ReP USP - Resultado da busca

Artigo de periodico
Deciphering complexity of GPCR signaling and modulation: implications and perspectives for drug discovery (2025)
Costa Neto, Claudio Miguel da ; Parreiras-e-Silva, Lucas Tabajara
Source: Clinical Science. Unidade: FM
Subjects: PROTEÍNAS, RECEPTORES, FÁRMACOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA NETO, Claudio Miguel da e PARREIRAS-E-SILVA, Lucas Tabajara. Deciphering complexity of GPCR signaling and modulation: implications and perspectives for drug discovery. Clinical Science, v. 139, n. 10, p. 463-477, 2025Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/87157. Acesso em: 25 fev. 2026.
APA
Costa Neto, C. M. da, & Parreiras-e-Silva, L. T. (2025). Deciphering complexity of GPCR signaling and modulation: implications and perspectives for drug discovery. Clinical Science, 139( 10), 463-477. doi:10.1042/CS20245182
NLM
Costa Neto CM da, Parreiras-e-Silva LT. Deciphering complexity of GPCR signaling and modulation: implications and perspectives for drug discovery [Internet]. Clinical Science. 2025 ; 139( 10): 463-477.[citado 2026 fev. 25 ] Available from: https://observatorio.fm.usp.br/handle/OPI/87157
Vancouver
Costa Neto CM da, Parreiras-e-Silva LT. Deciphering complexity of GPCR signaling and modulation: implications and perspectives for drug discovery [Internet]. Clinical Science. 2025 ; 139( 10): 463-477.[citado 2026 fev. 25 ] Available from: https://observatorio.fm.usp.br/handle/OPI/87157
Artigo de periodico
Endothelin-1 down-regulates nuclear factor erythroid 2-related factor-2 and contributes to perivascular adipose tissue dysfunction in obesity (2024)
Lima, Anna Flavia R.; Rodrigues, Daniel; Machado, Mirele Resende ; Oliveira Neto, José Teles de ; Bressan, Alecsander Fabrício Moreira ; Pedersoli, Carina Amarante ; Alves, Juliano Vilela ; Silva Neto, Júlio Alves da ; Barros, Paula Rodrigues de ; Dias, Thiago B.; Garcia, Luis Vicente ; Bruder-Nascimento, Ariane; Bruder-Nascimento, Thiago; Carneiro, Fernando Silva ; Leiria, Luiz Osório Silveira ; Tostes, Rita de Cássia Aleixo ; Costa, Rafael Menezes da
Source: Clinical Science. Unidade: FMRP
Subjects: ENDOTELINAS, OBESIDADE, ESTRESSE OXIDATIVO, TECIDO ADIPOSO, AORTA TORÁCICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Anna Flavia R. et al. Endothelin-1 down-regulates nuclear factor erythroid 2-related factor-2 and contributes to perivascular adipose tissue dysfunction in obesity. Clinical Science, v. 138, n. 17, p. 1071-1087, 2024Tradução . . Disponível em: https://doi.org/10.1042/CS20240624. Acesso em: 25 fev. 2026.
APA
Lima, A. F. R., Rodrigues, D., Machado, M. R., Oliveira Neto, J. T. de, Bressan, A. F. M., Pedersoli, C. A., et al. (2024). Endothelin-1 down-regulates nuclear factor erythroid 2-related factor-2 and contributes to perivascular adipose tissue dysfunction in obesity. Clinical Science, 138( 17), 1071-1087. doi:10.1042/CS20240624
NLM
Lima A FR, Rodrigues D, Machado MR, Oliveira Neto JT de, Bressan AFM, Pedersoli CA, Alves JV, Silva Neto JA da, Barros PR de, Dias T B, Garcia LV, Bruder-Nascimento A, Bruder-Nascimento T, Carneiro FS, Leiria LOS, Tostes R de CA, Costa RM da. Endothelin-1 down-regulates nuclear factor erythroid 2-related factor-2 and contributes to perivascular adipose tissue dysfunction in obesity [Internet]. Clinical Science. 2024 ; 138( 17): 1071-1087.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20240624
Vancouver
Lima A FR, Rodrigues D, Machado MR, Oliveira Neto JT de, Bressan AFM, Pedersoli CA, Alves JV, Silva Neto JA da, Barros PR de, Dias T B, Garcia LV, Bruder-Nascimento A, Bruder-Nascimento T, Carneiro FS, Leiria LOS, Tostes R de CA, Costa RM da. Endothelin-1 down-regulates nuclear factor erythroid 2-related factor-2 and contributes to perivascular adipose tissue dysfunction in obesity [Internet]. Clinical Science. 2024 ; 138( 17): 1071-1087.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20240624
Artigo de periodico
"Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training " (2023)
Raquel, Hiviny de Ataides ; Perego, Sany M. ; Masson, Gustavo S.; Jensen, Leonardo ; Colquhoun, Alison ; Michelini, Lisete Compagno
