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Artigo de periodico
Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids (2023)
Costa, Tiago Januário da ; Barros, Paula Rodrigues de ; Duarte, Diego A. ; Silva-Neto, Júlio Alves da; Hott, Sara Cristina ; Santos-Silva, Thamyris ; Costa-Neto, Claudio M. ; McCarthy, Cameron G. ; Jiménez-Altayó, Francesc ; Dantas, Ana Paula Villela ; Passaglia, Rita de Cassia Aleixo Tostes ; Akamine, Eliana Hiromi ; Gomes, Felipe Villela
Fonte: American Journal of Physiology. Heart and Circulatory Physiology. Unidades: FMRP, ICB
Assuntos: FARMACOLOGIA, PROSTAGLANDINAS, RECEPTORES ADRENÉRGICOS, MENOPAUSA, FÁRMACOS (SISTEMA CARDIOVASCULAR), ARTÉRIAS CARÓTIDAS, VASOS SANGUÍNEOS, CAMUNDONGOS, VASODILATAÇÃO, MODELOS ANIMAIS DE DOENÇAS, ENVELHECIMENTO
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ABNT
COSTA, Tiago Januário da et al. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, v. 324, n. 4, p. H417-H429, 2023Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00495.2022. Acesso em: 27 jun. 2025.
APA
Costa, T. J. da, Barros, P. R. de, Duarte, D. A., Silva-Neto, J. A. da, Hott, S. C., Santos-Silva, T., et al. (2023). Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids. American Journal of Physiology. Heart and Circulatory Physiology, 324( 4), H417-H429. doi:10.1152/ajpheart.00495.2022
NLM
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 324( 4): H417-H429.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Vancouver
Costa TJ da, Barros PR de, Duarte DA, Silva-Neto JA da, Hott SC, Santos-Silva T, Costa-Neto CM, McCarthy CG, Jiménez-Altayó F, Dantas APV, Passaglia R de CAT, Akamine EH, Gomes FV. Carotid dysfunction in senescent female mice is mediated by increased α1A-adrenoceptor activity and COX-derived vasoconstrictor prostanoids [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 324( 4): H417-H429.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00495.2022
Artigo de periodico
Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor (2023)
Rodrigues, Daniel ; Machado, Mirele Resende ; Alves, Juliano Vilela ; Fraga-Silva, Thais Fernanda de Campos ; Martins, Ronaldo Bragança ; Campos, Lígia Cristina Borges ; Francisco, Daniely Franco ; Couto, Ariel Emiliano Souza do ; Bonato, Vânia Luiza Deperon ; Arruda, Eurico ; Becari, Christiane ; Auxiliadora-Martins, Maria ; Louzada Júnior, Paulo ; Costa, Rafael Menezes da ; Tostes, Rita de Cássia Aleixo
Fonte: American Journal of Physiology. Heart and Circulatory Physiology. Unidades: FCFRP, FMRP, FOB
Assuntos: CÉLULAS ENDOTELIAIS, CITOCINAS, COVID-19, ANORMALIDADES CARDIOVASCULARES
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RODRIGUES, Daniel et al. Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor. American Journal of Physiology. Heart and Circulatory Physiology, v. 325, n. 2, p. H252–H263 Aug. 2023, 2023Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00096.2023. Acesso em: 27 jun. 2025.
APA
Rodrigues, D., Machado, M. R., Alves, J. V., Fraga-Silva, T. F. de C., Martins, R. B., Campos, L. C. B., et al. (2023). Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor. American Journal of Physiology. Heart and Circulatory Physiology, 325( 2), H252–H263 Aug. 2023. doi:10.1152/ajpheart.00096.2023
NLM
Rodrigues D, Machado MR, Alves JV, Fraga-Silva TF de C, Martins RB, Campos LCB, Francisco DF, Couto AES do, Bonato VLD, Arruda E, Becari C, Auxiliadora-Martins M, Louzada Júnior P, Costa RM da, Tostes R de CA. Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 325( 2): H252–H263 Aug. 2023.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00096.2023
Vancouver
Rodrigues D, Machado MR, Alves JV, Fraga-Silva TF de C, Martins RB, Campos LCB, Francisco DF, Couto AES do, Bonato VLD, Arruda E, Becari C, Auxiliadora-Martins M, Louzada Júnior P, Costa RM da, Tostes R de CA. Cytokine storm in individuals with severe COVID-19 decreases endothelial cell antioxidant defense via downregulation of the Nrf2 transcriptional factor [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2023 ; 325( 2): H252–H263 Aug. 2023.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00096.2023
Artigo de periodico
Enhanced sympathetic neurotransduction in the superior mesenteric artery in a rat model of heart failure: role of noradrenaline and ATP (2020)
Rivero, Javier Blanco ; Couto, Gisele Kruger; Paula, Suliana Mesquita ; Fontes, Milene Tavares ; Rossoni, Luciana Venturini
Fonte: American Journal of Physiology. Heart and Circulatory Physiology. Unidade: ICB
Assuntos: FISIOLOGIA, INSUFICIÊNCIA CARDÍACA ANIMAL, NORADRENALINA, MODELOS ANIMAIS DE DOENÇAS, ARTÉRIAS, VASCULARIZAÇÃO EM ANIMAL, ADRENORECEPTORES, RATOS WISTAR, HIDROXILASE
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RIVERO, Javier Blanco et al. Enhanced sympathetic neurotransduction in the superior mesenteric artery in a rat model of heart failure: role of noradrenaline and ATP. American Journal of Physiology. Heart and Circulatory Physiology, 2020Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00444.2020. Acesso em: 27 jun. 2025.
