Fernandes IG, Fernandes JR, Pinto TNC, Benard G, Sato MN. Deciphering the IgG idiotype network through proteomic analysis of potential targets in SARS-CoV-2-induced immune responses [Internet]. Immunology. 2025 ; 175( 2): 226-239.[citado 2026 abr. 24 ] Available from: https://observatorio.fm.usp.br/handle/OPI/87159
Vancouver
Machado NR, Fagundes BO, Nascimento LA do, Bergamasco IS, Sgnotto F da R, Fernandes IG, Fernandes JR, Pinto TNC, Benard G, Sato MN. Deciphering the IgG idiotype network through proteomic analysis of potential targets in SARS-CoV-2-induced immune responses [Internet]. Immunology. 2025 ; 175( 2): 226-239.[citado 2026 abr. 24 ] Available from: https://observatorio.fm.usp.br/handle/OPI/87159
Artigo de periodico
Limitation of deleterious iNOS-Expressing interstitial macrophages is dependent on protective IL-1α and IL-17 in tuberculosis and existing obesity (2025)
Appolini, Vinicius Bottura ; Oliveira, Rômulo Silva de; Rosa, Ualter Guilherme Cipriano ; Corrêa, Giseli Furlan ; Gembre, Ana Flávia; Fraga-Silva, Thais Fernanda de Campos ; Bonato, Vânia Luiza Deperon ; Ramalho, Leandra Naira Zambelli ; Costa, Diego Luís
Source: Immunology. Unidades: FMRP, ICB
Subjects: IMUNOLOGIA, MODELOS ANIMAIS DE DOENÇAS, INTERFERONS, INTERLEUCINAS, PULMÃO, MACRÓFAGOS, MYCOBACTERIUM TUBERCULOSIS, ÓXIDO NÍTRICO, OBESIDADE, TUBERCULOSE PULMONAR, CAMUNDONGOS
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APPOLINI, Vinicius Bottura et al. Limitation of deleterious iNOS-Expressing interstitial macrophages is dependent on protective IL-1α and IL-17 in tuberculosis and existing obesity. Immunology, v. 175, n. 4, p. 518-533, 2025Tradução . . Disponível em: https://doi.org/10.1111/imm.13947. Acesso em: 24 abr. 2026.
APA
Appolini, V. B., Oliveira, R. S. de, Rosa, U. G. C., Corrêa, G. F., Gembre, A. F., Fraga-Silva, T. F. de C., et al. (2025). Limitation of deleterious iNOS-Expressing interstitial macrophages is dependent on protective IL-1α and IL-17 in tuberculosis and existing obesity. Immunology, 175( 4), 518-533. doi:10.1111/imm.13947
NLM
Appolini VB, Oliveira RS de, Rosa UGC, Corrêa GF, Gembre AF, Fraga-Silva TF de C, Bonato VLD, Ramalho LNZ, Costa DL. Limitation of deleterious iNOS-Expressing interstitial macrophages is dependent on protective IL-1α and IL-17 in tuberculosis and existing obesity [Internet]. Immunology. 2025 ; 175( 4): 518-533.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13947
Vancouver
Appolini VB, Oliveira RS de, Rosa UGC, Corrêa GF, Gembre AF, Fraga-Silva TF de C, Bonato VLD, Ramalho LNZ, Costa DL. Limitation of deleterious iNOS-Expressing interstitial macrophages is dependent on protective IL-1α and IL-17 in tuberculosis and existing obesity [Internet]. Immunology. 2025 ; 175( 4): 518-533.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13947
Artigo de periodico
Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection (2023)
Moreira, Thaiane Pinto; Sousa, Carla Daiane Ferreira de; Costa, Victor Rodrigues de Melo; Queiroz Junior, Celso Martins; Santos, Franciele Martins; Bonilha, Caio Santos ; Ésper, Lísia Maria; Nogueira, Mauricio Lacerda ; Cunha, Thiago Mattar ; Teixeira, Mauro Martins ; Costa, Vivian Vasconcelos; Souza, Daniele da Glória de
Source: Immunology. Unidade: FMRP
Subjects: ARTRALGIA, VÍRUS CHIKUNGUNYA, NECROSE, REPLICAÇÃO VIRAL
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MOREIRA, Thaiane Pinto et al. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection. Immunology, v. 168, n. 3, p. 444-458, 2023Tradução . . Disponível em: https://doi.org/10.1111/imm.13583. Acesso em: 24 abr. 2026.