Source: Clinical Science. Unidade: ICB
Subjects: BIOLOGIA CELULAR, FISIOLOGIA, INSUFICIÊNCIA CARDÍACA ANIMAL, BARREIRA HEMATO-ENCEFÁLICA, RATOS WISTAR, SISTEMA NERVOSO AUTÔNOMO, CIRCULAÇÃO ANIMAL, EXERCÍCIO FÍSICO, PROTEÍNAS DA MEMBRANA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RAQUEL, Hiviny de Ataides et al. "Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training ". Clinical Science, v. 137, n. 15, p. 1049-1066, 2023Tradução . . Disponível em: https://doi.org/10.1042/CS20230489. Acesso em: 25 fev. 2026.
APA
Raquel, H. de A., Perego, S. M., Masson, G. S., Jensen, L., Colquhoun, A., & Michelini, L. C. (2023). "Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training ". Clinical Science, 137( 15), 1049-1066. doi:10.1042/CS20230489
NLM
Raquel H de A, Perego SM, Masson GS, Jensen L, Colquhoun A, Michelini LC. "Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training " [Internet]. Clinical Science. 2023 ; 137( 15): 1049-1066.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20230489
Vancouver
Raquel H de A, Perego SM, Masson GS, Jensen L, Colquhoun A, Michelini LC. "Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training " [Internet]. Clinical Science. 2023 ; 137( 15): 1049-1066.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20230489
Artigo de periodico
Murine endometrial-derived mesenchymal stem cells suppress experimental autoimmune encephalomyelitis depending on indoleamine-2,3-dioxygenase expression (2021)
Polonio, Carolina Manganeli ; Freitas, Carla Longo de ; Oliveira, Marília Garcia de ; Rossato, Cristiano ; Brandão, Wesley Nogueira ; Zanluqui, Nágela Ghabdan ; Oliveira, Lilian Gomes de ; Mimura, Luiza Ayumi Nishiyama ; Silva, Maysa Braga Barros ; Calich, Vera Lúcia Garcia ; Nisenbaum, Marcelo Gil; Halpern, Silvio ; Evangelista, Lucila; Maluf, Mariangela; Perin, Paulo Marcelo ; Czeresnia, Carlos Eduardo ; Peron, Jean Pierre Schatzmann
Source: Clinical Science. Unidades: ICB, FM, FCF
Subjects: IMUNOLOGIA, CÉLULAS-TRONCO, ENCEFALOMIELITE ANIMAL, ADJUVANTES IMUNOLÓGICOS, LINFÓCITOS, CITOCINAS, LINFONODOS DE ANIMAL, SISTEMA NERVOSO CENTRAL, REAÇÃO EM CADEIA POR POLIMERASE, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
POLONIO, Carolina Manganeli et al. Murine endometrial-derived mesenchymal stem cells suppress experimental autoimmune encephalomyelitis depending on indoleamine-2,3-dioxygenase expression. Clinical Science, v. 135, p. 1065–1082, 2021Tradução . . Disponível em: https://doi.org/10.1042/CS20201544. Acesso em: 25 fev. 2026.
APA
Polonio, C. M., Freitas, C. L. de, Oliveira, M. G. de, Rossato, C., Brandão, W. N., Zanluqui, N. G., et al. (2021). Murine endometrial-derived mesenchymal stem cells suppress experimental autoimmune encephalomyelitis depending on indoleamine-2,3-dioxygenase expression. Clinical Science, 135, 1065–1082. doi:10.1042/CS20201544
NLM
Polonio CM, Freitas CL de, Oliveira MG de, Rossato C, Brandão WN, Zanluqui NG, Oliveira LG de, Mimura LAN, Silva MBB, Calich VLG, Nisenbaum MG, Halpern S, Evangelista L, Maluf M, Perin PM, Czeresnia CE, Peron JPS. Murine endometrial-derived mesenchymal stem cells suppress experimental autoimmune encephalomyelitis depending on indoleamine-2,3-dioxygenase expression [Internet]. Clinical Science. 2021 ; 135 1065–1082.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20201544
Vancouver
Polonio CM, Freitas CL de, Oliveira MG de, Rossato C, Brandão WN, Zanluqui NG, Oliveira LG de, Mimura LAN, Silva MBB, Calich VLG, Nisenbaum MG, Halpern S, Evangelista L, Maluf M, Perin PM, Czeresnia CE, Peron JPS. Murine endometrial-derived mesenchymal stem cells suppress experimental autoimmune encephalomyelitis depending on indoleamine-2,3-dioxygenase expression [Internet]. Clinical Science. 2021 ; 135 1065–1082.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20201544
Artigo de periodico
Inflammatory effect of Bothropstoxin-I from Bothrops jararacussu venom mediated by NLRP3 inflammasome involves ATP and P2X7 receptor (2021)
Silva, Priscila de Andrade Ranéia e; Lima, Dhêmerson Souza de ; Luiz, João Paulo Mesquita ; Câmara, Niels Olsen Saraiva ; Alves Filho, José Carlos Farias ; Pontillo, Alessandra ; Bortoluci, Karina Ramalho ; Mauro, Eliana Faquim de Lima
