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Artigo de periodico
Profiling the immune epigenome across global cattle breeds (2023)
Powell, Jessica; Talenti, Andrea; Fisch, Andressa ; Hemmink, Johanneke D.; Paxton, Edith; Toye, Philip; Santos, Isabel Kinney Ferreira de Miranda ; Ferreira, Beatriz Rossetti ; Connelley, Tim K.; Morrison, Liam J.; Prendergast, James G. D.
Source: Genome Biology. Unidades: FMRP, EERP
Subjects: BOVINOS, METILAÇÃO, DNA, CROMATINA, CÉLULAS, IMUNOLOGIA
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POWELL, Jessica et al. Profiling the immune epigenome across global cattle breeds. Genome Biology, v. 24, n. 1, p. 1-31, 2023Tradução . . Disponível em: https://doi.org/10.1186/s13059-023-02964-3. Acesso em: 02 nov. 2024.
APA
Powell, J., Talenti, A., Fisch, A., Hemmink, J. D., Paxton, E., Toye, P., et al. (2023). Profiling the immune epigenome across global cattle breeds. Genome Biology, 24( 1), 1-31. doi:10.1186/s13059-023-02964-3
NLM
Powell J, Talenti A, Fisch A, Hemmink JD, Paxton E, Toye P, Santos IKF de M, Ferreira BR, Connelley TK, Morrison LJ, Prendergast JGD. Profiling the immune epigenome across global cattle breeds [Internet]. Genome Biology. 2023 ; 24( 1): 1-31.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1186/s13059-023-02964-3
Vancouver
Powell J, Talenti A, Fisch A, Hemmink JD, Paxton E, Toye P, Santos IKF de M, Ferreira BR, Connelley TK, Morrison LJ, Prendergast JGD. Profiling the immune epigenome across global cattle breeds [Internet]. Genome Biology. 2023 ; 24( 1): 1-31.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1186/s13059-023-02964-3
Artigo de periodico
The K/HDEL receptor does not recycle but instead acts as a Golgi-gatekeeper (2023)
Alvim, Jonas C.; Bolt, Robert M.; An, Jing; Kamisugi, Yasuko; Cuming, Andrew; Silva-Alvim, Fernanda A. L.; Casaverde, Juan Oswaldo Concha; Silva, Luis Lamberti Pinto da ; Hu, Meiyi; Hirsz, Dominique; Denecke, Jurgen
Source: Nature Communications. Unidade: FMRP
Subjects: PROTEÍNAS DE TRANSPORTE, COMPLEXO DE GOLGI, LIGANTES, PEPTÍDEOS
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ALVIM, Jonas C. et al. The K/HDEL receptor does not recycle but instead acts as a Golgi-gatekeeper. Nature Communications, v. 14, p. 1-16, 2023Tradução . . Disponível em: https://doi.org/10.1038/s41467-023-37056-0. Acesso em: 02 nov. 2024.
APA
Alvim, J. C., Bolt, R. M., An, J., Kamisugi, Y., Cuming, A., Silva-Alvim, F. A. L., et al. (2023). The K/HDEL receptor does not recycle but instead acts as a Golgi-gatekeeper. Nature Communications, 14, 1-16. doi:10.1038/s41467-023-37056-0
NLM
Alvim JC, Bolt RM, An J, Kamisugi Y, Cuming A, Silva-Alvim FAL, Casaverde JOC, Silva LLP da, Hu M, Hirsz D, Denecke J. The K/HDEL receptor does not recycle but instead acts as a Golgi-gatekeeper [Internet]. Nature Communications. 2023 ; 14 1-16.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1038/s41467-023-37056-0
Vancouver
Alvim JC, Bolt RM, An J, Kamisugi Y, Cuming A, Silva-Alvim FAL, Casaverde JOC, Silva LLP da, Hu M, Hirsz D, Denecke J. The K/HDEL receptor does not recycle but instead acts as a Golgi-gatekeeper [Internet]. Nature Communications. 2023 ; 14 1-16.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1038/s41467-023-37056-0
Artigo de periodico
Mechanisms of bunyavirus morphogenesis and egress (2023)
Barker, Jake; Silva, Luis Lamberti Pinto da ; Crump, Colin M.
