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Artigo de periodico
Sympathetic Neural Overdrive, Aortic Stiffening, Endothelial Dysfunction, and Impaired Exercise Capacity in Severe COVID-19 Survivors: A Mid-Term Study of Cardiovascular Sequelae (2023)
Faria, Diego ; Moll-Bernardes, Renata J ; Testa, Laura; Moniz, Camila M. V ; Rodrigues, Erika C; Rodrigues, Amanda G ; Araujo, Amanda; Alves, Maria J. N. N ; Ono, Bruna E ; Izaias, João E ; Salemi, Vera M. C ; Jordão Camila P ; Amaro-Vicente, Graziela ; Rondon, Maria Urbana Pinto Brandão ; Ludwig, Katelyn R; Craighead, Daniel H ; Rossman, Matthew J ; Consolim-Colombo, Fernanda M; Angelis, Katia De ; Irigoyen, Maria C. C ; Seals, Douglas R ; Negrão, Carlos Eduardo ; Sales, Allan R. K
Source: Hypertension. Unidades: FM, EEFE
Subjects: RIGIDEZ VASCULAR, ENDOTÉLIO, ESTRESSE OXIDATIVO, COVID-19, CAPACIDADE FUNCIONAL, SISTEMA NERVOSO SIMPÁTICO, EXERCÍCIO FÍSICO
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ABNT
FARIA, Diego et al. Sympathetic Neural Overdrive, Aortic Stiffening, Endothelial Dysfunction, and Impaired Exercise Capacity in Severe COVID-19 Survivors: A Mid-Term Study of Cardiovascular Sequelae. Hypertension, v. 80, n. 2, p. 470-481, 2023Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.122.19958. Acesso em: 02 nov. 2024.
APA
Faria, D., Moll-Bernardes, R. J., Testa, L., Moniz, C. M. V., Rodrigues, E. C., Rodrigues, A. G., et al. (2023). Sympathetic Neural Overdrive, Aortic Stiffening, Endothelial Dysfunction, and Impaired Exercise Capacity in Severe COVID-19 Survivors: A Mid-Term Study of Cardiovascular Sequelae. Hypertension, 80( 2), 470-481. doi:10.1161/HYPERTENSIONAHA.122.19958
NLM
Faria D, Moll-Bernardes RJ, Testa L, Moniz CMV, Rodrigues EC, Rodrigues AG, Araujo A, Alves MJNN, Ono BE, Izaias JE, Salemi VMC, Jordão Camila P, Amaro-Vicente G, Rondon MUPB, Ludwig KR, Craighead DH, Rossman MJ, Consolim-Colombo FM, Angelis KD, Irigoyen MCC, Seals DR, Negrão CE, Sales ARK. Sympathetic Neural Overdrive, Aortic Stiffening, Endothelial Dysfunction, and Impaired Exercise Capacity in Severe COVID-19 Survivors: A Mid-Term Study of Cardiovascular Sequelae [Internet]. Hypertension. 2023 ; 80( 2): 470-481.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.122.19958
Vancouver
Faria D, Moll-Bernardes RJ, Testa L, Moniz CMV, Rodrigues EC, Rodrigues AG, Araujo A, Alves MJNN, Ono BE, Izaias JE, Salemi VMC, Jordão Camila P, Amaro-Vicente G, Rondon MUPB, Ludwig KR, Craighead DH, Rossman MJ, Consolim-Colombo FM, Angelis KD, Irigoyen MCC, Seals DR, Negrão CE, Sales ARK. Sympathetic Neural Overdrive, Aortic Stiffening, Endothelial Dysfunction, and Impaired Exercise Capacity in Severe COVID-19 Survivors: A Mid-Term Study of Cardiovascular Sequelae [Internet]. Hypertension. 2023 ; 80( 2): 470-481.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.122.19958
Artigo de periodico
Crosstalk between vascular redox and calcium signaling in hypertension involves TRPM2 (Transient Receptor Potential Melastatin 2) cation channel (2020)
Alves-Lopes, Rhéure ; Neves, Karla B. ; Anagnostopoulou, Aikaterini ; Rios, Francisco J. ; Montenzano, Augusto C.; Touyz, Rhian M. ; Lacchini, Silvia
Source: Hypertension. Unidade: ICB
Subjects: CANAIS DE CÁLCIO, HIPERTENSÃO, CÉLULAS MUSCULARES, OXIGÊNIO, CAMUNDONGOS, INIBIDORES DE ENZIMAS, CATALASE, ANGIOTENSINA II
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ABNT
ALVES-LOPES, Rhéure et al. Crosstalk between vascular redox and calcium signaling in hypertension involves TRPM2 (Transient Receptor Potential Melastatin 2) cation channel. Hypertension, v. 75, n. 1, p. 139-149, 2020Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.119.13861. Acesso em: 02 nov. 2024.