Source: Clinical Science. Unidades: ICB, FMRP
Subjects: IMUNOLOGIA, MACRÓFAGOS, INFLAMAÇÃO, VENENOS DE ORIGEM ANIMAL, SERPENTES, FOSFOLIPASES A, ATIVAÇÃO ENZIMÁTICA, CATALASE, INTERLEUCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Priscila de Andrade Ranéia e et al. Inflammatory effect of Bothropstoxin-I from Bothrops jararacussu venom mediated by NLRP3 inflammasome involves ATP and P2X7 receptor. Clinical Science, v. 135, n. 5, p. 687–701, 2021Tradução . . Disponível em: https://doi.org/10.1042/CS20201419. Acesso em: 25 fev. 2026.
APA
Silva, P. de A. R. e, Lima, D. S. de, Luiz, J. P. M., Câmara, N. O. S., Alves Filho, J. C. F., Pontillo, A., et al. (2021). Inflammatory effect of Bothropstoxin-I from Bothrops jararacussu venom mediated by NLRP3 inflammasome involves ATP and P2X7 receptor. Clinical Science, 135( 5), 687–701. doi:10.1042/CS20201419
NLM
Silva P de AR e, Lima DS de, Luiz JPM, Câmara NOS, Alves Filho JCF, Pontillo A, Bortoluci KR, Mauro EF de L. Inflammatory effect of Bothropstoxin-I from Bothrops jararacussu venom mediated by NLRP3 inflammasome involves ATP and P2X7 receptor [Internet]. Clinical Science. 2021 ; 135( 5): 687–701.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20201419
Vancouver
Silva P de AR e, Lima DS de, Luiz JPM, Câmara NOS, Alves Filho JCF, Pontillo A, Bortoluci KR, Mauro EF de L. Inflammatory effect of Bothropstoxin-I from Bothrops jararacussu venom mediated by NLRP3 inflammasome involves ATP and P2X7 receptor [Internet]. Clinical Science. 2021 ; 135( 5): 687–701.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20201419
Artigo de periodico
Vena cava presents endothelial dysfunction prior to thoracic aorta in heart failure: the pivotal role of nNOS uncoupling/ oxidative stress (2021)
Dardi, Patrizia ; Perazza, Laís Rossi; Couto, Gisele Kruger ; Campos, Gianne Paul; Capettini, Luciano dos Santos Aggum ; Rossoni, Luciana Venturini
Source: Clinical Science. Unidade: ICB
Subjects: FISIOLOGIA, VEIA CAVA, ANTIOXIDANTES, INSUFICIÊNCIA CARDÍACA ANIMAL, AORTA TORÁCICA, AORTA DE ANIMAL, ENDOTÉLIO VASCULAR, PERÓXIDO DE HIDROGÊNIO, INFARTO DO MIOCÁRDIO, ÓXIDO NÍTRICO, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DARDI, Patrizia et al. Vena cava presents endothelial dysfunction prior to thoracic aorta in heart failure: the pivotal role of nNOS uncoupling/ oxidative stress. Clinical Science, 2021Tradução . . Disponível em: https://doi.org/10.1042/CS20210810. Acesso em: 25 fev. 2026.
APA
Dardi, P., Perazza, L. R., Couto, G. K., Campos, G. P., Capettini, L. dos S. A., & Rossoni, L. V. (2021). Vena cava presents endothelial dysfunction prior to thoracic aorta in heart failure: the pivotal role of nNOS uncoupling/ oxidative stress. Clinical Science. doi:10.1042/CS20210810
NLM
Dardi P, Perazza LR, Couto GK, Campos GP, Capettini L dos SA, Rossoni LV. Vena cava presents endothelial dysfunction prior to thoracic aorta in heart failure: the pivotal role of nNOS uncoupling/ oxidative stress [Internet]. Clinical Science. 2021 ;[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20210810
Vancouver
Dardi P, Perazza LR, Couto GK, Campos GP, Capettini L dos SA, Rossoni LV. Vena cava presents endothelial dysfunction prior to thoracic aorta in heart failure: the pivotal role of nNOS uncoupling/ oxidative stress [Internet]. Clinical Science. 2021 ;[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20210810
Artigo de periodico
Lysophosphatidylcholine induces oxidative stress in human endothelial cells via NOX5 activation: implications in atherosclerosis (2021)
Silva, Josiane Fernandes da; Alves, Juliano Vilela; Silva Neto, Júlio Alves da; Costa, Rafael Menezes da; Neves, Karla B.; Lopes, Rheure Alves Moreira; Carmargo, Livia L.; Rios, Francisco J.; Montezano, Augusto C.; Touyz, Rhian M.; Tostes, Rita de Cássia Aleixo
Source: Clinical Science. Unidade: FMRP
Subjects: ARTERIOSCLEROSE, CÁLCIO, CÉLULAS CULTIVADAS, CÉLULAS ENDOTELIAIS, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Josiane Fernandes da et al. Lysophosphatidylcholine induces oxidative stress in human endothelial cells via NOX5 activation: implications in atherosclerosis. Clinical Science, v. 135, n. 15, p. 1845-1858, 2021Tradução . . Disponível em: https://doi.org/10.1042/CS20210468. Acesso em: 25 fev. 2026.