Source: Journal of General Virology. Unidade: FMRP
Subjects: BUNYAVIRIDAE, VÍRUS DE RNA, MORFOGÊNESE
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BARKER, Jake e SILVA, Luis Lamberti Pinto da e CRUMP, Colin M. Mechanisms of bunyavirus morphogenesis and egress. Journal of General Virology, v. 104, n. 4, p. 1-11, 2023Tradução . . Disponível em: https://doi.org/10.1099/jgv.0.001845. Acesso em: 02 nov. 2024.
APA
Barker, J., Silva, L. L. P. da, & Crump, C. M. (2023). Mechanisms of bunyavirus morphogenesis and egress. Journal of General Virology, 104( 4), 1-11. doi:10.1099/jgv.0.001845
NLM
Barker J, Silva LLP da, Crump CM. Mechanisms of bunyavirus morphogenesis and egress [Internet]. Journal of General Virology. 2023 ; 104( 4): 1-11.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1099/jgv.0.001845
Vancouver
Barker J, Silva LLP da, Crump CM. Mechanisms of bunyavirus morphogenesis and egress [Internet]. Journal of General Virology. 2023 ; 104( 4): 1-11.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1099/jgv.0.001845
Artigo de periodico
Optical mapping compendium of structural variants across global cattle breeds (2022)
Talenti, A.; Powell, J.; Wragg, D.; Chepkwony, M.; Fisch, Andressa ; Ferreira, Beatriz Rossetti ; Mercadante, M. E. Z.; Santos, Isabel Kinney Ferreira de Miranda; Ezeasor, C. K.; Obishakin, E. T.; Muhanguzi, D.; Amanyire, W.; Silwamba, I.; Muma, J. B.; Mainda, G.; Kelly, R. F.; Toye, P.; Connelley, T.; Prendergast, J.
Source: Scientific Data. Unidades: EERP, FMRP
Subjects: COMPÊNDIOS, RAÇAS ANIMAIS, GADO, VARIAÇÕES ESTRUTURAIS
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TALENTI, A. et al. Optical mapping compendium of structural variants across global cattle breeds. Scientific Data, v. 9, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41597-022-01684-w. Acesso em: 02 nov. 2024.
APA
Talenti, A., Powell, J., Wragg, D., Chepkwony, M., Fisch, A., Ferreira, B. R., et al. (2022). Optical mapping compendium of structural variants across global cattle breeds. Scientific Data, 9, 1-8. doi:10.1038/s41597-022-01684-w
NLM
Talenti A, Powell J, Wragg D, Chepkwony M, Fisch A, Ferreira BR, Mercadante MEZ, Santos IKF de M, Ezeasor CK, Obishakin ET, Muhanguzi D, Amanyire W, Silwamba I, Muma JB, Mainda G, Kelly RF, Toye P, Connelley T, Prendergast J. Optical mapping compendium of structural variants across global cattle breeds [Internet]. Scientific Data. 2022 ; 9 1-8.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1038/s41597-022-01684-w
Vancouver
Talenti A, Powell J, Wragg D, Chepkwony M, Fisch A, Ferreira BR, Mercadante MEZ, Santos IKF de M, Ezeasor CK, Obishakin ET, Muhanguzi D, Amanyire W, Silwamba I, Muma JB, Mainda G, Kelly RF, Toye P, Connelley T, Prendergast J. Optical mapping compendium of structural variants across global cattle breeds [Internet]. Scientific Data. 2022 ; 9 1-8.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1038/s41597-022-01684-w
Artigo de periodico
Differences in transporters rather than drug targets are the principal determinants of the different innate sensitivities of Trypanosoma congolense and Trypanozoon subgenus Trypanosomes to diamidines and melaminophenyl arsenicals (2022)
Ungogo, Marzuq A.; Campagnaro, Gustavo Daniel; Alghamdi, Ali H.; Natto, Manal J.; Koning, Harry P. de
Source: International Journal of Molecular Sciences. Unidade: FMRP
Subjects: TRYPANOSOMA, TRIPANOSSOMICIDAS, EXPRESSÃO GÊNICA, FÁRMACOS
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UNGOGO, Marzuq A. et al. Differences in transporters rather than drug targets are the principal determinants of the different innate sensitivities of Trypanosoma congolense and Trypanozoon subgenus Trypanosomes to diamidines and melaminophenyl arsenicals. International Journal of Molecular Sciences, v. 23, n. 5, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.3390/ijms23052844. Acesso em: 02 nov. 2024.