APA
Moreira, T. P., Sousa, C. D. F. de, Costa, V. R. de M., Queiroz Junior, C. M., Santos, F. M., Bonilha, C. S., et al. (2023). Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection. Immunology, 168( 3), 444-458. doi:10.1111/imm.13583
NLM
Moreira TP, Sousa CDF de, Costa VR de M, Queiroz Junior CM, Santos FM, Bonilha CS, Ésper LM, Nogueira ML, Cunha TM, Teixeira MM, Costa VV, Souza D da G de. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection [Internet]. Immunology. 2023 ; 168( 3): 444-458.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13583
Vancouver
Moreira TP, Sousa CDF de, Costa VR de M, Queiroz Junior CM, Santos FM, Bonilha CS, Ésper LM, Nogueira ML, Cunha TM, Teixeira MM, Costa VV, Souza D da G de. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection [Internet]. Immunology. 2023 ; 168( 3): 444-458.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13583
Artigo de periodico
The turning point of COVID‐19 severity is associated with a unique circulating neutrophil gene signature (2023)
Fuzo, Carlos Alessandro ; Silva, Thais Fernanda de Campos Fraga ; Maruyama, Sandra Regina Costa ; Bastos, Victor Alexandre Félix; Rogerio, Luana Aparecida; Takamiya, Nayore Tamie; Silva-Neto, Pedro V. da; Pimentel, Vinícius Eduardo; Toro, Diana Mota; Pérez, Malena Martínez ; Carvalho, Jonatan C S de; Carmona-Garcia, Ingryd; Oliveira, Camilla N. S.; Degiovani, Augusto Marcussi; Ostini, Fatima M.; Constant, Leticia F.; Amorim, Alessandro P. de; Vilar, Fernando Crivelenti ; Feitosa, Marley Ribeiro ; Parra, Rogério Serafim ; Rocha, José Joaquim Ribeiro da ; Féres, Omar ; Gaspar, Gilberto Gambero ; Viana, Angelina Lettiere ; Fernandes, Ana Paula Morais ; Santos, Isabel Kinney Ferreira de Miranda ; Russo, Elisa Maria de Sousa ; Cardoso, Cristina Ribeiro de Barros ; Sorgi, Carlos Arterio ; Faccioli, Lucia Helena ; Bonato, Vânia Luiza Deperon ; Baruffi, Marcelo Dias
Source: Immunology. Unidades: FMRP, EERP, FCFRP, FFCLRP
Subjects: BIOMARCADORES, COVID-19, NEUTRÓFILOS, EXPRESSÃO GÊNICA
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FUZO, Carlos Alessandro et al. The turning point of COVID‐19 severity is associated with a unique circulating neutrophil gene signature. Immunology, v. 169, p. 323-343, 2023Tradução . . Disponível em: https://doi.org/10.1111/imm.13631. Acesso em: 24 abr. 2026.
APA
Fuzo, C. A., Silva, T. F. de C. F., Maruyama, S. R. C., Bastos, V. A. F., Rogerio, L. A., Takamiya, N. T., et al. (2023). The turning point of COVID‐19 severity is associated with a unique circulating neutrophil gene signature. Immunology, 169, 323-343. doi:10.1111/imm.13631
NLM
Fuzo CA, Silva TF de CF, Maruyama SRC, Bastos VAF, Rogerio LA, Takamiya NT, Silva-Neto PV da, Pimentel VE, Toro DM, Pérez MM, Carvalho JCS de, Carmona-Garcia I, Oliveira CNS, Degiovani AM, Ostini FM, Constant LF, Amorim AP de, Vilar FC, Feitosa MR, Parra RS, Rocha JJR da, Féres O, Gaspar GG, Viana AL, Fernandes APM, Santos IKF de M, Russo EM de S, Cardoso CR de B, Sorgi CA, Faccioli LH, Bonato VLD, Baruffi MD. The turning point of COVID‐19 severity is associated with a unique circulating neutrophil gene signature [Internet]. Immunology. 2023 ; 169 323-343.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13631
Vancouver
Fuzo CA, Silva TF de CF, Maruyama SRC, Bastos VAF, Rogerio LA, Takamiya NT, Silva-Neto PV da, Pimentel VE, Toro DM, Pérez MM, Carvalho JCS de, Carmona-Garcia I, Oliveira CNS, Degiovani AM, Ostini FM, Constant LF, Amorim AP de, Vilar FC, Feitosa MR, Parra RS, Rocha JJR da, Féres O, Gaspar GG, Viana AL, Fernandes APM, Santos IKF de M, Russo EM de S, Cardoso CR de B, Sorgi CA, Faccioli LH, Bonato VLD, Baruffi MD. The turning point of COVID‐19 severity is associated with a unique circulating neutrophil gene signature [Internet]. Immunology. 2023 ; 169 323-343.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13631
Artigo de periodico
The role of annexin A1 in the modulation of the NLRP3 inflammasome (2020)
Galvão, Izabela; Carvalho, Renan Villanova Homem de; Vago, Juliana P.; Silva, Alexandre Luiz Neves; Carvalho, Toniana G.; Antunes, Maísa Mota; Ribeiro, Fabiola Mara; Menezes, Gustavo B.; Zamboni, Dario Simões; Sousa, Lirlândia Pires; Teixeira, Mauro Martins
Source: Immunology. Unidade: FMRP
Subjects: CARTILAGEM ARTICULAR, CÉLULAS CULTIVADAS, IMUNOLOGIA, INTERLEUCINA 1, PULMÃO, MACRÓFAGOS, CULTURA DE CÉLULAS, TRANSCRIÇÃO GÊNICA, ÁCIDO ÚRICO
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GALVÃO, Izabela et al. The role of annexin A1 in the modulation of the NLRP3 inflammasome. Immunology, v. 160, n. 1, p. 78-89, 2020Tradução . . Disponível em: https://doi.org/10.1111/imm.13184. Acesso em: 24 abr. 2026.