APA
Silva, J. F. da, Alves, J. V., Silva Neto, J. A. da, Costa, R. M. da, Neves, K. B., Lopes, R. A. M., et al. (2021). Lysophosphatidylcholine induces oxidative stress in human endothelial cells via NOX5 activation: implications in atherosclerosis. Clinical Science, 135( 15), 1845-1858. doi:10.1042/CS20210468
NLM
Silva JF da, Alves JV, Silva Neto JA da, Costa RM da, Neves KB, Lopes RAM, Carmargo L L, Rios FJ, Montezano AC, Touyz RM, Tostes R de CA. Lysophosphatidylcholine induces oxidative stress in human endothelial cells via NOX5 activation: implications in atherosclerosis [Internet]. Clinical Science. 2021 ; 135( 15): 1845-1858.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20210468
Vancouver
Silva JF da, Alves JV, Silva Neto JA da, Costa RM da, Neves KB, Lopes RAM, Carmargo L L, Rios FJ, Montezano AC, Touyz RM, Tostes R de CA. Lysophosphatidylcholine induces oxidative stress in human endothelial cells via NOX5 activation: implications in atherosclerosis [Internet]. Clinical Science. 2021 ; 135( 15): 1845-1858.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20210468
Artigo de periodico
High-volume endurance exercise training stimulates hematopoiesis by increasing ACE NH2-terminal activity (2021)
Magalhães, Flávio Castro ; Fernandes, Tiago ; Bassaneze, Vinicius; Mattos, Katt Coelho; Schettert, Isolmar; Marques, Fabio Luiz Navarro ; Krieger, Jose Eduardo ; Nava, Roberto; Barauna, Valério Garrone; Oliveira, Edilamar Menezes de
Source: Clinical Science. Unidades: EEFE, FM
Subjects: ANGIOTENSINAS, SISTEMA RENINA-ANGIOTENSINA, ERITROPOIETINA, TREINAMENTO FÍSICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MAGALHÃES, Flávio Castro et al. High-volume endurance exercise training stimulates hematopoiesis by increasing ACE NH2-terminal activity. Clinical Science, v. 135, n. 20, p. 2377-2391, 2021Tradução . . Disponível em: https://doi.org/10.1042/CS20210739. Acesso em: 25 fev. 2026.
APA
Magalhães, F. C., Fernandes, T., Bassaneze, V., Mattos, K. C., Schettert, I., Marques, F. L. N., et al. (2021). High-volume endurance exercise training stimulates hematopoiesis by increasing ACE NH2-terminal activity. Clinical Science, 135( 20), 2377-2391. doi:10.1042/CS20210739
NLM
Magalhães FC, Fernandes T, Bassaneze V, Mattos KC, Schettert I, Marques FLN, Krieger JE, Nava R, Barauna VG, Oliveira EM de. High-volume endurance exercise training stimulates hematopoiesis by increasing ACE NH2-terminal activity [Internet]. Clinical Science. 2021 ; 135( 20): 2377-2391.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20210739
Vancouver
Magalhães FC, Fernandes T, Bassaneze V, Mattos KC, Schettert I, Marques FLN, Krieger JE, Nava R, Barauna VG, Oliveira EM de. High-volume endurance exercise training stimulates hematopoiesis by increasing ACE NH2-terminal activity [Internet]. Clinical Science. 2021 ; 135( 20): 2377-2391.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20210739
Artigo de periodico
Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure (2020)
Fontes, Milene Tavares ; Paula, Suliana Mesquita ; Lino, Caroline Antunes ; Senger, Nathalia ; Couto, Gisele Kruger; Barreto-Chaves, Maria Luiza Morais ; Mill, José Geraldo ; Rossoni, Luciana Venturini
Source: Clinical Science. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA, SISTEMA RENINA-ANGIOTENSINA, TECIDO ADIPOSO, INSUFICIÊNCIA CARDÍACA ANIMAL, DOENÇAS CARDIOVASCULARES EM ANIMAL, ENDOTÉLIO VASCULAR, INFARTO DO MIOCÁRDIO, RATOS, ANGIOTENSINAS, AORTA DE ANIMAL, ESTRESSE OXIDATIVO, ÓXIDO NÍTRICO, AORTA TORÁCICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FONTES, Milene Tavares et al. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure. Clinical Science, 2020Tradução . . Disponível em: https://doi.org/10.1042/CS20201099. Acesso em: 25 fev. 2026.
APA
Fontes, M. T., Paula, S. M., Lino, C. A., Senger, N., Couto, G. K., Barreto-Chaves, M. L. M., et al. (2020). Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure. Clinical Science. doi:10.1042/CS20201099
NLM