APA
Ungogo, M. A., Campagnaro, G. D., Alghamdi, A. H., Natto, M. J., & Koning, H. P. de. (2022). Differences in transporters rather than drug targets are the principal determinants of the different innate sensitivities of Trypanosoma congolense and Trypanozoon subgenus Trypanosomes to diamidines and melaminophenyl arsenicals. International Journal of Molecular Sciences, 23( 5), 1-17. doi:10.3390/ijms23052844
NLM
Ungogo MA, Campagnaro GD, Alghamdi AH, Natto MJ, Koning HP de. Differences in transporters rather than drug targets are the principal determinants of the different innate sensitivities of Trypanosoma congolense and Trypanozoon subgenus Trypanosomes to diamidines and melaminophenyl arsenicals [Internet]. International Journal of Molecular Sciences. 2022 ; 23( 5): 1-17.[citado 2024 nov. 02 ] Available from: https://doi.org/10.3390/ijms23052844
Vancouver
Ungogo MA, Campagnaro GD, Alghamdi AH, Natto MJ, Koning HP de. Differences in transporters rather than drug targets are the principal determinants of the different innate sensitivities of Trypanosoma congolense and Trypanozoon subgenus Trypanosomes to diamidines and melaminophenyl arsenicals [Internet]. International Journal of Molecular Sciences. 2022 ; 23( 5): 1-17.[citado 2024 nov. 02 ] Available from: https://doi.org/10.3390/ijms23052844
Artigo de periodico
GLP1R attenuates sympathetic response to high glucose via carotid body inhibition (2022)
Pauza, Audrys G.; Thakkar, Pratik; Tasic, Tatjana; Felippe, Igor; Bishop, Paul; Greenwood, Michael P.; Rysevaite-Kyguoliene, Kristina; Ast, Julia; Broichhagen, Johannes; Hodson, David J.; Salgado, Hélio César ; Pauza, Dainius H.; Japundzic-Zigon, Nina; Paton, Julian F. R.; Murphy, David
Source: Circulation Research. Unidade: FMRP
Subjects: HIPERTENSÃO, INSULINA, OBESIDADE, FATORES DE RISCO, GLICOSE
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PAUZA, Audrys G. et al. GLP1R attenuates sympathetic response to high glucose via carotid body inhibition. Circulation Research, v. 130, n. 5, p. 694-707, 2022Tradução . . Disponível em: https://doi.org/10.1161/CIRCRESAHA.121.319874. Acesso em: 02 nov. 2024.
APA
Pauza, A. G., Thakkar, P., Tasic, T., Felippe, I., Bishop, P., Greenwood, M. P., et al. (2022). GLP1R attenuates sympathetic response to high glucose via carotid body inhibition. Circulation Research, 130( 5), 694-707. doi:10.1161/CIRCRESAHA.121.319874
NLM
Pauza AG, Thakkar P, Tasic T, Felippe I, Bishop P, Greenwood MP, Rysevaite-Kyguoliene K, Ast J, Broichhagen J, Hodson DJ, Salgado HC, Pauza DH, Japundzic-Zigon N, Paton JFR, Murphy D. GLP1R attenuates sympathetic response to high glucose via carotid body inhibition [Internet]. Circulation Research. 2022 ; 130( 5): 694-707.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/CIRCRESAHA.121.319874
Vancouver
Pauza AG, Thakkar P, Tasic T, Felippe I, Bishop P, Greenwood MP, Rysevaite-Kyguoliene K, Ast J, Broichhagen J, Hodson DJ, Salgado HC, Pauza DH, Japundzic-Zigon N, Paton JFR, Murphy D. GLP1R attenuates sympathetic response to high glucose via carotid body inhibition [Internet]. Circulation Research. 2022 ; 130( 5): 694-707.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1161/CIRCRESAHA.121.319874
Artigo de periodico
Effects of gonadotropin inducible ovarian transcription factor 1 in the paraventricular nucleus on fluid intake after dehydration of ovariectomized female rats (2021)
Santos, Ana Luiza Romero Terra dos ; Reis, Wagner Luis; Cognuck, Susana Quirós; Lima, Juliana Bezerra Medeiros de; Domingues, Juliana Tonietto; Araújo, Leonardo Domingues de; Greenwood, Michael Paul; Greenwood, Mingkwan; Elias, Lucila Leico Kagohara ; Murphy, David; Antunes-Rodrigues, José
Source: Experimental Physiology. Unidade: FMRP
Subjects: ARGININA, GONADOTROFINAS, DESIDRATAÇÃO, OVÁRIO, FATORES DE TRANSCRIÇÃO
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SANTOS, Ana Luiza Romero Terra dos et al. Effects of gonadotropin inducible ovarian transcription factor 1 in the paraventricular nucleus on fluid intake after dehydration of ovariectomized female rats. Experimental Physiology, v. 106, n. 12, p. 2391-2399, 2021Tradução . . Disponível em: https://doi.org/10.1113/EP089890. Acesso em: 02 nov. 2024.