APA
Alves-Lopes, R., Neves, K. B., Anagnostopoulou, A., Rios, F. J., Montenzano, A. C., Touyz, R. M., & Lacchini, S. (2020). Crosstalk between vascular redox and calcium signaling in hypertension involves TRPM2 (Transient Receptor Potential Melastatin 2) cation channel. Hypertension, 75( 1), 139-149. doi:10.1161/HYPERTENSIONAHA.119.13861
NLM
Alves-Lopes R, Neves KB, Anagnostopoulou A, Rios FJ, Montenzano AC, Touyz RM, Lacchini S. Crosstalk between vascular redox and calcium signaling in hypertension involves TRPM2 (Transient Receptor Potential Melastatin 2) cation channel [Internet]. Hypertension. 2020 ; 75( 1): 139-149.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.119.13861
Vancouver
Alves-Lopes R, Neves KB, Anagnostopoulou A, Rios FJ, Montenzano AC, Touyz RM, Lacchini S. Crosstalk between vascular redox and calcium signaling in hypertension involves TRPM2 (Transient Receptor Potential Melastatin 2) cation channel [Internet]. Hypertension. 2020 ; 75( 1): 139-149.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.119.13861
Artigo de periodico
Modeling pathways from the perinatal factors to the vascular risk phenotype at the end of the second decade of life: birth cohort, Brazil (2020)
Amaral, Marconi Satuf; Ribeiro, Cecília Cláudia Costa; Alves, Maria Teresa Seabra de Brito e; Ribeiro, Maria Jacqueline Silva; Nascimento, Joelma Ximenes Prado Teixeira; Simões, Vanda Maria Ferreira; Ferraro, Alexandre Archanjo ; Barbieri, Marco Antonio; Silva, Antônio Augusto Moura da
Source: Hypertension. Unidades: FM, FMRP
Subjects: ADOLESCENTES, PRESSÃO SANGUÍNEA, ÍNDICE DE MASSA CORPORAL, DOENÇAS CARDIOVASCULARES, IDADE GESTACIONAL, RECÉM-NASCIDO, OBESIDADE INFANTIL, FATORES SOCIOECONÔMICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AMARAL, Marconi Satuf et al. Modeling pathways from the perinatal factors to the vascular risk phenotype at the end of the second decade of life: birth cohort, Brazil. Hypertension, v. 76, n. 2, p. 359-365, 2020Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.119.14218. Acesso em: 02 nov. 2024.
APA
Amaral, M. S., Ribeiro, C. C. C., Alves, M. T. S. de B. e, Ribeiro, M. J. S., Nascimento, J. X. P. T., Simões, V. M. F., et al. (2020). Modeling pathways from the perinatal factors to the vascular risk phenotype at the end of the second decade of life: birth cohort, Brazil. Hypertension, 76( 2), 359-365. doi:10.1161/HYPERTENSIONAHA.119.14218
NLM
Amaral MS, Ribeiro CCC, Alves MTS de B e, Ribeiro MJS, Nascimento JXPT, Simões VMF, Ferraro AA, Barbieri MA, Silva AAM da. Modeling pathways from the perinatal factors to the vascular risk phenotype at the end of the second decade of life: birth cohort, Brazil [Internet]. Hypertension. 2020 ; 76( 2): 359-365.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.119.14218
Vancouver
Amaral MS, Ribeiro CCC, Alves MTS de B e, Ribeiro MJS, Nascimento JXPT, Simões VMF, Ferraro AA, Barbieri MA, Silva AAM da. Modeling pathways from the perinatal factors to the vascular risk phenotype at the end of the second decade of life: birth cohort, Brazil [Internet]. Hypertension. 2020 ; 76( 2): 359-365.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.119.14218
Artigo de periodico
Does obstructive sleep apnea influence blood pressure and arterial stiffness in response to antihypertensive treatment? (2018)
Fatureto-borges, Fernanda; Drager, Luciano Ferreira
Source: Hypertension. Unidade: FM
Subjects: HIPERTENSÃO, PRESSÃO SANGUÍNEA, APNEIA DO SONO, MONITORIZAÇÃO AMBULATORIAL DA PRESSÃO ARTERIAL
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ABNT
FATURETO-BORGES, Fernanda e DRAGER, Luciano Ferreira. Does obstructive sleep apnea influence blood pressure and arterial stiffness in response to antihypertensive treatment?. Hypertension, v. 72, n. 2, p. 399-407, 2018Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.118.10825. Acesso em: 02 nov. 2024.