Fontes MT, Paula SM, Lino CA, Senger N, Couto GK, Barreto-Chaves MLM, Mill JG, Rossoni LV. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure [Internet]. Clinical Science. 2020 ;[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20201099
Vancouver
Fontes MT, Paula SM, Lino CA, Senger N, Couto GK, Barreto-Chaves MLM, Mill JG, Rossoni LV. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure [Internet]. Clinical Science. 2020 ;[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20201099
Artigo de periodico
Role of the renin–angiotensin system in kidney development and programming of adult blood pressure (2020)
Almeida, Lucas Ferreira de ; Tofteng, Signe S.; Madsen, Kirsten; Jensen, Boye L.
Source: Clinical Science. Unidade: FMRP
Subjects: SISTEMA RENINA-ANGIOTENSINA, PRESSÃO SANGUÍNEA, ADULTOS, HIPERTENSÃO, RIM, ANGIOTENSINAS, RENINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Lucas Ferreira de et al. Role of the renin–angiotensin system in kidney development and programming of adult blood pressure. Clinical Science, v. 134, n. 6, p. 641-656, 2020Tradução . . Disponível em: https://doi.org/10.1042/CS20190765. Acesso em: 25 fev. 2026.
APA
Almeida, L. F. de, Tofteng, S. S., Madsen, K., & Jensen, B. L. (2020). Role of the renin–angiotensin system in kidney development and programming of adult blood pressure. Clinical Science, 134( 6), 641-656. doi:10.1042/CS20190765
NLM
Almeida LF de, Tofteng S S, Madsen K, Jensen B L. Role of the renin–angiotensin system in kidney development and programming of adult blood pressure [Internet]. Clinical Science. 2020 ; 134( 6): 641-656.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20190765
Vancouver
Almeida LF de, Tofteng S S, Madsen K, Jensen B L. Role of the renin–angiotensin system in kidney development and programming of adult blood pressure [Internet]. Clinical Science. 2020 ; 134( 6): 641-656.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20190765
Artigo de periodico
NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis (2019)
Braga, Tárcio Teodoro ; Brandão, Wesley Nogueira ; Azevedo, Hatylas ; Terra, Fernanda Fernandes; Feitosa, Amanda Campelo Lima Melo; Pereira, Felipe Valença; Oliveira, Vinicius de Andrade ; Hiyane, Meire Ioshie; Peron, Jean Pierre Schatzmann ; Câmara, Niels Olsen Saraiva
Source: Clinical Science. Unidade: ICB
Subjects: IMUNOLOGIA, AUTOIMUNIDADE, NUCLEOTÍDEOS, CITOCINAS, DOENÇAS AUTOIMUNES, LINFÓCITOS T, ENCEFALOMIELITE ANIMAL, MODELOS ANIMAIS, FATORES DE TRANSCRIÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRAGA, Tárcio Teodoro et al. NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis. Clinical Science, v. 133, n. 17, p. 1901-1916, 2019Tradução . . Disponível em: https://doi.org/10.1042/CS20190506. Acesso em: 25 fev. 2026.
APA
Braga, T. T., Brandão, W. N., Azevedo, H., Terra, F. F., Feitosa, A. C. L. M., Pereira, F. V., et al. (2019). NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis. Clinical Science, 133( 17), 1901-1916. doi:10.1042/CS20190506
NLM
Braga TT, Brandão WN, Azevedo H, Terra FF, Feitosa ACLM, Pereira FV, Oliveira V de A, Hiyane MI, Peron JPS, Câmara NOS. NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis [Internet]. Clinical Science. 2019 ; 133( 17): 1901-1916.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20190506
Vancouver
Braga TT, Brandão WN, Azevedo H, Terra FF, Feitosa ACLM, Pereira FV, Oliveira V de A, Hiyane MI, Peron JPS, Câmara NOS. NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis [Internet]. Clinical Science. 2019 ; 133( 17): 1901-1916.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20190506
Artigo de periodico
Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure (2019)
Andrade, David Campos ; Toledo, Camilo ; Díaz, Hugo Sebastián ; Lucero, Claudia; Álvarez, Alexis Arce ; Santos, Luiz Marcelo Oliveira ; Takakura, Ana Carolina ; Moreira, Thiago dos Santos ; Schultz, Harold D. ; Marcus, Noah J. ; Alcayaga, Julio ; Rio, Rodrigo Del
Source: Clinical Science. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, SISTEMA NERVOSO SIMPÁTICO, INSUFICIÊNCIA CARDÍACA ANIMAL, NEURÔNIOS, IMUNOTOXINAS, TOXINAS EM ANIMAL, RATOS, ARRITMIA, TRONCO DE ANIMAL, CATECOLAMINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANDRADE, David Campos et al. Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure. Clinical Science, v. 133, n. 3, p. 393-405, 2019Tradução . . Disponível em: https://doi.org/10.1042/CS20180589. Acesso em: 25 fev. 2026.