APA
Santos, A. L. R. T. dos, Reis, W. L., Cognuck, S. Q., Lima, J. B. M. de, Domingues, J. T., Araújo, L. D. de, et al. (2021). Effects of gonadotropin inducible ovarian transcription factor 1 in the paraventricular nucleus on fluid intake after dehydration of ovariectomized female rats. Experimental Physiology, 106( 12), 2391-2399. doi:10.1113/EP089890
NLM
Santos ALRT dos, Reis WL, Cognuck SQ, Lima JBM de, Domingues JT, Araújo LD de, Greenwood MP, Greenwood M, Elias LLK, Murphy D, Antunes-Rodrigues J. Effects of gonadotropin inducible ovarian transcription factor 1 in the paraventricular nucleus on fluid intake after dehydration of ovariectomized female rats [Internet]. Experimental Physiology. 2021 ; 106( 12): 2391-2399.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1113/EP089890
Vancouver
Santos ALRT dos, Reis WL, Cognuck SQ, Lima JBM de, Domingues JT, Araújo LD de, Greenwood MP, Greenwood M, Elias LLK, Murphy D, Antunes-Rodrigues J. Effects of gonadotropin inducible ovarian transcription factor 1 in the paraventricular nucleus on fluid intake after dehydration of ovariectomized female rats [Internet]. Experimental Physiology. 2021 ; 106( 12): 2391-2399.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1113/EP089890
Artigo de periodico
Physiological and transcriptomic changes in the hypothalamic-neurohypophysial system after 24 h of furosemide-induced sodium depletion (2021)
Dutra, Sabrina G. V; Paterson, Alex; Monteiro, Livia R. N; Greenwood, Michael P; Greenwood, Mingkwan P; Amaral, Ludimila S; Melo, Mariana R; Colombari, Débora S. A; Colombari, Eduardo; Reis, Luís C; Hindmarch, Charles C. T; Elias, Lucila Leico Kagohara; Antunes-Rodrigues, José; Murphy, David; Mecawi, Andre S.
Source: Neuroendocrinology. Unidade: FMRP
Subjects: HIPOTÁLAMO, OCITOCINA, APETITE, SÓDIO, VASOPRESSINAS
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DUTRA, Sabrina G. V et al. Physiological and transcriptomic changes in the hypothalamic-neurohypophysial system after 24 h of furosemide-induced sodium depletion. Neuroendocrinology, v. 111, n. 1-2, p. 70-86, 2021Tradução . . Disponível em: https://doi.org/10.1159/000505997. Acesso em: 02 nov. 2024.