APA
Rivero, J. B., Couto, G. K., Paula, S. M., Fontes, M. T., & Rossoni, L. V. (2020). Enhanced sympathetic neurotransduction in the superior mesenteric artery in a rat model of heart failure: role of noradrenaline and ATP. American Journal of Physiology. Heart and Circulatory Physiology. doi:10.1152/ajpheart.00444.2020
NLM
Rivero JB, Couto GK, Paula SM, Fontes MT, Rossoni LV. Enhanced sympathetic neurotransduction in the superior mesenteric artery in a rat model of heart failure: role of noradrenaline and ATP [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2020 ;[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00444.2020
Vancouver
Rivero JB, Couto GK, Paula SM, Fontes MT, Rossoni LV. Enhanced sympathetic neurotransduction in the superior mesenteric artery in a rat model of heart failure: role of noradrenaline and ATP [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2020 ;[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00444.2020
Artigo de periodico
Autonomic cardiocirculatory control in mice with reduced expression of the vesicular acetylcholine transporter (2015)
Durand, Marina Toledo; Becari, Christiane; Tezini, Geisa Cristina Singolani Vaccari; Fazan Júnior, Rubens; Oliveira, Mauro; Guatimosim, Silvia; Prado, Vania F.; Prado, Marco A. M.; Salgado, Hélio César
Fonte: American Journal of Physiology. Heart and Circulatory Physiology. Unidade: FMRP
Assuntos: FREQUÊNCIA CARDÍACA, HEMODINÂMICA, DOENÇAS CARDIOVASCULARES, COLINÉRGICOS, SISTEMA NERVOSO PARASSIMPÁTICO, MODELOS ANIMAIS
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ABNT
DURAND, Marina Toledo et al. Autonomic cardiocirculatory control in mice with reduced expression of the vesicular acetylcholine transporter. American Journal of Physiology. Heart and Circulatory Physiology, v. 308, p. H655-H662, 2015Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00114.2015. Acesso em: 27 jun. 2025.
APA
Durand, M. T., Becari, C., Tezini, G. C. S. V., Fazan Júnior, R., Oliveira, M., Guatimosim, S., et al. (2015). Autonomic cardiocirculatory control in mice with reduced expression of the vesicular acetylcholine transporter. American Journal of Physiology. Heart and Circulatory Physiology, 308, H655-H662. doi:10.1152/ajpheart.00114.2015
NLM
Durand MT, Becari C, Tezini GCSV, Fazan Júnior R, Oliveira M, Guatimosim S, Prado VF, Prado MAM, Salgado HC. Autonomic cardiocirculatory control in mice with reduced expression of the vesicular acetylcholine transporter [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2015 ; 308 H655-H662.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00114.2015
Vancouver
Durand MT, Becari C, Tezini GCSV, Fazan Júnior R, Oliveira M, Guatimosim S, Prado VF, Prado MAM, Salgado HC. Autonomic cardiocirculatory control in mice with reduced expression of the vesicular acetylcholine transporter [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2015 ; 308 H655-H662.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00114.2015
Artigo de periodico
Dysautonomias in Parkinson's disease: cardiovascular changes and autonomic modulation in conscious rats after infusion of bilateral 6-OHDA in substantia nigra (2015)
Ariza, D.; Sisdeli, L.; Crestani, Carlos César; Fazan Júnior, Rubens; Martins-Pinge, M. C.