APA
Fatureto-borges, F., & Drager, L. F. (2018). Does obstructive sleep apnea influence blood pressure and arterial stiffness in response to antihypertensive treatment? Hypertension, 72( 2), 399-407. doi:10.1161/HYPERTENSIONAHA.118.10825
NLM
Fatureto-borges F, Drager LF. Does obstructive sleep apnea influence blood pressure and arterial stiffness in response to antihypertensive treatment? [Internet]. Hypertension. 2018 ; 72( 2): 399-407.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.118.10825
Vancouver
Fatureto-borges F, Drager LF. Does obstructive sleep apnea influence blood pressure and arterial stiffness in response to antihypertensive treatment? [Internet]. Hypertension. 2018 ; 72( 2): 399-407.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.118.10825
Artigo de periodico
Nondipping blood pressure patterns predict obstructive sleep apnea in patients undergoing ambulatory blood pressure monitoring (2018)
Genta-pereira, Daniel Castanho; Lorenzi-filho, Geraldo; Drager, Luciano F; Furlan, Sofia F; Omote, Daniel Q; Giorgi, Dante M. A; Bortolotto, Luiz A
Source: Hypertension. Unidade: FM
Subjects: APNEIA DO SONO TIPO OBSTRUTIVA, HIPERTENSÃO, TRANSTORNOS DO SONO, MONITORIZAÇÃO AMBULATORIAL DA PRESSÃO ARTERIAL
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ABNT
GENTA-PEREIRA, Daniel Castanho et al. Nondipping blood pressure patterns predict obstructive sleep apnea in patients undergoing ambulatory blood pressure monitoring. Hypertension, v. 72, n. 4, p. 979-985, 2018Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.118.11525. Acesso em: 02 nov. 2024.
APA
Genta-pereira, D. C., Lorenzi-filho, G., Drager, L. F., Furlan, S. F., Omote, D. Q., Giorgi, D. M. A., & Bortolotto, L. A. (2018). Nondipping blood pressure patterns predict obstructive sleep apnea in patients undergoing ambulatory blood pressure monitoring. Hypertension, 72( 4), 979-985. doi:10.1161/HYPERTENSIONAHA.118.11525
NLM
Genta-pereira DC, Lorenzi-filho G, Drager LF, Furlan SF, Omote DQ, Giorgi DMA, Bortolotto LA. Nondipping blood pressure patterns predict obstructive sleep apnea in patients undergoing ambulatory blood pressure monitoring [Internet]. Hypertension. 2018 ; 72( 4): 979-985.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.118.11525
Vancouver
Genta-pereira DC, Lorenzi-filho G, Drager LF, Furlan SF, Omote DQ, Giorgi DMA, Bortolotto LA. Nondipping blood pressure patterns predict obstructive sleep apnea in patients undergoing ambulatory blood pressure monitoring [Internet]. Hypertension. 2018 ; 72( 4): 979-985.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.118.11525
Artigo de periodico
Spironolactone Versus Clonidine as a Fourth-Drug Therapy for Resistant Hypertension: The ReHOT Randomized Study (Resistant Hypertension Optimal Treatment) (2018)
Krieger, Eduardo M.; Drager, Luciano F.; Lotufo, Paulo A.; Krieger, Jose E.
Source: Hypertension. Unidade: FM
Subjects: HIPERTENSÃO, MONITORIZAÇÃO AMBULATORIAL DA PRESSÃO ARTERIAL, FÁRMACOS DO SANGUE E SISTEMA HEMATOPOÉTICO
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ABNT
KRIEGER, Eduardo M. et al. Spironolactone Versus Clonidine as a Fourth-Drug Therapy for Resistant Hypertension: The ReHOT Randomized Study (Resistant Hypertension Optimal Treatment). Hypertension, v. 71, n. 4, p. 681-690, 2018Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.117.10662. Acesso em: 02 nov. 2024.
APA
Krieger, E. M., Drager, L. F., Lotufo, P. A., & Krieger, J. E. (2018). Spironolactone Versus Clonidine as a Fourth-Drug Therapy for Resistant Hypertension: The ReHOT Randomized Study (Resistant Hypertension Optimal Treatment). Hypertension, 71( 4), 681-690. doi:10.1161/HYPERTENSIONAHA.117.10662
NLM
Krieger EM, Drager LF, Lotufo PA, Krieger JE. Spironolactone Versus Clonidine as a Fourth-Drug Therapy for Resistant Hypertension: The ReHOT Randomized Study (Resistant Hypertension Optimal Treatment) [Internet]. Hypertension. 2018 ; 71( 4): 681-690.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.117.10662
Vancouver
Krieger EM, Drager LF, Lotufo PA, Krieger JE. Spironolactone Versus Clonidine as a Fourth-Drug Therapy for Resistant Hypertension: The ReHOT Randomized Study (Resistant Hypertension Optimal Treatment) [Internet]. Hypertension. 2018 ; 71( 4): 681-690.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.117.10662
Trabalho de evento-resumo periodico
A new technique for selective denervation of the aortic and carotid baroreceptors (2017)
Castania, Jaci Airton; Fazan Júnior, Rubens; Salgado, Hélio César; Katayama, Pedro L.; Brognara, Fernanda; Sabino, João P.