APA
Dutra, S. G. V., Paterson, A., Monteiro, L. R. N., Greenwood, M. P., Greenwood, M. P., Amaral, L. S., et al. (2021). Physiological and transcriptomic changes in the hypothalamic-neurohypophysial system after 24 h of furosemide-induced sodium depletion. Neuroendocrinology, 111( 1-2), 70-86. doi:10.1159/000505997
NLM
Dutra S GV, Paterson A, Monteiro L RN, Greenwood M P, Greenwood M P, Amaral L S, Melo M R, Colombari D SA, Colombari E, Reis L C, Hindmarch C CT, Elias LLK, Antunes-Rodrigues J, Murphy D, Mecawi A S. Physiological and transcriptomic changes in the hypothalamic-neurohypophysial system after 24 h of furosemide-induced sodium depletion [Internet]. Neuroendocrinology. 2021 ; 111( 1-2): 70-86.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1159/000505997
Vancouver
Dutra S GV, Paterson A, Monteiro L RN, Greenwood M P, Greenwood M P, Amaral L S, Melo M R, Colombari D SA, Colombari E, Reis L C, Hindmarch C CT, Elias LLK, Antunes-Rodrigues J, Murphy D, Mecawi A S. Physiological and transcriptomic changes in the hypothalamic-neurohypophysial system after 24 h of furosemide-induced sodium depletion [Internet]. Neuroendocrinology. 2021 ; 111( 1-2): 70-86.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1159/000505997
Artigo de periodico
The E3 ubiquitin ligase SCF(Fbxo7) mediates proteasomal degradation of UXT isoform 2 (UXT-V2) to inhibit the NF-κB signaling pathway (2021)
Spagnol, Valentine; Oliveira, Caio A. B.; Randle, Suzanne J.; Passos, Patrícia M. S.; Correia, Camila R. S. T. B.; Simaroli, Natália B.; Oliveira, Joice S.; Mevissen, Tycho E. T.; Medeiros, Ana Carla; Gomes, Marcelo Damário
Source: Biochimica et Biophysica Acta. General Subjects. Unidade: FMRP
Subjects: PROTEÍNAS, CICLO CELULAR, LIGASES, LINHAGEM CELULAR, METABOLISMO DE PROTEÍNA, TRANSDUÇÃO DE SINAL CELULAR
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SPAGNOL, Valentine et al. The E3 ubiquitin ligase SCF(Fbxo7) mediates proteasomal degradation of UXT isoform 2 (UXT-V2) to inhibit the NF-κB signaling pathway. Biochimica et Biophysica Acta. General Subjects, v. 1865, n. 1, p. 1-12, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.bbagen.2020.129754. Acesso em: 02 nov. 2024.
APA
Spagnol, V., Oliveira, C. A. B., Randle, S. J., Passos, P. M. S., Correia, C. R. S. T. B., Simaroli, N. B., et al. (2021). The E3 ubiquitin ligase SCF(Fbxo7) mediates proteasomal degradation of UXT isoform 2 (UXT-V2) to inhibit the NF-κB signaling pathway. Biochimica et Biophysica Acta. General Subjects, 1865( 1), 1-12. doi:10.1016/j.bbagen.2020.129754
NLM
Spagnol V, Oliveira CAB, Randle SJ, Passos PMS, Correia CRSTB, Simaroli NB, Oliveira JS, Mevissen TET, Medeiros AC, Gomes MD. The E3 ubiquitin ligase SCF(Fbxo7) mediates proteasomal degradation of UXT isoform 2 (UXT-V2) to inhibit the NF-κB signaling pathway [Internet]. Biochimica et Biophysica Acta. General Subjects. 2021 ; 1865( 1): 1-12.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.bbagen.2020.129754
Vancouver
Spagnol V, Oliveira CAB, Randle SJ, Passos PMS, Correia CRSTB, Simaroli NB, Oliveira JS, Mevissen TET, Medeiros AC, Gomes MD. The E3 ubiquitin ligase SCF(Fbxo7) mediates proteasomal degradation of UXT isoform 2 (UXT-V2) to inhibit the NF-κB signaling pathway [Internet]. Biochimica et Biophysica Acta. General Subjects. 2021 ; 1865( 1): 1-12.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.bbagen.2020.129754
Artigo de periodico
Trypanosoma brucei ATR links DNA damage signaling during antigenic variation with regulation of RNA polymerase I-transcribed surface antigens (2020)
Black, Jennifer Ann; Crouch, Kathryn; Lemgruber, Leandro; Lapsley, Craig; Dickens, Nicholas; Tosi, Luíz Ricardo Orsini ; Mottram, Jeremy C.; McCulloch, Richard
Source: Cell Reports. Unidade: FMRP
Subjects: ALELOS, VARIAÇÃO GENÉTICA, NÚCLEO CELULAR, PROLIFERAÇÃO CELULAR, SOBREVIVÊNCIA CELULAR, DANO AO DNA, GENOMAS, RNA POLIMERASES
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BLACK, Jennifer Ann et al. Trypanosoma brucei ATR links DNA damage signaling during antigenic variation with regulation of RNA polymerase I-transcribed surface antigens. Cell Reports, v. 30, n. 3, p. 836-851, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2019.12.049. Acesso em: 02 nov. 2024.
