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Artigo de periodico
The sympathetic nervous system exacerbates carotid body sensitivity in hypertension (2023)
Felippe, Igor S. A.; Zera, Tymoteusz; Silva, Melina Pires da ; Moraes, Davi José de Almeida ; McBryde, Fiona; Paton, Julian F. R.
Source: Cardiovascular Research. Unidade: FMRP
Subjects: PRESSÃO SANGUÍNEA, HIPERTENSÃO, SISTEMA NERVOSO SIMPÁTICO, CORPO CAROTÍDEO DE ANIMAL
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ABNT
FELIPPE, Igor S. A. et al. The sympathetic nervous system exacerbates carotid body sensitivity in hypertension. Cardiovascular Research, v. 119, n. 1, p. 316-331, 2023Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvac008. Acesso em: 19 out. 2024.
APA
Felippe, I. S. A., Zera, T., Silva, M. P. da, Moraes, D. J. de A., McBryde, F., & Paton, J. F. R. (2023). The sympathetic nervous system exacerbates carotid body sensitivity in hypertension. Cardiovascular Research, 119( 1), 316-331. doi:10.1093/cvr/cvac008
NLM
Felippe ISA, Zera T, Silva MP da, Moraes DJ de A, McBryde F, Paton JFR. The sympathetic nervous system exacerbates carotid body sensitivity in hypertension [Internet]. Cardiovascular Research. 2023 ; 119( 1): 316-331.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/cvac008
Vancouver
Felippe ISA, Zera T, Silva MP da, Moraes DJ de A, McBryde F, Paton JFR. The sympathetic nervous system exacerbates carotid body sensitivity in hypertension [Internet]. Cardiovascular Research. 2023 ; 119( 1): 316-331.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/cvac008
Artigo de periodico
Autonomic innervation of the carotid body as a determinant of its sensitivity: implications for cardiovascular physiology and pathology (2021)
Brognara, Fernanda ; Felippe, Igor S. A.; Salgado, Hélio César ; Paton, Julian F. R.
Source: Cardiovascular Research. Unidades: FMRP, EERP
Subjects: ARTÉRIAS, SISTEMA NERVOSO AUTÔNOMO, DOENÇAS CARDIOVASCULARES, HEMODINÂMICA
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ABNT
BROGNARA, Fernanda et al. Autonomic innervation of the carotid body as a determinant of its sensitivity: implications for cardiovascular physiology and pathology. Cardiovascular Research, v. 117, n. 4, p. 1015-1032, 2021Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvaa250. Acesso em: 19 out. 2024.
APA
Brognara, F., Felippe, I. S. A., Salgado, H. C., & Paton, J. F. R. (2021). Autonomic innervation of the carotid body as a determinant of its sensitivity: implications for cardiovascular physiology and pathology. Cardiovascular Research, 117( 4), 1015-1032. doi:10.1093/cvr/cvaa250
NLM
Brognara F, Felippe ISA, Salgado HC, Paton JFR. Autonomic innervation of the carotid body as a determinant of its sensitivity: implications for cardiovascular physiology and pathology [Internet]. Cardiovascular Research. 2021 ; 117( 4): 1015-1032.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/cvaa250
Vancouver
Brognara F, Felippe ISA, Salgado HC, Paton JFR. Autonomic innervation of the carotid body as a determinant of its sensitivity: implications for cardiovascular physiology and pathology [Internet]. Cardiovascular Research. 2021 ; 117( 4): 1015-1032.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/cvaa250
Artigo de periodico
Periodontitis is associated with hypertension: a systematic review and meta-analysis (2020)
Aguilera, Eva Muñoz; Suvan, Jean; Buti, Jacopo; Czesnikiewicz-Guzik, Marta; Ribeiro, Aline Barbosa ; Orlandi, Marco; Guzik, Tomasz J.; Hingorani, Aroon D.; Nart, Jose; D'Aiuto, Francesco
Source: Cardiovascular Research. Unidade: FMRP
Subjects: PRESSÃO SANGUÍNEA, SAÚDE BUCAL, HIGIENE BUCAL, HIPERTENSÃO, HIPERTENSÃO, PERIODONTITE, PREVALÊNCIA
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ABNT
AGUILERA, Eva Muñoz et al. Periodontitis is associated with hypertension: a systematic review and meta-analysis. Cardiovascular Research, v. 116, n. 1, p. 28-39, 2020Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvz201. Acesso em: 19 out. 2024.