APA
Andrade, D. C., Toledo, C., Díaz, H. S., Lucero, C., Álvarez, A. A., Santos, L. M. O., et al. (2019). Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure. Clinical Science, 133( 3), 393-405. doi:10.1042/CS20180589
NLM
Andrade DC, Toledo C, Díaz HS, Lucero C, Álvarez AA, Santos LMO, Takakura AC, Moreira T dos S, Schultz HD, Marcus NJ, Alcayaga J, Rio RD. Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure [Internet]. Clinical Science. 2019 ; 133( 3): 393-405.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20180589
Vancouver
Andrade DC, Toledo C, Díaz HS, Lucero C, Álvarez AA, Santos LMO, Takakura AC, Moreira T dos S, Schultz HD, Marcus NJ, Alcayaga J, Rio RD. Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure [Internet]. Clinical Science. 2019 ; 133( 3): 393-405.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20180589
Artigo de periodico
Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway (2018)
Senger, Nathalia ; Melo, Marcos Barrouin; Diniz, Gabriela Placoná ; Santos, Maria José Campagnole dos ; Santos, Robson Augusto Souza dos; Barreto-Chaves, Maria Luiza Morais
Source: Clinical Science. Unidade: ICB
Subjects: ANGIOTENSINAS, CORAÇÃO, HIPERTIREOIDISMO, SISTEMA RENINA-ANGIOTENSINA, HORMÔNIOS TIREOIDIANOS, ANGIOTENSINA II, ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SENGER, Nathalia et al. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, v. 132, n. 11, p. 1117-1133, 2018Tradução . . Disponível em: https://doi.org/10.1042/CS20171606. Acesso em: 25 fev. 2026.
APA
Senger, N., Melo, M. B., Diniz, G. P., Santos, M. J. C. dos, Santos, R. A. S. dos, & Barreto-Chaves, M. L. M. (2018). Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, 132( 11), 1117-1133. doi:10.1042/CS20171606
NLM
Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Barreto-Chaves MLM. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20171606
Vancouver
Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Barreto-Chaves MLM. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20171606
Artigo de periodico
Hyperfiltration in ubiquitin C-terminal hydrolase L1-deleted mice (2018)
Boisvert, Naomi C.; Holterman, Chet E.; Thibodeau, Jean-François; Nasrallah, Rania; Kamto, Eldjonai; Comin, Cesar H.; Costa, Luciano da Fontoura ; Carter, Anthony; Hébert, Richard L.; Gutsol, Alex; Cron, Gregory O.; Lacoste, Baptiste; Gray, Douglas A.; Kennedy, Chris R.
Source: Clinical Science. Unidade: IFSC
Subjects: REDES COMPLEXAS, AUTOR, MINERAÇÃO DE DADOS, VISÃO COMPUTACIONAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOISVERT, Naomi C. et al. Hyperfiltration in ubiquitin C-terminal hydrolase L1-deleted mice. Clinical Science, v. 132, n. 13, p. 1453-1470, 2018Tradução . . Disponível em: https://doi.org/10.1042/CS20180085. Acesso em: 25 fev. 2026.
APA
Boisvert, N. C., Holterman, C. E., Thibodeau, J. -F., Nasrallah, R., Kamto, E., Comin, C. H., et al. (2018). Hyperfiltration in ubiquitin C-terminal hydrolase L1-deleted mice. Clinical Science, 132( 13), 1453-1470. doi:10.1042/CS20180085
NLM
Boisvert NC, Holterman CE, Thibodeau J-F, Nasrallah R, Kamto E, Comin CH, Costa L da F, Carter A, Hébert RL, Gutsol A, Cron GO, Lacoste B, Gray DA, Kennedy CR. Hyperfiltration in ubiquitin C-terminal hydrolase L1-deleted mice [Internet]. Clinical Science. 2018 ; 132( 13): 1453-1470.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20180085
Vancouver
Boisvert NC, Holterman CE, Thibodeau J-F, Nasrallah R, Kamto E, Comin CH, Costa L da F, Carter A, Hébert RL, Gutsol A, Cron GO, Lacoste B, Gray DA, Kennedy CR. Hyperfiltration in ubiquitin C-terminal hydrolase L1-deleted mice [Internet]. Clinical Science. 2018 ; 132( 13): 1453-1470.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20180085
Artigo de periodico
Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease (2017)
Festuccia, William Tadeu Lara