APA
Galvão, I., Carvalho, R. V. H. de, Vago, J. P., Silva, A. L. N., Carvalho, T. G., Antunes, M. M., et al. (2020). The role of annexin A1 in the modulation of the NLRP3 inflammasome. Immunology, 160( 1), 78-89. doi:10.1111/imm.13184
NLM
Galvão I, Carvalho RVH de, Vago JP, Silva ALN, Carvalho TG, Antunes MM, Ribeiro FM, Menezes GB, Zamboni DS, Sousa LP, Teixeira MM. The role of annexin A1 in the modulation of the NLRP3 inflammasome [Internet]. Immunology. 2020 ; 160( 1): 78-89.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13184
Vancouver
Galvão I, Carvalho RVH de, Vago JP, Silva ALN, Carvalho TG, Antunes MM, Ribeiro FM, Menezes GB, Zamboni DS, Sousa LP, Teixeira MM. The role of annexin A1 in the modulation of the NLRP3 inflammasome [Internet]. Immunology. 2020 ; 160( 1): 78-89.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13184
Artigo de periodico
High levels of serum glycans monovalent IgG immune complexes detected by dissociative ELISA in experimental visceral leishmaniasis (2019)
Carvalho, Camila Aparecida de; Ferrao, Thiago Fidelis; Freitas, Flavia Regina Novais de; Andrade Junior, Heitor Franco de
Source: Immunology. Unidade: FM
Subjects: LEISHMANIOSE VISCERAL, ELISA, IMUNOGLOBULINAS, HAPTENOS, PROTEÍNAS DE TRANSPORTE, CROMATOGRAFIA, POLISSACARÍDEOS, COMPLEXO ANTÍGENO-ANTICORPO, MODELOS ANIMAIS DE DOENÇAS, HAMSTERS, ALBUMINAS
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ABNT
CARVALHO, Camila Aparecida de et al. High levels of serum glycans monovalent IgG immune complexes detected by dissociative ELISA in experimental visceral leishmaniasis. Immunology, v. 158, n. 4, p. 314-321, 2019Tradução . . Disponível em: https://doi.org/10.1111/imm.13118. Acesso em: 24 abr. 2026.
APA
Carvalho, C. A. de, Ferrao, T. F., Freitas, F. R. N. de, & Andrade Junior, H. F. de. (2019). High levels of serum glycans monovalent IgG immune complexes detected by dissociative ELISA in experimental visceral leishmaniasis. Immunology, 158( 4), 314-321. doi:10.1111/imm.13118
NLM
Carvalho CA de, Ferrao TF, Freitas FRN de, Andrade Junior HF de. High levels of serum glycans monovalent IgG immune complexes detected by dissociative ELISA in experimental visceral leishmaniasis [Internet]. Immunology. 2019 ; 158( 4): 314-321.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13118
Vancouver
Carvalho CA de, Ferrao TF, Freitas FRN de, Andrade Junior HF de. High levels of serum glycans monovalent IgG immune complexes detected by dissociative ELISA in experimental visceral leishmaniasis [Internet]. Immunology. 2019 ; 158( 4): 314-321.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13118
Artigo de periodico
The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells (2019)
Melo-Lima, Breno Luiz ; Poras, Isabelle; Passos, Geraldo Aleixo Silva ; Carosella, Edgardo D.; Donadi, Eduardo Antônio ; Moreau, Philippe
Source: Immunology. Unidades: FORP, FMRP
Subjects: TIMO (SISTEMAS SANGUÍNEO E IMUNE), AUTOIMUNIDADE, ANTÍGENOS
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MELO-LIMA, Breno Luiz et al. The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells. Immunology, v. 158, p. 121-135, 2019Tradução . . Disponível em: https://doi.org/10.1111/imm.13099. Acesso em: 24 abr. 2026.
APA
Melo-Lima, B. L., Poras, I., Passos, G. A. S., Carosella, E. D., Donadi, E. A., & Moreau, P. (2019). The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells. Immunology, 158, 121-135. doi:10.1111/imm.13099
NLM
Melo-Lima BL, Poras I, Passos GAS, Carosella ED, Donadi EA, Moreau P. The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells [Internet]. Immunology. 2019 ; 158 121-135.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13099
Vancouver
Melo-Lima BL, Poras I, Passos GAS, Carosella ED, Donadi EA, Moreau P. The Autoimmune Regulator (Aire) transactivatesHLA-Ggeneexpression in thymic epithelial cells [Internet]. Immunology. 2019 ; 158 121-135.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13099
Artigo de periodico
Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model (2019)
Pérez, Malena Martínez ; Martins, Larissa Marfori Sampaio; Dias, Murilo Solano; Pereira, Camila André; Leite, Jefferson Antonio; Gonçalves, Enrico Cerioni Spiropulos; Almeida, Paula Zaghetto de ; Freitas, Emanuelle Neivert de; Tostes, Rita de Cássia Aleixo ; Ramos, Simone Gusmão ; Zoete, Marcel R. de; Ryffel, Bernhard; Silva, João Santana da ; Sartori, Daniela Carlos
Source: Immunology. Unidade: FMRP
Subjects: DOENÇAS METABÓLICAS, INFLAMAÇÃO, OBESIDADE, IMUNOLOGIA, HOMEOSTASE, INTESTINOS
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PÉREZ, Malena Martínez et al. Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model. Immunology, v. 156, n. 4, p. 339-355, 2019Tradução . . Disponível em: https://doi.org/10.1111/imm.13028. Acesso em: 24 abr. 2026.