APA
Aguilera, E. M., Suvan, J., Buti, J., Czesnikiewicz-Guzik, M., Ribeiro, A. B., Orlandi, M., et al. (2020). Periodontitis is associated with hypertension: a systematic review and meta-analysis. Cardiovascular Research, 116( 1), 28-39. doi:10.1093/cvr/cvz201
NLM
Aguilera EM, Suvan J, Buti J, Czesnikiewicz-Guzik M, Ribeiro AB, Orlandi M, Guzik TJ, Hingorani AD, Nart J, D'Aiuto F. Periodontitis is associated with hypertension: a systematic review and meta-analysis [Internet]. Cardiovascular Research. 2020 ; 116( 1): 28-39.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/cvz201
Vancouver
Aguilera EM, Suvan J, Buti J, Czesnikiewicz-Guzik M, Ribeiro AB, Orlandi M, Guzik TJ, Hingorani AD, Nart J, D'Aiuto F. Periodontitis is associated with hypertension: a systematic review and meta-analysis [Internet]. Cardiovascular Research. 2020 ; 116( 1): 28-39.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/cvz201
Artigo de periodico
Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR (2011)
Callera, Gláucia Elena; Yogi, Álvaro; Briones, Ana Maria; Montezano, Augusto Cesar Inforzato; He, Ying; Passaglia, Rita de Cassia Aleixo Tostes; Schiffrin, Ernesto L; Touyz, Rhian M.
Source: Cardiovascular Research. Unidade: FMRP
Subjects: ALDOSTERONA, CÉLULAS MUSCULARES
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ABNT
CALLERA, Gláucia Elena et al. Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR. Cardiovascular Research, v. 91, n. 4, p. 720-731, 2011Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvr131. Acesso em: 19 out. 2024.
APA
Callera, G. E., Yogi, Á., Briones, A. M., Montezano, A. C. I., He, Y., Passaglia, R. de C. A. T., et al. (2011). Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR. Cardiovascular Research, 91( 4), 720-731. doi:10.1093/cvr/cvr131
NLM
Callera GE, Yogi Á, Briones AM, Montezano ACI, He Y, Passaglia R de CAT, Schiffrin EL, Touyz RM. Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR [Internet]. Cardiovascular Research. 2011 ; 91( 4): 720-731.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/cvr131
Vancouver
Callera GE, Yogi Á, Briones AM, Montezano ACI, He Y, Passaglia R de CAT, Schiffrin EL, Touyz RM. Vascular proinflammatory responses by aldosterone are mediated via c-Src trafficking to cholesterol-rich microdomains: role of PDGFR [Internet]. Cardiovascular Research. 2011 ; 91( 4): 720-731.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/cvr131
Artigo de periodico
Atrogin-1 and MuRF1 regulate cardiac MyBP-C levels via different mechanisms (2010)
Mearini, Giulia; Gedicke, Christina; Schlossarek, Saskia; Witt, Christian C.; Krämer, Elisabeth; Cao, Peirang; Gomes, Marcelo Damario; Lecker, Stewart H.; Labeit, Siegfried; Willis, Monte S.; Eschenhagem, Thomas; Carrier, Lucie
Source: Cardiovascular Research. Unidade: FMRP
Subjects: CARDIOPATIAS, MIOSINA
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ABNT
MEARINI, Giulia et al. Atrogin-1 and MuRF1 regulate cardiac MyBP-C levels via different mechanisms. Cardiovascular Research, v. 85, n. 2, p. 357-366, 2010Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvp348. Acesso em: 19 out. 2024.