Source: Hypertension. Conference titles: American Heart Association's Council on Hypertension. Unidade: FMRP
Subjects: AORTA, PRESSORECEPTORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CASTANIA, Jaci Airton et al. A new technique for selective denervation of the aortic and carotid baroreceptors. Hypertension. Dallas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 02 nov. 2024. , 2017
APA
Castania, J. A., Fazan Júnior, R., Salgado, H. C., Katayama, P. L., Brognara, F., & Sabino, J. P. (2017). A new technique for selective denervation of the aortic and carotid baroreceptors. Hypertension. Dallas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Castania JA, Fazan Júnior R, Salgado HC, Katayama PL, Brognara F, Sabino JP. A new technique for selective denervation of the aortic and carotid baroreceptors. Hypertension. 2017 ; 70[citado 2024 nov. 02 ]
Vancouver
Castania JA, Fazan Júnior R, Salgado HC, Katayama PL, Brognara F, Sabino JP. A new technique for selective denervation of the aortic and carotid baroreceptors. Hypertension. 2017 ; 70[citado 2024 nov. 02 ]
Artigo de periodico-carta/editorial
Mean heart rate level does not affect all heart rate variability indices (2017)
Silva, Luiz Eduardo Virgilio; Salgado, Helio Cesar; Fazan Júnior, Rubens
Source: Hypertension. Unidades: ICMC, FMRP
Subjects: FREQUÊNCIA CARDÍACA, HIPERTENSÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Luiz Eduardo Virgilio e SALGADO, Helio Cesar e FAZAN JÚNIOR, Rubens. Mean heart rate level does not affect all heart rate variability indices. Hypertension. Philadelphia: Instituto de Ciências Matemáticas e de Computação, Universidade de São Paulo. Disponível em: https://doi.org/10.1161/hypertensionaha.117.09060. Acesso em: 02 nov. 2024. , 2017
APA
Silva, L. E. V., Salgado, H. C., & Fazan Júnior, R. (2017). Mean heart rate level does not affect all heart rate variability indices. Hypertension. Philadelphia: Instituto de Ciências Matemáticas e de Computação, Universidade de São Paulo. doi:10.1161/hypertensionaha.117.09060
NLM
Silva LEV, Salgado HC, Fazan Júnior R. Mean heart rate level does not affect all heart rate variability indices [Internet]. Hypertension. 2017 ; 69( 5): e21-e22.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/hypertensionaha.117.09060
Vancouver
Silva LEV, Salgado HC, Fazan Júnior R. Mean heart rate level does not affect all heart rate variability indices [Internet]. Hypertension. 2017 ; 69( 5): e21-e22.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/hypertensionaha.117.09060
Artigo de periodico
Myeloperoxidase in hypertensive disorders of pregnancy and its relation with nitric oxide (2017)
Rocha-Penha, Liliam; Dias, Mayara Caldeira; Tanus-Santos, José Eduardo; Cavalli, Ricardo de Carvalho; Sandrim, Valéria Cristina
Source: Hypertension. Unidade: FMRP
Subjects: ANÁLISE DE VARIÂNCIA, BIOMARCADORES, HIPERTENSÃO NA GRAVIDEZ, ÓXIDO NÍTRICO, CÉLULAS ENDOTELIAIS, CORDÃO UMBILICAL
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ABNT
ROCHA-PENHA, Liliam et al. Myeloperoxidase in hypertensive disorders of pregnancy and its relation with nitric oxide. Hypertension, v. 69, n. 6, p. 1173-1180, 2017Tradução . . Disponível em: https://doi.org/10.1161/hypertensionaha.116.08854. Acesso em: 02 nov. 2024.
APA
Rocha-Penha, L., Dias, M. C., Tanus-Santos, J. E., Cavalli, R. de C., & Sandrim, V. C. (2017). Myeloperoxidase in hypertensive disorders of pregnancy and its relation with nitric oxide. Hypertension, 69( 6), 1173-1180. doi:10.1161/hypertensionaha.116.08854
NLM
Rocha-Penha L, Dias MC, Tanus-Santos JE, Cavalli R de C, Sandrim VC. Myeloperoxidase in hypertensive disorders of pregnancy and its relation with nitric oxide [Internet]. Hypertension. 2017 ; 69( 6): 1173-1180.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/hypertensionaha.116.08854
Vancouver
Rocha-Penha L, Dias MC, Tanus-Santos JE, Cavalli R de C, Sandrim VC. Myeloperoxidase in hypertensive disorders of pregnancy and its relation with nitric oxide [Internet]. Hypertension. 2017 ; 69( 6): 1173-1180.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/hypertensionaha.116.08854
Trabalho de evento-resumo periodico
Mmp-2/timp-2 imbalance and increased collagen production are crucial to the transition from compensated cardiac hypertrophy to heart failure in rats subjected to abdominal aorta constriction (2017)
Prado, Cibele Maria; Pizzo, Thayane R.; Fernandes, Aline; Amorim, Vanessa A.; Rizzi, Ellen; Meirelles, Flávio Vieira; Salgado, Hélio César