APA
Pérez, M. M., Martins, L. M. S., Dias, M. S., Pereira, C. A., Leite, J. A., Gonçalves, E. C. S., et al. (2019). Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model. Immunology, 156( 4), 339-355. doi:10.1111/imm.13028
NLM
Pérez MM, Martins LMS, Dias MS, Pereira CA, Leite JA, Gonçalves ECS, Almeida PZ de, Freitas EN de, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Sartori DC. Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model [Internet]. Immunology. 2019 ; 156( 4): 339-355.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13028
Vancouver
Pérez MM, Martins LMS, Dias MS, Pereira CA, Leite JA, Gonçalves ECS, Almeida PZ de, Freitas EN de, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Sartori DC. Interleukin-17/interleukin-17 receptor axis elicits intestinal neutrophil migration, restrains gut dysbiosis and lipopolysaccharide translocation in high-fat diet-induced metabolic syndrome model [Internet]. Immunology. 2019 ; 156( 4): 339-355.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13028
Artigo de periodico
Evaluation of inflammatory skin infiltrate following Aedes aegypti bites in sensitized and non‐sensitized mice reveals saliva‐dependent and immune‐dependent phenotypes (2019)
Henrique, Maressa de Oliveira ; Santos Neto, Leila; Assis, Josiane Betim de ; Barros, Michele Silva de; Capurro, Margareth de Lara ; Lepique, Ana Paula ; Fonseca, Denise Morais da ; Nunes, Anderson de Sá
Source: Immunology. Unidades: ICB, ESALQ
Subjects: AEDES, CAMUNDONGOS, CITOMETRIA DE FLUXO, FENÓTIPOS, PELE DE ANIMAL, IMUNIDADE, INFLAMAÇÃO, PICADAS DE INSETOS, SALIVA
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HENRIQUE, Maressa de Oliveira et al. Evaluation of inflammatory skin infiltrate following Aedes aegypti bites in sensitized and non‐sensitized mice reveals saliva‐dependent and immune‐dependent phenotypes. Immunology, v. 158, n. 1, p. 47-59, 2019Tradução . . Disponível em: https://doi.org/10.1111/imm.13096. Acesso em: 24 abr. 2026.
APA
Henrique, M. de O., Santos Neto, L., Assis, J. B. de, Barros, M. S. de, Capurro, M. de L., Lepique, A. P., et al. (2019). Evaluation of inflammatory skin infiltrate following Aedes aegypti bites in sensitized and non‐sensitized mice reveals saliva‐dependent and immune‐dependent phenotypes. Immunology, 158( 1), 47-59. doi:10.1111/imm.13096
NLM
Henrique M de O, Santos Neto L, Assis JB de, Barros MS de, Capurro M de L, Lepique AP, Fonseca DM da, Nunes A de S. Evaluation of inflammatory skin infiltrate following Aedes aegypti bites in sensitized and non‐sensitized mice reveals saliva‐dependent and immune‐dependent phenotypes [Internet]. Immunology. 2019 ; 158( 1): 47-59.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13096
Vancouver
Henrique M de O, Santos Neto L, Assis JB de, Barros MS de, Capurro M de L, Lepique AP, Fonseca DM da, Nunes A de S. Evaluation of inflammatory skin infiltrate following Aedes aegypti bites in sensitized and non‐sensitized mice reveals saliva‐dependent and immune‐dependent phenotypes [Internet]. Immunology. 2019 ; 158( 1): 47-59.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.13096
Artigo de periodico
Update on aire and thymic negative selection (2018)
Passos Junior, Geraldo Aleixo da Silva; Speck-Hernandez, Cesar A.; Assis, Amanda F.; Mendes-da-Cruz, Daniella A.
Source: Immunology. Unidade: FORP
Subjects: AUTOIMUNIDADE, GENES REGULADORES, TOLERÂNCIA IMUNOLÓGICA, GENÉTICA MOLECULAR
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PASSOS JUNIOR, Geraldo Aleixo da Silva et al. Update on aire and thymic negative selection. Immunology, v. 153, p. 10-20, 2018Tradução . . Disponível em: https://doi.org/10.1111/imm.12831. Acesso em: 24 abr. 2026.