Fonte: American Journal of Physiology. Heart and Circulatory Physiology. Unidade: FMRP
Assuntos: DOENÇA DE PARKINSON, SISTEMA CARDIOVASCULAR, PRESSORECEPTORES, AGENTES DOPAMINÉRGICOS, MODELOS ANIMAIS
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ABNT
ARIZA, D. et al. Dysautonomias in Parkinson's disease: cardiovascular changes and autonomic modulation in conscious rats after infusion of bilateral 6-OHDA in substantia nigra. American Journal of Physiology. Heart and Circulatory Physiology, v. 308, n. 3, p. H250-H257, 2015Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00406.2014. Acesso em: 27 jun. 2025.
APA
Ariza, D., Sisdeli, L., Crestani, C. C., Fazan Júnior, R., & Martins-Pinge, M. C. (2015). Dysautonomias in Parkinson's disease: cardiovascular changes and autonomic modulation in conscious rats after infusion of bilateral 6-OHDA in substantia nigra. American Journal of Physiology. Heart and Circulatory Physiology, 308( 3), H250-H257. doi:10.1152/ajpheart.00406.2014
NLM
Ariza D, Sisdeli L, Crestani CC, Fazan Júnior R, Martins-Pinge MC. Dysautonomias in Parkinson's disease: cardiovascular changes and autonomic modulation in conscious rats after infusion of bilateral 6-OHDA in substantia nigra [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2015 ; 308( 3): H250-H257.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00406.2014
Vancouver
Ariza D, Sisdeli L, Crestani CC, Fazan Júnior R, Martins-Pinge MC. Dysautonomias in Parkinson's disease: cardiovascular changes and autonomic modulation in conscious rats after infusion of bilateral 6-OHDA in substantia nigra [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2015 ; 308( 3): H250-H257.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00406.2014
Artigo de periodico
Cholinergic stimulation with pyridostigmine protects myocardial infarcted rats against ischemic-induced arrhythmias and preserves connexin43 protein (2015)
Santos-Almeida, Fernanda Machado; Girão, Henrique; Silva, Carlos Alberto Aguiar da; Salgado, Hélio César; Fazan Júnior, Rubens
Fonte: American Journal of Physiology. Heart and Circulatory Physiology. Unidade: FMRP
Assuntos: ANTICOLINÉRGICOS, ARRITMIA, INFARTO DO MIOCÁRDIO, MODELOS ANIMAIS, SISTEMA NERVOSO PARASSIMPÁTICO
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ABNT
SANTOS-ALMEIDA, Fernanda Machado et al. Cholinergic stimulation with pyridostigmine protects myocardial infarcted rats against ischemic-induced arrhythmias and preserves connexin43 protein. American Journal of Physiology. Heart and Circulatory Physiology, v. 308, n. 2, p. H101-H107, 2015Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00591.2014. Acesso em: 27 jun. 2025.
APA
Santos-Almeida, F. M., Girão, H., Silva, C. A. A. da, Salgado, H. C., & Fazan Júnior, R. (2015). Cholinergic stimulation with pyridostigmine protects myocardial infarcted rats against ischemic-induced arrhythmias and preserves connexin43 protein. American Journal of Physiology. Heart and Circulatory Physiology, 308( 2), H101-H107. doi:10.1152/ajpheart.00591.2014
NLM
Santos-Almeida FM, Girão H, Silva CAA da, Salgado HC, Fazan Júnior R. Cholinergic stimulation with pyridostigmine protects myocardial infarcted rats against ischemic-induced arrhythmias and preserves connexin43 protein [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2015 ; 308( 2): H101-H107.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00591.2014
Vancouver
Santos-Almeida FM, Girão H, Silva CAA da, Salgado HC, Fazan Júnior R. Cholinergic stimulation with pyridostigmine protects myocardial infarcted rats against ischemic-induced arrhythmias and preserves connexin43 protein [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2015 ; 308( 2): H101-H107.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00591.2014
Artigo de periodico
Testosterone induces apoptosis in vascular smooth muscle cells via extrinsic apoptotic pathway with mitochondria-generated reactive oxygen species involvement (2014)
Lopes, Rheure Alves Moreira; Neves, Karla Bianca; Pestana, Cezar Rangel; Queiroz, André Lima; Zanotto, Camila Ziliotto; Chignalia, Andréia Zago; Valim, Yara Maria Lucisano; Silveira, Leonardo dos Reis; Curti, Carlos; Passaglia, Rita de Cassia Aleixo Tostes
Fonte: American Journal of Physiology. Heart and Circulatory Physiology. Unidades: FCFRP, EEFERP, FMRP
Assuntos: TESTOSTERONA, APOPTOSE, CÉLULAS MUSCULARES, MITOCÔNDRIAS
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LOPES, Rheure Alves Moreira et al. Testosterone induces apoptosis in vascular smooth muscle cells via extrinsic apoptotic pathway with mitochondria-generated reactive oxygen species involvement. American Journal of Physiology. Heart and Circulatory Physiology, v. 306, n. 11, p. H1485-H1494, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00809.2013. Acesso em: 27 jun. 2025.