Source: Clinical Science. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FESTUCCIA, William Tadeu Lara. Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease. Clinical Science, v. 131, n. 4, p. 327-328, 2017Tradução . . Disponível em: https://doi.org/10.1042/CS20160855. Acesso em: 25 fev. 2026.
APA
Festuccia, W. T. L. (2017). Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease. Clinical Science, 131( 4), 327-328. doi:10.1042/CS20160855
NLM
Festuccia WTL. Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease [Internet]. Clinical Science. 2017 ; 131( 4): 327-328.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20160855
Vancouver
Festuccia WTL. Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease [Internet]. Clinical Science. 2017 ; 131( 4): 327-328.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20160855
Artigo de periodico
A past and present overview of macrophage metabolism and functional outcomes (2017)
Curi, Rui ; Mendes, Renata de Siqueira; Crispin, Luiz Aurélio de Campos ; Norata, Giuseppe Danilo ; Sampaio, Sandra Coccuzzo ; Newsholme, Philip
Source: Clinical Science. Unidade: ICB
Subjects: FISIOLOGIA, MACRÓFAGOS, METABOLISMO ENERGÉTICO, ÁCIDOS GRAXOS, GLICOSE, GLUTAMINA, INSULINA, HORMÔNIOS TIREOIDIANOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CURI, Rui et al. A past and present overview of macrophage metabolism and functional outcomes. Clinical Science, v. 131, n. 12, p. 1329-1342, 2017Tradução . . Disponível em: https://doi.org/10.1042/CS20170220. Acesso em: 25 fev. 2026.
APA
Curi, R., Mendes, R. de S., Crispin, L. A. de C., Norata, G. D., Sampaio, S. C., & Newsholme, P. (2017). A past and present overview of macrophage metabolism and functional outcomes. Clinical Science, 131( 12), 1329-1342. doi:10.1042/CS20170220
NLM
Curi R, Mendes R de S, Crispin LA de C, Norata GD, Sampaio SC, Newsholme P. A past and present overview of macrophage metabolism and functional outcomes [Internet]. Clinical Science. 2017 ; 131( 12): 1329-1342.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20170220
Vancouver
Curi R, Mendes R de S, Crispin LA de C, Norata GD, Sampaio SC, Newsholme P. A past and present overview of macrophage metabolism and functional outcomes [Internet]. Clinical Science. 2017 ; 131( 12): 1329-1342.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20170220
Artigo de periodico
Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy (2017)
Diniz, Gabriela Placoná ; Huang, Zhan-Peng; Liu, Jianming; Chen, Jinghai; Ding, Jian; Fonseca, Renata Inzinna; Barreto-Chaves, Maria Luiza Morais ; Donato Junior, José ; Hu, Xiaoyun; Wang, Da-Zhi
Source: Clinical Science. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, v. 131, n. 24, p. 2885-2900, 2017Tradução . . Disponível em: https://doi.org/10.1042/CS20171368. Acesso em: 25 fev. 2026.
APA
Diniz, G. P., Huang, Z. -P., Liu, J., Chen, J., Ding, J., Fonseca, R. I., et al. (2017). Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, 131( 24), 2885-2900. doi:10.1042/CS20171368
NLM
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20171368
Vancouver
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20171368
Artigo de periodico
Sympathetic nervous activity in patients with acute coronary syndrome: a comparative study of inflammatory biomarkers (2017)
Moreira, Humberto G; Lage, Rony L; Martinez, Daniel G; Ferreira-Santos, Larissa; Rondon, M. U.; Negrão, C. E.; Nicolau, José Carlos
Source: Clinical Science. Unidades: EEFE, FM
Subjects: SISTEMA NERVOSO SIMPÁTICO, SÍNDROME CORONARIANA AGUDA, BIOMARCADORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MOREIRA, Humberto G et al. Sympathetic nervous activity in patients with acute coronary syndrome: a comparative study of inflammatory biomarkers. Clinical Science, v. 131, n. 9, p. 883-895, 2017Tradução . . Disponível em: https://doi.org/10.1042/CS20170049. Acesso em: 25 fev. 2026.
APA
Moreira, H. G., Lage, R. L., Martinez, D. G., Ferreira-Santos, L., Rondon, M. U., Negrão, C. E., & Nicolau, J. C. (2017). Sympathetic nervous activity in patients with acute coronary syndrome: a comparative study of inflammatory biomarkers. Clinical Science, 131( 9), 883-895. doi:10.1042/CS20170049
NLM