APA
Passos Junior, G. A. da S., Speck-Hernandez, C. A., Assis, A. F., & Mendes-da-Cruz, D. A. (2018). Update on aire and thymic negative selection. Immunology, 153, 10-20. doi:10.1111/imm.12831
NLM
Passos Junior GA da S, Speck-Hernandez CA, Assis AF, Mendes-da-Cruz DA. Update on aire and thymic negative selection [Internet]. Immunology. 2018 ; 153 10-20.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12831
Vancouver
Passos Junior GA da S, Speck-Hernandez CA, Assis AF, Mendes-da-Cruz DA. Update on aire and thymic negative selection [Internet]. Immunology. 2018 ; 153 10-20.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12831
Artigo de periodico
Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model (2018)
Martins, Larissa M. S.; Perez, Malena M.; Pereira, Camila A.; Costa, Frederico R. C.; Dias, Murilo S.; Tostes, Rita de Cássia Aleixo; Ramos, Simone Gusmão; Zoete, Marcel R. de; Ryffel, Bernhard; Silva, João Santana da; Carlos, Daniela
Source: Immunology. Unidade: FMRP
Subjects: INFLAMAÇÃO, LINFÓCITOS, INTERLEUCINAS, DOENÇAS METABÓLICAS, OBESIDADE
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MARTINS, Larissa M. S. et al. Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model. Immunology, v. 154, n. 4, p. 624-636, 2018Tradução . . Disponível em: https://doi.org/10.1111/imm.12946. Acesso em: 24 abr. 2026.
APA
Martins, L. M. S., Perez, M. M., Pereira, C. A., Costa, F. R. C., Dias, M. S., Tostes, R. de C. A., et al. (2018). Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model. Immunology, 154( 4), 624-636. doi:10.1111/imm.12946
NLM
Martins LMS, Perez MM, Pereira CA, Costa FRC, Dias MS, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Carlos D. Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model [Internet]. Immunology. 2018 ; 154( 4): 624-636.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12946
Vancouver
Martins LMS, Perez MM, Pereira CA, Costa FRC, Dias MS, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Carlos D. Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model [Internet]. Immunology. 2018 ; 154( 4): 624-636.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12946
Artigo de periodico
The NLRP3 inflammasome contributes to host protection during Sporothrix schenckii infection (2017)
Gonçalves, Amanda Costa; Ferreira, Lucas Souza; Manente, Francine Alessandra; Faria, Carolina Maria Quinello Gomes de; Polesi, Marisa Campos; Andrade, Cleverton Roberto de; Zamboni, Dario Simões; Carlos, Iracilda Zeppone
Source: Immunology. Unidade: FMRP
Subjects: ESPOROTRICOSE, RECEPTORES
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GONÇALVES, Amanda Costa et al. The NLRP3 inflammasome contributes to host protection during Sporothrix schenckii infection. Immunology, v. 151, n. 2, p. 154-166, 2017Tradução . . Disponível em: https://doi.org/10.1111/imm.12719. Acesso em: 24 abr. 2026.
APA
Gonçalves, A. C., Ferreira, L. S., Manente, F. A., Faria, C. M. Q. G. de, Polesi, M. C., Andrade, C. R. de, et al. (2017). The NLRP3 inflammasome contributes to host protection during Sporothrix schenckii infection. Immunology, 151( 2), 154-166. doi:10.1111/imm.12719
NLM
Gonçalves AC, Ferreira LS, Manente FA, Faria CMQG de, Polesi MC, Andrade CR de, Zamboni DS, Carlos IZ. The NLRP3 inflammasome contributes to host protection during Sporothrix schenckii infection [Internet]. Immunology. 2017 ; 151( 2): 154-166.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12719
Vancouver
Gonçalves AC, Ferreira LS, Manente FA, Faria CMQG de, Polesi MC, Andrade CR de, Zamboni DS, Carlos IZ. The NLRP3 inflammasome contributes to host protection during Sporothrix schenckii infection [Internet]. Immunology. 2017 ; 151( 2): 154-166.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12719
Artigo de periodico
NOD1 in the modulation of host-microbe interactions and inflammatory bone resorption in the periodontal disease model (2016)
Souza, João Antonio Chaves de; Frasnelli, Sabrina Cruz Tfaile; Curylofo-Zotti, Fabiana de Almeida; Ávila-Campos, Mário Júlio; Spolidório, Luis Carlos; Zamboni, Dario Simões; Graves, Dana T.; Rossa Júnior, Carlos
Source: Immunology. Unidades: ICB, FMRP
Subjects: REABSORÇÃO ÓSSEA ALVEOLAR, RELAÇÕES HOSPEDEIRO-PARASITA, INFLAMAÇÃO, IMUNIDADE NATURAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA, João Antonio Chaves de et al. NOD1 in the modulation of host-microbe interactions and inflammatory bone resorption in the periodontal disease model. Immunology, v. 149, p. 374-385, 2016Tradução . . Disponível em: https://doi.org/10.1111/imm.12654. Acesso em: 24 abr. 2026.