APA
Mearini, G., Gedicke, C., Schlossarek, S., Witt, C. C., Krämer, E., Cao, P., et al. (2010). Atrogin-1 and MuRF1 regulate cardiac MyBP-C levels via different mechanisms. Cardiovascular Research, 85( 2), 357-366. doi:10.1093/cvr/cvp348
NLM
Mearini G, Gedicke C, Schlossarek S, Witt CC, Krämer E, Cao P, Gomes MD, Lecker SH, Labeit S, Willis MS, Eschenhagem T, Carrier L. Atrogin-1 and MuRF1 regulate cardiac MyBP-C levels via different mechanisms [Internet]. Cardiovascular Research. 2010 ; 85( 2): 357-366.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/cvp348
Vancouver
Mearini G, Gedicke C, Schlossarek S, Witt CC, Krämer E, Cao P, Gomes MD, Lecker SH, Labeit S, Willis MS, Eschenhagem T, Carrier L. Atrogin-1 and MuRF1 regulate cardiac MyBP-C levels via different mechanisms [Internet]. Cardiovascular Research. 2010 ; 85( 2): 357-366.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/cvp348
Artigo de periodico
Ischaemic preconditioning improves proteasomal activity and increases the degradation of δPKC during reperfusion (2010)
Churchill, Eric N; Ferreira, Julio Cesar Batista ; Brum, Patrícia Chakur ; Szweda, Luke I.; Mochly-Rosen, Daria
Source: Cardiovascular Research. Unidade: EEFE
Subjects: CARDIOLOGIA, FISIOLOGIA
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ABNT
CHURCHILL, Eric N et al. Ischaemic preconditioning improves proteasomal activity and increases the degradation of δPKC during reperfusion. Cardiovascular Research, v. 85, n. ja 2010, p. 385-394, 2010Tradução . . Disponível em: https://doi.org/10.1093/cvr/cvp334. Acesso em: 19 out. 2024.
APA
Churchill, E. N., Ferreira, J. C. B., Brum, P. C., Szweda, L. I., & Mochly-Rosen, D. (2010). Ischaemic preconditioning improves proteasomal activity and increases the degradation of δPKC during reperfusion. Cardiovascular Research, 85( ja 2010), 385-394. doi:10.1093/cvr/cvp334
NLM
Churchill EN, Ferreira JCB, Brum PC, Szweda LI, Mochly-Rosen D. Ischaemic preconditioning improves proteasomal activity and increases the degradation of δPKC during reperfusion [Internet]. Cardiovascular Research. 2010 ; 85( ja 2010): 385-394.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/cvp334
Vancouver
Churchill EN, Ferreira JCB, Brum PC, Szweda LI, Mochly-Rosen D. Ischaemic preconditioning improves proteasomal activity and increases the degradation of δPKC during reperfusion [Internet]. Cardiovascular Research. 2010 ; 85( ja 2010): 385-394.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/cvp334
Artigo de periodico
Pharmacological and physiological stimuli do not promote 'Ca POT. 2+'-sensitive 'K POT. +' channel activity in isolated heart mitochondria (2007)
Cancherini, Douglas Vasconcelos; Queliconi, Bruno Barros; Kowaltowski, Alicia Juliana
Source: Cardiovascular Research. Unidade: IQ
Subjects: MITOCÔNDRIAS, ISQUEMIA, BIOQUÍMICA
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ABNT
CANCHERINI, Douglas Vasconcelos e QUELICONI, Bruno Barros e KOWALTOWSKI, Alicia Juliana. Pharmacological and physiological stimuli do not promote 'Ca POT. 2+'-sensitive 'K POT. +' channel activity in isolated heart mitochondria. Cardiovascular Research, v. 73, n. 4, p. 720-728, 2007Tradução . . Acesso em: 19 out. 2024.
APA
Cancherini, D. V., Queliconi, B. B., & Kowaltowski, A. J. (2007). Pharmacological and physiological stimuli do not promote 'Ca POT. 2+'-sensitive 'K POT. +' channel activity in isolated heart mitochondria. Cardiovascular Research, 73( 4), 720-728.
NLM
Cancherini DV, Queliconi BB, Kowaltowski AJ. Pharmacological and physiological stimuli do not promote 'Ca POT. 2+'-sensitive 'K POT. +' channel activity in isolated heart mitochondria. Cardiovascular Research. 2007 ; 73( 4): 720-728.[citado 2024 out. 19 ]
Vancouver
Cancherini DV, Queliconi BB, Kowaltowski AJ. Pharmacological and physiological stimuli do not promote 'Ca POT. 2+'-sensitive 'K POT. +' channel activity in isolated heart mitochondria. Cardiovascular Research. 2007 ; 73( 4): 720-728.[citado 2024 out. 19 ]
Artigo de periodico
The Effects of gemfibrozil upon the metabolism of chylomicron-like emulsions in patients with endogenous hypertriglyceridemia (2001)
Santos, Raul D.; Ventura, Laura Ines; Spósito, Andrei Carvalho; Schreiber, Roberto; Ramires, José Antônio Franchini; Maranhão, Raul Cavalcante
Source: Cardiovascular Research. Unidades: FM, FCF
Subjects: CARDIOLOGIA, LIPOPROTEÍNAS
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ABNT
SANTOS, Raul D. et al. The Effects of gemfibrozil upon the metabolism of chylomicron-like emulsions in patients with endogenous hypertriglyceridemia. Cardiovascular Research, v. 49, n. 2, p. 456-465, 2001Tradução . . Disponível em: https://doi.org/10.1016/s0008-6363(00)00274-1. Acesso em: 19 out. 2024.