APA
Black, J. A., Crouch, K., Lemgruber, L., Lapsley, C., Dickens, N., Tosi, L. R. O., et al. (2020). Trypanosoma brucei ATR links DNA damage signaling during antigenic variation with regulation of RNA polymerase I-transcribed surface antigens. Cell Reports, 30( 3), 836-851. doi:10.1016/j.celrep.2019.12.049
NLM
Black JA, Crouch K, Lemgruber L, Lapsley C, Dickens N, Tosi LRO, Mottram JC, McCulloch R. Trypanosoma brucei ATR links DNA damage signaling during antigenic variation with regulation of RNA polymerase I-transcribed surface antigens [Internet]. Cell Reports. 2020 ; 30( 3): 836-851.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.celrep.2019.12.049
Vancouver
Black JA, Crouch K, Lemgruber L, Lapsley C, Dickens N, Tosi LRO, Mottram JC, McCulloch R. Trypanosoma brucei ATR links DNA damage signaling during antigenic variation with regulation of RNA polymerase I-transcribed surface antigens [Internet]. Cell Reports. 2020 ; 30( 3): 836-851.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.celrep.2019.12.049
Artigo de periodico
Conditional knockout of RAD51-related genes in Leishmania major reveals a critical role for homologous recombination during genome replication (2020)
Damasceno, Jeziel D.; Reis-Cunha, João; Crouch, Kathryn; Beraldi, Dario; Lapsley, Craig; Tosi, Luíz Ricardo Orsini ; Bartholomeu, Daniella Castanheira; McCulloch, Richard
Source: PLOS Genetics. Unidade: FMRP
Subjects: DANO AO DNA, GENOMAS, LEISHMANIA, LEISHMANIOSE CUTÂNEA, PARASITOLOGIA
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DAMASCENO, Jeziel D. et al. Conditional knockout of RAD51-related genes in Leishmania major reveals a critical role for homologous recombination during genome replication. PLOS Genetics, v. 16, n. 7, 2020Tradução . . Disponível em: https://doi.org/10.1371/journal.pgen.1008828. Acesso em: 02 nov. 2024.
APA
Damasceno, J. D., Reis-Cunha, J., Crouch, K., Beraldi, D., Lapsley, C., Tosi, L. R. O., et al. (2020). Conditional knockout of RAD51-related genes in Leishmania major reveals a critical role for homologous recombination during genome replication. PLOS Genetics, 16( 7). doi:10.1371/journal.pgen.1008828
NLM
Damasceno JD, Reis-Cunha J, Crouch K, Beraldi D, Lapsley C, Tosi LRO, Bartholomeu DC, McCulloch R. Conditional knockout of RAD51-related genes in Leishmania major reveals a critical role for homologous recombination during genome replication [Internet]. PLOS Genetics. 2020 ; 16( 7):[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pgen.1008828
Vancouver
Damasceno JD, Reis-Cunha J, Crouch K, Beraldi D, Lapsley C, Tosi LRO, Bartholomeu DC, McCulloch R. Conditional knockout of RAD51-related genes in Leishmania major reveals a critical role for homologous recombination during genome replication [Internet]. PLOS Genetics. 2020 ; 16( 7):[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pgen.1008828
Artigo de periodico
Genome duplication in Leishmania major relies on persistent subtelomeric DNA replication (2020)
Damasceno, Jeziel Dener; Marques, Catarina A.; Beraldi, Dario; Crouch, Kathryn; Lapsley, Craig; Obonaga, Ricardo; Tosi, Luíz Ricardo Orsini ; McCulloch, Richard
Source: eLife. Unidade: FMRP
Subjects: CROMOSSOMOS, QUÍMICA, GENÉTICA, REPLICAÇÃO DO DNA, LEISHMANIA, METABOLISMO
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DAMASCENO, Jeziel Dener et al. Genome duplication in Leishmania major relies on persistent subtelomeric DNA replication. eLife, v. 9, 2020Tradução . . Disponível em: https://doi.org/10.7554/eLife.58030. Acesso em: 02 nov. 2024.