APA
Souza, J. A. C. de, Frasnelli, S. C. T., Curylofo-Zotti, F. de A., Ávila-Campos, M. J., Spolidório, L. C., Zamboni, D. S., et al. (2016). NOD1 in the modulation of host-microbe interactions and inflammatory bone resorption in the periodontal disease model. Immunology, 149, 374-385. doi:10.1111/imm.12654
NLM
Souza JAC de, Frasnelli SCT, Curylofo-Zotti F de A, Ávila-Campos MJ, Spolidório LC, Zamboni DS, Graves DT, Rossa Júnior C. NOD1 in the modulation of host-microbe interactions and inflammatory bone resorption in the periodontal disease model [Internet]. Immunology. 2016 ; 149 374-385.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12654
Vancouver
Souza JAC de, Frasnelli SCT, Curylofo-Zotti F de A, Ávila-Campos MJ, Spolidório LC, Zamboni DS, Graves DT, Rossa Júnior C. NOD1 in the modulation of host-microbe interactions and inflammatory bone resorption in the periodontal disease model [Internet]. Immunology. 2016 ; 149 374-385.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12654
Artigo de periodico
Immunosuppressive evidence of Tityus serrulatus toxins Ts6 and Ts15: insights of a novel K+ channel pattern in T cells (2016)
Pucca, Manuela B.; Bertolini, Thaís B.; Cerni, Felipe A.; Bordon, Karla de Castro Figueiredo; Peigneur, Steve; Tygat, Jan; Bonato, Vânia Luiza Deperon; Arantes, Eliane Candiani
Source: Immunology. Unidades: FCFRP, FMRP
Subjects: ESCORPIÕES, TOXINAS, CANAIS DE POTÁSSIO, LINFÓCITOS, MODELOS ANIMAIS DE DOENÇAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PUCCA, Manuela B. et al. Immunosuppressive evidence of Tityus serrulatus toxins Ts6 and Ts15: insights of a novel K+ channel pattern in T cells. Immunology, v. 147, n. 2, p. 240-250, 2016Tradução . . Disponível em: https://doi.org/10.1111/imm.12559. Acesso em: 24 abr. 2026.
APA
Pucca, M. B., Bertolini, T. B., Cerni, F. A., Bordon, K. de C. F., Peigneur, S., Tygat, J., et al. (2016). Immunosuppressive evidence of Tityus serrulatus toxins Ts6 and Ts15: insights of a novel K+ channel pattern in T cells. Immunology, 147( 2), 240-250. doi:10.1111/imm.12559
NLM
Pucca MB, Bertolini TB, Cerni FA, Bordon K de CF, Peigneur S, Tygat J, Bonato VLD, Arantes EC. Immunosuppressive evidence of Tityus serrulatus toxins Ts6 and Ts15: insights of a novel K+ channel pattern in T cells [Internet]. Immunology. 2016 ; 147( 2): 240-250.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12559
Vancouver
Pucca MB, Bertolini TB, Cerni FA, Bordon K de CF, Peigneur S, Tygat J, Bonato VLD, Arantes EC. Immunosuppressive evidence of Tityus serrulatus toxins Ts6 and Ts15: insights of a novel K+ channel pattern in T cells [Internet]. Immunology. 2016 ; 147( 2): 240-250.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12559
Artigo de periodico
Genetic background affects the expansion of macrophage subsets in the lungs of Mycobacterium tuberculosis-infected hosts (2016)
Bertolini, Thais Barboza; Souza, Alexandre Ignacio de; Piñeros, Annie Rocio; Prado, Rafael de Queiroz; Silva, João Santana da; Ramalho, Leandra Naíra Zambelli; Bonato, Vânia Luiza Deperon
Source: Immunology. Unidade: FMRP
Subjects: TUBERCULOSE, INFLAMAÇÃO, ÓXIDO NÍTRICO, MACRÓFAGOS, GENÉTICA, MYCOBACTERIUM TUBERCULOSIS, SISTEMA IMUNE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BERTOLINI, Thais Barboza et al. Genetic background affects the expansion of macrophage subsets in the lungs of Mycobacterium tuberculosis-infected hosts. Immunology, v. 148, p. 102-113, 2016Tradução . . Disponível em: https://doi.org/10.1111/imm.12591. Acesso em: 24 abr. 2026.