APA
Santos, R. D., Ventura, L. I., Spósito, A. C., Schreiber, R., Ramires, J. A. F., & Maranhão, R. C. (2001). The Effects of gemfibrozil upon the metabolism of chylomicron-like emulsions in patients with endogenous hypertriglyceridemia. Cardiovascular Research, 49( 2), 456-465. doi:10.1016/s0008-6363(00)00274-1
NLM
Santos RD, Ventura LI, Spósito AC, Schreiber R, Ramires JAF, Maranhão RC. The Effects of gemfibrozil upon the metabolism of chylomicron-like emulsions in patients with endogenous hypertriglyceridemia [Internet]. Cardiovascular Research. 2001 ; 49( 2): 456-465.[citado 2024 out. 19 ] Available from: https://doi.org/10.1016/s0008-6363(00)00274-1
Vancouver
Santos RD, Ventura LI, Spósito AC, Schreiber R, Ramires JAF, Maranhão RC. The Effects of gemfibrozil upon the metabolism of chylomicron-like emulsions in patients with endogenous hypertriglyceridemia [Internet]. Cardiovascular Research. 2001 ; 49( 2): 456-465.[citado 2024 out. 19 ] Available from: https://doi.org/10.1016/s0008-6363(00)00274-1
Artigo de periodico
Oxidative stress as a signaling mechanism of the vascular response to injury: the redox hypothesis of restenosis (2000)
Azevedo, Luciano César Pontes de; Pedro, Marcelo de A.; Souza, Liliete C.; Souza, Heraldo P. de; Janiszewski, Mariano; Luz, Protásio Lemos da; Laurindo, Francisco R. M
Source: Cardiovascular Research. Unidade: FM
Assunto: MEDICINA
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ABNT
AZEVEDO, Luciano César Pontes de et al. Oxidative stress as a signaling mechanism of the vascular response to injury: the redox hypothesis of restenosis. Cardiovascular Research, v. 47, p. 436-445, 2000Tradução . . Disponível em: https://doi.org/10.1016/s0008-6363(00)00091-2. Acesso em: 19 out. 2024.
APA
Azevedo, L. C. P. de, Pedro, M. de A., Souza, L. C., Souza, H. P. de, Janiszewski, M., Luz, P. L. da, & Laurindo, F. R. M. (2000). Oxidative stress as a signaling mechanism of the vascular response to injury: the redox hypothesis of restenosis. Cardiovascular Research, 47, 436-445. doi:10.1016/s0008-6363(00)00091-2
NLM
Azevedo LCP de, Pedro M de A, Souza LC, Souza HP de, Janiszewski M, Luz PL da, Laurindo FRM. Oxidative stress as a signaling mechanism of the vascular response to injury: the redox hypothesis of restenosis [Internet]. Cardiovascular Research. 2000 ; 47 436-445.[citado 2024 out. 19 ] Available from: https://doi.org/10.1016/s0008-6363(00)00091-2
Vancouver
Azevedo LCP de, Pedro M de A, Souza LC, Souza HP de, Janiszewski M, Luz PL da, Laurindo FRM. Oxidative stress as a signaling mechanism of the vascular response to injury: the redox hypothesis of restenosis [Internet]. Cardiovascular Research. 2000 ; 47 436-445.[citado 2024 out. 19 ] Available from: https://doi.org/10.1016/s0008-6363(00)00091-2
Artigo de periodico
Effects of long term metoprolol administration on the eletrocardiogram of rats infected with t. Cruzi (1990)
Bestetti, Reinaldo Bulgarelli; Sales Neto, V N; Pinto, L Z; Soares, E G; Muccillo, Gerson; Oliveira, João Samuel Meira de
Source: Cardiovascular Research. Unidades: FMRP, FFCLRP
Subjects: MATEMÁTICA, PATOLOGIA
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ABNT
BESTETTI, Reinaldo Bulgarelli et al. Effects of long term metoprolol administration on the eletrocardiogram of rats infected with t. Cruzi. Cardiovascular Research, v. 24, p. 521-7, 1990Tradução . . Disponível em: https://doi.org/10.1093/cvr/24.7.521. Acesso em: 19 out. 2024.