Moreira HG, Lage RL, Martinez DG, Ferreira-Santos L, Rondon MU, Negrão CE, Nicolau JC. Sympathetic nervous activity in patients with acute coronary syndrome: a comparative study of inflammatory biomarkers [Internet]. Clinical Science. 2017 ; 131( 9): 883-895.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20170049
Vancouver
Moreira HG, Lage RL, Martinez DG, Ferreira-Santos L, Rondon MU, Negrão CE, Nicolau JC. Sympathetic nervous activity in patients with acute coronary syndrome: a comparative study of inflammatory biomarkers [Internet]. Clinical Science. 2017 ; 131( 9): 883-895.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20170049
Artigo de periodico
PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis (2016)
Filgueiras, Luciano Ribeiro; Koga, Marianna Mainardi; Quaresma, Paula G.; Ishizuka, Edson Kiyotaka; Montes, Marlise B. A.; Prada, Patricia O.; Saad, Mario J.; Jancar, Sonia ; Rios, Francisco J.
Source: Clinical Science. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FILGUEIRAS, Luciano Ribeiro et al. PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis. Clinical Science, v. 130, n. 8, p. 601-612, 2016Tradução . . Disponível em: https://doi.org/10.1042/CS20150538. Acesso em: 25 fev. 2026.
APA
Filgueiras, L. R., Koga, M. M., Quaresma, P. G., Ishizuka, E. K., Montes, M. B. A., Prada, P. O., et al. (2016). PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis. Clinical Science, 130( 8), 601-612. doi:10.1042/CS20150538
NLM
Filgueiras LR, Koga MM, Quaresma PG, Ishizuka EK, Montes MBA, Prada PO, Saad MJ, Jancar S, Rios FJ. PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis [Internet]. Clinical Science. 2016 ; 130( 8): 601-612.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20150538
Vancouver
Filgueiras LR, Koga MM, Quaresma PG, Ishizuka EK, Montes MBA, Prada PO, Saad MJ, Jancar S, Rios FJ. PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis [Internet]. Clinical Science. 2016 ; 130( 8): 601-612.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20150538
Artigo de periodico
EEF1D modulates proliferation and epithelial–mesenchymal transition in oral squamous cell carcinoma (2016)
Flores, Isadora L; Kawahara, Rebeca; Miguel, Márcia C. C; Granato, Daniela Campos; Domingues, Romênia R; Macedo, Carolina C. S; Carnielli, Carolina M; Yokoo, Sami; Rodrigues, Priscila C; Monteiro, Bárbara V. B; Oliveira, Carine E; Salmon, Cristiane Ribeiro; Nociti, Francisco H; Lopes, Márcio A; Silva, Alan Santos; Winck, Flavia Vischi ; Coletta, Ricardo D; Leme, Adriana Franco Paes
Source: Clinical Science. Unidade: IQ
Subjects: CARCINOMA, CÉLULAS EPITELIAIS, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FLORES, Isadora L et al. EEF1D modulates proliferation and epithelial–mesenchymal transition in oral squamous cell carcinoma. Clinical Science, v. 130, p. 785-799, 2016Tradução . . Disponível em: https://doi.org/10.1042/CS20150646. Acesso em: 25 fev. 2026.
APA
Flores, I. L., Kawahara, R., Miguel, M. C. C., Granato, D. C., Domingues, R. R., Macedo, C. C. S., et al. (2016). EEF1D modulates proliferation and epithelial–mesenchymal transition in oral squamous cell carcinoma. Clinical Science, 130, 785-799. doi:10.1042/CS20150646
NLM
Flores IL, Kawahara R, Miguel MCC, Granato DC, Domingues RR, Macedo CCS, Carnielli CM, Yokoo S, Rodrigues PC, Monteiro BVB, Oliveira CE, Salmon CR, Nociti FH, Lopes MA, Silva AS, Winck FV, Coletta RD, Leme AFP. EEF1D modulates proliferation and epithelial–mesenchymal transition in oral squamous cell carcinoma [Internet]. Clinical Science. 2016 ; 130 785-799.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20150646
Vancouver
Flores IL, Kawahara R, Miguel MCC, Granato DC, Domingues RR, Macedo CCS, Carnielli CM, Yokoo S, Rodrigues PC, Monteiro BVB, Oliveira CE, Salmon CR, Nociti FH, Lopes MA, Silva AS, Winck FV, Coletta RD, Leme AFP. EEF1D modulates proliferation and epithelial–mesenchymal transition in oral squamous cell carcinoma [Internet]. Clinical Science. 2016 ; 130 785-799.[citado 2026 fev. 25 ] Available from: https://doi.org/10.1042/CS20150646

USP Schools

ReP

Filtros

USP Schools

Departament

Authors

USP affiliated authors

Date published

Subjects

Funding Agencies

Indexado em

Normalized affiliation

Non normalized affiliation

Countries of institutions of collaboration