APA
Bertolini, T. B., Souza, A. I. de, Piñeros, A. R., Prado, R. de Q., Silva, J. S. da, Ramalho, L. N. Z., & Bonato, V. L. D. (2016). Genetic background affects the expansion of macrophage subsets in the lungs of Mycobacterium tuberculosis-infected hosts. Immunology, 148, 102-113. doi:10.1111/imm.12591
NLM
Bertolini TB, Souza AI de, Piñeros AR, Prado R de Q, Silva JS da, Ramalho LNZ, Bonato VLD. Genetic background affects the expansion of macrophage subsets in the lungs of Mycobacterium tuberculosis-infected hosts [Internet]. Immunology. 2016 ; 148 102-113.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12591
Vancouver
Bertolini TB, Souza AI de, Piñeros AR, Prado R de Q, Silva JS da, Ramalho LNZ, Bonato VLD. Genetic background affects the expansion of macrophage subsets in the lungs of Mycobacterium tuberculosis-infected hosts [Internet]. Immunology. 2016 ; 148 102-113.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12591
Artigo de periodico
CD11c+ CD103+ cells of Mycobacterium tuberculosis-infected C57BL/6 but not of BALB/c mice induce a high frequency of interferon-γ- or interleukin-17-producing CD4+ cells (2015)
Sérgio, Cássia Alves; Bertolini, Thaís Barboza; Gembre, Ana Flávia; Prado, Rafael de Queiroz; Bonato, Vânia Luiza Deperon
Source: Immunology. Unidade: FMRP
Subjects: TUBERCULOSE PULMONAR (IMUNOLOGIA), MYCOBACTERIUM TUBERCULOSIS, PULMÃO, CÉLULAS DENDRÍTICAS, MODELOS ANIMAIS DE DOENÇAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SÉRGIO, Cássia Alves et al. CD11c+ CD103+ cells of Mycobacterium tuberculosis-infected C57BL/6 but not of BALB/c mice induce a high frequency of interferon-γ- or interleukin-17-producing CD4+ cells. Immunology, v. 144, n. 4, p. 574–586, 2015Tradução . . Disponível em: https://doi.org/10.1111/imm.12411. Acesso em: 24 abr. 2026.
APA
Sérgio, C. A., Bertolini, T. B., Gembre, A. F., Prado, R. de Q., & Bonato, V. L. D. (2015). CD11c+ CD103+ cells of Mycobacterium tuberculosis-infected C57BL/6 but not of BALB/c mice induce a high frequency of interferon-γ- or interleukin-17-producing CD4+ cells. Immunology, 144( 4), 574–586. doi:10.1111/imm.12411
NLM
Sérgio CA, Bertolini TB, Gembre AF, Prado R de Q, Bonato VLD. CD11c+ CD103+ cells of Mycobacterium tuberculosis-infected C57BL/6 but not of BALB/c mice induce a high frequency of interferon-γ- or interleukin-17-producing CD4+ cells [Internet]. Immunology. 2015 ; 144( 4): 574–586.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12411
Vancouver
Sérgio CA, Bertolini TB, Gembre AF, Prado R de Q, Bonato VLD. CD11c+ CD103+ cells of Mycobacterium tuberculosis-infected C57BL/6 but not of BALB/c mice induce a high frequency of interferon-γ- or interleukin-17-producing CD4+ cells [Internet]. Immunology. 2015 ; 144( 4): 574–586.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12411
Artigo de periodico
Genome-wide gene expression profiling reveals unsuspected molecular alterations in pemphigus foliaceus (2014)
Malheiros, Danielle; Panepucci, Rodrigo A.; Roselino, Ana Maria Ferreira; Araújo, Amélia de Goes; Zago, Marco Antonio; Petzl-Erler, Maria Luiza
Source: Immunology. Unidade: FMRP
Subjects: PÊNFIGO (GENÉTICA), EXPRESSÃO GÊNICA, LINFÓCITOS, AUTOIMUNIDADE, ESTUDOS DE CASOS E CONTROLES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MALHEIROS, Danielle et al. Genome-wide gene expression profiling reveals unsuspected molecular alterations in pemphigus foliaceus. Immunology, v. 143, n. 3, p. 381-395, 2014Tradução . . Disponível em: https://doi.org/10.1111/imm.12315. Acesso em: 24 abr. 2026.
APA
Malheiros, D., Panepucci, R. A., Roselino, A. M. F., Araújo, A. de G., Zago, M. A., & Petzl-Erler, M. L. (2014). Genome-wide gene expression profiling reveals unsuspected molecular alterations in pemphigus foliaceus. Immunology, 143( 3), 381-395. doi:10.1111/imm.12315
NLM
Malheiros D, Panepucci RA, Roselino AMF, Araújo A de G, Zago MA, Petzl-Erler ML. Genome-wide gene expression profiling reveals unsuspected molecular alterations in pemphigus foliaceus [Internet]. Immunology. 2014 ; 143( 3): 381-395.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12315
Vancouver
Malheiros D, Panepucci RA, Roselino AMF, Araújo A de G, Zago MA, Petzl-Erler ML. Genome-wide gene expression profiling reveals unsuspected molecular alterations in pemphigus foliaceus [Internet]. Immunology. 2014 ; 143( 3): 381-395.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/imm.12315
Artigo de periodico
Lower numbers of natural killer T cells in HIV-1 and mycobacterium leprae co-infected patients (2012)
Carvalho, Karina I.; Bruno, Fernanda R.; Cappione, Jennifer E. Snyder; Maeda, Solange M.; Tomimori, Jane; Xavier, Marilia B.; Haslett, Patrick A.; Nixon, Douglas F.; Kallas, Esper G.
Source: Immunology. Unidade: FM
Subjects: HANSENÍASE, MYCOBACTERIUM LEPRAE, INTERFERONS, INFECÇÕES POR HIV, MORBIDADDE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO, Karina I. et al. Lower numbers of natural killer T cells in HIV-1 and mycobacterium leprae co-infected patients. Immunology, v. 136, n. 3, p. 38-46, 2012Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2567.2012.03563.x. Acesso em: 24 abr. 2026.