APA
Bestetti, R. B., Sales Neto, V. N., Pinto, L. Z., Soares, E. G., Muccillo, G., & Oliveira, J. S. M. de. (1990). Effects of long term metoprolol administration on the eletrocardiogram of rats infected with t. Cruzi. Cardiovascular Research, 24, 521-7. doi:10.1093/cvr/24.7.521
NLM
Bestetti RB, Sales Neto VN, Pinto LZ, Soares EG, Muccillo G, Oliveira JSM de. Effects of long term metoprolol administration on the eletrocardiogram of rats infected with t. Cruzi [Internet]. Cardiovascular Research. 1990 ;24 521-7.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/24.7.521
Vancouver
Bestetti RB, Sales Neto VN, Pinto LZ, Soares EG, Muccillo G, Oliveira JSM de. Effects of long term metoprolol administration on the eletrocardiogram of rats infected with t. Cruzi [Internet]. Cardiovascular Research. 1990 ;24 521-7.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/24.7.521
Artigo de periodico
Ability of the electrocardiogram to detec myocardial lesion in isoproterenol -induced rat cardiomyopathy (1987)
Bestetti, Reinaldo Bulgarelli; Ramos, C P; Figueiredo, J; Sales Neto, V N; Oliveira, João Samuel Meira de
Source: Cardiovascular Research. Unidades: FMRP, COSEAS
Assunto: CARDIOLOGIA
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ABNT
BESTETTI, Reinaldo Bulgarelli et al. Ability of the electrocardiogram to detec myocardial lesion in isoproterenol -induced rat cardiomyopathy. Cardiovascular Research, v. 21, p. 916-21, 1987Tradução . . Disponível em: https://doi.org/10.1093/cvr/21.12.916. Acesso em: 19 out. 2024.
APA
Bestetti, R. B., Ramos, C. P., Figueiredo, J., Sales Neto, V. N., & Oliveira, J. S. M. de. (1987). Ability of the electrocardiogram to detec myocardial lesion in isoproterenol -induced rat cardiomyopathy. Cardiovascular Research, 21, 916-21. doi:10.1093/cvr/21.12.916
NLM
Bestetti RB, Ramos CP, Figueiredo J, Sales Neto VN, Oliveira JSM de. Ability of the electrocardiogram to detec myocardial lesion in isoproterenol -induced rat cardiomyopathy [Internet]. Cardiovascular Research. 1987 ;21 916-21.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/21.12.916
Vancouver
Bestetti RB, Ramos CP, Figueiredo J, Sales Neto VN, Oliveira JSM de. Ability of the electrocardiogram to detec myocardial lesion in isoproterenol -induced rat cardiomyopathy [Internet]. Cardiovascular Research. 1987 ;21 916-21.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/21.12.916
Artigo de periodico
Functional evaluation of sympathetic and parasympathetic system in chagas disease using dynamic exercise (1987)
Gallo Junior, L; Morelo Filho, J; Maciel, B C; Marin Neto, J A; Martins, L E B; Lima Filho, E C
Source: Cardiovascular Research. Unidade: FMRP
Subjects: DOENÇA DE CHAGAS, SISTEMA NERVOSO PARASSIMPÁTICO, SISTEMA NERVOSO SIMPÁTICO, SISTEMA NERVOSO AUTÔNOMO
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ABNT
GALLO JUNIOR, L et al. Functional evaluation of sympathetic and parasympathetic system in chagas disease using dynamic exercise. Cardiovascular Research, v. 21, p. 922-7, 1987Tradução . . Disponível em: https://doi.org/10.1093/cvr/21.12.922. Acesso em: 19 out. 2024.