Source: Hypertension. Conference titles: American Heart Association's Council on Hypertension. Unidades: FMRP, FZEA
Subjects: HIPERTENSÃO, METALOPROTEINASES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PRADO, Cibele Maria et al. Mmp-2/timp-2 imbalance and increased collagen production are crucial to the transition from compensated cardiac hypertrophy to heart failure in rats subjected to abdominal aorta constriction. Hypertension. Dallas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 02 nov. 2024. , 2017
APA
Prado, C. M., Pizzo, T. R., Fernandes, A., Amorim, V. A., Rizzi, E., Meirelles, F. V., & Salgado, H. C. (2017). Mmp-2/timp-2 imbalance and increased collagen production are crucial to the transition from compensated cardiac hypertrophy to heart failure in rats subjected to abdominal aorta constriction. Hypertension. Dallas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Prado CM, Pizzo TR, Fernandes A, Amorim VA, Rizzi E, Meirelles FV, Salgado HC. Mmp-2/timp-2 imbalance and increased collagen production are crucial to the transition from compensated cardiac hypertrophy to heart failure in rats subjected to abdominal aorta constriction. Hypertension. 2017 ; 70[citado 2024 nov. 02 ]
Vancouver
Prado CM, Pizzo TR, Fernandes A, Amorim VA, Rizzi E, Meirelles FV, Salgado HC. Mmp-2/timp-2 imbalance and increased collagen production are crucial to the transition from compensated cardiac hypertrophy to heart failure in rats subjected to abdominal aorta constriction. Hypertension. 2017 ; 70[citado 2024 nov. 02 ]
Trabalho de evento-resumo periodico
Two-kidney one clip (2K1c) hypertension reduces complexity of heart rate variability in mice (2017)
Rodrigues, Fernanda L.; Silva, Luiz E.; Silva, Carlos A.; Carneiro, Fernando Silva; Tostes, Rita de Cássia Aleixo; Fazan Júnior, Rubens; Salgado, Hélio César
Source: Hypertension. Conference titles: American Heart Association's Council on Hypertension. Unidade: FMRP
Subjects: HIPERTENSÃO, FÁRMACOS (SISTEMA CARDIOVASCULAR)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRIGUES, Fernanda L. et al. Two-kidney one clip (2K1c) hypertension reduces complexity of heart rate variability in mice. Hypertension. Dallas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 02 nov. 2024. , 2017
APA
Rodrigues, F. L., Silva, L. E., Silva, C. A., Carneiro, F. S., Tostes, R. de C. A., Fazan Júnior, R., & Salgado, H. C. (2017). Two-kidney one clip (2K1c) hypertension reduces complexity of heart rate variability in mice. Hypertension. Dallas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Rodrigues FL, Silva LE, Silva CA, Carneiro FS, Tostes R de CA, Fazan Júnior R, Salgado HC. Two-kidney one clip (2K1c) hypertension reduces complexity of heart rate variability in mice. Hypertension. 2017 ; 70[citado 2024 nov. 02 ]
Vancouver
Rodrigues FL, Silva LE, Silva CA, Carneiro FS, Tostes R de CA, Fazan Júnior R, Salgado HC. Two-kidney one clip (2K1c) hypertension reduces complexity of heart rate variability in mice. Hypertension. 2017 ; 70[citado 2024 nov. 02 ]
Trabalho de evento-resumo periodico
Acetylcholine stimulation with pyridostigmine prevents cardiac arrhythmias in conscious myocardial infarcted rats and upregulate connexin 43, hipoxic inducible factor-1α and vascular endothelial grow factor in cardiomyocytes culture (2016)
Santos-Almeida, Fernanda Machado; Meschiari, César A.; Martins-Marques, Tânia; Cury-Pavão, Maria L.; Salgado, Hélio César; Girão, Henrique; Fazan Júnior, Rubens
Source: Hypertension. Conference titles: Council on Hypertension Scientific Session. Unidade: FMRP
Subjects: INFARTO DO MIOCÁRDIO, ACETILCOLINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS-ALMEIDA, Fernanda Machado et al. Acetylcholine stimulation with pyridostigmine prevents cardiac arrhythmias in conscious myocardial infarcted rats and upregulate connexin 43, hipoxic inducible factor-1α and vascular endothelial grow factor in cardiomyocytes culture. Hypertension. Dallas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 02 nov. 2024. , 2016
APA
Santos-Almeida, F. M., Meschiari, C. A., Martins-Marques, T., Cury-Pavão, M. L., Salgado, H. C., Girão, H., & Fazan Júnior, R. (2016). Acetylcholine stimulation with pyridostigmine prevents cardiac arrhythmias in conscious myocardial infarcted rats and upregulate connexin 43, hipoxic inducible factor-1α and vascular endothelial grow factor in cardiomyocytes culture. Hypertension. Dallas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Santos-Almeida FM, Meschiari CA, Martins-Marques T, Cury-Pavão ML, Salgado HC, Girão H, Fazan Júnior R. Acetylcholine stimulation with pyridostigmine prevents cardiac arrhythmias in conscious myocardial infarcted rats and upregulate connexin 43, hipoxic inducible factor-1α and vascular endothelial grow factor in cardiomyocytes culture. Hypertension. 2016 ; 68[citado 2024 nov. 02 ]