APA
Lopes, R. A. M., Neves, K. B., Pestana, C. R., Queiroz, A. L., Zanotto, C. Z., Chignalia, A. Z., et al. (2014). Testosterone induces apoptosis in vascular smooth muscle cells via extrinsic apoptotic pathway with mitochondria-generated reactive oxygen species involvement. American Journal of Physiology. Heart and Circulatory Physiology, 306( 11), H1485-H1494. doi:10.1152/ajpheart.00809.2013
NLM
Lopes RAM, Neves KB, Pestana CR, Queiroz AL, Zanotto CZ, Chignalia AZ, Valim YML, Silveira L dos R, Curti C, Passaglia R de CAT. Testosterone induces apoptosis in vascular smooth muscle cells via extrinsic apoptotic pathway with mitochondria-generated reactive oxygen species involvement [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2014 ; 306( 11): H1485-H1494.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00809.2013
Vancouver
Lopes RAM, Neves KB, Pestana CR, Queiroz AL, Zanotto CZ, Chignalia AZ, Valim YML, Silveira L dos R, Curti C, Passaglia R de CAT. Testosterone induces apoptosis in vascular smooth muscle cells via extrinsic apoptotic pathway with mitochondria-generated reactive oxygen species involvement [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2014 ; 306( 11): H1485-H1494.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00809.2013
Artigo de periodico
Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart (2002)
Santos, Pierre dos; Kowaltowski, Alicia Juliana ; Laclau, Muriel N.; Seetharaman, Subramanian; Paucek, Petr; Boudina, Sihem; Thambo, Jean-Benoit; Tariosse, Liliane; Garlid, Keith D.
Fonte: American Journal of Physiology. Heart and Circulatory Physiology. Unidade: IQ
Assuntos: BIOQUÍMICA, MITOCÔNDRIAS, METABOLISMO
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ABNT
SANTOS, Pierre dos et al. Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart. American Journal of Physiology. Heart and Circulatory Physiology, v. 283, n. 1, p. H282-H295, 2002Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00034.2002. Acesso em: 27 jun. 2025.
APA
Santos, P. dos, Kowaltowski, A. J., Laclau, M. N., Seetharaman, S., Paucek, P., Boudina, S., et al. (2002). Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart. American Journal of Physiology. Heart and Circulatory Physiology, 283( 1), H282-H295. doi:10.1152/ajpheart.00034.2002
NLM
Santos P dos, Kowaltowski AJ, Laclau MN, Seetharaman S, Paucek P, Boudina S, Thambo J-B, Tariosse L, Garlid KD. Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2002 ; 283( 1): H282-H295.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00034.2002
Vancouver
Santos P dos, Kowaltowski AJ, Laclau MN, Seetharaman S, Paucek P, Boudina S, Thambo J-B, Tariosse L, Garlid KD. Mechanisms by which opening the mitochondrial ATP- sensitive 'K POT. +' channel protects the ischemic heart [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 2002 ; 283( 1): H282-H295.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.00034.2002
Artigo de periodico
Losartan improves baroreflex control of heart rate of coarcted hypertensive rats (1995)
Santos, C M M; Pontieri, V; Leomil Neto, M.; Michelini, Lisete Compagno
Fonte: American Journal of Physiology. Heart and Circulatory Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
SANTOS, C M M et al. Losartan improves baroreflex control of heart rate of coarcted hypertensive rats. American Journal of Physiology. Heart and Circulatory Physiology, v. 38, n. 3 , p. h.812-8, 1995Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.1995.269.3.h812. Acesso em: 27 jun. 2025.
APA
Santos, C. M. M., Pontieri, V., Leomil Neto, M., & Michelini, L. C. (1995). Losartan improves baroreflex control of heart rate of coarcted hypertensive rats. American Journal of Physiology. Heart and Circulatory Physiology, 38( 3 ), h.812-8. doi:10.1152/ajpheart.1995.269.3.h812
NLM
Santos CMM, Pontieri V, Leomil Neto M, Michelini LC. Losartan improves baroreflex control of heart rate of coarcted hypertensive rats [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 1995 ;38( 3 ): h.812-8.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.1995.269.3.h812
Vancouver
Santos CMM, Pontieri V, Leomil Neto M, Michelini LC. Losartan improves baroreflex control of heart rate of coarcted hypertensive rats [Internet]. American Journal of Physiology. Heart and Circulatory Physiology. 1995 ;38( 3 ): h.812-8.[citado 2025 jun. 27 ] Available from: https://doi.org/10.1152/ajpheart.1995.269.3.h812

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