APA
Gallo Junior, L., Morelo Filho, J., Maciel, B. C., Marin Neto, J. A., Martins, L. E. B., & Lima Filho, E. C. (1987). Functional evaluation of sympathetic and parasympathetic system in chagas disease using dynamic exercise. Cardiovascular Research, 21, 922-7. doi:10.1093/cvr/21.12.922
NLM
Gallo Junior L, Morelo Filho J, Maciel BC, Marin Neto JA, Martins LEB, Lima Filho EC. Functional evaluation of sympathetic and parasympathetic system in chagas disease using dynamic exercise [Internet]. Cardiovascular Research. 1987 ;21 922-7.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/21.12.922
Vancouver
Gallo Junior L, Morelo Filho J, Maciel BC, Marin Neto JA, Martins LEB, Lima Filho EC. Functional evaluation of sympathetic and parasympathetic system in chagas disease using dynamic exercise [Internet]. Cardiovascular Research. 1987 ;21 922-7.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/21.12.922
Artigo de periodico
Parasympathetic contribution to bradycardia induced by endurance training in man (1986)
Maciel, B C; Gallo Junior, L; Marin Netto, J A; Lima Filho, E C; Terra Filho, J; Manço, José Carlos
Source: Cardiovascular Research. Unidade: FMRP
Subjects: FREQUÊNCIA CARDÍACA, TREINAMENTO FÍSICO
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ABNT
MACIEL, B C et al. Parasympathetic contribution to bradycardia induced by endurance training in man. Cardiovascular Research, v. 19, p. 642-8, 1986Tradução . . Disponível em: https://doi.org/10.1093/cvr/19.10.642. Acesso em: 19 out. 2024.
APA
Maciel, B. C., Gallo Junior, L., Marin Netto, J. A., Lima Filho, E. C., Terra Filho, J., & Manço, J. C. (1986). Parasympathetic contribution to bradycardia induced by endurance training in man. Cardiovascular Research, 19, 642-8. doi:10.1093/cvr/19.10.642
NLM
Maciel BC, Gallo Junior L, Marin Netto JA, Lima Filho EC, Terra Filho J, Manço JC. Parasympathetic contribution to bradycardia induced by endurance training in man [Internet]. Cardiovascular Research. 1986 ;19 642-8.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/19.10.642
Vancouver
Maciel BC, Gallo Junior L, Marin Netto JA, Lima Filho EC, Terra Filho J, Manço JC. Parasympathetic contribution to bradycardia induced by endurance training in man [Internet]. Cardiovascular Research. 1986 ;19 642-8.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/19.10.642
Artigo de periodico
Parasympathetic contribution to bradycardia induce by endurance training in man (1985)
Maciel, B C; Gallo Junior, L; Marin Netto, J A; Lima Filho, E C; Terra Filho, J; Manço, José Carlos
Source: Cardiovascular Research. Unidade: FMRP
Subjects: ADAPTAÇÃO FISIOLÓGICA, CONSUMO DE OXIGÊNIO, RESPIRAÇÃO
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ABNT
MACIEL, B C et al. Parasympathetic contribution to bradycardia induce by endurance training in man. Cardiovascular Research, v. 19, n. 10, p. 642-8, 1985Tradução . . Disponível em: https://doi.org/10.1093/cvr/19.10.642. Acesso em: 19 out. 2024.
APA
Maciel, B. C., Gallo Junior, L., Marin Netto, J. A., Lima Filho, E. C., Terra Filho, J., & Manço, J. C. (1985). Parasympathetic contribution to bradycardia induce by endurance training in man. Cardiovascular Research, 19( 10), 642-8. doi:10.1093/cvr/19.10.642
NLM
Maciel BC, Gallo Junior L, Marin Netto JA, Lima Filho EC, Terra Filho J, Manço JC. Parasympathetic contribution to bradycardia induce by endurance training in man [Internet]. Cardiovascular Research. 1985 ;19( 10): 642-8.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/19.10.642
Vancouver
Maciel BC, Gallo Junior L, Marin Netto JA, Lima Filho EC, Terra Filho J, Manço JC. Parasympathetic contribution to bradycardia induce by endurance training in man [Internet]. Cardiovascular Research. 1985 ;19( 10): 642-8.[citado 2024 out. 19 ] Available from: https://doi.org/10.1093/cvr/19.10.642

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