Vancouver
Santos-Almeida FM, Meschiari CA, Martins-Marques T, Cury-Pavão ML, Salgado HC, Girão H, Fazan Júnior R. Acetylcholine stimulation with pyridostigmine prevents cardiac arrhythmias in conscious myocardial infarcted rats and upregulate connexin 43, hipoxic inducible factor-1α and vascular endothelial grow factor in cardiomyocytes culture. Hypertension. 2016 ; 68[citado 2024 nov. 02 ]
Trabalho de evento-resumo periodico
Sympathetic activation elicited by bilateral carotid occlusion attenuates cytokine release in conscious endotoxemic rats (2016)
Brognara, Fernanda; Castania, Jaci Airton; Dias, Daniel P.; Fazan Júnior, Rubens; Cunha, Fernando de Queiroz; Patel, Kaushik P.; Ulloa, Luis; Kanashiro, Alexandre; Salgado, Hélio César
Source: Hypertension. Conference titles: Council on Hypertension Scientific Session. Unidade: FMRP
Subjects: INFLAMAÇÃO, CITOCINAS, SISTEMA NERVOSO AUTÔNOMO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BROGNARA, Fernanda et al. Sympathetic activation elicited by bilateral carotid occlusion attenuates cytokine release in conscious endotoxemic rats. Hypertension. Dallas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 02 nov. 2024. , 2016
APA
Brognara, F., Castania, J. A., Dias, D. P., Fazan Júnior, R., Cunha, F. de Q., Patel, K. P., et al. (2016). Sympathetic activation elicited by bilateral carotid occlusion attenuates cytokine release in conscious endotoxemic rats. Hypertension. Dallas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Brognara F, Castania JA, Dias DP, Fazan Júnior R, Cunha F de Q, Patel KP, Ulloa L, Kanashiro A, Salgado HC. Sympathetic activation elicited by bilateral carotid occlusion attenuates cytokine release in conscious endotoxemic rats. Hypertension. 2016 ; 68[citado 2024 nov. 02 ]
Vancouver
Brognara F, Castania JA, Dias DP, Fazan Júnior R, Cunha F de Q, Patel KP, Ulloa L, Kanashiro A, Salgado HC. Sympathetic activation elicited by bilateral carotid occlusion attenuates cytokine release in conscious endotoxemic rats. Hypertension. 2016 ; 68[citado 2024 nov. 02 ]
Trabalho de evento-resumo periodico
Is ischemia/reperfusion an efficient method for producing heart failure in rats ? (2016)
Omoto, Ana Carolina M.; Gava, Fábio N.; Oliveira, Mauro de; Silva, Carlos A.; Fazan Júnior, Rubens; Salgado, Hélio César
Source: Hypertension. Conference titles: Council on Hypertension Scientific Session. Unidade: FMRP
Subjects: REPERFUSÃO, INSUFICIÊNCIA CARDÍACA, INFARTO DO MIOCÁRDIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OMOTO, Ana Carolina M. et al. Is ischemia/reperfusion an efficient method for producing heart failure in rats ?. Hypertension. Dallas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 02 nov. 2024. , 2016
APA
Omoto, A. C. M., Gava, F. N., Oliveira, M. de, Silva, C. A., Fazan Júnior, R., & Salgado, H. C. (2016). Is ischemia/reperfusion an efficient method for producing heart failure in rats ? Hypertension. Dallas: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Omoto ACM, Gava FN, Oliveira M de, Silva CA, Fazan Júnior R, Salgado HC. Is ischemia/reperfusion an efficient method for producing heart failure in rats ? Hypertension. 2016 ; 68[citado 2024 nov. 02 ]
Vancouver
Omoto ACM, Gava FN, Oliveira M de, Silva CA, Fazan Júnior R, Salgado HC. Is ischemia/reperfusion an efficient method for producing heart failure in rats ? Hypertension. 2016 ; 68[citado 2024 nov. 02 ]
Artigo de periodico
Respiratory network enhances the sympathoinhibitory component of baroreflex of rats submitted to chronic intermittent hypoxia (2016)
Moraes, Davi José de Almeida; Bonagamba, Leni Gomes Heck; Silva, Melina Pires da; Mecawi, André S.; Antunes-Rodrigues, José; Machado, Benedito Honório
Source: Hypertension. Unidade: FMRP
Subjects: PRESSÃO SANGUÍNEA, HIPERTENSÃO, RESPIRAÇÃO, SISTEMA NERVOSO SIMPÁTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MORAES, Davi José de Almeida et al. Respiratory network enhances the sympathoinhibitory component of baroreflex of rats submitted to chronic intermittent hypoxia. Hypertension, v. 68, n. 4, p. 1021-1030, 2016Tradução . . Disponível em: https://doi.org/10.1161/hypertensionaha.116.07731. Acesso em: 02 nov. 2024.