APA
Damasceno, J. D., Marques, C. A., Beraldi, D., Crouch, K., Lapsley, C., Obonaga, R., et al. (2020). Genome duplication in Leishmania major relies on persistent subtelomeric DNA replication. eLife, 9. doi:10.7554/eLife.58030
NLM
Damasceno JD, Marques CA, Beraldi D, Crouch K, Lapsley C, Obonaga R, Tosi LRO, McCulloch R. Genome duplication in Leishmania major relies on persistent subtelomeric DNA replication [Internet]. eLife. 2020 ; 9[citado 2024 nov. 02 ] Available from: https://doi.org/10.7554/eLife.58030
Vancouver
Damasceno JD, Marques CA, Beraldi D, Crouch K, Lapsley C, Obonaga R, Tosi LRO, McCulloch R. Genome duplication in Leishmania major relies on persistent subtelomeric DNA replication [Internet]. eLife. 2020 ; 9[citado 2024 nov. 02 ] Available from: https://doi.org/10.7554/eLife.58030
Artigo de periodico
Vasopressin and v1br gene expression is increased in the hypothalamic pvn of borderline hypertensive rats (2020)
Savic, Bojana; Martin, Andrew; Mecawi, Andre Souza ; Bukumiric, Zoran; Antunes-Rodrigues, José ; Murphy, David; Sarenac, Olivera; Japundzic-Zigon, Nina
Source: Hypertension Research. Unidade: FMRP
Subjects: BARORREFLEXO, HIPERTENSÃO, RECEPTORES, VASOPRESSINAS, ESTRESSE PSICOLÓGICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SAVIC, Bojana et al. Vasopressin and v1br gene expression is increased in the hypothalamic pvn of borderline hypertensive rats. Hypertension Research, v. 43, n. 11, p. 1165-1174, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41440-020-0469-2. Acesso em: 02 nov. 2024.
APA
Savic, B., Martin, A., Mecawi, A. S., Bukumiric, Z., Antunes-Rodrigues, J., Murphy, D., et al. (2020). Vasopressin and v1br gene expression is increased in the hypothalamic pvn of borderline hypertensive rats. Hypertension Research, 43( 11), 1165-1174. doi:10.1038/s41440-020-0469-2
NLM
Savic B, Martin A, Mecawi AS, Bukumiric Z, Antunes-Rodrigues J, Murphy D, Sarenac O, Japundzic-Zigon N. Vasopressin and v1br gene expression is increased in the hypothalamic pvn of borderline hypertensive rats [Internet]. Hypertension Research. 2020 ; 43( 11): 1165-1174.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1038/s41440-020-0469-2
Vancouver
Savic B, Martin A, Mecawi AS, Bukumiric Z, Antunes-Rodrigues J, Murphy D, Sarenac O, Japundzic-Zigon N. Vasopressin and v1br gene expression is increased in the hypothalamic pvn of borderline hypertensive rats [Internet]. Hypertension Research. 2020 ; 43( 11): 1165-1174.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1038/s41440-020-0469-2
Artigo de periodico
GPCR-mediated glucose sensing system regulates light-dependent fungal development and mycotoxin production (2019)
Reis, Thaila Fernanda dos ; Maronãs, Laura Mellado ; Lohmar, Jessica M.; Silva, Lilian Pereira ; Zhou, Jing-Jiang; Calvo, Ana M.; Goldman, Gustavo Henrique ; Brown, Neil A.
Source: PLOS Genetics. Unidades: FCFRP, FMRP
Subjects: GLICOSE, ASPERGILLUS, GENÉTICA MICROBIANA, REGULAÇÃO GÊNICA, MICOTOXINAS, DIFERENCIAÇÃO SEXUAL (NÚCLEO CELULAR)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
REIS, Thaila Fernanda dos et al. GPCR-mediated glucose sensing system regulates light-dependent fungal development and mycotoxin production. PLOS Genetics, v. 15, n. 10, p. 1-27, 2019Tradução . . Disponível em: https://doi.org/10.1371/journal.pgen.1008419. Acesso em: 02 nov. 2024.
APA
Reis, T. F. dos, Maronãs, L. M., Lohmar, J. M., Silva, L. P., Zhou, J. -J., Calvo, A. M., et al. (2019). GPCR-mediated glucose sensing system regulates light-dependent fungal development and mycotoxin production. PLOS Genetics, 15( 10), 1-27. doi:10.1371/journal.pgen.1008419
NLM
Reis TF dos, Maronãs LM, Lohmar JM, Silva LP, Zhou J-J, Calvo AM, Goldman GH, Brown NA. GPCR-mediated glucose sensing system regulates light-dependent fungal development and mycotoxin production [Internet]. PLOS Genetics. 2019 ; 15( 10): 1-27.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pgen.1008419
Vancouver
Reis TF dos, Maronãs LM, Lohmar JM, Silva LP, Zhou J-J, Calvo AM, Goldman GH, Brown NA. GPCR-mediated glucose sensing system regulates light-dependent fungal development and mycotoxin production [Internet]. PLOS Genetics. 2019 ; 15( 10): 1-27.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1371/journal.pgen.1008419

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