APA
Carvalho, K. I., Bruno, F. R., Cappione, J. E. S., Maeda, S. M., Tomimori, J., Xavier, M. B., et al. (2012). Lower numbers of natural killer T cells in HIV-1 and mycobacterium leprae co-infected patients. Immunology, 136( 3), 38-46. doi:10.1111/j.1365-2567.2012.03563.x
NLM
Carvalho KI, Bruno FR, Cappione JES, Maeda SM, Tomimori J, Xavier MB, Haslett PA, Nixon DF, Kallas EG. Lower numbers of natural killer T cells in HIV-1 and mycobacterium leprae co-infected patients [Internet]. Immunology. 2012 ; 136( 3): 38-46.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/j.1365-2567.2012.03563.x
Vancouver
Carvalho KI, Bruno FR, Cappione JES, Maeda SM, Tomimori J, Xavier MB, Haslett PA, Nixon DF, Kallas EG. Lower numbers of natural killer T cells in HIV-1 and mycobacterium leprae co-infected patients [Internet]. Immunology. 2012 ; 136( 3): 38-46.[citado 2026 abr. 24 ] Available from: https://doi.org/10.1111/j.1365-2567.2012.03563.x
Trabalho de evento-resumo periodico
Immunosenescence and dendritic cells: role of TLR2 and TLR4 in phagocytosis and cytokines production against candida albicans (2012)
Nascimento, M. P. P.; Lara, Vanessa Soares
Source: Immunology. Conference titles: European Congress of Immunology. Unidade: FOB
Subjects: IMUNOLOGIA, CÉLULAS DENDRÍTICAS, FAGOCITOSE, CITOCINAS, CANDIDA ALBICANS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NASCIMENTO, M. P. P. e LARA, Vanessa Soares. Immunosenescence and dendritic cells: role of TLR2 and TLR4 in phagocytosis and cytokines production against candida albicans. Immunology. Oxford: Faculdade de Odontologia de Bauru, Universidade de São Paulo. . Acesso em: 24 abr. 2026. , 2012
APA
Nascimento, M. P. P., & Lara, V. S. (2012). Immunosenescence and dendritic cells: role of TLR2 and TLR4 in phagocytosis and cytokines production against candida albicans. Immunology. Oxford: Faculdade de Odontologia de Bauru, Universidade de São Paulo.
NLM
Nascimento MPP, Lara VS. Immunosenescence and dendritic cells: role of TLR2 and TLR4 in phagocytosis and cytokines production against candida albicans. Immunology. 2012 ; 137 475.[citado 2026 abr. 24 ]
Vancouver
Nascimento MPP, Lara VS. Immunosenescence and dendritic cells: role of TLR2 and TLR4 in phagocytosis and cytokines production against candida albicans. Immunology. 2012 ; 137 475.[citado 2026 abr. 24 ]
Trabalho de evento-resumo periodico
TNFa modulates chemokine expression and leukocyte influx during alveolar bone repair process in mice (2012)
Vieira, Andreia Espindola; Repeke, Carlos Eduardo; Silveira, Elcia Maria; Biguetti, Claudia C.; Franchiscone, C. F.; Campanelli, Ana Paula; Trombone, A. P. F.; Assis, Gérson Francisco de; Taga, Rumio; Garlet, Gustavo Pompermaier
Source: Immunology. Conference titles: European Congress of Immunology. Unidade: FOB
Subjects: IMUNOLOGIA, LEUCÓCITOS, CITOCINAS, REPARAÇÃO ALVEOLAR, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VIEIRA, Andreia Espindola et al. TNFa modulates chemokine expression and leukocyte influx during alveolar bone repair process in mice. Immunology. Oxford: Faculdade de Odontologia de Bauru, Universidade de São Paulo. . Acesso em: 24 abr. 2026. , 2012
APA
Vieira, A. E., Repeke, C. E., Silveira, E. M., Biguetti, C. C., Franchiscone, C. F., Campanelli, A. P., et al. (2012). TNFa modulates chemokine expression and leukocyte influx during alveolar bone repair process in mice. Immunology. Oxford: Faculdade de Odontologia de Bauru, Universidade de São Paulo.
NLM
Vieira AE, Repeke CE, Silveira EM, Biguetti CC, Franchiscone CF, Campanelli AP, Trombone APF, Assis GF de, Taga R, Garlet GP. TNFa modulates chemokine expression and leukocyte influx during alveolar bone repair process in mice. Immunology. 2012 ; 137 194.[citado 2026 abr. 24 ]
Vancouver
Vieira AE, Repeke CE, Silveira EM, Biguetti CC, Franchiscone CF, Campanelli AP, Trombone APF, Assis GF de, Taga R, Garlet GP. TNFa modulates chemokine expression and leukocyte influx during alveolar bone repair process in mice. Immunology. 2012 ; 137 194.[citado 2026 abr. 24 ]

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