APA
Moraes, D. J. de A., Bonagamba, L. G. H., Silva, M. P. da, Mecawi, A. S., Antunes-Rodrigues, J., & Machado, B. H. (2016). Respiratory network enhances the sympathoinhibitory component of baroreflex of rats submitted to chronic intermittent hypoxia. Hypertension, 68( 4), 1021-1030. doi:10.1161/hypertensionaha.116.07731
NLM
Moraes DJ de A, Bonagamba LGH, Silva MP da, Mecawi AS, Antunes-Rodrigues J, Machado BH. Respiratory network enhances the sympathoinhibitory component of baroreflex of rats submitted to chronic intermittent hypoxia [Internet]. Hypertension. 2016 ; 68( 4): 1021-1030.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/hypertensionaha.116.07731
Vancouver
Moraes DJ de A, Bonagamba LGH, Silva MP da, Mecawi AS, Antunes-Rodrigues J, Machado BH. Respiratory network enhances the sympathoinhibitory component of baroreflex of rats submitted to chronic intermittent hypoxia [Internet]. Hypertension. 2016 ; 68( 4): 1021-1030.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/hypertensionaha.116.07731
Artigo de periodico
Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms (2016)
Lopes, Rhéure Alves Moreira; Neves, Karla B.; Montezano, Augusto C.; Harvey, Adam; Carneiro, Fernando Silva; Touyz, Rhian M.; Tostes, Rita de Cássia Aleixo
Source: Hypertension. Unidade: FMRP
Subjects: DIABETES MELLITUS, PROTEÍNAS QUINASES, DISFUNÇÃO ERÉTIL, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOPES, Rhéure Alves Moreira et al. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms. Hypertension, v. 68, n. 4, p. 1056-1064, 2016Tradução . . Disponível em: https://doi.org/10.1161/hypertensionaha.116.07518. Acesso em: 02 nov. 2024.
APA
Lopes, R. A. M., Neves, K. B., Montezano, A. C., Harvey, A., Carneiro, F. S., Touyz, R. M., & Tostes, R. de C. A. (2016). Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms. Hypertension, 68( 4), 1056-1064. doi:10.1161/hypertensionaha.116.07518
NLM
Lopes RAM, Neves KB, Montezano AC, Harvey A, Carneiro FS, Touyz RM, Tostes R de CA. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms [Internet]. Hypertension. 2016 ; 68( 4): 1056-1064.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/hypertensionaha.116.07518
Vancouver
Lopes RAM, Neves KB, Montezano AC, Harvey A, Carneiro FS, Touyz RM, Tostes R de CA. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms [Internet]. Hypertension. 2016 ; 68( 4): 1056-1064.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/hypertensionaha.116.07518
Artigo de periodico
Chemerin regulates crosstalk between adipocytes and vascular cells through nox (2015)
Neves, Karla Bianca; Cat, Aurelie Nguyen Dinh; Lopes, Rheure Alves Moreira; Rios, Francisco Jose; Anagnostopoulou, Aikaterini; Lobato, Nubia Souza; Oliveira, Ana Maria de; Tostes, Rita de Cássia Aleixo; Montezano, Augusto C.; Touyz, Rhian M.
Source: Hypertension. Unidades: FMRP, FCFRP
Subjects: APOPTOSE (EFEITOS DE DROGAS), INTERAÇÃO CELULAR, QUIMIOCINAS (FARMACOLOGIA), CÉLULAS ENDOTELIAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NEVES, Karla Bianca et al. Chemerin regulates crosstalk between adipocytes and vascular cells through nox. Hypertension, v. 66, n. 3, p. 657-666, 2015Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.115.05616. Acesso em: 02 nov. 2024.
APA
Neves, K. B., Cat, A. N. D., Lopes, R. A. M., Rios, F. J., Anagnostopoulou, A., Lobato, N. S., et al. (2015). Chemerin regulates crosstalk between adipocytes and vascular cells through nox. Hypertension, 66( 3), 657-666. doi:10.1161/HYPERTENSIONAHA.115.05616
NLM
Neves KB, Cat AND, Lopes RAM, Rios FJ, Anagnostopoulou A, Lobato NS, Oliveira AM de, Tostes R de CA, Montezano AC, Touyz RM. Chemerin regulates crosstalk between adipocytes and vascular cells through nox [Internet]. Hypertension. 2015 ; 66( 3): 657-666.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.05616
Vancouver
Neves KB, Cat AND, Lopes RAM, Rios FJ, Anagnostopoulou A, Lobato NS, Oliveira AM de, Tostes R de CA, Montezano AC, Touyz RM. Chemerin regulates crosstalk between adipocytes and vascular cells through nox [Internet]. Hypertension. 2015 ; 66( 3): 657-666.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.05616
Artigo de periodico
Role of chemoreceptor activation in hemodynamic responses to electrical stimulation of the carotid sinus in conscious rats (2015)
Katayama, Pedro L.; Castania, Jaci Airton; Dias, Daniel P. M.; Patel, Kaushik P.; Fazan Júnior, Rubens; Salgado, Helio Cesar
Source: Hypertension. Unidade: FMRP
Subjects: PRESSORECEPTORES, HEMODINÂMICA, HIPERTENSÃO, SISTEMA NERVOSO SIMPÁTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KATAYAMA, Pedro L. et al. Role of chemoreceptor activation in hemodynamic responses to electrical stimulation of the carotid sinus in conscious rats. Hypertension, v. 66, n. 3, p. 598-603, 2015Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.115.05316. Acesso em: 02 nov. 2024.
APA
Katayama, P. L., Castania, J. A., Dias, D. P. M., Patel, K. P., Fazan Júnior, R., & Salgado, H. C. (2015). Role of chemoreceptor activation in hemodynamic responses to electrical stimulation of the carotid sinus in conscious rats. Hypertension, 66( 3), 598-603. doi:10.1161/HYPERTENSIONAHA.115.05316
NLM
Katayama PL, Castania JA, Dias DPM, Patel KP, Fazan Júnior R, Salgado HC. Role of chemoreceptor activation in hemodynamic responses to electrical stimulation of the carotid sinus in conscious rats [Internet]. Hypertension. 2015 ; 66( 3): 598-603.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.05316
Vancouver
Katayama PL, Castania JA, Dias DPM, Patel KP, Fazan Júnior R, Salgado HC. Role of chemoreceptor activation in hemodynamic responses to electrical stimulation of the carotid sinus in conscious rats [Internet]. Hypertension. 2015 ; 66( 3): 598-603.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.05316
Artigo de periodico
Downregulation of nuclear factor erythroid 2-related factor and associated antioxidant genes contributes to redox-sensitive vascular dysfunction in hypertension (2015)
Lopes, Rhéure Alves Moreira; Neves, Karla B.; Tostes, Rita de Cássia Aleixo; Montezano, Augusto C.; Touyz, Rhian M.
Source: Hypertension. Unidade: FMRP
Subjects: ESTRESSE OXIDATIVO, HIPERTENSÃO, RESISTÊNCIA VASCULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOPES, Rhéure Alves Moreira et al. Downregulation of nuclear factor erythroid 2-related factor and associated antioxidant genes contributes to redox-sensitive vascular dysfunction in hypertension. Hypertension, v. 66, n. 6, p. 1240-1250, 2015Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.115.06163. Acesso em: 02 nov. 2024.
APA
Lopes, R. A. M., Neves, K. B., Tostes, R. de C. A., Montezano, A. C., & Touyz, R. M. (2015). Downregulation of nuclear factor erythroid 2-related factor and associated antioxidant genes contributes to redox-sensitive vascular dysfunction in hypertension. Hypertension, 66( 6), 1240-1250. doi:10.1161/HYPERTENSIONAHA.115.06163
NLM
Lopes RAM, Neves KB, Tostes R de CA, Montezano AC, Touyz RM. Downregulation of nuclear factor erythroid 2-related factor and associated antioxidant genes contributes to redox-sensitive vascular dysfunction in hypertension [Internet]. Hypertension. 2015 ; 66( 6): 1240-1250.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.06163
Vancouver
Lopes RAM, Neves KB, Tostes R de CA, Montezano AC, Touyz RM. Downregulation of nuclear factor erythroid 2-related factor and associated antioxidant genes contributes to redox-sensitive vascular dysfunction in hypertension [Internet]. Hypertension. 2015 ; 66( 6): 1240-1250.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.06163
Artigo de periodico
Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension (2015)
Martínez-Martínez, Ernesto; Calvier, Laurent; Fernández-Celis, Amaya; Rousseau, Elodie; Jurado-López, Raquel; Rossoni, Luciana Venturini ; Jaisser, Frederic; Zannad, Faiez; Rossignol, Patrick; Cachofeiro, Victoria; López-Andrés, Natalia
Source: Hypertension. Unidade: ICB
Subjects: FISIOLOGIA, FISIOLOGIA CARDIOVASCULAR, HIPERTENSÃO, HIPERTENSÃO, INIBIDORES QUÍMICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARTÍNEZ-MARTÍNEZ, Ernesto et al. Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension. Hypertension, v. 66, n. 4, p. 767-775, 2015Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.115.05876. Acesso em: 02 nov. 2024.
APA
Martínez-Martínez, E., Calvier, L., Fernández-Celis, A., Rousseau, E., Jurado-López, R., Rossoni, L. V., et al. (2015). Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension. Hypertension, 66( 4), 767-775. doi:10.1161/HYPERTENSIONAHA.115.05876
NLM
Martínez-Martínez E, Calvier L, Fernández-Celis A, Rousseau E, Jurado-López R, Rossoni LV, Jaisser F, Zannad F, Rossignol P, Cachofeiro V, López-Andrés N. Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension [Internet]. Hypertension. 2015 ; 66( 4): 767-775.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.05876
Vancouver
Martínez-Martínez E, Calvier L, Fernández-Celis A, Rousseau E, Jurado-López R, Rossoni LV, Jaisser F, Zannad F, Rossignol P, Cachofeiro V, López-Andrés N. Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension [Internet]. Hypertension. 2015 ; 66( 4): 767-775.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.05876

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